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Cyclovertical deviation
1.
2. Right eye hyperdeviation
Left eye covered and right forced to fixate
Left hypotropia of
same magnitude
True vertical tropia
Left -same position
Right DVD
Left eye raises
under cover
B/L DVD
Left hypotropia of
less magnitude
Combination
3. Principles in cyclovertical
deviations
Incomitant hyperdeviation in horizontal plane -Oblique muscle
Comitant hyperdeviation in horizontal plane - Vertical rectus
Most common causes of a true vertical tropia - fourth nerve palsy.
If the deviation is incomitant in the horizontal plane or is long-
standing and comitant, perform a head-tilt test .
In a case of superior oblique palsy, the clinician must assume the
palsy is bilateral until it is proven otherwise
4. Park 3 step test
Primary position – which eye ?
Gaze ?
Head tilt ?
Sensitivity -70%(1)
Sensitivity of the three-step test in diagnosis of superior oblique
palsy Ajay M. Manchandia et al J AAPOS. 2014 December ; 18(6): 567–571. doi:10.1016/j.jaapos.2014.08.007.
8. 1.Dissociated vertical deviation
Etiology-
50% of patients with infantile
esotropia
Clinical features-
Head tilt
Excycloduction of the elevated eye
and incycloduction of the fixating
eye
Latent nystagmus
9.
10. Diagnostic test
1. Spielmanns translucent
occluder
2. Bielschowsky phenomenon
3. Red filter test
Measurement
Using Red filter
Using base down prism
12. Condition Treatment
IOOA & mod. DVD Recession with
anterior positioning IO
IOOA & Severe DVD Recession with anterior positioning
IO +
Superior rectus-recession 7-10 mm
DVD & no IOOA: Superior rectus-recession 7-10 mm +
Inferior rectus resection
13. Inferior Oblique Muscle Over action
Etiology
1. Primary
2. Secondary
Clinical Features
Management
Weakening procedure on the
inferior oblique muscle
14. Dissociated vertical
deviation
Inferior oblique over
action
Elevation In adduction abduction
and primary postion
Maximal in adduction
never in abduction
Superior oblique action May over act Usually under action
V pattern Absent Present
Incycloduction on
refixation
Present Absent
Saccadic velocity of
refixation movement
10–200/s 200–400/s
Latent nystagmus Often present Absent
Bielschowsky
phenomenon
Often present Absent
15. Superior Oblique Muscle Over action
Clinical Features
1. Overdepression in
adduction.
2. Associated with exo
Management
Superior oblique tendon
weakening procedure
16. 4.Superior Oblique Muscle
Paralysis
Most common Single cyclovertical muscle paralysis.
Etiology
Congenital vs acquired
Head trauma
Vascular problems of the central nervous system,
Diabetes mellitus, or a brain tumor
21. Maddox wing
The amount of cyclophoria is determined by asking the patient to move
the red arrow so that it is parallel with the horizontal row of numbers
23. Treatment
Greatest deviation in Treatment
Affected eye elevated in adduction I/l IO recession
Affected eye depressed in adduction I/L SO tucking
In all contralateral gazes Hypertropia<25 pd I/L IO
recession
Hypertropia> 25 pd I/L IO
recession + RSO tuck
In all contralateral gazes and in all down
gazeS
As in class 3+ C/L IR
recession/ I/L SR recession
In all down gazes I/L SO tuck + C/L LIR recession
Bilateral with V pattern Bilateral IO weakening or
modified Harada Ito
In all contralatreral gazes , down gazes
and in primary position
Explore trochlea
25. IO paralysis Brown syndrome
Forced duction Negative positive
Strabismus pattern A pattern V pattern
SO overaction Present None or minimal
Management
I/L SO or C/L SR weakening
27. Three forms of monocular elevation deficiency are as
follows:
1. Restriction
2. Elevator muscle innervational deficit
3. Combination
Management
Restrictive –recession of IR
No restriction – Knapp procedure
28. Refernces
Binocular vision and occular motility - Von noorden
5th edition
Handbook of Pediatric Strabismus and Amblyopia
Kenneth W. Wright, MD
PEDIATRIC Ophthalmologyand Strabismus - Creig S
Hoyt -4thedition
Pediatric Ophthalmology and Strabismus -Section
6 2015-2016- American academy of ophthalmology.