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Tumours of
the Jaws
Benign tumor
  These
       lesions may be unsightly or
  may be traumatised repeatedly, for
  example during shaving.
   Odontogenic   cysts & non-odontogenic
    tumor
   Soft tissue tumor and hard tissue tumor
Malignant tumor
 Themajority of these lesions spread slowly
 over years but some may spread more
 rapidly to involve lymph glands in the
 local area or more distant areas. All will
 cause great damage if neglected.
Management of benign or
malignant tumor
 Biopsy
 Surgical
         treatment
 Radiotherapy
 Chemotherapy
 Tumor:
  Is a mass of cells,
   tissues or organs
   resembling those
   normally present but
   arranged atypically
   and behave
   abnormally.

    Behavior     is very
        essential and is of great
        importance.
   Classification:
       Histogenetic:
           Epithelial origin
           connective tissue
            origin

       Histological:
           Degree of
            differentiation.
               Well
               moderate
               poorly
                differentiated
Clinical      behavior:
  Benign:
   slowlygrowing and expanding causing pressure
    atrophy but remain within the capsule.
   Very few mitosis could be seen.

  Malignant:
   Invade   surrounding tissues and locally invasive.
   Progressive growth and metastasize to distant
    organs, embolic spread due to lack of cell
    adhesion
   Mitosis.
  Intermediate:
   Locally
          invasive, no metastasis. Basal cell
   carcinoma and Ameloblastoma
 Oral    lesion are
    Carcinomas:
      Non‑secreting    epithelial
         Squamous cell
               90%
      Secreting   epithelial
         Adenocarcinoma
          5%
  Sarcomas:
      Lymphomas
      Others
 Early diagnosis is very essential for management
 Clinical diagnosis from the signs and symptoms
 Referral for essential investigation
   CLINICAL DIAGNOSIS OF ORAL CANCER

       Symptoms vary according to the site of the lesion
         painless in the early stages


         painfuland tender when secondarily infected or
          involves a sensory nerve

         painless    lump or ulcer on the lip

         Posteriorly   no symptom until it reach a size of 2 ‑3 cm
          swelling,
             pain and difficulty in deglutition

         absence   of symptoms until the tumor metastasize to
          regional lymph nodes
             hard lump on the neck
late     symptoms:
    pain due to secondary infection or nerve involvement
    excessive salivation
    difficulty in deglutition, speech
    haemorrhage


Within      bone:
    painless swelling involving the buccal and lingual or palatal
     sulci
     teeth become loose and painful ‑ acute alveolar abscess
    edentulous pt. the denture does not fit
    denture hyperplasia
    anaesthesia of the upper or lower lip and the cheek.
 Carcinoma      of lip:
    age 50‑70 years. Male
     lower class.
  Predisposition     factor:
    dirty,   jagged and stained
     teeth
    irritation.
    tobacco smoker
    leukoplakia.
    intense solar radiation ‑
     blistering cheilitis due to
     sunshine.
 Carcinoma         of tongue
        Anterior 2/3, affect males
        Posterior 1/3 equal in both sexes.
        Age over 60 years.
    Predisposing factors:
        Female with cancer tongue suffer from Paterson ‑Kelly
         syndrome.
        Bad oral hygiene
        Heavy alcoholic with element of Vit.B deficiency.
         Producing precancerous mucosal atrophy
        Syphilitic and leukoplakia. 25% and 5%.
        Superficial glossitis, papilloma, fissures and
         non‑specific ulcers.
 Clinically:
     Painless swelling
     Painful infected ulcer, referred pain to the ear.
     Excessive salivation, marked factor oris,
      haemorrhage
     loss of mobility due to fixation to the floor of the
      mouth.
Malignant first on one side, when
      occur at Tumors
 Fixation
  tongue is protruded it deviate toward the
  affected side
 indurations, fungation or ulceration which
  spread to the floor of the mouth and alveolar
  process and from post. 1/3 to the fauces,
  valleculae and epiglottis bilaterally.
 Spread to regional lymph nodes.
 Death: Inhalation bronchopneumonia,
  haemorrhage, cachexia and starvation and
  asphyxia.
 Carcinoma        of the mouth:
    Floor of the mouth.
      Typicalmalignant ulcer extend to alveolar
       process & tongue.
    The cheek:
      warty   and proliferative.
    The alveolar process:
      warty,   nodules or proliferative.
   Palate:
     spread  extensively before involving bone
      papillary or ulcerative.
   Soft palate and fauces:
     Poor  prognosis. bilateral Lymph node
      involvement
     Proliferative, fungating lesion spread to base
      of tongue.
     Pain, dysphagia and death due to erosion of
      carotid artery

