THIS SEMINAR ON VENOUS DRAINAGE OF HEAD, FACE, NECK AND BRAIN ENCOMPASSES ALL THE POSSIBLE DETAILED EXPLANATION ALONG WITH DIAGRAMMATIC ILLUSTRATIONS OF THE SAME. APPLIED AND SURGICAL ANATOMY ALONG WITH RECENT MODALITIES HAS BEEN ADDED HEREIN..
3. Veins (vena) are blood vessels that carry blood
towards the heart.
Most veins carry deoxygenated blood from the
tissues back to the heart.
Exceptions are the pulmonary and umbilical veins.
3
6. Blood from veins
• It is dark red in colour.
• Blood flows steadily or gushes.
• Bleeding easier to control.
• Most veins collapse when cut.
• However bleeding from deep veins can be massive and hard to
control as arterial bleeding.
6
8. Veins are classified as:
• Superficial veins.
course is close to the surface of the body, and have no• Are those whose
corresponding arteries.
• Deep veins.
• Are deeper in the body and have corresponding arteries.
• Communicating veins.
• (Or perforator veins) are veins that directly connect superficial
veins to deepveins.
• Pulmonary veins.
• Are a set of veins that deliver oxygenated blood from the lungs
to the heart.
• Systemic veins.
• Drain the tissues of the body and deliver deoxygenated blood to
the heart.
8
11. All the venous drainage from the head and neck
terminate ????
11
12. Veins of head, face, neck and brain
Exterior of head Neck Brain
and face
*External Jugular *Diploic veins
*Cerebral
• Supratrochlear
• Supraorbital
vein
*DuralVenous
Sinuses*Internal Jugular
*Subclavian
*Brachiocephalic
• Facial vein
• Superficial temporal *Anterior Jugular
• Maxillary vein
• Pterygoid Venous
plexus
• Retromandibular
• Posterior auricular
• Occipital vein
12
13. • Venous drainage from the face is
entirely superficial.
• All the venous drainage from
the head and neck terminate in
the internal jugular vein which
join the subclavian vein to form
the brachiocephalic vein behind
the medial end of the clavicle.
• Two brachiocephalic veins unite
to form superior vena cava.
13
VENOUS DRAINAGE OF HEAD AND FACE
17. Supratrochlear and Supraorbital Vein
• Drains the scalp.
• Supratrochlear and supraorbital
vein starts on
the forehead from venous network
which connects to the frontal
tributaries of superficial temporal vein.
• Runs downward superficial to the
frontalis muscle, and joins at the
medial angle of the orbit to form the
Facial vein(angular vein).
17
18. The Facial Vein
primary superficial
• Coursing parallel to facial arteries
• valveless veins that provide the
drainage of the face.
• Commences at the side on the root
• Formed by the union of the
of the nose.
supraorbital and
supratrochlear veins.
18
19. Relationship between the Facial Vein and Facial Artery
• Facial vein lies posterior to the facial artery
and its course is less tortuous than the
artery.
• Facial vein is usually located more posterior
and superficial to the facial artery.
• Of surgical significance, is the fact that the
facial artery and vein are close to the
mandible in the region of the inferior
border.
• The only structure that separates the vessels
from the bone is the periosteum.
19
20. Tributaries Of Facial Vein
• Superior ophthalmic vein
• Veins of ala of nose
• Deep facial vein, from
pterygoid plexus
• Inferior palpebral
• Superior and inferior labial
(drains area of upper and
lower lip)
• Buccinators (drains area of
cheek)
• Parotid and masseteric veins
(drains area from parotid and
masseter)
• Below mandible it receives
submental , tonsillar , external
palatine and submandibular
vein
20
21. Superficial TemporalVein
• Drains the scalp.
• Begins in a widespread network
joined across scalp to contra-lateral
vein and with supratrochlear,
supraorbital, posterior auricular and
occipital veins, all draining same
network.
• Cross posterior root of zygoma &
enters parotid gland to unite with
maxillary vein to form
retromandibular vein.
23. Maxillary vein
the first part of• A short trunk accompanies
maxillary artery.
• It is confluence of vein from
plexus, passes back
pterygoid
between
sphenomandibular ligament and neck of
mandible, to enter the parotid gland and here
it unites with superficial temporal vein to
form retromandibular vein.
