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" Obstetrics emergency 2 "
1. Diaa Mohammad Srahin
5th year Medical Student
Al-Quds University
Obstetrics & Gynecology
March / 2018
Dr. Bassam Al-Akhdar
2.
3. Uterine rupture
disruption of the uterine musculature through all of
its layers.
It is rare, occurring with an incidence of 0.03–0.3 per cent
Complete separation of the wall of the pregnant uterus with or
without expulsion of the fetus
It occurs mainly in association with a previous Caesarean
section.
This is because scar tissue does not have the same inherent
strength as myometrium. However, scarring can also exist as a
result of previous uterine surgery, such as a surgical evacuation
of retained products of conception resulting in a perforation.
4. The vast majority of cases occur during labour, usually
during late first stage or the active second stage.
5. Risk factors for uterine rupture
Previous Caesarean section
Previous uterine surgery
Induction and augmentation of labour
High parity
Macrosomic fetus
Placenta percreta
Fetal version, e.g. breech extraction
Congenital uterine anomaly, e.g. unicornuate
uterus
6. Clinical presentation
The most common findings are :
Vaginal bleeding
Abdominal pain
Loss of fetal electronic heart rate signal
Loss of station of fetal head
Hematuria may be present if the uterus has ruptured
into the bladder
7.
8. Diagnosis
The diagnosis of uterine rupture is typically made at
laparotomy by observation of complete disruption of
all uterine layers with active bleeding.
11. Uterine inversion
Rare complication occurring during the third stage of
labour.
Incidence: 1:2000- 1: 6000
The uterine fundus descends either the uterine cavity,
through the cervix, and very rarely beyond the
introitus .
12. It is caused by traction on the umbilical cord before
the placenta has separated and can occur after vaginal
deliveries or Caesarean section.
Associated factors are a fundal placenta, a short cord
and a morbidly adherent placenta.
Risk factors : Myometrial weakness (most common)
and previous uterine inversion.
13. Diagnosis
The prolapsed uterus stretching the cervix causes
vagal stimulation, thus the woman will demonstrate
signs of cardiovascular collapse and shock.
Although haemorrhage is commonly present, the
symptoms will be out of proportion to the estimated
blood loss.
The inverted uterus may be obvious at the introitus, but
other signs include the lack of a palpable uterus in the
abdomen
14. Management
Resuscitate the patient using the structured ABC approach.
It is very important not to remove the placenta if it is still attached as this
will increase the bleeding.
Immediately replace the uterus through the cervix by manual compression.
If that fails, hydrostatic pressure can be applied by pouring warmed saline
into the vagina, usually via a silc cup ventouse.
Tocolysis may be helpful to relax the cervical ring.
Surgery, to reposition the uterus from above, should be used as a last
resort.
After replacement, uterine contraction is maintained with an oxytocic.
19. Sudden maternal collapse
Maternal collapse refer to :
an acute event involving the cardiorespiratory systems
and/or brain, resulting in a reduced or absent conscious
level (and potentially death), at any stage in pregnancy and
up to six weeks after delivery
Sudden collapse, as in non-pregnant adults, occurs due to
a multitude of reasons. Some will be benign, such as a
vasaovagal attack (simple faint) or an epileptic fit in a
known epileptic, but other causes are life threatening
24. There is no specific treatment
cardiovascular and respiratory resuscitation and
correction of the coagulopathy
25. Pulmonary embolism
leading cause of death among pregnant women in the
developed world
Result of DVT- emboli travel through the venous system to
the lungs
the source is most commonly in the lower extremities or
pelvis
more common in the puerperium (two thirds)
Treat (stabilization, oxygen, hemodynamic support) and
evaluate simultaneously
26. Findings
Symptoms include chest pain and dyspnea (80%).
Physical and imaging findings include:
tachypnea (90%)
CXR often normal
ABG showing low pO2 (but often in normal range)
EKG may show tachycardia
Right axis deviation but usually normal
27.
28. Treatment
Urgent resuscitation using the structured ABC approach
is needed. If PE is suspected, anticoagulation should be
instituted
Low-molecular-weight heparin is the treatment of
choice for PE in pregnancy
UFH is alternative if LMWH is not available or
contraindicated
Warfarin is contraindicated because of teratogenicity
( embryopathy, miscarriage, stillbirth)
30. Umbilical cord accidents
Cord presentation is defined as the presence of
umbilical cord below the fetal presenting part when
the membranes are intact.
Cord prolapse is the presence of the cord below the
presenting part when the membranes are ruptured.
31.
32. Incidence: 1/500 deliveries
occurs when the fetal presenting part does not fit well
into the maternal pelvis, giving ‘space’ for the cord to
prolapse when the membranes rupture
34. Diagnosis
Most commonly, it is diagnosed by
seeing the cord at the introitus.
or feeling it during a vaginal
examination.
an abnormal fetal heart rate
pattern.
35. Management
Immediate management aims to minimize the pressure of
the fetal presenting part on the cord.
This is achieved by moving the woman on to all fours with
the head down.
applying pressure vaginally to push the presenting part out
of the pelvis, or by filling the bladder with 500 mL of
saline.
Emergency Caesarean section is required unless the cervix
is fully dilated and an assisted vaginal delivery can be safely
and easily performed.
