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Physiotherapy assessment Traumatic brain injury
1. PHYSIOTHERAPY ASSESSMENT OF TRAUMATIC
BRAIN INJURY (UNCONSCIOUS)
PRESENTED BY
AHMAD MUKHTAR MAGAJI (B.PT-BUK)
TO
THE DEPARTMENT OF MEDICAL REHABILITATION USMANU
DANFODIYO UNIVERSITY TEACHING HOSPITAL SOKOTO
ON
JULY,2020
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3. Introduction
• Traumatic Brain injury is any physical damage to the brain caused
by external force.(Barr et. al., 2012)
• Traumatic brain injuries constitute a significant portion of injury
resulting from automotive collisions (john et. al., 2013)
• Brain injuries not only represent a serious trauma for those
involved but also place an enormous burden on society, often
exacting a heavy economical, social, and emotional price. (Wong
et al., 2005) 3
4. Intro cont.
• Development of intervention strategies to prevent or minimize
these injuries requires a complete understanding of injury
mechanisms, response and tolerance level (Anthony et. al., 1999)
• It is worthy of note that the long-term sequelae of traumatic brain
injury especially moderate to severe cases do not affect the survivor
alone, but also the community they live in, as well as their families
and caregivers, both psychologically and economically. (Dziadzko et.
al., 2016)
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5. Definition
• Traumatic brain injury (TBI) is a non degenerative, non
congenital insult to the brain from external mechanical force
possibly leading to permanent or temporary impairment of
cognitive, physical, and psychosocial functions, with an
associated diminished or altered level of consciousness
(Segun, T. D., 2019)
• Unconscious “the state of not being awake and not aware of
things around you especially as the result of a head injury”
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10. Epidemiology
According to research carried out in university of port harcourt
by Regina, C.O. (2018)
• 30.9% of patients with trauma and 3.6 % of all A&E
admissions present with TBI.
• 76.9% males and 23.1% females 3.3:1
• Fatality rate of 22.6% of presenting TBI cases.
• Highest Rate of TBI occur in older adults
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13. Response to injury
• Swelling of brain
• Vasodilatation with increased blood volume
• Increased ICP
• Decreased blood flow to brain
• Perfusion decreases
• Cerebral ischemia (hypoxia)
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14. Assessment
Subjective
o Chief complain
o History
the exact time and nature of the accident, including the direction of the blow;
First action taken
Headache, nausea, vomiting and convulsion
Loss of consciousness
Referral
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15. Subjective ass
• PMHx- HTN,DM,RTA, previous hx of accidents,
Surgery
• FSHx- marital status, No of children, occupation,
hobbies, social life (smoking, alcohol, kola nut, drug
addiction ).
• DHx- muscle relaxants, anticoagulants e.t.c
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16. Objective Ass.
• O/E- position, bruises, stitches, oxygen saturation,
jaundice, breathing pattern, i.v line, presence of
urinary catheter, Ng tube, swelling, racoon eyes, battle
signs.
• Examination-
Vital signs : BP,PR, Temperature, RR, SPO2.
Glasgow coma scale
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25. Focal
Cerebral contusion
– Blunt trauma to local
brain tissue
– Capillary bleeding into
brain tissue
– Common with blunt
head trauma
• Confusion
• Neurologic deficit
– Personality changes
– Vision changes
– Speech changes
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26. Epidural Hematoma
– Bleeding between dura mater
and skull
– Involves arteries
• Middle meningeal artery most
common
– Rapid bleeding & reduction of
oxygen to tissues
– Herniates brain toward
foramen magnum
• Associated symptoms
• Ipsilateral dilated fixed pupil, signs
of increasing ICP, unconsciousness,
contralateral paralysis, death
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27. Subdural Hematoma
• Subdural Hematoma
– Bleeding within meninges
• Beneath dura mater & within
subarachnoid space
• Above pia mater
– Slow bleeding
• Superior sagital sinus
– Signs progress over several
days
• Slow deterioration of mentation
• Associated symptoms
• Headache
• Focal neurologic signs
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28. Intracerebral Hemorrhage
– Rupture blood vessel within
the brain
– Presentation similar to stroke
symptoms
– Signs and symptoms worsen
over time
• Associated symptoms
• Varies with region and degree
• Pattern similar to stroke
• Headache and vomiting
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29. DIFFUSE
Moderate Diffuse Axonal injury
Most common type of injury
as a result of severe blunt
head trauma. Brain is injured
so diffusely that there is
generalized edema. Usually,
there is no evidence of a
structural lesion. In most cases
patient presents unconscious,
without focal deficits.
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30. conclusion
Proper assessment of patient with traumatic brain injury is
key to accurate diagnosis and subsequent management.
High priority should be given to the prevention of traumatic
brain injury
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31. Recommendation
• Subsequent presenter should Present on assessment and
management of non-traumatic brain injury .
• Subsequent presentation on management of traumatic brain
injury should be done
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33. Refrences
• Barr, R.M., Gean, A.D., Le, T.H. Craniofacial trauma. In: Brant WE, Helms CA, editors.
Fundamentals of Diagnostic Radiology. 4th ed. Philadelphia: Wolters Kluwer
Health/Lippincott Williams & Wilkins; 2012. p. 49–74.
• Dziadzko, V., Dziadzko, M.A., Gajic, O., Karnatovskaia, L. Approaching psychological
trauma of the critically ill: Patient and family perspectives. Am J Respir Crit Care Med
2016;193:A4744.
• Segun, T. D., 2019. Traumatic brain injury (TBI)- Definition, Epidemiology,
Pathophysiology. Physical Medicine and Rehabilitation.
• Regina, C. O., Richard, C. E., 2018. An epidemiologic study of traumatic head injuries in
the emergency department of a tertiary health institution. Journal of Medicine in the
Tropics.
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