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Obstructed Labor and Uterine
Rupture
Characteristic of Normal Labor
Friedman Curve
Abnormal Labor patterns
Abnormal Labor Diagnostic criteria
Nullipara Multipara
Prolongation disorder
Prolonged latent phase > 20 hours > 14 hours
Protraction disorder
Protracted active-phase dilatation < 1.2 cm/hr <1.5 cm/hr
Protracted descent < 1 cm/hr < 2 cm/hr
Arrest disorders
Prolonged deceleration phase > 3hours > 1hour
Secondary arrest of dilatation > 2 hours > 2 hours
Arrest of descent > 1 hour > 1 hour
Arrest of dilatation No descent in deceleration phage or
second stage
Why do abnormal pattern of Labor occur?
Fault in
Power Passage Passenger
Obstructed Labor
 Labor is considered obstructed when the
presenting part of the fetus cannot progress
into the birth canal, despite strong uterine
contractions because of insurmountable barrier
preventing its descent. WHO, 2003
 Usually occurs at the pelvic brim, but may
occur in the cavity or at the outlet.
 Accounts for 1-2% deliveries in referral
hospitals
 Obstructed labor accounts for 8% of all
maternal deaths in the developing world, and
plays a negligible role in all US maternal
deaths.
J Am Med Women Assoc, 2002
Causes of obstructed labor:
Passasge
 cephalopelvic disproportion
(small pelvis with normal
sized fetus)
 Contracted pelvis
Passage, Passenger
Power
Normal
Causes of obstructed labor:
Passasge
 Abnormality of bony
pelvis
Passage, Passenger
Power
Normal
Causes of obstructed labor:
Passasge
 abnormalities of the
reproductive tract
 pelvic tumour
 stenosis of cervix or
vagina
 tight perineum.
Passage, Passenger
Power
Normal
Causes of obstructed labor:
Passenger
Passage, Passenger
Power
Normal
 Cephalopelvic
disproportion (normal
sized pelvis with large
fetus)
 Abnormal lie
 Abnormal presentations,
e.g. – brow ,
shoulder, face with
chin posterior
 Abnormal position
Causes of obstructed labor:
Passenger
Passage, Passenger
Power
Normal
 Aftercoming head in
breech presentation
 fetal abnormalities,
e.g. – hydrocephalus
- fetal ascitis
- locked twins
Risk Factors for Obstructed Labor
1) early marriage and young age of mother
2) short height
3) previous prolonged labour
4) malnutrition or lack of exposure to sunlight,
resulting in rickets or osteomalacia
5) staff untrained to recognize obstructed labor
(partograph not used)
6) failure to act on risk factors and delay in referral
to higher level of care (e.g. for caesarean section)
7) long distance involved in obtaining skilled help,
lack of transport and communication
8) community distrust of health care personnel
Pathologic changes during Obstructed Labor
 In primigravida:
 Increasing frequency and intensity of uterine
contraction leads to state of “Uterine exhaution”→
Uterine Inertia
 In multigravida
 With tonic uterine contraction and retraction of
upper segment, the lower segment becomes
progressively thinner to accommodate fetus
 A groove is formed between active upper segment
and distended lower segment, called Bandl’s ring
 With persistent contraction and retraction→ Lower
segment rupture
Constriction Ring ( Schroeder’s
Ring): Result of incoordinate
uterine action
Pathologic changes during Obstructed Labor
 Compression of urethra between presenting
part and symphysis pubis, bladder gets
distended and becomes abdominal, trauma to
bladder wall and mucosa→ hematuria
Pathologic changes during Obstructed Labor

pressure necrosis→ fistula formation
Diagnosis of Obstructed Labor
1) Presence of risk factors
1) General Condition:
 Exhausted, anxious
 Tachycardia
 Blood pressure: Normal or Increased or Decreased
 Dehydrated
 Foul smelling breath ( ketotic smell)
 Scanty concentrated urine/ no urine output
Diagnosis of Obstructed Labor
3) Per abdomen
 Abdomen tender
 Abdominal contour
changed
 Fetal heart rate: normal
(rarely), abnormal or
absent
Diagnosis of Obstructed Labor
4) Local
 Vulva: edematous
 Per Vaginal:
 Vagina: hot, flushed
 Cervix: not well applied to
presenting part,
edematous
 Presenting part:
impacted, large caput
succedaneum, excessive
moulding
 Liquor: scanty or absent,
Meconium stained
Diagnosis of Obstructed Labor
Complications of Obstructed labor
Maternal:
 Maternal exhaustion
and dehydration→
metabolic acidosis
 Uterine rupture
 Postpartum
hemorrhage
 Puerperal Sepsis
 Maternal mortality
Late:
 Psychological trauma
 Genitourinary fistulae
 Vaginal stenosis
