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CONTENTS
❑ Anatomy of Orbit
❑ Classification of fracture
❑ Pathophysiology
❑ Blow In fracture
❑ Blow Out Fracture – Pathophysiology & Clinical features.
❑ Superior Orbital Fissure Syndrome - Pathophysiology &
Clinical features.
❑ CLINICAL EVALUATION – Forced Duction Test
❑ Management
❑ Reconstruction
❑ Complications
Anatomy of Orbit
❑ Orbit – conical cavity
❑ Base – Anteriorly
❑ Apex – Directed at Optic Foramen.
❑ Orbital Volume – 30cc ; Globe – 7cc
❑ Bones – 7 (Maxillary, Zygomatic, Frontal, Ethmoid,
Lacrimal, Palatine, and Sphenoid )
❑ Four Walls – roof, lateral, medial, floor
❑ Medial wall & Floor – Thin
❑ Lateral wall & Roof – Stronger.
❑ Floor weakened – due to – Infraorbital canal passing
through it.
❑ Medial wall – Formed by – Lamina Papyracea of Ethmoid bone
& Lacrimal bone.
❑ 6 EXTRAOCULAR MUSCLES –
❑ Originate from Common Tendinous fibrous ring
❑ Annulus of Zinn – Fibrous ring – Common origin of 4Recti Muscle
❑ OPTIC NERVE – direct extension of Gray Matter
❑ Very sensitive to Compression and once damaged fails to regenerate.
❑ SUPERIOR ORBITAL FISSURE
❑ Transmits – 3rd , 4th & 6th Cranial Nerves
❑ Opthalmic Division of 5th Cranial Nerve
Anatomy of Orbit
Anatomy of Bony Orbit
Floor Of Orbit
❑ Extends – Rim to approx.
2/3rd of the depth of orbit.
❑ Posteromedial aspect of Orbital
floor transits into Medial Orbital Wall
– to form
post. medial bulge.
❑ Only three of four orbital wall extend
into the apex – Medial, Superior,
Lateral.
❑ Ant. 3rd – Diameter widens
behind Superior Inferior margins
‘Post Entry Concavity’ –
Saggital plane – ‘Lazy S’ shape.
Peterson’s Principles of Oral and Maxillofacial Surgery ; 2nd Edition ; Page 463- 464
Surface Landmarks –
❑ Infraorbital Groove –
bony sulcus
❑ Inferior Orbital Fissure
–converts into canal –
continues to Infraorbital
Foramen.
❑ Origin Of Inferior
Oblique Muscle
❑ Posterior Lateral
Promontry – Posterior
medial bulge ending in a
raised promontory shaped
lateral plateau.
Floor Of Orbit
Medial wall
❑ Convex Rectangular shape
❑ Runs parallel to saggital
plane
❑ Medial wall – paper thin
Lamina paprycea
– (0.2-0.4mm)
Reinforced along the Maxillary-
Ethmoidal suture --- forming an
Internal Orbital Buttress.
Peterson’s Principles of Oral and Maxillofacial Surgery ; 2nd Edition ; Page –465
Surface Landmarks
❖ Lacrimal Fossa –Anterior 1/3rd of MedialWall
❖ Frontoethmoidal suture – Roof of ethmoid sinus at the level of
cribriform plate.
❖ Ant. & Post. Ethmoidal Foramen – along frontoethmoidal suture in
midorbit.
❖ Distances :
❖ Medial orbital rim – Ant. Ethmoid Foramen – 22- 25mm
❖ Post. Ethmoid Foramen to Ant Ethmoid Foramen – 12mm
Lateral wall
❑ Forms – Triangle with an ant. base.
❑ Forms 450
angle to its medial wall
counterpart.
❑ SURFACE LANDMARKS –
❑ Whitnall’s tubercle – small bony
projection – actual insertion is
‘Common lateral retinaculum’.
❑ Superior & Inferior Orbital Fissure
❑ Zygomaticosphenoid suture –
imp. landmark to verify proper
reduction of complex ZMC fracture.
Peterson’s Principles of Oral and Maxillofacial Surgery ; 2nd Edition ; Page –465
Roof Of Orbit
❑ Domed contour
❑ Comprised of – Orbital plate of Frontal Bone
❑ Extremely thin
❑ In apex – Roof terminates into Lesser Wing of Sphenoid
– where Oval shaped Optic foramen forms Optic canal.
❑ SURFACE LANDMARKS:
❑ Trochlear fossa : Zone of attachment of tendon of
Superior Oblique muscle
❑ Lacrimal Gland Fossa
Apex of Orbit
❑ Post. 3rd – made by sphenoid
bone
❑ Superior Orbital Fissure –
separates the Lesser wing of
sphenoid & Greater Wing of
Sphenoid
❑ Optic Canal – within the lesser
wing of sphenoid
❑ Annulus of Zinn – All Extraocular
muscles except Inf. Oblique arises
from this tendinous ring.
Limit of Dissection
❑ Inferiorly(Floor) – Upto 28-30mm (safe limit) –optic canal
is at around 40mm.
❑ Laterally – Superior Orbital Fissure
❑ Superiorly – Orbital roof dissection is stopped at periorbital
surrounding Recurrent Meningeal Artery – passing through
bony canal within the Sphenofrontal suture line
❑ Medially – Posterior extent – Posterior Ethmoidal vessels,
running in the Fronto-Ethmoidal Suture line Anterior to Optic
foramen.
