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Basic EKG and Rhythm
      Interpretation
 William M. Vosik M.D.
Milot, Haiti – January 8-12, 2012
Objectives:
1. Describe the basic conduction system
   within the heart
2. Learn basic rhythm strip analysis
3. Learn basic EKG morphology
4. Be able to recognize normal and
   abnormal rhythms and EKGs
Course Outline
• Part 1
  – Basic ECG and Arrythmias
• Part 2
  – “Heart block” and Pacemakers
• Part 3
  – Ischemia, Injury and Infract
Conduction System of the Heart
• Sinoatrial node (SA node): a small group of cells
  that function as the natural pacemaker of the
  heart (at 60-100 bpm)
• Atrioventricular node (AV node): a small group of
  cells that:
  – Slows the conduction impulse from atria to ventricles
    to allow for ventricular filling
  – Functions as the backup pacemaker if SA node fails
    (40-60 bpm)
  – Screens rapid atrial impulses to protect the ventricles
    from dangerously fast rates
Conduction System of the Heart
• Bundle of His: short bundle of nerve fibers at the
  bottom of the AV node, leading to the bundle
  branches
• Bundle branches: bundles of nerve fibers
  located along the septum that carry the impulse
  into the right and left ventricles
  – Right bundle branch: travels along the right side of
    the septum and carries impulse to the RV
  – Left bundle branch: travels along the left side of the
    septum and carries impulse to the LV
• Purkinje Fibers: hair-like fibers that spread
  across endocardial surface of both ventricles
  and rapidly carry impulse to the ventricular
  muscle cells
Conduction System of the Heart
1. Impulse originates in SA node and spreads
   through both atria simultaneously
2. Impulse is delayed in AV node so atria have
   time to contract and contribute to ventricular
   filling before ventricles contract
3. Impulse travels through Bundle of His, down
   both bundle branches into the ventricles and
   through the Purkinje fibers to the ventricular
   myocardium
Conduction System of the Heart

Right atrium         Left atrium




                               Left ventricle



Right ventricle
What am I looking for?
• 1. What is the rate? (fast, slow or normal)
• 2. What is the rhythm? (regular or
  irregular)
• 3. Is there a P wave before every QRS
  complex? (is it sinus rhythm or not)
• 4. Is the QRS complex wide or narrow?
Rhythm Strips
• Focus only on the rate and rhythm
• Evaluate each strip for:
   – Heart rate
   – Rhythm: regular/irregular
   – Location and morphology of P waves
   – QRS status
• *Cannot diagnose acute MI on rhythm strip!
EKG (electrocardiogram)
A picture of the electrical conduction through the heart
• P wave (atrial depolarization)
• QRS complex (ventricular depolarization)
    – Q wave: first negative deflection from baseline
    – R wave: positive deflection from baselinie
    – S wave: negative deflection following an R wave
• T wave (ventricular repolarization)
Intervals
• PR Interval: AV conduction
  time; the time for impulse to
  travel SA node atria AV
  node ventricles
    – Measured from beginning of P
      wave to beginning of QRS.
    – Normal= 120-200 ms (0.12-
      0.20 sec )
    – >200 ms: AV block
• QRS Duration: time for
  conduction to travel through LV
  and RV
    – Measured from beginning to
      end of QRS complex (QRS
      width)
    – Normal= < 120 ms (0.12 sec)
Intervals
•    ST segment: early
    repolarization phase; should be
    at baseline; elevation or
    depression indicates
    injury, ischemia or infarct
    – From end of S wave to beginning
      of T wave
    – More to come on this later…
• QT Interval: the time for both
  ventricular depolarization and
  repolarization
    – Dependent upon rate
Rate
• How fast the heart is going
  – How many times the heart beats in 1 minute
    (bpm)
  – A “beat” is a ventricular contraction or a QRS
    complex.
• Only reliable if rhythm is regular; if
  irregular then rate is just an estimation
Rate
• ON RHYTHM STRIP:
  – If paper speed is 25 mm/sec (standard), the
    vertical lines at top edge of paper represent 3
    second intervals
        – 1 small box = 0.04 sec
        – There are 5 small boxes within each large box
        – 1 large box = 0.20 sec
Rate
• ON RHYTHM STRIP:
 – Count the number of QRS complexes in a 6
   second strip and multiply by 10
Rate
• ON EKG:
  – Measured between two
    consecutive QRS waves
    (R-R interval)                                 300 150 100 75   60
  – Rule of 300’s:
     • Count the # of large boxes
       between R waves and
       divide into 300
     • Find an R wave on a dark
       line. The first big box
       would be 300, the second
       150, 100, 75, 60 bpm.*




