PEDIATRICS OSCES REVISION
BY: DR. NYAMBURA KARIUKI
2018
COMPILED BY EFFIE NAILA
OUTLINE
 GENERAL EXAM
 CENTRAL NERVOUS SYSTEM
 CARDIOVASCULAR SYSTEM
 RESPIRATORY SYSTEM
 THE ABDOMEN
 NEONATALOGY
 SPECIMENS
 Laboratory: blood, LP; Radiological: CXR, IVU, MCU, CT scan of brain;
Photographs with clinical diagnoses; Intraosseous line (procedure); IV
cannulation; BP checking
 COMMUNICATION SKILLS: INFORMED CONSENT,
 VACCINES
Examine the lymph nodes of the head & neck
 Check whether there is lymphadenopathy & the overlying
skin (scars, erythema, rashes)
 Palpate from behind the patient
 Look for: From in front: submental, occipital, anterior &
posterior cervical chain, posterior auricular, pre – auricular,
submandibular
 Do for both right & left
 Accurately palpate using pulp of finger & not tips
 Roll the LNs over the muscle or bones to feel them
CONT.
Accurately comment on:
Size
Tenderness
Consistency
Mobility
Tethering to underlying structure
Matted or discrete
THE CENTRAL NERVOUS
SYSTEM
FAQS
Cranial nerves
LL weakness
Reflexes
Motor exam
Sensory exam
1. Child with a history of weakness
Do a motor exam of the lower limbs
Professional etiquette: greet the patient,
introduce yourself & explain to them what you
want to do & obtain consent.
Stand on the right side of the patient &
expose them adequately.
Cont.
Motor examination:
 Inspect & comment on: posture & muscle bulk (visible wasting),
fasciculations, skin changes, wounds or scars
 Palpate the muscle bulk of all muscle groups.
 Assess power across ALL MUSCLE GROUPS & grade.
 Assess tone across ALL THE JOINTS. Comment: hypertonia or
hypotonia & impression.
 Assess reflexes (knee jerk, ankle reflex, ankle clonus, plantar reflex/
Babinski response) Comment: hyperreflexia or hyporeflexia &
impression.
Motor examination of the upper limbs
Professional etiquette.
Stand on the right side of the patient.
Inspection: posture of upper limbs, visible wasting
(comment appropriately)
Palpate: muscle bulk
Tone: across all joints
Power in all muscle groups
Reflexes: brachioradialis (supinator), biceps, triceps
Name 3 contraindications of a lumbar
puncture
Increased ICP
Coagulopathy
Soft tissue infection at site of LP
Cardiopulmonary instability
2. Interpret the following LP findings in a
patient with a chronic headache
Protein in g/L: 0.5
Glucose in mmol/L: 1.6
CSF: Serum ratio is <0.4, reduced (N: CSF: serum
glucose ratio is 0.6)
WBCs: 180
Appearance: fibrin webbed
Differentials: TB meningitis, fungal meningitis
What is the treatment plan
Working diagnosis: Tuberculous
meningitis
2RHZE + 10RH
Steroid therapy
Assess cranial nerve II
Professional etiquette
Stand on right side of bed, sanitize & warm hands
Visual acuity: Snellen’s chart
Distant vision by standing 1m away from child
Color perception: Ishihara charts
Visual field assessment
Pupillary reflexes: direct & consensual
Assess cranial nerves III, IV & VI
 Professional etiquette
 Stand on right side of bed, sanitize & warm hands
 Inspect for ptosis, eye ball symmetry (squint, abnormal
movements) pupil size at rest.
 Accommodation reflex
 Eye movements: hold finger at 30cm & with the head still
move the finger through the various axes of eye movements
(H – shape). Observe for any restrictions in eye movement.
 Ask for double vision
3. The following child presented at pediatric casualty after
falling from a motor cycle. Examine cranial nerves V & VII
Trigeminal nerve (V1, V2 & V3)
 Examine for light touch, pain & temperature in all 3 nerve
distributions: forehead, cheek & jaw
 Inspect & palpate the masseter & temporalis muscle
 Test for power of the masseter muscle: oppose jaw opening
 Test right & left pterygoids muscle power: oppose lateral jaw
movement
 Corneal reflex
Cont.
Facial nerve (VII)
 Frontalis: ask patient to raise eyebrows
 Orbicularis oculi: ask patient to tightly close eyes then you try
& open them
 Ask patient to smile & frown
 Ask patient to puff the cheeks & try to deflate them by taping
on them gently bilaterally
 Test taste on the anterior of the tongue
Child presents with unexplained lower limb (LL) weakness.
Examine the deep tendon reflexes (DTR) on the LLs.
 Professional etiquette.
 Examine both the right & left DTRs.
 Knee reflexes: slightly lift the leg under the knee or
perform the test it in a sitting position with the leg dangling.
Identify & tap the patella & check the CONTRACTION
OF THE QUADRICEPS.
 Remember to perform the Jendrassik’s maneuver
 Ankle reflexes: slightly extend the hip & dorsiflex the foot
Assume this child was alert, normal upper limbs, weakness
of LL & increased DTR, what are your differentials?
Tumor (SOL)
Schistosomiasis
TB spine
Child admitted with a 2 day history of LOC, fever & an
episode of convulsion lasting 3 minutes
Assess the LOC of this child & interpret
your findings.
Do a pediatric GCS scoring.
Give the scores for E, V & M (correctly
report the findings & interpret them)
Comment on the final GCS
Based on the given history give the most likely
diagnosis (ALWAYS GIVE A JUSTIFICATION)
Complicated meningitis: convulsions, fever, LOC &
duration
The LOC isn’t convulsion – induced hence it is not a
febrile seizure but a more ominous condition.
Differentials
CVA
Meningoencephalitis
Cerebral malaria
What is the difference between an UMN &
LMN facial nerve palsy
UMN:
Spares the brow muscles
Spares eye closing
This child has undergone removal of a craniopharyngioma/
Examine cranial nerves II, III, IV & VI
Optic nerve
Distant vision using Snellen’s chart (Distance from
child  1 m away)
Color sense using Ishihara chart
Visual field assessment
How far from you: ____
Where to place finger: ____
Direct & consensual pupillary reflex
Cont.
Oculomotor nerve
 Upward rotation & inward movement of eye ball
 Size of pupil
 Assess for accommodation
Trochlear
 SO4
 Downward movement of eyeball
Abduscens
 Lateral movement of eyeball
Differentials of a child with flaccid paralysis:
Polio
Transverse myelitis
GBS
Atonic cerebral palsy
Investigations for the child with flaccid
paralysis
MRI/CT scan spine: to R/O any
structural problem in the motor area of
spinal cord.
Nerve conduction studies to determine
peripheral neuropathy.
Stool for polio virus.
UMNL vs. LMNL
UMNL
•Hypertonia (spasticity vs. rigidity)
•Fasciculations absent
•Hyperreflexia
•Normal bulk then disuse atrophy sets in with time
•Reduced power (tends towards normal over time if
adequate stimulation is maintained)
•Plantar extensor response (Babinski’s sign)
•Clonus may be present
•Spastic paralysis
LMNL
•Hypotonia
•Fasciculations present
•Hypo/ areflexia
•Atrophy occurs early
•Power is severely decreased
•Plantar flexion response
•Clonus may be absent
•Flaccid paralysis
THE CARDIOVASCULAR
SYSTEM
Examine the precordium of this child while
narrating your findings
 Professional etiquette.
 Stand on the right side of the patient.
 Commence P/E.
 Expose child adequately
 Inspection (accurately report): hyperactive
 Palpation: apex beat, left parasternal heave (LPS)
 Auscultation: apex, tricuspid area, pulmonary area (+ S2) &
aortic area.
Examine the neck of this patient for Jugular
Venous Pulse
Requirements: good lighting, patient reclined at 450
Assess the waveform of the internal jugular vein
lying adjacent to the medial border of the SCM.
Look for pulsation on the right side of the neck
Press on RUQ while watching neck to differentiate
between carotid artery & JVP pulsation
Identify the highest point of pulsation of the JVP
Cont.
 Extend ruler horizontally from the highest pulsation point
 This ruler must cross with a ruler placed at the sternal angle of
Louis at 900
 Report the measurement in cm
 Add 4cm to get the distance to the right atrium
 Report the JVP
 Normal is 9 cm from the RA which corresponds to a pressure
of mmHg.
Features that distinguish arterial & venous
pulses
Carotid arterial pulses
• Pulsation per heart beat: single
• Palpable.
• Pressure at root of neck: Unaffected.
• Pulsation height:
• Unaffected by the hepatojugular
reflux (apply manual pressure over
the RUQ)
• Independent on the posture of the
patient.
• Not dependent on respiration.
Jugular venous pulses
• Pulsation per heat beat: double
waveform in sinus rhythm
• Not palpable.
• Pressure at root of neck: obliterated
• Pulsation height:
• Transient in volume & height by
the hepatojugular reflux.
• Varies with posture of the patient.
• Decreased on inspiration (EXCEPT
IN CONSTRICTIVE
PERICARDITIS; Kussmaul sign)
Possible causes of a raised JVP
 CCF
 Cor pulmonale
 Constrictive pericarditis
 Pulmonary embolism
 Cardiac tamponade
 Tricuspid valvular disease
 Right ventricular infarction
 HCM
 SVC obstruction (SVCO)
 Iatrogenic fluid overload esp. in surgical & renal patients
Measure the BP (sphygmomanometer)
 Professional etiquette.