Distance of Vital Orbital Structures
from Bony landmark
STRUCTURE REFERENCE
LANDMARK
MEAN DISTANCE (mm)
Midpoint of
inferior orbital
fissure
Infraorbital foramen 24
Anterior
Ethmoidal
Foramen
Anterior Lacrimal crest 24
Superior Orbital Fissure Zygomaticofrontal
suture
35
Superior orbital Fissure Supraorbital Notch 40
Optic Canal
(medial aspect)
Anterior Lacrimal Crest 42
Optic Canal (Superior
Aspect)
Supraorbital notch 45
Pathophysiology of Orbital
Fractures
Trauma
Thick Rims protect the Eyeball
Absorb shock by Fracturing themselves
Orbital walls (especially Medial Wall & Floor) fracture
in an isolated way
Gets displaces Inwards or Outwards
Called as ‘Blow-In’ or ‘Blow-Out’fractures
Classification
 ORBITAL WALL FRACTURES
1. Blow Out Fracture
2. Pure Blow Out Fracture
3. Impure Blow Out Fracture
4. Blow In Fracture
 ISOLATED ORBITAL WALL FRACTURE :
1. Roof
2. Floor
3. Medial wall
4. Lateral wall
❑ ISOLATED FRACTURES OF THE ORBITAL
RIM:
1. SUPERIOR RIM
2. INFERIOR RIM
3. MEDIAL RIM
4. LATERAL RIM
❑ COMPLEX COMMINUTED FRACTURES :
Nasoethmoidal and fronto-naso-orbital
fractures
❑ PURE Blow Out OR Blow In –
Orbital Walls fracture in Isolation
❑ IMPURE Blow Out or Blow In Fracture –
Walls + Rim
Blow Out Fracture
Blow Out Fracture
❑ Term coined by – Smith and Regan – 1957
❑ First described by MacKenzie in 1844
❑ PATHOPHYSIOLOGY
❑ Buckling Theory –
❑ If a force strikes at any part of orbital rim,
❑ it will cause walls to undergo a rippling effect &
the force striking the rim will transfer to weaker
portion especially floor & cause them to distort
& eventually fracture
Pathophysiology of blow out fracture
of the orbit
❑ Hydraulic Theory (Pfeiffer in
1943) – A generalized increase
in orbital content pressure
results in direct compression and
fracture of the thin orbital bone.
❑ Therefore direct injury to globe
forcing it into the orbit is
necessary.
❑ Medial Wall & Floor –Thin &
Fragile
❑ Fracture readily – Provide
natural compensation.
Clinical Features
❑ Circumorbital Edema
❑ Subconjunctival Bleeding – due to fracture →
subperiosteal bleeding → escapes in subconjuctival
plane.
❑ Enopthalmous → Increase in size of Orbit →
Eyeball sinks
❑ Periorbital fat herniates through fractured walls-‘Hanging-
drop’Appearance
❑ Unilateral Epistaxis – bleeding into antrum
❑ Numbness in area of distribution of Infraorbital Nerve
❑ Diplopia or Vertical gage – Inferior Rectus or Inferior
Obliqus gets entrapped in fracture → Inability of eyeball
to move in vertical direction.
Enopthalmus following Blow-Out
Fracture
❑ Retracting action of
extraocular muscles
– Enopthalmus.
❑ Diplopia –
Entrapment of
Inferior Rectus &
Inferior Obliqus
Superior Orbital Fissure
Syndrome
k/a – ‘Rochon Duvigneaud Syndrome’
Hirschfeld – first described it.
Pathophysiology
Raised Intraorbital Pressure (due to
Hematoma/Displaced fractured segments)
Compression of contents of Sup. Orbital Fissure
Paresis of Nerve
Neurological deficit in their distribution
❑ Due to paresis of these nerves → all these
extraocular muscles undergo paralysis →
eyeball fails to move → ‘External
Opthalmoplegia’
❑ As affected eye does not move → whereas
contralateral normal eyeball moves → focal
axis gets disturbed → two images -→
Diplopia
Clinical Manifestations
❑ External Opthalmoplegia – Eyeball fails to move
❑ Diplopia – Two images of one object
❑ Internal Opthalmoplegia – Fixed Dilated pupils
(parasympathetic – III cranial nerve –
Occulomotor )
❑ Ptosis of Upper Eyelid – upper eyelid drops
down like a curtain – parasympathetic supply.
❑ Orbital Apex Syndrome – If Optic Nerve
involvement is present.
BLOW IN FRACTURE
Fragmented bones of the orbital floor are
displaced into the orbit.
Proptosis – Exopthalmous.
More commonly seen in fractures of orbital roof.
CLINICAL EXAMINATION
 Initial Opthalmological evaluation –
1. Periorbital Examination
2. Visual acuity – SNELLEN CHART
3. Ocular motility – FORCED DUCTION TEST
4. Pupillary responses ,- pupillary size, shape & symmetry,
light reactivity,
5. Visual fields – HESS CHART
6. Fundoscopic examination -TONOMETRY – to assess Intraocular
pressure (Normal 10-20mmHg)
7. Hertel Exopthalmometer – measure exopthalmous
Forced Duction Test
❑ Prior to the test, a cotton-
tipped applicator soaked
with topical anesthetic
drops and held against
the limbus for few
minutes.
❑ Fine-toothed forceps
are then used to grasp
the conjunctiva and
Tenon’s capsule just
posterior to the limbus.
❑ The patient is then
asked to look in the
direction of restriction of
movement of the eye .
Snellen Chart
• To assess visual
acuity of the eye
Tonometer
• Measures IOP
• Greatly increased IOP
causes pulsatile optic
disk
Hess Chart
Opthalmoscopy
 Examination of the fundus
 TYPES:
Direct --- General examination
Indirect--- Better view of the eye
Pressure – eye ball – blunt instrument—edges
of fundus -- examination
 Retinal detachment requires
ophthalmologic attention.
Hyphaema
❑ Layering of blood in the anterior chamber.
❑ Direct blow to eye --fist or squash ball
❑ C/F: Pain
Blurred vision
Photophobia
Visual acuity decreased
Pupil dilated
❑ Vision worse supine, clears upright
❑ Can cause increased IOP
❑ Head of bed elevated
❑ Atropine –dilates pupil– prevents iris movements
❑ Mannitol
❑ Anti fibrinolytic agent 50mg/kg every 4 hrs reduces
recurrent bleeding.