                      *This applies only to EKGs printed at 25 mm/sec (standard)
Rhythm
• Regular: equal distance between R waves

• Irregular: varying distances between R waves
  – Regularly irregular: R waves occur in a pattern
  – Irregularly irregular: totally irregular without any
    pattern

• Mark out R-R intervals on a separate piece of
  paper and hold over EKG to see if all R-R’s map
  out
Arrhythmias…
       What you need to know:


1. Normal or Abnormal?
2. Name the Arrhythmia
3. Treat the Patient with the Arrhythmia
Basic Rhythms and Arrhythmias
•   Normal Sinus Rhythm   • Supraventricular
•   Sinus Tachycardia       Tachycardia (SVT)
•   Sinus Bradycardia     • Atrial Fibrillation (AF)
•   Sinus Arrhythmia      • Atrial Flutter
•   Sinus Arrest          • Ventricular Tachycardia
•   Ectopy (PACs, PVCs)     (VT)
                          • Torsades de Pointes
                          • Ventricular Fibrillation
                            (VF)
Normal Sinus Rhythm
•   Rate: 60-100 bpm
•   Rhythm: regular
•   P -> QRS -> T
•   Intervals: All normal
Sinus Tachycardia
•   Rate: >100 bpm
•   Rhythm: regular
•   P -> QRS -> T
•   Intervals: All normal
Sinus Tachycardia
Sinus Bradycardia
•   Rate: <60 bpm
•   Rhythm: Regular
•   P -> QRS -> T
•   Intervals: All normal
Sinus Bradycardia
Sinus Arrhythmia
•   Rate: Any rate, but usually 60-100 bpm
•   Rhythm: irregular
•   P -> QRS -> T
•   Intervals: All normal
Sinus Arrhythmia
Sinus Pause/Arrest
• A period of time where there is no heart beat (flat line)
   – Somewhat interchangeable, but technically…
       • Just one impulse fails to form: sinus pause
       • More than one impulse in a row fails to form: sinus arrest
• Rate: can be normal or bradycardia
• Rhythm: irregular due to absence of sinus node
  discharge (evaluate baseline rhythm independently)
Ectopy
• Premature atrial contractions (PACs)
  – Irritable focus in atria fires before next sinus
    impulse is due
  – A compensatory pause will follow an ectopic beat
  – Rate: Normal
  – Rhythm: Irregular because of the PAC, otherwise
    baseline rhythm is regular
Ectopy
• Premature ventricular contractions (PVCs)
   – Early beat followed by compensatory pause
   – Rhythm: Irregular because of the PVC, otherwise baseline
     rhythm is regular
   – QRS: Wide
PVCs
More Advanced Arrhythmias
•   Supraventricular Tachycardia (SVT)
•   Atrial Fibrillation (AF)
•   Atrial Flutter
•   Torsades de Pointes
•   Ventricular Tachycardia (VT)
•   Ventricular Fibrillation (VF)
Supraventricular Tachycardia
              (SVT)
• Narrow QRS tachycardia that originates
  above the ventricles; exact mechanism
  unknown
• Rate: 150-250+
• Rhythm: Regular
• P waves: Not visible
• QRS: Normal (narrow)
SVT
Atrial Fibrillation (A-fib, AF)
• Rate: Atrial rate so fast it can’t be counted
  Rhythm: Irregularly irregular (chaotic)
• P-waves: Not present; with no formed
  atrial impulses visible
• PR: not measurable
• QRS: usually normal
Atrial Fibrillation (A Fib, AF)
Atrial Fibrillation (A-fib, AF)
Atrial Flutter
• Rate: Atrial rate usually 300 bpm (250-350)
• 4:1 (V rate: 75 bpm), 3:1 (100 bpm), 2:1 (150 bpm), 1:1
  (300 bpm)
• Rhythm: Usually regular, can be irregular, depending on
  AV conduction
• P-wave: sawtooth pattern
• PR: not measurable
• QRS: normal
Atrial Flutter
Atrial Flutter
Multifocal Atrial Tachycardia
                  (MAT)
• Regularity      No
• Rate            Approximately 150’s
• P waves         Different appearing (3 different)
• PRI                    Variable
• QRS             < . 12 seconds
• Causes of MAT: pulmonary disease (especially
  during acute exacerbations), Dig
  toxicity, hypoxia, electrolyte
  imbalances, CHF, nicotine, alcohol, caffeine
• Treatment involves treating the underlying cause
  (sepsis, pulmonary disease) but Calcium Channel
MAT
Ventricular Tachycardia (V Tach, VT)

