This document discusses several common skin diseases and infections. It begins by explaining that skin diseases can involve changes to the epidermis, dermis or subcutaneous tissue, and that their appearance may vary over time and with treatment. It then covers several primary and secondary bacterial skin infections like impetigo, folliculitis, boils, and cellulitis. It also discusses viral infections like chickenpox and shingles, as well as conditions such as acne, hidradenitis suppurativa, allergic eczema, and scabies. For each condition, it provides details on symptoms, signs, causes and characteristics.
This document discusses bacterial infections of the skin. It begins by describing the natural defenses of skin and normal skin flora. It then discusses the different types of primary and secondary skin infections, including impetigo, folliculitis, furuncles, carbuncles, cellulitis, erysipelas, cutaneous abscesses, and necrotizing fasciitis. It provides details on specific bacterial infections caused by Staphylococcus aureus and streptococci. The document concludes with sections on laboratory diagnosis of skin infections and principles of therapy.
Bacterial infections of the skin can be primary, caused by a single pathogen affecting normal skin, or secondary, occurring in already diseased skin. Common primary bacterial skin infections include impetigo, folliculitis, and boils, often caused by Staphylococcus aureus or streptococci. Secondary infections may involve the skin folds, and include conditions like intertrigo and toe web infections. Specific bacterial infections are impetigo, folliculitis, furuncles, carbuncles, cellulitis, erysipelas, cutaneous abscesses, and necrotizing fasciitis. Acne is also discussed as an infection of hair follicles by Propionibacterium acnes.
Bacterial infections of the skin can be either primary (caused by a single pathogen affecting normal skin) or secondary (occurring in already diseased skin). Common primary pathogens include Staphylococcus aureus, beta-hemolytic streptococci, and coryneform bacteria. S. aureus is responsible for infections like impetigo, folliculitis, furuncles, carbuncles, scalded skin syndrome, and toxic shock syndrome. Group A beta-hemolytic streptococci cause impetigo and cellulitis. Other bacterial infections include ecthyma, erysipelas, cellulitis, cutaneous abscesses, and necrotizing fasciitis. Laboratory diagnosis involves specimen collection
Impetigo is a common superficial bacterial skin infection that usually affects children. It is classified as bullous or non-bullous based on presentation. Staphylococcus aureus and Streptococcus pyogenes are the main causes. Non-bullous impetigo starts as pink macules or vesicles that evolve into pustules and crusted erosions. Bullous impetigo causes fluid-filled blisters that rupture, leaving thin crusts. Treatment involves oral antibiotics such as dicloxacillin, cephalexin, or clindamycin. Proper hand hygiene and avoiding scratching the lesions is important to prevent spread.
This document provides information about various skin diseases and infections. It begins with an introduction to pathological skin changes and then discusses different types of primary and secondary skin infections. Specific bacterial infections caused by Staphylococcus aureus and group A streptococci are examined. Common skin infections and their symptoms are defined, including impetigo, ecthyma, folliculitis, furuncles, carbuncles, cellulitis, and acne. Diagnosis and treatment of bacterial skin infections is also covered. The document concludes with information about fungal infections such as tinea.
The document discusses various types of scalp infections including folliculitis, fungal infections, impetigo, lichen planus, ringworm, seborrheic dermatitis, sebaceous cysts, and cephalohematoma. Folliculitis is an infection of the hair follicles caused by bacteria, viruses, or fungi. Fungal infections like those caused by Candida or Malassezia can also lead to scalp infections. Impetigo and lichen planus cause rashes and swelling on the scalp through bacterial infections or autoimmune reactions. Ringworm and seborrheic dermatitis cause flaky, irritated patches through fungal infections or inflammatory reactions. Seb
Impetigo is a common bacterial skin infection in children that causes red sores that can break open and ooze fluid. It is often caused by Streptococcus pyogenes or Staphylococcus aureus bacteria entering through broken skin. Symptoms include itchy red sores that form crusty yellow patches. Treatment involves topical or oral antibiotics. Folliculitis is another bacterial skin infection affecting hair follicles, typically caused by Staph. aureus, that causes inflamed pus-filled lesions. Boils (furuncles) and carbuncles are deep skin abscesses linked to Staph. aureus infection of hair follicles that require antibiotics. Burns are classified by depth of skin damage from superficial to full
This document discusses several common skin diseases and infections. It begins by explaining that skin diseases can involve changes to the epidermis, dermis or subcutaneous tissue, and that their appearance may vary over time and with treatment. It then covers several primary and secondary bacterial skin infections like impetigo, folliculitis, boils, and cellulitis. It also discusses viral infections like chickenpox and shingles, as well as conditions such as acne, hidradenitis suppurativa, allergic eczema, and scabies. For each condition, it provides details on symptoms, signs, causes and characteristics.
