OKWEREKWU FRANCIS .C
 Introduction
 Epidemiology
 Aetiology
 Pathophysiology
 Clinical Features
 Investigations
 Treatment
 Prevention
 Conclusion
Introduction
 Burn injuries are a world wide
problem. They rank high among
injuries suffered by man.
 Since its inception in 1849, commercial
use of electricity has been one of the
most potentially dangerous
commodities in the world.
Introduction
 Electrical injury includes electrocution,
electric shock, burns and secondary
injuries.
 The term electrical burn is used widely
to describe the variety of injuries
created by supraphysiologic electrical
energy interacting with living tissue.
Epidemiology
 According to statistical data, 0.8-1% of
accidental deaths are caused by an
electric injury.
 Electric injury accounts for 1000 deaths
each year in the United States, with a
mortality rate of 3-15%
Epidemiology
 Approximately 20% of all electrical injuries
occur in children, with a bimodal peak incidence
highest in toddlers and adolescents.
 Most electrical injuries that occur in children are
at home, with extension cords (60-70%) and wall
outlets (10-15%) being by far the most common
sources in this age group.
 Electrical burns account for 2-3% of all burns in
children that require emergency room care.
Epidemiology
 Electrical burns constituted 2.8% of total
burn admissions.
 Male: Female ratio was 1.8:1
 44.9% of the patients were children with
age range between 22 days – 14 years.
 Department of Burns, Plastic & Reconstructive Surgery, National
Orthopaedic Hospital Enugu(Niger ian J Clin Pract. 2006 Dec;9(2):124-
7.)
Aetiology
 Electrical burns are usually divided into low
voltage and high voltage injuries, the threshold
being 1000volts.
 a) Low voltage injuries: do not have enough
energy to cause destruction to significant amt of
subcutaneous tissues. The entry and the exit
points, normally in the fingers and toes suffer
small deep burns.
 b) High voltage injuries
Pathophysiology
 Electrical energy causing direct tissue
damage, altering cell membrane resting
potential, and eliciting muscle tetany.
 Conversion of electrical energy to thermal
energy, causing massive tissue destruction
and coagulative necrosis.
 Mechanical injury with direct trauma
resulting from falls or violent muscle
contraction.
Pathophysiology
 Factors that determine the degree of injury
include
 Type of current
 Resistance encountered
 Current pathway
 Magnitude of energy delivered
 Duration of contact.
Type of current
 The repetitive nature of AC increases the
likelihood of current delivery to the
myocardium, which can precipitate
ventricular fibrillation.
 In contrast, DC usually causes a single
violent muscle contraction, often
thrusting the victim away from the
source.
Resistance
 In general, tissues with high fluid and
electrolyte content conduct electricity
better.
 Bone is the tissue most resistant to the
flow of electricity.
 Nerve tissue is the least resistant, and
together with blood vessels, muscles,
and mucous membranes offer a path of
low resistance for electricity.
Resistance
 Skin is the most important factor
impeding current flow. It is the primary
resistor against electrical current, and its
degree of resistance is determined by its
thickness and moisture.
 It varies from 1000 ohms for humid thin
skin to several thousand ohms for dry
calloused skin.
Pathway
 Transthoracic currents can cause fatal
arrhythmia, direct cardiac damage, or
respiratory arrest.
 Transcranial currents can cause direct
brain injury, seizure, respiratory arrest,
and paralysis.
Magnitude
 The current intensity will also
determine the magnitude of injury.
There may be individual variation on
the energy dose for a specific effect.
 Less energy is generally required in
children, who have more water content
and thin skin and, hence, better
conductivity and less resistance.
Current intensity Expected clinical manifestation
1 mA Probable tingling sensation
3-5 mA "Let go" current for an average
child
7-9 mA "Let go" current for an average man
16-20 mA Tetany of skeletal muscles
20-50 mA Paralysis of respiratory muscles
(respiratory arrest)
50-100 mA Threshold for ventricular
fibrillation
Greater than 2 A Threshold for ventricular
fibrillation
15-30 A Common household circuit
breakers
240 A Maximal intensity of US household
current
Clinical Features
 Skin burns/Mouth burns
 Unconscious
 Bleeding
 Skeletal injuries
 Shock
 Dark coloured urine/ARF
 Respiratory arrest
 Tissue oedema/ Compartment syndrome
 Numbness/Paraesthesia
 Seizures
Management
 Acute emergency, shout for help.