 Ensure the child is sitting or lying at ease & not anxious.
 The manometer should be at the same level of the cuff on the
patient’s arm & the observer’s eye.
 Identify a cuff that covers most of the upper arm leaving a gap
of 1 cm or so below the axilla & above the antecubital fossa.
 Measure the upper arm circumference using a tape measure
& identify the right cuff.
Cont.
 Palpate the radial pulse as the cuff is inflated to a pressure of
20mmHg above the level at which radial pulsation can no longer be
felt.
 Place stethoscope lightly over brachial artery & deflate cuff at a rate
of 2 – 3 mmHg/s until the first Korotkoff sound is heard. This
correlates with the SBP.
 With further deflation subtle changes occur in the pitch of the sounds.
These correspond to the 2nd
, 3rd
& 4th
(muffling) Korotkoff sounds that
are clinically insignificant.
 When the sounds die away completely (i.e. the 5th
Korotkoff sound),
this correlates with the DBP.
Investigations in a child with HTN
4 limb BP for vascular disease
U/E/Cs: abnormal kidney function can cause HTN
Potassium levels: K+
levels in Conn’s syndrome
(hyperaldosteronism)
Serum renin levels
Doppler U/S of kidneys: renal artery disease
24hr urinary VMA for Phaeochromocytoma
OVERALL ASSESSMENT
What are the causes of HTN in children
Management of CCF
Prop up the patient.
Administer oxygen if in distress.
Medical:
For preload: diuretics
For cardiac contractility: digoxin
For afterload: CCBs
Treat underlying problem
Examine the pulses & narrate your findings
 Professional etiquette
 Stand on the right side of patient
 Warm hands
 Radial pulse rate for a full minute
 Describe rate, rhythm, volume, character, symmetry & radio – femoral delay
(coarctation of aorta is a diagnosis made at birth!), collapsing pulse.
 Brachial pulse
 Carotid pulse (DONE UNILATERALLY)
 Popliteal pulse: Gently flex the knee and feel for the popliteal pulse by deep
palpation in midline in popliteal fossa.
 Posterior tibial artery: behind & slightly below the medial malleolus of ankle
 Dorsalis pedis pulse: lateral to extensor tendon of great toe
Name 3 clinical conditions with a collapsing
pulse
Aortic regurgitation
Hyper – dynamic circulation:
thyrotoxicosis, beriberi
PDA
Large atrio – ventricular defect
15 month old child brought to casualty poor feeding, poor weight
gain & recurrent infections. BF was stopped at 6 months &
supplemented with cow’s milk
Lab test:
RBCs 3.9 low (N: 4 – 6 X 1012
/L)
WBCs: 13.6 high (N: 4 – 11 X 109
/L)
Hb: 8  low (N: > 12g/dl)
MCV:65  low (N: 78 – 98fL)
MCH: 19 (N: 27 – 32 pg)
MCHC: 22
Platelets: 203 (N: 150 – 350 X 109
/L)
Diagnosis: hypochromic microcytic anemia
Cont.
Most important differential: IDA
Others: Anemia of chronic disease;
sideroblastic anemia; lead poisoning;
thalassemia
Underlying cause:
Helminthic infections
Diet
Cont.
Treatment: Elemental iron 6mg/kg/day for 4 – 6
months
To replenish stores
Stages of iron deficiency:
1. Reticuloendothelial stores are depleted.
2a. MCV, MCH.
2b: Hb.
3. Clinical manifestations
THE RESPIRATORY
SYSTEM
This child presents with fever, cough & DIB for 3 days. Examine the
respiratory system by inspection, percussion & auscultation.
 Inspection:
 Cyanosis (central & peripheral), pallor, head nodding, FAN, finger
clubbing; listen for audible sounds (grunting, stridor, wheeze); Use
of accessory muscles; Observe for supraclavicular, suprasternal &
intercostal in – drawing; LCWI; chest wall symmetry; look for
abdominal breathing
 Count RR for 1 minute
 Percussion: All areas & report findings correctly
 Auscultation: Listen to breath sounds & check for vocal fremitus
Comment on the following findings in a
tachypneic child:
 pH: 7.04 (7.35 – 7.45)  low; acidosis
 PO2: 80mmHg (60 – 80mmHg)  normal; not respiratory acidosis
 PCO2: 6mmHg (40 – 60mmHg)  low; respiratory compensation
 HCO3
-
: 6mmol/L (22 – 24 mmol/L)  low; metabolic acidosis
 Na+
: 140  normal
 Cl-
: 108  normal
 Calculated anion gap: 26 (12 – 16 mmol/L)  elevated
Cont.
Conclusion: metabolic acidosis
with respiratory compensation
Add: you must do anion gap
{(Na+
+ K+
) - (Cl-
+ HCO3)} &
comment on it:
Common causes of metabolic acidosis with
a high anion gap
Poisoning
DKA
lactic acidosis
Uremia
Pancreatitis
Causes of normal anion gap acidosis
Renal tubular acidosis
Causes of hypochloremic, hypokalemic
metabolic alkalosis
Pyloric stenosis
Bicarbonate ingestion
Steroids
Frusemide
Baters disease
Adrenal pathology
The following is a radiological specimen of a 2 year
old child with fever, cough & progressive DIB
 Name the radiological specimen completely e.g. CHEST X
RAY as opposed to X ray; remember to comment on the
view.
 Quality: Rotation, Inspiration, Penetration
 Skeletal thoracic cage
 Lung findings
 Cardiac silhouette
 Heart size: CTR
 Diaphragm
Treatment plan for above child (write on
Treatment sheet)
Classify: e.g. severe etc.
Oxygen administration (method plus
flow rates)
Penicillin & gentamicin: dosages &
duration are MUST KNOWS.
Feeding & fluids
This evaluation was performed on a refugee camp on
a patient with persistent cough & fever
Write out a report of this specimen
State that it is a CHEST X RAY
Quality
Skeletal cage
Lung fields: multiple, widespread opacities
Heart size
Diaphragm
Diagnosis: Miliary TB
Treatment of TB
Intensive phase  2 months; RHZE/
RHZS
Continuation phase  4 months: RH
Nutrition
Any other infection
Do PITC
Child with fever, cough & DIB for 3 days
associated with poor feeding
Examine the respiratory system through inspection &
relevant GE for purposes of determining the severity of
the disease
Outline:
Diagnosis
Classification
Definitive treatment: O2 therapy, penicillin &
gentamicin (KNOW DOSAGES)
Previously well child with cough & fever for 1
day, DIB
General exam relevant to respiratory
system: include lymphadenopathy
Highest priority investigations:
CXR, HIV test, BGAs
HIV test because of recurrent
LRTI
Pneumothorax
CXR: collapsed lung, mediastinal shift, gas
in pleural cavity
Management:
Chest tube insertion
Underlying pathology
Taking care of nutrition
Interpret the radiological image provided & asses as follows
 Name the specimen as Chest X – Ray & state the view. Evaluate lung fields &
airway & give findings
 Interpretation:
 Verify the biodata
 Quality: R (clavicle or anterior rib length); I (count the ribs); Penetration.
 Look at right lung; left lung
 Pleural borders
 Costophrenic angles
 Cardiophrenic angles
 Hilar regions
 Tracheal position
 State diagnosis & differentials
This child is being managed for ascites.
Examine for ascites.
 Professional etiquette + warming your hands
 Expose adequately
 Inspection:
 Stand at the foot of the bed to accurately assess abdominal fullness,
symmetry & hernia orifices
 On right side of patient: Accurate assessment of umbilical stump,
look for scars, superficial venous distention & MWR (movement
with respiration)
 Palpation: first ask whether the child is in pain while warming your
hands before touching the patient then remember to look at the face of
the patient)
Differentials
 Exudative
 Abdominal malignancy
 TB peritonitis
 Transudative
 High hydrostatic pressure:
 Chronic liver disease
 Portal HTN
 Low oncotic pressure
 Renal disease: nephrotic syndrome
 Protein losing enteropathy
This child presents with abdominal distention
for the 1st
3 months.
Active and playful but poor feeding habits
Inspect & palpate abdomen
2 most important differentials based on
imaging modality provided (CXR with fecal
loading & hepatomegaly)
Constipation & hepatomegaly
Cont.
 Bedside manner
 Ensure comfort is adequate + appropriate exposure
 Inspect the abdomen & give findings: shape, umbilicus, MWR, skin &
surface of abdomen, inguinal area & external genitalia
 Palpation: before starting ask for any abdominal tenderness; look at the
face of the patient (light & deep) various quadrants & be systematic;
feel for left kidney, spleen, right kidney, liver, urinary bladder, groins.
 Percussion: shifting dullness & fluid thrill
 Auscultate for bowel movements & vascular bruits
 Anal & rectal examination
Abdominal pain
This 6 year old boy is brought to the casualty
with abdominal pain for the past month. He
passes stool once a week & his stool looks
like hard pellets. Take additional relevant
history from the guardian to help you reach a
diagnosis (constipation, food allergy esp.
cow milk food allergy).
Relevant history
Any pain while passing stool (dyschezia)
Blood on the surface of hard stool.
