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CHF in the ED
Bryce C Inman, MD
Loma Linda University Medical Center
Congestive Heart Failure
• Congestive heart failure is an imbalance in
pump function in which the heart is unable to
maintain adequate forward blood flow.
• 10% of those > 80 years old
• Most common cause of death is progressive
heart failure
CHF: 2 types
Systolic
• EF < 40%
• Impaired ventricular
contraction
• Most commonly from
ischemic heart disease
Diastolic
• EF > 60%
• Impaired ventricular
relaxation
• Most commonly from
chronic HTN and LVH
Prognosis
• Heart failure has an overall poor prognosis
• Symptoms predict outcome
– 5-10% mortality per year in moderate CHF
– 30-40% mortality per year in severe CHF
Diagnosis: History
• Dyspnea at rest
• Dyspnea upon exertion
• Orthopnea
• Cough: Frothy pink sputum highly predictive
of CHF
• Nonspecifics: weakness, dizziness, malaise,
etc.
Diagnosis: Exam
• Acute pulmonary edema: Severe respiratory
distress , relative hypertension, diaphoretic
skin. Bilateral crackles can typically be heard
• An S3 has 99 percent specificity for an
elevated capillary wedge pressure (but 20%
sensitivity)
• JVD has 94 percent specificity for elevated
capillary wedge pressure (but 39% sensitivity)
Imaging
• 1/5 CHF patients admitted to the hospital
lacked signs on CXR
• Congestive signs on CXR are unreliable in
chronic CHF
• Sensitivity for CHF with a portable CXR is poor.
• CXR findings often lag behind clinical
manifestions by several hours
However, a CXR is useful to exclude other
processes (e.g., pneumothorax)
Pleural effusion
Pulmonary Edema
What about labs and EKG?
• Lack sensitivity and specificity
– Occasionally you might see an elevated AST/ALT
or prerenal azotemia
– EKG may show ischemia or previous MI,
dysrhythmias, etc.
Natriuretic peptides
• 70 y/o M presents with respiratory distress.
His 02 saturations are in the 70’s, he has mild
retractions, and breath sounds are difficult to
auscultate.
– Is this CHF or COPD?
• A BNP of <100 almost entirely excludes CHF
What else looks like acute CHF?
TREAT!
70 y/o M presents with respiratory distress. His 02
saturations are in the 70’s, he has mild retractions,
and breath sounds are difficult to auscultate.
Airway Management
• Airway management supercedes all other priorities in
these patients, particularly those who are critically ill.
• Hypoxia is a greater risk than hypercarbia so CO2
retention is not an immediate concern
o What is the best way to manage the airway?
Intubation vs NIPPV
Intubation
• Typically for those in severe
distress or those who are
non-cooperative.
BiPAP/CPAP
• May decrease the need for
intubations, but no
significant change in
mortality
Pressure Control
• Systolic pressure acceptable?
– Start nitroglycerin (0.4 mg PO q2-3 min)
– Nitrospray: Single spray (0.4 mg) up to a max of 1.2 mg
Ointment: Apply 1-2 inches of nitropaste to chest wall
IV: Start at 20 mcg/min IV and rate to effect in 5-10 mcg
increments q3-5min
• The failing heart is sensitive to increases in afterload;
these measures alleviate the pulmonary edema from
CHF.
Don’t venodilate when….
• Preload dependent states exist such as;
– Right ventricular infarct
– Critical aortic stenosis
– Volume depletion
Most require only oxygen, blood
pressure control, and diuresis
-Vasoconstricted patients require vasodilators.
-Congested patients required diuretics
★Diastolic HF patients respond better to BP
management than diuresis
Diuresis
• First line therapy is a diuretic such as furosemide.
– 10-20 mg IV for symptomatic CHF and diuretic naïve.
40-80 mg IV for patients already using diuretics
80-120 mg IV for patients whose symptoms are
refractory to the initial dose after 1 h of its
administration
• Metolazone, a thiazide diuretic, can be added for
effect.
If hypotensive…
• Inotropes including dobutamine and
dopamine are used primarily
– Dopamine starts at 5 mcg/kg/min IV and increase
at 5 mcg/kg/min increments to a 20 mcg/kg/min
dose
– Dobutamine starts at 2.5 mcg/kg/min IV; generally
therapeutic in the range of 10-40 mcg/kg/min
Admit or go home?
• With few exceptions, most patients
presenting with symptoms of CHF require
admission. Those who respond well to initial
interventions may require only basic ward
admission with telemetry.
• Those who had a gradual onset dyspnea, rapid
response to therapy, good oxygen saturations,
and ACS/MI unlikely as the inciting event may
be stable for discharge
In conclusion
• Airway management is goal
– IF NIPPV easily available, begin immediately and
monitor for progress or decline
• Control Pressure
– Use nitroglycerin and titrate to effect
– If known diastolic CHF, attempt to reduce
afterload
• Pressor support if hypotensive
– Dobutamine/dopamine