25. Retromandibular Vein
• Runs posterior to ramus of the
mandible within the substance of
parotid gland
• Superficial to external carotid
artery and deep to facial nerve
• Dividesinto an Anterior branch
going forwards to join with facial
vein and Posterior branch which
joins posterior auricular to
form external jugular vein
26. Posterior Auricular Vein
• Drains the scalp.
• Begins upon the side of neck,
in a plexus which
communicates with tributaries
of occipital vein and temporal
veins.
• Descends behind the auricle
and joins the posterior division
of retromandibular vein to
form external jugular vein.
• Receives mastoid emissary
veins from sigmoid sinus.
Infection here can be
dangerous or fatal from
retrograde thrombosis of
cerebellar and medullary
veins.
27. Occipital Vein
• Begins in posterior network in
scalp, pierce the cranial
attachment of trapezius, turns
into sub occipital triangle and
becomes deep.
• May follow occipital artery to
end in internal jugular vein; or
join posterior auricular &
hence external jugular vein.
28. Lingual Vein
Drains Tongue & Sublingual Region
3 branches:
• Dorsal lingualvein
• Deep lingual vein
• Sublingual vein
31. Tributaries:
1. Sphenopalatine v
2. Deep temporal v
3. Pterygoid v
4. Masseteric v
5. Buccal v
6. Dental v
7. Greater palatine v
8. Middle meningeal v
9. Inferior opthalmic v
32. b) SUBOCCIPITAL-
1. Located in suboccipital triangle
2. Receives blood from
1. Muscular veins
2. Transverse sinus
3. Occipital veins
4. Internal vertebral venous plexus
5. Condylar emmissary veins
3. Drains into vertebral veins
33. c) PHARYNGEAL VENOUS PLEXUS:
1. Located on posterolateral
region of pharynx.
2. Receives blood from-
Pharynx
Soft palate
Pre vertebral region
3. Drains into internal jugular and
facial veins.
34. Veins of the neck
The word "jugular" refers to the throat or neck.
It derives from the Latin ‘jugulum’ meaning throat or
collar bone.
The jugular veins are relatively superficial and not protected
by tissues such as bone or cartilage. This makes them
susceptible to damage. Due to the large volumes of blood that
flow though the jugular veins, damage to the jugulars can
quickly cause significant blood loss, which can lead to
hypovolemic shock and then death if not treated.
To go for the jugular is to attack a vital part that is
particularly vulnerable.
36. External Jugular Vein-
• External jugular vein largely drains
scalp and face, but also some
deeper parts.
• Formed by union of post division of
retromandibular vein & post
auricular vein.
• Begins near the angle of mandible,
just below the parotid gland and
drains into subclavian vein.
• Covered with platysma and
superficial fascia and separated
from sternocleidomastoid by deep
cervical fascia.
37.
38. TRIBUTARIES OF EXTERNAL JUGULAR VEIN
• Posterior division of
retromandibular vein
• Posterior auricular vein
• Posterior external jugular vein
• Transverse cervical vein
• Suprascapular vein
• Anterior jugular vein
• Occasionally joined by occipital
39. Anterior Jugular Vein
• Starts near the hyoid bone by confluence of superficial
submandibular veins.
• Descends between midline and anterior border of
Sternocleidomastoid turning laterally, low in neck, posterior
to this muscle but superficial to depressor of hyoid bone.
Joins external jugular vein or subclavian vein directly.•
• There are usually two anterior jugular veins, just above the
sternum, communicate by large transverse trunk, the venous
jugular arch
• Have no valves.
40. INTERIOR JUGULAR VEIN
• Begins atthe base of the skull in the posterior
compartment of jugular foramen collects
blood from brain.
• Immediately below the jugular foramen,
it is widened to form superior bulb of
internal jugular v,contained in jugular
fossa.
• Located post. to internal carotid artery
• At its lower end, at the junction with
subclavian vein , IJV is again widened to
form inferiorbulb
• Posterior to sternal end of clavicle, IJV
combines with subclavian vein to form
bracheocephalic vein.
It is a direct continuation of the sigmoid sinus.