This document discusses bacterial infections of the skin. It begins by describing the natural defenses of skin and normal skin flora. It then discusses the different types of primary and secondary skin infections, including impetigo, folliculitis, furuncles, carbuncles, cellulitis, erysipelas, cutaneous abscesses, and necrotizing fasciitis. It provides details on specific bacterial infections caused by Staphylococcus aureus and streptococci. The document concludes with sections on laboratory diagnosis of skin infections and principles of therapy.
Bacterial infections of the skin can be primary, caused by a single pathogen affecting normal skin, or secondary, occurring in already diseased skin. Common primary bacterial skin infections include impetigo, folliculitis, and boils, often caused by Staphylococcus aureus or streptococci. Secondary infections may involve the skin folds, and include conditions like intertrigo and toe web infections. Specific bacterial infections are impetigo, folliculitis, furuncles, carbuncles, cellulitis, erysipelas, cutaneous abscesses, and necrotizing fasciitis. Acne is also discussed as an infection of hair follicles by Propionibacterium acnes.
Bacterial infections of the skin can be either primary (caused by a single pathogen affecting normal skin) or secondary (occurring in already diseased skin). Common primary pathogens include Staphylococcus aureus, beta-hemolytic streptococci, and coryneform bacteria. S. aureus is responsible for infections like impetigo, folliculitis, furuncles, carbuncles, scalded skin syndrome, and toxic shock syndrome. Group A beta-hemolytic streptococci cause impetigo and cellulitis. Other bacterial infections include ecthyma, erysipelas, cellulitis, cutaneous abscesses, and necrotizing fasciitis. Laboratory diagnosis involves specimen collection
Impetigo is a common superficial bacterial skin infection that usually affects children. It is classified as bullous or non-bullous based on presentation. Staphylococcus aureus and Streptococcus pyogenes are the main causes. Non-bullous impetigo starts as pink macules or vesicles that evolve into pustules and crusted erosions. Bullous impetigo causes fluid-filled blisters that rupture, leaving thin crusts. Treatment involves oral antibiotics such as dicloxacillin, cephalexin, or clindamycin. Proper hand hygiene and avoiding scratching the lesions is important to prevent spread.
This document provides information about various skin diseases and infections. It begins with an introduction to pathological skin changes and then discusses different types of primary and secondary skin infections. Specific bacterial infections caused by Staphylococcus aureus and group A streptococci are examined. Common skin infections and their symptoms are defined, including impetigo, ecthyma, folliculitis, furuncles, carbuncles, cellulitis, and acne. Diagnosis and treatment of bacterial skin infections is also covered. The document concludes with information about fungal infections such as tinea.
The document discusses various types of scalp infections including folliculitis, fungal infections, impetigo, lichen planus, ringworm, seborrheic dermatitis, sebaceous cysts, and cephalohematoma. Folliculitis is an infection of the hair follicles caused by bacteria, viruses, or fungi. Fungal infections like those caused by Candida or Malassezia can also lead to scalp infections. Impetigo and lichen planus cause rashes and swelling on the scalp through bacterial infections or autoimmune reactions. Ringworm and seborrheic dermatitis cause flaky, irritated patches through fungal infections or inflammatory reactions. Seb
Impetigo is a common bacterial skin infection in children that causes red sores that can break open and ooze fluid. It is often caused by Streptococcus pyogenes or Staphylococcus aureus bacteria entering through broken skin. Symptoms include itchy red sores that form crusty yellow patches. Treatment involves topical or oral antibiotics. Folliculitis is another bacterial skin infection affecting hair follicles, typically caused by Staph. aureus, that causes inflamed pus-filled lesions. Boils (furuncles) and carbuncles are deep skin abscesses linked to Staph. aureus infection of hair follicles that require antibiotics. Burns are classified by depth of skin damage from superficial to full
This document discusses various bacterial infections that can affect the skin, including Staphylococcus aureus, Streptococcus pyogenes, Corynebacterium, and Actinomyces. It provides details on specific cutaneous infections such as impetigo, ecthyma, folliculitis, furuncles, carbuncles, erysipelas, bacterial scalded skin syndrome, and actinomycosis. It describes the causative bacteria, clinical presentation, diagnosis and treatment of these important skin infections.
