This document summarizes the duties and responsibilities of doctors in cases of suspected poisoning. It describes relevant sections of the Indian Penal Code and Criminal Procedure Code, including requirements to preserve evidence and inform authorities. It also provides guidance on treating specific types of poisoning through gastric lavage, activated charcoal, and antidotes. Common poisons discussed include organophosphates, heavy metals, acids, and other toxic substances. Post-mortem findings are also outlined.
1. Alcohol is a depressant that slows down the body and mind, affecting reaction time, decision making, and judgment. Its effects are felt quickly upon consumption as it is absorbed into the bloodstream within 5-10 minutes.
2. Drinking alcohol can damage many organs, especially the brain and liver. Even low doses can impair functions while high doses can cause loss of consciousness, coma, or death. The liver is responsible for filtering alcohol but can only process about one drink per hour.
3. Binge drinking and underage drinking are especially dangerous and illegal, increasing risks of injuries, alcohol poisoning, liver disease, and other health issues. The safest amount of alcohol is none. Treatment
This document discusses neurotics and alcohol. It defines different categories of neurotics such as cerebral, spinal, and peripheral neurotics. It then discusses alcohol in depth, including its absorption, distribution, metabolism, excretion, actions, acute and chronic poisoning, and treatment. It defines terms like inebriation and provides details on ethyl alcohol including its production, consumption levels, and percentages in different alcoholic beverages.
This document discusses neurotics and alcohol. It describes different categories of neurotics including cerebral, spinal and peripheral neurotics. It then discusses alcohol in depth, including its absorption, distribution, metabolism, excretion, actions, acute and chronic poisoning, and treatment for alcohol poisoning. Key points include alcohol is primarily metabolized in the liver, it has depressant effects on the central nervous system, and chronic alcohol use can lead to organ damage and conditions like cirrhosis of the liver.
The document discusses various types of alcohols such as ethyl alcohol and methyl alcohol, how alcohols like ethanol are manufactured through fermentation, and the pharmacological effects of acute and chronic alcohol consumption including intoxication, organ damage, and diseases like cirrhosis as well as the potential benefits and uses of alcohol in small amounts.
This document summarizes information on arsenic and lead poisoning. It discusses the sources, physical properties, uses, and toxic effects of arsenic and lead. For both poisons, it describes the absorption, distribution, and mechanisms of toxicity. The clinical manifestations of acute and chronic poisoning are outlined for each element. Diagnosis involves measuring levels in blood and urine. Treatment of arsenic poisoning involves chelation therapy with BAL, penicillamine or DMSA. For severe lead poisoning, chelation with CaNa2EDTA or BAL is recommended along with supportive care. Mild to moderate lead poisoning is treated with oral chelation agents like D-penicillamine.
This document discusses various corrosive poisons including acids and alkalies. It describes their properties, clinical effects, post-mortem findings, and medicolegal aspects. Major acids discussed are sulfuric acid, nitric acid, hydrochloric acid, oxalic acid, and carbolic acid. Their local tissue-destroying effects and systemic toxicity are outlined. Major alkalies covered are ammonia, potassium hydroxide, sodium hydroxide, and sodium/potassium carbonates. Their caustic properties, inhalation dangers, and causes of death are summarized. The document provides details on evaluating and treating corrosive poisoning cases.
This document summarizes the duties and responsibilities of doctors in cases of suspected poisoning. It describes relevant sections of the Indian Penal Code and Criminal Procedure Code, including requirements to preserve evidence and inform authorities. It also provides guidance on treating specific types of poisoning through gastric lavage, activated charcoal, and antidotes. Common poisons discussed include organophosphates, heavy metals, acids, and other toxic substances. Post-mortem findings are also outlined.
1. Alcohol is a depressant that slows down the body and mind, affecting reaction time, decision making, and judgment. Its effects are felt quickly upon consumption as it is absorbed into the bloodstream within 5-10 minutes.
2. Drinking alcohol can damage many organs, especially the brain and liver. Even low doses can impair functions while high doses can cause loss of consciousness, coma, or death. The liver is responsible for filtering alcohol but can only process about one drink per hour.
