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GENU VALGUS
Dr. Santosh Batajoo
 Genu valgum or knock knee
 Outward deviation of the longitudinal
axis of tibia.
DEFORMITY
 Medial angulation of the knee and outward
deviation of the longitudinal axis of both the tibia
and the femur.
 Advanced cases- the distal ends of the femur
and tibia are rotated externally by the pull of the
biceps femoris and the tensor fascia lata.
 Distal shaft of the tibia develops a
compensatory internal rotation
 Pronated and flat foot.
 Idiopathic variety is the commonest.
 Bilateral
 Physiological genu valgum appears at the
age of 2-3 years and nearly always corrects
by the age of 6.
PATHOPHYSIOLOGY
 The mechanical axis shifts
laterally, pathologic stress is
placed on the lateral femur and
tibia, inhibiting growth and
possibly leading to vicious cycle.
 Asymmetrical rate of growth of the
epiphyseal plate- trauma, infection.
 Static abnormality like deformed femur or the
tibia or muscle imbalance brings excessive
pressure on one end of the plate.
 Metabolic and nutritional disturbance
 Malunion of fracture at the metaphysis
 Developmental disturbances – osteogenesis
imperfecta, chondrodysplasia
 The medial condyle assumes larger proportions,
and the articular surfaces lie at an oblique
angle.
 The quadriceps extensor mechanism crosses
the joint over the lateral aspect where the
patella tends to subluxate or dislocate outward.
 Soft tissue structures on the lateral side are
shortened (biceps, iliotibial band, peroneal
nerve)
 Medial collateral ligament is lax.
TYPES
Physiological –
Compensatory-
 Varus, valgus and rotational deformities
of the proximal femur.
 Persistent anteversion of the femoral
neck
Pathological-
 Distorted epiphyseal or physeal growth.
 Rickets, injuries of the epiphyseal and
physeal growth cartilage.
In adults-
 Common in females.
 May be sequel to childhood deformity,
rheumatoid arthritis, ligament injury,
malunited fractures, paget’s disease.
 Complications-
 Patello-femoral osteoarthritis due to
abnormal tracking of the patella.
CAUSES
 Idiopathic
 Post-traumatic
fractures of the lateral femoral or tibial condyles
damage to lateral lower femoral or upper tibial
epiphyses.
 Post inflammatory
damage to lateral epiphyses by infection.
 Neoplastic
tumour causing growth disparity e.g.
chondroblastoma
 Bone softening
rickets and osteomalacia, bone dysplasias,
RA
 Stretching of joints
charcot’s disease, paralytic disease
 Cartilage thinning
OA of the lateral compartment of the knee
CLINICAL FEATURES
 Knee pain
 Gait- limp or circumduction
 Deformity of the knee.
 Intermalleolar gap- distance between the medial
malleoli when the knees are touching with the
patella facing forwards, is more than 8cm.
 Plumb line test.
 Knee flexion test to detect whether it lies in
the femur or tibia.
RADIOGRAPHS
X-ray
 Entire lower limb with the
patient weight bearing
 Angle formed between
femoral and tibial shafts
(normal 6º)
 Lateral distal femoral angle (normal - 84º)
 Proximal medial tibial angle (normal – 87º)
TREATMENT
CONSERVATIVE-
 Depends on the cause.
 vitamin D and calcium supplements
 Weight loss
 Exercise
 Pronation of the feet is corrected by elevating
the inner border of the shoes
 Knock knee braces are worn continuously
SURGICAL-
 More severe cases- intermalleolar distance
more than 10cm by the age 4.
 If the lateral portion of the
epiphyseal plate is intact-
stapling the medial portion
of the epiphyseal plate.
 If correction is achieved before growth is
complete, some overcorrection must be
attained.
Open wedge osteotomy-
 After epiphyseal fusion- an osteotomy at the site
of maximum deformity, either femur or the tibia.
 If the length of the extremity is adequate, a
wedge of bone is removed from the medial side.
 If the length is short, osteotomy from the outer
side lengthens the extremity.