41. TRIBUTARIES OF INTERNAL JUGULAR VEIN
Into Superior Bulb
Inferior petrosal sinus
Occipital vein
Pharyngeal veins
Common Facial vein
Lingual vein
Superior thyroid vein
Middle thyroid vein
43. RELATIONS
Surrounded By Accompanying Lymph Nodes
Relation To Internal Carotid Artery
C2: Posteriorly
C3: Posterolaterally
C4: Laterally
Vagus Nerve (CN X) Always
Situated Between The ICA And IJV
Anteriorly (I.E. Is Crossed By These
Structures)
Upper Third: Spinal Root Of Accessory
Nerve (CN XI)
Middle Third: Lower Root Of Ansa
Cervicalis
Lower Third: Sternocleidomastoid
Muscle, Tendon Of Omohyoid Muscle
Posteriorly (From Superior To Inferior As The
IJV Descends In The Neck)
Lateral Mass Of C1 (Atlas)
Middle Scalene Muscle
Anterior Scalene Muscle
Pleura Of Lung Apices
44. 36
• Each subclavian vein is a
continuation of the axillary
vein runs from the outer
border of the first rib to the
medial border of anterior
scalene muscle.
• From here it joins with the
internal jugular vein to form
the brachiocephalic vein (also
known as “innominate vein”).
The angle of union is termed
the Venous Angle.
• The thoracic duct drains into
the left subclavian vein, near
its junction with the left
internal jugular vein.
SUBCLAVIAN VEIN
46. RIGHT BRACHIOCEPHALIC VEIN
• 2.5 cm long
• Runs vertically downwards
• TRIBUTARIES are namely :
1. Vertebral vein
2. Internal thoracic vein
3. Inferior thyroid vein
4. First posterior intercostal vein
47. LEFT BRACHIOCEPHALIC VEIN
• 6 cm long
• Runs obliquely downwards and to the right behind the half of
manubrium sterni
• TRIBUTARIES are namely :
1. Vertebral vein
2. Internal thoracic vein
3. Inferior thyroid vein
4. First posterior intercostal
5. Left superior intercostal
6. Thymic and pericardial veins
48. SUPERIOR VENA CAVA
FORMED by union of right and left Brachiocephalic
veins.
Formed at lower border of the right first costal cartilage.
It pours blood into right atrium of the heart.
50. DIPLOIC VEINS
These veins occupy channels in diploe of
some cranial bones and are devoid of valves.
They are large with dilation at regular
interval; their thin wall is merely
endothelium. Absent at birth, begin to
develop at about 2yrs. They communicate
with meningeal veins, Dural sinuses &
pericranial veins.
Frontal diploic vein
An anterior temporal diploic vein
A posterior temporal diploic vein
An occipital diploic vein
51. CEREBRAL
VEINS
They are divided into external
& internal groups according to
the outer surface or inner parts
of hemispheres they drain into.
External cerebral veins drain
into superior saggital sinus
Internal cerebral veins drain
into great cerebral veins of
Galen
52. Dural Venous Sinuses
• They are blood-filled spaces situated between
the layers of the duramater,
• Lined by endothelium,
• Walls are thick and composed of fibrous
tissue.
• They have no muscular tissue.
• They have no valves.
53.
54. • They receive blood from:
1.The Brain
2.The Meninges
3.Bones of the skull
• CSF is also poured in some of them.
55. Types of Dural sinuses
• There are 23venous sinuses, of which –
•8 are PAIRED
•7 are UNPAIRED
58. SUPERIOR AND INFERIOR PETROSAL SINUS
They are small and situated
on the superior and inferior
borders of the petrous part of
the temporal bone on each
side.
Each superior sinus drains
the cavernous sinus into the
transverse sinus.
Each inferior sinus drains
the cavernous sinus into the
internal jugular vein.
59. TRANSVERSESINUS
They are paired and begin
at the internal occipital
protuberance.
The right sinus is direct
continuation of the superior
sagittal sinus.
The left is continuation of the
straight sinus.
They drain from
the confluence of
sinuses to the sigmoid
sinuses, which ultimately
connect to the Internal
jugular vein.
60. SIGMOID SINUS
They are a direct continuation
of the transverse sinuses.
Each sinus turns downward and
medially and grooves the mastoid
part of the temporal bone.
Here it lies behind the mastoid
antrum.
It then turns downward through the
posterior part of the jugular foramen
to become continuous with the
superior bulb of the internal jugular
vein.
61. SUPERIOR SAGITTAL SINUS
It occupies the upper fixed
border of the falx cerebri.
It begins in the front at the
foramen cecum where it receives
a vein from the nasal cavity.
It runs backward, grooving the
vault of the skull and at the
internal occipital protuberance it
deviates to one side and becomes
continuous with the transverse
sinus.
62. It communicates through small
openings with 2 or 3 venous lacunae
on each side.
