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SEMINAR, LATIHAN, DAN
FAMILY GATHERING
GABUNGAN WILAYAH 2
DR. DENISH GUNAWAN
TOPIC
Environmental
Injury
Heat & Cold-
related Illness
Envenomation Dehydration
ENVIRONMENTAL
INJURY
PHYSIOLOGICAL CHANGES
Hiperventilasi (Hypoxic Ventilatory Response)
Homeostasis
↑ Sistem Saraf Simpatik
↑ Erythropoietin
↑ Mitokondria
↓ Exercise capacity (±1% / 100m above 1500m)
AMS & HACE
Acute Mountain Sickness
Nausea, vomiting, loss of appetite,
disturbed sleep, fatigue, dizziness
Paucity of physical finding
6-12 h after ascent
May progress to HACE
High Altitude Cerebral Edema
Hallmarks: ataxia & altered
consciousness
Diffuse cerebral involvement
Without focal neurologic deficit
HAPE
High Altitude Pulmonary
Edema
Onset: 2-4 days
Features:
• ↓ exercise tolerance
greater than expected
• Dry, persistent cough/
Blood-tinged sputum
• Tachypnea & tachycardia at
rest
• Crackles
• Hypoxemia & respiratory
alkalosis
MANAGEMENT
Discontinuation of ascent
Descent
Assess ABCDE
Consider comorbidities
If readily available:
O2 administration
Hyperbaric therapy (portable)
Pharmacological treatment 
acetazolamide ,
dexamethasone, PDEi
(sildenafil, tadalafil),
acetaminophen & ibuprofen
PREVENTION
• Climb high, sleep low
• Above 3000m  increase your altitude by 300m/day and
every 900m of elevation gained, take a rest day
• If begin to show symptoms don’t go higher until
symptoms decrease
• Keep hydrated
• Don’t over-exert yourself
• Avoid tobacco, alcohol, and other antidepressant drugs
• Eat a high carbohydrate diet while at altitude
• Medication (acetazolamide 24-48h before ascent and
continue for 48h at high altitude or longer,
dexamethasone)
HEAT & COLD
RELATED ILLNESS
Heat Related Illness
Hypothermia
Burns
HEAT LOSS & PRODUCTION
HEAT-RELATED ILLNESS
Heat Stroke
• Core temp ≥ 40oC
• Dehydration, hot flushed dry skin + CNS Dysfunction
Heat Exhaustion
• Ill defined spectrum of disease
• Mental function intact
• Core temperature usually < 39oC
Heat Cramps
• Painful, brief muscle spasm
PERBEDAAN MANIFESTASI
TATALAKSANA HEAT STRESS/
CRAMPS
Move :
• Pindahkan korban ke tempat
yang jauh dari keramaian dan
teduh.
Assess :
• Identifikasi derajat serta jenis
heat injury. (perhatikan ABC)
Decide :
• Cool & Call. Panggil bantuan.
Kipas korban, lepaskan
segala sesuatu yang terlalu
mengikat (belt, dll) dan
berikan kompres dingin di 4
titik. Posisikan korban
dengan posisi
Trendelenberg. Jika korban
sadar beri minum.Jika tidak
sadar, jangan beri minum.
Evaluate :
• Evaluasi terus kondisi
korban, jangan langsung
ditinggal
Catatan :
4 tempat kompres =leher, aksila,
dahi, lipat paha
Jangan diberi minum jika korban
tidak sadar agar tidak tersedak!!!
TATALAKSANA HEAT
EXHAUSTION/ HEAT STROKE
DRHABC!!
1. Pindahkan pasien ketempat dingin, lepaskan pakaian dan lakukan
pendinginan eksternal :
• Konduktor dingin disisi dahi, kiri/kanan leher, aksila, dan
inguinal;
• Pendingin/ penyejuk ruangan
• Usapkan kulit dengan air suhu dingin
(target penurunan suhu ialah <39,4 oC pada rektal dan 30-33o C
pada kulit)
2. Posisikan pasien miring (recovery position) dan pastikan jalan
napas terbuka,
3. Berikan O2 4 L/menit. Upayakan agar saturasi selalu >90%.
4. Berikan cairan kristaloid isotonik untuk ekspansi volume
HIPOTERMIA
Core temperature < 35oC
Primary & Secondary hypothermia
Mild
(32oC – 35oC)
Moderate
(28oC – 32oC)
Severe
(<28oC)
1. Hipotermia primer
• Produksi panas dalam tubuh tidak dapat
mengimbangi adanya stress dingin, terutama
bila cadangan energi dalam tubuh sedang
berkurang.
