15. Foreword
Anaemia, a manifestation of under-nutrition and poor dietary intake of iron is
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levels of care.
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and control anaemia. I call upon all stakeholders and partners for their continued support
in this respect.
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(Dr. Rakesh Kumar)
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D . RAKESH K MAR, I.A.S
INT SE RETAR
Telefax : 23061723
E-mail : r umar 2@hotmail.com
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N B , N D - 110108
16.
17. Contents
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1.1. What is Anaemia? 1
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24. Acronyms
AG Adolescent Girl
ANC Antenatal Care
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ASHA Accredited Social Health Activist
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CHC Community Health Centre
CNS Central Nervous System
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DH District Hospital
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FRU First Referral Unit
GDP Gross Domestic Product
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IFA Iron and Folic Acid
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ITBN Insecticide Treated Bed Nets
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MCP Card Mother Child Protection Card
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MoHFW Ministry of Health and Family Welfare
MUAC Mid Upper Arm Circumference
MWCD Ministry of Women and Child Development
NFHS National Family Health Survey
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RBC Red Blood Cells
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WIFS Weekly Iron and Folic Acid Supplementation
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25. 1
Anaemia – $ 3XEOLF +HDOWK KDOOHQJH
1.1. What is Anaemia?
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function of the RBCs is to deliver oxygen from the lungs to the tissues and carbon
dioxide from the tissues to the lungs. This is accomplished by using haemoglobin
(Hb), a tetramer protein composed of haem and globin. Anaemia impairs the body’s
ability for gas exchange by decreasing the number of RBCs transporting oxygen and
carbon dioxide. $QDHPLD UHVXOWV IURP RQH RU PRUH RI WKH IROORZLQJ SURFHVV GHIHFWLYH UHG
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Fig. 1.1: Adverse effects of anaemia
ANAEMIA
Reduced
physical
development
Impaired
sexual and
reproductive
development
Reduced
cognitive
development
Decreased work output
Decreased work capacity
Diminished concentration
Disturbance in perception
Poor learning ability
Irregular menstruation
Low pre-pregnancy iron stores
LBW babies and preterm delivery
1
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2
Table 1.1: Haemoglobin levels to diagnose anaemia (g/dl)
Age groups No Anaemia Mild Moderate Severe
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Source: Haemoglobin concentration for the diagnosis of anaemia and assessment of severity. WHO
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causative factor.
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Storage iron
Transport iron
RBC iron
Normal Iron depletion
Iron deficient
erythropoiesis
Iron deficiency
anaemia
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in chronic anaemia. Kidney failure can also cause anaemia.
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Fig. 2.1: Global picture –Anaemia as a public health problem in preschool children by country
Category of Public
Health Significance
Normal (5.0%)
Mild (5.0-19.9%)
Moderate (20.0-39.9%)
Severe (40.0%)
No Data
Source: WHO Global Database on Anaemia
2
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Table 2.1: Prevalence of anaemia in India and neighbouring countries
Country
Proportion of population with
anaemia (Hb 11 g/dl)
Public health problem
%DQJODGHVK Severe
Bhutan Severe
India Severe
Nepal Severe
Pakistan Severe
6UL /DQND Moderate
Source: WHO Global Database on Anaemia
2.2. Indian Scenario
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58. Table 2.3: Prevalence of anaemia among children aged 6 to 35 months (per cent)
Anaemia level
NFHS-2 NFHS-3
Urban Rural Total Urban Rural Total
0LOG ± JGO
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8
Fig. 2.2: Prevalence of anaemia among adolescent girls (12–19 years) and young women
(20–29 years) in India6,7
80
12--14 years
68.6 69.7
55.8 56.1
47.2
27
39.1 38.5
20.5 20.7
14.9
1.1 1.7 1.71
16
15--17 years 15--19 years 20--29 years
70
60
Any anaemia
(12.0 g/dl)
Mild anaemia
(10.0–11.9 g/dl)
Severe anaemia
(7.0 g/dl)
Moderate anaemia
(7.0–9.9 g/dl)
50
40
30
Percentage
20
10
0
Source: NFHS-3, 2005-06 and the National Nutrition Monitoring Bureau Survey (NNMBS), 2006
Prevalence of anaemia among pregnant women, men and women of reproductive
age (WRA)
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68. Fig. 2.3: Prevalence of anaemia among pregnant women
Percentage
60
50
40
30
20
10
0
26
2
58
Mild Any anaemiaSevere
31
Moderate
Source: NFHS-3, 2005-06
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111. Table 6.1: IFA supplementation programme and service delivery
Age group Intervention/Dose Regime Service delivery
± PRQWKV 1ml of IFA syrup
FRQWDLQLQJ PJ RI
elemental iron and
PFJ RI IROLF DFLG
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children 12 months
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Table 7.1: Management of anaemia on the basis of haemoglobin levels in children
6 months–5 years
Level of Hb Treatment Follow-up Referral
No Anaemia
! JPGO
154. PJ RI HOHPHQWDO LURQ DQG PFJ RI IROLF DFLG LQ ELZHHNO UHJLPHQ
Mild Anaemia
± JPGO
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ANM
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treatment to document
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responded to the treatment of
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for 2 months, refer the child to the
)58'+ ZLWK ),01, WUDLQHG 02
Paediatrician/Physician for further
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± JPGO
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treatment to document
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for 2 months, refer the child to the
)58'+ ZLWK ),01, WUDLQHG 02
Paediatrician/Physician for further
LQYHVWLJDWLRQV
Severe Anaemia
JPGO
157. Refer
XUJHQWO WR
DH/FRU
Table 7.2: Dose of IFA syrup for anaemic children 6 months–5 years
Age of child Dose Frequency
PRQWKV± PRQWKV ± NJ
160. 2 ml of IFA syrup Once a day
Follow-up of children undergoing treatment of anaemia to be done by ANM
onitoring by SH for co liance of yru e ery
ay for a erio of onth
ollo u by e ery ay
f chil continue to ha e anae ia Hb e ti ation at
ub centre after onth of yru refer the
chil to PHC for further anage ent
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the merit of the individual case.
Management of severe anaemia at FRU/DH (as per F-IMNCI) in children
6 months–5 years
History to be taken for Examination for
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Table 7.3: Management of anaemia on the basis of haemoglobin levels in children
5–10 years
Level of Hb Treatment Follow-up Referral
Mild Anaemia
(11–11.4
gm/dl)
PJ RI LURQ.J
day for
2 months
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WR WKH WUHDWPHQW RI DQDHPLD ZLWK GDLO
dose of iron for 2 months, refer the
FKLOG WR WKH )58'+ ZLWK ),01,
trained MO/Paediatrician/Physician
IRU IXUWKHU LQYHVWLJDWLRQ
Moderate
Anaemia
(8–10.9 gm/dl)
PJ RI LURQ.J
day for
2 months
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FRPSOHWLQJ PRQWKV RI
WUHDWPHQW WR DVVHVV LI +E
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In case the child has not responded
WR WKH WUHDWPHQW RI DQDHPLD ZLWK GDLO
dose of iron for 2 months, refer the
FKLOG WR WKH )58'+ ZLWK ),01,
trained MO/Paediatrician/Physician
IRU IXUWKHU LQYHVWLJDWLRQV
Severe
Anaemia
(8 gm/dl)
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to DH/FRU
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Management of severe anaemia at FRU/DH in children 5–10 years
History taking for Examination for
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