IMAGING
❑ Caldwell view
❑ Waters view
❑ Lateral skull
❑ CT Scan – Orbit
- To visualize the fractured segments
- Also helps evaluate the Intraorbital volume.
By using a software to compare the normal
orbital volume to the affected.
❑ MRI
Imaging
 Ultrasound
 High resolution CT is used the
management of enophthalmos
 Sinuscopy
 3D reconstruction volumetric
assessment
Management
Management
Indications for surgical
managment
Unresolving soft tissue entrapment with disabling
diplopia
Enopthalmous greater than 2mm
CT scan evidence of a large fracture
SURGICAL MANAGEMENT :
1.CLOSED REDUCTION –
Transantrally.- Caldwell Luc Procedure
Trannasally – Through inferior turbinate – foley’s
catheter
Incisions
❑ Existing lacerations
❑ Lower Eyelid –
1)Subciliary
2) Subtarsal
3) Infraorbital approaches
❑ Transconjuctival Approach
SAGITAL SECTION THROUGH ORBIT &
GLOBE
C- Palpebral
Conjuntiva
IO- Inferior Oblique
muscle
IR- Inferior Rectus
Muscle
OO- Orbicularis Oculi
OS – Orbital Septum
P -
Periosteum/Periorbita TP-
Tarsal Plate.
Orbicularis Oris Muscle
Innervated by Cranial Nerve VII
Upper Eyelid – Levator
Palpebral Superioris – Cranial
Nerve III
Orbicularis Oculi -
Orbital and
Palpebral Portions
Palpebral Portion is divided into
fibers -
Pretarsal Portion
Preseptal Portion
Subciliary
Incision
The incision is approximately 2 mm below eyelashes, can be
extended laterally as necessary (top dashed line). It is made
through skin only.
Subcutaneous dissection of skin, leaving
pretarsal portion of orbicularis muscle
attached to tarsus. Dissection 4-6mm
inferiorly in this plane is adequate.
Subcutaneous dissection
through lid margin
Use scissors to dissect through orbicularis oculi muscle
over lateral orbital rim to identify periosteum.
Incision through the bridge of orbicularis
oculi muscle.
Saggital plane through orbit showing
incision of the bridge of orbicularis oculi
muscle.
❑ Incision through periosteum
along ant. maxilla, 3 to 4 mm
inferior to infraorbital rim.
❑ Note the pretarsal muscle still
remaining on the inferior tarsus
and orbital septum, which restricts
orbital fat from entering field.
❑ Subperiosteal dissection of anterior
maxilla and orbital floor. Periosteal
elevator entering the orbit is
placed almost vertically as
dissection proceeds behind rim.
❑ In ant. region, floor of the orbit is at a
lower level than crest of the rim,
necessitating dissection inferiorly.
Sagital plane through orbit showing subperiosteal dissection of
the anterior maxilla and orbital floor.
Transconjuctival approach
•Fig.1 - Incision of conjuctiva below the tarsalplate.
•Fig 2 - Incision through periosteum. To facilitate this maneuver, a traction
suture is placed through the cut end of conjunctiva to retract the tissueand
maintain the position of corneal shield.
• Small retractors are placed so that lower lid is retracted to the level of ant.
surface of infraorbital rim.
• Intervening tissue along infraorbital rim is the periosteum. Theincision
is made through the periosteum just post. to infraorbitalrim.
•Saggital plane through the orbit and globe demonstrating
level and plane of incision. The conjunctiva and lower lid
are incised with scissors.
SUBPERIOSTEAL DISSECTION OF THE ORBITAL FLOOR.
Note the traction suture placed through the cut end of the
conjunctiva, which assists in retracting the conjunctiva and
maintains the corneal shield in place.
Surgical Exposure
❑ After periorbital
dissection is performed,
adequate exposure and
illumination of the
fractured area is
imperative.
❑ Malleable retractors,
spoons and special
orbital retractors
designed for the globe.
❑ Transition between anterior
mid orbit , the orbital floor
slopes upwards giving rise to
posterior medial bulge &
slightly convex bony platform.
❑ Elevator passes transversely
along inferior orbital fissure.
❑ Infraorbital neurovascular
bundle can be visualized first
shining through the thin bony
roof of its canal.
❑ Then it becomes directly visible
in infraorbital groove.
EXTENT OF DISSECTION
❑ Taking into account extent of
fracture, the periorbital dissection
stops at medial border of the
inferior orbital fissure leaving
the soft tissue invagination intact.
❑ Laterally, the dissection is
continued to posterior edge of
the floor to orbital process of
the palatine bone. The suture
between the maxilla and the
palatine bone is indistinguishable
in the adult skull.
❑ Medially the periorbital
dissection extends to the zone
over the internal orbital
buttress.
❑ For an EXTENDED ACCESS
to posterior orbital floor, the
contents of the inferior orbital
fissure must be addressed
and transected to allow for
this additional access.
❑ The transsection is prepared
with a dissection along the
inferior portion of the lateral
orbital wall in order to create a
second tunnel alongside the
vertical soft tissue.
❑ The intervening soft tissue
invagination is transected in a
stepwise fashion using
bipolar electrocautery and
sharp dissection across the
fissure above the level of
Müller’s muscle, stripping
the periorbita along the
lateral edge of the inferior
orbital fissure.
Limit of Dissection
❑ Inferiorly – Upto 28-30mm.
❑ Laterally – Superior Orbital Fissure
❑ Superiorly – Orbital roof dissection is stopped
at periorbital surrounding Recurrent Meningeal
Artery – passing through bony canal within the
Sphenofrontal suture line
❑ Medially – Posterior extent – Posterior
Ethmoidal vessels , running in the Fronto-
Ethmoidal Suture line Anterior to Optic
foramen.