This document summarizes various infections and disorders of the integumentary system. It describes cellulitis as a bacterial skin infection causing red, swollen skin. Furuncles are acute abscesses caused by Staphylococcus aureus, often appearing as boils. Carbuncles are clusters of interconnected furuncles. Fungal infections like ringworm can cause patches of scaling on the scalp or body. Viral infections like herpes simplex cause clusters of blisters on the lips or genitals. Skin cancers can develop from abnormal cell growth in the skin.
This document describes vesiculo-bullous lesions, which present clinically as vesicles or bullae that often rupture early, appearing as ulcerated or erosive areas. Some key points:
- Vesicles are fluid-filled lesions less than 1 cm, while bullae contain fluid and are over 1 cm.
- Causes include trauma, infection, autoimmunity, and genetic factors.
- Examples described include herpes simplex infection, varicella zoster infection, and hand foot and mouth disease. Clinical features, pathogenesis, management are provided for each. Classification is discussed based on acute vs chronic presentation, clinical presentation, and histopathological location.
This document provides information about Impetigo, a common and contagious skin infection that mainly affects children. It describes the symptoms and signs of Impetigo such as reddish sores that develop into honey-colored crusts. Risk factors include age 2-5 years old, close contact with others, warm weather, and broken skin. Impetigo is caused by bacteria like Staphylococcus aureus and Streptococcus pyogenes. Prevention methods include washing clothes and bedding daily, wearing gloves when applying ointment, and keeping infected children home until no longer contagious.
Skin infections can be caused by bacteria, viruses, or fungi entering through breaks in the skin or spreading from other sites. Staphylococcus aureus is a common cause and can result in impetigo (pus-filled vesicles), folliculitis (infection of hair follicles), furuncles (boils), or carbuncles (coalesced furuncles). Toxic shock syndrome and scalded skin syndrome are severe illnesses caused by S. aureus toxins. Streptococcus pyogenes commonly causes impetigo, erysipelas (painful skin infection), or scarlet fever (rash following strep throat). Proper treatment depends on identifying the causative agent.
This document provides information on extrapulmonary manifestations of tuberculosis (TB), including cutaneous TB. It discusses various types of cutaneous TB such as tuberculous chancre, lupus vulgaris, scrofuloderma, and tuberculids. Risk factors, clinical features, diagnosis, and treatment are described for each type. Causative organisms include Mycobacterium tuberculosis as well as atypical mycobacteria. Diagnostic tests include tuberculin skin tests, interferon gamma release assays, and sputum/tissue cultures. Treatment typically involves a combination of anti-TB medications.
The document discusses bacterial and viral infections of the skin. It provides details on the resident skin flora, including Staphylococcus epidermidis and aerobic diphtheroids that help defend the skin. Staphylococcal infections like impetigo and furunculosis are described. Impetigo can be caused by S. aureus or streptococci and presents as blisters or crusts. Furuncles are acute hair follicle infections while carbuncles involve adjacent follicles. Treatment options including antibiotics are outlined. Viral warts caused by HPV are also summarized, including common, plantar, plane and anogenital presentations and treatments.
Non-bullous impetigo is a superficial bacterial skin infection that causes clusters of vesicles or pustules that rupture, forming honey-colored crusts. Bullous impetigo causes larger bullae that rupture, leaving larger crusts. Ecthyma is a deeper skin infection than impetigo that can cause scarring. Folliculitis causes pustules in hair follicles while furuncles are deeper infected follicles and carbuncles involve connected furuncles. Erysipelas presents as shiny, raised plaques caused mainly by streptococci bacteria. Treatment focuses on good hygiene, treating predisposing factors, cleaning and antibiotics.
This document provides information about impetigo, a common skin infection seen mostly in children. It defines impetigo and describes how it is transmitted through direct contact. It notes that impetigo occurs more commonly in hot, humid weather and when the skin barrier is disrupted. The document outlines the epidemiology of impetigo, describing who is most affected. It discusses the etiology, describing the bacteria that cause impetigo and the different types. It provides details on clinical manifestations, diagnosis, pathophysiology, treatment options including antibiotics, and concludes by thanking the reader.
