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FLUID OVERLOAD
INTRODUCTION
PERSONAL HISTORY
FAMILY HISTORY
PAST & PRESENT MEDICAL HISTORY
Physical examination
Vital signs
• RR- 30/min
• PR- 114/min
• Temp- 97.8
• Spo2 – 92% RA
• BP – 84/60 mm Hg
Abnormal findings
Integumentary
• Pedal edema – Grade 1 – 2mm
• Skin peeling over nasal bridge
1 x 1 cms
Chest
• Mild crepitus heard over bilateral lungs
• Decreased bilateral air entry
Nutritional Assessment
CALORIE PROTEIN FAT IRON CALCIUM
TOTAL 704.8 29.64 9.1 1 363.7
RDA 2730 60 30 17 600
EXCESS
DEFICIT 2025.2 30.36 20.9 16 236.3
Investigations
Date Investigation done Patient value Inference
14/3/23 Troponin T 1.641ng/ml High ( Myocardial
Infarction)
Bicarb 13mmol/l Acidosis
Potassium 4.1meq/l Hypokalemia
Sodium 135mmol/l hyponatremia
LDL 232mg/dl High
Triglyceride 158mg/dl High
Cholesterol 283 High
HBA1C 7.6 Diabetes mellitus
15/3/23 Hb 12 Anemia
Creatinine 1.63 Increased
16/3/23 Sodium 135 Hyponatremia
Other Investigations
Date Investigations done Patient value Inference
Urea 71 Uremia
17/3/2023 Potassium 3.4 Hypokalemia
Urea 73 Uremia
18/3/23 Potassium 3.1 Hypokalemia
Other Investigations
14/3/2023
ECG : Sinus tachycardia , ST
elevation V3-V6, I avL
ECHO: moderate left ventricular
systolic dysfunction EF- 34%
RWMA+ basal and mild anterior
antero septal apical hypokinesia
Mild MR
Mild TR
Grade I diastolic dysfunction
TRANS CORONAL ANGIOGRAM +
RESCUE PTCA
Triple vessel coronary artery
disease
Chest xray :
Alveolar opacities
18/3/2023:
Extensive AWMI
Killips classification IV
LV dysfunction ( EF : 28%)
Medications
• Tab Plavix 75 mg OD
• Tab Colchicine 0.6 mg OD PO
• Stator 80mg OD PO
• Deplatt A 75 OD PO
• Inj Lasix- infusion
• Inj Noradrenaline – Infusion
• Injection piptaz 4.5 gm OD IV
• Syp Kmac
CONCEPT
ON FLUID
OVERLOAD
• Fluid volume excess
• Hyper volemia
• Isotonic expansion of the extracellular fluid
Abnormal retention of water & sodium
Pathophysiology
Related to :
• Simple fluid overload
• Diminished function of the homeostatic mechanisms – responsible for
regulating fluid balance
Contributing factors
Book picture Patient picture
Heart failure
✔
Renal failure
Cirrhosis of the liver
Consumption/Administration of excessive sodium
Clinical Manifestation
Book picture Patient picture
Edema ✔
Distended neck veins
Crackles ( abnormal lung sounds ) ✔
Tachycardia ✔
Increased blood pressure
Shortness of breath ✔
Assessment & diagnostic findings
Book picture Patient picture
Low protein intake
Anemia ✔
Hyponatremia ( excessive retention of water) ✔
Chest X ray – pulmonary congestion ✔
Edema ✔
Medical Management
Book picture Patient picture
Administering diuretics ✓
Restricting fluid & sodium ✓
Pharmacological
Therapy
Dialysis Nutritional
Therapy
Diuretics
Thiazide
diuretics
Loop
diuretics
Potassium-
sparing
diuretics
Osmotic
diuretics
Carbonic
anhydrase
inhibitors
Preventing
Hypervolemia
Sodium
restricted diet
Avoid OTC
Detecting &
controlling
Promoting
rest
Restricting
sodium
intake
Monitoring
parenteral
fluid therapy
Regular rest
periods
Drinking
water
Nursing Management
Book picture Patient picture
Intake & output ✔
Weighed daily ✔
Breath sounds Crepts
Dyspnea / orthopnea PND
Position Semi fowlers
Journal information
Theory Application
Nursing process
Ineffective airway clearance related to presence of fluid in
the plural cavity secondary to pericardial effusion
Assessed the lungs for adventitious breath sounds – crepts found in the B/L lungs
Assessed the RR, rhythm , & depth – used accessory muscles for breathing , has shallow
breathing
Monitored spo2 – 89-RA
Performed chest physio
Auscultated lungs for bilateral equal air entry
Ineffective breathing pattern related to altered oxygen supply secondary to
presence of fluid in the pericardial space & increased intra thoracic pressure
Assessed RR, depth , effort &use of accessory muscles RR=36/min
Used pulse oximeter & ABG to assess the status of the patient – respiratory acidosis
Positioned him in semi fowlers
Administered oxygen – NIV bipap – fio2 – 60%
Monitored chest x ray & ECHO findings
Ensured that o2 supply is intact
