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Management of Cataract
Dr. Abhishek Onkar
Symptom Pathogenesis Type of cataract
Frequent change of glasses Rapid change in µ of lens Cortical/nuclear
↓ VA (Gradual, painless,
progressive
↓ transparency of lens All types → ← ↑
Second sight/myopic shift ↑ µ of nucleus → index myopia Nuclear
↓ ability to see in bright sunlight,
blinded by light from headlights
while driving at night
Loss of contrast sensitivity which is
greater at higher spatial
frequencies; constriction of pupil
cuts off peripheral vision from non-
cataractous area
PSC
Monocular diplopia/ polyopia Cortical spokes with water clefts of
↓ µ than surrounding lens
Cortical cataract( spoke/cuneiform)
Glare ↑ scattering of light Cortical/ PSC
Coloured haloes around light Irregularities of µ Cortical
Colour shift (more obvious after
surgery)
Blue specta absorbed more by
cataractous lens
Nuclear
Visual field loss Generalized ↓ in sensitivity d/t loss
of lens transparency
All types
Signs
• Iris shadow
• Depth of anterior chamber
• Pupillary reflex
• Visual acuity
• Plain mirror examination under mydriasis
Iris
shadow
A.C.
Depth
Pupillary
reflex
Visual
acuity
Intumescent Present Shallow Greyish white FC to 6/18
Incipient Present Normal Greyish white FC to 6/18
Mature Absent Normal Pearly white HM to FC
close to face
Hypermature
Morgagnian
Absent Shallow Milky white HM +
Hypermature
Calcified
Absent Normal or
deep
Milky
chalky
HM +
Differential diagnosis
Painless, progressive diminution of vision
• Cataract
• Primary open angle glaucoma
• Diabetic retinopathy
• Age related macular degeneration
• Corneal dystrophies and degenerations
• Retinitis pigmentosa
• Refractive errors
Management
• An un-operated eye is more comfortable than an operated eye if
visual diminution is mild.
• Early cataract :
-Refraction and glasses
-Dark glasses or photochromatic glasses for nuclear cataract
-Rule out other causes of visual diminution
-If BCVA not to patient’s satisfaction, then operate.
Patient workup
• Retinoscopy and best corrected visual acuity
• Intraocular pressure
• Slit lamp examination
• DDO
• Fundus evaluation – direct & indirect
• Macular function tests
• Ultrasonography
• IOL power calculation
• Complete Ocular Examination including adnexa
• IOP
• Syringing
• Tear film evaluation
• Keratometry
• Axial length measurement: A-scan
• IOL power calculation
SRK Formula: Sanders, Retzlaff and Kraff
P = A – 0.9 K – 2.5 AL
General investigations
• Blood pressure
• Blood sugar
• Complete haemogram
• HIV, Hepatitis B & C
• Causes of straining
• Foci of infection
• Systemic examination
Evolution of cataract surgery
ICCE
• TRAUMATIC SUBLUXTED LENS
ECCE: Steps in short
• 1. +/-Bridle traction suture
• 2. Peritomy (Conj opening)
• 3. Partial thickness corneoscleral wound and AC Entering
• 4. Can-opener capsulotomy
• 5. Extend corneoscleral wound and Lens extraction
• 6. remove cortex +/- suture
• 7. IOL implantation and close wound
Phacoemulsification : Steps in short
• 1. Capsulorhexis
• 2. Corneal/ scleral incision
• 3. Hydrodissection and hydrodelineation
• 4. Phacoemulsification- Divide and Conquer
• 5. Cortex removal/ capsule polishing
• 6. IOL implantation
Potential Postoperative Complications
• Corneal edema
• Descemet's membrane detachment
• Induced astigmatism
• Corneal melting
• Conjunctival ballooning
• Epithelial downgrowth
• Toxic anterior segment syndrome (TASS)
• Flat chamber
• Elevated intraocular pressure
• Intraoperative floppy iris syndrome (IFIS)
• Iridodialysis
• Cyclodialysis
• Ciliary block glaucoma
• Chronic postoperative uveitis
• Macular infarction
• Retinal detachment
• Retained lens material
• Capsular rupture/PCR
• Vitreous prolapse
• Dropped nucleus
• IOL complications (decentration and dislocation, pupillary
capture, capsular block syndrome, uveitis-glaucoma-
hyphema syndrome, pseudophakic bullous keratopathy,
incorrect IOL power, IOL glare, IOL opacification)
• Anterior capsular fibrosis and phimosis
• Posterior capsular opacification
• Hemorrhage (retrobulbar hemorrhage, suprachoroidal
effusion, expulsive suprachoroidal hemorrhage, hyphema)
• Endophthalmitis
• Cystoid macular edema
• Uretts Zavala syndrome
• Retinal light toxicity

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Cataract management

  • 1. Management of Cataract Dr. Abhishek Onkar
  • 2. Symptom Pathogenesis Type of cataract Frequent change of glasses Rapid change in µ of lens Cortical/nuclear ↓ VA (Gradual, painless, progressive ↓ transparency of lens All types → ← ↑ Second sight/myopic shift ↑ µ of nucleus → index myopia Nuclear ↓ ability to see in bright sunlight, blinded by light from headlights while driving at night Loss of contrast sensitivity which is greater at higher spatial frequencies; constriction of pupil cuts off peripheral vision from non- cataractous area PSC Monocular diplopia/ polyopia Cortical spokes with water clefts of ↓ µ than surrounding lens Cortical cataract( spoke/cuneiform) Glare ↑ scattering of light Cortical/ PSC Coloured haloes around light Irregularities of µ Cortical Colour shift (more obvious after surgery) Blue specta absorbed more by cataractous lens Nuclear Visual field loss Generalized ↓ in sensitivity d/t loss of lens transparency All types
  • 3. Signs • Iris shadow • Depth of anterior chamber • Pupillary reflex • Visual acuity • Plain mirror examination under mydriasis
  • 4.