• Terjadi melalui radiasi (55-65%), konduksi
(10-15%), konveksi, respirasi, evaporasi.
2. Hipotermia sekunder
• Penyakit atau pengobatan tertentu yang
menyebabkan penurunan suhu.
MANAGEMENT
Ensure Safety
Do the ABCDE
Rewarm the patient
Beware of
complications
FROSTBITE
MANAGEMENT
• Do not attempt rewarming if there is a risk of refreezing
• Replace constricting, damp clothing with warm blankets
• Give the patient hot fluids by mouth (if able to drink)
• Place the injured part in circulating water at constant 40⁰C
until pink color and perfusion return (usually 20-30 min)
• Do not dry heat may cause injury due to skin insensation
• Rewarming can be extremely painful  adequate analgesic
• Beware of the reperfusion synd.  monitor patient’s cardiac
status and peripheral perfusion during rewarming
• Wound care (clean, and leave blisters intact for 7-10 days to
provide sterile biologic dressing to protect underlying
epithelization)
COMPLICATIONS OF
REPERFUSION
1. Acidosis
2. Hyperkalemia
3. Local swelling
4. Rewarming Shock
5. After Drop
6. Dysrhythmia
LUKA BAKAR
Penyebab utama morbiditas dan mortalitas:
• Penerapan prinsip dasar  menurunkan angka morbiditas
dan mortalitas
Prinsip dasar:
• Curiga masalah airway
• Identifikasi dan penanganan cedera mekanis
• Resusitasi cairan
• Kontrol suhu
Luka bakar  ekstravasasi plasma ke luka 
edema, bullae  hipovolemia
Jaringan terbakar  mediator inflamasi lokal &
sistemik, hormon stress  hiperpermeabilitas
vaskular, retensi natrium, vasokonstriksi 
gangguan paru, jantung dan mikrosirkulasi
Komplikasi:
• Gagal ginjal akut
• Disfungsi organ akibat iskemia
• Kolaps kardiovaskular
• Asidosis metabolik
PRIORITAS
• AIRWAY
• STOP BURNING PROCESS
• INTRAVENOUS ACCESS
TANDA TRAUMA
INHALASI
• Luka bakar leher dan/atau wajah
• Alis atau bulu hidung terbakar
• Arang sputum / deposit karbon di
mulut/hidung
• Eritema orofaring
• Serak
• Riwayat terkurung
• Luka ledakan mengenai kepala dan
badan
• Kadar karboksihemoglobin > 10%
BURN AREA ASSESMENT
Palmar surface—The surface area of a patient's palm (including
fingers) is roughly 0.8% of total body surface area. Palmar surface
are can be used to estimate relatively small burns (< 15% of total
surface area) or very large burns (> 85%, when unburnt skin is
counted). For medium sized burns, it is inaccurate.
Wallace rule of nines—This is a good, quick way of estimating
medium to large burns in adults. The body is divided into areas of
9%, and the total burn area can be calculated. It is not accurate in
children.
DERAJAT LUKA
BAKAR
DERAJAT LUKA
BAKAR (2)
What causes a superficial first-degree burn?
In most cases,superficial first-degree burns are caused by the
following:
Mild sunburn
Flash burn -- a sudden, brief burst of heat
What causes a second-degree burn?
In most cases, partial thickness second-degree burns are caused
by the following:
Scald injuries
Flames
Skin that briefly comes in contact with a hot object
Sunburn
Chemicals
Electricity
What causes a third-degree burn?
