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Clinical neurophysiology END-Techniques Case studies
Diabetic neuropathy ,[object Object],[object Object],[object Object]
Causes of diabetic neuropathies ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
symptoms ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
Types of diabetic neuropathy ,[object Object],[object Object],[object Object],[object Object],[object Object]
Principle of axonal degeneration:dying back hypothesis
Nerve conduction studies or electromyography   ,[object Object],[object Object],[object Object]
[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
Ulnar nerve entrapment
F-response ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
Axonal reflex In normal conditions, they are only seen in tibial nerve. In earlier GBS,IDD can be seen. They are also called IDD(Intermediate Double Dischares) or M-satellites.
 
Fine-fibers and autonomic neuropathy testing ,[object Object],[object Object],[object Object],[object Object],[object Object],[object Object]
Psychophysic  ,[object Object],[object Object],[object Object],[object Object]
Charcot-Marie-Tooth (CMT)  Hereditary Motor and Sensory Neuropathies   The legs showed mild atrophy of the anterior tibialis and peroneal muscles  Hypertrophic polyneuropathy. The most likely diagnosis was chronic inflammatory demyelinating polyneuropathy Her nerve conduction velocities (NCV) were 24.7m/s in both median and ulnar nerves without  multifocal blocks   Motor NCV of the right peroneal nerve was 14.7m/s; the motor NCV of the left ulnar nerve was 16.9m/s; and the motor NCV of the right median nerve was 21.2m/s  Lack of conduction blocks argued against chronic inflammatory demyelinating polyneuropathy (CIDP)  Based on the molecular studies and  electrophysiological examination , the diagnosis of Charcot-Marie-Tooth type 1A polyneuropathy was made.
[object Object],[object Object]
GBS ,[object Object],[object Object],[object Object]
Multiple Motor Neuropathy   ,[object Object],[object Object],[object Object],[object Object]
[object Object],[object Object],[object Object],[object Object],[object Object]
[object Object],[object Object],[object Object]
Conduction block
 
Absence of F-response
Conduction block outside of common entrapment sites.  The conduction block should not be an entrapment
EMG: Normal activity Fibrilation  ,[object Object],[object Object]
Muscle physiology
Motor neuron Degenerative motor neuron

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END-Techniques

  • 2.
  • 3.
  • 4.
  • 5.
  • 6. Principle of axonal degeneration:dying back hypothesis
  • 7.
  • 8.
  • 10.
  • 11. Axonal reflex In normal conditions, they are only seen in tibial nerve. In earlier GBS,IDD can be seen. They are also called IDD(Intermediate Double Dischares) or M-satellites.
  • 12.  
  • 13.
  • 14.
  • 15. Charcot-Marie-Tooth (CMT) Hereditary Motor and Sensory Neuropathies The legs showed mild atrophy of the anterior tibialis and peroneal muscles Hypertrophic polyneuropathy. The most likely diagnosis was chronic inflammatory demyelinating polyneuropathy Her nerve conduction velocities (NCV) were 24.7m/s in both median and ulnar nerves without multifocal blocks Motor NCV of the right peroneal nerve was 14.7m/s; the motor NCV of the left ulnar nerve was 16.9m/s; and the motor NCV of the right median nerve was 21.2m/s Lack of conduction blocks argued against chronic inflammatory demyelinating polyneuropathy (CIDP) Based on the molecular studies and electrophysiological examination , the diagnosis of Charcot-Marie-Tooth type 1A polyneuropathy was made.
  • 16.
  • 17.
  • 18.
  • 19.
  • 20.
  • 22.  
  • 24. Conduction block outside of common entrapment sites. The conduction block should not be an entrapment
  • 25.