Call Girls Madurai Just Call 9630942363 Top Class Call Girl Service Available
Cataract complications
1. Complications of Cataract Surgery
Prof. Naimatullah Khan Kundi
Head, Department of Ophthalmology
Khyber Medical College Peshawar
2. Complications of cataract surgery
Complications are varied in time and
scope
1. Intraoperative
2. Immediate postoperative
3. Late postoperative
Therefore it is necessary to observe the
postoperative patients at periodic intervals
3. Major postoperative complications
of
cataract surgery
Endophthalmitis
Expulsive Haemorrhage
Corneal edema
Delayed choroidal haemorrhage
Wound distortion or disruption
Hyphaema
Shallow or flat anterior chamber
Elevated IOP
Corneal edema
Glaucoma
Detachment of descemet’s
Malignant glaucoma
membrane
Retained lens material
Suprachoroidal haemorrhage or
effusion
4. Major postoperative complications
of
cataract surgery
•
Vitreous disruption incarceration in the wound
•
Suture induced astigmatism
•
Pupillary capture
•
Complications of IOL implantation
•
Uveitis
•
IOL dislocation
•
Hemorrhage
•
Retinal detachment
•
Cystoid macular edema
•
Retianed lens material
•
Capsular rupture
•
Vitreous loss
6. Acute bacterial endophthalmitis
Incidence - about 1:1,000
Common causative
organisms
• Staph. epidermidis
• Staph. aureus
• Pseudomonas sp.
Source of infection
•
Patient’s own external
bacterial flora is most
frequent culprit
•
Contaminated solutions
and instruments
Environmental flora including
that of surgeon and
operating room personnel
•
7. Signs of severe endophthalmitis
• Pain and marked visual loss
• Absent or poor red reflex
• Corneal haze, fibrinous exudate and • Inability to visualize fundus with
hypopyon
indirect ophthalmoscope
8. Signs of mild endophthalmitis
• Mild pain and visual loss
• Anterior chamber cells
• Small hypopyon
• Fundus visible with indirect
ophthalmoscope
9. Management of Acute Endophthalmitis
1. Preparation of intravitreal injections
2. Identification of causative organisms
• Aqueous samples
• Vitreous samples
3. Intravitreal injections of antibiotics
4. Vitrectomy - only if VA is PL
5. Subsequent treatment
10. Subsequent Treatment
1. Periocular injections
• Vancomycin 25 mg with ceftazidime 100 mg
or gentamicin 20 mg with cefuroxime 125 mg
• Betamethasone 4 mg (1 ml)
2. Topical therapy
• Fortified gentamicin 15 mg/ml and vancomycin 50 mg/ml drops
• Dexamethasone 0.1%
3. Systemic therapy
• Antibiotics are not beneficial
• Steroids only in very severe cases
11. Major postoperative complications of
cataract surgery
Corneal edema
Detached Descemet’s membrane
Mechanical trauma
Vitreo-endothelial touch
IOL-endothelial touch
Toxic solutions
12. Major postoperative complications of
cataract surgery
Wound distortion or disruption
Astigmatism
Wound leak
Inadvertent filtering bledb
Iris prolapse
Hypotony
13. Major postoperative complications of
cataract surgery
Shallow or flat anterior chamber
Wound leak
Choroidal detachment or hemorrhage
Pupillary block
Ciliary block
14. Shallow or flat AC
A. Intraoperative
1. Inadequate infusion of BSS
2. Leakage over sized wound
3. External pressure on the globe
4. Positive vitreous pressure more common in:
Obese
Bull necked pts.
COPD
Anxious Pts. Who perform valsalva maneuver
5. Supachoroidal haemorrhage or effusion
15. Shallow or flat AC (cont’d)
Intraoperative shallow AC
Management
Raise infusion bottle
Place suture across the wound to ↓ its size
External pressure: Readjust surgical drapes or eye
lid speculum
Positive vitreous pressure:
I/V manitol ↓ the ↑ positive pressure and
Allow the case to continue uneventfully
Suprachoroidal hemorrhage or effusion:
Check red reflex
Examine fundus with indirect ophthalmoscope to confirm
diagnosis
16. Shallow or flat AC (cont’d)
B. Postoperative shallow AC
Postoperative shallow AC → opposition of iris to
angle → PAS → chronic ACG
Irido-vitreal (ICCE) / irido-capsular (ECCE)
synechiae
→ pupillary block
Corneal contact with vitreous / IOL → endothelial
cell loss → chronic corneal edema
17. Shallow or flat AC (cont’d)
B. Postoperative shallow AC
Causes
1.
