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DR JOEL ARUDCHELVAM
CONSULTANT VASCULAR AND TRANSPLANT
SURGEON
 Dilated tortuous superficial veins (derived
from the Greek word "varix," - “grapelike”)
 Old disease
 Hippocrates and Galen described the disease
 Prevalence of varicose veins - 2% to over 60% in
population studies
 Recurrence rate of 20%-60% after 5 years
 Epidemiology of chronic venous disease.Robertson L, Evans C, Fowkes FG Phlebology. 2008; 23(3):103-11
 Allaf N, Welch M. Recurrent varicose veins: Inadequate surgery remains a problem. Phlebology. 2005;20:138–
40.
Venous anatomy
Named perforators along
Greater saphenous
distribution
New perforator vein terminology
 Inadequate initial procedure
 Not stripping
 Neo vascularisation -new veins appearing in the granulation
tissue connecting the end of the ligated sapheno-femoral
junction (SFJ)
 Recanalisation – after thermal, chemical ablation
 New source of reflux
 Accessory Long Saphenous Vein (LSV)
 Accessory Short Saphenous Vein (SSV)
 Deep venous disease
 Inadequate initial procedure
 Not stripping
 Neo vascularisation -new veins appearing in the
granulation tissue connecting the end of the SFJ
 Recanalisation – after thermal, chemical ablation
 New source of reflux
 Accessory Long Saphenous Vein (LSV)
 Accessory Short Saphenous Vein (SSV)
 Deep venous disease
 Inadequate initial procedure
 Not stripping
 Neo vascularisation -new veins appearing in the
granulation tissue connecting the end of the SFJ
 Recanalisation – after thermal, chemical ablation
 New source of reflux
 Accessory Long Saphenous Vein (LSV)
 Accessory Short Saphenous Vein (SSV)
 Deep venous disease
 C0: No Varicose Veins
 C1: Telangiectasia ( reticular veins , spider veins)
 C2: Varicose veins
 C3: Edema
 C4: Skin changes
 C4a: pigmentation and eczema
 C4b: lipodermatosclerosis and atrophie blanche
 C5: Healed venous ulcer
 C6: Active venous ulcer
 C0: No Varicose Veins
 C1: Telangiectasia ( reticular veins , spider veins)
 C2: Varicose veins
 C3: Edema
 C4: Skin changes
 C4a: pigmentation and eczema
 C4b: lipodermatosclerosis and atrophie blanche
 C5: Healed venous ulcer
 C6: Active venous ulcer
 C0: No Varicose Veins
 C1: Telangiectasia ( reticular veins , spider veins)
 C2: Varicose veins
 C3: Edema
 C4: Skin changes
 C4a: pigmentation and eczema
 C4b: lipodermatosclerosis and atrophie blanche
 C5: Healed venous ulcer
 C6: Active venous ulcer
 To identify the cause of recurrence
 SFJ incompetence (SFI), Sapheno poplieal junction
incompetence (SPI)
 Acc LSV, Acc SSV
 DVT
 Perforators / site
 Neo vascularisation
 COSMETIC
 SYMPTOMATIC
 COMPLICATED
 Oedema
 C4 – skin changes
 C4a: pigmentation and eczema.
 C4b: lipodermatosclerosis and atrophie blanche.
 C5: healed venous ulcer.
 C6: active venous ulcer
 Surgery
 Thermal Ablation ( Radio frequncy ablation
/LASER )
 Sclerotherapy
 Dangers
 Risk of injury to femoral vein
 Lymphatic leak
 Expose artery first and approach vein from
lateral side
• Energy source
• Gain medium
• Resonant cavity
• LASER
• Monochromatic – same wave
length
• Coherent – unidirectional
• Collimated - parallel
Laser energy is absorbed
by vein wall and
hemoglobin producing heat
and vein wall destruction
 Scleroscents used
 SODIUM TETRADECYL SULPHATE(STD)
 HYPERTONIC SALINE SOL
 POLYDOCANOL
 SOTRADECOL
 ETHANOLAMINE OLEATE
 GLUCOSE COMBINATIONS
• sclerosant is taken in 20 ml
syringe ,another syringe with
4 times the amount of air
• By repeated to and fro motion
,dense white foam prepared
 Mechanism of action
 Endothelial damage
 Inflammation
 obliteration
 Recurrent SFJ /SFI
 Surgical,Laser
 neovascularisation
 Sclerotherapy, surgery
 LSV
 Thermal ablation (LASER, RFA), Stripping
 Varicosities
 Sclerotherapy, Avulsion, thermal ablation (LASER,
RFA)
 Pelvic DVT - Venous Angioplasty
 Duplex scanning and identifying the right
source - accessory LSV, (Giacomini vein),
correct site of reflux of SSV. Ect
 Stripping of Long Saphenous Vein
 Duplex scanning and Avoiding varicose vein
intervention in patients with past DVT
 Duplex scanning and identifying the right
source - acc LSV, (Giacomini vein), correct site
of reflux of SSV. Ect
 Stripping of Long Saphenous Vein , Ligation of
tributaries
 Duplex scanning and Avoiding varicose vein
intervention in patients with past DVT
Thank You

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Recurrent varicose veins and its management

  • 1. DR JOEL ARUDCHELVAM CONSULTANT VASCULAR AND TRANSPLANT SURGEON
  • 2.  Dilated tortuous superficial veins (derived from the Greek word "varix," - “grapelike”)  Old disease  Hippocrates and Galen described the disease
  • 3.  Prevalence of varicose veins - 2% to over 60% in population studies  Recurrence rate of 20%-60% after 5 years  Epidemiology of chronic venous disease.Robertson L, Evans C, Fowkes FG Phlebology. 2008; 23(3):103-11  Allaf N, Welch M. Recurrent varicose veins: Inadequate surgery remains a problem. Phlebology. 2005;20:138– 40.