❑ The subperiosteal dissection is
continued using a periosteal or
freer elevator in a lateromedial
direction and lifted up and
retracted by and by with the
ribbon retractor until the
entrance of apex is reached.
❑ Closure (layer-by-layer)
Reconstruction Material
Autogenous Material
• Graft transferred from one part
of patient’s body to another
• Ex. –1. Septal cartilage
2. Ear cartilage
3. Maxillary wall bone
4. Iliac crest
5. Rib
6. Split calvaria
Bone Graft
Advantages:
1. Provides strength and stability to the orbital
floor
2. Relative resistance to infection
3. Lack of host response against the graft
4. Lack of concern for late extrusion
5. Long term efficacy and reliability
6. Can be easily accessed and harvested from
same operative area ( cartilage – ear, nose;
bone- maxilla, mandible )
Disadvantages :
1. Need for graft donor site
2. Increased operative time
3. Complications related to donor site wound
4. Donor site morbidity ( rib – pneumothorax,
infection; calvarial – dural tears,
subarachnoid haemorrhage, etc)
5. Unpredictable graft resorption
6. Limited ability to contour same type of bone
7. Limited availability
Allogenic Materials
 Graft transferred from another
person (homogenic/ allogenic) or
from any animal of different
species (xenograft)
 Ex. 1. Lyophilized dura
2. Lyophilized cartilage
3. Banked bone
Advantages:
1. No need for graft donor site
2. Decreases operative time
3. Eliminates complications
related to donor site wound
morbidity
4. Inertness
5. Resistant to resorption
Alloplastic Materials
 Grafts derived from non-living source, synthetic
 Ex. 1. Gelfilm
2. Medpor
3. Polytetrafluoroethylene
4. Methylmethacrylate
5. Polyamide mesh
6. Titanium, vitallium, other metallic meshes
7. Hydroxyapatite – block, granular
8. Hydroxyapatite – cement, vicryl mesh
Pre-formed Orbital Implant
ADVANTAGES :
•Radiopacity
•Smooth Surface
•Minimal or no countouring necessary
DISADVANTAGE :
•Cost
Porous Polyethylene
Sheet (PPE)
Disadvantages :
• Not Radiopaque (Not visible on Post OP Radiographs)
•Lack of Rigidity when very thin wafer of PPE is used. When a
more thick rigid wafer is used there is a risk of causing
dystopia.
Polyethylene and titanium mesh
❑ By combining titanium mesh with
porous polyethylene –
❑ Material becomes radioopaque
❑ More rigid than porous PPE.
❑ ADVANTAGE :
❑ Stability
❑ Contouring
❑ Adequate in large three wall
fractures
❑ Radiopacity
❑ No Donor Site Needed
❑ DISADVANTAGES : Less
drainage from orbit.
Resorbable materials
❑ Thermoplastic and Non
thermoplastic materials.
❑ Advantages :
❑ Availability Handling/
❑ Contourability (only for
thermoplastics)
❑ Smooth surface and smooth edges
❑ Disadvantages:
❑ No radiopacity
❑ Degradation of material with possible
contour loss Sterile infection /
inflammatory response
❑ Difficult to shape according to patients
anatomy (only for non- thermoplastics)
❑ Less drainage from the orbit.
COMPLICATIONS
EARLY COMPLICATIONS :
1. Hemorrhagic or orbital hematoma – treated by
-lateral cathotomy immediately, lateral canthal
Tendonlysis ,
Lateral canthotomy – indicated when –
- Decreased visual acuity
- Intarocular pressure more than 40mmHg
- Proptosis
- Opthalmoplegia
Retrobulbar hemorrhage
❑ Rare, rapidly progressive
life threatening emergency
that results in accumulation
of blood in the retrobulbar
space
❑ Increased IOP → stretching
of the optic nerve &
blockage of ocular
perfusion
❑ Proptosis , marked
subconjuctival ecchymosis
& edema.
Symptoms– Pain , decreasing visual acuity, diplopia.
Treatment includes
• I.v mannitol(200 ml of 20% solution)
• i.v acetazolamide 500 mg
• i.v papaverine 40 mg
• i.v hydrocortisone 100 mg
• Dexamethasone sodium phosphate-3-4mg/kg body
weight followed by 1-3 mg /kg 6th hourly.
• If no evidence of improvement stop after 48 hours.
3. Blindness
OCULOCARDIAC REFLEX/ TRIGEMINOCARDIAC/
TRIGEMINOVAGAL REFLEX
- The oculocardiac reflex pathway begins with the
- afferent fibres of the long & short ciliary nerves that
travel with
- the opthalmic division of the trigeminal nerve to
- the gasserion ganglion via
- the sensory nucleus of the trigeminal nerve.
- In the floor of the 4th ventricle short internuncial
fibres in the reticular formation connect them with the
efferent pathway from the motor nucleus of the
vagus nerve to the depressor nerve ending in the
mucle tissue of the heart.
CLINICAL FEATURES –
- Bradycardia
- Faintness
- Further stimulation can lead to cardiac
dysrhythmias
- Atrioventricular blocks
- Asystole
- Bradycardia has been attributed to Trigeminal
derived vagal reflex
LATE COMPLICATIONS
❑ Altered vision
❑ Diplopia
❑ Ectropion – Lower eyelid turns outwards.
❑ Epiphora – Overflow of tears onto the face
❑ Enopthalmous
References
❑ Rowe & William’s Maxillofacial Injuries
❑ Maxillofacial Surgery-Peter Ward Booth
Vol-1
❑ Peterson’s Principles of Oral and
Maxillofacial Surgery ; 2nd Edition
❑ Textbook of Oral & Maxillofacial Surgery –
R.M. Borle
Orbital Fracture & Management

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Orbital Fracture & Management

  • 1.