The document discusses different types of ectoparasite infestations including tungiasis (jiggers), fleas, lice, and their prevention and treatment. It provides details on the clinical features and life cycles of each parasite. For all infestations, prevention focuses on hygiene, environmental controls like cleaning living areas, and application of insecticides. Treatment involves carefully removing parasites from the skin followed by cleaning and medicating the affected area.
This document summarizes various neonatal dermatoses (skin conditions in newborns). It describes common viral infections like herpes simplex virus and varicella zoster virus. Bacterial infections from Staphylococcus aureus, Group B streptococci, and Listeria monocytogenes are outlined. Congenital conditions like congenital syphilis, epidermolysis bullosa, incontinentia pigmenti, and cutaneous mastocytosis are also summarized. For each condition, the document provides details on presentation, diagnosis, and treatment.
This document provides a detailed overview of various rashes and eruptions that can present with fever. It begins by outlining the important aspects of a patient's history for evaluating rashes. It then describes the characteristics of different skin lesions and provides a classification system for rashes. The remainder of the document discusses specific rash-causing conditions categorized by the classification, outlining their etiology, appearance, and associated clinical features in 1-3 sentences for each. Key conditions covered include measles, rubella, erythema infectiosum, dengue fever, scarlet fever, chickenpox, and smallpox.
This document summarizes various bacterial skin infections. It discusses the normal skin flora and pathogens that can cause infection. Common infections covered include impetigo, folliculitis, furuncles, carbuncles, ecthyma, erysipelas, cellulitis, and cutaneous tuberculosis. Causative organisms, characteristics, diagnosis and treatment are described for each infection. The document also provides overview of leprosy, its various clinical forms, routes of transmission and immunological factors that determine presentation.
This document provides information on different types of rashes that can accompany febrile illness. It begins with an introduction on febrile rashes and their diagnostic approach. It then describes various rash patterns such as maculopapular, vesicular, and urticarial rashes. Specific infectious diseases that can present with rashes like measles, rubella, and scarlet fever are discussed. It concludes by summarizing diagnostic tests and distinguishing features of different febrile rashes.
Cutaneous Infections of skin disease managementkareniedavid7
Impetigo is a common, highly contagious bacterial skin infection that primarily affects children. It is caused by Staphylococcus aureus or Streptococcus pyogenes. Non-bullous impetigo presents as vesicles or pustules surrounded by erythema, while bullous impetigo features flaccid bullae without surrounding erythema. Both are usually treated with oral antibiotics like erythromycin. Tinea cruris is a fungal infection of the groin caused by dermatophytes like Trichophyton rubrum. It presents as well-demarcated erythematous scaly plaques and is diagnosed by microscopy of skin scrapings in potassium hydroxide.
Cutaneous manifestations of systemic infections can include:
1. Osler nodes and Janeway lesions in infective endocarditis, appearing as tender nodules on fingers or palms.
2. Rashes caused by viral exanthems like measles, rubella, enteroviruses. Measles causes Koplik's spots.
3. Fungal infections in immunocompromised patients like candidiasis, dermatophytosis, cryptococcus causing papules or plaques.
4. Bacterial infections may cause cellulitis, ecthyma gangrenosum from Pseudomonas, or disseminated lesions in meningococcemia.
The document discusses various disorders, conditions, and diseases that affect the integumentary system. It provides definitions and details on common skin issues like acne, psoriasis, eczema, contact dermatitis, athlete's foot, hives, rosacea, vitiligo, impetigo, boils, carbuncles, ringworm, warts, keloids, MRSA, gangrene, harlequin ichthyosis, scleroderma, alopecia areata, and burns. Treatment options are mentioned for some conditions. A wide range of both infectious and non-infectious skin problems are examined.