• Ectopic focus in ventricle
• Rate: 140 – 250, may start at lower rate
• Rhythm: usually regular, may be slightly
  irregular
• P-wave: Not usually present, however, can
  occasionally see P waves.
• PR: not measurable
• QRS: wide > 0.12 sec
• ACLS protocol to manage (CPR/SHOCK!)
Ventricular Tachycardia (V
        Tach, VT)
Idioventricular Rhythm
  Regularity   Yes
  Rate         20-40 bpm
  P waves      none
  QRS          WIDE
Ventricular Tachycardia
Regularity      Yes
Rate            > 100 bpm
P waves         Can’t always see them
QRS             Wide
Torsades de Pointes
 Multifocal Ventricular Tachycardia
Ventricular Fibrillation (V Fib, VF)
• Chaotic electrical activity in ventricles resulting in
  the ventricles quivering; Total loss of cardiac
  output
• Rate: Zero
• Rhythm: Chaotic
• P-waves: None
• QRS: None
   – Treat per ACLS protocol (CPR/SHOCK!)
Ventricular Fibrillation (V Fib, VF)
In Summary….
1. Is it fast or slow?
2. Is it regular or irregular?2.
3. Is there a P wave before every QRS
 complex?
Part 2 – Heart Blocks
Heart Blocks
• A blockage at any level of the electrical
  conduction system of the heart
• 1st degree heart block
• 2nd degree heart block
  – Type I (Wenkebach)
  – Type II
• 3rd degree (complete) heart block
1st degree heart block
• AV node conducts the electrical activity
  more slowly than normal
• PR interval > 200 ms.
• Often seen with bradycardia; usually an
  incidental finding on a routine EKG
2nd Degree Heart Block
• Disturbance, delay, or interruption of atrial
  impulse conduction to the ventricles
  through the atrioventricular node (AVN).
• Type I (Wenckebach, Mobitz I)
  – progressive prolongation of the PR interval until ultimately, the
    atrial impulse fails to conduct, a QRS complex is not generated,
    and there is no ventricular contraction
2nd Degree Heart Block
• Type II (Mobitz II)
  – an unexpected nonconducted atrial
    impulse, without prior measurable
    lengthening of the conduction time. Thus,
    the PR and R-R intervals between
    conducted beats are constant.
3rd Degree Heart Block
• “Complete” heart block
• Complete separation of atria from ventricles
• The P waves with a regular P to P interval
  represents the first rhythm.
• The QRS complexes with a regular R to R
  interval represent the second rhythm. The
  PR interval will be variable, as the hallmark
  of complete heart block is no apparent
  relationship between P waves and QRS
  complexes.
Moving on to bundle branch blocks
 • A reminder of the conduction system…
Bundle Branch Blocks
• A condition in which there's a delay or
  obstruction along the pathway that
  electrical impulses travel to the ventricles
• The blockage may occur on the pathway
  that sends electrical impulses to the left or
  the right side of your heart.
Bundle Branch Blocks
• Wide QRS complex (>= 120 ms)
• Left bundle branch block (LBBB): wide,
  purely positive QRS deflection in V5, V6
A bit about devices…
• Pacemakers- Used to treat
  bradyarrhythmias
• Pacemakers have two functions: pace or
  sense
• If pacing in the ventricle will make a wide
  QRS complex
Paced Rhythms
  Chamber         Chamber        Response to
   Paced           Sensed         Sensing