  • 2. CONTENTS ❑ Anatomy of Orbit ❑ Classification of fracture ❑ Pathophysiology ❑ Blow In fracture ❑ Blow Out Fracture – Pathophysiology & Clinical features. ❑ Superior Orbital Fissure Syndrome - Pathophysiology & Clinical features. ❑ CLINICAL EVALUATION – Forced Duction Test ❑ Management ❑ Reconstruction ❑ Complications
  • 3. Anatomy of Orbit ❑ Orbit – conical cavity ❑ Base – Anteriorly ❑ Apex – Directed at Optic Foramen. ❑ Orbital Volume – 30cc ; Globe – 7cc ❑ Bones – 7 (Maxillary, Zygomatic, Frontal, Ethmoid, Lacrimal, Palatine, and Sphenoid ) ❑ Four Walls – roof, lateral, medial, floor ❑ Medial wall & Floor – Thin ❑ Lateral wall & Roof – Stronger. ❑ Floor weakened – due to – Infraorbital canal passing through it.
  • 4. ❑ Medial wall – Formed by – Lamina Papyracea of Ethmoid bone & Lacrimal bone. ❑ 6 EXTRAOCULAR MUSCLES – ❑ Originate from Common Tendinous fibrous ring ❑ Annulus of Zinn – Fibrous ring – Common origin of 4Recti Muscle ❑ OPTIC NERVE – direct extension of Gray Matter ❑ Very sensitive to Compression and once damaged fails to regenerate. ❑ SUPERIOR ORBITAL FISSURE ❑ Transmits – 3rd , 4th & 6th Cranial Nerves ❑ Opthalmic Division of 5th Cranial Nerve Anatomy of Orbit
  • 6. Floor Of Orbit ❑ Extends – Rim to approx. 2/3rd of the depth of orbit. ❑ Posteromedial aspect of Orbital floor transits into Medial Orbital Wall – to form post. medial bulge. ❑ Only three of four orbital wall extend into the apex – Medial, Superior, Lateral. ❑ Ant. 3rd – Diameter widens behind Superior Inferior margins ‘Post Entry Concavity’ – Saggital plane – ‘Lazy S’ shape. Peterson’s Principles of Oral and Maxillofacial Surgery ; 2nd Edition ; Page 463- 464
  • 7. Surface Landmarks – ❑ Infraorbital Groove – bony sulcus ❑ Inferior Orbital Fissure –converts into canal – continues to Infraorbital Foramen. ❑ Origin Of Inferior Oblique Muscle ❑ Posterior Lateral Promontry – Posterior medial bulge ending in a raised promontory shaped lateral plateau. Floor Of Orbit
  • 8.
  • 9. Medial wall ❑ Convex Rectangular shape ❑ Runs parallel to saggital plane ❑ Medial wall – paper thin Lamina paprycea – (0.2-0.4mm) Reinforced along the Maxillary- Ethmoidal suture --- forming an Internal Orbital Buttress. Peterson’s Principles of Oral and Maxillofacial Surgery ; 2nd Edition ; Page –465
  • 10. Surface Landmarks ❖ Lacrimal Fossa –Anterior 1/3rd of MedialWall ❖ Frontoethmoidal suture – Roof of ethmoid sinus at the level of cribriform plate. ❖ Ant. & Post. Ethmoidal Foramen – along frontoethmoidal suture in midorbit. ❖ Distances : ❖ Medial orbital rim – Ant. Ethmoid Foramen – 22- 25mm ❖ Post. Ethmoid Foramen to Ant Ethmoid Foramen – 12mm
  • 11. Lateral wall ❑ Forms – Triangle with an ant. base. ❑ Forms 450 angle to its medial wall counterpart. ❑ SURFACE LANDMARKS – ❑ Whitnall’s tubercle – small bony projection – actual insertion is ‘Common lateral retinaculum’. ❑ Superior & Inferior Orbital Fissure ❑ Zygomaticosphenoid suture – imp. landmark to verify proper reduction of complex ZMC fracture. Peterson’s Principles of Oral and Maxillofacial Surgery ; 2nd Edition ; Page –465
  • 12. Roof Of Orbit ❑ Domed contour ❑ Comprised of – Orbital plate of Frontal Bone ❑ Extremely thin ❑ In apex – Roof terminates into Lesser Wing of Sphenoid – where Oval shaped Optic foramen forms Optic canal. ❑ SURFACE LANDMARKS: ❑ Trochlear fossa : Zone of attachment of tendon of Superior Oblique muscle ❑ Lacrimal Gland Fossa
  • 13. Apex of Orbit ❑ Post. 3rd – made by sphenoid bone ❑ Superior Orbital Fissure – separates the Lesser wing of sphenoid & Greater Wing of Sphenoid ❑ Optic Canal – within the lesser wing of sphenoid ❑ Annulus of Zinn – All Extraocular muscles except Inf. Oblique arises from this tendinous ring.
  • 14.
  • 15. Limit of Dissection ❑ Inferiorly(Floor) – Upto 28-30mm (safe limit) –optic canal is at around 40mm. ❑ Laterally – Superior Orbital Fissure ❑ Superiorly – Orbital roof dissection is stopped at periorbital surrounding Recurrent Meningeal Artery – passing through bony canal within the Sphenofrontal suture line ❑ Medially – Posterior extent – Posterior Ethmoidal vessels, running in the Fronto-Ethmoidal Suture line Anterior to Optic foramen.
