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SHOCK
CIRCULATORY SHOCK
INTRODUCTION
• SHOCK IS A DECREASE BLOOD SUPPLY TO THE
TISSUE AND ORGANS OF THE BODY.
• IT IS A WHOLE BODY CIRCULATORY FAILURE.
• BODY TISSUES IS NOT GETTING ENOUGH BLOOD
FROM CIRCULATION.
• BLOOD FLOW TO THE TISSUE IS DANGEROUSLY
LOW AND ITS LEADS TO CELLULAR DAMAGE AND
FAILURE OF MULTIPLE ORGAN FAILURE.
DEFINITION
• SHOCK MAY BE DEFINED AS INADEQUATE DELIVERY OF
OXYGEN AND NUTRIENTS TO MAINTAIN NORMAL TISSUE
AND CELLULAR FUNCTIONS.
- SCHWARTZ’S
• IT IS A CLINICO-PATHOLOGICAL CONDITION IN WHICH
CARDIOVASCULAR SYSTEM COLLAPSED RESULTED IN
INADEQUATE TISSUE PERFUSION AND GENERALIZED
HYPOXIA TO THE MULTIPLE SYSTEMS OF THE BODY.
OBJECTIVES
• BASIC INTRODUCTION OF SHOCK
• DEFINITION OF SHOCK
• TO KNOW ABOUT STAGES OF SHOCK
• REVIEW ABOUT CLASSIFICATION OF SHOCK
• TO ELABORATE ABOUT ETIOLOGY AND CLINICAL
FEATURES
• MANAGEMENT ASPECT ABOUT SHOCK
ETIOLOGY
 BLOOD LOSS  CARDIAC
TEMPONADE
PLASMA LOSS  PULMONARY
EMBOLISM
 ELECTROLYTES IMBALANCE  TENSION
PNEUMOTHORAX
 VALVULAR DYSFUNCTION  AIR EMBOLISM
 MI  SPINAL ANAESTHESIA
 ARRHYTHMIAS  TRAUMA TO SPINAL
CLASSIFICATION
1.HYPOVOLEMIC SHOCK
2.CARDIOGENIC SHOCK
3.OBSTRUCTIVE SHOCK
4.DISTRIBUTIVE SHOCK
I. ANAPHYLATIC SHOCK
II. NEUROGENIC SHOCK
III. SEPTIC SHOCK
STAGES OF SHOCK
• 1. INITIATION STAGE :- CELLS BECOME LEAKY AND
SWITCH TO ANAEROBIC METABOLISM.
• 2. COMPENSATED STAGE :- ATTEMPT TO CORRECT
METABOLIC UPSET OF SHOCK VIA SNS REGULATION AND
RAAS.
• 3. DECOMPENSATED STAGE :- COMPENSATORY
MECHANISM WILL BE FAILED AND SHOCK IS IN MORE
PROGRESSIVE STAGE.
• 4. REFRACTORY STAGE :- ORGANS FAIL AND SHOCK CAN
NO LONGER BE REVERSED.
EFFECT ON ORGANS
• HEART :- TACHYCARDIA, DECREASE CO, HYPOTENSION.
• LUNGS :- TACHYPNEA, DECREASE GAS EXCHANGE,
PULMONARY EDEMA.
• ENDOCRINE :- INCREASE ADH, INCREASE SODIUM AND
WATER REABSORPTION.
• CNS :- DECREASE PERFUSION PRESSURE,
DROWSINESS.
• BLOOD :- COAGULATION ABNORMALITIES.
• RENAL :- DECREASE GFR, DECREASE UO.
CLINICAL FEATURES
• TACHYCARDIA (RAPID, WEAK, THREADY PULSE)
• BRADYCARDIA IN NEUROGENIC SHOCK
• TACHYPNEA (RAPID, SHALLOW RESPIRATION)
• HYPOTENSION
• COOL AND CALMY SKIN (WARM SKIN IN NEUROGENIC
SHOCK)
• OLIGURIA (< 30ML/HOURS)
• ANURIA
CONTINUED...
