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Pneumothorax
By
Dr. MD ABDULLAH SALEEM
1
What is pneumothorax
 Pneumothorax is defined as
presence of air or gas in the
pleural space
(OR)
 air between the lung and chest wall,
in other term, air between the
visceral pleura and the parietal
pleura
2
Mechanism
 In normal people, the pressure in
pleural space is negative during the
entire respiratory cycle.
 Two opposite forces result in negative
pressure in pleural space.
(outward pull of the chest wall and
elastic recoil of the lung)
 The negative pressure will
be disappeared if any communication
develops .
3
 When a
communication
develops between an
alveolus or other
intrapulmonary air
space and pleural
space, air will flow
into the pleural space
until there is no
longer a pressure
difference or the
communication is
sealed 4
Pathophysiology
Negative pressure eliminated
The lung recoil-& lung-volume decrease
V/Q low –anatomic shunt
hypoxia
Positive pressure
◦ Compress blood vessels and heart
◦ Decreased cardiac output
◦ Impaired venous return
◦ Hypotension
Result in
◦ A decrease in vital capacity
◦ A decrease in PaO2
5
CLASSIFICATION OF PNEUMOTHORAX
6
Pneumothorax
Spontaneous
Primary Secondary
Traumatic
Iatrogenic
Interventional
procedures.
Positive pressure
ventilation
Non iatrogenic
Penetrating
trauma
Blunt trauma.
Primary spontaneous
pneumothorax
 It occurs in young healthy individuals
without underlying lung disease.
 It is due to rupture of apical
sub-pleural bleb or bullae
Predisposing factors:
 Smoking.
 Tall, thin male.
 Airway inflammation (distal)
 Structural abnormalities of bronchial tree
 Genetic contribution
7
Secondary spontaneous
pneumothorax:
It is seen in pt with underlying lung disease.
INFECTION:
Tb
Acute bacteria pneumonia
Copd
Obstructive lung disease
ILD
Fibrosis
Eosinophilic granuloma
Sarcoidosis
Lymphangioleiomyomatosis..
Malignancy:
Primary lung carcinoma
Complication of
chemotherpy
Connective tissue disease
Scleroderma
Marfans syndrome
Catamenial pneumothorax
Pulmonary infract
Wegegner’s
granulomatosis… 8
Clinical manifestation
Dyspnea
Chest pain ( pleuritic)
Uncommon manifestation
Cough
Hemoptysis
Orthopnea
Cyanosis
Mod tachycardia
9
Traumatic pneumothorax
Accidental trauma:(non-iatrogenic)
Blunt trauma: with fracture ribs.
Penetrating trauma: stab wound or
gun shot
injury.
Iatrogenic :
Positive pressure ventilation:
Alveolar rupture  interstitial
emphysema  pneumothorax.
Interventional procedures:
Biopsy, thoraco-centesis, CVP
line,trachestomy etc.. 10
11
clinical type of PNX
Pneumothorax
Closed
pneumothorax
Open
pneumothorax
Tension
pneumothorax
12
Closed
pneumothorax
Open
pneumothorax
Tension
pneumothorax
The pleural tear
Is sealed
The pleural tear
is open
The pleural tear
act as a ball &
valve mechanism
The pleural
cavity pressure
is < the
atmospheric
pressure
The pleural
cavity pressure
is = the
atmospheric
pressure
The pleural cavity
pressure is > the
atmospheric
pressure
Clinical manifestation
 Tension pneumothorax
◦ Distressed with rapid labored respiration
◦ Cyanosis
◦ Marked tachycardia
 Patient who suddenly deteriorate
clinically,
be suspected in the patient with
◦ Mechanical ventilation
◦ Cardiopulmonary resuscitation
13
Physical examination
◦ Depend on size of pneumothorax
◦ The vital signs usually normal
◦ Unilateral Chest movements
◦ The trachea may be shifted toward the
contralateral side if the pneumothorax is
large
◦ Tactile fremitus is absent
◦ The percussion note is hypersonant
◦ The breath sounds are reduced or absent
on the affected side
◦ The lower edge of the liver may be shifted
inferiorly with a right-side pneumothorax
14
Lab investigations
and Diagnosis
15
Radiological manifestation
cxray in erect position cxray in supine position
16
Small pneumothorax
17
18
Small pneumothorax
19
pneumothorax with mediastinal
shift
20
Pneumothorax ?