Has the child attained toilet training.
Does the child soil his pants lately (encopresis)
Associated symptoms: abdominal swelling, vomiting
Etiology: diet, water intake, allergy to cow’s milk &
dairy, similar history amongst family members,
psychosocial factors
How would you manage this condition
Reassure parents & advice them to stay calm
Address any psychosocial issues
Toilet training (regular sitting on the toilet for long
enough; steps to ensure proper seating)
Encourage physical activity
Encourage fluid intake
Diet: fiber rich fruits & vegetables
Medical: laxatives, peglec
Palpate the abdomen of this child.
Professional etiquette
Stand on right side
Expose adequately & appropriately
Ask for abdominal tenderness
Light & deep palpation while looking at the patient’s
face.
Liver, spleen & bimanual palpation of the kidneys.
6 year child; history of vomiting blood; scheduled for Upper
GI endoscopy
Take a focused history
 Professional etiquette
 General condition of child to assess need for resuscitation.
 Characterize symptoms: onset, volume, color (frank blood
or coffee ground), frequency, prior episodes of bleeding
 Site of bleeding: accompanying melena stool
 Etiology: preceding retching episodes (Mallory Weis tear);
PUD; chronic liver disease; bleeding disorders; drug history
(anticoagulants e.g. warfarin, NSAIDs, steroids)
Procurement of an informed consent for this
procedure
 Explain the procedure that is going to be done.
 Risks: Procedural risks (further bleeding & perforation) & anesthetic risks
 Benefits (so that they can see the procedure is worth it)
 It will identify the source of bleeding
 It can be therapeutic: allowing direct arrest of the bleeding & direct
treatment e.g. sclerotherapy or banding in variceal bleeding
 A sample can be taken
 Alternatives: Trial symptomatic treatment but further bleeding could occur
 Closure: Ask whether they have understood; Ask for any questions; ask what
they have decided & thank the patient.
Features of enlarged spleen
Firm
Beneath the left subcostal margin in LUQ
Dull to percussion
Moves downwards on inspiration
Not bimanually palpable
One “cannot get above it”
Notched in lower medial border
Causes of massive splenomegaly in children
Increased function: Chronic malaria/ TSS,
visceral leishmaniasis, Thalassemia major,
spherocytosis
Impaired blood flow: hepatic cirrhosis,
thrombosis, CCF
Infiltration: ALL, Gaucher’s disease,
Niemann – Pick disease, amyloidosis
Causes of hepatomegaly in children
 Hematological malignancies, thalassemia
 EBV, CMV, rubella
 Portal HTN
 Amyloidosis
 Toxoplasmosis
 Sarcoidosis
 Pernicious anemia
 Leishmaniasis, malaria, filariasis
 Niemann – Pick disease, Gaucher’s disease
 Acromegaly
 Reye’s syndrome
 Budd – Chiari syndrome
Image of a child with head light sign.
Child with itchy skin lesions since they were 1
year old:
Dry, scaly skin; poorly demarcated hyper –
pigmentation; Edematous; exudations (oozing
& crusting); excoriations; Lichenification
(thickened skin) suggestive of chronicity;
symmetrical
Impression: atopic dermatitis/ eczema
Cont.
Management
 Supportive:
 Trigger avoidance: elimination of allergens, heat & low humidity.
 Emollients & adequate water intake: to keep the skin hydrated
 Antihistamines or calamine lotion: Reduce pruritus
 Antibiotics covering S. aureus: to treat superinfections
 Definitive: anti – inflammatory therapy
 Topical steroids (mainstay of management)
 Calcineurin inhibitors: tacrolimus, pimecrolimus
 Phototherapy
 There is no cure but it clears off in adolescence for most patients.
Image of child with skin lesions & koplik
spots
Child in the picture above reported that skin lesions
started appearing 3 days earlier. There is a history of
fever & cough; other children have a similar condition.
Diagnosis: clinical diagnosis of measles based on 
History of fever & cough  prodrome
Maculopapular rash on the 3rd
day
Koplik spots opposite lower molar teeth
Similar symptoms amongst other children
Community level risk factors (name &
explain):
Inadequate immunization health systems in the
community  difficulties accessing services
Low socioeconomic status  leading to difficulty in
accessing preventive services
Low immunization coverage  low community
protection, i.e., low herd immunity
High undernutrition rates  leading to large numbers
of susceptible population
Complications
Secondary streptococcal infection:
Acute otitis media
Sinusitis & tonsillopharyngitis
Laryngitis & tracheobronchiolitis
Pneumonia (may be viral)
Sub – acute sclerosing panencephalitis
Aseptic meningitis
Management
Symptomatic treatment
Eye care
Antibiotics for secondary infection
Vitamin A
Prevention
Measles vaccine
Passive immunization with measles
immunoglobulin
Vitamin A supplementation
This is a picture of a 3 year old child. Presents with a rash on
her body, following a prodrome of fever and abdominal
pain.
Chicken pox: widespread, vesicular rash on the face at
different stages (pleomorphic)  papules, vesicles, pustules
& crusts on an ill looking child; impression varicella.
4 complications:
Skin & soft tissue infection
Encephalitis
Pneumonia
Otitis media
Hepatitis
Cont.
 Treatment & management
 Prevention: vaccine for contacts
 Supportive treatment:
 Hydrate & ensure good nutrition
 Antipyretic: paracetamol
 Antipruritic: calamine lotion
 Antihistamine
 Antibiotics (esp. those covering S. aureus)
 AVOID ASPIRIN which may precipitate Reye syndrome
 Definitive: acyclovir 20mg/kg/dose at 4 doses per day for 5 days (children > 12
months)
NEONATOLOGY
Neonatal resuscitation
You are called in the labor ward to
resuscitate a term baby born via SVD;
doesn’t cry when born; no meconium
staining of liquor; resuscitate baby & ask
for responses at every stage.
Refer to protocol
Examine at 1, 5 minutes
If there’s MSAF, FIRST ACTION IS TO SUCTION
THE AIRWAY.
Dry & stimulate the baby while examining for color,
tone, breathing & activity
Open airway using chin lift maneuver
Look listen feel for breathing… proceed as in protocol.
Explain plan of management to the nurse as
resuscitation reaches 5 minutes
Cover up the baby for warmth.
Give it to the mother once the baby
is stable for rooming in.
Delivery of a term neonate cause of sever fetal
bradycardia
Equipment check
Firm surface & heater (radiant warmer or resuscitairre
with a heater)
Source of Oxygen & oxygen delivery devices
2 cloths (wiper & wrapper)
Functioning ambu bag (confirm it is functioning)
Face mask of the right size (covering nose up to level
of chin & not to eyes)
Cont.
Working suction apparatus
Different size catheters
Laryngoscope and appropriate blades
Gloves
Different sizes of ET tubes & tapes
Guedel airway
A 4 day old neonate was admitted because of
yellowness of skin
Relevant questions to reach a diagnosis
GA
Onset of jaundice & How it progressed
Irritability
Fever
Abnormal cry
Cont.
Refusal to feed
Convulsions
Maternal blood group
Maternal infection: TORCHES
Previous history of a baby with jaundice
Any inherited conditions like G6PDH deficiency
Clinically observe & comment on the baby
(do not touch the baby)
Color: jaundiced?
Activity
Posture
Size of baby
Any obvious dysmorphic features
Any obvious rashes
Any obvious respiratory distress
List 5 investigations you would do for this
indication
FBC
PBF & Reticulocyte count
DCT
Blood group (of both mother)
TSB & direct bilirubin (LFTs)
How to work out investigations for NNJ
It is either indirect or direct
hyperbilirubinemia so to differentiate:
TSB & direct bilirubin
If indirect hyperbilirubinemia differentiate
between hemolytic & non – hemolytic causes
by: Reticulocyte count, PBF & DCT
A 16 hour term new born in nursery is
noted to have jaundice
What are the 10 most important questions you would
like to ask the mother:
 To do with the child: Onset & progression (don’t belabor),
Ability to BF, history of fever, lethargy, convulsions,
delayed cry.
 To do with mother: history of prolonged labor, antenatal
history of infection in mother (ask about fever, discharge),
PROM; Blood group of mother; previous baby with
jaundice; GA; Birth weight
2 day old baby is brought to casualty and reported to be
unwell. List 10 important questions you would ask.
Changes in levels of activity
Ability to BF
History of fever in baby
History of lethargy & convulsions
Prolonged labor/ delayed cry
Cont.
Antenatal history of infection in mother
History of Prolonged ROM
Place & mode of delivery
Maternal fever in labor
Gestation at delivery (preterm or term)
DIB, cyanosis, LCWI, jaundice
Given a prenatal diagnosis of extreme preterm, what are the
slight differences in immediate management of the infant?
Warmth:
Cover the baby with some cotton wool.
You can use your face mask to make the baby a
hood for the head.
Place baby in a plastic bag.
In congenital diaphragmatic hernia: Avoid BVM
& intubate immediately.
SPECIMENS
Intraosseous line insertion
10 month old infant has history of
diarrhea & vomiting & is now lethargic
on examination was found to be sickly,
CRT of 5 seconds, IV cannulation failed,
explain the process of your intervention
giving details:
Procedure
 Greet patient & parent & explain what you are going to do & why
 Collect equipment: IO or BM needle 15 – 18G or 16 – 21G
hypodermic needle
 Locate site: proximal tibia (2cm below tibial tuberosity & 1cm
medially)
 Cleanse site with betadine or surgical spirit
 Hold the leg with the non – dominant hand
 Insert needle vertically, slowly & steadily with bevel facing the toes
until there is a give
Cont.
