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MDCT of Solid Abdominal
Organ Injury
3rd
National Conference SER 2016
Bengaluru, India 23-25 September 2016
K.SHANMUGANATHAN M.D.
ABDOMINAL TRAUMA
OBJECTIVES
• Splenic injury
• Late arterial / early p-v phase imaging
• Liver injury
Blunt Splenic Injury
BLUNT SPLENIC INJURY
• Most commonly injured organ
• MDCT - 98% accurate
• Vascular lesions (AB & VI) – 83%
• 20% rib fractures
• Grading systems
HM, KS, SEM, et al. JACS 2008 ; 206:685-93
Splenic Grading systems
Grade Description of Injury
I Subcapsular hematoma <1 cm thick
Laceration <1 cm parenchymal depth
Parenchymal hematoma <1 cm diameter
II Subcapsular hematoma 1–3 cm thick
Laceration 1–3 cm parenchymal depth
Parenchymal hematoma 1–3 cm in diameter
III Splenic capsular disruption.
Subcapsular hematoma >3 cm thick
Laceration >3 cm parenchymal depth
Parenchymal hematoma >3 cm in diameter
IVA Active intraparenchymal and subcapsular splenic bleeding
Splenic vascular injury (pseudoaneurysm or arteriovenous fistula)
Shattered spleen
IVB Active intraperitoneal bleeding
GRADE INJURY DESCRIPTION
I Hematoma
Laceration
Subcapsular, < 10% surface area
Capsular tear, < 1 cm parenchymal depth
II Hematoma
Laceration
Subcapsular, 10-50% surface area;
Intraparenchymal, < 5cm in diameter
1-3 cm parenchymal depth which does
not involve a trabecular vessel
III Hematoma
Laceration
Subcapsular, > 50% surface area or expanding;
ruptured subcapsular or parenchymal hematoma
> 3 cm parenchymal depth or involving trabacular
vessels
IV Laceration Laceration involving segmental or hilar vessels
producing major devascularization (>25% of spleen)
V Laceration
Vascular
Completely shattered spleen
Hilar vascular injury which devascularizes spleen
TRAUMA “WHOLE BODY”
MDCT PROTOCOLS
c
MDCT Contrast
Volume
I2
mg/mL Rate Delay Detector
width
Pitch Rotation
time
Bolus *
Pro
16- slice 150 mL
300 mg/mL
N.Saline
90ml – 6mL/sec
60ml – 4mL/sec
50ml – 5mL/sec
Bolus pro
90 HU
0.75 mm 0.938 0.5 sec Ascending
Aorta
40 & 64-slice
< 50 yrs 100 mL
350 mg/mL
N.Saline
50ml – 6mL/sec
50ml – 4mL/sec
50ml - 5mL/sec
18 sec 0.625
mm
0.976 0.5 sec
Nil
40 & 64 -slice
> 50 yrs
100 mL 350 mg/mL
N.Saline
50ml – 6mL/sec
50ml – 4mL/sec
50ml - 5mL/sec
Bolus pro
90 HU
0.625
mm
0.976 0.5 sec Ascending
Aorta
Late arterial P-V phase
Optimizing Trauma MDCT Protocol for
Blunt Splenic Injury: Need for Arterial and
Portal Venous Phase Scans
96 / 100
Sens/ Spec
PA
(pseudo-
aneurysm)
AB
(active
bleeding)
NVI
(nonvascular
injury)
PSH
(perisplenic
hematoma)
Arterial 70 / 95 70 / 98 76 / 97 95 / 95
Portal
Venous
17 / 100 93 / 100 93 / 100 98 / 97
Combined 92 / 92 96 / 100 100 / 100 100 / 97
Boscak AR, Shanmuganathan K, Mirvis SE et al. Radiology. 2013 Feb 28.