  • 16. Distance of Vital Orbital Structures from Bony landmark STRUCTURE REFERENCE LANDMARK MEAN DISTANCE (mm) Midpoint of inferior orbital fissure Infraorbital foramen 24 Anterior Ethmoidal Foramen Anterior Lacrimal crest 24 Superior Orbital Fissure Zygomaticofrontal suture 35 Superior orbital Fissure Supraorbital Notch 40 Optic Canal (medial aspect) Anterior Lacrimal Crest 42 Optic Canal (Superior Aspect) Supraorbital notch 45
  • 17. Pathophysiology of Orbital Fractures Trauma Thick Rims protect the Eyeball Absorb shock by Fracturing themselves Orbital walls (especially Medial Wall & Floor) fracture in an isolated way Gets displaces Inwards or Outwards Called as ‘Blow-In’ or ‘Blow-Out’fractures
  • 18. Classification  ORBITAL WALL FRACTURES 1. Blow Out Fracture 2. Pure Blow Out Fracture 3. Impure Blow Out Fracture 4. Blow In Fracture  ISOLATED ORBITAL WALL FRACTURE : 1. Roof 2. Floor 3. Medial wall 4. Lateral wall
  • 19. ❑ ISOLATED FRACTURES OF THE ORBITAL RIM: 1. SUPERIOR RIM 2. INFERIOR RIM 3. MEDIAL RIM 4. LATERAL RIM ❑ COMPLEX COMMINUTED FRACTURES : Nasoethmoidal and fronto-naso-orbital fractures
  • 20. ❑ PURE Blow Out OR Blow In – Orbital Walls fracture in Isolation ❑ IMPURE Blow Out or Blow In Fracture – Walls + Rim
  • 22. Blow Out Fracture ❑ Term coined by – Smith and Regan – 1957 ❑ First described by MacKenzie in 1844 ❑ PATHOPHYSIOLOGY ❑ Buckling Theory – ❑ If a force strikes at any part of orbital rim, ❑ it will cause walls to undergo a rippling effect & the force striking the rim will transfer to weaker portion especially floor & cause them to distort & eventually fracture
  • 23. Pathophysiology of blow out fracture of the orbit ❑ Hydraulic Theory (Pfeiffer in 1943) – A generalized increase in orbital content pressure results in direct compression and fracture of the thin orbital bone. ❑ Therefore direct injury to globe forcing it into the orbit is necessary. ❑ Medial Wall & Floor –Thin & Fragile ❑ Fracture readily – Provide natural compensation.
  • 24. Clinical Features ❑ Circumorbital Edema ❑ Subconjunctival Bleeding – due to fracture → subperiosteal bleeding → escapes in subconjuctival plane. ❑ Enopthalmous → Increase in size of Orbit → Eyeball sinks ❑ Periorbital fat herniates through fractured walls-‘Hanging- drop’Appearance ❑ Unilateral Epistaxis – bleeding into antrum ❑ Numbness in area of distribution of Infraorbital Nerve ❑ Diplopia or Vertical gage – Inferior Rectus or Inferior Obliqus gets entrapped in fracture → Inability of eyeball to move in vertical direction.
  • 25. Enopthalmus following Blow-Out Fracture ❑ Retracting action of extraocular muscles – Enopthalmus. ❑ Diplopia – Entrapment of Inferior Rectus & Inferior Obliqus
  • 26. Superior Orbital Fissure Syndrome k/a – ‘Rochon Duvigneaud Syndrome’ Hirschfeld – first described it.
  • 27. Pathophysiology Raised Intraorbital Pressure (due to Hematoma/Displaced fractured segments) Compression of contents of Sup. Orbital Fissure Paresis of Nerve Neurological deficit in their distribution
  • 28. ❑ Due to paresis of these nerves → all these extraocular muscles undergo paralysis → eyeball fails to move → ‘External Opthalmoplegia’ ❑ As affected eye does not move → whereas contralateral normal eyeball moves → focal axis gets disturbed → two images -→ Diplopia
  • 29. Clinical Manifestations ❑ External Opthalmoplegia – Eyeball fails to move ❑ Diplopia – Two images of one object ❑ Internal Opthalmoplegia – Fixed Dilated pupils (parasympathetic – III cranial nerve – Occulomotor ) ❑ Ptosis of Upper Eyelid – upper eyelid drops down like a curtain – parasympathetic supply. ❑ Orbital Apex Syndrome – If Optic Nerve involvement is present.
  • 30. BLOW IN FRACTURE Fragmented bones of the orbital floor are displaced into the orbit. Proptosis – Exopthalmous. More commonly seen in fractures of orbital roof.
  • 31. CLINICAL EXAMINATION  Initial Opthalmological evaluation – 1. Periorbital Examination 2. Visual acuity – SNELLEN CHART 3. Ocular motility – FORCED DUCTION TEST 4. Pupillary responses ,- pupillary size, shape & symmetry, light reactivity, 5. Visual fields – HESS CHART 6. Fundoscopic examination -TONOMETRY – to assess Intraocular pressure (Normal 10-20mmHg) 7. Hertel Exopthalmometer – measure exopthalmous
  • 32. Forced Duction Test ❑ Prior to the test, a cotton- tipped applicator soaked with topical anesthetic drops and held against the limbus for few minutes. ❑ Fine-toothed forceps are then used to grasp the conjunctiva and Tenon’s capsule just posterior to the limbus. ❑ The patient is then asked to look in the direction of restriction of movement of the eye .
  • 33. Snellen Chart • To assess visual acuity of the eye Tonometer • Measures IOP • Greatly increased IOP causes pulsatile optic disk Hess Chart
  • 34. Opthalmoscopy  Examination of the fundus  TYPES: Direct --- General examination Indirect--- Better view of the eye Pressure – eye ball – blunt instrument—edges of fundus -- examination  Retinal detachment requires ophthalmologic attention.