In most cases, full thickness, third-degree burns are caused by
the following:
A scalding liquid
Skin that comes in contact with a hot object for an extended
period of time
Flames from a fire
An electrical source
A chemical source
ENVENOMATION
SNAKE BITE
PRINCIPAL
Snake identification: venomous/ not
Clinical manifestation varies among species
Do DRABCDE
Early Evacuation
MANAGEMENT
DO
• DRABCDE
• Pressure immobilization
• Splint the bitten extremity
• Keep extremity at heart level
• Take digital photograph of the
snake
DON’T
• Incise
• Apply suction
• Apply ice/ electric shock
• Tourniquet
• Attempt to capture & transport
offending snake
https://biomedicalsciences.unimelb.edu.au/__data/assets/pdf_file
/0006/2106393/2.1-Snakebite_firstaid_ANG_AVRU.pdf
BEES & WASPS
STINGS
• DRABCDE
• Remove the sting (by any means) ASAP
• Apply a cold compress to reduce pain & swelling
• Transport to hospital
• Monitor for signs of allergic reaction  anaphylactic
shock  injection of epinephrine / adrenaline
DEHIDRASI
PENANGANAN
Oral Rehydration Therapy (ORT) untuk dehidrasi mild-moderate
Jika muntah-muntah, ORT bukan terapi yang tepat, namun rehidrasi
dilakukan secara intravena.
Jika ada tanda-tanda dehidrasi berat (HR meningkat, BP rendah), cairan
diberikan secara intravena.
Cairan intravena diberikan sebanyak 20-30 mL/ kg dari larutan
isotonik NaCl 0.9% selama 1-2 jam.
PENANGANAN (2)
Buatlah pasien menjadi lebih dingin, jika pasien mengalami kenaikan
temperatur atau setelah terpapar panas, dengan cara:
• Membuka pakaian berlebih dan kendorkan pakaian lain
• Dinginkan area di ruangan berAC merupakan cara terbaik untuk
membantu mengembalikan temperatur ke normal setelah
paparan panas
• Jika tidak ada AC, tingkatkan pendinginan dengan evaporasi
dengan meletakkan pasien dekat kipas angin, atau balut pasien
dengan handuk basah di sekitarnya
• Hindari pemberian paparan yang terlalu dingin (es)! Karena
mengakibatkan vasokonstriksi dan mengurangi pelepasan panas,
selain itu juga dapat mengakibatkan menggigil yang menaikkan
temperatur tubuh.
• Jika ada, semprot dengan spray air hangat di permukaan kulit
untuk membantu pendinginan dengan evaporasi
REFERENCE
• https://biomedicalsciences.unimelb.edu.au/departments/p
harmacology/engage/avru/discover/first-aid
• https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3678789/#!
po=7.65766
• https://www.princeton.edu/~oa/safety/altitude.html
• https://www.ncbi.nlm.nih.gov/books/NBK430916/
• https://biomedicalsciences.unimelb.edu.au/__data/assets/
pdf_file/0006/2106393/2.1-
Snakebite_firstaid_ANG_AVRU.pdf
• ATLS 2018
• Lintas Diare 2010
TERIMA
KASIH

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Ppt kegawatdaruratan environmental

  • 1. SEMINAR, LATIHAN, DAN FAMILY GATHERING GABUNGAN WILAYAH 2 DR. DENISH GUNAWAN
  • 2. TOPIC Environmental Injury Heat & Cold- related Illness Envenomation Dehydration
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  • 5. PHYSIOLOGICAL CHANGES Hiperventilasi (Hypoxic Ventilatory Response) Homeostasis ↑ Sistem Saraf Simpatik ↑ Erythropoietin ↑ Mitokondria ↓ Exercise capacity (±1% / 100m above 1500m)
  • 6. AMS & HACE Acute Mountain Sickness Nausea, vomiting, loss of appetite, disturbed sleep, fatigue, dizziness Paucity of physical finding 6-12 h after ascent May progress to HACE High Altitude Cerebral Edema Hallmarks: ataxia & altered consciousness Diffuse cerebral involvement Without focal neurologic deficit
  • 7. HAPE High Altitude Pulmonary Edema Onset: 2-4 days Features: • ↓ exercise tolerance greater than expected • Dry, persistent cough/ Blood-tinged sputum • Tachypnea & tachycardia at rest • Crackles • Hypoxemia & respiratory alkalosis
  • 8. MANAGEMENT Discontinuation of ascent Descent Assess ABCDE Consider comorbidities If readily available: O2 administration Hyperbaric therapy (portable) Pharmacological treatment  acetazolamide , dexamethasone, PDEi (sildenafil, tadalafil), acetaminophen & ibuprofen
  • 9. PREVENTION • Climb high, sleep low • Above 3000m  increase your altitude by 300m/day and every 900m of elevation gained, take a rest day • If begin to show symptoms don’t go higher until symptoms decrease • Keep hydrated • Don’t over-exert yourself • Avoid tobacco, alcohol, and other antidepressant drugs • Eat a high carbohydrate diet while at altitude • Medication (acetazolamide 24-48h before ascent and continue for 48h at high altitude or longer, dexamethasone)