Withdraw stylet once catheter is inside
Confirm with saline 10ml/kg
Remove samples for U/E/Cs, RBS, FBC
Stabilize IO line with a tape
Give fluids as needed while watching led/ calf muscles
for swelling.
Replace IO access with IV within 8 hrs.
Name 3 complications of the procedure
Extravasation
Osteomyelitis
Compartment syndrome
Lumbar puncture
IV CANNULATION
Examine this growth chart & discuss it under
the following tasks
Describe growth pattern.
 State birth weight
 Comment on curve e.g.
 First few months doing well
 There was no/ decreased weight gain between ___ & __ months
 Later the baby grew steadily or weight remained the same
 Overall impression: good weight gain, loss of weight, keeping the same weight
 Growth faltering: weight is still increasing but at a reduced velocity
 Identify the growth faltering, mention the age of onset & duration of faltering
 Conclusion
Preventive measures in growth faltering
1. BF: start at birth, EBF for the 1st
6 months; continue for at least 2
years
2. Complementary feeding: start at 6 months; animal proteins to
prevent PEM; 4 feeds per day then as child grows give appropriate
number off feeds per day as per child’s age.
3. Good maternal nutrition
4. Reduce impact of illness: early identification of illness & appropriate
treatment
5. Family: increase family income, increase birth spacing to reduce the
size of the family
6. Education: by health worker of the family & the comunity
Anthropometric measurements
Take MUAC of this child (review procedure)
Acromion & olecranon fossa.
Non – dominant arm in flexed position when
measuring.
What is nutritional status.
Interpret provided growth curve in 1st
year.
MCUG
COMMUNICATION SKILLS
You are called to see a child in the post – natal clinic with a TS whose
mother complains she has insufficient milk & pain around the nipples
when breastfeeding
Comment on the position of the mother & baby
The mother is bending towards baby
The baby’s ventral surface is not touching that
of the mother
The mother isn’t supporting the whole body
The baby isn’t facing the breast
The baby’s nose is not at the level of nipple.
Explain to the mother how to breastfeed
First of all, commend the mother for
breastfeeding.
Advice her that she needs to hold & latch
the baby properly to avoid pain on BF.
Technique
 Ensure mother’s comfort: The back should be supported either
using a pillow or the back of the seat.
 Proper positioning of the baby: the mother should 
 Ensure the baby is facing the nipple with the nose at the
level of the nipple (she can use a BF pillow or she can place
a foot on a BF stool to bring her thigh up)
 Ensure the baby’s tummy is touching hers.
 Support the baby’s whole body ensuring the baby’s body &
head are along a straight line.
Cont.
 Proper latching of the baby: the mother should 
 Use the C – grip & stimulate baby to open the mouth by touching
the baby’s upper lip with the nipple. Once the baby opens the mouth
wide, she should introduce the breast deep into the mouth while
pulling the baby towards her.
 Ensure more of the areola is visible above the baby’s top lip than
below the lower lip, the lower lip is curled outwards & the chin is
touching (or almost touching) the breast.
 Give the mother an opportunity to ask any questions.
 Commend the mother for seeking help about her problems.
Talk to a mother whose baby has a life threatening congenital heart
disease that is potentially treatable. Discuss the situation with the
mother
Professional etiquette
Introduce situation to mother
Ascertain that the mother is aware about the situation,
i.e., “the baby was not properly formed. We are not
sure of the cause yet but we will do our best to take
care of the baby.”
Inform her about the care & also that a team is looking
after her baby (multidisciplinary).
Cont.
Inform her that the baby will need several
investigations.
Inform her that there are good chances for cure, “With
surgical care the baby can heal completely but will
require long term follow up.”
Inform her that the condition may recur in subsequent
pregnancies & that they will need to be followed up.
Allow mother to ask questions
PMTCT
This mother comes to you at ANC having been
referred by the obstetrician after a diagnosis of
HIV. She has been counselled & informed of her
diagnosis.
Discuss the plan of feeding of her baby giving
reasons.
Discuss the follow up investigations to be done
giving timing & reasons.
Plan of feeding
 Professional etiquette (make mother sit down; you should also sit down
appropriately; have appropriate position & mannerisms)
 Confirm with the mother that she understands about her diagnosis of
HIV & how it impacts her baby. Ask her of any concerns she may want
to raise regarding the feeding of her baby
 Give options of feeding: EBF vs. RF
 She can either Exclusively Breast Feed for the first 6 months or not
BF at all (discourage her from mixed feeding).
 If she decides not to BF, advice on Replacement Feeding using the
AFASS criteria, i.e., Affordable, Feasible, Acceptable, Sustainable &
Safe.
Cont.
 Risks of BF: she may transmit the HIV to her uninfected baby.
 Risks of RF:
 Clean water is required to prepare & the infant is more likely to get sick
 Formula feeds are expensive so the supply may not be reliable
 Preparing formula feeds is labor intensive as they must be made fresh each
time
 Not BF may raise questions about HIV status resulting in stigmatization
 Remind mother about prompt onset of appropriate complementary feeding at
6 months.
 Make sure mother understands the plan; make her review it
 Give opportunity to ask questions & make her choice
Testing
If infected: start ART
Give NVP/AZT for 6 weeks; then continue NVP
for an additional 6 weeks
HIV DNA PCR for baby at birth, at 6 weeks, at 6
months, at 12 months.
HIV ELISA antibodies at 18 months.
You’ve been asked by the obstetric team to talk to a newly
diagnosed HIV+ mother in preterm labor at 24 weeks
 Explain to mother immediate & long term risks to the baby in lieu of the
above history:
 Immediate risks to baby:
 Vertical transmission of the HIV because:
 The mother is newly diagnosed & she has not been on drugs (high
viral load)
 Instrumentation during labor  increases risk of transmission
 Preterm birth: increased need for resuscitation  increases risk of
transmission
 Risk arising from prematurity  RDS, IVH, NNS, feeding difficulties.
Cont.
Long term complications:
Increased susceptibility to infection
Chronic lung disease & subsequent
respiratory insufficiency.
Growth & developmental delay.
High mortality rate both in the neonatal
period & early childhood.
Advice for subsequent pregnancies
Use & adherence to ART long before she
conceives again to maintain a low viral load.
Contraception to ensure planned pregnancies.
Once she gets pregnant, encourage on ANC
follow up esp. since she is HIV positive & since
she is at a risk of having a recurrence of the PTL.
Results of a 9 month old child
Brought in very sick
No urine output for 2 days
Height is 70 cm
Write out interpretation of result
How do you manage the most potentially lethal
electrolyte imbalance: HYPERKALEMIA
Cont.
 Specimen
 Urea  high (N: 2.5 – 6.6. mmol/L)
 Sodium  low (N: 135 – 145 mmol/L)
 Potassium  High (N  3.5 – 4.5 mmol/L)
 Chloride  (N  95 – 107 mmol/L)
 Creatinine  (N  60 – 120 µmol/L)
 Bicarbonate  low, metabolic acidosis (N  21 – 29 mmol/L)
 Schwartz formula: eGFR (mL/min/1.73m2
) =
Manage hyperkalemia
 > 6mEq/L
 Resonium calcium PO or in sorbitol PR (enema)
 Restrict K+
intake
 > 7 mEq/L
 Place on cardiac monitor
 Calcium gluconate: stabilize membrane
 Insulin/ glucose
 Salbutamol nebulization
 Sodium bicarbonate
 If refractory: dialysis
Indications of dialysis
Tumor lysis syndrome
Severe, refractory hyperkalemia
Severe intractable metabolic acidosis
Uremia (Urea > 30mmol/L with clinical signs)
Volume overload with CCF not responding to treatment.
To facilitate nutritional support & fluids (When
requiring space for nutrition: dialyze to remove fluid so
that the neonate can be fed)
ENDOCRINOLOGY
READ AROUND:
AMBIGUOUS GENITALIA
PRECOCIOUS PUBERTY
TYPED BY EFFIE NAILA
For I know the thoughts & plans that I
have for you, says the Lord, thoughts &
plans for welfare & peace & not for evil,
to give you hope in your FINAL
OUTCOME.
Jer. 29:11, AMP.

5.Pediatrics OSCEs by Effie. pptx

  • 1.
    PEDIATRICS OSCES REVISION BY:DR. NYAMBURA KARIUKI 2018 COMPILED BY EFFIE NAILA
  • 2.
    OUTLINE  GENERAL EXAM CENTRAL NERVOUS SYSTEM  CARDIOVASCULAR SYSTEM  RESPIRATORY SYSTEM  THE ABDOMEN  NEONATALOGY  SPECIMENS  Laboratory: blood, LP; Radiological: CXR, IVU, MCU, CT scan of brain; Photographs with clinical diagnoses; Intraosseous line (procedure); IV cannulation; BP checking  COMMUNICATION SKILLS: INFORMED CONSENT,  VACCINES
  • 3.