  • 4.
  • 5. Venous anatomy Named perforators along Greater saphenous distribution
  • 6. New perforator vein terminology
  • 7.  Inadequate initial procedure  Not stripping  Neo vascularisation -new veins appearing in the granulation tissue connecting the end of the ligated sapheno-femoral junction (SFJ)  Recanalisation – after thermal, chemical ablation  New source of reflux  Accessory Long Saphenous Vein (LSV)  Accessory Short Saphenous Vein (SSV)  Deep venous disease
  • 8.
  • 9.  Inadequate initial procedure  Not stripping  Neo vascularisation -new veins appearing in the granulation tissue connecting the end of the SFJ  Recanalisation – after thermal, chemical ablation  New source of reflux  Accessory Long Saphenous Vein (LSV)  Accessory Short Saphenous Vein (SSV)  Deep venous disease
  • 10.
  • 11.  Inadequate initial procedure  Not stripping  Neo vascularisation -new veins appearing in the granulation tissue connecting the end of the SFJ  Recanalisation – after thermal, chemical ablation  New source of reflux  Accessory Long Saphenous Vein (LSV)  Accessory Short Saphenous Vein (SSV)  Deep venous disease
  • 12.
  • 13.  C0: No Varicose Veins  C1: Telangiectasia ( reticular veins , spider veins)  C2: Varicose veins  C3: Edema  C4: Skin changes  C4a: pigmentation and eczema  C4b: lipodermatosclerosis and atrophie blanche  C5: Healed venous ulcer  C6: Active venous ulcer
  • 14.
  • 15.  C0: No Varicose Veins  C1: Telangiectasia ( reticular veins , spider veins)  C2: Varicose veins  C3: Edema  C4: Skin changes  C4a: pigmentation and eczema  C4b: lipodermatosclerosis and atrophie blanche  C5: Healed venous ulcer  C6: Active venous ulcer
  • 16.
  • 17.  C0: No Varicose Veins  C1: Telangiectasia ( reticular veins , spider veins)  C2: Varicose veins  C3: Edema  C4: Skin changes  C4a: pigmentation and eczema  C4b: lipodermatosclerosis and atrophie blanche  C5: Healed venous ulcer  C6: Active venous ulcer
  • 18.
  • 19.
  • 20.  To identify the cause of recurrence  SFJ incompetence (SFI), Sapheno poplieal junction incompetence (SPI)  Acc LSV, Acc SSV  DVT  Perforators / site  Neo vascularisation
  • 21.  COSMETIC  SYMPTOMATIC  COMPLICATED  Oedema  C4 – skin changes  C4a: pigmentation and eczema.  C4b: lipodermatosclerosis and atrophie blanche.  C5: healed venous ulcer.  C6: active venous ulcer
  • 22.  Surgery  Thermal Ablation ( Radio frequncy ablation /LASER )  Sclerotherapy
  • 23.  Dangers  Risk of injury to femoral vein  Lymphatic leak  Expose artery first and approach vein from lateral side
  • 24. • Energy source • Gain medium • Resonant cavity • LASER • Monochromatic – same wave length • Coherent – unidirectional • Collimated - parallel
  • 25.
  • 26. Laser energy is absorbed by vein wall and hemoglobin producing heat and vein wall destruction
  • 27.  Scleroscents used  SODIUM TETRADECYL SULPHATE(STD)  HYPERTONIC SALINE SOL  POLYDOCANOL  SOTRADECOL  ETHANOLAMINE OLEATE  GLUCOSE COMBINATIONS
  • 28. • sclerosant is taken in 20 ml syringe ,another syringe with 4 times the amount of air • By repeated to and fro motion ,dense white foam prepared
  • 29.  Mechanism of action  Endothelial damage  Inflammation  obliteration
  • 30.  Recurrent SFJ /SFI  Surgical,Laser  neovascularisation  Sclerotherapy, surgery  LSV  Thermal ablation (LASER, RFA), Stripping  Varicosities  Sclerotherapy, Avulsion, thermal ablation (LASER, RFA)  Pelvic DVT - Venous Angioplasty
  • 31.  Duplex scanning and identifying the right source - accessory LSV, (Giacomini vein), correct site of reflux of SSV. Ect  Stripping of Long Saphenous Vein  Duplex scanning and Avoiding varicose vein intervention in patients with past DVT
  • 32.
  • 33.  Duplex scanning and identifying the right source - acc LSV, (Giacomini vein), correct site of reflux of SSV. Ect  Stripping of Long Saphenous Vein , Ligation of tributaries  Duplex scanning and Avoiding varicose vein intervention in patients with past DVT