This document provides information about various skin infestations caused by parasites, including scabies, pediculosis (lice infestations), and myasis (fly infestations). It describes the causative agents, symptoms, diagnosis, and treatment of each condition. Scabies is caused by the mite Sarcoptes scabiei and results in a very itchy rash and burrows in the skin. It spreads through direct skin-to-skin contact. Pediculosis involves infestation by head, body, or pubic lice, which feed on human blood and spread through close physical contact or fomites. Proper treatment requires killing the parasites as well as cleaning environments and belong
share - Lions, tigers, AI and health misinformation, oh my!.pptxTina Purnat
• Pitfalls and pivots needed to use AI effectively in public health
• Evidence-based strategies to address health misinformation effectively
• Building trust with communities online and offline
• Equipping health professionals to address questions, concerns and health misinformation
• Assessing risk and mitigating harm from adverse health narratives in communities, health workforce and health system
This document discusses various bacterial infections that can affect the skin, including Staphylococcus aureus, Streptococcus pyogenes, Corynebacterium, and Actinomyces. It provides details on specific cutaneous infections such as impetigo, ecthyma, folliculitis, furuncles, carbuncles, erysipelas, bacterial scalded skin syndrome, and actinomycosis. It describes the causative bacteria, clinical presentation, diagnosis and treatment of these important skin infections.
This document summarizes various infections and disorders of the integumentary system. It describes cellulitis as a bacterial skin infection causing red, swollen skin. Furuncles are acute abscesses caused by Staphylococcus aureus, often appearing as boils. Carbuncles are clusters of interconnected furuncles. Fungal infections like ringworm can cause patches of scaling on the scalp or body. Viral infections like herpes simplex cause clusters of blisters on the lips or genitals. Skin cancers can develop from abnormal cell growth in the skin.
This document describes vesiculo-bullous lesions, which present clinically as vesicles or bullae that often rupture early, appearing as ulcerated or erosive areas. Some key points:
- Vesicles are fluid-filled lesions less than 1 cm, while bullae contain fluid and are over 1 cm.
- Causes include trauma, infection, autoimmunity, and genetic factors.
- Examples described include herpes simplex infection, varicella zoster infection, and hand foot and mouth disease. Clinical features, pathogenesis, management are provided for each. Classification is discussed based on acute vs chronic presentation, clinical presentation, and histopathological location.
This document provides information about Impetigo, a common and contagious skin infection that mainly affects children. It describes the symptoms and signs of Impetigo such as reddish sores that develop into honey-colored crusts. Risk factors include age 2-5 years old, close contact with others, warm weather, and broken skin. Impetigo is caused by bacteria like Staphylococcus aureus and Streptococcus pyogenes. Prevention methods include washing clothes and bedding daily, wearing gloves when applying ointment, and keeping infected children home until no longer contagious.
Skin infections can be caused by bacteria, viruses, or fungi entering through breaks in the skin or spreading from other sites. Staphylococcus aureus is a common cause and can result in impetigo (pus-filled vesicles), folliculitis (infection of hair follicles), furuncles (boils), or carbuncles (coalesced furuncles). Toxic shock syndrome and scalded skin syndrome are severe illnesses caused by S. aureus toxins. Streptococcus pyogenes commonly causes impetigo, erysipelas (painful skin infection), or scarlet fever (rash following strep throat). Proper treatment depends on identifying the causative agent.
This document provides information on extrapulmonary manifestations of tuberculosis (TB), including cutaneous TB. It discusses various types of cutaneous TB such as tuberculous chancre, lupus vulgaris, scrofuloderma, and tuberculids. Risk factors, clinical features, diagnosis, and treatment are described for each type. Causative organisms include Mycobacterium tuberculosis as well as atypical mycobacteria. Diagnostic tests include tuberculin skin tests, interferon gamma release assays, and sputum/tissue cultures. Treatment typically involves a combination of anti-TB medications.
The document discusses bacterial and viral infections of the skin. It provides details on the resident skin flora, including Staphylococcus epidermidis and aerobic diphtheroids that help defend the skin. Staphylococcal infections like impetigo and furunculosis are described. Impetigo can be caused by S. aureus or streptococci and presents as blisters or crusts. Furuncles are acute hair follicle infections while carbuncles involve adjacent follicles. Treatment options including antibiotics are outlined. Viral warts caused by HPV are also summarized, including common, plantar, plane and anogenital presentations and treatments.
Non-bullous impetigo is a superficial bacterial skin infection that causes clusters of vesicles or pustules that rupture, forming honey-colored crusts. Bullous impetigo causes larger bullae that rupture, leaving larger crusts. Ecthyma is a deeper skin infection than impetigo that can cause scarring. Folliculitis causes pustules in hair follicles while furuncles are deeper infected follicles and carbuncles involve connected furuncles. Erysipelas presents as shiny, raised plaques caused mainly by streptococci bacteria. Treatment focuses on good hygiene, treating predisposing factors, cleaning and antibiotics.
