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ANTEPARTUM HAEMORRHAGE
GOALS
1. To identify and recognize causes of APH
2. To be able to resuscitate properly
3. To be able to manage appropriately
ANTEPARTUM HAEMORRHAGE
 PV bleeding after 22 weeks till birth of baby
 Incidence: 3 – 5%
 Before 22 weeks – miscarriage/abortion
 E.g. threatened miscarriage
 Have to differentiate from “show”
 blood-stained mucoid discharge
CAUSES
 Indeterminate 45%
 Abruptio Placenta 30%
 Placenta Praevia 20%
 Local causes 5%
(e.g. vaginitis, cervical polyp, ectropion,
carcinoma)
HISTORY
 Colour, quantity of bleeding
 Precipitating factors e.g. trauma, intercourse
 Contractions
 Leaking liquor
 Fetal movement
 Previous ultrasound
GENERAL MEASURES
 Call for help
 Resuscitation: A, B, C
 BP,PR
 2 IV access
 Blood ix
 FBC, coagulation profile
 Rhesus
 Cross match blood 4 units
 Ultrasound
 Identify Cause of bleeding
 NO VE until cause determined
PLACENTA PRAEVIA
 Placenta lying partly or wholly within the lower uterine
segment (> 28 weeks)
 < 28 weeks - Low-lying placenta
Upper segment
Lower segment
CLASSIFICATION
 PP I : Within lower segment (5cm)
 PP II : Reaches internal os
 PP III : Partially covers os
 PP IV: Completely covers os
INCIDENCE
 18 weeks: 26%
 32 weeks: 5%
 Term: 0.5%
 Only about 1 in 10 of low lying placenta persists as
clinically relevant placenta praevia towards term
RISK FACTORS
 Maternal
 Previous caesarean section (associated with placenta
accreta)
 Previous placenta praevia
 Grandmultip
 Elderly
 Smoking
 Fetal
 Twins
COMPLICATIONS
 Maternal
 PPH
 Placenta accreta
 Maternal death
 Fetal
 Prematurity
 IUGR
 Congenital malformations (2-fold increase
in PP and abruptio)
MANAGEMENT
 Diagnosis by U/S (ideally with transvaginal U/S)
 No V/E
 Active PV bleeding- immediate delivery
 Bleeding stop- hospitalized till delivery
MORBIDLY ADHERENT PLACENTA
 Placenta penetrates through the
myometrium of uterus
 Includes
- Placenta accreta
- Placenta increta
- Placenta percreta
 Risk factors
- Previous scar
- Previous D&C
 Women with previous caesarean section and at the
same time had placenta praevia at increase risk of
morbidly adherent placenta
 Increase in maternal morbidity and mortality
LINK BETWEEN NUMBER OF PREVIOUS CAESAREAN
SECTION AND RISK OF MORBIDLY ADHERENT PLACENTA
& HYSTERECTOMY
NUMBER OF
PREVIOUS C-
SECTION
NUMBER OF
WOMEN
NUMBER OF
WOMEN WITH
PLACENTA
ACCRETA
CHANCE OF
PLACENTA
ACCRETA IF
PRAEVIA
NUMBER OF
HYSTERECTOMIES
0 6201 15 (0.24%) 3% 40 (0.65%)
1 15808 49 (0.31%) 11% 67 (0.42%)
2 6324 36 (0.57%) 40% 57 (0.9%)
3 1452 31 (2.13%) 61 % 35 (2.4%)
4 258 6 (2.33%) 67 % 9 (3.49%)
PLACENTA ABRUPTIO
 Premature separation of the placenta prior to the 3rd
stage of labour
 Incidence: 1-2%
 Placental separation if severe enough, can cause
inadequate nutrition and oxygenation of the fetus –
fetal death
 Can be concealed, revealed or mixed (most common)
RISK FACTORS
 Maternal
 Previous abruptio
 Grandmultipara
 PIH
 Trauma, fall, massage
 ECV
 Substance abuse
 Smoking
 Fetal
 Twins
 Polyhydramnios (with sudden decompression on rupture of
membrane)
MANAGEMENT (FETUS ALIVE)
 Assess maternal and fetal stability
 ARM to augment labour and prevent amniotic fluid
embolism
 Continuos CTG monitoring
 LSCS for:
 Fetal distress
 Persistent bleeding
 Vaginal delivery not imminent
 If mother not stable (need to resuscitate first!)
