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CASE PRESENTATION
PRESENTED BY
DR. ABDUL SALAM
FCPS RESIDENT
PAEDS WARD, LIYARI GOVT. HOSPITAL
HISTORY
6 year old femlae child sadia d/o shoaib , wt 20 kg, 5 DOA Resident Of Lyari admitted through opd
with Complaint of:
Fever 6 days
Leg pain 3 days
HISTORY OF PRESENTING
COMPLAINT
 A/C to mother , My pt. Was alright 6 days back then she developed fever that was sudden in
onset,high grade documented as 103 F, continuous in nature, No aggrevating and relieving factor
associated with rigors and chills.pt also have leg pain that was gradual in onset,mild , Present
through out the day,aching in character No aggrevating and relieving factor and no referred. Or
shifting of pain.hx of nausea was present that was occasional with no hx of rashes
PAST HISTORY
PAST MEDICAL:
No Previous Significant Medical Admission
PAST SURGICAL:
 Non significant
 Hx of birth
 Non significant
 Allergic hx
 Non significant
 Tranfusion hx
 Non significant
Development hx
All milestones are appropriate according to age
Studying in class 1
VACCINATION HISTORY
 Completely Vaccinated Child
Nutritional Hx:
before illness pt was having one paratha chai at morning along with one roti and salan at lunch
dinner Along with one snacks
Now she is having biscuits chai at morning ,. ½ roti with salan at lunch and dinner With no
snacks in between
Apetite decreases after illness
FAMILY HISTORY
 Product of consanguineous marriage
 Mother age 38 yrs and Father 42 yrs
 3 siblings ( 1 sister and 2 brother) alive and healthy
 No hx of any chronic illness in family
 Personal hx
 Sleep normal
 Apetite dec
 Bowel habits normal
 Hx of pica positive
SOCIOECONOMIC HISTORY
 Live in rented house
 6 members
 One earner
 earning is 28250 per month (security guard)
 Use tap water
 Ventilated house
EXAMINATION
My pt was lying on bed with cannula on her right hand fully oriented as per time place and
person
VITALS:
 HR:115 bpm
 RR: 32/min APM
 Temp: 101o F wt: 20 kg length 115cm ( SD Median)
 BP : 110/95 mm Hg
 CRT: 2sec
o2 sat :98 percent
SUBVITALS:
 A+, J-, CL-, C-, D-, E-, LN-
 HEENT appears to be normal
SYSTEMIC EXAMINATION
CARDIOVASCULAR:
 S1+ S2 + 0
CENTRAL NERVOUS SYSTEM:
 Intact
ABDOMEN:
 On inspection there Was normal breathing pattern umbilicus was placed centrally with
no buldging or scar marks Or prominent veins
 On palpation liver was palpable 2 cm below subcostal margins Smooth regular surface
and borders ,non tender with an average span of 11 cm rest of the findings were non
significant
 On percussion dull
 On ascultation gut sounds were audible
 CHEST
 Examination of chest was non significant with bilateral air entery and no added sounds
centrally placed trachea and apex beat at 4 ics
DIFFERENTIAL DIAGNOSIS
 Typhoid
 Dengue
 malaria
INVESTIGATIONS
 CBC
 UCE
 MP ICT
 Blood C/S
 Dengue Serology
Labs: CBC
Date 20/08 24/ 08 28/08
HB 10g/dl 9.5g/dl 8.6g/dl
TLC 7200 6500 7000
Hematocrit 45 47 47
Neut 40 47 46
Lymph 55 55 45
Eisno 02 05 06
Mono 03 04 04
Plat C 100000 45000 75000
Normal Values: Hb: 13.0-16.5 . TLC: 4.0-11.0*109 L, Neut: 50-70%, Lymph: 20-50%, Eisno: 1-6%, Mono: 2-10%, Plat C: 150-400
uc
e
MPICT
 P
. Falciparum = Negative
 P Vivax = Negative
Dengue Serology:
 Dengue IgM Antibodies: positive
 Dengue IgG Antibodies: negative
Blood C/S:
No Organism Seen
Treatment Given
 Admit In Paeds Ward
 TPR Monitoring 4 hourly
 Maintain IV Line
 Orally allowed
 0.45 percent D/S 1500 ml @ 60 ml/ hr
 Inj Ceftriaxone 700mg IV BD
 Inj Provas 20 ml IV SOS
 Syp Panadol 1 TSF SOS
DEFINITIVE DIAGNOSIS
Dengue
DENGUE
INTRODUCTION:
Dengue viruses are spread to people through the
bite of an infected Aedes species
Dengue virus (DENV) is the cause of dengue fever. It
is a mosquito-borne, single positive-stranded RNA
virus of the family Flaviviridae; genus Flavivirus. Four
serotypes of the virus have been found DENV-1,
DENV-2, DENV-3 and DENV-4, a reported fifth has yet
to be confirmed, all of which can cause the full
spectrum of disease.
