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Developmental Disturbances
of Oral & Paraoral structures-2
INDIAN DENTAL ACADEMY
Leader in continuing Dental Education
www.indiandentalacademy.com
OBJECTIVES
 At the end of the lecture student should be able to understand
the
– Developmental disturbances associated with:
•Buccal mucosa
•Gingiva
•Salivary gland
www.indiandentalacademy.com
Developmental Disturbances
BUCCAL MUCOSA
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Fordyce’s Granules
• Heterotopic collection of sebaceous glands
• Due to inclusion of ectoderm having some potentialities
of skin
• Small yellow spots
• Bilaterally symmetrical
• Commonly involved areas- Buccal mucosa, lips, tongue,
gingiva, frenum
• Adults > Children
• Males=Females
www.indiandentalacademy.com
www.indiandentalacademy.com
Histopathology
• Resembles normal
sebaceous glands
• A hair shaft may be
present
• Few to many lobules
• Grouped around ducts
opening onto surface
• Keratin plugging
Treatment
• No treatment required
• Benign sebaceous
adenoma may develop
sebaceous glands
www.indiandentalacademy.com
Focal Epithelial hyperplasia
Heck’s Disease
• Multiple nodular lesion with sessile base
• Common seen on lips, buccal mucosa, commisure,
tongue,
• 1-5 mm in diameter
• Same color as adjacent mucosa
• Seen commonly in children 3-18 yrs
• Undergo spontaneous regression in 4-6 months
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www.indiandentalacademy.com
• Histopathology
– Nodular elevations
– Acanthosis
– Fusion of rete pegs
– Mild hyperkeratosis
– Clumps of lymphocytes,
PMN’s
– Liquefaction degeneration
of basal layer
Treatment
– No treatment required
Acanthosis
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Developmental Disturbances
GINGIVA
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Fibromatosis Gingivae
• Dense, diffuse, smooth, nodular overgrowth of gingival tissues
of one or both arches
• Appears at the time of eruption of incisors
• Normal color, not painful, non hemorrhagic
• H/F
– Thick epithelium
– Elongated rete pegs
– Dense fibrous connective tissue
– Few interspersed fibroblasts
– Inflammation may or may not be present
• Treatment
– Surgical resection
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Narrow,slender elongated rete pegs
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Retrocuspid papillae
• Small elevated nodule on lingual mucosa of mandibular
cuspids
• Sessile, bilateral, regresses with age
• Lies between free gingival margin and mucogingival
junction
• Hyperparakertatinized/ Hyperorthokeratinized
• No treatment required
www.indiandentalacademy.com
Developmental Disturbances
Salivary Glands
DEPARTMENT OF ORAL PATHOLOGY AND MICROBIOOGY
SHARAD PAWAR DENTAL COLLEGE
www.indiandentalacademy.com
• Aplasia
– Congenital absence of salivary glands
– Xerostomia – Dry mouth
– Oral mucosa appears dry, smooth, pebbly
– Lips are crackled and fissures at corner of mouth
– Rampant caries and early loss of teeth
– No specific treatment
www.indiandentalacademy.com
• Atresia
– Congenital occlusion or absence of salivary gland duct
– Retention cysts
– Severe Xerostomia
• Aberrancy
– Presence of salivary glands in abnormal locations
– Lead to formation of retention cyst
– Neoplasm like Central mucoeidermoid carcinoma
www.indiandentalacademy.com
Xerostomia
• Dry or burning sensation of mucosa
• Mucosa may become atrophic, inflamed, pale & translucent
• Atrophy of lingual papillae
• Fissuring, cracking, areas of denudation
• Rampant caries
• Lack of tolerance of appliances
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Etiology
• Temporary / Transient xerostomia due to emotional reaction,
blockage of ducts, drugs,
• Salivary gland aplasia
• X Ray radiation
• Vitamin deficiency
• Sjogren’s syndrome
• Miscellaneous causes
• Treatment.-
• Correction of cause
• Saliva substitutes
www.indiandentalacademy.com
Hyperplasia of Palatal Glands
• Hyperplasia of minor salivary glands
• Etiology
– Endocrinal disorders, Gout, Diabetes mellitus, menopause, hepatic
disease, starvation, alcoholism, inflammation, benign
lymphoepithelial lesion, sjogren’s syndrome, aglossia-adactylia
syndrome, uveoparotid fever etc.