 Secondary
amenorrhea, infertility
 Neurological injury to
perineal nerve, foot
drop (compression of
lumbosacral plexus),
bladder (neurogenic
bladder)
Complications of Obstructed labor
Fetal:
 Excessive moulding of
fetal head→ Intracranial
hemorrhage
 Perinatal asphyxia
 Fetal death/ still birth
 Neonatal Sepsis
Management of obstructed labor
 Resuscitation: ABC
 Catheterization
 Arrange blood and blood products
 Arrange uterotonics
 Prepare for emergency cesarean section
 Explain the condition, plan of management and
posible complications to patient and her
relatives
 Pre-operative prophylaxis with broad spectrum
antibiotics
Management of obstructed labor
 Beware of bladder and ureteric injury
 Address intraoperative complication: extension
of uterine incision, atonic or traumatic PPH
 Continue broad spectrum antibiotics
postoperatively
 Continue catheter for at least 14 days
 Inspect vaginal mucosa before removing
catheter for slough, necrosis→ may need to
prolong catheter
 Counsel regarding possibility of fistula
Complications of Obstructed labor
Maternal:
 Maternal exhaustion and
dehydration→ metabolic
acidosis
 Uterine rupture
 Postpartum hemorrhage
 Puerperal Sepsis
 Maternal mortality
Late:
 Psychological trauma
 Genitourinary fistulae
 Vaginal stenosis
 Secondary amenorrhea
Fetal:
 Excessive moulding of
fetal head→ Intracranial
hemorrhage
 Perinatal ashyxia
 Fetal death
 Neonatal Sepsis
Prevention of Obstructed Labor
SAFE PREGNANCY
AND DELIVERY
Raising
Awareness
Medical Intervention
Capacity
Building
Proper
Antenatal
Care Labor
Monitoring
LSCS
Prevention of Obstructed Labor
Labor monitoring by
WHO partograph
Prevention of Obstructed Labor
 Referring and recognising women who are at
increased risk of obstructed labour.
 Women who are pregnant very young.
 Previous history of prolonged or obstructed labor.
 Antenatal care:
 Pelvimetry to rule out inadequate pelvis
 Identify women with abnormal lie, presentations and
referring to centre with emergency c-section.
Uterine Rupture
Full thickness disruption of uterine wall
i) Complete:
All layers of uterine wall are separated (the uterus
communicates directly with the peritoneal cavity)
ii) Incomplete:
Uterine muscle is separated but visceral peritoneum
is intact (rupture does not reach the visceral
peritoneum)
Also referred as uterine dehiscence
Uterine Rupture
Risk factors
1) Previous uterine scar
 Prior cesarean delivery
 Prior uterine rupture
 Myomectomy
 Uterine perforation
Prior Incision Estimated rate of rupture
One low transverse 0.2-0.9%
Multiple low transverse 0.9-1.8%
Low vertical 1-7%
T-shaped 4-9%
Classical 2-9%
Prior preterm CS Increased
Lower segment 2-6%
Upper segment 9-32%
Risk factors
2) Multiparity
3) Prolonged or obstructed labor
4) Difficult instrumental delivery
5) Intra uterine manipulation: ECV, internal
podalic version
6) Injudicious use of oxytocin
7) Fundal pressure during delivery
Diagnosis
 History:
 Previous uterine surgeries: LSCS, Myomectomy
 Previous history of rupture
 Obstructed labor
 Prolonged labor with difficult instrumental deliveries
 Clinical features
 Complain of sudden sharp pain with sensation of tear
inside
 Loss of labor pain, instead continuous pain
 Abdominal distension
 Per vaginal bleeding
 Hematuria
Diagnosis
 Patient’s condition deteriorate suddenly due to
massive blood loss
 Pale
 Tachycardia
 hypotension
 Sudden death if major vessels involved
 Abdominal examination:
 loss of uterine contour
 fetal parts easily palpable superficially
 PV examination:
 loss of presenting part ( the presenting part moves
higher)
Diagnosis
Alteration in fetal heart rate
 Fetal tachycardia
 Deceleration and bradycardia
 Absent fetal heart sounds
Management
 Resuscitation: ABC
 Catheterization
 Arrange blood and blood products
 Prepare for emergency laparotomy
 Explain the condition to patient and her
relatives, need of blood transfusion, emergency
laparotomy, possibility of hysterectomy if
rupture is irreparable, possible need of ICU
care, chances of sepsis postoperatively, risk of
mortality
 Pre-operative prophylaxis with broad spectrum
antibiotics
Management
 Emergency laparotomy under GA
 Midline vertical incision
 Deliver the baby and placenta
 Assess the site and size of the rupture
 Repairable: Repair
 Irreparable: Hysterectomy (Total/Subtotal)
 If the tear extends up to cervix and vagina:
Exploration of vagina and repair of tear.