Numerous arachnoid villi and
granulations project into these lacunae
which also receive the diploic;
emissary and meningeal veins.
It receives the superior cerebral veins .
At the internal occipital protuberance
it is dilated to form the confluence of
the sinuses which is connected to the
opposite transverse sinus and receives
the occipital sinus.
63. INFERIOR SAGITTAL
SINUS
It occupies the free lower margin of
the falx cerebri.
It runs backward and joins the great
cerebral vein which is formed by the
union of the two internal cerebral
veins at the free margin of the
tentorium cerebelli to form the straight
sinus.
It receives cerebral veins from the
medial surface of the cerebral
hemisphere.
64. STRAIGHT
SINUS
It occupies the line of junction
of the falx cerebri with the
tentorium cerebelli.
It is formed by the union of
the inferior sagittal sinus with
the great cerebral vein.
It ends by turning to the left (
sometimes to the right ) to
form the transverse sinus.
65. OCCIPITAL SINUS
It is a small sinus occupying the
attached margin of the falx
cerebelli.
It communicates with the
vertebral veins near the foramen
magnum.
Superiorly it drains into the
confluence of sinuses.
66. CAVERNOUS
SINUS
Situated in the middle cranial
fossa on each side of the body
of the sphenoid bone.
Each sinus extends from the
superior orbital fissure in front
to the apex of the petrous part
of the temporal bone behind.
The 3rd & 4th cranial nerves and
the ophthalmic & maxillary
divisions of the trigeminal nerve
run forward in the lateral wall of
this sinus.
The internal carotid artery, its
sympathetic nerve plexus and
abducent nerve run forward
through it.
68. COMMUNICATIONS
Transverse sinus through superior
petrosal sinus
Internal jugular vein trough inferior
petrosalsinus
Pterygoid plexus through emissary
veins
Facial vein through superior
opthalmicvein
Cavernous sinus of opposite side
through anterior and posterior
intercavernous sinus.
69. EMISSARY VEINS
• Cranial venous
sinuses communicate
with veins outside
the skull through
EMISSARY VEINS
• These comminication
s help to keep the
pressure of blood in
the sinuses constant
72. Facial vein
• Applied Importance:
• Facial veins have no valves and it connects
to cavernous sinus by 2 routes.
• 1.) via ophthalmic vein or supraorbital vein.
• 2)Via deep facial vein to pterygoid plexus
and hence to cavernous sinus.
• Thus infective thrombosis of facial vein
may extend to intracranial venous
sinuses.
73
73. • Anatomical variations of the facial veins and arteries are of
great importance for facial transplantations because they are
the main vascular pedicles that will be connected to the
patient. A good arterial inflow and venous outflow are
essential for the free flap survival.
74. RETROMANDIBULAR VEIN(RMV)
74
• Formation of the RMV by union of the superficial temporal
vein and maxillary vein mostly occurs at a level higher than
the passage of the main trunk and branches of the FN,
where they laterally pass to the vein.
• In open surgical reduction of mandibular condyle fractures,
the FN and its branches can be localized by using the
superficial temporal veins and the RMVs as a guide, even in
abnormal course of the nerve.
75. Lateral view of right facial nerve (FN) giving superior division
(SD) and inferior division (ID); each of them pass through a
separate ring in the retro-mandibular vein (RMV).
75
77. 77
In 13%, major divisions (temporal and facial) are independent;
in 11%, anastomoses occur between rami of the temporal division;
in 22%, connections occur between adjacent rami from the major
divisions;
in 21%, anastomoses representing a composite of those in the11% and
22% categories occur;
in 12%, proximal anastomoses occur within the temporal component,
as well as distal interconnection between the latter and the cervical
component;
in 9%, two anastomotic rami connect the buccal divisin of the cervical to
the zygomatic part of the temporal;
in 5%, a transverse ramus, from the trunk of the nerve, contributes to
the buccal ramus formed by anastomosis between the two major
divisions;
in 7% richly plexiform communications occur, especially within the
temporal portion of the nerve.
78. Communications of the Pterygoid plexus
1. With Inferior opthalmic vein through the inferior
orbital fissure
2. With Cavernous sinus through emissary veins
3. With facial vein through the deep facial vein
79. Applied anatomy:
PSA block
-haematoma
-black eye
Serves as media for spread of external
infection to the cavernous sinus
80. A black eye is bruising around the eye commonly due to an
injury to the face rather than to the eye. The name is given due
to the color of bruising.
