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REACTION OF TEETH TO TRAUMATIC
INJURIES
Presented by
Dr. Rasha Hatem
Lecturer of Pediatric Dentistry and Dental Public
Health
Faculty of Oral and Dental Medicine
Cairo University
Treatment of Traumatic Dental Injuries in
The Primary Dentition
Primary teeth are more likely to be displaced than
fractured because of:
• The thinner and more elastic alveolar bone.
• Physiological resorption, which reduces the root length.
The effect of injury in the deciduous dentition falls into
two categories:
1) Immediate effect on the primary teeth:
Concussion
Displacement: Intrusion, extrusion or avulsion.
Fracture: Crown-root fracture (very rare).
2) Indirect effect on the unerupted permanent teeth:
Hypoplasia: Turner’s hypoplasia.
Hypomineralization
Dilaceration
I. Treatment of fractured primary teeth
- Enamel and dentin fractures
- Enamel fractures
- Fractures involving pulp:
- Traumatized anterior teeth that have become non-vital:
 No treatment is required unless there are signs of a
pathological condition (i.e. pain, abscess and fistula).
 Treatment can be either pulpectomy then filling with
resorbable paste or extraction.
- Fractures of root of primary teeth
II. Treatment of displaced primary teeth
The intruded primary tooth will re-erupt spontaneously
[either partially or completely] over a period of few
months.
 Extraction is indicated when the apex of intruded tooth
approaches the permanent tooth germ.
 Infection of the intruded primary tooth e.g. periapical
abscess necessitates its extraction.
1. Concussion:No treatment, just soft diet & follow-up.
2. Intrusion:
Complications following intrusion of
primary teeth
2- Gemination: Partial duplication in the part of the affected
tooth formed after injury.
1- Dilaceration:
3-Turner’s hypoplasia: localized small pigmented
hypoplastic area in the permanent successor.
4- Appearance of additional cusps or denticles.
3. Extrusion:
The extruded primary tooth is usually extracted if severely
loose. Repositioning such tooth may result in damage to the
underlying permanent successor. In addition, providing
adequate splint to support the repositioned tooth may be
difficult in a very young child.
4. Avulsion:
Avulsed primary teeth are not replanted to avoid injury to
the underlying permanent successor. The tooth should be
discarded
Reactions of tooth to trauma
1. Pulpal hyperemia:
 Follows minor trauma where there is congestion of
blood vessels in pulp chamber which may lead to
pulp necrosis .
 The hyperemic tooth appears reddish in colour as
compared to adjacent teeth.
 The hyperemic tooth may undergo resolution or necrosis
and is treated with root canal therapy.
2. Internal haemorrhage:
• Occurs following trauma which results in hyperemia
and increased blood pressure in the pulp leading to
rupture of capillaries and escape of RBCs with
subsequent breakdown and pigment formation.
• Discoloration may be temporary in mild cases where
reabsorption of RBCs occurs before reaching the
dentinal tubules.
• Discoloration may be permanent in severe cases due
to pigment formation in dentinal tubules.
3. Pulp calcification:
• It is a rapid physiologic repair response of the pulp to
trauma which may continue until the pulp is
completely replaced by calcified tissue. The clinical
crowns appear opaque yellow in colour and show no
response to various pulp tests
• Primary teeth will undergo normal physiologic
resorption while permanent teeth will be indefinitely
retained.
4. Internal resorption:
• It is a destructive process caused by osteoclastic
activity and maybe seen radiographically in pulp
chamber or root canals a few weeks following trauma.
• It may lead to perforation of the root or the crown
may appear with a “pink spot” where vascular pulp
tissue shines through the remaining thin shell of the
tooth.
5. External root resorption:
Occurs following trauma with damage to the
periodontal structures or when trauma causes tooth
displacement. The process usually continues until gross
areas of the root may be destroyed.
• If internal resorption is detected early, the pulp tissue is
extirpated and Ca(OH)2 is placed in the canal to create an
environment unfavourable for root resorption. Repeated
applications of Ca(OH)2 may be necessary until radiographs
confirm cessation of the process. Gutta percha is placed as
the final filling material.
6. Pulpal necrosis:
• Occurs following trauma which causes severance of
apical vessels and decreased blood supply to the pulp.
• A necrosed tooth can be treated with root canal therapy.
7. Ankylosis:
• It is the fusion of alveolar bone and root surface.
• Radiographically:
There is interruption in periodontal membrane space of
ankylosed tooth and continuity of dentin and alveolar bone.
• Clinically:
There is difference in the incisal plane of ankylosed
tooth and the adjacent (submerged)
.Treatment:
 In ankylosed primary anterior teeth, extraction is done
followed by a space maintainer.
 In ankylosed permanent anterior teeth, the tooth can be
covered by a jacket crown.
Reaction of teeth to trauma