 If associated bladder rupture: Repair
 If hemostasis cannot be obtained: Internal iliac
artery ligation
Management
 Continue broad spectrum antibiotics
 Blood and blood products ( PCV: FFP: PRP in 1:1:1)
 Continue catheter for 14 days
 Inspect anterior vaginal wall before removing
catheter: if necrotic, slough→ may need to prolong
the catheter
Contraception
 If family is complete:
 Perform tubal ligation with uterine repair
 If wants pregnancy in future:
 effective reversible contraception , at least for 5 years,
 Explain the risk of rupture in next pregnancy
Complications
 Hemorrhage → hypovolemic shock and its
complications (Coagulopathy, AKI), complications
related to transfusion
 Puerperal sepsis
 Maternal mortality (up to 30%)
 Perinatal mortality (10-80%)
Late complications
 Urogenital fistulae (VVF, ureteric)
 Secondary amenorrhoea, lactational failure
 Increased chances of rupture in future pregnancy
Prevention
1) Proper antenatal care
2) Identification of high risk patients
3) Labor monitoring
4) Judicious use of oxytocin
5) Avoid fundal pressure during labor
6) Always rule out genital tract injuries in
instrumental deliveries
Thank You !

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Obstructed labor and uterine rupture

  • 1. Obstructed Labor and Uterine Rupture
  • 2. Characteristic of Normal Labor Friedman Curve
  • 3. Abnormal Labor patterns Abnormal Labor Diagnostic criteria Nullipara Multipara Prolongation disorder Prolonged latent phase > 20 hours > 14 hours Protraction disorder Protracted active-phase dilatation < 1.2 cm/hr <1.5 cm/hr Protracted descent < 1 cm/hr < 2 cm/hr Arrest disorders Prolonged deceleration phase > 3hours > 1hour Secondary arrest of dilatation > 2 hours > 2 hours Arrest of descent > 1 hour > 1 hour Arrest of dilatation No descent in deceleration phage or second stage
  • 4. Why do abnormal pattern of Labor occur? Fault in Power Passage Passenger
  • 5. Obstructed Labor  Labor is considered obstructed when the presenting part of the fetus cannot progress into the birth canal, despite strong uterine contractions because of insurmountable barrier preventing its descent. WHO, 2003  Usually occurs at the pelvic brim, but may occur in the cavity or at the outlet.