36. move the woman on to all fours
with head down.
Apply pressure vaginally to push
the presenting part out of the
pelvis.
Emergency Caesarean section is
required unless the cervix is fully
dilated
Management
37. Fetal outcome
Depends upon the gestation, other complicating factors
such as IUGR and for how long the cord has been
compressed.
With a term baby and a prompt diagnosis in hospital-
excellent
Total cord compression ˃ 10 minutes - cerebral damage
If continued for around 20 minutes - death
38. Shoulder dystocia
“The need for additional obstetric
maneuvers to release the shoulders
after gentle downward traction has
failed”.
results in excessive morbidity for
both mother and fetus.
Incidence: 0.6 % in the UK
>50% occur in normal weight
infants (less than 4 kg)
41. Diagnosis
Diagnosis is usually obvious when the shoulders fail
to deliver during the next contraction after delivery of
the head.
It is sometimes preceded by the ‘turtle sign’, which is
the head appearing to be pulled back on to
the perineum at delivery.
Uterine rupture, or a tear in the uterus, usually happens due to a previous uterine injury.
A unicornuate uterus represents a uterine malformation where the uterus is formed from one only of the paired Müllerian ducts while the other Müllerian duct does not develop or only in a rudimentary fashio
or the feeling of a ‘dimple’ in the uterinefundus on abdominal examination.
There is no specific treatment for amniotic fluid embolism, and initial emergency management is the same as for any other cause of sudden maternal collapse with cardiovascular and respiratory resuscitation and correction of the coagulopathy
Admit the patient into the intensive care unit
Treatment is supportive and includes :
cardiopulmonary resuscitation, mechanical ventilation, Dopamine, steroids, correction of acidosis, blood transfusion, and blood component therapy to correct DIC.
If the mother is undelivered, a decision must be made as to whether the baby can be delivered
Puerperium is defined as the time from the delivery of the placenta through the first few weeks after the delivery. This period is usually considered to be 6 weeks in duration
more common in the puerperium, but it can occur at any time in the antenatal and post-natal period
The fact that PE remains such a threat is a clear indication of the difficulty in diagnosing it
there are 2 patients at risk rather than 1.
overdiagnosis results in unnecessary, dangerous treatments that jeopardize both patients and makes the pregnancy and delivery far more complicated.
the usual imaging modalities, which we use without a second thought in nonpregnant patients, suddenly become more complicated in pregnant patients .The decision to use imaging modalities that produce radiation exposure in pregnant patients is difficult because of concerns with teratogenicity.
Presentation varies from mild dyspnea and tachycardia to cardiopulmonary collapse
but no single symptom predominate because thrombi location varies
Spiral CT scan of the chest is the best initial test for suspected PE
Pulmonary angiograph is the definitive diagnostic method- most common indication is a negative spiral CT scan in a high risk and symptomatic patient
Neither clinical judgment nor clinical decision rules that are commonly used in nonpregnant patients have proven to be effective for diagnosing pregnant patients with PE. For example, D-dimer levels rise gradually during pregnancy and then drop in the immediate postpartum period but do not return to normal until 4-6 weeks postpartum. A normal D-dimer level appears to have a high negative predictive value in patients with a low clinical suspicion for VTE,
but false-positive levels are very common.
Ventilation Perfusion ScintigraphyThe negative predictive value of a normal VQ scan in pregnant patients is excellent, making this a useful first test for evaluating PE. However, 21% of women in the study had nondiagnostic scans, necessitating additional imaging tests
Pulmonary Computed Tomographic AngiographyA major benefit of CTA over other imaging modalities is that it often provides alternative pulmonary diagnoses
PE can be a cause of sudden cardiorespiratory collapse. In this situation, diagnosis and management should occur simultaneously.
Low-molecular-weight heparin is the treatment of choice for PE in pregnancy, as it can be continued on an outpatient basis.
Warfarin is contraindicated because of teratogenicity.
Intravenous unfractionated heparin is preferred in patients with renal failure and if urgent reversal of anticoagulation is anticipated (eg, high risk for bleeding or surgery is anticipated).
Thrombolytic drugs (eg, 100 mg tPA over 2 hours) can be considered in patients who are hemodynamically unstable or in patients with intractable hypoxemia. In these patients, the benefit is likely to outweigh the minor risks for bleeding and fetal loss.
an abnormal fetal heart rate pattern may suggest it, as compression of the umbilical veinbetween the presenting part and the pelvis, reducesor stops the fl ow of oxygenated blood to the fetus,causing deep variable decelerations, then bradycardiaif the situation is not relieved
Immediate management aims to minimize the pressure of the fetal presenting part on the cord.
This is achieved by moving the woman on to all fours with the head down.
applying pressure vaginally to push the presenting part out of the pelvis, or by filling the bladder with 500 mL of saline.
Emergency Caesarean section is required unless the cervix is fully dilated and an assisted vaginal delivery can be safely and easily performed.
There should be minimal handling of the cord, as this causes spasm which will worsen blood flow.
if the cord is beyond the introitus it should be replaced into the vagina to keep it warmer.
Second stage of labor
Head has delivered
No further delivery of body
Usually ass with fetal shoulders in the anterior-posterior plane, with anterior shoulder impacted behind the pubic symphasis