  • 35. Hyphaema ❑ Layering of blood in the anterior chamber. ❑ Direct blow to eye --fist or squash ball ❑ C/F: Pain Blurred vision Photophobia Visual acuity decreased Pupil dilated ❑ Vision worse supine, clears upright ❑ Can cause increased IOP
  • 36. ❑ Head of bed elevated ❑ Atropine –dilates pupil– prevents iris movements ❑ Mannitol ❑ Anti fibrinolytic agent 50mg/kg every 4 hrs reduces recurrent bleeding.
  • 37. IMAGING ❑ Caldwell view ❑ Waters view ❑ Lateral skull ❑ CT Scan – Orbit - To visualize the fractured segments - Also helps evaluate the Intraorbital volume. By using a software to compare the normal orbital volume to the affected. ❑ MRI
  • 38. Imaging  Ultrasound  High resolution CT is used the management of enophthalmos  Sinuscopy  3D reconstruction volumetric assessment
  • 40.
  • 41. Indications for surgical managment Unresolving soft tissue entrapment with disabling diplopia Enopthalmous greater than 2mm CT scan evidence of a large fracture SURGICAL MANAGEMENT : 1.CLOSED REDUCTION – Transantrally.- Caldwell Luc Procedure Trannasally – Through inferior turbinate – foley’s catheter
  • 42. Incisions ❑ Existing lacerations ❑ Lower Eyelid – 1)Subciliary 2) Subtarsal 3) Infraorbital approaches ❑ Transconjuctival Approach
  • 43. SAGITAL SECTION THROUGH ORBIT & GLOBE C- Palpebral Conjuntiva IO- Inferior Oblique muscle IR- Inferior Rectus Muscle OO- Orbicularis Oculi OS – Orbital Septum P - Periosteum/Periorbita TP- Tarsal Plate.
  • 44. Orbicularis Oris Muscle Innervated by Cranial Nerve VII Upper Eyelid – Levator Palpebral Superioris – Cranial Nerve III Orbicularis Oculi - Orbital and Palpebral Portions Palpebral Portion is divided into fibers - Pretarsal Portion Preseptal Portion
  • 45. Subciliary Incision The incision is approximately 2 mm below eyelashes, can be extended laterally as necessary (top dashed line). It is made through skin only.
  • 46. Subcutaneous dissection of skin, leaving pretarsal portion of orbicularis muscle attached to tarsus. Dissection 4-6mm inferiorly in this plane is adequate. Subcutaneous dissection through lid margin
  • 47. Use scissors to dissect through orbicularis oculi muscle over lateral orbital rim to identify periosteum.
  • 48. Incision through the bridge of orbicularis oculi muscle. Saggital plane through orbit showing incision of the bridge of orbicularis oculi muscle.
  • 49. ❑ Incision through periosteum along ant. maxilla, 3 to 4 mm inferior to infraorbital rim. ❑ Note the pretarsal muscle still remaining on the inferior tarsus and orbital septum, which restricts orbital fat from entering field. ❑ Subperiosteal dissection of anterior maxilla and orbital floor. Periosteal elevator entering the orbit is placed almost vertically as dissection proceeds behind rim. ❑ In ant. region, floor of the orbit is at a lower level than crest of the rim, necessitating dissection inferiorly.
  • 50. Sagital plane through orbit showing subperiosteal dissection of the anterior maxilla and orbital floor.
  • 51. Transconjuctival approach •Fig.1 - Incision of conjuctiva below the tarsalplate. •Fig 2 - Incision through periosteum. To facilitate this maneuver, a traction suture is placed through the cut end of conjunctiva to retract the tissueand maintain the position of corneal shield. • Small retractors are placed so that lower lid is retracted to the level of ant. surface of infraorbital rim. • Intervening tissue along infraorbital rim is the periosteum. Theincision is made through the periosteum just post. to infraorbitalrim.
  • 52. •Saggital plane through the orbit and globe demonstrating level and plane of incision. The conjunctiva and lower lid are incised with scissors.
  • 53. SUBPERIOSTEAL DISSECTION OF THE ORBITAL FLOOR. Note the traction suture placed through the cut end of the conjunctiva, which assists in retracting the conjunctiva and maintains the corneal shield in place.
  • 54. Surgical Exposure ❑ After periorbital dissection is performed, adequate exposure and illumination of the fractured area is imperative. ❑ Malleable retractors, spoons and special orbital retractors designed for the globe.
  • 55. ❑ Transition between anterior mid orbit , the orbital floor slopes upwards giving rise to posterior medial bulge & slightly convex bony platform. ❑ Elevator passes transversely along inferior orbital fissure. ❑ Infraorbital neurovascular bundle can be visualized first shining through the thin bony roof of its canal. ❑ Then it becomes directly visible in infraorbital groove.
  • 56. EXTENT OF DISSECTION ❑ Taking into account extent of fracture, the periorbital dissection stops at medial border of the inferior orbital fissure leaving the soft tissue invagination intact. ❑ Laterally, the dissection is continued to posterior edge of the floor to orbital process of the palatine bone. The suture between the maxilla and the palatine bone is indistinguishable in the adult skull. ❑ Medially the periorbital dissection extends to the zone over the internal orbital buttress.
  • 57. ❑ For an EXTENDED ACCESS to posterior orbital floor, the contents of the inferior orbital fissure must be addressed and transected to allow for this additional access. ❑ The transsection is prepared with a dissection along the inferior portion of the lateral orbital wall in order to create a second tunnel alongside the vertical soft tissue.
  • 58. ❑ The intervening soft tissue invagination is transected in a stepwise fashion using bipolar electrocautery and sharp dissection across the fissure above the level of Müller’s muscle, stripping the periorbita along the lateral edge of the inferior orbital fissure.
  • 59. Limit of Dissection ❑ Inferiorly – Upto 28-30mm. ❑ Laterally – Superior Orbital Fissure ❑ Superiorly – Orbital roof dissection is stopped at periorbital surrounding Recurrent Meningeal Artery – passing through bony canal within the Sphenofrontal suture line ❑ Medially – Posterior extent – Posterior Ethmoidal vessels , running in the Fronto- Ethmoidal Suture line Anterior to Optic foramen.
