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Thermal injuries
Dr Rupesh
ASSISTANT PROFESSOR
Classification
• Thermal deaths may result from
1. Extreme Heat 4. Scalds
2. Extreme Cold 5. Electrical
3. Burns 6. Lightning
Due to heat
General –
heat stroke,
heat
cramps,
heat
exhaustion
Local –
Burns and
scalds
Due to cold
General –
hypothermia
Local –
Frost bite
Trench foot
Definition
It is an injury which is caused by the
application of heat, chemical
substances to the external or
internal surfaces of the body
causing tissue destruction.
Exclusion --?
Temp req-
Min time-
44-51
65-
>>70 –
Varieties of burns
• Chemical UV RAYS
• Electrical x rays
• Explosions
• Flame
• Heated solid body
• Infrared rays
• Laser
• Microwave burns
Classification
• Dupuytren
• Wilson
• Hebra
• Hebra and wilson
• Epidermal
• Dermo- epidermal
• Deep
Clinical classification
• Partial thickness
• Full thickness
Effects of burns
• Surface area
• The degree of heat
• Duration of exposure
• Age
• Sex
• Site of burns
Surface area
• Rule of wallace
• Rule of five
• Rule of palm
• Lund and browder chart
Cause of death
• Immediate – neurogenic, Co monoxide intox, acute respir intox.
• Late – gangrene, embolism, biochemical disturbances, secondary
shock
• Very late –marjolins ulcer
• Scene of death
• PM appearances – external
• Clothes, position of body, facial features,
• Crows feet
Pugilistic attitude
• Causes
• Mechanism
• Features
• Interpretation
• d/d
PM appearences
• Burnt areas- irregular, blisters, microscopy
• Flash burns
• Heat rupture
• Hair -
• Internal findings
• 1. heat hematoma
• Heat fractures
• GIT
• Viscera
• Blood
S.no Feature Heat rupture Lacerated wound
1 Cause Exposure to intense heat Blunt force
2 Site Fatty tissue Anywhere
3 Bleeding Absent Present
4 Margins No bruise and vital reactions Present
5 Floor Intact nerves and vessels Nerves and vessels cut
6 Associated
findings
Of burns Of trauma
S.NO Trait Heat hematoma EDH
1 CAUSE INTENSE HEAT BLUNT TRAUMA
2 LOCATION PARIETO TEMPORAL AT THE SITE OF TRAUMA
3 DISTRIBUTION DIFFUSE LOCALISED
4 MECHANISM EXPANSION OF BLOOD IN
DIPOLE
RUPTURE OF MMA
5 Appearance Honey comb Rubbery
6 CNS No injury Injury present
7 Co in clot Yes no
Establishment of identity
• Teeth
• Finger prints
• Gums
• Autopsy findings
Age of burns
• Immediate – erythema
• 1 hour- vesication
• 6 hours – epidermis thinned out, inflammatory reaction in dermis
• 12-24 hours –exudate will begins to dry
• 36 hours – pus formation (if not healthy)
• 2-3 weeks – granulation tissue occurs
• Several weeks to months- scar formation
• 10-25 yrs – ulcer
s.No Trait AM BURNS PM BURNS
1 LINE OF
REDNESS
PRESENT ABSENT
2 BLISTERS BASE IS RED AND INFLAMED
EXUDATE –RICH IN PROTIENS
AND CHLORIDES
BASE IS PALE,DRY AND HARD
CONTAINS AIR
3 VITAL
REACTION
MARKED NONE
4 ENZYMES INCREASED ENZYMES NO SUCH
5 Co Hb Present Absent
MLI OF BURNS
• BRANDING
• MANNER OF INFLICTION
• NECKLACING
• SCTION 285 IPC
Cold injuries
General – hypothermia
Local – with tissue freezing
- Without tissue freezing
• Definition – a condition in which core temperature <35* c
• Stages of hypothermia
• 1st stage
• 2nd stage
• 3 rd stage
• Types – primary , secondary
• - acute, chronic
• Local effects
• Frost nip
• Frost bite
With out tissue freezing
• Trench foot
• Chilblains
Treatment
• External rewarming
• Internal rewarming
• Iv fluids, humidified o2, peritoneal lavage etc
• Antibiotics
PM appearances
• External –
• Skin-
• White death
• Internal – skull
• Heart-
• Stomach – wischnewski erosions
• Fat necrosis
Mli of hypothermia
• Paradoxical undressing
• Neonatal cold injury
• Accidental hypothermia
Heat
• Factors affecting heat loss
• Hot and humid conditions
• Heavy tight waterproof clothes
• Diseases- CF, psoriasis, eczema etc
• Obesity -
•Heat cramps
•Heat syncope
•Heat exhaustion
•Heat stroke
Heat cramps
• Muscle spasm resulting from combination of
• Prolonged excercise,
• Heavy sweating
• Excessive water repalacement in extreme heat
• Pathophysiology -?