 Secure yourself.
 Separate the victim from the current's source.
The safest way to do so is to shut off the current
e.g by throwing a circuit breaker or switch or by
disconnecting the device from an electrical
outlet.
 Treat as multi-trauma patient with cervical
immobilisation at least until the full extent of
injuries has been quantified
Management
 ABC’s
 IV access
 IV fluids-Ringer’s Lactate
 Monitor urine output -100ml/hr
 ARF prevention(in case of myoglobinuria)
 Tetanus prophylaxis
 Analgesia, ulcer prophylaxis, antibiotics
 Wound dressing
Management
 Defribillation
 Associated injuries-Splint limb, refer
 Extremities – Fasciotomies for compartment
syndrome
 Debridement/amputation -non-viable tissue
 Reconstructive surgery for mouth burns
Investigations
 FBC
 E,U,Cr
 ECG x 24 hrs
 CXR
 ABG
 X-ray, CT
 Wound swab- MCS
Complications
 Seizures
 Peripheral nerve damage
 Psychiatric problems from depression to
aggressive behaviour
 Cataracts
 Paraplegia/Quadriplegia
 Deformities
Prevention
 Education about and respect for electricity are
essential.
 Ensure electrical devices are properly designed,
installed, and maintained.
 Electrical wiring should be installed and serviced
by properly trained personnel.
 Outlet guards reduce risk in homes with infants
or young children.
PREVENTION CONTD.
 Any electrical device that touches or may be
touched by the body should be properly
grounded.
 Three-pronged outlets are safest.
 Circuit breakers that interrupt (trip) circuits
when current as low as 5 milliamperes leaks are
advisable in areas that get wet, such as kitchens
and bathrooms and outdoors.
CONCLUSION
 Although electrical burns represent only a small
proportion of burn injuries, the incidence of
complications, mortality and morbidity, and
disability is high. Such injuries can be prevented
with proper educational programmes designed
to suit the community.
THANK YOU FOR LISTENING

Electrical-Burns effect on OT performance .ppt

  • 1.
  • 2.
     Introduction  Epidemiology Aetiology  Pathophysiology  Clinical Features  Investigations  Treatment  Prevention  Conclusion
  • 3.
    Introduction  Burn injuriesare a world wide problem. They rank high among injuries suffered by man.  Since its inception in 1849, commercial use of electricity has been one of the most potentially dangerous commodities in the world.
  • 4.
    Introduction  Electrical injuryincludes electrocution, electric shock, burns and secondary injuries.  The term electrical burn is used widely to describe the variety of injuries created by supraphysiologic electrical energy interacting with living tissue.
  • 5.
    Epidemiology  According tostatistical data, 0.8-1% of accidental deaths are caused by an electric injury.  Electric injury accounts for 1000 deaths each year in the United States, with a mortality rate of 3-15%
  • 6.
    Epidemiology  Approximately 20%of all electrical injuries occur in children, with a bimodal peak incidence highest in toddlers and adolescents.  Most electrical injuries that occur in children are at home, with extension cords (60-70%) and wall outlets (10-15%) being by far the most common sources in this age group.  Electrical burns account for 2-3% of all burns in children that require emergency room care.
  • 7.
    Epidemiology  Electrical burnsconstituted 2.8% of total burn admissions.  Male: Female ratio was 1.8:1  44.9% of the patients were children with age range between 22 days – 14 years.  Department of Burns, Plastic & Reconstructive Surgery, National Orthopaedic Hospital Enugu(Niger ian J Clin Pract. 2006 Dec;9(2):124- 7.)
  • 8.
    Aetiology  Electrical burnsare usually divided into low voltage and high voltage injuries, the threshold being 1000volts.  a) Low voltage injuries: do not have enough energy to cause destruction to significant amt of subcutaneous tissues. The entry and the exit points, normally in the fingers and toes suffer small deep burns.  b) High voltage injuries
  • 9.
    Pathophysiology  Electrical energycausing direct tissue damage, altering cell membrane resting potential, and eliciting muscle tetany.  Conversion of electrical energy to thermal energy, causing massive tissue destruction and coagulative necrosis.  Mechanical injury with direct trauma resulting from falls or violent muscle contraction.