2.
Choroidal detachment
3.
Pupillary block
4.
Ciliary block
5.
Wound leak
Suprachoroidal hemorrhage
Cases associated with ocular hypotension are 2ndry to wound
leakage / choroidal detachment
Slow or intermittent wound leaks may coexist with formed AC
18. Shallow or flat AC (cont’d)
B. Postoperative shallow AC
Seidel Test:
To detect an area of wound leakage
Instill one drop of 2% fluorescein and examine the incision
with cobalt blue filter on the SL
Aqueous dilution of fluorescein at the site of leakage will
produce contrasting area of green stain
Occasionally aqueous flow is so slight that gentle pressure on
the globe is necessary to confirm the site of leakage
19. Postoperative shallow AC (cont’d)
Management
Several Options
1.
Cycloplegics and pressure patching
2.
CAI and topical beta blockers: ↓ Aqueous flow through
the woung
3.
Corticosteroid avoidence: Enhance local wound
reaction to faciliatte spontaneous closure
4.
Therapeutic contact lens help in opposing wound
edges and ↓ aqueous flow through the wound
5.
Tissue adhesive: may seal the wound
6.
Surgical
20. Postoperative shallow AC (cont’d)
Management
These measures are appropriate for minor wound
leaks
Many patients develop associated ciliochoroidal
detachment which resolves spontaneously after
wound closure
21. Postoperative shallow AC (cont’d)
Management
Surgical approach with reformation of AC and
wound repair indicated:
If no improvement occurs in 24 – 46 hours
If obvious wound separation is present
Iris prolapse
IOL contact with corneal endothelium
22. Postoperative shallow AC(cont’d)
Complications (shallow AC)
Early postoperative Pupillary block glaucoma may follow
resolved wound leak
Late Pupillary block glaucoma is caused by postoperative
uveitis with irido-vitreous / irido-capsular synechiae
formation
AC IOL Placement without PI may be associated with early
or late postoperative pupillary block glaucoma
Ciliary block glaucoma caused by aqueous sequestration
within the vitreous body with flat AC & ↑ IOP
23. Postoperative shallow AC (cont’d)
Pupillary block glaucoma
Treatment
1. Pupillary dilation
2. Laser or surgical iridotomy
3. Vitretcomy preferred treatment for ciliary
block glaucoma
24. Corneal edema
Factors:
↑IOP
Endothelial cell damage
Edema in the immediate postoperative period
Incidence is increased in preexisting endothelial
Dysfunction
Acute endothelial decompensation with increase in
corneal thickness
25. Corneal edema (cont’d)
Causes:
1. Mechanical trauma
2. Prolonged intraocular irrigation
3. Inflammmation
4. Increased IOP
Resolves in 4 – 6 weeks
Corneal edema persisting after 3 months will usually
not clear and may require penetrating keratoplasty
26. Brown McLean Syndrome
This clinical condition occurs after cataract surgery
(most frequently ICCE)
Etiology unknown
Consists of peripheral corneal edema with clear
central cornea
Edema typically starts inferiorly and progresses
circumfrentially but spares the central cornea
It rarely progresses to clinically significant central
corneal edema
27. Vitreo-corneal adherence and persistent
corneal edema
Early / late
Uncomplicated ICCE or complicated ECCE
Early recognition and treatment are essential to
prevent development of irreversible corneal edema
Treatment:
1. Anterior vitrectomy (Limbus / PP)
2. Penetrating keratoplasty with vitrectomy in more
advanced cases
28. Tixic solutions
Certain solutions can be toxic to corneal
endothelium when:
Irrigated
Inadvertently injected
Cause:
Temporary
Permanent
Into AC
Corneal edema
29. Corneal complications of phacoemulsification
Heat: transferred from the vibrating probe to the cornea
Tight wound prevents adequate irrigation fluid along
the probe
Occlusion of irrigation / aspiration tubing
Holding phaco tip too close to the corneal endothelium:
30. Corneal complications of phacoemulsification
The US energy causes:
Injury to cornea
Loss of endothelial cell
C. Edema on 1st postoperative
day/delayed for months to years
In corneal edema develops during:
Phacoemulsification and
Decreases visualization
Convert to nuclear expression
technique
31. Detachment of descemet’s membrane
Results in stromal swelling and epithelial bullae
localized in the area of detachment
Causes:
When Instrument / IOL is introduced through
cataract incision.