    Examine the lymphnodes of the head & neck  Check whether there is lymphadenopathy & the overlying skin (scars, erythema, rashes)  Palpate from behind the patient  Look for: From in front: submental, occipital, anterior & posterior cervical chain, posterior auricular, pre – auricular, submandibular  Do for both right & left  Accurately palpate using pulp of finger & not tips  Roll the LNs over the muscle or bones to feel them
  • 4.
  • 5.
  • 6.
  • 7.
    1. Child witha history of weakness Do a motor exam of the lower limbs Professional etiquette: greet the patient, introduce yourself & explain to them what you want to do & obtain consent. Stand on the right side of the patient & expose them adequately.
  • 8.
    Cont. Motor examination:  Inspect& comment on: posture & muscle bulk (visible wasting), fasciculations, skin changes, wounds or scars  Palpate the muscle bulk of all muscle groups.  Assess power across ALL MUSCLE GROUPS & grade.  Assess tone across ALL THE JOINTS. Comment: hypertonia or hypotonia & impression.  Assess reflexes (knee jerk, ankle reflex, ankle clonus, plantar reflex/ Babinski response) Comment: hyperreflexia or hyporeflexia & impression.
  • 9.
    Motor examination ofthe upper limbs Professional etiquette. Stand on the right side of the patient. Inspection: posture of upper limbs, visible wasting (comment appropriately) Palpate: muscle bulk Tone: across all joints Power in all muscle groups Reflexes: brachioradialis (supinator), biceps, triceps
  • 10.
    Name 3 contraindicationsof a lumbar puncture Increased ICP Coagulopathy Soft tissue infection at site of LP Cardiopulmonary instability
  • 11.
    2. Interpret thefollowing LP findings in a patient with a chronic headache Protein in g/L: 0.5 Glucose in mmol/L: 1.6 CSF: Serum ratio is <0.4, reduced (N: CSF: serum glucose ratio is 0.6) WBCs: 180 Appearance: fibrin webbed Differentials: TB meningitis, fungal meningitis
  • 12.
    What is thetreatment plan Working diagnosis: Tuberculous meningitis 2RHZE + 10RH Steroid therapy
  • 13.
    Assess cranial nerveII Professional etiquette Stand on right side of bed, sanitize & warm hands Visual acuity: Snellen’s chart Distant vision by standing 1m away from child Color perception: Ishihara charts Visual field assessment Pupillary reflexes: direct & consensual
  • 14.
    Assess cranial nervesIII, IV & VI  Professional etiquette  Stand on right side of bed, sanitize & warm hands  Inspect for ptosis, eye ball symmetry (squint, abnormal movements) pupil size at rest.  Accommodation reflex  Eye movements: hold finger at 30cm & with the head still move the finger through the various axes of eye movements (H – shape). Observe for any restrictions in eye movement.  Ask for double vision
  • 15.
    3. The followingchild presented at pediatric casualty after falling from a motor cycle. Examine cranial nerves V & VII Trigeminal nerve (V1, V2 & V3)  Examine for light touch, pain & temperature in all 3 nerve distributions: forehead, cheek & jaw  Inspect & palpate the masseter & temporalis muscle  Test for power of the masseter muscle: oppose jaw opening  Test right & left pterygoids muscle power: oppose lateral jaw movement  Corneal reflex
  • 16.
    Cont. Facial nerve (VII) Frontalis: ask patient to raise eyebrows  Orbicularis oculi: ask patient to tightly close eyes then you try & open them  Ask patient to smile & frown  Ask patient to puff the cheeks & try to deflate them by taping on them gently bilaterally  Test taste on the anterior of the tongue
  • 17.
    Child presents withunexplained lower limb (LL) weakness. Examine the deep tendon reflexes (DTR) on the LLs.  Professional etiquette.  Examine both the right & left DTRs.  Knee reflexes: slightly lift the leg under the knee or perform the test it in a sitting position with the leg dangling. Identify & tap the patella & check the CONTRACTION OF THE QUADRICEPS.  Remember to perform the Jendrassik’s maneuver  Ankle reflexes: slightly extend the hip & dorsiflex the foot
  • 18.
    Assume this childwas alert, normal upper limbs, weakness of LL & increased DTR, what are your differentials? Tumor (SOL) Schistosomiasis TB spine
  • 19.
    Child admitted witha 2 day history of LOC, fever & an episode of convulsion lasting 3 minutes Assess the LOC of this child & interpret your findings. Do a pediatric GCS scoring. Give the scores for E, V & M (correctly report the findings & interpret them) Comment on the final GCS
  • 20.
    Based on thegiven history give the most likely diagnosis (ALWAYS GIVE A JUSTIFICATION) Complicated meningitis: convulsions, fever, LOC & duration The LOC isn’t convulsion – induced hence it is not a febrile seizure but a more ominous condition. Differentials CVA Meningoencephalitis Cerebral malaria
  • 21.
    What is thedifference between an UMN & LMN facial nerve palsy UMN: Spares the brow muscles Spares eye closing
  • 22.
    This child hasundergone removal of a craniopharyngioma/ Examine cranial nerves II, III, IV & VI Optic nerve Distant vision using Snellen’s chart (Distance from child  1 m away) Color sense using Ishihara chart Visual field assessment How far from you: ____ Where to place finger: ____ Direct & consensual pupillary reflex
  • 23.
    Cont. Oculomotor nerve  Upwardrotation & inward movement of eye ball  Size of pupil  Assess for accommodation Trochlear  SO4  Downward movement of eyeball Abduscens  Lateral movement of eyeball
  • 24.
    Differentials of achild with flaccid paralysis: Polio Transverse myelitis GBS Atonic cerebral palsy
  • 25.
    Investigations for thechild with flaccid paralysis MRI/CT scan spine: to R/O any structural problem in the motor area of spinal cord. Nerve conduction studies to determine peripheral neuropathy. Stool for polio virus.
  • 26.
    UMNL vs. LMNL UMNL •Hypertonia(spasticity vs. rigidity) •Fasciculations absent •Hyperreflexia •Normal bulk then disuse atrophy sets in with time •Reduced power (tends towards normal over time if adequate stimulation is maintained) •Plantar extensor response (Babinski’s sign) •Clonus may be present •Spastic paralysis LMNL •Hypotonia •Fasciculations present •Hypo/ areflexia •Atrophy occurs early •Power is severely decreased •Plantar flexion response •Clonus may be absent •Flaccid paralysis
  • 27.
  • 28.
    Examine the precordiumof this child while narrating your findings  Professional etiquette.  Stand on the right side of the patient.  Commence P/E.  Expose child adequately  Inspection (accurately report): hyperactive  Palpation: apex beat, left parasternal heave (LPS)  Auscultation: apex, tricuspid area, pulmonary area (+ S2) & aortic area.
  • 30.
    Examine the neckof this patient for Jugular Venous Pulse Requirements: good lighting, patient reclined at 450 Assess the waveform of the internal jugular vein lying adjacent to the medial border of the SCM. Look for pulsation on the right side of the neck Press on RUQ while watching neck to differentiate between carotid artery & JVP pulsation Identify the highest point of pulsation of the JVP
  • 31.
    Cont.  Extend rulerhorizontally from the highest pulsation point  This ruler must cross with a ruler placed at the sternal angle of Louis at 900  Report the measurement in cm  Add 4cm to get the distance to the right atrium  Report the JVP  Normal is 9 cm from the RA which corresponds to a pressure of mmHg.
  • 32.
    Features that distinguisharterial & venous pulses Carotid arterial pulses • Pulsation per heart beat: single • Palpable. • Pressure at root of neck: Unaffected. • Pulsation height: • Unaffected by the hepatojugular reflux (apply manual pressure over the RUQ) • Independent on the posture of the patient. • Not dependent on respiration. Jugular venous pulses • Pulsation per heat beat: double waveform in sinus rhythm • Not palpable. • Pressure at root of neck: obliterated • Pulsation height: • Transient in volume & height by the hepatojugular reflux. • Varies with posture of the patient. • Decreased on inspiration (EXCEPT IN CONSTRICTIVE PERICARDITIS; Kussmaul sign)
  • 33.
    Possible causes ofa raised JVP  CCF  Cor pulmonale  Constrictive pericarditis  Pulmonary embolism  Cardiac tamponade  Tricuspid valvular disease  Right ventricular infarction  HCM  SVC obstruction (SVCO)  Iatrogenic fluid overload esp. in surgical & renal patients
  • 34.
    Measure the BP(sphygmomanometer)  Professional etiquette.  Ensure the child is sitting or lying at ease & not anxious.  The manometer should be at the same level of the cuff on the patient’s arm & the observer’s eye.  Identify a cuff that covers most of the upper arm leaving a gap of 1 cm or so below the axilla & above the antecubital fossa.  Measure the upper arm circumference using a tape measure & identify the right cuff.
  • 35.
    Cont.  Palpate theradial pulse as the cuff is inflated to a pressure of 20mmHg above the level at which radial pulsation can no longer be felt.  Place stethoscope lightly over brachial artery & deflate cuff at a rate of 2 – 3 mmHg/s until the first Korotkoff sound is heard. This correlates with the SBP.  With further deflation subtle changes occur in the pitch of the sounds. These correspond to the 2nd , 3rd & 4th (muffling) Korotkoff sounds that are clinically insignificant.  When the sounds die away completely (i.e. the 5th Korotkoff sound), this correlates with the DBP.