  • 10.
    Pathophysiology  Factors thatdetermine the degree of injury include  Type of current  Resistance encountered  Current pathway  Magnitude of energy delivered  Duration of contact.
  • 11.
    Type of current The repetitive nature of AC increases the likelihood of current delivery to the myocardium, which can precipitate ventricular fibrillation.  In contrast, DC usually causes a single violent muscle contraction, often thrusting the victim away from the source.
  • 12.
    Resistance  In general,tissues with high fluid and electrolyte content conduct electricity better.  Bone is the tissue most resistant to the flow of electricity.  Nerve tissue is the least resistant, and together with blood vessels, muscles, and mucous membranes offer a path of low resistance for electricity.
  • 13.
    Resistance  Skin isthe most important factor impeding current flow. It is the primary resistor against electrical current, and its degree of resistance is determined by its thickness and moisture.  It varies from 1000 ohms for humid thin skin to several thousand ohms for dry calloused skin.
  • 14.
    Pathway  Transthoracic currentscan cause fatal arrhythmia, direct cardiac damage, or respiratory arrest.  Transcranial currents can cause direct brain injury, seizure, respiratory arrest, and paralysis.
  • 15.
    Magnitude  The currentintensity will also determine the magnitude of injury. There may be individual variation on the energy dose for a specific effect.  Less energy is generally required in children, who have more water content and thin skin and, hence, better conductivity and less resistance.
  • 16.
    Current intensity Expectedclinical manifestation 1 mA Probable tingling sensation 3-5 mA "Let go" current for an average child 7-9 mA "Let go" current for an average man 16-20 mA Tetany of skeletal muscles 20-50 mA Paralysis of respiratory muscles (respiratory arrest) 50-100 mA Threshold for ventricular fibrillation Greater than 2 A Threshold for ventricular fibrillation 15-30 A Common household circuit breakers 240 A Maximal intensity of US household current
  • 17.
    Clinical Features  Skinburns/Mouth burns  Unconscious  Bleeding  Skeletal injuries  Shock  Dark coloured urine/ARF  Respiratory arrest  Tissue oedema/ Compartment syndrome  Numbness/Paraesthesia  Seizures
  • 20.
    Management  Acute emergency,shout for help.  Secure yourself.  Separate the victim from the current's source. The safest way to do so is to shut off the current e.g by throwing a circuit breaker or switch or by disconnecting the device from an electrical outlet.  Treat as multi-trauma patient with cervical immobilisation at least until the full extent of injuries has been quantified
  • 21.
    Management  ABC’s  IVaccess  IV fluids-Ringer’s Lactate  Monitor urine output -100ml/hr  ARF prevention(in case of myoglobinuria)  Tetanus prophylaxis  Analgesia, ulcer prophylaxis, antibiotics  Wound dressing
  • 22.
    Management  Defribillation  Associatedinjuries-Splint limb, refer  Extremities – Fasciotomies for compartment syndrome  Debridement/amputation -non-viable tissue  Reconstructive surgery for mouth burns
  • 23.
    Investigations  FBC  E,U,Cr ECG x 24 hrs  CXR  ABG  X-ray, CT  Wound swab- MCS
  • 24.
    Complications  Seizures  Peripheralnerve damage  Psychiatric problems from depression to aggressive behaviour  Cataracts  Paraplegia/Quadriplegia  Deformities
  • 25.
    Prevention  Education aboutand respect for electricity are essential.  Ensure electrical devices are properly designed, installed, and maintained.  Electrical wiring should be installed and serviced by properly trained personnel.  Outlet guards reduce risk in homes with infants or young children.
  • 26.
    PREVENTION CONTD.  Anyelectrical device that touches or may be touched by the body should be properly grounded.  Three-pronged outlets are safest.  Circuit breakers that interrupt (trip) circuits when current as low as 5 milliamperes leaks are advisable in areas that get wet, such as kitchens and bathrooms and outdoors.
  • 27.
    CONCLUSION  Although electricalburns represent only a small proportion of burn injuries, the incidence of complications, mortality and morbidity, and disability is high. Such injuries can be prevented with proper educational programmes designed to suit the community.
  • 28.
    THANK YOU FORLISTENING