  • 10. HEAT & COLD RELATED ILLNESS Heat Related Illness Hypothermia Burns
  • 11. HEAT LOSS & PRODUCTION
  • 12. HEAT-RELATED ILLNESS Heat Stroke • Core temp ≥ 40oC • Dehydration, hot flushed dry skin + CNS Dysfunction Heat Exhaustion • Ill defined spectrum of disease • Mental function intact • Core temperature usually < 39oC Heat Cramps • Painful, brief muscle spasm
  • 14. TATALAKSANA HEAT STRESS/ CRAMPS Move : • Pindahkan korban ke tempat yang jauh dari keramaian dan teduh. Assess : • Identifikasi derajat serta jenis heat injury. (perhatikan ABC) Decide : • Cool & Call. Panggil bantuan. Kipas korban, lepaskan segala sesuatu yang terlalu mengikat (belt, dll) dan berikan kompres dingin di 4 titik. Posisikan korban dengan posisi Trendelenberg. Jika korban sadar beri minum.Jika tidak sadar, jangan beri minum. Evaluate : • Evaluasi terus kondisi korban, jangan langsung ditinggal Catatan : 4 tempat kompres =leher, aksila, dahi, lipat paha Jangan diberi minum jika korban tidak sadar agar tidak tersedak!!!
  • 15. TATALAKSANA HEAT EXHAUSTION/ HEAT STROKE DRHABC!! 1. Pindahkan pasien ketempat dingin, lepaskan pakaian dan lakukan pendinginan eksternal : • Konduktor dingin disisi dahi, kiri/kanan leher, aksila, dan inguinal; • Pendingin/ penyejuk ruangan • Usapkan kulit dengan air suhu dingin (target penurunan suhu ialah <39,4 oC pada rektal dan 30-33o C pada kulit) 2. Posisikan pasien miring (recovery position) dan pastikan jalan napas terbuka, 3. Berikan O2 4 L/menit. Upayakan agar saturasi selalu >90%. 4. Berikan cairan kristaloid isotonik untuk ekspansi volume
  • 16. HIPOTERMIA Core temperature < 35oC Primary & Secondary hypothermia Mild (32oC – 35oC) Moderate (28oC – 32oC) Severe (<28oC)
  • 17. 1. Hipotermia primer • Produksi panas dalam tubuh tidak dapat mengimbangi adanya stress dingin, terutama bila cadangan energi dalam tubuh sedang berkurang. • Terjadi melalui radiasi (55-65%), konduksi (10-15%), konveksi, respirasi, evaporasi. 2. Hipotermia sekunder • Penyakit atau pengobatan tertentu yang menyebabkan penurunan suhu.
  • 18. MANAGEMENT Ensure Safety Do the ABCDE Rewarm the patient Beware of complications
  • 19.
  • 21. MANAGEMENT • Do not attempt rewarming if there is a risk of refreezing • Replace constricting, damp clothing with warm blankets • Give the patient hot fluids by mouth (if able to drink) • Place the injured part in circulating water at constant 40⁰C until pink color and perfusion return (usually 20-30 min) • Do not dry heat may cause injury due to skin insensation • Rewarming can be extremely painful  adequate analgesic • Beware of the reperfusion synd.  monitor patient’s cardiac status and peripheral perfusion during rewarming • Wound care (clean, and leave blisters intact for 7-10 days to provide sterile biologic dressing to protect underlying epithelization)
  • 22. COMPLICATIONS OF REPERFUSION 1. Acidosis 2. Hyperkalemia 3. Local swelling 4. Rewarming Shock 5. After Drop 6. Dysrhythmia
  • 23. LUKA BAKAR Penyebab utama morbiditas dan mortalitas: • Penerapan prinsip dasar  menurunkan angka morbiditas dan mortalitas Prinsip dasar: • Curiga masalah airway • Identifikasi dan penanganan cedera mekanis • Resusitasi cairan • Kontrol suhu
  • 24. Luka bakar  ekstravasasi plasma ke luka  edema, bullae  hipovolemia Jaringan terbakar  mediator inflamasi lokal & sistemik, hormon stress  hiperpermeabilitas vaskular, retensi natrium, vasokonstriksi  gangguan paru, jantung dan mikrosirkulasi Komplikasi: • Gagal ginjal akut • Disfungsi organ akibat iskemia • Kolaps kardiovaskular • Asidosis metabolik
  • 25. PRIORITAS • AIRWAY • STOP BURNING PROCESS • INTRAVENOUS ACCESS
  • 26. TANDA TRAUMA INHALASI • Luka bakar leher dan/atau wajah • Alis atau bulu hidung terbakar • Arang sputum / deposit karbon di mulut/hidung • Eritema orofaring • Serak • Riwayat terkurung • Luka ledakan mengenai kepala dan badan • Kadar karboksihemoglobin > 10%
  • 27. BURN AREA ASSESMENT Palmar surface—The surface area of a patient's palm (including fingers) is roughly 0.8% of total body surface area. Palmar surface are can be used to estimate relatively small burns (< 15% of total surface area) or very large burns (> 85%, when unburnt skin is counted). For medium sized burns, it is inaccurate. Wallace rule of nines—This is a good, quick way of estimating medium to large burns in adults. The body is divided into areas of 9%, and the total burn area can be calculated. It is not accurate in children.