  • 36.
    Investigations in achild with HTN 4 limb BP for vascular disease U/E/Cs: abnormal kidney function can cause HTN Potassium levels: K+ levels in Conn’s syndrome (hyperaldosteronism) Serum renin levels Doppler U/S of kidneys: renal artery disease 24hr urinary VMA for Phaeochromocytoma OVERALL ASSESSMENT
  • 37.
    What are thecauses of HTN in children
  • 38.
    Management of CCF Propup the patient. Administer oxygen if in distress. Medical: For preload: diuretics For cardiac contractility: digoxin For afterload: CCBs Treat underlying problem
  • 39.
    Examine the pulses& narrate your findings  Professional etiquette  Stand on the right side of patient  Warm hands  Radial pulse rate for a full minute  Describe rate, rhythm, volume, character, symmetry & radio – femoral delay (coarctation of aorta is a diagnosis made at birth!), collapsing pulse.  Brachial pulse  Carotid pulse (DONE UNILATERALLY)  Popliteal pulse: Gently flex the knee and feel for the popliteal pulse by deep palpation in midline in popliteal fossa.  Posterior tibial artery: behind & slightly below the medial malleolus of ankle  Dorsalis pedis pulse: lateral to extensor tendon of great toe
  • 41.
    Name 3 clinicalconditions with a collapsing pulse Aortic regurgitation Hyper – dynamic circulation: thyrotoxicosis, beriberi PDA Large atrio – ventricular defect
  • 42.
    15 month oldchild brought to casualty poor feeding, poor weight gain & recurrent infections. BF was stopped at 6 months & supplemented with cow’s milk Lab test: RBCs 3.9 low (N: 4 – 6 X 1012 /L) WBCs: 13.6 high (N: 4 – 11 X 109 /L) Hb: 8  low (N: > 12g/dl) MCV:65  low (N: 78 – 98fL) MCH: 19 (N: 27 – 32 pg) MCHC: 22 Platelets: 203 (N: 150 – 350 X 109 /L) Diagnosis: hypochromic microcytic anemia
  • 43.
    Cont. Most important differential:IDA Others: Anemia of chronic disease; sideroblastic anemia; lead poisoning; thalassemia Underlying cause: Helminthic infections Diet
  • 44.
    Cont. Treatment: Elemental iron6mg/kg/day for 4 – 6 months To replenish stores Stages of iron deficiency: 1. Reticuloendothelial stores are depleted. 2a. MCV, MCH. 2b: Hb. 3. Clinical manifestations
  • 45.
  • 46.
    This child presentswith fever, cough & DIB for 3 days. Examine the respiratory system by inspection, percussion & auscultation.  Inspection:  Cyanosis (central & peripheral), pallor, head nodding, FAN, finger clubbing; listen for audible sounds (grunting, stridor, wheeze); Use of accessory muscles; Observe for supraclavicular, suprasternal & intercostal in – drawing; LCWI; chest wall symmetry; look for abdominal breathing  Count RR for 1 minute  Percussion: All areas & report findings correctly  Auscultation: Listen to breath sounds & check for vocal fremitus
  • 47.
    Comment on thefollowing findings in a tachypneic child:  pH: 7.04 (7.35 – 7.45)  low; acidosis  PO2: 80mmHg (60 – 80mmHg)  normal; not respiratory acidosis  PCO2: 6mmHg (40 – 60mmHg)  low; respiratory compensation  HCO3 - : 6mmol/L (22 – 24 mmol/L)  low; metabolic acidosis  Na+ : 140  normal  Cl- : 108  normal  Calculated anion gap: 26 (12 – 16 mmol/L)  elevated
  • 48.
    Cont. Conclusion: metabolic acidosis withrespiratory compensation Add: you must do anion gap {(Na+ + K+ ) - (Cl- + HCO3)} & comment on it:
  • 49.
    Common causes ofmetabolic acidosis with a high anion gap Poisoning DKA lactic acidosis Uremia Pancreatitis
  • 50.
    Causes of normalanion gap acidosis Renal tubular acidosis
  • 51.
    Causes of hypochloremic,hypokalemic metabolic alkalosis Pyloric stenosis Bicarbonate ingestion Steroids Frusemide Baters disease Adrenal pathology
  • 52.
    The following isa radiological specimen of a 2 year old child with fever, cough & progressive DIB  Name the radiological specimen completely e.g. CHEST X RAY as opposed to X ray; remember to comment on the view.  Quality: Rotation, Inspiration, Penetration  Skeletal thoracic cage  Lung findings  Cardiac silhouette  Heart size: CTR  Diaphragm
  • 53.
    Treatment plan forabove child (write on Treatment sheet) Classify: e.g. severe etc. Oxygen administration (method plus flow rates) Penicillin & gentamicin: dosages & duration are MUST KNOWS. Feeding & fluids
  • 54.
    This evaluation wasperformed on a refugee camp on a patient with persistent cough & fever Write out a report of this specimen State that it is a CHEST X RAY Quality Skeletal cage Lung fields: multiple, widespread opacities Heart size Diaphragm Diagnosis: Miliary TB
  • 55.
    Treatment of TB Intensivephase  2 months; RHZE/ RHZS Continuation phase  4 months: RH Nutrition Any other infection Do PITC
  • 56.
    Child with fever,cough & DIB for 3 days associated with poor feeding Examine the respiratory system through inspection & relevant GE for purposes of determining the severity of the disease Outline: Diagnosis Classification Definitive treatment: O2 therapy, penicillin & gentamicin (KNOW DOSAGES)
  • 57.
    Previously well childwith cough & fever for 1 day, DIB General exam relevant to respiratory system: include lymphadenopathy Highest priority investigations: CXR, HIV test, BGAs HIV test because of recurrent LRTI
  • 58.
    Pneumothorax CXR: collapsed lung,mediastinal shift, gas in pleural cavity Management: Chest tube insertion Underlying pathology Taking care of nutrition
  • 59.
    Interpret the radiologicalimage provided & asses as follows  Name the specimen as Chest X – Ray & state the view. Evaluate lung fields & airway & give findings  Interpretation:  Verify the biodata  Quality: R (clavicle or anterior rib length); I (count the ribs); Penetration.  Look at right lung; left lung  Pleural borders  Costophrenic angles  Cardiophrenic angles  Hilar regions  Tracheal position  State diagnosis & differentials
  • 61.
    This child isbeing managed for ascites. Examine for ascites.  Professional etiquette + warming your hands  Expose adequately  Inspection:  Stand at the foot of the bed to accurately assess abdominal fullness, symmetry & hernia orifices  On right side of patient: Accurate assessment of umbilical stump, look for scars, superficial venous distention & MWR (movement with respiration)  Palpation: first ask whether the child is in pain while warming your hands before touching the patient then remember to look at the face of the patient)
  • 63.
    Differentials  Exudative  Abdominalmalignancy  TB peritonitis  Transudative  High hydrostatic pressure:  Chronic liver disease  Portal HTN  Low oncotic pressure  Renal disease: nephrotic syndrome  Protein losing enteropathy
  • 64.
    This child presentswith abdominal distention for the 1st 3 months. Active and playful but poor feeding habits Inspect & palpate abdomen 2 most important differentials based on imaging modality provided (CXR with fecal loading & hepatomegaly) Constipation & hepatomegaly
  • 65.
    Cont.  Bedside manner Ensure comfort is adequate + appropriate exposure  Inspect the abdomen & give findings: shape, umbilicus, MWR, skin & surface of abdomen, inguinal area & external genitalia  Palpation: before starting ask for any abdominal tenderness; look at the face of the patient (light & deep) various quadrants & be systematic; feel for left kidney, spleen, right kidney, liver, urinary bladder, groins.  Percussion: shifting dullness & fluid thrill  Auscultate for bowel movements & vascular bruits  Anal & rectal examination
  • 67.
    Abdominal pain This 6year old boy is brought to the casualty with abdominal pain for the past month. He passes stool once a week & his stool looks like hard pellets. Take additional relevant history from the guardian to help you reach a diagnosis (constipation, food allergy esp. cow milk food allergy).
  • 68.
    Relevant history Any painwhile passing stool (dyschezia) Blood on the surface of hard stool. Has the child attained toilet training. Does the child soil his pants lately (encopresis) Associated symptoms: abdominal swelling, vomiting Etiology: diet, water intake, allergy to cow’s milk & dairy, similar history amongst family members, psychosocial factors
  • 69.
    How would youmanage this condition Reassure parents & advice them to stay calm Address any psychosocial issues Toilet training (regular sitting on the toilet for long enough; steps to ensure proper seating) Encourage physical activity Encourage fluid intake Diet: fiber rich fruits & vegetables Medical: laxatives, peglec
  • 70.
    Palpate the abdomenof this child. Professional etiquette Stand on right side Expose adequately & appropriately Ask for abdominal tenderness Light & deep palpation while looking at the patient’s face. Liver, spleen & bimanual palpation of the kidneys.
  • 71.
    6 year child;history of vomiting blood; scheduled for Upper GI endoscopy Take a focused history  Professional etiquette  General condition of child to assess need for resuscitation.  Characterize symptoms: onset, volume, color (frank blood or coffee ground), frequency, prior episodes of bleeding  Site of bleeding: accompanying melena stool  Etiology: preceding retching episodes (Mallory Weis tear); PUD; chronic liver disease; bleeding disorders; drug history (anticoagulants e.g. warfarin, NSAIDs, steroids)
  • 72.