THANK YOU…

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GENU VALGUS: CAUSES, SIGNS AND TREATMENT OF KNOCK KNEE

  • 2.  Genu valgum or knock knee  Outward deviation of the longitudinal axis of tibia.
  • 3. DEFORMITY  Medial angulation of the knee and outward deviation of the longitudinal axis of both the tibia and the femur.  Advanced cases- the distal ends of the femur and tibia are rotated externally by the pull of the biceps femoris and the tensor fascia lata.  Distal shaft of the tibia develops a compensatory internal rotation  Pronated and flat foot.
  • 4.  Idiopathic variety is the commonest.  Bilateral  Physiological genu valgum appears at the age of 2-3 years and nearly always corrects by the age of 6.
  • 5. PATHOPHYSIOLOGY  The mechanical axis shifts laterally, pathologic stress is placed on the lateral femur and tibia, inhibiting growth and possibly leading to vicious cycle.
  • 6.  Asymmetrical rate of growth of the epiphyseal plate- trauma, infection.  Static abnormality like deformed femur or the tibia or muscle imbalance brings excessive pressure on one end of the plate.  Metabolic and nutritional disturbance  Malunion of fracture at the metaphysis  Developmental disturbances – osteogenesis imperfecta, chondrodysplasia
  • 7.  The medial condyle assumes larger proportions, and the articular surfaces lie at an oblique angle.  The quadriceps extensor mechanism crosses the joint over the lateral aspect where the patella tends to subluxate or dislocate outward.  Soft tissue structures on the lateral side are shortened (biceps, iliotibial band, peroneal nerve)  Medial collateral ligament is lax.
  • 8. TYPES Physiological – Compensatory-  Varus, valgus and rotational deformities of the proximal femur.  Persistent anteversion of the femoral neck Pathological-  Distorted epiphyseal or physeal growth.  Rickets, injuries of the epiphyseal and physeal growth cartilage.
  • 9. In adults-  Common in females.  May be sequel to childhood deformity, rheumatoid arthritis, ligament injury, malunited fractures, paget’s disease.  Complications-  Patello-femoral osteoarthritis due to abnormal tracking of the patella.
  • 10. CAUSES  Idiopathic  Post-traumatic fractures of the lateral femoral or tibial condyles damage to lateral lower femoral or upper tibial epiphyses.  Post inflammatory damage to lateral epiphyses by infection.  Neoplastic tumour causing growth disparity e.g. chondroblastoma
  • 11.  Bone softening rickets and osteomalacia, bone dysplasias, RA  Stretching of joints charcot’s disease, paralytic disease  Cartilage thinning OA of the lateral compartment of the knee
  • 12. CLINICAL FEATURES  Knee pain  Gait- limp or circumduction  Deformity of the knee.  Intermalleolar gap- distance between the medial malleoli when the knees are touching with the patella facing forwards, is more than 8cm.
  • 13.  Plumb line test.  Knee flexion test to detect whether it lies in the femur or tibia.
  • 14. RADIOGRAPHS X-ray  Entire lower limb with the patient weight bearing  Angle formed between femoral and tibial shafts (normal 6º)
  • 15.  Lateral distal femoral angle (normal - 84º)  Proximal medial tibial angle (normal – 87º)
  • 16. TREATMENT CONSERVATIVE-  Depends on the cause.  vitamin D and calcium supplements  Weight loss  Exercise
  • 17.  Pronation of the feet is corrected by elevating the inner border of the shoes  Knock knee braces are worn continuously
  • 18. SURGICAL-  More severe cases- intermalleolar distance more than 10cm by the age 4.  If the lateral portion of the epiphyseal plate is intact- stapling the medial portion of the epiphyseal plate.  If correction is achieved before growth is complete, some overcorrection must be attained.
  • 19. Open wedge osteotomy-  After epiphyseal fusion- an osteotomy at the site of maximum deformity, either femur or the tibia.  If the length of the extremity is adequate, a wedge of bone is removed from the medial side.  If the length is short, osteotomy from the outer side lengthens the extremity.