 Paeds for neonatal resuscitation
 Watch for PPH
MANAGEMENT (FETUS DEAD)
 Assess maternal stability and coagulopathy
 Vigorous replacement of fluid and blood products
 Correct any coagulopathy
 Vaginal delivery unless severe haemorrhage
 Watch for PPH
MATERNAL COMPLICATIONS
 Couvelaire uterus
 Blood seeping into uterus (myometrium) causing
uterine atony
 Coagulopathy
 Due to profuse bleeding
 Due to thromboplastin release from decidua
 Hypovolaemic shock
 Renal cortical necrosis
 Amniotic fluid embolism
 Maternal death
FETAL COMPLICATIONS
 IUGR
 Birth asphyxia
 Perinatal death
PRAEVIA ABRUPTIO
Painless Painful
Revealed Can be concealed
Uterus soft Uterus tense/ tender
FH usually present FH may be absent
May have abnormal lie
or malpresentation
May be hard to feel
fetal parts
How to differentiate? Clinical presentation
 If there is no identifiable cause of APH
 Deliver by 40 weeks – due to possibility of minor
abruption (may lead to placental insufficiency)
 Refer if undelivered by 40 wks + 0 days
INDETERMINATE APH
 Bleeding with onset at rupture of
membranes
 Immediate delivery
 High perinatal mortality from fetal
exsanguination
VASA PRAEVIA
PRINCIPLES OF MANAGEMENT
 Assess maternal condition and stabilize if necessary.
 Then assess the fetal condition.
 Determine the cause of the bleeding
 Manage according to underlying cause.
Manage Antepartum Haemorrhage (APH) Effectively in Pregnancy

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Manage Antepartum Haemorrhage (APH) Effectively in Pregnancy

  • 2. GOALS 1. To identify and recognize causes of APH 2. To be able to resuscitate properly 3. To be able to manage appropriately
  • 3. ANTEPARTUM HAEMORRHAGE  PV bleeding after 22 weeks till birth of baby  Incidence: 3 – 5%  Before 22 weeks – miscarriage/abortion  E.g. threatened miscarriage  Have to differentiate from “show”  blood-stained mucoid discharge
  • 4. CAUSES  Indeterminate 45%  Abruptio Placenta 30%  Placenta Praevia 20%  Local causes 5% (e.g. vaginitis, cervical polyp, ectropion, carcinoma)
  • 5. HISTORY  Colour, quantity of bleeding  Precipitating factors e.g. trauma, intercourse  Contractions  Leaking liquor  Fetal movement  Previous ultrasound
  • 6. GENERAL MEASURES  Call for help  Resuscitation: A, B, C  BP,PR  2 IV access  Blood ix  FBC, coagulation profile  Rhesus  Cross match blood 4 units  Ultrasound  Identify Cause of bleeding  NO VE until cause determined
  • 7. PLACENTA PRAEVIA  Placenta lying partly or wholly within the lower uterine segment (> 28 weeks)  < 28 weeks - Low-lying placenta Upper segment Lower segment
  • 8. CLASSIFICATION  PP I : Within lower segment (5cm)  PP II : Reaches internal os  PP III : Partially covers os  PP IV: Completely covers os
  • 9. INCIDENCE  18 weeks: 26%  32 weeks: 5%  Term: 0.5%  Only about 1 in 10 of low lying placenta persists as clinically relevant placenta praevia towards term
  • 10. RISK FACTORS  Maternal  Previous caesarean section (associated with placenta accreta)  Previous placenta praevia  Grandmultip  Elderly  Smoking  Fetal  Twins
  • 11. COMPLICATIONS  Maternal  PPH  Placenta accreta  Maternal death  Fetal  Prematurity  IUGR  Congenital malformations (2-fold increase in PP and abruptio)