3. Binge drinking and underage drinking are especially dangerous and illegal, increasing risks of injuries, alcohol poisoning, liver disease, and other health issues. The safest amount of alcohol is none. Treatment
This document discusses neurotics and alcohol. It defines different categories of neurotics such as cerebral, spinal, and peripheral neurotics. It then discusses alcohol in depth, including its absorption, distribution, metabolism, excretion, actions, acute and chronic poisoning, and treatment. It defines terms like inebriation and provides details on ethyl alcohol including its production, consumption levels, and percentages in different alcoholic beverages.
This document discusses neurotics and alcohol. It describes different categories of neurotics including cerebral, spinal and peripheral neurotics. It then discusses alcohol in depth, including its absorption, distribution, metabolism, excretion, actions, acute and chronic poisoning, and treatment for alcohol poisoning. Key points include alcohol is primarily metabolized in the liver, it has depressant effects on the central nervous system, and chronic alcohol use can lead to organ damage and conditions like cirrhosis of the liver.
The document discusses various types of alcohols such as ethyl alcohol and methyl alcohol, how alcohols like ethanol are manufactured through fermentation, and the pharmacological effects of acute and chronic alcohol consumption including intoxication, organ damage, and diseases like cirrhosis as well as the potential benefits and uses of alcohol in small amounts.
This document summarizes information on arsenic and lead poisoning. It discusses the sources, physical properties, uses, and toxic effects of arsenic and lead. For both poisons, it describes the absorption, distribution, and mechanisms of toxicity. The clinical manifestations of acute and chronic poisoning are outlined for each element. Diagnosis involves measuring levels in blood and urine. Treatment of arsenic poisoning involves chelation therapy with BAL, penicillamine or DMSA. For severe lead poisoning, chelation with CaNa2EDTA or BAL is recommended along with supportive care. Mild to moderate lead poisoning is treated with oral chelation agents like D-penicillamine.
This document discusses various corrosive poisons including acids and alkalies. It describes their properties, clinical effects, post-mortem findings, and medicolegal aspects. Major acids discussed are sulfuric acid, nitric acid, hydrochloric acid, oxalic acid, and carbolic acid. Their local tissue-destroying effects and systemic toxicity are outlined. Major alkalies covered are ammonia, potassium hydroxide, sodium hydroxide, and sodium/potassium carbonates. Their caustic properties, inhalation dangers, and causes of death are summarized. The document provides details on evaluating and treating corrosive poisoning cases.
This document provides information on corrosive poisons including sulfuric acid, oxalic acid, phenol, and nitric acid. It describes their physical and chemical properties, mechanisms of action, clinical features of poisoning, treatment approaches, causes of death, and post-mortem appearances. It also discusses their judicious and injudicious uses and medicolegal significance. The document is intended to educate about these dangerous corrosive poisons through detailed descriptions and comparisons.
Mercury is a liquid metal that is highly toxic, especially in its vaporized form and when ingested as certain mercury compounds. The document discusses mercury's properties and various forms, how mercury poisoning affects the body and can cause damage to organs like the kidneys and brain, symptoms of both acute and chronic mercury toxicity, treatment options, and postmortem findings related to mercury poisoning.
Ethanol acts as a central nervous system depressant by enhancing the effects of the inhibitory neurotransmitter GABA at GABA-A receptors. It is metabolized in the liver by alcohol dehydrogenase and aldehyde dehydrogenase. Acute intoxication can cause impairment, loss of coordination, and respiratory depression, while chronic use is associated with conditions like cirrhosis of the liver and cardiomyopathy. Treatment for acute intoxication involves maintaining airway and circulation, gastric lavage, glucose supplementation, and benzodiazepines for seizures. Chronic alcoholism is treated with supervised withdrawal, benzodiazepine substitution, psychotherapy, and disulfiram to produce an aversive reaction if alcohol is consumed. Methanol poisoning is treated
This document discusses various post-mortem changes that can help estimate time since death, including lividity, rigor mortis, decomposition, and biochemical changes in vitreous humor, blood, and cerebrospinal fluid over time. Measurement of facial hair growth or food digestion can also provide clues to estimate how long someone survived after their last shave or meal. Radiocarbon dating of bone can also potentially estimate time of death for skeletal remains.
Methanol poisoning causes metabolic acidosis, optic neuritis, renal toxicity, and CNS depression. Symptoms include odor on breath, acid urine with acetone and albumin, retinal ganglion cell degeneration, and convulsions. Death is mainly due to metabolic acidosis from formic acid production and respiratory depression from CNS effects.