Although most black eye injuries are not serious, bleeding
within the eye, called a hyphema, is serious and can reduce
vision and damage the cornea.
Hyphema can cause rapid increase eye pressure and vision
loss from glaucoma if left untreated.
BLACK EYE
81. Abnormaly dilated, tortuous veins produced by
prolonged, increase intraluminal pressure. Small
purplish or blue-black round swellings under the
tongue with age and are known as “caviar lesions”
No treatment is indicated for lingual varices..
Lingualvaricosity
80
82. EXTERNAL JUGULAR VEIN(EJV)
• EJV is examined to assess the venous pressure; the
right atrial pressure is reflected in it because of
absence of valves.
• Vein is visible through skin and can be made more
prominent by blowing with mouth and nostrils closed.
• Normal JVP is 5 to 8 cm.
• Vene puncture performed on this vein
• Surgical division of sternocleidomastoid muscle
requires special care of the vein
• Increased venous pressure indicates congestive cardiac
failure
83. Visualization (LEWIS METHOD)
The veins of the neck, viewed from in
front.
The patient is positioned under 45°,
and the filling level of the jugular vein
determined. Visualize the internal
jugular vein when looking for the
pulsation. In healthy people, the filling
level of the jugular vein should be less
than 3 centimetres vertical height
above the sternal angle.
JUGULAR VENOUS PULSE
82
84. Jugular venous pulse (JVP)
• Determine activity of
atrium
• Seen better then felt
• Preferable over EJV
• Elevation of JVP
indicative of
cardiac failure
84
85. A man with severe
congestive cardiac failure
with marked jugular
venous distension.
External jugular vein
marked by arrow.
84
86. INTERNAL JUGULAR VEIN(IJV)
Infection from middle ear spreads to IJV
Surgical removal of deep cervical nodes can
puncture IJV
Easy accessibility between two heads of
sternocleidomastoid muscle for introduction of
cannula
86
87. Thrombophlebitis can occur by spread of
infection in caverous sinus
Systolic thrill felt over the vein in mitral
stenosis
During CCF dilatation of vein occur
In congestive cardiac failure, venous
pressure is markedly increased, the IJV is
dilated and engorged with blood.
87
88. • The deep cervical
lymph node lie on
IJV. These nodes
become adherant
to veins in
malignancy or in
T.B. Therefore
during such
operations the vein
is also resected.
88
89. Applied anatomy:
1. Special care required
to preserve the vein
during surgical
treatment of WRY
NECK
ANTERIOR JUGULAR VEIN(AJV)
89
90. Jugular phlebectasia in children
• Jugular phlebectasia is a congenital dilatation of
jugular vein which appears as a soft, compressible
mass in the neck only during straining or crying. It
should be differentiated from laryngocele, cysts and
tumors of neck which may also appear during
straining.
• Ultrasonography (US) and computerized tomography
(CT) are diagnostic methods to distinguish the
pathology
• More common in internal jugular vein.
89
91. DURAL SINUS THROMBOSIS
Common causes of dural venous sinus
include
fractures
head and neck infections, head
or intracranial hematomas either
thrombosis
injury,skull
by direct
compression of the sinus or endothelial damage within
the sinus can cause the activation of coagulation system
resulting in sinus occlusion.
90
92. 91
Brain cells contain an abundance of thromboplastin that is
released after injury inducing an hypercoagulable state
leading to destruction of platelets & erythrocytes followed by
thrombus formation.
Most common thrombosed sinuses are tranSverse,cavernous
& superior saggital sinus
Clinical symptoms
headache, papilloedema, impairedconsciousness, vomitting.
93. Metastasisof tumour cells to
dural sinuses
Tha basilar and occipital sinuses communicate through the
foramen magnum with the internal venous plexuses.
Because these venous channels are valveless,
compression of the thorax, abdomen, or pelvis as occurs
during heavy coughing and straining may force venous
blood from these regions into vertebral venous system and
from it into dural venous sinuses.
As a a result, pus in the abscesses and tumour cells in
these regions may spread to vertebrae and brain.
92
95. Thrombosis caused by sepsis in the danger
area of face, nasal cavity, paranasal sinuses give
rise to :
Nervous symptoms:
1)severe pain in the eye and forehead in the
area of distribution of opthalmic nerve.
2)Involvement of 3rd, 4th and 6th nerve resulting
in paralysis of muscle supplied.
