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PRESENTED BY:
LOKESH SAINI
“
“SILLICA “
MAY BE, A KILLER
1
WHAT IS Crystalline Silica?
Silica is one of the most abundant
minerals in the earths crust
composed of silicon and oxygen.
Silica is found commonly in
crystalline quartz state and rarely
in tridymite & cristobalite state.
Silica is found in varying amounts
in almost all types of rock, sands,
clays, shale and gravel. Sandstone
is almost pure quartz whereas
granite might contain 15-30%
quartz.
2/22
wikipedia
1 to 10 µm in size
3/22
WHAT IS respirable Silica?
SOURCES OF RESPIRABLE SILLICA
DRILLING
DEMOLITION
GRINDING
CUTTING
ROAD WORK
4/22
CONSTRUCTION
WORKS
SOURCES OF RESPIRABLE SILLICA
MINING
5/22
CERAMIC GLASS
INDUSTRIAL
ABRASIVE
FOUNDRY
Other Sources
•Sand blasting
•Paint & Coating
•Gem cutting &
Polishing
•Slate/pencil
Silicosis is a slow, progressive, occupational, irreversible
pulmonary lung disease that usually occurs, after many years
of exposure with fine respirable crystalline silica particles (RCS).
Full name :- pneumonoultramicroscopicsilicovolcanoconiosis.
Health Hazards
False-colour x-ray of the chest area of a person suffering from
silicosis. Image sourced from Science Photo Library 2011
X-ray of the chest area of a person suffering from
silicosis. Image sourced from Science Photo Library 2011
WHAT IS silicosis?
(WHO)
6/22
SYMPTOMS
wikipedia
It is hard to diagnosis at early stage.
In early stage , the symptoms may be
Dyspnea (shortness of breath) exacerbated by excertion
Cough, often persistent and sometime severe
Fatigue, Chest pain, Fever
Tachypnea (rapid breathing) which is often labored
Loss of appetite and weight loss (Anorexia)
Gradual dark shallow rifts in nails eventually leading to cracks as
protein fibers within nail beds are destroyed.
In advance cases, the following may occurs
•Cyanosis(Blue Skin)
•Cor pulmonale (right venticle heart disease)
•Respiratory insufficiency, Tuberculosis, Lung Cancer
7/22
HOW DOES IT AFFECT
8/22
Physically Weak
Respiratory
System & Lungs
Skin Problems
PATHPHYSIOLOGY OF SILICOSIS
9/22
Inhalation of
Silica Dust
Reach upto deep
lung (Aleveoli)
O2 - CO2 are exchanged here
gets inhibits due to dust deposit. The
dust can not be cleared by mucus or
coughing
Tumor necrosis factor, interleukin-I,
leukotriene-B4 & cytokines.
These stimulate
fibroblasts to
proliferate & produce
collagen around silica
particle
Fibrosis and
formation of the
nodular lesions
Silicosis affected lung slice Wikipedia
Exact
Pathphysiology
is still unknown
SILICOSIS DEPENDS ON
10/22
 The amount and kind of dust inhaled.
 The percentage of free silica in the dust.
 The form of silica.
 The size & shape of silica particle.
 The duration of exposure.
 The individual’s natural body resistance.
 The presence or absence of complicating factors (such as
infection vectors).
Luton Lina
DETECTION OF SILICOSIS
11/22
 Early detection is difficult-
Absence of symptoms
 Radiography-X ray of chest
(Shows fibrous tissues)
 Work History - Helps
differentiating Silicosis with
other dust causing disease such
as Asbestosis.
 Lung function test using
spirometer.
Luton Tina
12
PREVENTION & CONTROL OF SILICOSIS
• elimination of process.