Inadvertent fluid injection between descemet’s
membrane and stroma
Treatment:
Small detachments can be reattached with air
tamponade in AC
Large detachments can be sutured back into place
32. Suprachoroidal haemorrhage or effusion
Occurs intraoperatively
Choroidal effusion with or without suprachoroidal
haemorrhage
Choroidal effusion may be difficult to differentiate from
choroidal haemorrhage (clinically)
Both complications may occur in patients with:
HT
Obesity
Glaucoma
Chronic ocular inflammation
33. Suprachoroidal haemorrhage or effusion
Choroidal effusion may be precursor of suprachoroidal
haemorrhage
Or haemorrhage may represent spontaneous rupture of
choroidal vasculature (in patients with underlying vascular
disease)
Choroidal effusion tents veins and arteries that course
through sclera and supply choroid
Disruption of these vessels lead to suprachoroidal
haemorrhage
(cont’d)
34. Suprachoroidal haemorrhage or effusion
(cont’d)
Treatment:
Rapid wound closure with elevation of IOP to tamponade
the extravasated plasma or blood
Sclerostomy in one or more quadrants posterior to ora
serrata to drain blood
Elevated IOP serves both to stop bleeding and to
extravasate suprachoroidal blood
35. Expulsive Haemorrhage
Rare but serious intraoperative problem
Requires immediate action
Presentation:
Sudden ↑ IOP
Darkening of red reflex
Wound gap
Iris prolapse
Expulsion of lens and vitreous
Bright red blood
36. Expulsive Haemorrhage (cont’d)
Treatment:
Immediate closure of the wound with sutures /
digital pressure
Perform posterior selerotomies (5 – 7 mm
posterior to limbus) to permit suprachoroidal
haemorrhage blood to escape and allow
repositioning of prolapsed intraocualr tissues
and closure of the wound
37. Delayed choroidal haemorrhage
Early postoperative period (less common)
Presentation:
Sudden onset of pain
Loss of vision
Shallow AC
↑ IOP
Wound intact / disrupted
38. Delayed choroidal haemorrhage
(cont’d)
Management:
Observation:
If wound intact and IOP controlled, limited
haemorrhage may be observed and resolve
spontaneously
Surgical drainage:
1.
2.
3.
4.
5.
Wound disruption
Persistent shallow AC
Uncontrolled glaucoma
Adherent choroidals (kissing)
Persistent choroidal detachment
40. Hyphaema
Early / Late
Early:
Immediate postoperative period
Late:
Origin: Incision / Iris
Mild resolves spontaneously
Mixed with blood / viscoelastic –
resolution longer
Months / years after surgery
Origin: wound vascularization / erosion
of vascular tissue by lens implant
41. Hyphaema (cont’d)
Complications (prolonged hyphaema):
↑ IOP
Corneal blood staining
Management:
IOP monitored closely and treated in the usual medial
fashion
Argon laser photocoagulation of the bleeding vessels
stop / prevent rebleeding
With-holding antiplatelet therapy (Those who receive)
until hyphaema resolves. Also risk of continued /
recurrent bleeding reduced
42. Elevated IOP
Mild and selflimiting
Significant and sustained
Causes:
Retained viscoelastic material in AC, PC, behind the IOL]
Pupillary block
Ciliary block
Hyphaema
Endophthalmitis
Retained lens material (phacolytic / phacoanaphylatic
reaction)
Iris pigment release
Preexisting glaucoma
Corticosteroid usage
PAS (early postoperative flat AC when eye inflammed) →
2ndry glaucoma
43. Elevated IOP
(cont’d)
Treatment:
Mild and selflimiting:
Does not require prolonged anti-glaucoma
therapy
IOP elevation lasts for a few days and is
amenable to medical treatment
Significant and sustained rise of IOP:
May necessitate timely and specific
management in several circumstances
Treat the underlying cause of IOP elevation
44. Malignant glaucoma (ciliary block glaucoma
Posterior dissection of aqueous into the vitreous body
and 2ndry rise of IOP
IOP rise may occur inspite of patent iridectomy
Treatment
Cycloplegics – to move lens-iris diaphragm posteriorly
Disruption of anterior hyaloid face and vitreous to reestablish
a channel for aqueous to come forward
Techniques:
Mechanical disruption (knife)
ND: YAG Laser
PPV
45. Retained lens material
Small lens material (cortical) better tolerated
and require no surgical intervention
More likely resorb over time
Nuclear material incite significant inflammatory
reaction
Inflammatory reaction may be difficult to
differentiate from microbial endophthalmitis