  • 6.  Accounts for 1-2% deliveries in referral hospitals  Obstructed labor accounts for 8% of all maternal deaths in the developing world, and plays a negligible role in all US maternal deaths. J Am Med Women Assoc, 2002
  • 7. Causes of obstructed labor: Passasge  cephalopelvic disproportion (small pelvis with normal sized fetus)  Contracted pelvis Passage, Passenger Power Normal
  • 8. Causes of obstructed labor: Passasge  Abnormality of bony pelvis Passage, Passenger Power Normal
  • 9. Causes of obstructed labor: Passasge  abnormalities of the reproductive tract  pelvic tumour  stenosis of cervix or vagina  tight perineum. Passage, Passenger Power Normal
  • 10. Causes of obstructed labor: Passenger Passage, Passenger Power Normal  Cephalopelvic disproportion (normal sized pelvis with large fetus)  Abnormal lie  Abnormal presentations, e.g. – brow , shoulder, face with chin posterior  Abnormal position
  • 11. Causes of obstructed labor: Passenger Passage, Passenger Power Normal  Aftercoming head in breech presentation  fetal abnormalities, e.g. – hydrocephalus - fetal ascitis - locked twins
  • 12. Risk Factors for Obstructed Labor 1) early marriage and young age of mother 2) short height 3) previous prolonged labour 4) malnutrition or lack of exposure to sunlight, resulting in rickets or osteomalacia 5) staff untrained to recognize obstructed labor (partograph not used) 6) failure to act on risk factors and delay in referral to higher level of care (e.g. for caesarean section) 7) long distance involved in obtaining skilled help, lack of transport and communication 8) community distrust of health care personnel
  • 13. Pathologic changes during Obstructed Labor  In primigravida:  Increasing frequency and intensity of uterine contraction leads to state of “Uterine exhaution”→ Uterine Inertia  In multigravida  With tonic uterine contraction and retraction of upper segment, the lower segment becomes progressively thinner to accommodate fetus  A groove is formed between active upper segment and distended lower segment, called Bandl’s ring  With persistent contraction and retraction→ Lower segment rupture Constriction Ring ( Schroeder’s Ring): Result of incoordinate uterine action
  • 14. Pathologic changes during Obstructed Labor  Compression of urethra between presenting part and symphysis pubis, bladder gets distended and becomes abdominal, trauma to bladder wall and mucosa→ hematuria
  • 15. Pathologic changes during Obstructed Labor  pressure necrosis→ fistula formation
  • 16. Diagnosis of Obstructed Labor 1) Presence of risk factors 1) General Condition:  Exhausted, anxious  Tachycardia  Blood pressure: Normal or Increased or Decreased  Dehydrated  Foul smelling breath ( ketotic smell)  Scanty concentrated urine/ no urine output
  • 17. Diagnosis of Obstructed Labor 3) Per abdomen  Abdomen tender  Abdominal contour changed  Fetal heart rate: normal (rarely), abnormal or absent
  • 18. Diagnosis of Obstructed Labor 4) Local  Vulva: edematous  Per Vaginal:  Vagina: hot, flushed  Cervix: not well applied to presenting part, edematous  Presenting part: impacted, large caput succedaneum, excessive moulding  Liquor: scanty or absent, Meconium stained
  • 20. Complications of Obstructed labor Maternal:  Maternal exhaustion and dehydration→ metabolic acidosis  Uterine rupture  Postpartum hemorrhage  Puerperal Sepsis  Maternal mortality Late:  Psychological trauma  Genitourinary fistulae  Vaginal stenosis  Secondary amenorrhea, infertility  Neurological injury to perineal nerve, foot drop (compression of lumbosacral plexus), bladder (neurogenic bladder)
  • 21. Complications of Obstructed labor Fetal:  Excessive moulding of fetal head→ Intracranial hemorrhage  Perinatal asphyxia  Fetal death/ still birth  Neonatal Sepsis
  • 22. Management of obstructed labor  Resuscitation: ABC  Catheterization  Arrange blood and blood products  Arrange uterotonics  Prepare for emergency cesarean section  Explain the condition, plan of management and posible complications to patient and her relatives  Pre-operative prophylaxis with broad spectrum antibiotics
  • 23. Management of obstructed labor  Beware of bladder and ureteric injury  Address intraoperative complication: extension of uterine incision, atonic or traumatic PPH  Continue broad spectrum antibiotics postoperatively  Continue catheter for at least 14 days  Inspect vaginal mucosa before removing catheter for slough, necrosis→ may need to prolong catheter  Counsel regarding possibility of fistula
  • 24. Complications of Obstructed labor Maternal:  Maternal exhaustion and dehydration→ metabolic acidosis  Uterine rupture  Postpartum hemorrhage  Puerperal Sepsis  Maternal mortality Late:  Psychological trauma  Genitourinary fistulae  Vaginal stenosis  Secondary amenorrhea Fetal:  Excessive moulding of fetal head→ Intracranial hemorrhage  Perinatal ashyxia  Fetal death  Neonatal Sepsis
  • 25. Prevention of Obstructed Labor SAFE PREGNANCY AND DELIVERY Raising Awareness Medical Intervention Capacity Building Proper Antenatal Care Labor Monitoring LSCS
  • 26. Prevention of Obstructed Labor Labor monitoring by WHO partograph
  • 27. Prevention of Obstructed Labor  Referring and recognising women who are at increased risk of obstructed labour.  Women who are pregnant very young.  Previous history of prolonged or obstructed labor.  Antenatal care:  Pelvimetry to rule out inadequate pelvis  Identify women with abnormal lie, presentations and referring to centre with emergency c-section.