This document provides information about impetigo, a common skin infection seen mostly in children. It defines impetigo and describes how it is transmitted through direct contact. It notes that impetigo occurs more commonly in hot, humid weather and when the skin barrier is disrupted. The document outlines the epidemiology of impetigo, describing who is most affected. It discusses the etiology, describing the bacteria that cause impetigo and the different types. It provides details on clinical manifestations, diagnosis, pathophysiology, treatment options including antibiotics, and concludes by thanking the reader.
The document discusses different types of ectoparasite infestations including tungiasis (jiggers), fleas, lice, and their prevention and treatment. It provides details on the clinical features and life cycles of each parasite. For all infestations, prevention focuses on hygiene, environmental controls like cleaning living areas, and application of insecticides. Treatment involves carefully removing parasites from the skin followed by cleaning and medicating the affected area.
This document summarizes various neonatal dermatoses (skin conditions in newborns). It describes common viral infections like herpes simplex virus and varicella zoster virus. Bacterial infections from Staphylococcus aureus, Group B streptococci, and Listeria monocytogenes are outlined. Congenital conditions like congenital syphilis, epidermolysis bullosa, incontinentia pigmenti, and cutaneous mastocytosis are also summarized. For each condition, the document provides details on presentation, diagnosis, and treatment.
This document provides a detailed overview of various rashes and eruptions that can present with fever. It begins by outlining the important aspects of a patient's history for evaluating rashes. It then describes the characteristics of different skin lesions and provides a classification system for rashes. The remainder of the document discusses specific rash-causing conditions categorized by the classification, outlining their etiology, appearance, and associated clinical features in 1-3 sentences for each. Key conditions covered include measles, rubella, erythema infectiosum, dengue fever, scarlet fever, chickenpox, and smallpox.
This document summarizes various bacterial skin infections. It discusses the normal skin flora and pathogens that can cause infection. Common infections covered include impetigo, folliculitis, furuncles, carbuncles, ecthyma, erysipelas, cellulitis, and cutaneous tuberculosis. Causative organisms, characteristics, diagnosis and treatment are described for each infection. The document also provides overview of leprosy, its various clinical forms, routes of transmission and immunological factors that determine presentation.
This document provides information on different types of rashes that can accompany febrile illness. It begins with an introduction on febrile rashes and their diagnostic approach. It then describes various rash patterns such as maculopapular, vesicular, and urticarial rashes. Specific infectious diseases that can present with rashes like measles, rubella, and scarlet fever are discussed. It concludes by summarizing diagnostic tests and distinguishing features of different febrile rashes.
Cutaneous Infections of skin disease managementkareniedavid7
Impetigo is a common, highly contagious bacterial skin infection that primarily affects children. It is caused by Staphylococcus aureus or Streptococcus pyogenes. Non-bullous impetigo presents as vesicles or pustules surrounded by erythema, while bullous impetigo features flaccid bullae without surrounding erythema. Both are usually treated with oral antibiotics like erythromycin. Tinea cruris is a fungal infection of the groin caused by dermatophytes like Trichophyton rubrum. It presents as well-demarcated erythematous scaly plaques and is diagnosed by microscopy of skin scrapings in potassium hydroxide.
Cutaneous manifestations of systemic infections can include:
1. Osler nodes and Janeway lesions in infective endocarditis, appearing as tender nodules on fingers or palms.
2. Rashes caused by viral exanthems like measles, rubella, enteroviruses. Measles causes Koplik's spots.
3. Fungal infections in immunocompromised patients like candidiasis, dermatophytosis, cryptococcus causing papules or plaques.
4. Bacterial infections may cause cellulitis, ecthyma gangrenosum from Pseudomonas, or disseminated lesions in meningococcemia.
The document discusses various disorders, conditions, and diseases that affect the integumentary system. It provides definitions and details on common skin issues like acne, psoriasis, eczema, contact dermatitis, athlete's foot, hives, rosacea, vitiligo, impetigo, boils, carbuncles, ringworm, warts, keloids, MRSA, gangrene, harlequin ichthyosis, scleroderma, alopecia areata, and burns. Treatment options are mentioned for some conditions. A wide range of both infectious and non-infectious skin problems are examined.