A = Atrium      A = Atrium      T = Triggered
V = Ventricle   V = Ventricle   I = Inhibited
D = Dual
                D = Dual        D = Dual (D+I)
  (A+V)
O = None        (A+V)           O = None

                O = None
What Type of Pacemaker Is It?
Part 3 – Ischemia, Injury or Infarct
Coronary Athrosclerosis
Ischemia, Injury or Infarct
• Ischemia- diminished blood flow to the heart muscle
       • ST segment depression and/or T-wave inversion.
• Injury- due to prolonged ischemia and lack of blood flow
  to the myocardium
       • ST segment elevation and/or tall, peaky T-waves over area of injury.
       • Can be reversed with prompt intervention and reperfusion
• Infarction- myocardial cells in the infarcted zone are
  dead and cannot be reperfused
   – Acute: ST elevation, Q waves may be present
   – Old: Significant Q waves present
           – Greater than ¼ amplitude of the R-wave OR
           – Greater than 0.04 seconds wide (one small box)
Time Passing
Ischemia
12 Lead EKG Practice
ST elevation with AMI


• Look for ST elevation >
  1mm in two or more
  anatomically contiguous
  leads
Coronary Circulation:

                                 Left
                                 Main

                                             Circumfl
                                             ex



                                                       Left
      Right                                            Anterior
      Coronar                                          Descendi
      y                                                ng
      Artery




Right Coronary Artery (RCA)            Left Main Coronary Artery
- Left Ventricle
  - Inferior Wall
  - Posterior Wall            Left Anterior DescendingCircumflex
- Right Ventricle             - Left Ventricle - Left Ventricle
- SA Node (55%)                 - Anterior Wall         - Lateral wall
- AV Node (90%)                 - Interventricular Septum
Leads tell location:
• Septal
  – V1-V2
• Anterior
  – V3-V4
• Inferior
  – II, III, AVF
• Lateral
  – I, AVL, V5-V6
Localization of an MI:
Inferior MI with reciprocal
 changes in lateral leads
12 Lead EKG Practice
12 Lead EKG Practice
12 Lead EKG Practice
Pericarditis:
• Patients present with s/s of MI
• Sharp, pleuritic, and POSITIONAL-
  relieved by leaning forward
• Often accompanied by fever and
  palpitations
• Treated with ASA or NSAID
• EKG shows DIFFUSE ST elevation in all
  leads except aVR
Cases….
• A 59 year-old female patient is sitting in
  the lobby when she begins to feel her
  heart race. You get her back to a patient
  room and hook her up to the heart
  monitor.
• This is what you see….
• What is the rhythm?
• A 73 year-old male diabetic patient is in
  the lobby waiting to see his internist. He
  clutches his chest and slumps onto the
  floor. As a member of the Code Blue
  Team you respond and after swift
  completion of your ABC’s (airway,
  breathing, circulation- ACLS protocol!) you
  hook him up to the monitor.
• This is what you see……
• What is the rhythm?
• What do you do next?
• You now see this on the monitor…..




• What is the rhythm?
• What else to you see?
• What do you do next?
• What do you see?
• What do you do next?
• A 32 year-old male patient comes into the
  clinic complaining of his heart racing. He
  has just come up from the Wellness
  Center downstairs and says he finished
  his “No Explode” energy drink just prior to
  beginning his workout. You swiftly take
  him back to a patient room and hook up
  the monitor.
• This is what you see……
• What is the rhythm?
• A 65 year-old male patient comes into the
  clinic to be evaluated for a syncopal
  episode. He admits to frequent episodes
  of lightheadedness and feeling like he
  could pass out. You hook him up to the
  monitor this is what you see…..
• What do you see?
• What is the underlying rhythm?