21
Quantification of the size
The simple method to estimate the size
Small, a visible rim of < 2 cm between the
lung margin and the chest wall
Large, a visible rim of ≥2 cm between the
lung margin and chest wall
Light index
Measure transverse
Diameters of lung and
Compare it with diameter
hemithorax
22
22
Hemithorax (HT)
Lung (L)
CT scanning
It is recommended in difficult cases
such as patients in whom the lungs
are obscured by overlying surgical
emphysema
To differentiate a pneumothorax from
suspected bulla in complex cystic lung
disease
23
24
CT can diagnose easily
pneumothroax
25
25
CT scanning
Small pneumothorax
Subcutaneous emphysema
MANAGMENT
 Goals
◦ To promote lung expansion
◦ To eliminate the pathogenesis
◦ To decrease pneumothorax recurrence
 Treatment options according to
◦ Classification of pneumothorax
◦ Pathogenesis
◦ The extension of lung collapse
◦ Severity of disease
◦ Complication and concomitant underlying
diseases
26
TREATMENT OPTIONS FOR PSP AND SSP
Observation
O2 treatment
Simple aspiration
Small catheter aspiration
Chest tube drainage
Thoracoscopy (VAT with blebtomy &
 VAT with pleurectomy)
Open (axillary) thoracotomy
27
Observation - PSP
 Small, closed mildly symptomatic
spontaneous pneumothorax.
 Do not require hospital admission
 It should be stressed to patient that they
should be return directly to hospital in
the event of developing breathlessness.
28
Observation - SSP
 Observation along is only recommend
in patients with small SSP of less than
1 cm depth or isolated apical
pneumothorax in asymptomatic
patients
 Hospitalization is recommended in
these cases
 All other cases will require active
intervention ( aspiration or chest drain
insertion)
29
30
 Marked breathlessness in a patient with a
small (<2 cm) PSP may develop tension
pneumothorax
 Observation is inappropriate and active
intervation is required
 If a patient is hospitalised for observation,
supplemental high flow (10 l/min) oxygen
should be given where feasible
Observation - PSP or SSP
31
 Inhalation of high concentration of oxygen
may reduce the total pressure of gases in
pleural capillaries by reducing the partial
pressure of nitrogen
 This should increase the pressure gradient
between the pleural capillaries and the
pleural cavity
 Thereby increasing absorption of air from
the pleural cavity
O2 TREATMENT-- PSP or
SSP
32
 The rate of resolution/reabsorption of
spontaneous pneumothorax is 1.25 –
1.8% of volume of hemithorax every 24
hours
 The addition of high flow oxygen
therapy has been shown to result in a
4-fold increase in the rate of
peumothorax reabsorption during the
periods of oxygen supplementation
33
Simple aspiration
 Simple aspiration is recommended as first line
treatment for all PSP requiring intervention
 Simple aspiration is less likely to succeed in
secondary pneumothoraces and in this situation,
is only recommended as an initial treatment in
small (<2 cm) pneumothoraces in minimally
breathless patients under the age of 50 years
 Patients with secondary pneumothoraces
treated successfully with simple aspiration
should be admitted to hospital and observed for
at least 24 hours before discharge
34
 Repeated aspiration is reasonable for
primary pneumothorax when the first
aspiration has been unsuccessful
 A volume of < 2.5 L has been aspirated on
the first attempt
 The aspiration can be used by needle or
catheter
Catheter aspiration
Intercostal tube drainage
 INDICATIONS
◦ Unstable
pneumothorax
◦ Severe dyspnea
◦ Large lung collapse
◦ Open or tension
pneumothorax
◦ Recurrent
pneumothorax
◦ Simple aspiration or
catheter aspiration
drainage is
unsuccessful in
controlling symptoms 35
36
36
 The site of chest tube
insertion is in the
midclavicular line of
second and third
intercostal
 or anterior axillary line
of fifth and sixth
intercostal
Intercostal tube drainage
37
37
Intercostal tube drainage
Fix the catheter and cover with gauze
Making a small incision
Using a forceps to extend the hole
Inserting a catheter into pleural cavity
38
Trocar tube thoracostomy
 Insertion of trocar into the pleural space.