www.indiandentalacademy.com
• C/F
– Small localized swelling, usually on hard palate or junction of
hard and soft palate,
– Intact surface, sessile, normal color, asymptomatic
• H/F
– Closely packed collection of normal mucous acini, normal ducts,
• Treatment
– Surgical excision
www.indiandentalacademy.com
STAFNE CYST
• Due to inclusion of glandular tissue adjacent to lingual surface
of the body of mandible
• R/F
– Ovoid radiolucency between mandibular canal and inferior
border of mandible in the molar region
– Should be differentiated from traumatic bone cyst
www.indiandentalacademy.com
Anterior lingual depression
– Poorly defined radiolucency in anterior region between central
incisor and premolar
– Due to sublingual gland
www.indiandentalacademy.com
www.indiandentalacademy.com
SUMMARY
 By the end of the lecture student should be able to conclude the
– Developmental disturbances associated with:
•Buccal mucosa
•Gingiva
• Salivary gland
www.indiandentalacademy.com
www.indiandentalacademy.com
www.indiandentalacademy.com
www.indiandentalacademy.com

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Developmental Disturbances of Oral & Paraoral structures/ dental crown & bridge courses

  • 1. Developmental Disturbances of Oral & Paraoral structures-2 INDIAN DENTAL ACADEMY Leader in continuing Dental Education www.indiandentalacademy.com
  • 2. OBJECTIVES  At the end of the lecture student should be able to understand the – Developmental disturbances associated with: •Buccal mucosa •Gingiva •Salivary gland www.indiandentalacademy.com
  • 4. Fordyce’s Granules • Heterotopic collection of sebaceous glands • Due to inclusion of ectoderm having some potentialities of skin • Small yellow spots • Bilaterally symmetrical • Commonly involved areas- Buccal mucosa, lips, tongue, gingiva, frenum • Adults > Children • Males=Females www.indiandentalacademy.com
  • 6. Histopathology • Resembles normal sebaceous glands • A hair shaft may be present • Few to many lobules • Grouped around ducts opening onto surface • Keratin plugging Treatment • No treatment required • Benign sebaceous adenoma may develop sebaceous glands www.indiandentalacademy.com
  • 7. Focal Epithelial hyperplasia Heck’s Disease • Multiple nodular lesion with sessile base • Common seen on lips, buccal mucosa, commisure, tongue, • 1-5 mm in diameter • Same color as adjacent mucosa • Seen commonly in children 3-18 yrs • Undergo spontaneous regression in 4-6 months www.indiandentalacademy.com
  • 9. • Histopathology – Nodular elevations – Acanthosis – Fusion of rete pegs – Mild hyperkeratosis – Clumps of lymphocytes, PMN’s – Liquefaction degeneration of basal layer Treatment – No treatment required Acanthosis www.indiandentalacademy.com
  • 11. Fibromatosis Gingivae • Dense, diffuse, smooth, nodular overgrowth of gingival tissues of one or both arches • Appears at the time of eruption of incisors • Normal color, not painful, non hemorrhagic • H/F – Thick epithelium – Elongated rete pegs – Dense fibrous connective tissue – Few interspersed fibroblasts – Inflammation may or may not be present • Treatment – Surgical resection www.indiandentalacademy.com
  • 12. Narrow,slender elongated rete pegs www.indiandentalacademy.com
  • 13. Retrocuspid papillae • Small elevated nodule on lingual mucosa of mandibular cuspids • Sessile, bilateral, regresses with age • Lies between free gingival margin and mucogingival junction • Hyperparakertatinized/ Hyperorthokeratinized • No treatment required www.indiandentalacademy.com
  • 14. Developmental Disturbances Salivary Glands DEPARTMENT OF ORAL PATHOLOGY AND MICROBIOOGY SHARAD PAWAR DENTAL COLLEGE www.indiandentalacademy.com
  • 15. • Aplasia – Congenital absence of salivary glands – Xerostomia – Dry mouth – Oral mucosa appears dry, smooth, pebbly – Lips are crackled and fissures at corner of mouth – Rampant caries and early loss of teeth – No specific treatment www.indiandentalacademy.com
  • 16. • Atresia – Congenital occlusion or absence of salivary gland duct – Retention cysts – Severe Xerostomia • Aberrancy – Presence of salivary glands in abnormal locations – Lead to formation of retention cyst – Neoplasm like Central mucoeidermoid carcinoma www.indiandentalacademy.com
  • 17. Xerostomia • Dry or burning sensation of mucosa • Mucosa may become atrophic, inflamed, pale & translucent • Atrophy of lingual papillae • Fissuring, cracking, areas of denudation • Rampant caries • Lack of tolerance of appliances www.indiandentalacademy.com
  • 18. Etiology • Temporary / Transient xerostomia due to emotional reaction, blockage of ducts, drugs, • Salivary gland aplasia • X Ray radiation • Vitamin deficiency • Sjogren’s syndrome • Miscellaneous causes • Treatment.- • Correction of cause • Saliva substitutes www.indiandentalacademy.com
  • 19. Hyperplasia of Palatal Glands • Hyperplasia of minor salivary glands • Etiology – Endocrinal disorders, Gout, Diabetes mellitus, menopause, hepatic disease, starvation, alcoholism, inflammation, benign lymphoepithelial lesion, sjogren’s syndrome, aglossia-adactylia syndrome, uveoparotid fever etc. www.indiandentalacademy.com
  • 20. • C/F – Small localized swelling, usually on hard palate or junction of hard and soft palate, – Intact surface, sessile, normal color, asymptomatic • H/F – Closely packed collection of normal mucous acini, normal ducts, • Treatment – Surgical excision www.indiandentalacademy.com
  • 21. STAFNE CYST • Due to inclusion of glandular tissue adjacent to lingual surface of the body of mandible • R/F – Ovoid radiolucency between mandibular canal and inferior border of mandible in the molar region – Should be differentiated from traumatic bone cyst www.indiandentalacademy.com
  • 22. Anterior lingual depression – Poorly defined radiolucency in anterior region between central incisor and premolar – Due to sublingual gland www.indiandentalacademy.com
  • 24. SUMMARY  By the end of the lecture student should be able to conclude the – Developmental disturbances associated with: •Buccal mucosa •Gingiva • Salivary gland www.indiandentalacademy.com