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Basic EKG and Rhythm Interpretation Symposia - The CRUDEM Foundation

  • 1. Basic EKG and Rhythm Interpretation William M. Vosik M.D. Milot, Haiti – January 8-12, 2012
  • 2. Objectives: 1. Describe the basic conduction system within the heart 2. Learn basic rhythm strip analysis 3. Learn basic EKG morphology 4. Be able to recognize normal and abnormal rhythms and EKGs
  • 3. Course Outline • Part 1 – Basic ECG and Arrythmias • Part 2 – “Heart block” and Pacemakers • Part 3 – Ischemia, Injury and Infract
  • 4. Conduction System of the Heart • Sinoatrial node (SA node): a small group of cells that function as the natural pacemaker of the heart (at 60-100 bpm) • Atrioventricular node (AV node): a small group of cells that: – Slows the conduction impulse from atria to ventricles to allow for ventricular filling – Functions as the backup pacemaker if SA node fails (40-60 bpm) – Screens rapid atrial impulses to protect the ventricles from dangerously fast rates
  • 5. Conduction System of the Heart • Bundle of His: short bundle of nerve fibers at the bottom of the AV node, leading to the bundle branches • Bundle branches: bundles of nerve fibers located along the septum that carry the impulse into the right and left ventricles – Right bundle branch: travels along the right side of the septum and carries impulse to the RV – Left bundle branch: travels along the left side of the septum and carries impulse to the LV • Purkinje Fibers: hair-like fibers that spread across endocardial surface of both ventricles and rapidly carry impulse to the ventricular muscle cells
  • 6. Conduction System of the Heart 1. Impulse originates in SA node and spreads through both atria simultaneously 2. Impulse is delayed in AV node so atria have time to contract and contribute to ventricular filling before ventricles contract 3. Impulse travels through Bundle of His, down both bundle branches into the ventricles and through the Purkinje fibers to the ventricular myocardium
  • 7. Conduction System of the Heart Right atrium Left atrium Left ventricle Right ventricle
  • 8. What am I looking for? • 1. What is the rate? (fast, slow or normal) • 2. What is the rhythm? (regular or irregular) • 3. Is there a P wave before every QRS complex? (is it sinus rhythm or not) • 4. Is the QRS complex wide or narrow?
  • 9. Rhythm Strips • Focus only on the rate and rhythm • Evaluate each strip for: – Heart rate – Rhythm: regular/irregular – Location and morphology of P waves – QRS status • *Cannot diagnose acute MI on rhythm strip!
  • 10. EKG (electrocardiogram) A picture of the electrical conduction through the heart • P wave (atrial depolarization) • QRS complex (ventricular depolarization) – Q wave: first negative deflection from baseline – R wave: positive deflection from baselinie – S wave: negative deflection following an R wave • T wave (ventricular repolarization)
  • 11. Intervals • PR Interval: AV conduction time; the time for impulse to travel SA node atria AV node ventricles – Measured from beginning of P wave to beginning of QRS. – Normal= 120-200 ms (0.12- 0.20 sec ) – >200 ms: AV block • QRS Duration: time for conduction to travel through LV and RV – Measured from beginning to end of QRS complex (QRS width) – Normal= < 120 ms (0.12 sec)
  • 12. Intervals • ST segment: early repolarization phase; should be at baseline; elevation or depression indicates injury, ischemia or infarct – From end of S wave to beginning of T wave – More to come on this later… • QT Interval: the time for both ventricular depolarization and repolarization – Dependent upon rate
  • 13. Rate • How fast the heart is going – How many times the heart beats in 1 minute (bpm) – A “beat” is a ventricular contraction or a QRS complex. • Only reliable if rhythm is regular; if irregular then rate is just an estimation
  • 14. Rate • ON RHYTHM STRIP: – If paper speed is 25 mm/sec (standard), the vertical lines at top edge of paper represent 3 second intervals – 1 small box = 0.04 sec – There are 5 small boxes within each large box – 1 large box = 0.20 sec
  • 15. Rate • ON RHYTHM STRIP: – Count the number of QRS complexes in a 6 second strip and multiply by 10
  • 16. Rate • ON EKG: – Measured between two consecutive QRS waves (R-R interval) 300 150 100 75 60 – Rule of 300’s: • Count the # of large boxes between R waves and divide into 300 • Find an R wave on a dark line. The first big box would be 300, the second 150, 100, 75, 60 bpm.* *This applies only to EKGs printed at 25 mm/sec (standard)
  • 17. Rhythm • Regular: equal distance between R waves • Irregular: varying distances between R waves – Regularly irregular: R waves occur in a pattern – Irregularly irregular: totally irregular without any pattern • Mark out R-R intervals on a separate piece of paper and hold over EKG to see if all R-R’s map out