• substitution of materials
• substitution/modification of processes and equipment
• maintenance of equipment
• wet methods
• work practices
At
Source
• isolation: - of the source (closed systems,
enclosures)
• - of workers (control cabins)
• local exhaust ventilation
Transmission
Path
• Work Practices
• Education and training
• Personal hygiene
• Personal protective equipment
• Health surveillance
At
worker
At Work
environment
Luton Tina
HIERARCHY OF
CONTROL
At Source
Path
Worker
•Layout Change, storage, labeling
•Good housekeeping, Env. Monitoring/alarm
•Warning sign for restricted area
WHO
PERMISSIBLE EXPOSURE LIMIT
(PEL)
As per OSHA,
USA
Limit is
100µg/m³
Statistics
0
0.1
0.2
0.3
0.4
0.5
0.6
1 2 3 4
Predicted risks of developing silicosis within 15
years following exposure
15 years exposure to respirable crystalline silica (8
hour TWA) mg.m3
Exposure
(mg/m³)
Risk (%)
0.02 0.25
0.04 0.5
0.1 2.5
0.3 20
Source: COSHH Regulations 2002 Consultative Document
14/22
Industry wise prevalence of silicosis in India
15/22
Industry Prevalence(%) Reference
Gold Mines 9-14 Caplan, Gowda
Mica Mines 34 CAF
Lead & Zinc 30.4 CAF
Stone Cutter 35 Gupta
Foundry 27 Samel
Slate Pencil 54 Saiyed
Stone quarry 22 NIOH
Sand Grinding 28 NIOH
Ceramics & potteries 15 Saiyed ICMR
1
2
3
AFFECTED DISTRICTS IN RAJASTHAN
16/22
JODHPUR
KOTA
BUNDI
JHALAWAR
CASE STUDY
Workers of stone mine of District Jodhpur
17/22
 There are 15000 unorganized stone mines.
 By random sampling , 49 mines and 376 workers, taken
for study
<20, 27
20-29, 17430-39, 112
40-49, 43
>50, 20 Numbers with Age Distribution
<5Yr, 185
5-9Yr, 126
10-15Yr, 45
>16Yr, 20 Numbers With Duration of Work
Yadav, Anand
CASE STUDY
Workers of stone mine of District Jodhpur
18/22
Silica dust<10µm,
263
Impure Water, 14
Poor Hygiene, 13
Changing
environment, 21
impure food, 26
Don't
Know,
39
Numbers with cause
Yadav, Anand
CASE STUDY
Workers of stone mine of District Jodhpur
19/22
Malaise, disturbed
sleep, hoarseness,
giddiness and
headache with
fever, 77
Fever with loss
of wieght, 78Cough with
breathlessness
and chest pain,
123
Weakness, loss of
appetite and fever,
46
Don't Know,
53
Numbers with Symptom
Yadav, Anand
GOVERNMENT RESPONCE
20/22
•Rs 100,000 to each silicosis-affected person.
•Rs 300,000 to family of a person dies from silicosis.
PAYMENTS ARE MADE BY
RAJASTHAN ENVIRONMENT AND HEALTH
ADMINISTRATIVE BOARD
Ahmad Absar
REFERENCES
21/22
1) GOHNET, The Global Occupational Health Network, WHO, Issue No.12-2007.
2) Ahmad Abrar, Silicosis, Mining, and Occupational Health in India’s Sandstone
Industry; Dec 24th , 2015.
3) www.google.co.in
4) Wikipedia; Silicosis.
5) ICMR Bulletin, Vol29, No 9, September, 1999.
6) National Conference on Silicosis, July 24th 2014.
7) Tina Luton, Silicosis, Educate Eliminate Eradicate, Issue12.
8) Recommendation of NHRC on preventive, remedial, rehabilitative and
compensation aspects of silicosis.
9) Absar Ahmad,”Are the Sandstone Miners Abused in India?” Indian Journal of
Community Health 2015;27,2:286-289.
10) Yadav S. P. , Anand P.K. ; Awareness And Practices About Silicosis Among The Sand
Stone Quarry Workers in Desert Ecology of Jodhpur, Rajasthan.