  • 28. Uterine Rupture Full thickness disruption of uterine wall i) Complete: All layers of uterine wall are separated (the uterus communicates directly with the peritoneal cavity) ii) Incomplete: Uterine muscle is separated but visceral peritoneum is intact (rupture does not reach the visceral peritoneum) Also referred as uterine dehiscence
  • 30. Risk factors 1) Previous uterine scar  Prior cesarean delivery  Prior uterine rupture  Myomectomy  Uterine perforation Prior Incision Estimated rate of rupture One low transverse 0.2-0.9% Multiple low transverse 0.9-1.8% Low vertical 1-7% T-shaped 4-9% Classical 2-9% Prior preterm CS Increased Lower segment 2-6% Upper segment 9-32%
  • 31. Risk factors 2) Multiparity 3) Prolonged or obstructed labor 4) Difficult instrumental delivery 5) Intra uterine manipulation: ECV, internal podalic version 6) Injudicious use of oxytocin 7) Fundal pressure during delivery
  • 32. Diagnosis  History:  Previous uterine surgeries: LSCS, Myomectomy  Previous history of rupture  Obstructed labor  Prolonged labor with difficult instrumental deliveries  Clinical features  Complain of sudden sharp pain with sensation of tear inside  Loss of labor pain, instead continuous pain  Abdominal distension  Per vaginal bleeding  Hematuria
  • 33. Diagnosis  Patient’s condition deteriorate suddenly due to massive blood loss  Pale  Tachycardia  hypotension  Sudden death if major vessels involved  Abdominal examination:  loss of uterine contour  fetal parts easily palpable superficially  PV examination:  loss of presenting part ( the presenting part moves higher)
  • 34. Diagnosis Alteration in fetal heart rate  Fetal tachycardia  Deceleration and bradycardia  Absent fetal heart sounds
  • 35. Management  Resuscitation: ABC  Catheterization  Arrange blood and blood products  Prepare for emergency laparotomy  Explain the condition to patient and her relatives, need of blood transfusion, emergency laparotomy, possibility of hysterectomy if rupture is irreparable, possible need of ICU care, chances of sepsis postoperatively, risk of mortality  Pre-operative prophylaxis with broad spectrum antibiotics
  • 36. Management  Emergency laparotomy under GA  Midline vertical incision  Deliver the baby and placenta  Assess the site and size of the rupture  Repairable: Repair  Irreparable: Hysterectomy (Total/Subtotal)  If the tear extends up to cervix and vagina: Exploration of vagina and repair of tear.  If associated bladder rupture: Repair  If hemostasis cannot be obtained: Internal iliac artery ligation
  • 37. Management  Continue broad spectrum antibiotics  Blood and blood products ( PCV: FFP: PRP in 1:1:1)  Continue catheter for 14 days  Inspect anterior vaginal wall before removing catheter: if necrotic, slough→ may need to prolong the catheter Contraception  If family is complete:  Perform tubal ligation with uterine repair  If wants pregnancy in future:  effective reversible contraception , at least for 5 years,  Explain the risk of rupture in next pregnancy
  • 38. Complications  Hemorrhage → hypovolemic shock and its complications (Coagulopathy, AKI), complications related to transfusion  Puerperal sepsis  Maternal mortality (up to 30%)  Perinatal mortality (10-80%) Late complications  Urogenital fistulae (VVF, ureteric)  Secondary amenorrhoea, lactational failure  Increased chances of rupture in future pregnancy
  • 39. Prevention 1) Proper antenatal care 2) Identification of high risk patients 3) Labor monitoring 4) Judicious use of oxytocin 5) Avoid fundal pressure during labor 6) Always rule out genital tract injuries in instrumental deliveries