EPIDEMIOLOGY:
 Before 1970, only 9 countries had experienced severe dengue epidemics. The disease is
now endemic in more than 100 countries in the WHO regions of Africa, the Americas, the
Eastern Mediterranean, South-East Asia and the Western Pacific.
 Since 2010, Pakistan has been experiencing an epidemic of dengue fever that has
caused 16 580 confirmed cases and 257 deaths in Lahore and nearly 5000 cases and 60
deaths reported from the rest of the country. The three provinces facing the epidemic
are Khyber Pakhtunkhwa, Punjab and Sindh.
Dengue Hemorrhagic Fever
 The risk factors for developing dengue hemorrhagic fever include
 This rare form of the disease is characterized by:
 high fever
 damage to the lymphatic system
 damage to blood vessels
 bleeding from the nose
 bleeding under the skin
 internal bleeding
 bleeding from the gums
 liver enlargement
 circulatory system failure
 The symptoms of dengue hemorrhagic fever can trigger dengue shock
syndrome, which is also characterized by low blood pressure, weak pulse, cold,
clammy skin, and restlessness. Dengue shock syndrome is severe and can lead
to excessive bleeding and even death.
SYMPTOMS:
Common Dengue symptoms include:
 Symptoms, which usually begin four to six days after infection and last for up to 10 days,
may include
 Sudden, high fever
 Severe headaches
 Pain behind the eyes
 Severe joint and muscle pain
 Fatigue
 Nausea
 Vomiting
 Skin rash, which appears two to five days after the onset of fever
 Mild bleeding (such a nose bleed, bleeding gums, or easy bruising)
INVESTIGATIONS
 Dengue NS1: The presence of dengue nonstructural protein 1 (NS1) antigen is consistent
with acute-phase infection with dengue virus. The NS1 antigen is typically detectable within
1 to 2 days following infection and up to 9 days following symptom onset.
 Dengue Serology: The IgM become detectable on Day 3 to 5 of illness in case of primary
dengue infection and persist for 2 to 3 months, whereas IgG appear by the fourteenth day
and persist for life. Secondary infection shows that IgG rises within 1 to 2 days after onset of
symptoms, simultaneously with IgM antibodies.:
 Positive IgM and IgG tests for dengue antibodies detected in an initial blood sample mean
that it is likely that the person became infected with dengue virus within recent weeks.
TREATMENT
 There is no specific medicine to treat dengue infection. If you think you may have
dengue fever, you should use pain relievers with acetaminophen and avoid medicines
with aspirin, which could worsen bleeding. You should also rest, drink plenty of fluids,
and see your doctor. If you start to feel worse in the first 24 hours after your fever goes
down, you should get to a hospital immediately to be checked for complications.
PREVENTION
The best way to prevent the disease is to prevent bites by infected mosquitoes, particularly if you
are living in or traveling to a tropical area. This involves protecting yourself and making efforts to
keep the mosquito population down. In 2019, the FDA approved a vaccine called Dengvaxia to
help prevent the disease from occurring in adolescents aged 9 to 16 who have already been
infected by dengue. But, there currently is no vaccine to prevent the general population from
contracting it.