Common Splenic Injuries
BLUNT SPLENIC INJURY
VASCULAR LESIONS
• Active bleeding
• Vascular injury – PSA & A-V fistula
• Infarcts
Contrast Extravasation
BLUNT SPLENIC INJURY
ACTIVE BLEEDING
• Irregular or linear area
• Increases in size ( delayed phase)
• Seen - only on delayed imaging
BLUNT SPLENIC INJURY
MDCT – Active Bleeding
• AB – Seen in 10% (40/392)
• AB – MDCT (16 slice) 84% Sen, 98% Spc,
95% Acc
• Splenectomy - 60% (24/40)
• Embolization – 94% (16/17)
HM, KS, SEM, et al. JACS 2008; 206:685-93
Arterial P-V phase
Vascular Injury
BLUNT SPLENIC INJURY
VASCULAR INJURY
• Pseudoaneurysm
• Arterio-venous fistula
BLUNT SPLENIC INJURY
VASCULAR INJURY
• Defined
• Low attenuation area
• Washout – isoattenuation or
hyperattenuation
BLUNT SPLENIC INJURY
MDCT – Vascular injury (PSA & AVF)
• VI – Seen in 12% (46/392)
• VI – MDCT (16 slice) – 63% Sen, 94% Spc,
95% Acc
• Embolization – 95% (40/42)
• Splenectomy - 9% (4/46)
HM, KS, SEM, et al. JACS 2008 ; 206:685-93
Why Late arterial/ Early P-V
Images?
• Demonstrates parenchymal vascular
injury (PSA & fistulas) not seen on p-v
phase images
48 – year old male with past history of
cirrhosis (Hep C), GI bleeds,
thrombocytopenia (platelets 78 K/Mc L) was
admitted following a fall down multiple steps
at home. Had an abnormal INR/ prothrombin
time 15.7
Admission
3 day follow-up Arterial phase
P-V phase
Arterial phase
P-V Phase
40 keV
Mixed
Iodine maps
Splenic Infarcts
Liver Injury
BLUNT LIVER INJURY
• Injury - 2ND
most common
• Nonsurgical management - 50 % - 96%
• Hemorrhage - mortality
• Rx – multi-disciplinary approach
IR & SURGERY
PAP,SEM,KS, et al. Radiology 2000;216:417-27
BLUNT LIVER INJURY
VASCULAR INJURY
• Active bleeding – hepatic artery,
portal, or hepatic veins
• Pseudoaneurysm - rare
• Fistula - three types
A-V fistula, P-V
P-V phase
Late arterial phase
Late arterial phase – 3 days post injury
P-Vphase

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Mdct of solid organ injury india 2014

  • 1. MDCT of Solid Abdominal Organ Injury 3rd National Conference SER 2016 Bengaluru, India 23-25 September 2016 K.SHANMUGANATHAN M.D.
  • 2. ABDOMINAL TRAUMA OBJECTIVES • Splenic injury • Late arterial / early p-v phase imaging • Liver injury
  • 4. BLUNT SPLENIC INJURY • Most commonly injured organ • MDCT - 98% accurate • Vascular lesions (AB & VI) – 83% • 20% rib fractures • Grading systems HM, KS, SEM, et al. JACS 2008 ; 206:685-93
  • 5. Splenic Grading systems Grade Description of Injury I Subcapsular hematoma <1 cm thick Laceration <1 cm parenchymal depth Parenchymal hematoma <1 cm diameter II Subcapsular hematoma 1–3 cm thick Laceration 1–3 cm parenchymal depth Parenchymal hematoma 1–3 cm in diameter III Splenic capsular disruption. Subcapsular hematoma >3 cm thick Laceration >3 cm parenchymal depth Parenchymal hematoma >3 cm in diameter IVA Active intraparenchymal and subcapsular splenic bleeding Splenic vascular injury (pseudoaneurysm or arteriovenous fistula) Shattered spleen IVB Active intraperitoneal bleeding GRADE INJURY DESCRIPTION I Hematoma Laceration Subcapsular, < 10% surface area Capsular tear, < 1 cm parenchymal depth II Hematoma Laceration Subcapsular, 10-50% surface area; Intraparenchymal, < 5cm in diameter 1-3 cm parenchymal depth which does not involve a trabecular vessel III Hematoma Laceration Subcapsular, > 50% surface area or expanding; ruptured subcapsular or parenchymal hematoma > 3 cm parenchymal depth or involving trabacular vessels IV Laceration Laceration involving segmental or hilar vessels producing major devascularization (>25% of spleen) V Laceration Vascular Completely shattered spleen Hilar vascular injury which devascularizes spleen
