brief account on fish disease myxospiridia and trichordiniasis

1. Submitted By:- Soumya Ranjan Dutta
1708041230004
B.F.Sc. 5th sem, 3rd year
Submitted to:- Mr Abhijit Mallick
Asst. Prof. , MSSSOA
Centurion University

2. TRICHODINIASIS

3. Causative Agent :- Species of genus tricordinella, tricodina
Mostly Affects on:- Indian major carps and Catfishes
Taxonomy of the causative agent:-
Domain: Eukaryota
Phylum: Ciliophora
Subphylum: Intramacronucleata
Class: Oligohymenophorea
Subclass: Peritrichia
Order: Mobilida
Family: Trichodinidae
Genus: Trichodina

5. Epidemic condition:-
The disease is more likely to occur in small, shallow ponds .
Poor water quality
High stocking density
Continuous rain condition
Symptoms :-
Can infect fish at any developmental stage
 Trichodina mainly invades the skin of juvenile fish, feeding on the tissue,
and destroying the skin.
Trichodinelia mainly parasitizes the gills,
Gill and Fin rot
Hyperplasia

6. Signs:-
Appeared a creamish coating on gill.
Mucus secreation

7. Treatment and Doses:-
Water quality improvement.
Diminish the stocking density.
 8ppm copper sulphate solution for 20–30 min
The reproduction of Trichodina can be inhibited by adding 15–20 kg/mu of
chinaberry (Melia azedarach) leaves once a week.
Treat the pond with a mixture of copper sulphate and ferrous sulphate (5:2)
to a concentration of 0.7 ppm.
Formalin at a concentration of 30 ppm can kill Trichodina on juvenile eel
gill.

8. MYXOSPOREAN
Causative agent:- Myxobolus bengalensis, M. catalae, M.
hosadurgensis
Mostly affects on :- Indian major carps
Taxonomy
Kingdom: Animalia
Phylum: Cnidaria
Class: Myxozoa
Order: Bivalvulida
Family: Myxobolidae
Genus: Myxobolus

9. Structure
A spore of Myxosporidia consists of two identical chitinous shells
surrounding plasmodium. The line where the two shells join is called the
sutural line. There is a ridge along the sutural line called the sutural ridge.
The side with the sutural ridge is called the sutural side or lateral side. The
side without the sutural ridge is called the shell side or front side. The
plasmodium consists of a polar capsule(s) and the sporolasm.
1-Sporoshell, 2-polar vacuole,3-polar filament, 4-embrvonic nucleus
5-iodiophilous vacuole, 6-sutural line, 7-polar vacuole pore

10. Life cycle
Its life occurs in 2 host primary host is annelids and secondary host is fish
The inactive stage is ingested by the annelid
Then spores comes out called amoebulla and penetrate the gut
Then become trophozoit and form cyst
Cyst formation is occurs in the host body.
Then it comes out by faecal matter and attacked fish epidermis
Then it causes the disease and multiplies on fish body.

12. Symptoms:-
Lethargic
Blocking of respiratory tract
Excessive mucus secretion
Hyperplasia
Perforated scales
ulceration
Epidemic condition:-
The disease is more likely to occur in small, shallow ponds .
Poor water quality
High stocking density

13. Control measures:-
Control spores entering the fish pond
Segregation of age group of fishes
Treatment:-
Eradicate the spore by sterilizing the pond with 100 kg/mu of nitrolime
(CaN2) or 125 kg/mu of quicklime
 In the fry and fingerling stages sprinkle dipterex powder into the pond to
a concentration of 5 ppm twice monthly.
Calcium oxcide treatment after dewatering and dry for 1 month
Source
Handbook of Fisheries & Aquaculture,FAO