  • 18. Arrhythmias… What you need to know: 1. Normal or Abnormal? 2. Name the Arrhythmia 3. Treat the Patient with the Arrhythmia
  • 19. Basic Rhythms and Arrhythmias • Normal Sinus Rhythm • Supraventricular • Sinus Tachycardia Tachycardia (SVT) • Sinus Bradycardia • Atrial Fibrillation (AF) • Sinus Arrhythmia • Atrial Flutter • Sinus Arrest • Ventricular Tachycardia • Ectopy (PACs, PVCs) (VT) • Torsades de Pointes • Ventricular Fibrillation (VF)
  • 20. Normal Sinus Rhythm • Rate: 60-100 bpm • Rhythm: regular • P -> QRS -> T • Intervals: All normal
  • 21.
  • 22. Sinus Tachycardia • Rate: >100 bpm • Rhythm: regular • P -> QRS -> T • Intervals: All normal
  • 24. Sinus Bradycardia • Rate: <60 bpm • Rhythm: Regular • P -> QRS -> T • Intervals: All normal
  • 26. Sinus Arrhythmia • Rate: Any rate, but usually 60-100 bpm • Rhythm: irregular • P -> QRS -> T • Intervals: All normal
  • 28. Sinus Pause/Arrest • A period of time where there is no heart beat (flat line) – Somewhat interchangeable, but technically… • Just one impulse fails to form: sinus pause • More than one impulse in a row fails to form: sinus arrest • Rate: can be normal or bradycardia • Rhythm: irregular due to absence of sinus node discharge (evaluate baseline rhythm independently)
  • 29. Ectopy • Premature atrial contractions (PACs) – Irritable focus in atria fires before next sinus impulse is due – A compensatory pause will follow an ectopic beat – Rate: Normal – Rhythm: Irregular because of the PAC, otherwise baseline rhythm is regular
  • 30. Ectopy • Premature ventricular contractions (PVCs) – Early beat followed by compensatory pause – Rhythm: Irregular because of the PVC, otherwise baseline rhythm is regular – QRS: Wide
  • 31. PVCs
  • 32. More Advanced Arrhythmias • Supraventricular Tachycardia (SVT) • Atrial Fibrillation (AF) • Atrial Flutter • Torsades de Pointes • Ventricular Tachycardia (VT) • Ventricular Fibrillation (VF)
  • 33. Supraventricular Tachycardia (SVT) • Narrow QRS tachycardia that originates above the ventricles; exact mechanism unknown • Rate: 150-250+ • Rhythm: Regular • P waves: Not visible • QRS: Normal (narrow)
  • 34. SVT
  • 35. Atrial Fibrillation (A-fib, AF) • Rate: Atrial rate so fast it can’t be counted Rhythm: Irregularly irregular (chaotic) • P-waves: Not present; with no formed atrial impulses visible • PR: not measurable • QRS: usually normal
  • 38.
  • 39. Atrial Flutter • Rate: Atrial rate usually 300 bpm (250-350) • 4:1 (V rate: 75 bpm), 3:1 (100 bpm), 2:1 (150 bpm), 1:1 (300 bpm) • Rhythm: Usually regular, can be irregular, depending on AV conduction • P-wave: sawtooth pattern • PR: not measurable • QRS: normal
  • 42. Multifocal Atrial Tachycardia (MAT) • Regularity No • Rate Approximately 150’s • P waves Different appearing (3 different) • PRI Variable • QRS < . 12 seconds • Causes of MAT: pulmonary disease (especially during acute exacerbations), Dig toxicity, hypoxia, electrolyte imbalances, CHF, nicotine, alcohol, caffeine • Treatment involves treating the underlying cause (sepsis, pulmonary disease) but Calcium Channel
  • 43. MAT
  • 44. Ventricular Tachycardia (V Tach, VT) • Ectopic focus in ventricle • Rate: 140 – 250, may start at lower rate • Rhythm: usually regular, may be slightly irregular • P-wave: Not usually present, however, can occasionally see P waves. • PR: not measurable • QRS: wide > 0.12 sec • ACLS protocol to manage (CPR/SHOCK!)
  • 46. Idioventricular Rhythm Regularity Yes Rate 20-40 bpm P waves none QRS WIDE
  • 47. Ventricular Tachycardia Regularity Yes Rate > 100 bpm P waves Can’t always see them QRS Wide
  • 48. Torsades de Pointes Multifocal Ventricular Tachycardia
  • 49. Ventricular Fibrillation (V Fib, VF) • Chaotic electrical activity in ventricles resulting in the ventricles quivering; Total loss of cardiac output • Rate: Zero • Rhythm: Chaotic • P-waves: None • QRS: None – Treat per ACLS protocol (CPR/SHOCK!)
  • 51. In Summary…. 1. Is it fast or slow? 2. Is it regular or irregular?2. 3. Is there a P wave before every QRS complex?
  • 52. Part 2 – Heart Blocks
  • 53. Heart Blocks • A blockage at any level of the electrical conduction system of the heart • 1st degree heart block • 2nd degree heart block – Type I (Wenkebach) – Type II • 3rd degree (complete) heart block
  • 54. 1st degree heart block • AV node conducts the electrical activity more slowly than normal • PR interval > 200 ms. • Often seen with bradycardia; usually an incidental finding on a routine EKG
  • 55. 2nd Degree Heart Block • Disturbance, delay, or interruption of atrial impulse conduction to the ventricles through the atrioventricular node (AVN). • Type I (Wenckebach, Mobitz I) – progressive prolongation of the PR interval until ultimately, the atrial impulse fails to conduct, a QRS complex is not generated, and there is no ventricular contraction
