1. The document describes different types of myocardial infarctions (MIs) that can occur in different regions of the heart.
2. It provides details on the causes, risk factors, and characteristic ECG findings for inferior wall MIs, anterior wall MIs, posterior wall MIs, anteroseptal MIs, lateral wall MIs, and anterolateral MIs.
3. The risk factors described that can lead to MIs include high blood pressure, stress, family history of heart disease, drug usage, and physical inactivity.
2. ɪɴꜰᴇʀɪᴏʀ ᴡᴀʟʟ ᴍɪ
• An inferior wall mi also known as lWMI occurs
when inferior myocardial tissue supplied by the
right coronary artery or RCA is injured due to
thrombosis of that vessel.
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The ecg findings of acute inferior myocardial
infarction include the following ;
1.ST seg elevation in the inferior leads (II,III,and
aVF)
2.Reciprocal ST segment depression in the lateral
leads (I , aVL ,v5 and V6 )
3.progressive development of Q wave in II III aVF.
4.Hyperacute ( peaked ) T wave in II III aVF with
relative loss of r wave height
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5. ᴀɴᴛᴇʀɪᴏʀ ᴡᴀʟʟ ᴍɪ
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An anterior wall myocardial infarction occurs
when anterior myocardial tissue usually by the
Left Anterior Descending coronary artery
suffers injury due to lack of blood supply.
When an AWMI extends to the septal and
lateral regions as well , the culprit lession is
usually more proximal in the LAD or even in the
Left Main Coronary artery.
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ECG findings
1. ST seg elevation in the anterior leads (v3 and
v4) at the J point and sometimes in the septal
or lateral leads .This ST seg elevation is
concave and frequently overwhelms the T
wave This is called 'tomb stoning ' for obvious
reason the shape is similar to tombstone
2.reciprocal ST depression in the Inferior leads
II III aVF
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8. ᴩᴏꜱᴛᴇʀɪᴏʀ ᴡᴀʟʟ ᴍɪ
• A posterior wall mi occurs when the posterior
myocardial tissue usually supplied by the
posterior descending artery ( A branch of RCA )
actually loses blood supply due to intracoronay
thrombus in that vessel .This frequently
coincides with an inferior wall mi due to shared
blood supply
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Ecg findings
ST segment depression in the septal and anterior
precordial leads (v1 - v4).this is because the ECG leads
will see the MI backwards ,the leads are replaced
anteriorly but the injury is posterior.
2.chance of ST seg elevation in inferior leads (II III aVF )
in an inferior MI present
3.dominant r wave (r/s ratio >1) in v2
4.tall broad R Wave (>30ms)
5.upright T wave
6.horizontal ST depression.
10. ᴀɴᴛᴇʀᴏꜱᴇᴩᴛᴀʟ ᴍɪ
• Anteroseptal mi are commonly caused by
rupture of an unstable atherosclerotic plaque in
the left anterior descending artery or Diagonal
branch.
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ECG finding
1. ST elevation and hyperacute T wave in v2 -
v4
2.ST elevation in I and aVL with reciprocal ST
depression in lead III q waves are represent in
the septal leads v1 - v2
12. ʟᴀᴛᴇʀᴀʟᴡᴀʟʟ ᴍɪ
• Infarction of the lateral wall usually occurs as a
part of a larger teritery infarction. Isolated
lateral STEMI is less common,but may be
produced by occlusion of smaller branch
arteries that supplies the lateral wall eg:-first
diagonal branch(D1)of LAD,OM of the LCX or
the ramus intermedias
13. Ecg changes
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ST elevation in v5 v6 1&aVL
Reciprocal ST depression in III &aVF
ST elevation primarly localised to lead 1& aVL
is referred to as high lateral MI
14. ᴀɴᴛᴇʀᴏ ʟᴀᴛᴇʀᴀʟ ᴍɪ
• Anterolateral infarcts results from the
occlusion of the LMCA and changes appear in
leads I aVL and V2 - v6 . Anterolateral mi
frequently are caused by occlusion of the LAD
together with RCA or LCX
15. ʀɪꜱᴋ ꜰᴀᴄᴛᴏʀꜱ ᴛʜᴀᴛ ʟᴇᴀᴅꜱ ᴛᴏ ᴍɪ
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1.high blood pressure can damage your arteries and
also accelerate the buildup of plaque
2.Stress: stress may also be a trigger for acute
myocardial. Infarction
3.Family history : you are more likely to have a heart
attack if you have a family history of early heart disease
4.Drug usage
5.physical inactivity ; getting exercise done regularly
helps you to maintain moderate weight as well as
reduce your blood pressure , cholesterol and blood sugar
level