  • 12. MANAGEMENT  Diagnosis by U/S (ideally with transvaginal U/S)  No V/E  Active PV bleeding- immediate delivery  Bleeding stop- hospitalized till delivery
  • 13. MORBIDLY ADHERENT PLACENTA  Placenta penetrates through the myometrium of uterus  Includes - Placenta accreta - Placenta increta - Placenta percreta  Risk factors - Previous scar - Previous D&C
  • 14.  Women with previous caesarean section and at the same time had placenta praevia at increase risk of morbidly adherent placenta  Increase in maternal morbidity and mortality
  • 15. LINK BETWEEN NUMBER OF PREVIOUS CAESAREAN SECTION AND RISK OF MORBIDLY ADHERENT PLACENTA & HYSTERECTOMY NUMBER OF PREVIOUS C- SECTION NUMBER OF WOMEN NUMBER OF WOMEN WITH PLACENTA ACCRETA CHANCE OF PLACENTA ACCRETA IF PRAEVIA NUMBER OF HYSTERECTOMIES 0 6201 15 (0.24%) 3% 40 (0.65%) 1 15808 49 (0.31%) 11% 67 (0.42%) 2 6324 36 (0.57%) 40% 57 (0.9%) 3 1452 31 (2.13%) 61 % 35 (2.4%) 4 258 6 (2.33%) 67 % 9 (3.49%)
  • 16. PLACENTA ABRUPTIO  Premature separation of the placenta prior to the 3rd stage of labour  Incidence: 1-2%
  • 17.  Placental separation if severe enough, can cause inadequate nutrition and oxygenation of the fetus – fetal death
  • 18.  Can be concealed, revealed or mixed (most common)
  • 19. RISK FACTORS  Maternal  Previous abruptio  Grandmultipara  PIH  Trauma, fall, massage  ECV  Substance abuse  Smoking  Fetal  Twins  Polyhydramnios (with sudden decompression on rupture of membrane)
  • 20. MANAGEMENT (FETUS ALIVE)  Assess maternal and fetal stability  ARM to augment labour and prevent amniotic fluid embolism  Continuos CTG monitoring  LSCS for:  Fetal distress  Persistent bleeding  Vaginal delivery not imminent  If mother not stable (need to resuscitate first!)  Paeds for neonatal resuscitation  Watch for PPH
  • 21. MANAGEMENT (FETUS DEAD)  Assess maternal stability and coagulopathy  Vigorous replacement of fluid and blood products  Correct any coagulopathy  Vaginal delivery unless severe haemorrhage  Watch for PPH
  • 22. MATERNAL COMPLICATIONS  Couvelaire uterus  Blood seeping into uterus (myometrium) causing uterine atony  Coagulopathy  Due to profuse bleeding  Due to thromboplastin release from decidua  Hypovolaemic shock  Renal cortical necrosis  Amniotic fluid embolism  Maternal death
  • 23. FETAL COMPLICATIONS  IUGR  Birth asphyxia  Perinatal death
  • 24. PRAEVIA ABRUPTIO Painless Painful Revealed Can be concealed Uterus soft Uterus tense/ tender FH usually present FH may be absent May have abnormal lie or malpresentation May be hard to feel fetal parts How to differentiate? Clinical presentation
  • 25.  If there is no identifiable cause of APH  Deliver by 40 weeks – due to possibility of minor abruption (may lead to placental insufficiency)  Refer if undelivered by 40 wks + 0 days INDETERMINATE APH
  • 26.  Bleeding with onset at rupture of membranes  Immediate delivery  High perinatal mortality from fetal exsanguination VASA PRAEVIA
  • 27. PRINCIPLES OF MANAGEMENT  Assess maternal condition and stabilize if necessary.  Then assess the fetal condition.  Determine the cause of the bleeding  Manage according to underlying cause.