    Procurement of aninformed consent for this procedure  Explain the procedure that is going to be done.  Risks: Procedural risks (further bleeding & perforation) & anesthetic risks  Benefits (so that they can see the procedure is worth it)  It will identify the source of bleeding  It can be therapeutic: allowing direct arrest of the bleeding & direct treatment e.g. sclerotherapy or banding in variceal bleeding  A sample can be taken  Alternatives: Trial symptomatic treatment but further bleeding could occur  Closure: Ask whether they have understood; Ask for any questions; ask what they have decided & thank the patient.
  • 73.
    Features of enlargedspleen Firm Beneath the left subcostal margin in LUQ Dull to percussion Moves downwards on inspiration Not bimanually palpable One “cannot get above it” Notched in lower medial border
  • 74.
    Causes of massivesplenomegaly in children Increased function: Chronic malaria/ TSS, visceral leishmaniasis, Thalassemia major, spherocytosis Impaired blood flow: hepatic cirrhosis, thrombosis, CCF Infiltration: ALL, Gaucher’s disease, Niemann – Pick disease, amyloidosis
  • 75.
    Causes of hepatomegalyin children  Hematological malignancies, thalassemia  EBV, CMV, rubella  Portal HTN  Amyloidosis  Toxoplasmosis  Sarcoidosis  Pernicious anemia  Leishmaniasis, malaria, filariasis  Niemann – Pick disease, Gaucher’s disease  Acromegaly  Reye’s syndrome  Budd – Chiari syndrome
  • 76.
    Image of achild with head light sign. Child with itchy skin lesions since they were 1 year old: Dry, scaly skin; poorly demarcated hyper – pigmentation; Edematous; exudations (oozing & crusting); excoriations; Lichenification (thickened skin) suggestive of chronicity; symmetrical Impression: atopic dermatitis/ eczema
  • 78.
    Cont. Management  Supportive:  Triggeravoidance: elimination of allergens, heat & low humidity.  Emollients & adequate water intake: to keep the skin hydrated  Antihistamines or calamine lotion: Reduce pruritus  Antibiotics covering S. aureus: to treat superinfections  Definitive: anti – inflammatory therapy  Topical steroids (mainstay of management)  Calcineurin inhibitors: tacrolimus, pimecrolimus  Phototherapy  There is no cure but it clears off in adolescence for most patients.
  • 79.
    Image of childwith skin lesions & koplik spots Child in the picture above reported that skin lesions started appearing 3 days earlier. There is a history of fever & cough; other children have a similar condition. Diagnosis: clinical diagnosis of measles based on  History of fever & cough  prodrome Maculopapular rash on the 3rd day Koplik spots opposite lower molar teeth Similar symptoms amongst other children
  • 80.
    Community level riskfactors (name & explain): Inadequate immunization health systems in the community  difficulties accessing services Low socioeconomic status  leading to difficulty in accessing preventive services Low immunization coverage  low community protection, i.e., low herd immunity High undernutrition rates  leading to large numbers of susceptible population
  • 81.
    Complications Secondary streptococcal infection: Acuteotitis media Sinusitis & tonsillopharyngitis Laryngitis & tracheobronchiolitis Pneumonia (may be viral) Sub – acute sclerosing panencephalitis Aseptic meningitis
  • 82.
  • 83.
    Prevention Measles vaccine Passive immunizationwith measles immunoglobulin Vitamin A supplementation
  • 84.
    This is apicture of a 3 year old child. Presents with a rash on her body, following a prodrome of fever and abdominal pain. Chicken pox: widespread, vesicular rash on the face at different stages (pleomorphic)  papules, vesicles, pustules & crusts on an ill looking child; impression varicella. 4 complications: Skin & soft tissue infection Encephalitis Pneumonia Otitis media Hepatitis
  • 85.
    Cont.  Treatment &management  Prevention: vaccine for contacts  Supportive treatment:  Hydrate & ensure good nutrition  Antipyretic: paracetamol  Antipruritic: calamine lotion  Antihistamine  Antibiotics (esp. those covering S. aureus)  AVOID ASPIRIN which may precipitate Reye syndrome  Definitive: acyclovir 20mg/kg/dose at 4 doses per day for 5 days (children > 12 months)
  • 86.
  • 87.
    Neonatal resuscitation You arecalled in the labor ward to resuscitate a term baby born via SVD; doesn’t cry when born; no meconium staining of liquor; resuscitate baby & ask for responses at every stage.
  • 88.
    Refer to protocol Examineat 1, 5 minutes If there’s MSAF, FIRST ACTION IS TO SUCTION THE AIRWAY. Dry & stimulate the baby while examining for color, tone, breathing & activity Open airway using chin lift maneuver Look listen feel for breathing… proceed as in protocol.
  • 91.
    Explain plan ofmanagement to the nurse as resuscitation reaches 5 minutes Cover up the baby for warmth. Give it to the mother once the baby is stable for rooming in.
  • 92.
    Delivery of aterm neonate cause of sever fetal bradycardia Equipment check Firm surface & heater (radiant warmer or resuscitairre with a heater) Source of Oxygen & oxygen delivery devices 2 cloths (wiper & wrapper) Functioning ambu bag (confirm it is functioning) Face mask of the right size (covering nose up to level of chin & not to eyes)
  • 93.
    Cont. Working suction apparatus Differentsize catheters Laryngoscope and appropriate blades Gloves Different sizes of ET tubes & tapes Guedel airway
  • 94.
    A 4 dayold neonate was admitted because of yellowness of skin Relevant questions to reach a diagnosis GA Onset of jaundice & How it progressed Irritability Fever Abnormal cry
  • 95.
    Cont. Refusal to feed Convulsions Maternalblood group Maternal infection: TORCHES Previous history of a baby with jaundice Any inherited conditions like G6PDH deficiency
  • 96.
    Clinically observe &comment on the baby (do not touch the baby) Color: jaundiced? Activity Posture Size of baby Any obvious dysmorphic features Any obvious rashes Any obvious respiratory distress
  • 97.
    List 5 investigationsyou would do for this indication FBC PBF & Reticulocyte count DCT Blood group (of both mother) TSB & direct bilirubin (LFTs)
  • 98.
    How to workout investigations for NNJ It is either indirect or direct hyperbilirubinemia so to differentiate: TSB & direct bilirubin If indirect hyperbilirubinemia differentiate between hemolytic & non – hemolytic causes by: Reticulocyte count, PBF & DCT
  • 99.
    A 16 hourterm new born in nursery is noted to have jaundice What are the 10 most important questions you would like to ask the mother:  To do with the child: Onset & progression (don’t belabor), Ability to BF, history of fever, lethargy, convulsions, delayed cry.  To do with mother: history of prolonged labor, antenatal history of infection in mother (ask about fever, discharge), PROM; Blood group of mother; previous baby with jaundice; GA; Birth weight
  • 100.
    2 day oldbaby is brought to casualty and reported to be unwell. List 10 important questions you would ask. Changes in levels of activity Ability to BF History of fever in baby History of lethargy & convulsions Prolonged labor/ delayed cry
  • 101.
    Cont. Antenatal history ofinfection in mother History of Prolonged ROM Place & mode of delivery Maternal fever in labor Gestation at delivery (preterm or term) DIB, cyanosis, LCWI, jaundice
  • 102.
    Given a prenataldiagnosis of extreme preterm, what are the slight differences in immediate management of the infant? Warmth: Cover the baby with some cotton wool. You can use your face mask to make the baby a hood for the head. Place baby in a plastic bag. In congenital diaphragmatic hernia: Avoid BVM & intubate immediately.
  • 103.
  • 104.
    Intraosseous line insertion 10month old infant has history of diarrhea & vomiting & is now lethargic on examination was found to be sickly, CRT of 5 seconds, IV cannulation failed, explain the process of your intervention giving details:
  • 105.
    Procedure  Greet patient& parent & explain what you are going to do & why  Collect equipment: IO or BM needle 15 – 18G or 16 – 21G hypodermic needle  Locate site: proximal tibia (2cm below tibial tuberosity & 1cm medially)  Cleanse site with betadine or surgical spirit  Hold the leg with the non – dominant hand  Insert needle vertically, slowly & steadily with bevel facing the toes until there is a give
  • 106.
    Cont. Withdraw stylet oncecatheter is inside Confirm with saline 10ml/kg Remove samples for U/E/Cs, RBS, FBC Stabilize IO line with a tape Give fluids as needed while watching led/ calf muscles for swelling. Replace IO access with IV within 8 hrs.
  • 108.
    Name 3 complicationsof the procedure Extravasation Osteomyelitis Compartment syndrome
  • 109.
  • 110.
  • 111.
    Examine this growthchart & discuss it under the following tasks Describe growth pattern.  State birth weight  Comment on curve e.g.  First few months doing well  There was no/ decreased weight gain between ___ & __ months  Later the baby grew steadily or weight remained the same  Overall impression: good weight gain, loss of weight, keeping the same weight  Growth faltering: weight is still increasing but at a reduced velocity  Identify the growth faltering, mention the age of onset & duration of faltering  Conclusion
  • 112.