Decreased cardiac output related to increased pressure on the heart resulting
in inefficient and impaired ventricular filling & decreased ejection fraction
Assessed heart rate & BP- HR-140/mt, BP-80/70
Checked peripheral pulses – present
Auscultated for abnormal heart & breath sounds – crepts present B/L
Assessed for presence of paroxysmal nocturnal dyspnea – present
Monitored urine output – has positive balance
Activity intolerance related to imbalance between oxygen supply & demand
secondary to increased thoracic pressure leading to inadequate expansion of lungs
Assessed the patients level of physical activity & mobility – he is tired , unable
to perform any activity
Assessed cardio pulmonary status before initiating activity
Observed & documented response of activity
Used pulse oximetry to assess for o2 desaturation during activity
Fatigue related to poor physical condition , decreased
nutritional status , decreased cardiac output
Assessed the patients description of fatigue – He felt tired even on rest periods
Assessed the possible cause of fatigue – disease condition , breathing difficulty
Assessed his ability to perform ADL- he is unable to do
Assessed his emotional response to fatigue – he is very fearful
Minimized the environmental stimuli
Anxiety related to change in health status and fear of death
Assessed his level of anxiety- very anxious
Determined how he used the defense mechanism – he cries
Assured that he is safe and he is not alone – his wife was with him
Gave spiritual & psychological support
Imbalanced nutrition less than body requirement related to decreased
appetite , increased metabolic needs caused by disease process
Auscultated for bowel sounds – heard
Recognized the signs of hypoglycemia
Monitored the laboratory studies
Performed GRBS -286mg/dl
Impaired spontaneous ventilation related to non complainant lung tissue as
evidenced by adventitious breath sounds , dyspnea , apnea, decreased spo2 levels
& decreased RR rates
Monitored ABG values – respiratory acidosis
Monitored GCS periodically – 15/15
Assessed his ability to breath- unable to breathe on his own
Monitored the parameters of the ventilators – fio2 – 60%
Explained to the relatives about the future treatment plan
fluid overload.pptx
fluid overload.pptx

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fluid overload.pptx

  • 3. Physical examination Vital signs • RR- 30/min • PR- 114/min • Temp- 97.8 • Spo2 – 92% RA • BP – 84/60 mm Hg
  • 4. Abnormal findings Integumentary • Pedal edema – Grade 1 – 2mm • Skin peeling over nasal bridge 1 x 1 cms Chest • Mild crepitus heard over bilateral lungs • Decreased bilateral air entry
  • 5. Nutritional Assessment CALORIE PROTEIN FAT IRON CALCIUM TOTAL 704.8 29.64 9.1 1 363.7 RDA 2730 60 30 17 600 EXCESS DEFICIT 2025.2 30.36 20.9 16 236.3
  • 6. Investigations Date Investigation done Patient value Inference 14/3/23 Troponin T 1.641ng/ml High ( Myocardial Infarction) Bicarb 13mmol/l Acidosis Potassium 4.1meq/l Hypokalemia Sodium 135mmol/l hyponatremia LDL 232mg/dl High Triglyceride 158mg/dl High Cholesterol 283 High HBA1C 7.6 Diabetes mellitus 15/3/23 Hb 12 Anemia Creatinine 1.63 Increased 16/3/23 Sodium 135 Hyponatremia
  • 7. Other Investigations Date Investigations done Patient value Inference Urea 71 Uremia 17/3/2023 Potassium 3.4 Hypokalemia Urea 73 Uremia 18/3/23 Potassium 3.1 Hypokalemia
  • 8. Other Investigations 14/3/2023 ECG : Sinus tachycardia , ST elevation V3-V6, I avL ECHO: moderate left ventricular systolic dysfunction EF- 34% RWMA+ basal and mild anterior antero septal apical hypokinesia Mild MR Mild TR Grade I diastolic dysfunction TRANS CORONAL ANGIOGRAM + RESCUE PTCA Triple vessel coronary artery disease Chest xray : Alveolar opacities
  • 9. 18/3/2023: Extensive AWMI Killips classification IV LV dysfunction ( EF : 28%)
  • 10. Medications • Tab Plavix 75 mg OD • Tab Colchicine 0.6 mg OD PO • Stator 80mg OD PO • Deplatt A 75 OD PO • Inj Lasix- infusion • Inj Noradrenaline – Infusion • Injection piptaz 4.5 gm OD IV • Syp Kmac
  • 12. • Fluid volume excess • Hyper volemia • Isotonic expansion of the extracellular fluid Abnormal retention of water & sodium