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Reaction of teeth to trauma

  • 1. REACTION OF TEETH TO TRAUMATIC INJURIES Presented by Dr. Rasha Hatem Lecturer of Pediatric Dentistry and Dental Public Health Faculty of Oral and Dental Medicine Cairo University
  • 2. Treatment of Traumatic Dental Injuries in The Primary Dentition Primary teeth are more likely to be displaced than fractured because of: • The thinner and more elastic alveolar bone. • Physiological resorption, which reduces the root length. The effect of injury in the deciduous dentition falls into two categories: 1) Immediate effect on the primary teeth: Concussion Displacement: Intrusion, extrusion or avulsion. Fracture: Crown-root fracture (very rare).
  • 3. 2) Indirect effect on the unerupted permanent teeth: Hypoplasia: Turner’s hypoplasia. Hypomineralization Dilaceration I. Treatment of fractured primary teeth - Enamel and dentin fractures - Enamel fractures
  • 4. - Fractures involving pulp: - Traumatized anterior teeth that have become non-vital:  No treatment is required unless there are signs of a pathological condition (i.e. pain, abscess and fistula).  Treatment can be either pulpectomy then filling with resorbable paste or extraction. - Fractures of root of primary teeth
  • 5. II. Treatment of displaced primary teeth The intruded primary tooth will re-erupt spontaneously [either partially or completely] over a period of few months.  Extraction is indicated when the apex of intruded tooth approaches the permanent tooth germ.  Infection of the intruded primary tooth e.g. periapical abscess necessitates its extraction. 1. Concussion:No treatment, just soft diet & follow-up. 2. Intrusion:
  • 6. Complications following intrusion of primary teeth 2- Gemination: Partial duplication in the part of the affected tooth formed after injury. 1- Dilaceration:
  • 7. 3-Turner’s hypoplasia: localized small pigmented hypoplastic area in the permanent successor. 4- Appearance of additional cusps or denticles.
  • 8. 3. Extrusion: The extruded primary tooth is usually extracted if severely loose. Repositioning such tooth may result in damage to the underlying permanent successor. In addition, providing adequate splint to support the repositioned tooth may be difficult in a very young child. 4. Avulsion: Avulsed primary teeth are not replanted to avoid injury to the underlying permanent successor. The tooth should be discarded
  • 9. Reactions of tooth to trauma 1. Pulpal hyperemia:  Follows minor trauma where there is congestion of blood vessels in pulp chamber which may lead to pulp necrosis .  The hyperemic tooth appears reddish in colour as compared to adjacent teeth.  The hyperemic tooth may undergo resolution or necrosis and is treated with root canal therapy.
  • 10. 2. Internal haemorrhage: • Occurs following trauma which results in hyperemia and increased blood pressure in the pulp leading to rupture of capillaries and escape of RBCs with subsequent breakdown and pigment formation. • Discoloration may be temporary in mild cases where reabsorption of RBCs occurs before reaching the dentinal tubules. • Discoloration may be permanent in severe cases due to pigment formation in dentinal tubules.
  • 11. 3. Pulp calcification: • It is a rapid physiologic repair response of the pulp to trauma which may continue until the pulp is completely replaced by calcified tissue. The clinical crowns appear opaque yellow in colour and show no response to various pulp tests • Primary teeth will undergo normal physiologic resorption while permanent teeth will be indefinitely retained.
  • 12. 4. Internal resorption: • It is a destructive process caused by osteoclastic activity and maybe seen radiographically in pulp chamber or root canals a few weeks following trauma. • It may lead to perforation of the root or the crown may appear with a “pink spot” where vascular pulp tissue shines through the remaining thin shell of the tooth.
  • 13. 5. External root resorption: Occurs following trauma with damage to the periodontal structures or when trauma causes tooth displacement. The process usually continues until gross areas of the root may be destroyed. • If internal resorption is detected early, the pulp tissue is extirpated and Ca(OH)2 is placed in the canal to create an environment unfavourable for root resorption. Repeated applications of Ca(OH)2 may be necessary until radiographs confirm cessation of the process. Gutta percha is placed as the final filling material.
  • 14. 6. Pulpal necrosis: • Occurs following trauma which causes severance of apical vessels and decreased blood supply to the pulp. • A necrosed tooth can be treated with root canal therapy.
  • 15. 7. Ankylosis: • It is the fusion of alveolar bone and root surface. • Radiographically: There is interruption in periodontal membrane space of ankylosed tooth and continuity of dentin and alveolar bone. • Clinically: There is difference in the incisal plane of ankylosed tooth and the adjacent (submerged)
  • 16. .Treatment:  In ankylosed primary anterior teeth, extraction is done followed by a space maintainer.  In ankylosed permanent anterior teeth, the tooth can be covered by a jacket crown.

Editor's Notes

  1. Dilaceration: occurs in permanent successors of intruded primary teeth , where the calcified portion of permanent successor is twisted or bent upon itself and from this new position growth progresses. It may lead to a sharp angle between the crown and root.
  2. Trauma which causes injury and inflammation to periodontal membrane is associated with osteoclastic activity which may result in external root resorption. This process may be followed by repair. If repair occurs at a higher rate than resorption, this may lead to fusion of alveolar bone and root surface.