47. Retained lens material (cont’d)
Treatment:
Surgical intervention
Large amount of lens material
Inflammation not controlled by topical
medication
2ndry hypotony / increased IOP from
inflammation
PC intact:
Simple aspiration
PC ruptured: (Potential for lens-vitreous admixture)
Vitrectomy
48. Vitreous disruption incarceration in the wound
Rupture of anterior vitreous face (ICCE/ECCE)
Anterior migration through pupil
Vitreous traction: - Retinal breaks and RD
Vitreous incarceration in the wound → chronic ocular
inflammation with / without CME
Vitreous transparent, its presence datected by: Touching / Manipulating the wound / iris with sponge or spatula:Adherent vitreous becomes apparent / cause movement of the
pupil
49. Vitreous disruption incarceration in the wound (cont’d)
Management
Cutting vitreous strands and removed by suction cutter /
cellulose sponges
ND:YAG laser / anterior vitrecotmy
PPV:
Chronic ocular
inflammation with CME
and vitreous incarcerated
in the wound
if cornea shows considerable compromise (to
reduce surgical trauma)
50. Suture induced astigmatism
Tight sutures:
post-operative astigmatism, Steepens the cornea
in the direction of sutures
Removing sutures 6 – 8 wks postoperatively
may alleviate astigmatism
Wound leak:
Significant against the rule astigmatism
Secondary intra-ocular infection:
Entry of organisms into the eye through suture
tract
51. Pupillary capture
Causes
PS (Iris and PC Adhesions)
Improper placement of IOL haptics
Anterior displacement of PC IOL optic (non
angulated IOL in ciliary sulcus)
Inadvertent flipping over of angulated IOL so
it angles anteriorly
Positive vitreous pressure from behind the
optic of IOL
52. Pupillary capture (cont’d)
Management
Asymptomatic:
Problem cosmetic – patient can be left untreated
Occasionally glare, photophobia, monocular diplopia
In bag placement has decreased the occurrence of
pupillary capture
Symptomatic:
Pharmacological manipulation of pupil with mydriatics to
free iris
Surgical intervention – free iris / break synechiae
54. Complications of IOL implantation
1.
Decentration and dislocation
2.
Uveitis – glaucoma – hyphaema (UGH)
syndrome
3.
Corneal edema and pseudo-phakic
bullous keratopathy
4.
Wrong power IOL
55. Complications of IOL implantation (cont’d)
Decentration and dislocation
Causes
Asymmetric hapitc placemnt:
One in bag and other in sulcus. IOL designed for
bag fixation prone to decentration / dislocation
when one / both haptics are placed in sulcus
Insufficient zonular support
Irregular fibrosis of posterior capsule
56. Complications of IOL implantation (cont’d)
Decentration and dislocation
(cont’d)
Management
Rotation of IOL
Reposition IOL haptics
Replace capsule fixated IOL with PC sulcus
fixated IOL
IOL exchange with AC IOL / Trans-sclerally
sutured PC IOL (complete IOL dislocation)
57. Complications of IOL implantation (cont’d)
Uveitis-glaucoma-hyphaema (UGH) syndrome
UGH syndrome was first described in the context of
rigid AC IOLs
Classic triad (UGH) or individual elements may occur
Causative Factors:
1. Inappropriate IOL size
2. Contact between implant and vascular structures
3. Defects in implant manufacturing
4. Idiosyncretic reaction of patient to implant
58. Complications of IOL implantation (cont’d)
Uveitis-glaucoma-hyphaema (UGH) syndrome
Treatment
Topical anti-inflammatory medications
Topical anti-glaucoma medications
IOL removal (symptoms not alleviated / threaten
retinal or corneal function)
59. Complications of IOL implantation (cont’d)
Corneal edema and pseudo-phalic bullous
keratopathy
Causes
1.
2.
3.
4.
5.
Surgical trauma
IOL type: - Iris fixated / closed loop flexible AC IOL
Vitreous contact with corneal edothelium
Glaucoma
Corneal endothelial dystrophy (Fuchs) – increased
risk of developing postoperative corneal edema
even after smooth, a traumatic surgery
60. Complications of IOL implantation (cont’d)
Corneal edema and pseudo-phalic bullous
keratopathy
Symptoms
1.
2.
3.
4.
5.
Corneal edema → BK
↓ VA
Irritation
FB Sensation
Epiphora
6. Infective keratitis (occasionally)
62. Complications of IOL implantation (cont’d)
Wrong power IOL
A.
1. Miscalculation
2. Manufacturing defect
B. If magnitude of implant error produce symptomatic
anisometropia; replace IOL with appropriate power