To protect yourself:
•Use mosquito repellents, even indoors.
•When outdoors, wear long-sleeved shirts and long pants tucked into socks.
•When indoors, use air conditioning if available.
•Make sure window and door screens are secure and free of holes. If sleeping areas are not
screened or air conditioned, use mosquito nets.
•If you have symptoms of dengue, speak to your doctor.
To reduce the mosquito population, get rid of places where mosquitoes can breed. These include
old tires, cans, or flower pots that collect rain. Regularly change the water in outdoor bird baths
and pets' water dishes.
If someone in your home gets dengue fever, be especially vigilant about efforts to protect
yourself and other family members from mosquitoes. Mosquitoes that bite the infected family
member could spread the infection to others in your home.
THANK YOU
 How can pneumonia be described more exactly?
 Experts also classify pneumonia according to factors other than where the patient was infected and the severity. But that typically
doesn't affect how the pneumonia is treated. Instead, it's useful for getting a better description of the illness.
 Atypical pneumonia
 Typical pneumonia generally begins with a sudden high fever and chills, and then coughing with phlegm coming later.
 Atypical pneumonia is caused by other germs, which are also referred to as "atypical." Older people in particular have fewer or slightly
different symptoms if they have atypical pneumonia: It then starts off rather slowly with a mild fever and/or headache and aching
Rather than coughing with phlegm, they have a dry, tickly cough.
 Atypical symptoms don't mean that the lungs are less severely inflamed or that the disease will take a milder course though.
 Upper, middle and lower lobe pneumonia
 X-rays play an important role in distinguishing between these types: the term lobar pneumonia is used if an entire lung lobe is visibly
inflamed. Depending on which lung lobe is affected, the pneumonia is referred to as upper, middle or lower lobe pneumonia.
 If there are several multi-lobe focal inflammations in the lungs, the term focal pneumonia is used. Some people use the term
bronchopneumonia if the focal inflammations started in inflamed airways (bronchi).
 Sometimes, it's the air sacs that are more inflamed (alveolar pneumonia), and sometimes it's the tissue between the sacs (interstitial
pneumonia).

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DOC-20220906-WA0008..pptx

  • 1. CASE PRESENTATION PRESENTED BY DR. ABDUL SALAM FCPS RESIDENT PAEDS WARD, LIYARI GOVT. HOSPITAL
  • 2. HISTORY 6 year old femlae child sadia d/o shoaib , wt 20 kg, 5 DOA Resident Of Lyari admitted through opd with Complaint of: Fever 6 days Leg pain 3 days HISTORY OF PRESENTING COMPLAINT  A/C to mother , My pt. Was alright 6 days back then she developed fever that was sudden in onset,high grade documented as 103 F, continuous in nature, No aggrevating and relieving factor associated with rigors and chills.pt also have leg pain that was gradual in onset,mild , Present through out the day,aching in character No aggrevating and relieving factor and no referred. Or shifting of pain.hx of nausea was present that was occasional with no hx of rashes
  • 3. PAST HISTORY PAST MEDICAL: No Previous Significant Medical Admission PAST SURGICAL:  Non significant  Hx of birth  Non significant  Allergic hx  Non significant  Tranfusion hx  Non significant
  • 4. Development hx All milestones are appropriate according to age Studying in class 1
  • 5. VACCINATION HISTORY  Completely Vaccinated Child Nutritional Hx: before illness pt was having one paratha chai at morning along with one roti and salan at lunch dinner Along with one snacks Now she is having biscuits chai at morning ,. ½ roti with salan at lunch and dinner With no snacks in between Apetite decreases after illness