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Jaw Tumor Types, Causes, Symptoms & Treatment

  • 2. Benign tumor  These lesions may be unsightly or may be traumatised repeatedly, for example during shaving.  Odontogenic cysts & non-odontogenic tumor  Soft tissue tumor and hard tissue tumor
  • 3. Malignant tumor  Themajority of these lesions spread slowly over years but some may spread more rapidly to involve lymph glands in the local area or more distant areas. All will cause great damage if neglected.
  • 4. Management of benign or malignant tumor  Biopsy  Surgical treatment  Radiotherapy  Chemotherapy
  • 5.  Tumor:  Is a mass of cells, tissues or organs resembling those normally present but arranged atypically and behave abnormally.  Behavior is very essential and is of great importance.
  • 6. Classification:  Histogenetic:  Epithelial origin  connective tissue origin  Histological:  Degree of differentiation.  Well  moderate  poorly differentiated
  • 7. Clinical behavior:  Benign:  slowlygrowing and expanding causing pressure atrophy but remain within the capsule.  Very few mitosis could be seen.  Malignant:  Invade surrounding tissues and locally invasive.  Progressive growth and metastasize to distant organs, embolic spread due to lack of cell adhesion  Mitosis.  Intermediate:  Locally invasive, no metastasis. Basal cell carcinoma and Ameloblastoma
  • 8.  Oral lesion are  Carcinomas:  Non‑secreting epithelial  Squamous cell 90%  Secreting epithelial  Adenocarcinoma 5%  Sarcomas:  Lymphomas  Others
  • 9.  Early diagnosis is very essential for management  Clinical diagnosis from the signs and symptoms  Referral for essential investigation
  • 10. CLINICAL DIAGNOSIS OF ORAL CANCER  Symptoms vary according to the site of the lesion  painless in the early stages  painfuland tender when secondarily infected or involves a sensory nerve  painless lump or ulcer on the lip  Posteriorly no symptom until it reach a size of 2 ‑3 cm swelling,  pain and difficulty in deglutition  absence of symptoms until the tumor metastasize to regional lymph nodes  hard lump on the neck
  • 11. late symptoms:  pain due to secondary infection or nerve involvement  excessive salivation  difficulty in deglutition, speech  haemorrhage Within bone:  painless swelling involving the buccal and lingual or palatal sulci  teeth become loose and painful ‑ acute alveolar abscess  edentulous pt. the denture does not fit  denture hyperplasia  anaesthesia of the upper or lower lip and the cheek.
  • 12.  Carcinoma of lip:  age 50‑70 years. Male lower class.  Predisposition factor:  dirty, jagged and stained teeth  irritation.  tobacco smoker  leukoplakia.  intense solar radiation ‑ blistering cheilitis due to sunshine.
  • 13.
  • 14.  Carcinoma of tongue  Anterior 2/3, affect males  Posterior 1/3 equal in both sexes.  Age over 60 years.  Predisposing factors:  Female with cancer tongue suffer from Paterson ‑Kelly syndrome.  Bad oral hygiene  Heavy alcoholic with element of Vit.B deficiency. Producing precancerous mucosal atrophy  Syphilitic and leukoplakia. 25% and 5%.  Superficial glossitis, papilloma, fissures and non‑specific ulcers.
  • 15.  Clinically:  Painless swelling  Painful infected ulcer, referred pain to the ear.  Excessive salivation, marked factor oris, haemorrhage  loss of mobility due to fixation to the floor of the mouth.
  • 16. Malignant first on one side, when occur at Tumors  Fixation tongue is protruded it deviate toward the affected side  indurations, fungation or ulceration which spread to the floor of the mouth and alveolar process and from post. 1/3 to the fauces, valleculae and epiglottis bilaterally.  Spread to regional lymph nodes.  Death: Inhalation bronchopneumonia, haemorrhage, cachexia and starvation and asphyxia.
  • 17.  Carcinoma of the mouth:  Floor of the mouth.  Typicalmalignant ulcer extend to alveolar process & tongue.  The cheek:  warty and proliferative.  The alveolar process:  warty, nodules or proliferative.
  • 18. Palate:  spread extensively before involving bone papillary or ulcerative.  Soft palate and fauces:  Poor prognosis. bilateral Lymph node involvement  Proliferative, fungating lesion spread to base of tongue.  Pain, dysphagia and death due to erosion of carotid artery