  • 56.
  • 57. 2nd Degree Heart Block • Type II (Mobitz II) – an unexpected nonconducted atrial impulse, without prior measurable lengthening of the conduction time. Thus, the PR and R-R intervals between conducted beats are constant.
  • 58.
  • 59. 3rd Degree Heart Block • “Complete” heart block • Complete separation of atria from ventricles • The P waves with a regular P to P interval represents the first rhythm. • The QRS complexes with a regular R to R interval represent the second rhythm. The PR interval will be variable, as the hallmark of complete heart block is no apparent relationship between P waves and QRS complexes.
  • 60.
  • 61. Moving on to bundle branch blocks • A reminder of the conduction system…
  • 62. Bundle Branch Blocks • A condition in which there's a delay or obstruction along the pathway that electrical impulses travel to the ventricles • The blockage may occur on the pathway that sends electrical impulses to the left or the right side of your heart.
  • 63. Bundle Branch Blocks • Wide QRS complex (>= 120 ms) • Left bundle branch block (LBBB): wide, purely positive QRS deflection in V5, V6
  • 64. A bit about devices… • Pacemakers- Used to treat bradyarrhythmias • Pacemakers have two functions: pace or sense • If pacing in the ventricle will make a wide QRS complex
  • 65. Paced Rhythms Chamber Chamber Response to Paced Sensed Sensing A = Atrium A = Atrium T = Triggered V = Ventricle V = Ventricle I = Inhibited D = Dual D = Dual D = Dual (D+I) (A+V) O = None (A+V) O = None O = None
  • 66. What Type of Pacemaker Is It?
  • 67.
  • 68.
  • 69. Part 3 – Ischemia, Injury or Infarct
  • 71. Ischemia, Injury or Infarct • Ischemia- diminished blood flow to the heart muscle • ST segment depression and/or T-wave inversion. • Injury- due to prolonged ischemia and lack of blood flow to the myocardium • ST segment elevation and/or tall, peaky T-waves over area of injury. • Can be reversed with prompt intervention and reperfusion • Infarction- myocardial cells in the infarcted zone are dead and cannot be reperfused – Acute: ST elevation, Q waves may be present – Old: Significant Q waves present – Greater than ¼ amplitude of the R-wave OR – Greater than 0.04 seconds wide (one small box)
  • 74. 12 Lead EKG Practice
  • 75. ST elevation with AMI • Look for ST elevation > 1mm in two or more anatomically contiguous leads
  • 76. Coronary Circulation: Left Main Circumfl ex Left Right Anterior Coronar Descendi y ng Artery Right Coronary Artery (RCA) Left Main Coronary Artery - Left Ventricle - Inferior Wall - Posterior Wall Left Anterior DescendingCircumflex - Right Ventricle - Left Ventricle - Left Ventricle - SA Node (55%) - Anterior Wall - Lateral wall - AV Node (90%) - Interventricular Septum
  • 77. Leads tell location: • Septal – V1-V2 • Anterior – V3-V4 • Inferior – II, III, AVF • Lateral – I, AVL, V5-V6
  • 79. Inferior MI with reciprocal changes in lateral leads
  • 80. 12 Lead EKG Practice
  • 81. 12 Lead EKG Practice
  • 82. 12 Lead EKG Practice
  • 83. Pericarditis: • Patients present with s/s of MI • Sharp, pleuritic, and POSITIONAL- relieved by leaning forward • Often accompanied by fever and palpitations • Treated with ASA or NSAID • EKG shows DIFFUSE ST elevation in all leads except aVR
  • 84.
  • 85.
  • 86.
  • 87. Cases…. • A 59 year-old female patient is sitting in the lobby when she begins to feel her heart race. You get her back to a patient room and hook her up to the heart monitor. • This is what you see….
  • 88. • What is the rhythm?
  • 89. • A 73 year-old male diabetic patient is in the lobby waiting to see his internist. He clutches his chest and slumps onto the floor. As a member of the Code Blue Team you respond and after swift completion of your ABC’s (airway, breathing, circulation- ACLS protocol!) you hook him up to the monitor. • This is what you see……
  • 90. • What is the rhythm? • What do you do next?
  • 91. • You now see this on the monitor….. • What is the rhythm? • What else to you see? • What do you do next?
  • 92. • What do you see? • What do you do next?
  • 93. • A 32 year-old male patient comes into the clinic complaining of his heart racing. He has just come up from the Wellness Center downstairs and says he finished his “No Explode” energy drink just prior to beginning his workout. You swiftly take him back to a patient room and hook up the monitor. • This is what you see……
  • 94. • What is the rhythm?
  • 95. • A 65 year-old male patient comes into the clinic to be evaluated for a syncopal episode. He admits to frequent episodes of lightheadedness and feeling like he could pass out. You hook him up to the monitor this is what you see…..
  • 96. • What do you see? • What is the underlying rhythm?