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  • 32. What causes a superficial first-degree burn? In most cases,superficial first-degree burns are caused by the following: Mild sunburn Flash burn -- a sudden, brief burst of heat
  • 33. What causes a second-degree burn? In most cases, partial thickness second-degree burns are caused by the following: Scald injuries Flames Skin that briefly comes in contact with a hot object Sunburn Chemicals Electricity
  • 34. What causes a third-degree burn? In most cases, full thickness, third-degree burns are caused by the following: A scalding liquid Skin that comes in contact with a hot object for an extended period of time Flames from a fire An electrical source A chemical source
  • 36. SNAKE BITE PRINCIPAL Snake identification: venomous/ not Clinical manifestation varies among species Do DRABCDE Early Evacuation
  • 37. MANAGEMENT DO • DRABCDE • Pressure immobilization • Splint the bitten extremity • Keep extremity at heart level • Take digital photograph of the snake DON’T • Incise • Apply suction • Apply ice/ electric shock • Tourniquet • Attempt to capture & transport offending snake
  • 39.
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  • 41. BEES & WASPS STINGS • DRABCDE • Remove the sting (by any means) ASAP • Apply a cold compress to reduce pain & swelling • Transport to hospital • Monitor for signs of allergic reaction  anaphylactic shock  injection of epinephrine / adrenaline
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  • 47. PENANGANAN Oral Rehydration Therapy (ORT) untuk dehidrasi mild-moderate Jika muntah-muntah, ORT bukan terapi yang tepat, namun rehidrasi dilakukan secara intravena. Jika ada tanda-tanda dehidrasi berat (HR meningkat, BP rendah), cairan diberikan secara intravena. Cairan intravena diberikan sebanyak 20-30 mL/ kg dari larutan isotonik NaCl 0.9% selama 1-2 jam.
  • 48. PENANGANAN (2) Buatlah pasien menjadi lebih dingin, jika pasien mengalami kenaikan temperatur atau setelah terpapar panas, dengan cara: • Membuka pakaian berlebih dan kendorkan pakaian lain • Dinginkan area di ruangan berAC merupakan cara terbaik untuk membantu mengembalikan temperatur ke normal setelah paparan panas • Jika tidak ada AC, tingkatkan pendinginan dengan evaporasi dengan meletakkan pasien dekat kipas angin, atau balut pasien dengan handuk basah di sekitarnya • Hindari pemberian paparan yang terlalu dingin (es)! Karena mengakibatkan vasokonstriksi dan mengurangi pelepasan panas, selain itu juga dapat mengakibatkan menggigil yang menaikkan temperatur tubuh. • Jika ada, semprot dengan spray air hangat di permukaan kulit untuk membantu pendinginan dengan evaporasi
  • 49. REFERENCE • https://biomedicalsciences.unimelb.edu.au/departments/p harmacology/engage/avru/discover/first-aid • https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3678789/#! po=7.65766 • https://www.princeton.edu/~oa/safety/altitude.html • https://www.ncbi.nlm.nih.gov/books/NBK430916/ • https://biomedicalsciences.unimelb.edu.au/__data/assets/ pdf_file/0006/2106393/2.1- Snakebite_firstaid_ANG_AVRU.pdf • ATLS 2018 • Lintas Diare 2010