Inorganic (non metallic) irritant Poisons by Sunil Kumar Dahasunil kumar daha
Please find the power point on Inorganic (non metallic) irritants poisons. I tried to present it on understandable way and all the contents are reviewed by experts and from very reliable references. Thank you
A presentation on Arsenic Poisoning, from a brief history, compounds, uses, circumstances of poisoning, types with clinical symptoms, diagnosis, treatment and postmortem findings. Subject from Forensic Medicine and Toxicology.
#arsenicpoisoning #arsenic
This document summarizes information about gastric acid secretion and factors that influence it. It discusses:
1) Gastric acid is produced by parietal cells in the stomach and plays a key role in protein digestion. Parietal cells secrete acid in response to gastrin, histamine, and acetylcholine.
2) Peptic ulcers are caused by an imbalance between gastric acid and the protective mucus lining, often due to H. pylori infection. Antacids and proton pump inhibitors are used to treat ulcers by reducing acid.
3) Proton pump inhibitors irreversibly block the proton pump in parietal cells, strongly inhibiting gastric acid secretion.
This document discusses corrosive poisoning from ingesting or contacting strong acids and bases. It notes that these chemicals can erode and destroy surfaces and cause systemic effects. Treatment involves dilution, avoiding gastric lavage, and symptomatic care. Complications can be acute like perforations or chronic like strictures. Death typically occurs from circulatory collapse within 12-24 hours. Postmortem findings include blackened and eroded stomach lining with potential perforations. The document also discusses vitriolage, which is the malicious throwing of corrosives like sulfuric acid to disfigure victims, which causes severe burns.
1. The document discusses acid-base disturbances and defines acids, bases, and pH. It explains how acids and bases are produced and balanced in the body to maintain a normal blood pH.
2. When there is excess acid or base in the blood, compensation mechanisms attempt to return the pH to normal through buffers, lungs, and kidneys. However, disease can cause acidosis or alkalosis if the imbalance persists.
3. Respiratory and metabolic acidosis and alkalosis are described based on their causes and effects on pH, pCO2, and HCO3 levels. Compensation and treatment approaches are also outlined.
Burns are one of the most common household injuries, especially among children. The term “burn” means more than the burning sensation associated with this injury. Burns are characterized by severe skin damage that causes the affected skin cells to die.
Most people can recover from burns without serious health consequences, depending on the cause and degree of injury. More serious burns require immediate emergency medical care to prevent complications and death
This document provides information about gastrointestinal agents (GI agents), which are drugs used to treat GI disorders. It discusses the classifications of GI agents including acidifying agents, antacids, protectives, adsorbents, and cathartics. It then describes common antacids including aluminum hydroxide gel, calcium carbonate, and magnesium salts. The ideal characteristics of antacids are outlined. Common calcium-containing and magnesium-containing antacids are also discussed in more detail.
Heavy metal poisoning, especially from arsenic, is a major global health issue. Arsenic is commonly found in soil, water, and foods in some areas. It interferes with cellular energy production. Acute arsenic poisoning causes nausea, vomiting, and diarrhea resembling cholera. Chronic arsenic poisoning over years causes skin pigmentation, keratosis, and neurological symptoms. The diagnosis is confirmed by detecting high arsenic levels in urine, hair, and nails. Treatment involves removing the patient from exposure and giving dimercaprol as an antidote.
Uric acid is the end product of purine metabolism and is excreted in urine. It is produced from the breakdown of purines from food and cells. There are two main methods for determining serum uric acid levels - the Caraway method which uses phosphotungstic acid and measures absorbance at 700nm, and the enzymatic method using uricase and peroxidase to produce a colored product measured at 500-540nm. Normal serum uric acid levels are 3.5-7.2 mg/dl for males and 2.5-6.2 mg/dl for females. Hyperuricemia can be caused by reduced excretion due to renal disorders or increased production from metabolic disorders and diseases
This document provides information about antacids, including their mechanism of action, classification, and examples. It discusses how antacids work by neutralizing gastric acid through chemical reactions or by forming protective coatings in the stomach. Antacids are classified as systemic or non-systemic. Systemic antacids like sodium bicarbonate can cause alkalosis while non-systemic antacids like aluminum hydroxide, magnesium hydroxide, and magnesium carbonate act locally in the stomach without systemic absorption. Common antacids are discussed in detail with their chemical reactions, effects, uses, and side effects.
