Brain Injury Power Point
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This document discusses how concussions can cause brain injuries and long-term health issues by impacting hormone production in the hypothalamus, pituitary gland, and adrenal glands. Specifically, it notes that concussions have been associated with illnesses like Alzheimer's, Parkinson's, and depression. The key to recovery is restoring hormone levels like human growth hormone, cortisol, and thyroid hormones to their pre-injury levels through testing done at 3 and 12 months post-injury.
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This document discusses the link between nutrition and depression. It begins by providing background on depression, including prevalence rates and causes. It then explains how certain nutrients are important for neurotransmitter production and can impact mood if deficient. Specific amino acids like tryptophan, 5-HTP, and phenylalanine are highlighted as being able to reduce depression when supplemented. The role of other nutrients like B vitamins, essential fats, and methyl donors is also outlined. Overall, the document establishes that nutrition plays a key role in depression through its effect on neurotransmission.
endocrine system.pptx
The pituitary gland has three lobes - anterior, posterior, and intermediate. The anterior lobe secretes growth hormone and others to regulate various processes. The posterior lobe stores and releases oxytocin and antidiuretic hormone. Disorders include dwarfism, gigantism, and others related to hormone imbalances. The pancreas contains islets of Langerhans that secrete insulin and glucagon to regulate blood sugar. Diabetes occurs when there is insufficient insulin. The thyroid gland produces T3 and T4 which regulate metabolism; disorders include hypothyroidism, hyperthyroidism, Graves' disease, and goiter. The parathyroid glands secrete PTH which regulates blood calcium levels.
Mental health and hormones cady
This lecture on the relevance of hormonal optimization in mental health, was presented by Dr. Cady in Salt Lake City, UT at the 2012 Medical Seminar Series coordinated by World Link Medical.
CNS STIMULANTS & NOOTROPICS (COGNITION ENHANCERS) for B.Pharm & Pharm.D
This document discusses CNS stimulants and nootropics, or cognition enhancers. It describes how CNS stimulants produce generalized stimulation of the central nervous system and lists various convulsants, analeptics, and psychostimulants. Nootropics are meant to enhance cerebral functions like memory and are used to treat conditions like Alzheimer's disease, dementia, and learning defects. Common nootropics discussed include cholinergic activators like donepezil and rivastigmine, the NMDA antagonist memantine, and various other drugs like piracetam. Rivastigmine inhibits acetylcholinesterase to increase cholinergic transmission in the brain. Memantine
Presentation on Huntington's disease.pptx
Huntington’s Disease
It is basal ganglia disease. It is caused by degeneration of caudate nucleus and putamen, especially of GABAergic and acetylcholinergic neurons.
A Brief History of Huntington's Disease
Huntington's disease (HD) is named after George Huntington, who described it among residents of East Hampton, Long Island in 1872. It is a hereditary neurodegenerative disease. In 1993, a collaborative group of investigators discovered the gene that causes HD. As a result of this discovery it is now possible to diagnose HD with blood or tissue samples.
Detail of Degenerated Parts of the Brain during Huntington’s Disease
The detail of degenerated parts of the brain during huntington’s disease are given below:
Caudate nucleus
The basal ganglia are a group of structures found deep within the cerebral hemispheres. The structures generally included in the basal ganglia are the caudate, putamen, and globus pallidus in the cerebrum, the substantia nigra in the midbrain, and the subthalamic nucleus in the diencephalon. Despite the name, the basal ganglia are not actually ganglia.
The separate nuclei of the basal ganglia all have extensive roles of their own in the brain, but when referring to them as one network the function most frequently associated with the basal ganglia involves movement.
The basal ganglia receive information from the cortex, much of which is sent first to the caudate and putamen (which together are often referred to as the striatum). After the information is processed by the basal ganglia, it is sent back to the cortex by way of the thalamus. Thus, the pathway from the cortex to the basal ganglia and then back to the cortex via the thalamus forms a loop.
In simplistic terms, the functions of the basal ganglia in motor control are to facilitate movement and inhibit competing movements. For example, when someone tries to make an intentional movement like reaching for a pencil, the basal ganglia help to facilitate the movement by allowing motor plans associated with that movement (reaching and grasping in this case) to be activated. At the same time, the basal ganglia cause motor plans that might counteract the movement (perhaps flexing in this case) to be inhibited. The result is a smooth and fluid movement.
Putamen
The putamen is a round structure located at the base of the forebrain (telencephalon). The putamen and caudate nucleus together form the dorsal striatum. It is also one of the structures that composes the basal ganglia. A primary function of the putamen is to regulate movements at various stages (e.g. preparation and execution) and influence various types of learning. It employs GABA, acetylcholine, and enkephalin to perform its functions.
GABA
Gamma-Amino Butyric acid (GABA) is an amino acid which acts as a neurotransmitter in the central nervous system. It inhibits nerve transmission in the brain, calming nervous activity.
Acetylecholine
Use acetylcholine as a neurotransmitter to innervate cholinergic neurons.
Psychiatric nursing review
The document provides an overview of foundations of psychiatric mental health nursing. It discusses definitions of mental health and mental illness. It describes the Diagnostic and Statistical Manual of Mental Disorders (DSM-IV-TR) and issues of self-awareness for nurses. Neurobiological theories of mental illness are presented, including the roles of neurotransmitters and neuroanatomic structures. Psychopharmacological treatments for conditions such as schizophrenia, depression, and anxiety are summarized. Finally, psychosocial theories of Sigmund Freud are briefly introduced.
Similar to Brain Injury Power Point (20)
CNS STIMULANTS & NOOTROPICS (COGNITION ENHANCERS) for B.Pharm & Pharm.D
CNS STIMULANTS & NOOTROPICS (COGNITION ENHANCERS) for B.Pharm & Pharm.D
Brain Injury Power Point
- 1. Serious ScienceYour text here
- 4. ILLNESS REPORTABLY ASSOCIATED TO BRAIN INJURY • Alzheimer's • Parkinson's • MS • Motor Neuron's • Chronic fatigue syndrome • Adrenal fatigue / Failure • Depression • Anxiety • Diabetes
- 6. THE COCK PIT • Hypothalamus • Pituitary Gland • Pineal Gland • Thyroid Gland • Adrenal Glands
- 7. The Critical fuels 1. Human Growth Hormone HGH 2. IGF – 1 Insulin like Growth factor 1 3. Free Testosterone / 4. Cortisol- Adrenals 5. TSH Thyroid Stimulating Hormone 6. T3 / T4 Thyroid 7. DHEAs - Adrenals • ACTH Pituitary • LH – Luteinising Hormone • FSH - Follical Stimulating Hormone
- 9. The Damage
- 10. TO SUMMERISE • Main Cause of Brain Injury is caused by Concussion or head impact • Brain Injury Can have everlasting effects • Natural Nuero Steroid ( Hormonal) Testing is King !! • 3 months post Injury 12 months post Injury • Restoring hormone levels to their optimal, pre-injury and youthful levels results in remarkable recovery of many of the impaired functions. • Duty of Care
Editor's Notes
- What research shows is that