• DIZZINESS OR FAINTING
• CONFUSED AND DROWSINESS
• NAUSEA, VOMITING, DIARRHOEA
• THIRST, DRY MOUTH
• UNCONSCIOUSNESS
• MULTI ORGAN FAILURE
DIAGNOSTIC EVALUATION
• HISTORY COLLECTION
• PHYSICAL EXAMINATION
• BP MEASUREMENTS
• ECG
• ECHO
• X-RAY
• BLOOD TESTS LIKE ABG ANALYSIS, RFT, LFT, CBC, ESR,
TROP-I.
• ANGIOGRAPHY
MANAGEMENT OF SHOCK
• PHARMACOLOGICAL MANAGEMENT
• INOTROPES – DOBUTAMINE, DOPAMINE,
ADRENALINE.
• VASOPRESSOR – PHENYLEPHRINE, NE.
• FLUID REPLACEMENT – COLLOIDS AND
CRYSTALLOIDS.
• ANTICOAGULANT – HEPARIN.
• VASODILATOR – NTG.
CONTINUED...
• DIURETICS – FRUSEMIDE OR FUROSEMIDE.
• VASOCONSTRICTION – NE.
• ANTIHISTAMINE – CHLORPHENIRAMINE,
CETRIZINE.
• ANTIBIOTICS
• SPECIFIC GAMMA GLOBULIN
• CORTICOSTEROIDS
NURSING MANAGEMENT
• CAB – CIRCULATION, AIRWAY, BREATHING
• ASSESSMENT OF TISSUE PERFUSION
• VITAL SIGNS MONITORING
• PERIPHERAL PULSE
• LEVEL OF CONSCIOUSNESS
• CALILLARY REFILL
• ALLERGIES
• ONSET AND DURATION OF SYMPTOMS
CONTINUED...
• RESPONSE OF PATIENT GIVE CPR IF NEEDED
• LAY TCHECK HE PERSON FLAT AND FACE-UP
• DO NOMOVE PATIENT IF CEREBRAL AND SPINAL INJURY
IS SUSPECTED
• RAISE PERSON FEET ABOUT 12 INCHES
• CHECK SIGN OF CIRCULATION
• KEEP THE PERSON WT ARM AND COMFORTABLE
• NBM, IF PERSON WANTS WATER JUST MOISTEN LIPS
• IDENTIFY CAUSE AND TREAT ACCORDINGLY
THANK YOU🙏

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shock by Arpit Partil.pptx

  • 2. INTRODUCTION • SHOCK IS A DECREASE BLOOD SUPPLY TO THE TISSUE AND ORGANS OF THE BODY. • IT IS A WHOLE BODY CIRCULATORY FAILURE. • BODY TISSUES IS NOT GETTING ENOUGH BLOOD FROM CIRCULATION. • BLOOD FLOW TO THE TISSUE IS DANGEROUSLY LOW AND ITS LEADS TO CELLULAR DAMAGE AND FAILURE OF MULTIPLE ORGAN FAILURE.
  • 3. DEFINITION • SHOCK MAY BE DEFINED AS INADEQUATE DELIVERY OF OXYGEN AND NUTRIENTS TO MAINTAIN NORMAL TISSUE AND CELLULAR FUNCTIONS. - SCHWARTZ’S • IT IS A CLINICO-PATHOLOGICAL CONDITION IN WHICH CARDIOVASCULAR SYSTEM COLLAPSED RESULTED IN INADEQUATE TISSUE PERFUSION AND GENERALIZED HYPOXIA TO THE MULTIPLE SYSTEMS OF THE BODY.
  • 4. OBJECTIVES • BASIC INTRODUCTION OF SHOCK • DEFINITION OF SHOCK • TO KNOW ABOUT STAGES OF SHOCK • REVIEW ABOUT CLASSIFICATION OF SHOCK • TO ELABORATE ABOUT ETIOLOGY AND CLINICAL FEATURES • MANAGEMENT ASPECT ABOUT SHOCK
  • 5. ETIOLOGY  BLOOD LOSS  CARDIAC TEMPONADE PLASMA LOSS  PULMONARY EMBOLISM  ELECTROLYTES IMBALANCE  TENSION PNEUMOTHORAX  VALVULAR DYSFUNCTION  AIR EMBOLISM  MI  SPINAL ANAESTHESIA  ARRHYTHMIAS  TRAUMA TO SPINAL
  • 6. CLASSIFICATION 1.HYPOVOLEMIC SHOCK 2.CARDIOGENIC SHOCK 3.OBSTRUCTIVE SHOCK 4.DISTRIBUTIVE SHOCK I. ANAPHYLATIC SHOCK II. NEUROGENIC SHOCK III. SEPTIC SHOCK
  • 7.