39
Trocar tube thoracostomy
 Insertion of the chest tube through the
trocar
4
Operative tube thoracostomy
The physician’s index
finger is used to
enlarge the opening
and to explore the
pleural space
Placement of chest
tube
Intrapleurally using
large hemostat
41
Observation of drainage
 No bubble released
◦ The lung reexpansion
◦ The chest tube is obstructed by secretion or blood clot
◦ The chest tube shift to chest wall, the hole of the chest
tube is located in the chest wall
 If the lung reexpansion, removing the chest tube 24
hours after reexpansion.
 Otherwise, the chest tube will be inserted again or
regulated the position.
42
Complications of intercostal tube
drainage
 Penetration of major organs
◦ Lung, stomach, spleen, liver, heart and great
vessels
◦ It occurs more commonly when a sharp metal
trocar is inappropriately applied
 Pleural infection
◦ Empyema, the rate of 1%
 Surgical emphysema
◦ Subcutaneous emphysema
43
Chemical pleurodesis
 Goals
◦ To prevent pneumothorax recurrence
◦ To produce inflammation of pleura and
adhesions
 Indications
◦ Persist air leak and repeated pneumothorax
◦ Bilateral pneumothorax
◦ Complicated with bullae
◦ Lung dysfunction, not tolerate to operation
44
Chemical pleurodesis
 Sclerosing agents
◦ Tetracycline
◦ Doxycline
◦ Talc
◦ Erythromycin
 The instillation of sclerosing agents into the pleural
space lead to an aseptic inflammation with dense
adhesions.
45
 Methods
◦ Via chest tube or by surgical mean
◦ Administration of intrapleural local anaesthesia, 200 – 300
mg lidocaine intrapleurally injection
◦ Agents diluted by 60 – 100 ml saline
◦ Injected to pleural space
◦ Clamp the tube 4hours
◦ Drainage again
◦ Observed by chest X-ray film, if air of pleural space is
absorption, remove the chest tube
◦ If pneumothorax still exist, repeated pleurodesis.
Chemical pleurodesis
46
46
 Side effct
◦ Chest pain
◦ Fever
◦ Dyspnea
◦ Acute respiratory distress syndrome
◦ Acute respiratory failure
Chemical pleurodesis
47
Surgical treatment
 Indications
◦ No response to medical treatment
◦ Persist air leak
◦ Hemopneumothorax
◦ Bilateral pneumothoraces
◦ Recurrent pneumothorax
◦ Tension pneumothorax failed to dainage
◦ Thicken pleura makes lung unable to
reexpansion
◦ Multiple blebs or bullae
Thoracoscopy- VATs
 It is being increasly used in ot with
recurrent psp-ssp
 Recent studies show VATs it may be
the procedure of choice and it has
very less complication when compare
to other open surgery
 Pleural belbs causes pneumothorax
which can be treated by a method
called endostapling or
suturefollowed by pleurodosis
48
Axillary (open) thoracotomy
 Transaxillary minithoracotomy is preferred
.when VATs isnot available
 Open thotacotomy with suturing of blebs
and pleural abrasion is done
 Recurrence
 High risk professions
 B/L or tension pneumothorax
49
INDICATIONS
50
Complications of pneumothorax
 Pyopneumothorax
◦ Caused by aspiration or intercostal chest tube
insertion (iatrogenic)
◦ Also results from necrotic pneumonia, lung
abscess, or caseous pneumonia
 Hydropneumothorax.
 Hemopneumotorax
◦ Bleeding in pleural space.
◦ Common cause is rupture of vessels in
adhesions.
◦ When lung re-expansion, bleeding will stop.
◦ When bleeding persists, surgical ligation will
be needed.
51
 Mediastinal and subcutaneous
emphysema
◦ Alveoli rupture, the air enter into pulmonary interstitial
and then goes into mediastinal and subcutaneous
tissues.