This document summarizes post-mortem changes that can help estimate time of death. It describes immediate changes that occur within 30 minutes of death, early changes within 36 hours such as algor mortis (body cooling), rigor mortis (muscle stiffening), and livor mortis (post-mortem lividity). Late changes after 36 hours include decomposition stages of bloating, decay, and skeletonization. Specific changes in the skin, eyes, and potassium levels in vitreous humor are also noted.
This document discusses acidifying agents used to treat conditions caused by low stomach acid such as achlorhydria. It describes two main acidifying agents - ammonium chloride and hydrochloric acid. Ammonium chloride is used as a systemic acidifier, expectorant, and diuretic. It is prepared by neutralizing hydrochloric acid with ammonia. Hydrochloric acid is used as an acidifying agent, solvent, and catalyst. It is prepared by reacting sulfuric acid with sodium chloride. Both agents work to increase stomach acid levels and support digestion when acid production is impaired.
Gastrointestinal agents are classified into different groups based on their mechanism of action. These include acidifying agents, antacids, adsorbents, laxatives, cathartics, purgatives, and protective agents. Acidifying agents like dilute hydrochloric acid are used to increase acid levels in the stomach. Antacids like aluminum hydroxide gel and magnesium hydroxide are used to neutralize excess stomach acid in conditions like hyperacidity. Laxatives, cathartics and purgatives help promote bowel movements and treat constipation.
This document provides information on corrosive poisons including sulfuric acid, oxalic acid, phenol, and nitric acid. It describes their physical and chemical properties, mechanisms of action, clinical features of poisoning, treatment approaches, causes of death, and post-mortem appearances. It also discusses their judicious and injudicious uses and medicolegal significance. The document is intended to educate about these dangerous corrosive poisons through detailed descriptions and comparisons.
Mercury is a liquid metal that is highly toxic, especially in its vaporized form and when ingested as certain mercury compounds. The document discusses mercury's properties and various forms, how mercury poisoning affects the body and can cause damage to organs like the kidneys and brain, symptoms of both acute and chronic mercury toxicity, treatment options, and postmortem findings related to mercury poisoning.
Ethanol acts as a central nervous system depressant by enhancing the effects of the inhibitory neurotransmitter GABA at GABA-A receptors. It is metabolized in the liver by alcohol dehydrogenase and aldehyde dehydrogenase. Acute intoxication can cause impairment, loss of coordination, and respiratory depression, while chronic use is associated with conditions like cirrhosis of the liver and cardiomyopathy. Treatment for acute intoxication involves maintaining airway and circulation, gastric lavage, glucose supplementation, and benzodiazepines for seizures. Chronic alcoholism is treated with supervised withdrawal, benzodiazepine substitution, psychotherapy, and disulfiram to produce an aversive reaction if alcohol is consumed. Methanol poisoning is treated
This document discusses various post-mortem changes that can help estimate time since death, including lividity, rigor mortis, decomposition, and biochemical changes in vitreous humor, blood, and cerebrospinal fluid over time. Measurement of facial hair growth or food digestion can also provide clues to estimate how long someone survived after their last shave or meal. Radiocarbon dating of bone can also potentially estimate time of death for skeletal remains.
Methanol poisoning causes metabolic acidosis, optic neuritis, renal toxicity, and CNS depression. Symptoms include odor on breath, acid urine with acetone and albumin, retinal ganglion cell degeneration, and convulsions. Death is mainly due to metabolic acidosis from formic acid production and respiratory depression from CNS effects.
Inorganic (non metallic) irritant Poisons by Sunil Kumar Dahasunil kumar daha
Please find the power point on Inorganic (non metallic) irritants poisons. I tried to present it on understandable way and all the contents are reviewed by experts and from very reliable references. Thank you
A presentation on Arsenic Poisoning, from a brief history, compounds, uses, circumstances of poisoning, types with clinical symptoms, diagnosis, treatment and postmortem findings. Subject from Forensic Medicine and Toxicology.
#arsenicpoisoning #arsenic
This document summarizes information about gastric acid secretion and factors that influence it. It discusses:
1) Gastric acid is produced by parietal cells in the stomach and plays a key role in protein digestion. Parietal cells secrete acid in response to gastrin, histamine, and acetylcholine.