11) Indian Bureau of Mines(IBM), Indian Minerals Year book (Nagpur: Ministry of
Mines, Government of India, 2012).
22/22

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2015 pce5271lokeshsilicosis

  • 2. WHAT IS Crystalline Silica? Silica is one of the most abundant minerals in the earths crust composed of silicon and oxygen. Silica is found commonly in crystalline quartz state and rarely in tridymite & cristobalite state. Silica is found in varying amounts in almost all types of rock, sands, clays, shale and gravel. Sandstone is almost pure quartz whereas granite might contain 15-30% quartz. 2/22 wikipedia
  • 3. 1 to 10 µm in size 3/22 WHAT IS respirable Silica?
  • 4. SOURCES OF RESPIRABLE SILLICA DRILLING DEMOLITION GRINDING CUTTING ROAD WORK 4/22 CONSTRUCTION WORKS
  • 5. SOURCES OF RESPIRABLE SILLICA MINING 5/22 CERAMIC GLASS INDUSTRIAL ABRASIVE FOUNDRY Other Sources •Sand blasting •Paint & Coating •Gem cutting & Polishing •Slate/pencil
  • 6. Silicosis is a slow, progressive, occupational, irreversible pulmonary lung disease that usually occurs, after many years of exposure with fine respirable crystalline silica particles (RCS). Full name :- pneumonoultramicroscopicsilicovolcanoconiosis. Health Hazards False-colour x-ray of the chest area of a person suffering from silicosis. Image sourced from Science Photo Library 2011 X-ray of the chest area of a person suffering from silicosis. Image sourced from Science Photo Library 2011 WHAT IS silicosis? (WHO) 6/22
  • 7. SYMPTOMS wikipedia It is hard to diagnosis at early stage. In early stage , the symptoms may be Dyspnea (shortness of breath) exacerbated by excertion Cough, often persistent and sometime severe Fatigue, Chest pain, Fever Tachypnea (rapid breathing) which is often labored Loss of appetite and weight loss (Anorexia) Gradual dark shallow rifts in nails eventually leading to cracks as protein fibers within nail beds are destroyed. In advance cases, the following may occurs •Cyanosis(Blue Skin) •Cor pulmonale (right venticle heart disease) •Respiratory insufficiency, Tuberculosis, Lung Cancer 7/22
  • 8. HOW DOES IT AFFECT 8/22 Physically Weak Respiratory System & Lungs Skin Problems
  • 9. PATHPHYSIOLOGY OF SILICOSIS 9/22 Inhalation of Silica Dust Reach upto deep lung (Aleveoli) O2 - CO2 are exchanged here gets inhibits due to dust deposit. The dust can not be cleared by mucus or coughing Tumor necrosis factor, interleukin-I, leukotriene-B4 & cytokines. These stimulate fibroblasts to proliferate & produce collagen around silica particle Fibrosis and formation of the nodular lesions Silicosis affected lung slice Wikipedia Exact Pathphysiology is still unknown
  • 10. SILICOSIS DEPENDS ON 10/22  The amount and kind of dust inhaled.  The percentage of free silica in the dust.  The form of silica.  The size & shape of silica particle.  The duration of exposure.  The individual’s natural body resistance.  The presence or absence of complicating factors (such as infection vectors). Luton Lina
  • 11. DETECTION OF SILICOSIS 11/22  Early detection is difficult- Absence of symptoms  Radiography-X ray of chest (Shows fibrous tissues)  Work History - Helps differentiating Silicosis with other dust causing disease such as Asbestosis.  Lung function test using spirometer. Luton Tina