  • 5. Iris shadow A.C. Depth Pupillary reflex Visual acuity Intumescent Present Shallow Greyish white FC to 6/18 Incipient Present Normal Greyish white FC to 6/18 Mature Absent Normal Pearly white HM to FC close to face Hypermature Morgagnian Absent Shallow Milky white HM + Hypermature Calcified Absent Normal or deep Milky chalky HM +
  • 6. Differential diagnosis Painless, progressive diminution of vision • Cataract • Primary open angle glaucoma • Diabetic retinopathy • Age related macular degeneration • Corneal dystrophies and degenerations • Retinitis pigmentosa • Refractive errors
  • 7. Management • An un-operated eye is more comfortable than an operated eye if visual diminution is mild. • Early cataract : -Refraction and glasses -Dark glasses or photochromatic glasses for nuclear cataract -Rule out other causes of visual diminution -If BCVA not to patient’s satisfaction, then operate.
  • 8. Patient workup • Retinoscopy and best corrected visual acuity • Intraocular pressure • Slit lamp examination • DDO • Fundus evaluation – direct & indirect • Macular function tests • Ultrasonography • IOL power calculation
  • 9. • Complete Ocular Examination including adnexa • IOP • Syringing • Tear film evaluation • Keratometry • Axial length measurement: A-scan • IOL power calculation
  • 10. SRK Formula: Sanders, Retzlaff and Kraff P = A – 0.9 K – 2.5 AL
  • 11. General investigations • Blood pressure • Blood sugar • Complete haemogram • HIV, Hepatitis B & C • Causes of straining • Foci of infection • Systemic examination
  • 13.
  • 14.
  • 16. ECCE: Steps in short • 1. +/-Bridle traction suture • 2. Peritomy (Conj opening) • 3. Partial thickness corneoscleral wound and AC Entering • 4. Can-opener capsulotomy • 5. Extend corneoscleral wound and Lens extraction • 6. remove cortex +/- suture • 7. IOL implantation and close wound
  • 17. Phacoemulsification : Steps in short • 1. Capsulorhexis • 2. Corneal/ scleral incision • 3. Hydrodissection and hydrodelineation • 4. Phacoemulsification- Divide and Conquer • 5. Cortex removal/ capsule polishing • 6. IOL implantation
  • 18.
  • 19.
  • 20. Potential Postoperative Complications • Corneal edema • Descemet's membrane detachment • Induced astigmatism • Corneal melting • Conjunctival ballooning • Epithelial downgrowth • Toxic anterior segment syndrome (TASS) • Flat chamber • Elevated intraocular pressure • Intraoperative floppy iris syndrome (IFIS) • Iridodialysis • Cyclodialysis • Ciliary block glaucoma • Chronic postoperative uveitis • Macular infarction • Retinal detachment • Retained lens material • Capsular rupture/PCR • Vitreous prolapse • Dropped nucleus • IOL complications (decentration and dislocation, pupillary capture, capsular block syndrome, uveitis-glaucoma- hyphema syndrome, pseudophakic bullous keratopathy, incorrect IOL power, IOL glare, IOL opacification) • Anterior capsular fibrosis and phimosis • Posterior capsular opacification • Hemorrhage (retrobulbar hemorrhage, suprachoroidal effusion, expulsive suprachoroidal hemorrhage, hyphema) • Endophthalmitis • Cystoid macular edema • Uretts Zavala syndrome • Retinal light toxicity