8. History
Chest pain
Fatigue (end of the day)
dyspnea
Orthopnea & PND
Palpitations
Syncope
Body swelling
9. History cont’
Other illnesses :
Thyroid, connective tissue, neoplastic disorders, Rheumatic fever in
childhood , HTN, DM & dyslipidemia, Smoking, Alcohol (arrhythmias,
CMP)
Family Hx - CAD, HTN, HCMP
Family Hx of sudden death - single most important indicator of risk in
HCMP
Drug Hx :
β-Blockers & some CCB(diltiazem, verapamil), bradycardias,TCA & β-
agoniststachyarrhythmias, Vasodilators hypotension syncopal
attack ,Doxorubicin & related compounds toxic cardiomyopathy
10. History
CHEST PAIN
cardiac origin -Myocardial ischemia, pericarditis, aortic dissection
Myocardial ischemia(angina)
Retrosternal pain which may radiate into the arms, the throat or the jaw
Has a constricting/squeezing character, is provoked by exertion & relieved
rapidly by rest or nitroglycerin
11. History cont’
Pericarditis
Central chest pain, sharp in character & aggravated by deep inspiration,
cough or postural changes, radiates to trapezius muscle
Aortic dissection
Severe tearing pain in either the front or the back of the chest, abrupt
onset
12. History cont’
SYNCOPE
Transient, self-limited loss of consciousness due to acute global
impairment of blood flow to the brain.
Rapid onset , brief duration, & recovery spontaneous & complete
DDX - seizures, hypoxemia, hypoglycemia, overdose…
Prodrome (presyncope) is common, although syncope may occur
without prodrome
dizziness, lightheadedness or faintness, weakness, fatigue, visual
& auditory disturbances
Causes : neurally mediated syncope (reflex syncope), orthostatic
hypotension, cardiac syncope
13. Examination
Inspection (Observation)
Examination of the arteries & veins:
o BP, Radial pulse (HR, rhythm, volume)
o Examination of the neck (Carotid pulse, ± distended neck veins JVP)
o Examination of the peripheral pulses & auscultation for carotid &
femoral arterial bruits
o ± Varicose veins
Palpation
Auscultation- heart sounds, murmurs, friction rubb
Percussion & auscultation of the lung bases (supportive for Dx)
14. Examination
Inspection
General appearance
Look for Sign of cyanosis
Look for Sign of Anemia
Look for Clubbing of finger or toe
15. Examination cont’
CYANOSIS
Blue discoloration of the skin & mucous membranes caused by reduced O2 in the superficial blood vessels
Can be Central or peripheral cyanosis
Peripheral cyanosis
o May result when cutaneous vasoconstriction slows the blood flow & ↑ oxygen extraction in the
perpheripheral tissue in the capillary bed.
o Usually in the upper and lower extremities where the blood flow is less rapid.
o Physiological during cold exposure
o HF - ↓COP produces reflex cutaneous vasoconstriction
16. Examination cont’
Central cyanosis
o Result from the reduced arterial oxygen saturation caused by cardiac or
pulmonary disease
o level of deoxygenated hemoglobin in the arteries is below 5 g/dL with oxygen
saturation below 85%
o Conditions associated with central cyanosis includes;
Septal defects, PDA,TOF
Plumonary conditions like severe pneumonia, Massive PE
18. Examination cont’
Clubbing
Caused by hypervascularity & the opening of anastomotic channels in the nail bed. Rarely, it
may be congenital
Fingers & toes
Clubbing is not a cardiac specific sign
pulmonary 75 – 80 %
Cardiac 10 – 15 %
Hepatic and GI 5 – 10 %
19. Examination cont’
Grade I –softened nail beds or nail bed fluctuation
Grade II – obliteration of lovibond angle
Grade III –parrot peaking
Grade IV – drum stick like finger tips
20. Causes of clubbing
Cardiopulmonary disorders
Severe chronic cyanosis
Cyanotic congenital heart disease
Chronic fibrosing alveolitis
Chronic suppuration in the lungs
bronchiectasis
empyema
lung abscess
Bronchial Ca
Subacute bacterial endocarditis (SBE)
Chronic abdominal disorders
Crohn's disease
Ulcerative colitis
Cirrhosis of the liver
Schamroth's window test
21.
22. Other cutanoues manifestation
Splinter hemorrhages in the nail-bed (very non-specific finding)
Osler's nodes (tender erythematous nodules in the pulps of the fingers)
Janeway lesions (painless erythematous lesions on the palms)
Roth's spots (erythematous lesions in the optic fundi)
25. Examination cont’
Edema
Cardinal feature of CHF
Pitting - tibia, lateral malleoli, sacrum
Caused by salt & water retention by the kidney
Most prominent around the ankles in the ambulant pt & over the sacrum
in the bedridden pt (gravity effect)
Advanced HF- anasarca (generalized edema)
28. Examination BP
Instrument
Sphygmomanometer, stetoscope
Anaeroid and mercury, digital type
Blood pressure has two component
Systolic
Diastolic pressure
Normal <140/90 mmHg
Korotkoff sounds
29.
30. Examination BP cont’
Width of the inflatable bladder of the cuff should be about 40% of upper arm
circumference (about 12–14 cm in the average adult)
Length of inflatable bladder should be about 80% of upper arm circumference (almost long
enough to encircle the arm)
Ideally, ask the patient to avoid smoking or drinking caffeinated beverages for 30 minutes before
the blood pressure is taken and to rest for at least 5 minutes.
Check to make sure the examining room is quiet and comfortably warm.
Make sure the arm selected is free of clothing or any problem on the hand
31. Examination BP
Position the arm at the level of heart
If the patient is seated, rest the arm on a table a little above the patient’s waist; if
standing, try to support the patient’s arm at the midchest level.
33. Examiantion JVP
Fluctuations in Rt atrial pressure during the cardiac cycle generate a pulse that is transmitted
backwards into the jugular veins
Best examined while the pt reclines at 45°
If the Rt atrial pressure is very low - smaller reclining angle
Alternatively, manual pressure over the upper abdomen may be used to produce a transient
↑in venous return to the heart which elevates the jugular venous pulse (hepatojugular
reflux)
34. Examination JVP
NV :
o 4cm vertically above the sternal angle
o 9cm above the Rt atrium
o 6mmHg
Elevation of the JVP elevation of the RA pressure unless the superior
vena cava is obstructed
During inspiration the pressure within the chest falls & there is a fall in
the JVP
In constrictive pericarditis & tamponade, inspiration produces a
paradoxical rise (Kussmaul's sign) in JVP because the ↑ venous return
cannot be accommodated within the constricted Rt side of the heart
35. Examination JVP
Inspection
o Venous pulse - double undulation, sharper inward movement
o Carotid pulse - easily visible medially & higher in the neck, generally in the
submandibular region, single, sharp outward movement
The amplitude of the venous pulse can be manipulated by changing the
venous pressure :
o ↓ by raising the head & trunk above the level of the Rt atrium (e.g., sitting
or standing)
o ↑by enhancing the venous return to the Rt side of the heart by raising the
legs or compressing the abdomen(RUQ) (Hepatojugular reflux) for at
least 10s
o positive response is defined by a sustained rise of >3 cm in JVP
for at least 15 sec after release of the hand
37. Examination JVP
Steps
Position patient 30/45
Tangential light
Identify internal jugular venousepulsation(right)
Extend a long rectangular object or card horizontally from this point and a
centimeter ruler vertically from the sternal angle, making an exact right angle.
Measure the vertical distance in centimeters