  • 60. ❑ The subperiosteal dissection is continued using a periosteal or freer elevator in a lateromedial direction and lifted up and retracted by and by with the ribbon retractor until the entrance of apex is reached. ❑ Closure (layer-by-layer)
  • 62. Autogenous Material • Graft transferred from one part of patient’s body to another • Ex. –1. Septal cartilage 2. Ear cartilage 3. Maxillary wall bone 4. Iliac crest 5. Rib 6. Split calvaria
  • 63. Bone Graft Advantages: 1. Provides strength and stability to the orbital floor 2. Relative resistance to infection 3. Lack of host response against the graft 4. Lack of concern for late extrusion 5. Long term efficacy and reliability 6. Can be easily accessed and harvested from same operative area ( cartilage – ear, nose; bone- maxilla, mandible )
  • 64. Disadvantages : 1. Need for graft donor site 2. Increased operative time 3. Complications related to donor site wound 4. Donor site morbidity ( rib – pneumothorax, infection; calvarial – dural tears, subarachnoid haemorrhage, etc) 5. Unpredictable graft resorption 6. Limited ability to contour same type of bone 7. Limited availability
  • 65. Allogenic Materials  Graft transferred from another person (homogenic/ allogenic) or from any animal of different species (xenograft)  Ex. 1. Lyophilized dura 2. Lyophilized cartilage 3. Banked bone Advantages: 1. No need for graft donor site 2. Decreases operative time 3. Eliminates complications related to donor site wound morbidity 4. Inertness 5. Resistant to resorption
  • 66. Alloplastic Materials  Grafts derived from non-living source, synthetic  Ex. 1. Gelfilm 2. Medpor 3. Polytetrafluoroethylene 4. Methylmethacrylate 5. Polyamide mesh 6. Titanium, vitallium, other metallic meshes 7. Hydroxyapatite – block, granular 8. Hydroxyapatite – cement, vicryl mesh
  • 67. Pre-formed Orbital Implant ADVANTAGES : •Radiopacity •Smooth Surface •Minimal or no countouring necessary DISADVANTAGE : •Cost
  • 68. Porous Polyethylene Sheet (PPE) Disadvantages : • Not Radiopaque (Not visible on Post OP Radiographs) •Lack of Rigidity when very thin wafer of PPE is used. When a more thick rigid wafer is used there is a risk of causing dystopia.
  • 69. Polyethylene and titanium mesh ❑ By combining titanium mesh with porous polyethylene – ❑ Material becomes radioopaque ❑ More rigid than porous PPE. ❑ ADVANTAGE : ❑ Stability ❑ Contouring ❑ Adequate in large three wall fractures ❑ Radiopacity ❑ No Donor Site Needed ❑ DISADVANTAGES : Less drainage from orbit.
  • 70. Resorbable materials ❑ Thermoplastic and Non thermoplastic materials. ❑ Advantages : ❑ Availability Handling/ ❑ Contourability (only for thermoplastics) ❑ Smooth surface and smooth edges ❑ Disadvantages: ❑ No radiopacity ❑ Degradation of material with possible contour loss Sterile infection / inflammatory response ❑ Difficult to shape according to patients anatomy (only for non- thermoplastics) ❑ Less drainage from the orbit.
  • 71. COMPLICATIONS EARLY COMPLICATIONS : 1. Hemorrhagic or orbital hematoma – treated by -lateral cathotomy immediately, lateral canthal Tendonlysis , Lateral canthotomy – indicated when – - Decreased visual acuity - Intarocular pressure more than 40mmHg - Proptosis - Opthalmoplegia
  • 72. Retrobulbar hemorrhage ❑ Rare, rapidly progressive life threatening emergency that results in accumulation of blood in the retrobulbar space ❑ Increased IOP → stretching of the optic nerve & blockage of ocular perfusion ❑ Proptosis , marked subconjuctival ecchymosis & edema.
  • 73. Symptoms– Pain , decreasing visual acuity, diplopia. Treatment includes • I.v mannitol(200 ml of 20% solution) • i.v acetazolamide 500 mg • i.v papaverine 40 mg • i.v hydrocortisone 100 mg • Dexamethasone sodium phosphate-3-4mg/kg body weight followed by 1-3 mg /kg 6th hourly. • If no evidence of improvement stop after 48 hours. 3. Blindness
  • 74. OCULOCARDIAC REFLEX/ TRIGEMINOCARDIAC/ TRIGEMINOVAGAL REFLEX - The oculocardiac reflex pathway begins with the - afferent fibres of the long & short ciliary nerves that travel with - the opthalmic division of the trigeminal nerve to - the gasserion ganglion via - the sensory nucleus of the trigeminal nerve. - In the floor of the 4th ventricle short internuncial fibres in the reticular formation connect them with the efferent pathway from the motor nucleus of the vagus nerve to the depressor nerve ending in the mucle tissue of the heart.
  • 75. CLINICAL FEATURES – - Bradycardia - Faintness - Further stimulation can lead to cardiac dysrhythmias - Atrioventricular blocks - Asystole - Bradycardia has been attributed to Trigeminal derived vagal reflex
  • 76. LATE COMPLICATIONS ❑ Altered vision ❑ Diplopia ❑ Ectropion – Lower eyelid turns outwards. ❑ Epiphora – Overflow of tears onto the face ❑ Enopthalmous
  • 77. References ❑ Rowe & William’s Maxillofacial Injuries ❑ Maxillofacial Surgery-Peter Ward Booth Vol-1 ❑ Peterson’s Principles of Oral and Maxillofacial Surgery ; 2nd Edition ❑ Textbook of Oral & Maxillofacial Surgery – R.M. Borle