2) Peptic ulcers are caused by an imbalance between gastric acid and the protective mucus lining, often due to H. pylori infection. Antacids and proton pump inhibitors are used to treat ulcers by reducing acid.
3) Proton pump inhibitors irreversibly block the proton pump in parietal cells, strongly inhibiting gastric acid secretion.
This document discusses corrosive poisoning from ingesting or contacting strong acids and bases. It notes that these chemicals can erode and destroy surfaces and cause systemic effects. Treatment involves dilution, avoiding gastric lavage, and symptomatic care. Complications can be acute like perforations or chronic like strictures. Death typically occurs from circulatory collapse within 12-24 hours. Postmortem findings include blackened and eroded stomach lining with potential perforations. The document also discusses vitriolage, which is the malicious throwing of corrosives like sulfuric acid to disfigure victims, which causes severe burns.
1. The document discusses acid-base disturbances and defines acids, bases, and pH. It explains how acids and bases are produced and balanced in the body to maintain a normal blood pH.
2. When there is excess acid or base in the blood, compensation mechanisms attempt to return the pH to normal through buffers, lungs, and kidneys. However, disease can cause acidosis or alkalosis if the imbalance persists.
3. Respiratory and metabolic acidosis and alkalosis are described based on their causes and effects on pH, pCO2, and HCO3 levels. Compensation and treatment approaches are also outlined.
Burns are one of the most common household injuries, especially among children. The term “burn” means more than the burning sensation associated with this injury. Burns are characterized by severe skin damage that causes the affected skin cells to die.
Most people can recover from burns without serious health consequences, depending on the cause and degree of injury. More serious burns require immediate emergency medical care to prevent complications and death
This document provides information about gastrointestinal agents (GI agents), which are drugs used to treat GI disorders. It discusses the classifications of GI agents including acidifying agents, antacids, protectives, adsorbents, and cathartics. It then describes common antacids including aluminum hydroxide gel, calcium carbonate, and magnesium salts. The ideal characteristics of antacids are outlined. Common calcium-containing and magnesium-containing antacids are also discussed in more detail.
Heavy metal poisoning, especially from arsenic, is a major global health issue. Arsenic is commonly found in soil, water, and foods in some areas. It interferes with cellular energy production. Acute arsenic poisoning causes nausea, vomiting, and diarrhea resembling cholera. Chronic arsenic poisoning over years causes skin pigmentation, keratosis, and neurological symptoms. The diagnosis is confirmed by detecting high arsenic levels in urine, hair, and nails. Treatment involves removing the patient from exposure and giving dimercaprol as an antidote.
Uric acid is the end product of purine metabolism and is excreted in urine. It is produced from the breakdown of purines from food and cells. There are two main methods for determining serum uric acid levels - the Caraway method which uses phosphotungstic acid and measures absorbance at 700nm, and the enzymatic method using uricase and peroxidase to produce a colored product measured at 500-540nm. Normal serum uric acid levels are 3.5-7.2 mg/dl for males and 2.5-6.2 mg/dl for females. Hyperuricemia can be caused by reduced excretion due to renal disorders or increased production from metabolic disorders and diseases
This document provides information about antacids, including their mechanism of action, classification, and examples. It discusses how antacids work by neutralizing gastric acid through chemical reactions or by forming protective coatings in the stomach. Antacids are classified as systemic or non-systemic. Systemic antacids like sodium bicarbonate can cause alkalosis while non-systemic antacids like aluminum hydroxide, magnesium hydroxide, and magnesium carbonate act locally in the stomach without systemic absorption. Common antacids are discussed in detail with their chemical reactions, effects, uses, and side effects.
This document summarizes post-mortem changes that can help estimate time of death. It describes immediate changes that occur within 30 minutes of death, early changes within 36 hours such as algor mortis (body cooling), rigor mortis (muscle stiffening), and livor mortis (post-mortem lividity). Late changes after 36 hours include decomposition stages of bloating, decay, and skeletonization. Specific changes in the skin, eyes, and potassium levels in vitreous humor are also noted.
This document discusses acidifying agents used to treat conditions caused by low stomach acid such as achlorhydria. It describes two main acidifying agents - ammonium chloride and hydrochloric acid. Ammonium chloride is used as a systemic acidifier, expectorant, and diuretic. It is prepared by neutralizing hydrochloric acid with ammonia. Hydrochloric acid is used as an acidifying agent, solvent, and catalyst. It is prepared by reacting sulfuric acid with sodium chloride. Both agents work to increase stomach acid levels and support digestion when acid production is impaired.