    Preventive measures ingrowth faltering 1. BF: start at birth, EBF for the 1st 6 months; continue for at least 2 years 2. Complementary feeding: start at 6 months; animal proteins to prevent PEM; 4 feeds per day then as child grows give appropriate number off feeds per day as per child’s age. 3. Good maternal nutrition 4. Reduce impact of illness: early identification of illness & appropriate treatment 5. Family: increase family income, increase birth spacing to reduce the size of the family 6. Education: by health worker of the family & the comunity
  • 114.
    Anthropometric measurements Take MUACof this child (review procedure) Acromion & olecranon fossa. Non – dominant arm in flexed position when measuring. What is nutritional status. Interpret provided growth curve in 1st year.
  • 115.
  • 116.
  • 117.
    You are calledto see a child in the post – natal clinic with a TS whose mother complains she has insufficient milk & pain around the nipples when breastfeeding Comment on the position of the mother & baby The mother is bending towards baby The baby’s ventral surface is not touching that of the mother The mother isn’t supporting the whole body The baby isn’t facing the breast The baby’s nose is not at the level of nipple.
  • 118.
    Explain to themother how to breastfeed First of all, commend the mother for breastfeeding. Advice her that she needs to hold & latch the baby properly to avoid pain on BF.
  • 119.
    Technique  Ensure mother’scomfort: The back should be supported either using a pillow or the back of the seat.  Proper positioning of the baby: the mother should   Ensure the baby is facing the nipple with the nose at the level of the nipple (she can use a BF pillow or she can place a foot on a BF stool to bring her thigh up)  Ensure the baby’s tummy is touching hers.  Support the baby’s whole body ensuring the baby’s body & head are along a straight line.
  • 120.
    Cont.  Proper latchingof the baby: the mother should   Use the C – grip & stimulate baby to open the mouth by touching the baby’s upper lip with the nipple. Once the baby opens the mouth wide, she should introduce the breast deep into the mouth while pulling the baby towards her.  Ensure more of the areola is visible above the baby’s top lip than below the lower lip, the lower lip is curled outwards & the chin is touching (or almost touching) the breast.  Give the mother an opportunity to ask any questions.  Commend the mother for seeking help about her problems.
  • 121.
    Talk to amother whose baby has a life threatening congenital heart disease that is potentially treatable. Discuss the situation with the mother Professional etiquette Introduce situation to mother Ascertain that the mother is aware about the situation, i.e., “the baby was not properly formed. We are not sure of the cause yet but we will do our best to take care of the baby.” Inform her about the care & also that a team is looking after her baby (multidisciplinary).
  • 122.
    Cont. Inform her thatthe baby will need several investigations. Inform her that there are good chances for cure, “With surgical care the baby can heal completely but will require long term follow up.” Inform her that the condition may recur in subsequent pregnancies & that they will need to be followed up. Allow mother to ask questions
  • 124.
    PMTCT This mother comesto you at ANC having been referred by the obstetrician after a diagnosis of HIV. She has been counselled & informed of her diagnosis. Discuss the plan of feeding of her baby giving reasons. Discuss the follow up investigations to be done giving timing & reasons.
  • 125.
    Plan of feeding Professional etiquette (make mother sit down; you should also sit down appropriately; have appropriate position & mannerisms)  Confirm with the mother that she understands about her diagnosis of HIV & how it impacts her baby. Ask her of any concerns she may want to raise regarding the feeding of her baby  Give options of feeding: EBF vs. RF  She can either Exclusively Breast Feed for the first 6 months or not BF at all (discourage her from mixed feeding).  If she decides not to BF, advice on Replacement Feeding using the AFASS criteria, i.e., Affordable, Feasible, Acceptable, Sustainable & Safe.
  • 126.
    Cont.  Risks ofBF: she may transmit the HIV to her uninfected baby.  Risks of RF:  Clean water is required to prepare & the infant is more likely to get sick  Formula feeds are expensive so the supply may not be reliable  Preparing formula feeds is labor intensive as they must be made fresh each time  Not BF may raise questions about HIV status resulting in stigmatization  Remind mother about prompt onset of appropriate complementary feeding at 6 months.  Make sure mother understands the plan; make her review it  Give opportunity to ask questions & make her choice
  • 127.
    Testing If infected: startART Give NVP/AZT for 6 weeks; then continue NVP for an additional 6 weeks HIV DNA PCR for baby at birth, at 6 weeks, at 6 months, at 12 months. HIV ELISA antibodies at 18 months.
  • 129.
    You’ve been askedby the obstetric team to talk to a newly diagnosed HIV+ mother in preterm labor at 24 weeks  Explain to mother immediate & long term risks to the baby in lieu of the above history:  Immediate risks to baby:  Vertical transmission of the HIV because:  The mother is newly diagnosed & she has not been on drugs (high viral load)  Instrumentation during labor  increases risk of transmission  Preterm birth: increased need for resuscitation  increases risk of transmission  Risk arising from prematurity  RDS, IVH, NNS, feeding difficulties.
  • 130.
    Cont. Long term complications: Increasedsusceptibility to infection Chronic lung disease & subsequent respiratory insufficiency. Growth & developmental delay. High mortality rate both in the neonatal period & early childhood.
  • 131.
    Advice for subsequentpregnancies Use & adherence to ART long before she conceives again to maintain a low viral load. Contraception to ensure planned pregnancies. Once she gets pregnant, encourage on ANC follow up esp. since she is HIV positive & since she is at a risk of having a recurrence of the PTL.
  • 132.
    Results of a9 month old child Brought in very sick No urine output for 2 days Height is 70 cm Write out interpretation of result How do you manage the most potentially lethal electrolyte imbalance: HYPERKALEMIA
  • 133.
    Cont.  Specimen  Urea high (N: 2.5 – 6.6. mmol/L)  Sodium  low (N: 135 – 145 mmol/L)  Potassium  High (N  3.5 – 4.5 mmol/L)  Chloride  (N  95 – 107 mmol/L)  Creatinine  (N  60 – 120 µmol/L)  Bicarbonate  low, metabolic acidosis (N  21 – 29 mmol/L)  Schwartz formula: eGFR (mL/min/1.73m2 ) =
  • 134.
    Manage hyperkalemia  >6mEq/L  Resonium calcium PO or in sorbitol PR (enema)  Restrict K+ intake  > 7 mEq/L  Place on cardiac monitor  Calcium gluconate: stabilize membrane  Insulin/ glucose  Salbutamol nebulization  Sodium bicarbonate  If refractory: dialysis
  • 135.
    Indications of dialysis Tumorlysis syndrome Severe, refractory hyperkalemia Severe intractable metabolic acidosis Uremia (Urea > 30mmol/L with clinical signs) Volume overload with CCF not responding to treatment. To facilitate nutritional support & fluids (When requiring space for nutrition: dialyze to remove fluid so that the neonate can be fed)
  • 136.
  • 137.
    TYPED BY EFFIENAILA For I know the thoughts & plans that I have for you, says the Lord, thoughts & plans for welfare & peace & not for evil, to give you hope in your FINAL OUTCOME. Jer. 29:11, AMP.

Editor's Notes

  • #11 Normal ranges CSF protein: 0.15 – 0.45 g/L (0.2 – 1.7g/L in premature & term neonates due to a highly permeable BBB) CSF: serum glucose ratio  >0.5 Typical laboratory findings in bacterial meningitis include: CSF WBC count above 1000 /uL , usually with a neutrophilia. A CSF protein concentration above 2.5g/L. A CSF glucose concentrations below 2.5 mmol/L. Typical laboratory findings in viral meningitis: A CSF WBC count > 250/uL and almost always < than 2000/uL. The differential shows a lymphocytosis CSF proteins concentration is typically < 1.5 g/L. CSF protein concentration > 2.2.g/L reduces probability of viral infection to 1% or less CSF glucose will typically be normal (>50% of serum concentration) in viral CNS infections
  • #30 JVP is due to fluctuations in right atrial pressure during the cardiac cycle. Kussmaul’s sign: this is a paradoxical increase in JVP with inspiration due to the inspiratory increase in venous return that cannot be accommodated in a tamponaded right atrium. It is seen in constrictive pericarditis, cardiac tamponade
  • #34 Also examine pulses
  • #46 If it is just inspection of respiratory It could be normal. Most likely diagnosis & differentials giving reasons Depend on the patient
  • #47 Causes of normal anion gap acidosis - Renal tubular acidosis  hyperchloremia
  • #58 CXR Pneumonia Miliary TB Pleural effusion Pneumothorax Lung consolidation
  • #67 Don’t eat: chips & mandazis 2 or 3 satchets of peglec 1L at once 500mL per hour
  • #76 General principles: elimination of allergens, maintenance of skin hydration, control pruritus, anti – inflammatory therapy with steroids, manage secondary infection
  • #125 Acceptable: Replacement feeding for breast milk is acceptable by the mother, the family and others who are close to the family. Feasible: The mother has access to clean and safe water for cleaning the feeding bottles, teats, measuring cup and spoon, and diluting the formula milk if it comes as a powder. Affordable: The family can afford to buy enough formula milk or animal milk to feed the baby adequately. Sustainable: The mother is able to prepare feeds for the child as frequently as recommended and as the baby demands. Safe: The formula milk should be safe and nutritious for the health of the baby.
  • #127 RNA PCR is viral load