  • 8.
  • 9. STAGES OF SHOCK • 1. INITIATION STAGE :- CELLS BECOME LEAKY AND SWITCH TO ANAEROBIC METABOLISM. • 2. COMPENSATED STAGE :- ATTEMPT TO CORRECT METABOLIC UPSET OF SHOCK VIA SNS REGULATION AND RAAS. • 3. DECOMPENSATED STAGE :- COMPENSATORY MECHANISM WILL BE FAILED AND SHOCK IS IN MORE PROGRESSIVE STAGE. • 4. REFRACTORY STAGE :- ORGANS FAIL AND SHOCK CAN NO LONGER BE REVERSED.
  • 10. EFFECT ON ORGANS • HEART :- TACHYCARDIA, DECREASE CO, HYPOTENSION. • LUNGS :- TACHYPNEA, DECREASE GAS EXCHANGE, PULMONARY EDEMA. • ENDOCRINE :- INCREASE ADH, INCREASE SODIUM AND WATER REABSORPTION. • CNS :- DECREASE PERFUSION PRESSURE, DROWSINESS. • BLOOD :- COAGULATION ABNORMALITIES. • RENAL :- DECREASE GFR, DECREASE UO.
  • 11. CLINICAL FEATURES • TACHYCARDIA (RAPID, WEAK, THREADY PULSE) • BRADYCARDIA IN NEUROGENIC SHOCK • TACHYPNEA (RAPID, SHALLOW RESPIRATION) • HYPOTENSION • COOL AND CALMY SKIN (WARM SKIN IN NEUROGENIC SHOCK) • OLIGURIA (< 30ML/HOURS) • ANURIA
  • 12. CONTINUED... • DIZZINESS OR FAINTING • CONFUSED AND DROWSINESS • NAUSEA, VOMITING, DIARRHOEA • THIRST, DRY MOUTH • UNCONSCIOUSNESS • MULTI ORGAN FAILURE
  • 13. DIAGNOSTIC EVALUATION • HISTORY COLLECTION • PHYSICAL EXAMINATION • BP MEASUREMENTS • ECG • ECHO • X-RAY • BLOOD TESTS LIKE ABG ANALYSIS, RFT, LFT, CBC, ESR, TROP-I. • ANGIOGRAPHY
  • 14. MANAGEMENT OF SHOCK • PHARMACOLOGICAL MANAGEMENT • INOTROPES – DOBUTAMINE, DOPAMINE, ADRENALINE. • VASOPRESSOR – PHENYLEPHRINE, NE. • FLUID REPLACEMENT – COLLOIDS AND CRYSTALLOIDS. • ANTICOAGULANT – HEPARIN. • VASODILATOR – NTG.
  • 15. CONTINUED... • DIURETICS – FRUSEMIDE OR FUROSEMIDE. • VASOCONSTRICTION – NE. • ANTIHISTAMINE – CHLORPHENIRAMINE, CETRIZINE. • ANTIBIOTICS • SPECIFIC GAMMA GLOBULIN • CORTICOSTEROIDS
  • 16. NURSING MANAGEMENT • CAB – CIRCULATION, AIRWAY, BREATHING • ASSESSMENT OF TISSUE PERFUSION • VITAL SIGNS MONITORING • PERIPHERAL PULSE • LEVEL OF CONSCIOUSNESS • CALILLARY REFILL • ALLERGIES • ONSET AND DURATION OF SYMPTOMS
  • 17. CONTINUED... • RESPONSE OF PATIENT GIVE CPR IF NEEDED • LAY TCHECK HE PERSON FLAT AND FACE-UP • DO NOMOVE PATIENT IF CEREBRAL AND SPINAL INJURY IS SUSPECTED • RAISE PERSON FEET ABOUT 12 INCHES • CHECK SIGN OF CIRCULATION • KEEP THE PERSON WT ARM AND COMFORTABLE • NBM, IF PERSON WANTS WATER JUST MOISTEN LIPS • IDENTIFY CAUSE AND TREAT ACCORDINGLY