  • 6. FAMILY HISTORY  Product of consanguineous marriage  Mother age 38 yrs and Father 42 yrs  3 siblings ( 1 sister and 2 brother) alive and healthy  No hx of any chronic illness in family  Personal hx  Sleep normal  Apetite dec  Bowel habits normal  Hx of pica positive
  • 7. SOCIOECONOMIC HISTORY  Live in rented house  6 members  One earner  earning is 28250 per month (security guard)  Use tap water  Ventilated house
  • 8. EXAMINATION My pt was lying on bed with cannula on her right hand fully oriented as per time place and person VITALS:  HR:115 bpm  RR: 32/min APM  Temp: 101o F wt: 20 kg length 115cm ( SD Median)  BP : 110/95 mm Hg  CRT: 2sec o2 sat :98 percent SUBVITALS:  A+, J-, CL-, C-, D-, E-, LN-  HEENT appears to be normal
  • 9. SYSTEMIC EXAMINATION CARDIOVASCULAR:  S1+ S2 + 0 CENTRAL NERVOUS SYSTEM:  Intact ABDOMEN:  On inspection there Was normal breathing pattern umbilicus was placed centrally with no buldging or scar marks Or prominent veins  On palpation liver was palpable 2 cm below subcostal margins Smooth regular surface and borders ,non tender with an average span of 11 cm rest of the findings were non significant  On percussion dull  On ascultation gut sounds were audible  CHEST  Examination of chest was non significant with bilateral air entery and no added sounds centrally placed trachea and apex beat at 4 ics
  • 11. INVESTIGATIONS  CBC  UCE  MP ICT  Blood C/S  Dengue Serology
  • 12. Labs: CBC Date 20/08 24/ 08 28/08 HB 10g/dl 9.5g/dl 8.6g/dl TLC 7200 6500 7000 Hematocrit 45 47 47 Neut 40 47 46 Lymph 55 55 45 Eisno 02 05 06 Mono 03 04 04 Plat C 100000 45000 75000 Normal Values: Hb: 13.0-16.5 . TLC: 4.0-11.0*109 L, Neut: 50-70%, Lymph: 20-50%, Eisno: 1-6%, Mono: 2-10%, Plat C: 150-400
  • 13. uc e
  • 14. MPICT  P . Falciparum = Negative  P Vivax = Negative Dengue Serology:  Dengue IgM Antibodies: positive  Dengue IgG Antibodies: negative Blood C/S: No Organism Seen
  • 15. Treatment Given  Admit In Paeds Ward  TPR Monitoring 4 hourly  Maintain IV Line  Orally allowed  0.45 percent D/S 1500 ml @ 60 ml/ hr  Inj Ceftriaxone 700mg IV BD  Inj Provas 20 ml IV SOS  Syp Panadol 1 TSF SOS
  • 17. DENGUE INTRODUCTION: Dengue viruses are spread to people through the bite of an infected Aedes species Dengue virus (DENV) is the cause of dengue fever. It is a mosquito-borne, single positive-stranded RNA virus of the family Flaviviridae; genus Flavivirus. Four serotypes of the virus have been found DENV-1, DENV-2, DENV-3 and DENV-4, a reported fifth has yet to be confirmed, all of which can cause the full spectrum of disease.
  • 18. EPIDEMIOLOGY:  Before 1970, only 9 countries had experienced severe dengue epidemics. The disease is now endemic in more than 100 countries in the WHO regions of Africa, the Americas, the Eastern Mediterranean, South-East Asia and the Western Pacific.  Since 2010, Pakistan has been experiencing an epidemic of dengue fever that has caused 16 580 confirmed cases and 257 deaths in Lahore and nearly 5000 cases and 60 deaths reported from the rest of the country. The three provinces facing the epidemic are Khyber Pakhtunkhwa, Punjab and Sindh.
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  • 20. Dengue Hemorrhagic Fever  The risk factors for developing dengue hemorrhagic fever include  This rare form of the disease is characterized by:  high fever  damage to the lymphatic system  damage to blood vessels  bleeding from the nose  bleeding under the skin  internal bleeding  bleeding from the gums  liver enlargement  circulatory system failure  The symptoms of dengue hemorrhagic fever can trigger dengue shock syndrome, which is also characterized by low blood pressure, weak pulse, cold, clammy skin, and restlessness. Dengue shock syndrome is severe and can lead to excessive bleeding and even death.