◦ After aspiration or intercostal chest tube insertion, the
air enters the subcutaneous by the needle hole or
incision – surgical emphysema
◦ Physical exam – crepitus is present.
Subcutaneous
emphysema
52
THANK YOU
53
54

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(Pneumothorax

  • 2. What is pneumothorax  Pneumothorax is defined as presence of air or gas in the pleural space (OR)  air between the lung and chest wall, in other term, air between the visceral pleura and the parietal pleura 2
  • 3. Mechanism  In normal people, the pressure in pleural space is negative during the entire respiratory cycle.  Two opposite forces result in negative pressure in pleural space. (outward pull of the chest wall and elastic recoil of the lung)  The negative pressure will be disappeared if any communication develops . 3
  • 4.  When a communication develops between an alveolus or other intrapulmonary air space and pleural space, air will flow into the pleural space until there is no longer a pressure difference or the communication is sealed 4
  • 5. Pathophysiology Negative pressure eliminated The lung recoil-& lung-volume decrease V/Q low –anatomic shunt hypoxia Positive pressure ◦ Compress blood vessels and heart ◦ Decreased cardiac output ◦ Impaired venous return ◦ Hypotension Result in ◦ A decrease in vital capacity ◦ A decrease in PaO2 5
  • 6. CLASSIFICATION OF PNEUMOTHORAX 6 Pneumothorax Spontaneous Primary Secondary Traumatic Iatrogenic Interventional procedures. Positive pressure ventilation Non iatrogenic Penetrating trauma Blunt trauma.
  • 7. Primary spontaneous pneumothorax  It occurs in young healthy individuals without underlying lung disease.  It is due to rupture of apical sub-pleural bleb or bullae Predisposing factors:  Smoking.  Tall, thin male.  Airway inflammation (distal)  Structural abnormalities of bronchial tree  Genetic contribution 7
  • 8. Secondary spontaneous pneumothorax: It is seen in pt with underlying lung disease. INFECTION: Tb Acute bacteria pneumonia Copd Obstructive lung disease ILD Fibrosis Eosinophilic granuloma Sarcoidosis Lymphangioleiomyomatosis.. Malignancy: Primary lung carcinoma Complication of chemotherpy Connective tissue disease Scleroderma Marfans syndrome Catamenial pneumothorax Pulmonary infract Wegegner’s granulomatosis… 8
  • 9. Clinical manifestation Dyspnea Chest pain ( pleuritic) Uncommon manifestation Cough Hemoptysis Orthopnea Cyanosis Mod tachycardia 9
  • 10. Traumatic pneumothorax Accidental trauma:(non-iatrogenic) Blunt trauma: with fracture ribs. Penetrating trauma: stab wound or gun shot injury. Iatrogenic : Positive pressure ventilation: Alveolar rupture  interstitial emphysema  pneumothorax. Interventional procedures: Biopsy, thoraco-centesis, CVP line,trachestomy etc.. 10
  • 11. 11 clinical type of PNX Pneumothorax Closed pneumothorax Open pneumothorax Tension pneumothorax
  • 12. 12 Closed pneumothorax Open pneumothorax Tension pneumothorax The pleural tear Is sealed The pleural tear is open The pleural tear act as a ball & valve mechanism The pleural cavity pressure is < the atmospheric pressure The pleural cavity pressure is = the atmospheric pressure The pleural cavity pressure is > the atmospheric pressure
  • 13. Clinical manifestation  Tension pneumothorax ◦ Distressed with rapid labored respiration ◦ Cyanosis ◦ Marked tachycardia  Patient who suddenly deteriorate clinically, be suspected in the patient with ◦ Mechanical ventilation ◦ Cardiopulmonary resuscitation 13
  • 14. Physical examination ◦ Depend on size of pneumothorax ◦ The vital signs usually normal ◦ Unilateral Chest movements ◦ The trachea may be shifted toward the contralateral side if the pneumothorax is large ◦ Tactile fremitus is absent ◦ The percussion note is hypersonant ◦ The breath sounds are reduced or absent on the affected side ◦ The lower edge of the liver may be shifted inferiorly with a right-side pneumothorax 14
  • 16. Radiological manifestation cxray in erect position cxray in supine position 16
  • 21. 21
