Publications sima hadidi
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This document lists 13 scientific publications that involve researcher S. Hadidi. The publications span from 2000-2013 and cover topics such as: identifying genetic factors related to disease resistance in rainbow trout; examining the role of the protein Ship1 in regulating immune responses to helminth infections; investigating the functions of the histone methyltransferase G9a in T cell differentiation; studying genetic selection for bacterial cold water disease resistance in trout; identifying Toll-like receptor genes in trout; and exploring the role of spleen size in disease resistance of trout. Many publications also involve examining the regulatory functions of the protein MD-1.
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This document lists 44 original publications with their respective impact factors. The publications are grouped by year and cover kidney disease research from 1998-2006. They examine topics like the expression of chemokines and their receptors in various human kidney diseases, animal models of glomerulonephritis, and the role of toll-like receptors in immune complex glomerulonephritis. The impact factors range from 0.3 to 7.1, with most papers published in high impact journals like the Journal of the American Society of Nephrology.
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2. One study immunized rabbits with the BCG vaccine, which contains HSP-65, and found higher levels of atherosclerotic plaques in immunized rabbits compared to controls. This suggests HSP-65 immunity may enhance atherosclerosis.
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1. The document discusses the potential role of heat shock protein 65 (HSP-65) as an autoantigen in the development of atherosclerotic plaques. Several studies have found associations between antibodies to HSP-65 and various measures of atherosclerosis.
2. One study immunized rabbits with the BCG vaccine, which contains HSP-65, and found higher levels of atherosclerotic plaques in immunized rabbits compared to controls. This suggests HSP-65 immunity may enhance atherosclerosis.
3. However, the role of HSP-65 is unclear - it may be more important for plaque vulnerability rather than generation, and different antigens could be involved at different stages or between patients.
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II. One study found that RAG-1 deficient mice, which lack functional B and T cells, exhibited increased neointimal formation following arterial injury compared to wild type mice. Transferring B cells from wild type mice to RAG-1 deficient mice reduced neointimal formation.
III. Overall, the studies suggest that B cells may play a protective role against atherosclerosis by reducing plaque formation and progression. The mechanism may involve antibodies that target oxidation-specific epitopes, but this requires further investigation.
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I. The document summarizes several studies that investigated the role of the immune system, particularly B cells, in atherosclerosis using mouse models.
II. One study found that RAG-1 deficient mice, which lack functional B and T cells, exhibited increased neointimal formation following arterial injury compared to wild type mice. Transferring B cells from wild type mice to RAG-1 deficient mice reduced neointimal formation.
III. Overall, the studies suggest that B cells may play a protective role against atherosclerosis by reducing plaque formation and progression. The mechanism may involve antibodies that target oxidation-specific epitopes, but this requires further investigation.
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This document lists 44 original publications with their respective impact factors. The publications are grouped by year and cover kidney disease research from 1998-2006. They examine topics like the expression of chemokines and their receptors in various human kidney diseases, animal models of glomerulonephritis, and the role of toll-like receptors in immune complex glomerulonephritis. The impact factors range from 0.3 to 7.1, with most papers published in high impact journals like the Journal of the American Society of Nephrology.
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1. The document discusses the potential role of heat shock protein 65 (HSP-65) as an autoantigen in the development of atherosclerotic plaques. Several studies have found associations between antibodies to HSP-65 and various measures of atherosclerosis.
2. One study immunized rabbits with the BCG vaccine, which contains HSP-65, and found higher levels of atherosclerotic plaques in immunized rabbits compared to controls. This suggests HSP-65 immunity may enhance atherosclerosis.
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1. The document discusses the potential role of heat shock protein 65 (HSP-65) as an autoantigen in the development of atherosclerotic plaques. Several studies have found associations between antibodies to HSP-65 and various measures of atherosclerosis.
2. One study immunized rabbits with the BCG vaccine, which contains HSP-65, and found higher levels of atherosclerotic plaques in immunized rabbits compared to controls. This suggests HSP-65 immunity may enhance atherosclerosis.
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I. The document summarizes several studies that investigated the role of the immune system, particularly B cells, in atherosclerosis using mouse models.