• Symptoms
• Treatment
Heat syncope
• Def- is a condition resulting from ?
• NEITHER WATER OR SALT DEPLETION
• Predisposing factors- sudden change in environ
• prolonged standing
• Signs and symptoms
• Management
Heat exhaustion
• Is a condition resulting from severe
Dehydration after a huge amount of
Sweet has been lost
Pathophysiology
Thermo regulation is maintained
Sequelae –
Management – cold water with salt
Heat stroke
• Heat hyperpyrexia, siriasis, sun stroke, thermic fever
• Complete break down of TR mechanisms
• Complete loss of sweating
• Temp >41 *c
Signs and symptoms
PM Appearences
• Brain – petechial hemorrhages on white matter
• Degeneration of neurons- GP
• Diffuse proliferation of microglia
• Swollen dendrites
• Cerebellum – purkinje layer- edema
• Purkinje cells- degeneration
• Hypothalamus- edema of nuclei
• Heart – sub epicardial and sub endocardial
• Lungs
• Liver
• Kidneys
• Adrenals -
MLI
• DEATHS IN PRISONS
• WORK ENVIRONMENT
burn injuries - Copy.pptx

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burn injuries - Copy.pptx

  • 1.
  • 3.
  • 4. Classification • Thermal deaths may result from 1. Extreme Heat 4. Scalds 2. Extreme Cold 5. Electrical 3. Burns 6. Lightning
  • 5. Due to heat General – heat stroke, heat cramps, heat exhaustion Local – Burns and scalds
  • 6. Due to cold General – hypothermia Local – Frost bite Trench foot
  • 7. Definition It is an injury which is caused by the application of heat, chemical substances to the external or internal surfaces of the body causing tissue destruction. Exclusion --?
  • 9. Varieties of burns • Chemical UV RAYS • Electrical x rays • Explosions • Flame • Heated solid body • Infrared rays • Laser • Microwave burns
  • 11.
  • 12. • Hebra and wilson • Epidermal • Dermo- epidermal • Deep
  • 13. Clinical classification • Partial thickness • Full thickness
  • 14. Effects of burns • Surface area • The degree of heat • Duration of exposure • Age • Sex • Site of burns
  • 15. Surface area • Rule of wallace • Rule of five
  • 16. • Rule of palm • Lund and browder chart
  • 17. Cause of death • Immediate – neurogenic, Co monoxide intox, acute respir intox. • Late – gangrene, embolism, biochemical disturbances, secondary shock • Very late –marjolins ulcer
  • 18. • Scene of death • PM appearances – external • Clothes, position of body, facial features, • Crows feet
  • 19. Pugilistic attitude • Causes • Mechanism • Features • Interpretation • d/d
  • 20. PM appearences • Burnt areas- irregular, blisters, microscopy • Flash burns • Heat rupture • Hair - • Internal findings • 1. heat hematoma • Heat fractures • GIT • Viscera • Blood
  • 21. S.no Feature Heat rupture Lacerated wound 1 Cause Exposure to intense heat Blunt force 2 Site Fatty tissue Anywhere 3 Bleeding Absent Present 4 Margins No bruise and vital reactions Present 5 Floor Intact nerves and vessels Nerves and vessels cut 6 Associated findings Of burns Of trauma