  • 22. Quantification of the size The simple method to estimate the size Small, a visible rim of < 2 cm between the lung margin and the chest wall Large, a visible rim of ≥2 cm between the lung margin and chest wall Light index Measure transverse Diameters of lung and Compare it with diameter hemithorax 22 22 Hemithorax (HT) Lung (L)
  • 23. CT scanning It is recommended in difficult cases such as patients in whom the lungs are obscured by overlying surgical emphysema To differentiate a pneumothorax from suspected bulla in complex cystic lung disease 23
  • 24. 24 CT can diagnose easily pneumothroax
  • 26. MANAGMENT  Goals ◦ To promote lung expansion ◦ To eliminate the pathogenesis ◦ To decrease pneumothorax recurrence  Treatment options according to ◦ Classification of pneumothorax ◦ Pathogenesis ◦ The extension of lung collapse ◦ Severity of disease ◦ Complication and concomitant underlying diseases 26
  • 27. TREATMENT OPTIONS FOR PSP AND SSP Observation O2 treatment Simple aspiration Small catheter aspiration Chest tube drainage Thoracoscopy (VAT with blebtomy &  VAT with pleurectomy) Open (axillary) thoracotomy 27
  • 28. Observation - PSP  Small, closed mildly symptomatic spontaneous pneumothorax.  Do not require hospital admission  It should be stressed to patient that they should be return directly to hospital in the event of developing breathlessness. 28
  • 29. Observation - SSP  Observation along is only recommend in patients with small SSP of less than 1 cm depth or isolated apical pneumothorax in asymptomatic patients  Hospitalization is recommended in these cases  All other cases will require active intervention ( aspiration or chest drain insertion) 29
  • 30. 30  Marked breathlessness in a patient with a small (<2 cm) PSP may develop tension pneumothorax  Observation is inappropriate and active intervation is required  If a patient is hospitalised for observation, supplemental high flow (10 l/min) oxygen should be given where feasible Observation - PSP or SSP
  • 31. 31  Inhalation of high concentration of oxygen may reduce the total pressure of gases in pleural capillaries by reducing the partial pressure of nitrogen  This should increase the pressure gradient between the pleural capillaries and the pleural cavity  Thereby increasing absorption of air from the pleural cavity O2 TREATMENT-- PSP or SSP
  • 32. 32  The rate of resolution/reabsorption of spontaneous pneumothorax is 1.25 – 1.8% of volume of hemithorax every 24 hours  The addition of high flow oxygen therapy has been shown to result in a 4-fold increase in the rate of peumothorax reabsorption during the periods of oxygen supplementation
  • 33. 33 Simple aspiration  Simple aspiration is recommended as first line treatment for all PSP requiring intervention  Simple aspiration is less likely to succeed in secondary pneumothoraces and in this situation, is only recommended as an initial treatment in small (<2 cm) pneumothoraces in minimally breathless patients under the age of 50 years  Patients with secondary pneumothoraces treated successfully with simple aspiration should be admitted to hospital and observed for at least 24 hours before discharge
  • 34. 34  Repeated aspiration is reasonable for primary pneumothorax when the first aspiration has been unsuccessful  A volume of < 2.5 L has been aspirated on the first attempt  The aspiration can be used by needle or catheter Catheter aspiration
  • 35. Intercostal tube drainage  INDICATIONS ◦ Unstable pneumothorax ◦ Severe dyspnea ◦ Large lung collapse ◦ Open or tension pneumothorax ◦ Recurrent pneumothorax ◦ Simple aspiration or catheter aspiration drainage is unsuccessful in controlling symptoms 35
  • 36. 36 36  The site of chest tube insertion is in the midclavicular line of second and third intercostal  or anterior axillary line of fifth and sixth intercostal Intercostal tube drainage
  • 37. 37 37 Intercostal tube drainage Fix the catheter and cover with gauze Making a small incision Using a forceps to extend the hole Inserting a catheter into pleural cavity
  • 38. 38 Trocar tube thoracostomy  Insertion of trocar into the pleural space.