This document provides information about various skin infestations caused by parasites, including scabies, pediculosis (lice infestations), and myasis (fly infestations). It describes the causative agents, symptoms, diagnosis, and treatment of each condition. Scabies is caused by the mite Sarcoptes scabiei and results in a very itchy rash and burrows in the skin. It spreads through direct skin-to-skin contact. Pediculosis involves infestation by head, body, or pubic lice, which feed on human blood and spread through close physical contact or fomites. Proper treatment requires killing the parasites as well as cleaning environments and belong
share - Lions, tigers, AI and health misinformation, oh my!.pptxTina Purnat
• Pitfalls and pivots needed to use AI effectively in public health
• Evidence-based strategies to address health misinformation effectively
• Building trust with communities online and offline
• Equipping health professionals to address questions, concerns and health misinformation
• Assessing risk and mitigating harm from adverse health narratives in communities, health workforce and health system
Here is the updated list of Top Best Ayurvedic medicine for Gas and Indigestion and those are Gas-O-Go Syp for Dyspepsia | Lavizyme Syrup for Acidity | Yumzyme Hepatoprotective Capsules etc
TEST BANK For An Introduction to Brain and Behavior, 7th Edition by Bryan Kol...rightmanforbloodline
TEST BANK For An Introduction to Brain and Behavior, 7th Edition by Bryan Kolb, Ian Q. Whishaw, Verified Chapters 1 - 16, Complete Newest Versio
TEST BANK For An Introduction to Brain and Behavior, 7th Edition by Bryan Kolb, Ian Q. Whishaw, Verified Chapters 1 - 16, Complete Newest Version
TEST BANK For An Introduction to Brain and Behavior, 7th Edition by Bryan Kolb, Ian Q. Whishaw, Verified Chapters 1 - 16, Complete Newest Version
Cell Therapy Expansion and Challenges in Autoimmune DiseaseHealth Advances
There is increasing confidence that cell therapies will soon play a role in the treatment of autoimmune disorders, but the extent of this impact remains to be seen. Early readouts on autologous CAR-Ts in lupus are encouraging, but manufacturing and cost limitations are likely to restrict access to highly refractory patients. Allogeneic CAR-Ts have the potential to broaden access to earlier lines of treatment due to their inherent cost benefits, however they will need to demonstrate comparable or improved efficacy to established modalities.
In addition to infrastructure and capacity constraints, CAR-Ts face a very different risk-benefit dynamic in autoimmune compared to oncology, highlighting the need for tolerable therapies with low adverse event risk. CAR-NK and Treg-based therapies are also being developed in certain autoimmune disorders and may demonstrate favorable safety profiles. Several novel non-cell therapies such as bispecific antibodies, nanobodies, and RNAi drugs, may also offer future alternative competitive solutions with variable value propositions.
Widespread adoption of cell therapies will not only require strong efficacy and safety data, but also adapted pricing and access strategies. At oncology-based price points, CAR-Ts are unlikely to achieve broad market access in autoimmune disorders, with eligible patient populations that are potentially orders of magnitude greater than the number of currently addressable cancer patients. Developers have made strides towards reducing cell therapy COGS while improving manufacturing efficiency, but payors will inevitably restrict access until more sustainable pricing is achieved.
Despite these headwinds, industry leaders and investors remain confident that cell therapies are poised to address significant unmet need in patients suffering from autoimmune disorders. However, the extent of this impact on the treatment landscape remains to be seen, as the industry rapidly approaches an inflection point.
Basavarajeeyam is a Sreshta Sangraha grantha (Compiled book ), written by Neelkanta kotturu Basavaraja Virachita. It contains 25 Prakaranas, First 24 Chapters related to Rogas& 25th to Rasadravyas.
- Video recording of this lecture in English language: https://youtu.be/kqbnxVAZs-0
- Video recording of this lecture in Arabic language: https://youtu.be/SINlygW1Mpc
- Link to download the book free: https://nephrotube.blogspot.com/p/nephrotube-nephrology-books.html
- Link to NephroTube website: www.NephroTube.com
- Link to NephroTube social media accounts: https://nephrotube.blogspot.com/p/join-nephrotube-on-social-media.html
Promoting Wellbeing - Applied Social Psychology - Psychology SuperNotesPsychoTech Services
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Rasamanikya is a excellent preparation in the field of Rasashastra, it is used in various Kushtha Roga, Shwasa, Vicharchika, Bhagandara, Vatarakta, and Phiranga Roga. In this article Preparation& Comparative analytical profile for both Formulationon i.e Rasamanikya prepared by Kushmanda swarasa & Churnodhaka Shodita Haratala. The study aims to provide insights into the comparative efficacy and analytical aspects of these formulations for enhanced therapeutic outcomes.