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TUT CHF

  • 1. CHF in the ED Bryce C Inman, MD Loma Linda University Medical Center
  • 2. Congestive Heart Failure • Congestive heart failure is an imbalance in pump function in which the heart is unable to maintain adequate forward blood flow. • 10% of those > 80 years old • Most common cause of death is progressive heart failure
  • 3. CHF: 2 types Systolic • EF < 40% • Impaired ventricular contraction • Most commonly from ischemic heart disease Diastolic • EF > 60% • Impaired ventricular relaxation • Most commonly from chronic HTN and LVH
  • 4.
  • 5. Prognosis • Heart failure has an overall poor prognosis • Symptoms predict outcome – 5-10% mortality per year in moderate CHF – 30-40% mortality per year in severe CHF
  • 6. Diagnosis: History • Dyspnea at rest • Dyspnea upon exertion • Orthopnea • Cough: Frothy pink sputum highly predictive of CHF • Nonspecifics: weakness, dizziness, malaise, etc.
  • 7. Diagnosis: Exam • Acute pulmonary edema: Severe respiratory distress , relative hypertension, diaphoretic skin. Bilateral crackles can typically be heard • An S3 has 99 percent specificity for an elevated capillary wedge pressure (but 20% sensitivity) • JVD has 94 percent specificity for elevated capillary wedge pressure (but 39% sensitivity)
  • 8. Imaging • 1/5 CHF patients admitted to the hospital lacked signs on CXR • Congestive signs on CXR are unreliable in chronic CHF • Sensitivity for CHF with a portable CXR is poor. • CXR findings often lag behind clinical manifestions by several hours However, a CXR is useful to exclude other processes (e.g., pneumothorax)
  • 11. What about labs and EKG? • Lack sensitivity and specificity – Occasionally you might see an elevated AST/ALT or prerenal azotemia – EKG may show ischemia or previous MI, dysrhythmias, etc.
  • 12. Natriuretic peptides • 70 y/o M presents with respiratory distress. His 02 saturations are in the 70’s, he has mild retractions, and breath sounds are difficult to auscultate. – Is this CHF or COPD? • A BNP of <100 almost entirely excludes CHF
  • 13. What else looks like acute CHF?
  • 14. TREAT! 70 y/o M presents with respiratory distress. His 02 saturations are in the 70’s, he has mild retractions, and breath sounds are difficult to auscultate.
  • 15. Airway Management • Airway management supercedes all other priorities in these patients, particularly those who are critically ill. • Hypoxia is a greater risk than hypercarbia so CO2 retention is not an immediate concern o What is the best way to manage the airway?
  • 16. Intubation vs NIPPV Intubation • Typically for those in severe distress or those who are non-cooperative. BiPAP/CPAP • May decrease the need for intubations, but no significant change in mortality
  • 17. Pressure Control • Systolic pressure acceptable? – Start nitroglycerin (0.4 mg PO q2-3 min) – Nitrospray: Single spray (0.4 mg) up to a max of 1.2 mg Ointment: Apply 1-2 inches of nitropaste to chest wall IV: Start at 20 mcg/min IV and rate to effect in 5-10 mcg increments q3-5min • The failing heart is sensitive to increases in afterload; these measures alleviate the pulmonary edema from CHF.
  • 18. Don’t venodilate when…. • Preload dependent states exist such as; – Right ventricular infarct – Critical aortic stenosis – Volume depletion
  • 19. Most require only oxygen, blood pressure control, and diuresis -Vasoconstricted patients require vasodilators. -Congested patients required diuretics ★Diastolic HF patients respond better to BP management than diuresis
  • 20. Diuresis • First line therapy is a diuretic such as furosemide. – 10-20 mg IV for symptomatic CHF and diuretic naïve. 40-80 mg IV for patients already using diuretics 80-120 mg IV for patients whose symptoms are refractory to the initial dose after 1 h of its administration • Metolazone, a thiazide diuretic, can be added for effect.
  • 21. If hypotensive… • Inotropes including dobutamine and dopamine are used primarily – Dopamine starts at 5 mcg/kg/min IV and increase at 5 mcg/kg/min increments to a 20 mcg/kg/min dose – Dobutamine starts at 2.5 mcg/kg/min IV; generally therapeutic in the range of 10-40 mcg/kg/min
  • 22. Admit or go home? • With few exceptions, most patients presenting with symptoms of CHF require admission. Those who respond well to initial interventions may require only basic ward admission with telemetry. • Those who had a gradual onset dyspnea, rapid response to therapy, good oxygen saturations, and ACS/MI unlikely as the inciting event may be stable for discharge
  • 23. In conclusion • Airway management is goal – IF NIPPV easily available, begin immediately and monitor for progress or decline • Control Pressure – Use nitroglycerin and titrate to effect – If known diastolic CHF, attempt to reduce afterload • Pressor support if hypotensive – Dobutamine/dopamine

Editor's Notes

  1. RAAS and sympathetic nervous system activation lead to increased norepin, vasopression  sodium/water retention Cardiac remodeling
  2. Often missed on CXR, especially if patient is intubated and supine.
  3. High output state e.g. anemia or sepsis