  • 13. Pathophysiology Related to : • Simple fluid overload • Diminished function of the homeostatic mechanisms – responsible for regulating fluid balance
  • 14. Contributing factors Book picture Patient picture Heart failure ✔ Renal failure Cirrhosis of the liver Consumption/Administration of excessive sodium
  • 15. Clinical Manifestation Book picture Patient picture Edema ✔ Distended neck veins Crackles ( abnormal lung sounds ) ✔ Tachycardia ✔ Increased blood pressure Shortness of breath ✔
  • 16. Assessment & diagnostic findings Book picture Patient picture Low protein intake Anemia ✔ Hyponatremia ( excessive retention of water) ✔ Chest X ray – pulmonary congestion ✔ Edema ✔
  • 17. Medical Management Book picture Patient picture Administering diuretics ✓ Restricting fluid & sodium ✓
  • 22. Nursing Management Book picture Patient picture Intake & output ✔ Weighed daily ✔ Breath sounds Crepts Dyspnea / orthopnea PND Position Semi fowlers
  • 24.
  • 27. Ineffective airway clearance related to presence of fluid in the plural cavity secondary to pericardial effusion Assessed the lungs for adventitious breath sounds – crepts found in the B/L lungs Assessed the RR, rhythm , & depth – used accessory muscles for breathing , has shallow breathing Monitored spo2 – 89-RA Performed chest physio Auscultated lungs for bilateral equal air entry
  • 28. Ineffective breathing pattern related to altered oxygen supply secondary to presence of fluid in the pericardial space & increased intra thoracic pressure Assessed RR, depth , effort &use of accessory muscles RR=36/min Used pulse oximeter & ABG to assess the status of the patient – respiratory acidosis Positioned him in semi fowlers Administered oxygen – NIV bipap – fio2 – 60% Monitored chest x ray & ECHO findings Ensured that o2 supply is intact
  • 29. Decreased cardiac output related to increased pressure on the heart resulting in inefficient and impaired ventricular filling & decreased ejection fraction Assessed heart rate & BP- HR-140/mt, BP-80/70 Checked peripheral pulses – present Auscultated for abnormal heart & breath sounds – crepts present B/L Assessed for presence of paroxysmal nocturnal dyspnea – present Monitored urine output – has positive balance
  • 30. Activity intolerance related to imbalance between oxygen supply & demand secondary to increased thoracic pressure leading to inadequate expansion of lungs Assessed the patients level of physical activity & mobility – he is tired , unable to perform any activity Assessed cardio pulmonary status before initiating activity Observed & documented response of activity Used pulse oximetry to assess for o2 desaturation during activity
  • 31. Fatigue related to poor physical condition , decreased nutritional status , decreased cardiac output Assessed the patients description of fatigue – He felt tired even on rest periods Assessed the possible cause of fatigue – disease condition , breathing difficulty Assessed his ability to perform ADL- he is unable to do Assessed his emotional response to fatigue – he is very fearful Minimized the environmental stimuli
  • 32. Anxiety related to change in health status and fear of death Assessed his level of anxiety- very anxious Determined how he used the defense mechanism – he cries Assured that he is safe and he is not alone – his wife was with him Gave spiritual & psychological support
  • 33. Imbalanced nutrition less than body requirement related to decreased appetite , increased metabolic needs caused by disease process Auscultated for bowel sounds – heard Recognized the signs of hypoglycemia Monitored the laboratory studies Performed GRBS -286mg/dl
  • 34. Impaired spontaneous ventilation related to non complainant lung tissue as evidenced by adventitious breath sounds , dyspnea , apnea, decreased spo2 levels & decreased RR rates Monitored ABG values – respiratory acidosis Monitored GCS periodically – 15/15 Assessed his ability to breath- unable to breathe on his own Monitored the parameters of the ventilators – fio2 – 60% Explained to the relatives about the future treatment plan