Editor's Notes

  1. Write this down- not in your handout
  2. Usually doesn’t need treatment;Can be caused by drugs such as CCBs and BBs;Also seen with acute myositis, AMI, and electrolyte disturbances
  3. the impulse generated in the SA node in the atrium does not propagate to the ventricles; Because the impulse is blocked, an accessory pacemaker in the lower chambers will typically activate the ventricles.Often significantlybradycardic
  4. You will often find P waves buried within QRS or T wave complexes
  5. Because it is an prolongation of the ventricular depolarization, the QRS is wide (takes longer for the impulse to go through both ventricles)LEFT: activation of the left ventricle is delayed, which results in the left ventricle contracting later than the right ventricle.
  6. Extreme sinus bradycardia, sinus arrest/pauses &gt;2.0 s duration, chronotropic incompetence, heart blocks, tachybrady syndrome (sick sinus syndrome)
  7. Atrial fibrillation
  8. Ventricular tachycardiaAttach the AED and shock the patient! Get HELP!
  9. Sinus rhythmST elevationGet a 12-lead and HELP!
  10. Acute MI! Tombstoning!GET HELP! (aka a cardiologist!)
  11. How is this different from the atrial fibrillation rhythm we just saw? REGULAR!!!
  12. Sinus arrest13 big boxes x .20 seconds = 2.6 second pause!(Much greater than 2.0 seconds and you’re at risk for passing out)