II. One study found that RAG-1 deficient mice, which lack functional B and T cells, exhibited increased neointimal formation following arterial injury compared to wild type mice. Transferring B cells from wild type mice to RAG-1 deficient mice reduced neointimal formation.
III. Overall, the studies suggest that B cells may play a protective role against atherosclerosis by reducing plaque formation and progression. The mechanism may involve antibodies that target oxidation-specific epitopes, but this requires further investigation.
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I. The document summarizes several studies that investigated the role of the immune system, particularly B cells, in atherosclerosis using mouse models.
II. One study found that RAG-1 deficient mice, which lack functional B and T cells, exhibited increased neointimal formation following arterial injury compared to wild type mice. Transferring B cells from wild type mice to RAG-1 deficient mice reduced neointimal formation.
III. Overall, the studies suggest that B cells may play a protective role against atherosclerosis by reducing plaque formation and progression. The mechanism may involve antibodies that target oxidation-specific epitopes, but this requires further investigation.
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Meningitis is a medical emergency that requires a multi-disciplinary approach. Tuberculous meningitis (TBM) is one of the most common types and if not diagnosed and treated promptly, it can cause permanent neurological damage in 25% of patients. Adenosine deaminase (ADA) is an enzyme involved in purine metabolism that is released by T cells during the immune response to TBM. Measuring ADA levels in cerebrospinal fluid is a simple, cost-effective and rapid way to help diagnose TBM with high sensitivity of 84.21%, specificity of 95.45%, and diagnostic accuracy of 71.32%.
ANA498Y_Final
This study aimed to differentiate between invasive squamous cell carcinoma (SCC) and carcinoma-in-situ (CIS) of the uterine cervix using immunohistochemical markers. Tissue samples from 37 patients were stained for CD3, CD4, CD8, CD20, and CD138. Scoring showed decreased expression of CD8, CD20, and CD138 in invasive SCC compared to CIS, indicating differences in immune cell infiltration between invasive and non-invasive lesions. The results provide potential markers for differentiating invasive SCC from CIS of the cervix.
A Report of the Alopecia Areata Registry, Biobank and Clinical Trials Network...
A Report of the Alopecia Areata Registry, Biobank and Clinical Trials Network...National Alopecia Areata Foundation
To date, the Registry has epidemiology and quality-of-life data from 11,180 self-registered patients with 4,196
well-characterized samples of DNA, lymphoblast lines, and sera for future research studies.Publications
This document lists 16 publications by Peter M. Power related to research on pilin glycosylation in Neisseria meningitidis and other gram-negative bacteria. The publications span from 2000 to 2014 and include studies characterizing the genetic basis of pilin glycosylation, the role of specific genes in the glycosylation pathway, comparative analyses of glycosylation mechanisms between bacteria, and investigations into genome diversity and phase variation.
224 k ras polymorphism in cancer, for atherosclerosis
This document discusses several studies that have examined gene polymorphisms related to atherosclerosis. It focuses on polymorphisms in genes involved in inflammation, immune response, endothelial function, and lipid metabolism. The studies presented had varying and sometimes conflicting results on the association between specific gene variants and coronary artery disease or myocardial infarction. In general, the document indicates that coronary artery disease has a complex genetic basis, with many genes and environmental factors involved.
Similar to Publications sima hadidi (20)
The Role of CSF Adenosine Deaminase in Detecting Tuberculous Meningitis
The Role of CSF Adenosine Deaminase in Detecting Tuberculous Meningitis
A Report of the Alopecia Areata Registry, Biobank and Clinical Trials Network...
A Report of the Alopecia Areata Registry, Biobank and Clinical Trials Network...