  • 21. SYMPTOMS: Common Dengue symptoms include:  Symptoms, which usually begin four to six days after infection and last for up to 10 days, may include  Sudden, high fever  Severe headaches  Pain behind the eyes  Severe joint and muscle pain  Fatigue  Nausea  Vomiting  Skin rash, which appears two to five days after the onset of fever  Mild bleeding (such a nose bleed, bleeding gums, or easy bruising)
  • 22. INVESTIGATIONS  Dengue NS1: The presence of dengue nonstructural protein 1 (NS1) antigen is consistent with acute-phase infection with dengue virus. The NS1 antigen is typically detectable within 1 to 2 days following infection and up to 9 days following symptom onset.  Dengue Serology: The IgM become detectable on Day 3 to 5 of illness in case of primary dengue infection and persist for 2 to 3 months, whereas IgG appear by the fourteenth day and persist for life. Secondary infection shows that IgG rises within 1 to 2 days after onset of symptoms, simultaneously with IgM antibodies.:  Positive IgM and IgG tests for dengue antibodies detected in an initial blood sample mean that it is likely that the person became infected with dengue virus within recent weeks.
  • 23. TREATMENT  There is no specific medicine to treat dengue infection. If you think you may have dengue fever, you should use pain relievers with acetaminophen and avoid medicines with aspirin, which could worsen bleeding. You should also rest, drink plenty of fluids, and see your doctor. If you start to feel worse in the first 24 hours after your fever goes down, you should get to a hospital immediately to be checked for complications.
  • 24. PREVENTION The best way to prevent the disease is to prevent bites by infected mosquitoes, particularly if you are living in or traveling to a tropical area. This involves protecting yourself and making efforts to keep the mosquito population down. In 2019, the FDA approved a vaccine called Dengvaxia to help prevent the disease from occurring in adolescents aged 9 to 16 who have already been infected by dengue. But, there currently is no vaccine to prevent the general population from contracting it. To protect yourself: •Use mosquito repellents, even indoors. •When outdoors, wear long-sleeved shirts and long pants tucked into socks. •When indoors, use air conditioning if available. •Make sure window and door screens are secure and free of holes. If sleeping areas are not screened or air conditioned, use mosquito nets. •If you have symptoms of dengue, speak to your doctor. To reduce the mosquito population, get rid of places where mosquitoes can breed. These include old tires, cans, or flower pots that collect rain. Regularly change the water in outdoor bird baths and pets' water dishes. If someone in your home gets dengue fever, be especially vigilant about efforts to protect yourself and other family members from mosquitoes. Mosquitoes that bite the infected family member could spread the infection to others in your home.
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  • 29.  How can pneumonia be described more exactly?  Experts also classify pneumonia according to factors other than where the patient was infected and the severity. But that typically doesn't affect how the pneumonia is treated. Instead, it's useful for getting a better description of the illness.  Atypical pneumonia  Typical pneumonia generally begins with a sudden high fever and chills, and then coughing with phlegm coming later.  Atypical pneumonia is caused by other germs, which are also referred to as "atypical." Older people in particular have fewer or slightly different symptoms if they have atypical pneumonia: It then starts off rather slowly with a mild fever and/or headache and aching Rather than coughing with phlegm, they have a dry, tickly cough.  Atypical symptoms don't mean that the lungs are less severely inflamed or that the disease will take a milder course though.  Upper, middle and lower lobe pneumonia  X-rays play an important role in distinguishing between these types: the term lobar pneumonia is used if an entire lung lobe is visibly inflamed. Depending on which lung lobe is affected, the pneumonia is referred to as upper, middle or lower lobe pneumonia.  If there are several multi-lobe focal inflammations in the lungs, the term focal pneumonia is used. Some people use the term bronchopneumonia if the focal inflammations started in inflamed airways (bronchi).  Sometimes, it's the air sacs that are more inflamed (alveolar pneumonia), and sometimes it's the tissue between the sacs (interstitial pneumonia).