94
96. Venous symptoms:
1) Marked edema of eyelid, cornea & exopthalmos due to
congestion of orbital vein.
Carotid and cavernous communication: because of
peculiar relationship of cavernous sinus to internal carotid
artery a communication may occur between the two as a
result of injury.
When this happens the arterial pressure is communicated
through the sinus to vein of orbit & as a result the eye
become prominent & pulsate with each heart beat(
pulsating exopthalmos)
95
97. Emissary veins are connection between the extracranial scalp veins and
the diploic and intracranial venous systems. These veins are valveless
and therefore can transmit infection from the extracranial to the
intracranial compartment.
The meningeal veins are epidural veins that lie within the dura draining
the falx cerebri, the tentorium, and the cranial dura. They run in shallow
grooves on the inner table of the skull to communicate with the dural
sinuses or traverse extracranially to the pterygoid plexus in the deep
face or vertebral plexus around the cervical spine.
The diploic veins are small irregular endothelial-lined channels coursing
between the inner and outer tables of the skull. These communicate
with the extracranial venous system, the meningeal veins, and the dural
sinuses. They are rarely seen using angiography unless enlarged, as in
the case of an arterial-venous malformation.
Communication between extracranial and intracranial veins
96
98. 97
Danger triangle of the face
consists of the area from the corners
of the mouth to the bridge of
the nose, including the nose
and maxilla.
The presence of loose areolar tissue
containing the emissary veins allows
the spread of retrograde infections
from the nasal area to spread to
the brain causing
cavernous sinus thrombosis,
meningitis
brain abscess.
99. • Sinus pericranii (SP) is a rare disorder
characterized by a congenital (or occasionally,
acquired) epicranial venous malformation of
the scalp.
• Sinus pericranii is an abnormal
communication between the intracranial and
extracranial venous drainage pathways.
• Treatment of this condition has mainly been
recommended for aesthetic reasons and
prevention of hemorrhage.
Sinuspericranii
98
100. VASCULAR
MALFORMATIONS
Described as abnormalities of
blood and lymphatic vessels,
many hemangiomas,
vascular malformations, like
present at birth but do not
undergo proliferation and
do not spontaneously
involute.
• Do not regress with age and
may
severe
be associated with
or life threatening
haemorrhage.
• A large venous malformation
appears as bluish, soft,
compressible lesion,no bruit
or pulsation is present.
99
101.
102. • TREATMENT: May be treated with
sclerotherapy as well as direct injections of
sodium morrhuate, boiling water, alcohol &
ethibloc.
• Combined application of sodium tetradecyl
sulfate sclerothearpy & conservative ablative
surgery when larger lesions are involved.
100
103. VENOUS MALFORMATIONS
Low flow lesion
Composed of ectatic venous channels that will continue to grow throughout
the patient’s lifetime. The overall incidence of venous malformation is about
1 in 10 000.
These lesions commonly occur in the head and neck area with a predilection
for the oral cavity, airway, and muscle groups.
Several syndromes can be
associated with venous
malformations Including Blue
Rubber Bleb Nevus Syndrome,
Glomuvenous Malformation
(Associated With Glomulin
Mutation), Sturge-weber
Syndrome, Klippel– Trenaunay
Syndrome etc.
104. The lesions vary in colour depending on depth of involvement, from undetectable
colour differences to deep purple. The lesions fill with dependency and are
compressible, which helps to distinguish them from lymphatic malformations on
physical examination. Patients may present with complaints of pain and swelling
and this is usually related to clot formation either from trauma or venous stasis.
Venous malformations have a propensity to occur in muscle groups but can also
involve skin and mucosa. Areas frequently involved in the head and neck are
masseter, temporalis, tongue musculature, as well as oral and airway mucosa.
MRI remains the diagnostic modality of choice to assess extent and plan
treatment for these lesions.
105. Telangiectasias or angioectasias are small dilated
blood vessels near the surface of the skin or
mucous membranes,
measuring between 0.5 and 1 millimeter in
diameter.
They can develop anywhere on the body but are
commonly seen on the face around the nose,
cheeks, and chin.
Some telangiectasia are due to developmental
abnormalities that can closely mimic the behaviour
of benign vascular neoplasms.
They may be composed of abnormal aggregations
of arterioles, capillaries, or venules.
Telangiectasia
101
106. • Telangiectasias on the face are often treated with
a laser.
• Laser therapy uses a light beam that is pulsed
onto the veins in order to seal them off, causing
them to dissolve.