224 k ras polymorphism in cancer, for atherosclerosis
224 k ras polymorphism in cancer, for atherosclerosis
Publications sima hadidi
- 1. 1: Wiens GD, Vallejo RL, Leeds TD, Palti Y, Hadidi S, Liu S, Evenhuis JP, Welch TJ, Rexroad CE 3rd. Assessment of genetic correlation between bacterial cold water disease resistance and spleen index in a domesticated population of rainbow trout: identification of QTL on chromosome Omy19. PLoS One. 2013 Oct 9;8(10):e75749. doi: 10.1371/journal.pone.0075749. eCollection 2013. PubMed PMID: 24130739; PubMed Central PMCID: PMC3794016. 2: Hadidi S, Antignano F, Hughes MR, Wang SK, Snyder K, Sammis GM, Kerr WG, McNagny KM, Zaph C. Myeloid cell-specific expression of Ship1 regulates IL-12 production and immunity to helminth infection. Mucosal Immunol. 2012 Sep;5(5):535-43. doi: 10.1038/mi.2012.29. Epub 2012 Apr 25. PubMed PMID: 22535180. 3: Lehnertz B, Northrop JP, Antignano F, Burrows K, Hadidi S, Mullaly SC, Rossi FM, Zaph C. Activating and inhibitory functions for the histone lysine methyltransferase G9a in T helper cell differentiation and function. J Exp Med. 2010 May 10;207(5):915-22. doi: 10.1084/jem.20100363. Epub 2010 Apr 26. PubMed PMID: 20421388; PubMed Central PMCID: PMC2867284. 4: Leeds TD, Silverstein JT, Weber GM, Vallejo RL, Palti Y, Rexroad CE 3rd, Evenhuis J, Hadidi S, Welch TJ, Wiens GD. Response to selection for bacterial cold water disease resistance in rainbow trout. J Anim Sci. 2010 Jun;88(6):1936-46. doi: 10.2527/jas.2009-2538. Epub 2010 Feb 12. PubMed PMID: 20154172. 5: Palti Y, Gahr SA, Purcell MK, Hadidi S, Rexroad CE 3rd, Wiens GD. Identification, characterization and genetic mapping of TLR7, TLR8a1 and TLR8a2 genes in rainbow trout (Oncorhynchus mykiss). Dev Comp Immunol. 2010 Feb;34(2):219-33. doi: 10.1016/j.dci.2009.10.002. Epub 2009 Oct 23. PubMed PMID: 19825389. 6: Hadidi S, Glenney GW, Welch TJ, Silverstein JT, Wiens GD. Spleen size predicts resistance of rainbow trout to Flavobacterium psychrophilum challenge. J Immunol. 2008 Mar 15;180(6):4156-65. PubMed PMID: 18322227. 7: Hongo D, Hadidi S, Damrauer S, Garrigue V, Kraft D, Sachs DH, Nikolic B, Sykes
- 2. M. Porcine thymic grafts protect human thymocytes from HIV-1-induced destruction. J Infect Dis. 2007 Sep 15;196(6):900-10. Epub 2007 Aug 7. PubMed PMID: 17703422. 8: Hadidi S, Gorczynski RM. MD-1 expression regulates direct and indirect allorecognition. Tissue Antigens. 2004 Feb;63(2):132-41. PubMed PMID: 14705984. 9: Gorczynski RM, Hadidi S, Yu G, Clark DA. The same immunoregulatory molecules contribute to successful pregnancy and transplantation. Am J Reprod Immunol. 2002 Jul;48(1):18-26. PubMed PMID: 12322892. 10: Hadidi S, Chen Z, Phillips J, Yu K, Gorczynski RM. Antisense deoxyoligonucleotides or antibodies to murine MD-1 inhibit rejection of allogeneic and xenogeneic skin grafts in C3H mice. Transplantation. 2002 Jun 15;73(11):1771-9. PubMed PMID: 12085000. 11: Hadidi S, Yu K, Chen Z, Gorczynski RM. Preparation and functional properties of polyclonal and monoclonal antibodies to murine MD-1. Immunol Lett. 2001 Jun 1;77(2):97-103. PubMed PMID: 11377703. 12: Gorczynski RM, Chen Z, Clark D, Hu J, Kai Y, Li X, Tsang W, Hadidi S. Expression of murine MD-1 regulates T-Cell activation/cytokine production. Transplant Proc. 2001 Feb-Mar;33(1-2):1585. PubMed PMID: 11267430. 13: Gorczynski RM, Chen Z, Clark DA, Hu J, Yu G, Li X, Tsang W, Hadidi S. Regulation of gene expression of murine MD-1 regulates subsequent T cell activation and cytokine production. J Immunol. 2000 Aug 15;165(4):1925-32. PubMed PMID: 10925274.