Gastrointestinal agents are classified into different groups based on their mechanism of action. These include acidifying agents, antacids, adsorbents, laxatives, cathartics, purgatives, and protective agents. Acidifying agents like dilute hydrochloric acid are used to increase acid levels in the stomach. Antacids like aluminum hydroxide gel and magnesium hydroxide are used to neutralize excess stomach acid in conditions like hyperacidity. Laxatives, cathartics and purgatives help promote bowel movements and treat constipation.
Similar to Acid and Alkali Poisoning.pdf ......... (20)
This document provides an overview of wound healing, its functions, stages, mechanisms, factors affecting it, and complications.
A wound is a break in the integrity of the skin or tissues, which may be associated with disruption of the structure and function.
Healing is the body’s response to injury in an attempt to restore normal structure and functions.
Healing can occur in two ways: Regeneration and Repair
There are 4 phases of wound healing: hemostasis, inflammation, proliferation, and remodeling. This document also describes the mechanism of wound healing. Factors that affect healing include infection, uncontrolled diabetes, poor nutrition, age, anemia, the presence of foreign bodies, etc.
Complications of wound healing like infection, hyperpigmentation of scar, contractures, and keloid formation.
LAND USE LAND COVER AND NDVI OF MIRZAPUR DISTRICT, UPRAHUL
This Dissertation explores the particular circumstances of Mirzapur, a region located in the
core of India. Mirzapur, with its varied terrains and abundant biodiversity, offers an optimal
environment for investigating the changes in vegetation cover dynamics. Our study utilizes
advanced technologies such as GIS (Geographic Information Systems) and Remote sensing to
analyze the transformations that have taken place over the course of a decade.
The complex relationship between human activities and the environment has been the focus
of extensive research and worry. As the global community grapples with swift urbanization,
population expansion, and economic progress, the effects on natural ecosystems are becoming
more evident. A crucial element of this impact is the alteration of vegetation cover, which plays a
significant role in maintaining the ecological equilibrium of our planet.Land serves as the foundation for all human activities and provides the necessary materials for
these activities. As the most crucial natural resource, its utilization by humans results in different
'Land uses,' which are determined by both human activities and the physical characteristics of the
land.
The utilization of land is impacted by human needs and environmental factors. In countries
like India, rapid population growth and the emphasis on extensive resource exploitation can lead
to significant land degradation, adversely affecting the region's land cover.
Therefore, human intervention has significantly influenced land use patterns over many
centuries, evolving its structure over time and space. In the present era, these changes have
accelerated due to factors such as agriculture and urbanization. Information regarding land use and
cover is essential for various planning and management tasks related to the Earth's surface,
providing crucial environmental data for scientific, resource management, policy purposes, and
diverse human activities.
Accurate understanding of land use and cover is imperative for the development planning
of any area. Consequently, a wide range of professionals, including earth system scientists, land
and water managers, and urban planners, are interested in obtaining data on land use and cover
changes, conversion trends, and other related patterns. The spatial dimensions of land use and
cover support policymakers and scientists in making well-informed decisions, as alterations in
these patterns indicate shifts in economic and social conditions. Monitoring such changes with the
help of Advanced technologies like Remote Sensing and Geographic Information Systems is
crucial for coordinated efforts across different administrative levels. Advanced technologies like
Remote Sensing and Geographic Information Systems
9
Changes in vegetation cover refer to variations in the distribution, composition, and overall
structure of plant communities across different temporal and spatial scales. These changes can
occur natural.
Temple of Asclepius in Thrace. Excavation resultsKrassimira Luka
The temple and the sanctuary around were dedicated to Asklepios Zmidrenus. This name has been known since 1875 when an inscription dedicated to him was discovered in Rome. The inscription is dated in 227 AD and was left by soldiers originating from the city of Philippopolis (modern Plovdiv).
Leveraging Generative AI to Drive Nonprofit InnovationTechSoup
In this webinar, participants learned how to utilize Generative AI to streamline operations and elevate member engagement. Amazon Web Service experts provided a customer specific use cases and dived into low/no-code tools that are quick and easy to deploy through Amazon Web Service (AWS.)