• These light-based treatments require adequate
heating of the veins.
• These treatments can result in the destruction
of sweat glands, and the risk increases with the
number of treatments.
102
108. • Indication: Tointroduce or replace fluids in
circulation
• Complications:
1.Overloading
2.Thrombophlebitis
3.Haematoma
4.Infection
5.Air embolism
108
109. Venesection ( venous cut down)
• As an alternative to venipuncture in
critically ill patients in need of
vascular access, and in whom
venipuncture may be difficult
• An emergency procedure in which the
vein is exposed surgically and then a
cannula is inserted into the vein under
direct vision.
• It is used to get vascular access in
trauma and hypovolemic shock
patients when peripheral cannulation
is difficult or impossible.
• Sites for venesection great saphenous
vein (most common, usually at the
ankle) ,basilic vein, cephalic vein.
110. • The skin is cleaned, draped, and
anesthetized.
• The greater saphenous vein is identified
on the surface above the medial
malleolus, a full-thickness transverse skin
incision is made, and 2 cm of the vein is
freed from the surrounding structures.
• The vessel is tied closed distally, the
proximal portion is transected
(venotomy) and gently dilated, and a
cannula is introduced through the
venotomy and secured in place with a
more proximal ligature around the vein
and cannula.
• An intravenous line is connected to the
cannula to complete the procedure.
111. Free flap transplantation is an important method for the surgical resurfacing of soft
tissue defects in all parts of the body. The arterialized venous flap technique depends
on the use of two veins in the skin flap that are used respectively for arterial flow and
venous drainage. Survival depends on the size of the flap, arterial inflow, and venous
outflow. For good results, an arterialized venous flap should be designed to contain
most of the venous network in the centre, the arterial inflow should be anastomosed
to one afferent vein, and two or more efferent veins should drain the arterialized
venous flap.
ARTERIALIZED VENOUS FLAP
Ozek et al. studied the changes occurring in the
venous system after its arterialization and showed
that the thickening and hypertrophy of the vessel
walls indicated adaptive changes to withstand the
increased stress experienced in their new roles as
arterial conduits.
112. Central venous line placement
A central venous catheter is a special IV line that is inserted into a large vein in the body.
Several veins are used for central venous catheters including those located in the
shoulder (subclavian vein), neck (jugular vein), and groin (femoral vein). It is used to
administer medication or fluids, obtain blood tests (specifically the "mixed venous
oxygen saturation"), and directly obtain cardiovascular measurements such as the
central venous pressure.
113. INDICATIONS AND USES
Monitoring of the central venous pressure (CVP) in acutely ill patients
to quantify fluid balance
Long-term Intravenous antibiotics
Long-term Parenteral nutrition especially in chronically ill patients
Long-term pain medications
Chemotherapy
Seldinger Technique
1.desired vessel or cavity is punctured
using a trocar (hollow needle).
2.soft curved tip guide wire is then
inserted through the trocar and advanced
into the lumen.
3.guidewire is held secured in place whilst
the introducer trocar is removed.
4.large-bore sheath/cannula/catheter is
passed over the guidewire into the
lumen/cavity.
5.guidewire is withdrawn leaving the
introducer sheath in situ through which
catheters and other medical devices can
be introduced.
114. Air Embolism
• External jugular vein pierces the investing
layer of deep cervical fascia
• The margins of the vein get adherent to the
fascia
• So, if the vein gets cut, it can not close and air
enters into it causing air embolism
114
117. Bibliography
• B.D. Chaurasia’s Human Anatomy Volume 3
• Gray’s Textbook of Anatomy
• Hine, Levy, Shafer’s Textbook of Oral
Pathology
• Hutchinson’s Textbook of Medicine
• Peterson’s Textbook of Oral Surgery
• Internet Sources
117
Chronic Cerebrospinal Venous Insufficiency (CCSVI) is a term used to describe a potential reduction in blood flow in the major veins that drain blood from the brain and spinal cord over a prolonged period. The condition was first described as being a possible contributor to MS by Italian physician, Dr Zamboni in 2009. The proposed treatment for CCSVI, sometimes referred to as “liberation therapy,” is an angioplasty procedure, which involves opening blocked or narrowed veins by inflating a small balloon or inserting a stent to allow for better blood flow and improve drainage of blood from the brain. In May 2012, the U.S. FDA issued a safety communication about procedures to treat CCSVI in people with MS.