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Dr Jaffar Raza Syed
SmokingSmoking & PDD
Page 1
Dr Jaffar Raza Syed Page 2
Dr Jaffar Raza Syed Page 3
Microbiology
--Smokers had higher level of
B. forsythus.
A. actinomycetemcomitans
P. gingivalis
Remaining in the pocket after therapy when compared to nonsmokers.
--Fusobacterium nucleatum,
--S.vincentii,
--P. gingivalis,
--P. intermedia,
--Peptostreptococcus micros,
--Prevotella nigrescens,
--B. forsythus
were significantly more prevalent in current smokers than in nonsmokers and
former smokers
Dr Jaffar Raza Syed Page 4
Immunology
• Altered neutrophil chemotaxis, phagocytosis and oxidative burst.
• Increased TNF- α, and PGE2 in GCF.
• Increased production of PGE2 by monocyte in response to LPS.
• IgG2 level is reduced suggesting reduced protection against
periodontal infection.
• Nicotine, a major component of tobacco adversely affect
fibroblast function.
• Tobacco products alter normal reparative and regeneration potential
of periodontium.
Dr Jaffar Raza Syed
Physiology
• Reduced signs of inflamation
• Decreased gingival vessels
• Decreased GCF flow and bleeding on probing
• Decreased subgingival temperature
• Increased time needed to recover from
inflamation due to alter inflamatory response
gingival vessels with increased inflammation.
bleeding on probing with increased inflammation
temperature.
• Increased time needed to recover from local anesthesia.
Page 5
due to alter inflamatory response
increased inflammation
Dr Jaffar Raza Syed Page 6
Dr Jaffar Raza Syed Page 7
Effect Of Smoking On The Response To Periodontal Therapy
Non-surgical
• Decreased clinical response to scaling and root planing
• Decreased reduction in pocket depth
• Decreased gain in clinical attachment level.
• Decreased negative impact of smoking with increased level of plaque control.
Dr Jaffar Raza Syed Page 8
Surgery and implants
• Decreased pocket depth reduction post surgery.
• Increased deterioration of furcation post surgery.
• Decreased gain in clinical attachment level, decreased bone fill,
increased recession and increased membrane exposure following
guided tissue regeneration (GTR).
• Decreased pocket depth reduction after DFDBA allograft.
• Decreased pocket depth reduction and gain in clinical attachment level
after open flap debridement.
• Conflicting data on the impact of smoking on implant success.
• Smoking cessation should be recommended prior to implant.
Dr Jaffar Raza Syed Page 9
Maintenance
• Increased pocket depth during maintenance.
• Decreased gain in clinical attachment level.
Recurrent (refractory) Disease
• Increased recurrent/refractory disease in smokers.
• Increased need for retreatment in smokers.
• Increased need for antibiotics in smokers to control the negative effect
of periodontal infection onsurgical outcome.
• Increased tooth loss in smokers after surgical therapy.

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016.smoking and periodontal diseases

  • 1. Dr Jaffar Raza Syed SmokingSmoking & PDD Page 1
  • 2. Dr Jaffar Raza Syed Page 2
  • 3. Dr Jaffar Raza Syed Page 3 Microbiology --Smokers had higher level of B. forsythus. A. actinomycetemcomitans P. gingivalis Remaining in the pocket after therapy when compared to nonsmokers. --Fusobacterium nucleatum, --S.vincentii, --P. gingivalis, --P. intermedia, --Peptostreptococcus micros, --Prevotella nigrescens, --B. forsythus were significantly more prevalent in current smokers than in nonsmokers and former smokers
  • 4. Dr Jaffar Raza Syed Page 4 Immunology • Altered neutrophil chemotaxis, phagocytosis and oxidative burst. • Increased TNF- α, and PGE2 in GCF. • Increased production of PGE2 by monocyte in response to LPS. • IgG2 level is reduced suggesting reduced protection against periodontal infection. • Nicotine, a major component of tobacco adversely affect fibroblast function. • Tobacco products alter normal reparative and regeneration potential of periodontium.
  • 5. Dr Jaffar Raza Syed Physiology • Reduced signs of inflamation • Decreased gingival vessels • Decreased GCF flow and bleeding on probing • Decreased subgingival temperature • Increased time needed to recover from inflamation due to alter inflamatory response gingival vessels with increased inflammation. bleeding on probing with increased inflammation temperature. • Increased time needed to recover from local anesthesia. Page 5 due to alter inflamatory response increased inflammation
  • 6. Dr Jaffar Raza Syed Page 6
  • 7. Dr Jaffar Raza Syed Page 7 Effect Of Smoking On The Response To Periodontal Therapy Non-surgical • Decreased clinical response to scaling and root planing • Decreased reduction in pocket depth • Decreased gain in clinical attachment level. • Decreased negative impact of smoking with increased level of plaque control.
  • 8. Dr Jaffar Raza Syed Page 8 Surgery and implants • Decreased pocket depth reduction post surgery. • Increased deterioration of furcation post surgery. • Decreased gain in clinical attachment level, decreased bone fill, increased recession and increased membrane exposure following guided tissue regeneration (GTR). • Decreased pocket depth reduction after DFDBA allograft. • Decreased pocket depth reduction and gain in clinical attachment level after open flap debridement. • Conflicting data on the impact of smoking on implant success. • Smoking cessation should be recommended prior to implant.
  • 9. Dr Jaffar Raza Syed Page 9 Maintenance • Increased pocket depth during maintenance. • Decreased gain in clinical attachment level. Recurrent (refractory) Disease • Increased recurrent/refractory disease in smokers. • Increased need for retreatment in smokers. • Increased need for antibiotics in smokers to control the negative effect of periodontal infection onsurgical outcome. • Increased tooth loss in smokers after surgical therapy.