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Head InjuryHead Injury
Definition of head injury
Discus the types of head injury
Understand the different sign and symptom of head
injury
Describe the pathophysiol0gy of head injury
Explain the diagnostic evaluation of head injury
Enlist the complication of head injury
Detail the management of head injury
Any trauma to the scalp, skull, or brain
Head trauma includes an alteration in
consciousness no matter how brief
Causes
Motor vehicle accidents
Firearm-related injuries
Falls
Assaults
Sports-related injuries
Recreational accidents
High potential for poor outcome
Deaths occur at three points in time after
injury:
Immediately after the injury
Within 2 hours after injury
3 weeks after injury
Scalp lacerations
The most minor type of head
trauma
Scalp is highly vascular → profuse
bleeding
Major complication is infection
Types of Head Injuries
Skull fractures
Linear or depressed
Simple, comminuted, or compound
Closed or open
Direct & Indirect
Coup & Contrecoup
Skull fractures
Location of fracture alters the
presentation of the manifestations
Facial paralysis
Conjugate deviation of gaze palsy
Battle’s sign
Basal Skull fractures
CSF leak (extravasation) into ear (Otorrhea) or
nose (Rhinorrhea)
High risk infection or meningitis
“HALO Sign (Battle Sign)” on clothes of linen
Possible injury to Internal carotid artery
Permanent CSF leaks possible
Fig. 55-13
Minor head trauma
Concussion
A sudden transient mechanical head
injury with disruption of neural
activity and a change in LOC
Brief disruption in LOC
Amnesia
Headache
Short duration
Minor head trauma
Postconcussion syndrome
2 weeks to 2 months
Persistent headache
Lethargy
Personality and behavior
changes
Major head trauma
Includes cerebral contusions and
lacerations
Both injuries represent severe
trauma to the brain
Major head trauma
Contusion
The bruising of brain tissue within a
focal area that maintains the integrity
of the pia mater and arachnoid layers
Lacerations
Involve actual tearing of the brain
tissue
Intracerebral hemorrhage is generally
associated with cerebral laceration
Diffuse axonal injury (DAI)
Widespread axonal damage
occurring after a mild, moderate, or
severe TBI
Process takes approximately 12-24
hours
Diffuse axonal injury (DAI)
Clinical signs:
↓ LOC
↑ ICP
Decerebration or decortication
Global cerebral edema
Epidural hematoma
Results from bleeding between the
dura and the inner surface of the
skull
A neurologic emergency
Venous or arterial origin
Subdural hematoma
Occurs from bleeding between the
dura mater and arachnoid layer of
the meningeal covering of the brain
Fig. 55-15
Subdural Hematoma
Epidural Hematoma
Subdural hematoma
Usually venous in origin
Much slower to develop into a mass
large enough to produce symptoms
May be caused by an arterial
hemorrhage
Subdural hematoma
Acute subdural hematoma
High mortality
Signs within 48 hours of the injury
Associated with major trauma
(Shearing Forces)
Patient appears drowsy and confused
Pupils dilate and become fixed
Subdural hematoma
Subacute subdural hematoma
Occurs within 2-14 days of the
injury
Failure to regain consciousness
may be an indicator
Subdural hematoma
Chronic subdural hematoma
Develops over weeks or months
after a seemingly minor head
injury
CT scan considered the best diagnostic test
to determine craniocerebral trauma
MRI
Cervical spine x-ray
Glasgow Coma Scale (GCS)
Craniotomy
Craniectomy
Cranioplasty
Burr-hole
Nursing Assessment
GCS score
Neurologic status
Presence of CSF leak
Nursing Diagnoses
Ineffective tissue perfusion
Hyperthermia
Acute pain
Anxiety
Impaired physical mobility
Planning
Overall goals:
Maintain adequate cerebral
perfusion
Remain normothermic
Be free from pain, discomfort, and
infection
Attain maximal cognitive, motor,
and sensory function
Nursing implementation
Health Promotion
Prevent car and motorcycle
accidents
Wear safety helmets
Acute Intervention
Maintain cerebral perfusion and
prevent secondary cerebral ischemia
Monitor for changes in neurologic
status
Ambulatory and Home Care
Nutrition
Bowel and bladder management
Spasticity
Dysphagia
Seizure disorders
Family participation and education
Expected Outcomes
Maintain normal cerebral perfusion
pressure
Achieve maximal cognitive, motor,
and sensory function
Experience no infection,
hyperthermia, or pain
 McCaffrey RJ (1997). "Special Issues in the Evaluation of Mild 
Traumatic Brain Injury". The Practice of Forensic
Neuropsychology: Meeting Challenges in the Courtroom. New 
York: Plenum Press. pp. 71–75. ISBN 0-306-45256-1.
Jump up^ "What is Head Trauma?". News Medical. 
Retrieved 2013-05-04.
^ Jump up to:a
 b
 "Head injury- first aid". MedlinePlus. 
Retrieved 2013-05-04.
Jump up^ "Head Injury (Brain Injury)". eMedicinehealth. 
Retrieved 2013-05-04.
Jump up^ name="Head Injury (Brain Injury)"
Jump up^ Carlson, Neil R. (2013). "Physiology of Behavior". In 
Campanella, Craig.Neurological Disorders. Pearson Education, 
Inc. pp. 526–527. ISBN 0-205-23939-0.

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Types of Head Injuries and Their Signs & Symptoms