  • 6. TRAUMA “WHOLE BODY” MDCT PROTOCOLS c MDCT Contrast Volume I2 mg/mL Rate Delay Detector width Pitch Rotation time Bolus * Pro 16- slice 150 mL 300 mg/mL N.Saline 90ml – 6mL/sec 60ml – 4mL/sec 50ml – 5mL/sec Bolus pro 90 HU 0.75 mm 0.938 0.5 sec Ascending Aorta 40 & 64-slice < 50 yrs 100 mL 350 mg/mL N.Saline 50ml – 6mL/sec 50ml – 4mL/sec 50ml - 5mL/sec 18 sec 0.625 mm 0.976 0.5 sec Nil 40 & 64 -slice > 50 yrs 100 mL 350 mg/mL N.Saline 50ml – 6mL/sec 50ml – 4mL/sec 50ml - 5mL/sec Bolus pro 90 HU 0.625 mm 0.976 0.5 sec Ascending Aorta Late arterial P-V phase
  • 7. Optimizing Trauma MDCT Protocol for Blunt Splenic Injury: Need for Arterial and Portal Venous Phase Scans 96 / 100 Sens/ Spec PA (pseudo- aneurysm) AB (active bleeding) NVI (nonvascular injury) PSH (perisplenic hematoma) Arterial 70 / 95 70 / 98 76 / 97 95 / 95 Portal Venous 17 / 100 93 / 100 93 / 100 98 / 97 Combined 92 / 92 96 / 100 100 / 100 100 / 97 Boscak AR, Shanmuganathan K, Mirvis SE et al. Radiology. 2013 Feb 28.
  • 9. BLUNT SPLENIC INJURY VASCULAR LESIONS • Active bleeding • Vascular injury – PSA & A-V fistula • Infarcts
  • 11. BLUNT SPLENIC INJURY ACTIVE BLEEDING • Irregular or linear area • Increases in size ( delayed phase) • Seen - only on delayed imaging
  • 12. BLUNT SPLENIC INJURY MDCT – Active Bleeding • AB – Seen in 10% (40/392) • AB – MDCT (16 slice) 84% Sen, 98% Spc, 95% Acc • Splenectomy - 60% (24/40) • Embolization – 94% (16/17) HM, KS, SEM, et al. JACS 2008; 206:685-93
  • 13.
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  • 17. BLUNT SPLENIC INJURY VASCULAR INJURY • Pseudoaneurysm • Arterio-venous fistula
  • 18. BLUNT SPLENIC INJURY VASCULAR INJURY • Defined • Low attenuation area • Washout – isoattenuation or hyperattenuation
  • 19. BLUNT SPLENIC INJURY MDCT – Vascular injury (PSA & AVF) • VI – Seen in 12% (46/392) • VI – MDCT (16 slice) – 63% Sen, 94% Spc, 95% Acc • Embolization – 95% (40/42) • Splenectomy - 9% (4/46) HM, KS, SEM, et al. JACS 2008 ; 206:685-93
  • 20. Why Late arterial/ Early P-V Images? • Demonstrates parenchymal vascular injury (PSA & fistulas) not seen on p-v phase images
  • 21. 48 – year old male with past history of cirrhosis (Hep C), GI bleeds, thrombocytopenia (platelets 78 K/Mc L) was admitted following a fall down multiple steps at home. Had an abnormal INR/ prothrombin time 15.7
  • 22. Admission 3 day follow-up Arterial phase P-V phase
  • 26.
  • 28. BLUNT LIVER INJURY • Injury - 2ND most common • Nonsurgical management - 50 % - 96% • Hemorrhage - mortality • Rx – multi-disciplinary approach IR & SURGERY
  • 29. PAP,SEM,KS, et al. Radiology 2000;216:417-27
  • 30. BLUNT LIVER INJURY VASCULAR INJURY • Active bleeding – hepatic artery, portal, or hepatic veins • Pseudoaneurysm - rare • Fistula - three types A-V fistula, P-V
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  • 37. Late arterial phase – 3 days post injury P-Vphase

Editor's Notes

  1. American Association for the Surgery of Trauma
  2. 6 radiologist reviewed at 3 different time period
  3. active bleeding spleen
  4. -- Year old male admitted following a fall with abdominal pain. Admission CT &amp; 3 day follow-up CT
  5. hypoperfusion syndrome
  6. Blunt trauma with arterio-portal fistula
  7. Portal vein PSA
  8. Transfer with out side CT showing a grade II injury (only p-v phase images) comes back for follow-up CT.