This document provides an overview of myasthenia gravis (MG), an autoimmune disease where antibodies target acetylcholine receptors (AchRs) at the neuromuscular junction, inhibiting its activity. It discusses the adaptive immune response producing AchR antibodies, anatomy and physiology of the neuromuscular junction, symptoms of MG, diagnostic tests, and treatments. A research proposal is presented to identify the mechanism of AchR antibody inhibition, hypothesizing the mechanism depends on the percentage of AchRs bound rather than antibody type. A series of experiments are proposed to test the three proposed mechanisms: blocking Ach binding, inducing membrane endocytosis, and complement-mediated membrane damage.