Does Over-Masturbation Contribute to Chronic Prostatitis.pptxwalterHu5
In some case, your chronic prostatitis may be related to over-masturbation. Generally, natural medicine Diuretic and Anti-inflammatory Pill can help mee get a cure.
Muktapishti is a traditional Ayurvedic preparation made from Shoditha Mukta (Purified Pearl), is believed to help regulate thyroid function and reduce symptoms of hyperthyroidism due to its cooling and balancing properties. Clinical evidence on its efficacy remains limited, necessitating further research to validate its therapeutic benefits.
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4. Bacterial skin infection
• Most common bacterial skin infections
are caused by coagulase-positive
Staphylococcus aureus or group A
beta hemolytic streptococci.
• Syphilis has been called “the great
masquerader” and “the great imitator”
based on the many varied
presentations of the cutaneous and
other organ system findings.
7. Clinical feature
Bullous
• begins as a superficial vesicle
• Rapidly progresses to a flaccid bulla,
with sharp margins and no
surrounding erythema.
• the bulla ruptures,a moist yellow
crust forms.
• arises on grossly normal skin and
favors moist intertriginous areas,
• Complication :SSSS
Non –bullous
• begins as a single lesion
• manifesting as a red macule or
papule that becomes a vesicle
• The vesicle ruptures, forming an
erosion
• honey-colored crust commonly seen
with impetigo
• occurs on the face or extremities
• Complication :PSGN
8. Management
• simple incision with a number 11 scalpel blade and drainage with
evacuation of the pus.
• oral antibiotics
• for MSSA infections include dicloxacillin, cephalexin,clindamycin,
doxycycline, minocycline, and trimethoprim sulfamethoxazole.
carriers of MSSA and MRSA should be
• treated with intranasal mupirocin (Bactroban) ointment twice a
day for 5 days and 4% chlorhexidine (Hibiclens) wash daily.
9. Erysipelas
• infection of the dermis
and subcutaneous tissue
• poorly demarcated
borders
• caused by Streptococcus
or Staphylococcus species
• Associated with
lymphangitis .
cellulitis
• superficial form of
cellulitis
• sharply demarcated
borders
• caused almost exclusively
by Streptococcus.
• Associated with
lymphangitis less
common .
12. carbuncle
• Carbuncles are an
aggregate of infected
hair follicles that form
broad, swollen,
erythematous, deep,
and painful masses
that usually open and
drain through
multiple tracts.
furuncle
• a furuncle is a tender,
erythematous, firm or
fluctuant mass of
walled-off purulent
material, arising from
the hair follicle.
13. Sychosis vulgaris
• It’s a cutaneous condition characterized by a chronic
infection of the chin or bearded region.
• The irritation is caused by a deep infection of hair
follicles.
• species of Staphylococcus or Propionibacterium bacteria.
• Asymptomatic or painful and tender
erythematouspapules and pustules may form around
coarse hair in the beard
14. • Sycosis vulgaris is a cutaneous condition characterized by a chronic
infection of the chin or bearded region. The irritation is caused by a deep
infection of hair follicles, often by species of Staphylococcus or
Propionibacterium bacteria. Asymptomatic or painful and tender
erythematouspapules and pustules may form around coarse hair in the
beard
16. syphilis
• Syphilis is a sexually transmitted disease with worldwide
distribution.
• caused by Treponema pallidum , a spirochetal bacterium.
• After inoculation through skin or mucous membranes, the
bacterium spreads throughout the body via the lymphatic
system and blood.
17. Primary
• At the site of entry .
• 10-90 days after
infection.
• dusky red macule
evolves into a papule.
Surface necrosis occurs
with
• indurated, firm ulcer
(chancre), with a raised
border.
• Ulcer spontaneously
heals 2-10 weeks .
Secondary
• Systemic symptoms
• 4to 10 weeks after onset
of infection.
• pink, violaceous or red-
brown macular, r or
papulosquamous
eruption appears on the
face, trunk, extremities.
• A patchy, “moth-eaten”
and/or diffuse alopecia
may develop
• Wart-like moist papules
condylomata lata appear.
• Heal within 3-12 wk
tertiary
• primarily affects the
central nervous system
and the cardiovascular
system.
• Small group of people.