  • 12. 12 PREVENTION & CONTROL OF SILICOSIS • elimination of process. • substitution of materials • substitution/modification of processes and equipment • maintenance of equipment • wet methods • work practices At Source • isolation: - of the source (closed systems, enclosures) • - of workers (control cabins) • local exhaust ventilation Transmission Path • Work Practices • Education and training • Personal hygiene • Personal protective equipment • Health surveillance At worker At Work environment Luton Tina HIERARCHY OF CONTROL At Source Path Worker •Layout Change, storage, labeling •Good housekeeping, Env. Monitoring/alarm •Warning sign for restricted area WHO
  • 13. PERMISSIBLE EXPOSURE LIMIT (PEL) As per OSHA, USA Limit is 100µg/m³
  • 14. Statistics 0 0.1 0.2 0.3 0.4 0.5 0.6 1 2 3 4 Predicted risks of developing silicosis within 15 years following exposure 15 years exposure to respirable crystalline silica (8 hour TWA) mg.m3 Exposure (mg/m³) Risk (%) 0.02 0.25 0.04 0.5 0.1 2.5 0.3 20 Source: COSHH Regulations 2002 Consultative Document 14/22
  • 15. Industry wise prevalence of silicosis in India 15/22 Industry Prevalence(%) Reference Gold Mines 9-14 Caplan, Gowda Mica Mines 34 CAF Lead & Zinc 30.4 CAF Stone Cutter 35 Gupta Foundry 27 Samel Slate Pencil 54 Saiyed Stone quarry 22 NIOH Sand Grinding 28 NIOH Ceramics & potteries 15 Saiyed ICMR 1 2 3
  • 16. AFFECTED DISTRICTS IN RAJASTHAN 16/22 JODHPUR KOTA BUNDI JHALAWAR
  • 17. CASE STUDY Workers of stone mine of District Jodhpur 17/22  There are 15000 unorganized stone mines.  By random sampling , 49 mines and 376 workers, taken for study <20, 27 20-29, 17430-39, 112 40-49, 43 >50, 20 Numbers with Age Distribution <5Yr, 185 5-9Yr, 126 10-15Yr, 45 >16Yr, 20 Numbers With Duration of Work Yadav, Anand
  • 18. CASE STUDY Workers of stone mine of District Jodhpur 18/22 Silica dust<10µm, 263 Impure Water, 14 Poor Hygiene, 13 Changing environment, 21 impure food, 26 Don't Know, 39 Numbers with cause Yadav, Anand
  • 19. CASE STUDY Workers of stone mine of District Jodhpur 19/22 Malaise, disturbed sleep, hoarseness, giddiness and headache with fever, 77 Fever with loss of wieght, 78Cough with breathlessness and chest pain, 123 Weakness, loss of appetite and fever, 46 Don't Know, 53 Numbers with Symptom Yadav, Anand
  • 20. GOVERNMENT RESPONCE 20/22 •Rs 100,000 to each silicosis-affected person. •Rs 300,000 to family of a person dies from silicosis. PAYMENTS ARE MADE BY RAJASTHAN ENVIRONMENT AND HEALTH ADMINISTRATIVE BOARD Ahmad Absar
  • 21. REFERENCES 21/22 1) GOHNET, The Global Occupational Health Network, WHO, Issue No.12-2007. 2) Ahmad Abrar, Silicosis, Mining, and Occupational Health in India’s Sandstone Industry; Dec 24th , 2015. 3) www.google.co.in 4) Wikipedia; Silicosis. 5) ICMR Bulletin, Vol29, No 9, September, 1999. 6) National Conference on Silicosis, July 24th 2014. 7) Tina Luton, Silicosis, Educate Eliminate Eradicate, Issue12. 8) Recommendation of NHRC on preventive, remedial, rehabilitative and compensation aspects of silicosis. 9) Absar Ahmad,”Are the Sandstone Miners Abused in India?” Indian Journal of Community Health 2015;27,2:286-289. 10) Yadav S. P. , Anand P.K. ; Awareness And Practices About Silicosis Among The Sand Stone Quarry Workers in Desert Ecology of Jodhpur, Rajasthan. 11) Indian Bureau of Mines(IBM), Indian Minerals Year book (Nagpur: Ministry of Mines, Government of India, 2012).
  • 22. 22/22