Strategies for Effective Upskilling is a presentation by Chinwendu Peace in a Your Skill Boost Masterclass organisation by the Excellence Foundation for South Sudan on 08th and 09th June 2024 from 1 PM to 3 PM on each day.
Chapter wise All Notes of First year Basic Civil Engineering.pptxDenish Jangid
Chapter wise All Notes of First year Basic Civil Engineering
Syllabus
Chapter-1
Introduction to objective, scope and outcome the subject
Chapter 2
Introduction: Scope and Specialization of Civil Engineering, Role of civil Engineer in Society, Impact of infrastructural development on economy of country.
Chapter 3
Surveying: Object Principles & Types of Surveying; Site Plans, Plans & Maps; Scales & Unit of different Measurements.
Linear Measurements: Instruments used. Linear Measurement by Tape, Ranging out Survey Lines and overcoming Obstructions; Measurements on sloping ground; Tape corrections, conventional symbols. Angular Measurements: Instruments used; Introduction to Compass Surveying, Bearings and Longitude & Latitude of a Line, Introduction to total station.
Levelling: Instrument used Object of levelling, Methods of levelling in brief, and Contour maps.
Chapter 4
Buildings: Selection of site for Buildings, Layout of Building Plan, Types of buildings, Plinth area, carpet area, floor space index, Introduction to building byelaws, concept of sun light & ventilation. Components of Buildings & their functions, Basic concept of R.C.C., Introduction to types of foundation
Chapter 5
Transportation: Introduction to Transportation Engineering; Traffic and Road Safety: Types and Characteristics of Various Modes of Transportation; Various Road Traffic Signs, Causes of Accidents and Road Safety Measures.
Chapter 6
Environmental Engineering: Environmental Pollution, Environmental Acts and Regulations, Functional Concepts of Ecology, Basics of Species, Biodiversity, Ecosystem, Hydrological Cycle; Chemical Cycles: Carbon, Nitrogen & Phosphorus; Energy Flow in Ecosystems.
Water Pollution: Water Quality standards, Introduction to Treatment & Disposal of Waste Water. Reuse and Saving of Water, Rain Water Harvesting. Solid Waste Management: Classification of Solid Waste, Collection, Transportation and Disposal of Solid. Recycling of Solid Waste: Energy Recovery, Sanitary Landfill, On-Site Sanitation. Air & Noise Pollution: Primary and Secondary air pollutants, Harmful effects of Air Pollution, Control of Air Pollution. . Noise Pollution Harmful Effects of noise pollution, control of noise pollution, Global warming & Climate Change, Ozone depletion, Greenhouse effect
Text Books:
1. Palancharmy, Basic Civil Engineering, McGraw Hill publishers.
2. Satheesh Gopi, Basic Civil Engineering, Pearson Publishers.
3. Ketki Rangwala Dalal, Essentials of Civil Engineering, Charotar Publishing House.
4. BCP, Surveying volume 1
हिंदी वर्णमाला पीपीटी, hindi alphabet PPT presentation, hindi varnamala PPT, Hindi Varnamala pdf, हिंदी स्वर, हिंदी व्यंजन, sikhiye hindi varnmala, dr. mulla adam ali, hindi language and literature, hindi alphabet with drawing, hindi alphabet pdf, hindi varnamala for childrens, hindi language, hindi varnamala practice for kids, https://www.drmullaadamali.com
Philippine Edukasyong Pantahanan at Pangkabuhayan (EPP) CurriculumMJDuyan
(𝐓𝐋𝐄 𝟏𝟎𝟎) (𝐋𝐞𝐬𝐬𝐨𝐧 𝟏)-𝐏𝐫𝐞𝐥𝐢𝐦𝐬
𝐃𝐢𝐬𝐜𝐮𝐬𝐬 𝐭𝐡𝐞 𝐄𝐏𝐏 𝐂𝐮𝐫𝐫𝐢𝐜𝐮𝐥𝐮𝐦 𝐢𝐧 𝐭𝐡𝐞 𝐏𝐡𝐢𝐥𝐢𝐩𝐩𝐢𝐧𝐞𝐬:
- Understand the goals and objectives of the Edukasyong Pantahanan at Pangkabuhayan (EPP) curriculum, recognizing its importance in fostering practical life skills and values among students. Students will also be able to identify the key components and subjects covered, such as agriculture, home economics, industrial arts, and information and communication technology.