  • 39. 39 Trocar tube thoracostomy  Insertion of the chest tube through the trocar
  • 40. 4 Operative tube thoracostomy The physician’s index finger is used to enlarge the opening and to explore the pleural space Placement of chest tube Intrapleurally using large hemostat
  • 41. 41 Observation of drainage  No bubble released ◦ The lung reexpansion ◦ The chest tube is obstructed by secretion or blood clot ◦ The chest tube shift to chest wall, the hole of the chest tube is located in the chest wall  If the lung reexpansion, removing the chest tube 24 hours after reexpansion.  Otherwise, the chest tube will be inserted again or regulated the position.
  • 42. 42 Complications of intercostal tube drainage  Penetration of major organs ◦ Lung, stomach, spleen, liver, heart and great vessels ◦ It occurs more commonly when a sharp metal trocar is inappropriately applied  Pleural infection ◦ Empyema, the rate of 1%  Surgical emphysema ◦ Subcutaneous emphysema
  • 43. 43 Chemical pleurodesis  Goals ◦ To prevent pneumothorax recurrence ◦ To produce inflammation of pleura and adhesions  Indications ◦ Persist air leak and repeated pneumothorax ◦ Bilateral pneumothorax ◦ Complicated with bullae ◦ Lung dysfunction, not tolerate to operation
  • 44. 44 Chemical pleurodesis  Sclerosing agents ◦ Tetracycline ◦ Doxycline ◦ Talc ◦ Erythromycin  The instillation of sclerosing agents into the pleural space lead to an aseptic inflammation with dense adhesions.
  • 45. 45  Methods ◦ Via chest tube or by surgical mean ◦ Administration of intrapleural local anaesthesia, 200 – 300 mg lidocaine intrapleurally injection ◦ Agents diluted by 60 – 100 ml saline ◦ Injected to pleural space ◦ Clamp the tube 4hours ◦ Drainage again ◦ Observed by chest X-ray film, if air of pleural space is absorption, remove the chest tube ◦ If pneumothorax still exist, repeated pleurodesis. Chemical pleurodesis
  • 46. 46 46  Side effct ◦ Chest pain ◦ Fever ◦ Dyspnea ◦ Acute respiratory distress syndrome ◦ Acute respiratory failure Chemical pleurodesis
  • 47. 47 Surgical treatment  Indications ◦ No response to medical treatment ◦ Persist air leak ◦ Hemopneumothorax ◦ Bilateral pneumothoraces ◦ Recurrent pneumothorax ◦ Tension pneumothorax failed to dainage ◦ Thicken pleura makes lung unable to reexpansion ◦ Multiple blebs or bullae
  • 48. Thoracoscopy- VATs  It is being increasly used in ot with recurrent psp-ssp  Recent studies show VATs it may be the procedure of choice and it has very less complication when compare to other open surgery  Pleural belbs causes pneumothorax which can be treated by a method called endostapling or suturefollowed by pleurodosis 48
  • 49. Axillary (open) thoracotomy  Transaxillary minithoracotomy is preferred .when VATs isnot available  Open thotacotomy with suturing of blebs and pleural abrasion is done  Recurrence  High risk professions  B/L or tension pneumothorax 49 INDICATIONS
  • 50. 50 Complications of pneumothorax  Pyopneumothorax ◦ Caused by aspiration or intercostal chest tube insertion (iatrogenic) ◦ Also results from necrotic pneumonia, lung abscess, or caseous pneumonia  Hydropneumothorax.  Hemopneumotorax ◦ Bleeding in pleural space. ◦ Common cause is rupture of vessels in adhesions. ◦ When lung re-expansion, bleeding will stop. ◦ When bleeding persists, surgical ligation will be needed.
  • 51. 51  Mediastinal and subcutaneous emphysema ◦ Alveoli rupture, the air enter into pulmonary interstitial and then goes into mediastinal and subcutaneous tissues. ◦ After aspiration or intercostal chest tube insertion, the air enters the subcutaneous by the needle hole or incision – surgical emphysema ◦ Physical exam – crepitus is present. Subcutaneous emphysema
  • 53. 53
  • 54. 54