  • 22. S.NO Trait Heat hematoma EDH 1 CAUSE INTENSE HEAT BLUNT TRAUMA 2 LOCATION PARIETO TEMPORAL AT THE SITE OF TRAUMA 3 DISTRIBUTION DIFFUSE LOCALISED 4 MECHANISM EXPANSION OF BLOOD IN DIPOLE RUPTURE OF MMA 5 Appearance Honey comb Rubbery 6 CNS No injury Injury present 7 Co in clot Yes no
  • 23. Establishment of identity • Teeth • Finger prints • Gums • Autopsy findings
  • 24. Age of burns • Immediate – erythema • 1 hour- vesication • 6 hours – epidermis thinned out, inflammatory reaction in dermis • 12-24 hours –exudate will begins to dry • 36 hours – pus formation (if not healthy) • 2-3 weeks – granulation tissue occurs • Several weeks to months- scar formation • 10-25 yrs – ulcer
  • 25. s.No Trait AM BURNS PM BURNS 1 LINE OF REDNESS PRESENT ABSENT 2 BLISTERS BASE IS RED AND INFLAMED EXUDATE –RICH IN PROTIENS AND CHLORIDES BASE IS PALE,DRY AND HARD CONTAINS AIR 3 VITAL REACTION MARKED NONE 4 ENZYMES INCREASED ENZYMES NO SUCH 5 Co Hb Present Absent
  • 26. MLI OF BURNS • BRANDING • MANNER OF INFLICTION • NECKLACING • SCTION 285 IPC
  • 27.
  • 28.
  • 29.
  • 30.
  • 31. Cold injuries General – hypothermia Local – with tissue freezing - Without tissue freezing
  • 32. • Definition – a condition in which core temperature <35* c • Stages of hypothermia • 1st stage • 2nd stage • 3 rd stage
  • 33. • Types – primary , secondary • - acute, chronic • Local effects • Frost nip • Frost bite
  • 34. With out tissue freezing • Trench foot • Chilblains
  • 35. Treatment • External rewarming • Internal rewarming • Iv fluids, humidified o2, peritoneal lavage etc • Antibiotics
  • 36. PM appearances • External – • Skin- • White death • Internal – skull • Heart- • Stomach – wischnewski erosions • Fat necrosis
  • 37. Mli of hypothermia • Paradoxical undressing • Neonatal cold injury • Accidental hypothermia
  • 38.
  • 39. Heat • Factors affecting heat loss • Hot and humid conditions • Heavy tight waterproof clothes • Diseases- CF, psoriasis, eczema etc • Obesity -
  • 40. •Heat cramps •Heat syncope •Heat exhaustion •Heat stroke
  • 41. Heat cramps • Muscle spasm resulting from combination of • Prolonged excercise, • Heavy sweating • Excessive water repalacement in extreme heat • Pathophysiology -? • Symptoms • Treatment
  • 42. Heat syncope • Def- is a condition resulting from ? • NEITHER WATER OR SALT DEPLETION • Predisposing factors- sudden change in environ • prolonged standing • Signs and symptoms • Management
  • 43. Heat exhaustion • Is a condition resulting from severe Dehydration after a huge amount of Sweet has been lost Pathophysiology Thermo regulation is maintained Sequelae – Management – cold water with salt
  • 44. Heat stroke • Heat hyperpyrexia, siriasis, sun stroke, thermic fever • Complete break down of TR mechanisms • Complete loss of sweating • Temp >41 *c
  • 46. PM Appearences • Brain – petechial hemorrhages on white matter • Degeneration of neurons- GP • Diffuse proliferation of microglia • Swollen dendrites • Cerebellum – purkinje layer- edema • Purkinje cells- degeneration • Hypothalamus- edema of nuclei
  • 47. • Heart – sub epicardial and sub endocardial • Lungs • Liver • Kidneys • Adrenals -
  • 48. MLI • DEATHS IN PRISONS • WORK ENVIRONMENT