𝐄𝐱𝐩𝐥𝐚𝐢𝐧 𝐭𝐡𝐞 𝐍𝐚𝐭𝐮𝐫𝐞 𝐚𝐧𝐝 𝐒𝐜𝐨𝐩𝐞 𝐨𝐟 𝐚𝐧 𝐄𝐧𝐭𝐫𝐞𝐩𝐫𝐞𝐧𝐞𝐮𝐫:
-Define entrepreneurship, distinguishing it from general business activities by emphasizing its focus on innovation, risk-taking, and value creation. Students will describe the characteristics and traits of successful entrepreneurs, including their roles and responsibilities, and discuss the broader economic and social impacts of entrepreneurial activities on both local and global scales.
2. Acids
concentration, time of act
precipitation and dissolving proteins – coagulation
necrosis
cause dehydratation of tissue and with water some acids
are heat up and create termic changes
after consume of acid it can immediately come collapse
and quickly death
Autopsy finding: constricted stomach, stomach wall is
oedematous and fat up to 1cm. Vessels are filled by black
altered blood, up to tarry clots (acid hematin).
3. Sulphuric acid - H2SO4
deadly dose is dependent on concentration
approximately 4 – 6 g
symptoms immediately after swallow - burning pain in the mouth,
neck, oesophagus, stomach and in the abdomen, very painful and
continuous, than man screaming of pain and faint
soon after consume begin vomiting of acid
intestine constipation and in the urinary blader is only little of urine
intensive thirst with swallowing disorders, attempt to drink lead to
new vomiting
dehydratation occur
heavy and noisy breathing, hoarse voice, speaking latter impossible
after absorbtion – nervous symptoms, alcali of blood decrease
4. Hydrochloric acid - HCl
Deadly dose at concentrate acid is 10 – 15 g,
at children approximately 2 g
Symptoms are similar as at sulphuric acid
5. Nitric acid - HNO3
Deadly dose is approximately 8 g, death be
coming approximately 12 hours after consume.
Vomit liquid has lemon up to orange coloration.
Autopsy: at nose is yellow foam, finding is
similar as at another acids, but epithelium is
yellow.
6. Acetic acid - CH3COOH
Deadly dose is approximately 12 g, death be coming 1,5
hours up to 2 days after consume.
Symptoms: burning pain in the mouth and pharynx, pain in
the stomach and abdomen. Becam thirst, vomiting, noisy
breathing, irritable cough, temperature grow up.
Nervous signs – tremors, paralysis of the extremites.
Autopsy: similar finding on the body as at anorganic acids.
7. Oxalic acid - (COOH)2 -
CH3COOH
Deadly dose is 15- 20 g, death be coming after 10
minutes up to 30 hours.
After consume begin burning in the mouth and in
the oesophagus, vomiting, pains in the abdomen, hoarse
voice. Vomit liquid is greenbrown up to black. Rising
consciousness disorders, fibrilations of muscles,
spasms, paleness, skin is cold, temperature go down.
Autopsy finding: basicly same as at acid. Oxalic acid
uptake calcium from tissue and form insoluble calcium
oxalate, calcium oxalate crystals can find under
epithelium of stomach and intestine. Body loss calcium,
it lead to muscle malfunction. In the kidney we can see
crystals already macroscopically.
8. Lemon, tartaric and lactic acid
Deadly doses
30 g lemon acid
10 g tartaric acid
Rising acidosis and cause of death is
methaemoglobinaemia and production of acid
haematin.
10. Alkali
Hydroxide create with proteins alkalic substances
(albuminates) and not coagulate proteins.
Dissolving proteins, thereby rising gelatine masses
and colliquative necrosis, which is sticky.
Acid create coagulation necrosis!!!
11. Potassium and sodium hydroxide
KOH & NaOH
Deadly dose: 80 – 100 g
Symptoms: burn on the skin or necrosis, healing by scars.
Per os: promptly burning pain in the mouth, oesophagus,
stomach and in the abdomen. Vomit liquid have strong alkali
reaction and there are parts of epithelium and blood. Cold
feelings, salivation and tremor.
Autopsy finding: burn around mouth, epithelium in the
stomach is incrassate(fat) and sticky. Lining is gelatinous,
toxic damage of myocardium, in the liver necrosis.