This document summarizes a presentation on using trichometry and cross-sectional trichometry (CST) to objectively measure hair loss and monitor treatment outcomes. CST allows clinicians to accurately track changes in hair mass index (HMI) over time to determine treatment effectiveness and progression of hair loss. The document provides examples of how CST can answer questions about treatment responses, compare different treatments, and distinguish shedding from permanent hair loss. CST provides a quantitative tool to optimize hair loss management.
THE ART of SKIN & BODY CARE by ASEPTA LABORATOIRES MONACOAsepta Laboratoires
INNOVATIVE & HIGHLY EFFECTIVE TARGETED PROFESSIONAL TREATMENTS for professional & home use - MADE IN MONACO by ASEPTA LABORATOIRES MONACO | The Laboratory Expert in Dermo-Pharmacy and Leader in Foot Care since 1943.
Discover the benefits of ASEPTA world renown brands: AKILEINE, CYCALEINE, ONYKOLEINE, AKILDIA, AKILWINTER, HELIABRINE, HELIXIENCE, HELIASLIM, COUP D'ECLAT, ECRINAL
Androgenetic Alopecia or hereditary hair loss, considered by many as an outward and undesirable sign of premature aging that can be ‘seen from across the room,’ currently affects an estimated 50 Million men and 30 Million women in the U.S. Approximately 40% of men in their 40’s are experiencing hair loss and 50% of men in their 50’s with over a billion dollars spent in the U.S. annually on medical treatments, hair transplants, non-medical ‘cures’ and cover-ups to address the condition.
Hormone replacement therapy (HRT) that includes androgens/testosterone may accelerate the progression of unwanted androgenetic alopecia, especially in those patients whose hair follicles are highly androgen sensitive.
OBJECTIVE: Patients who undergo testosterone replacement may be at risk for hair loss related side effects. It is our goal to present an overview of the mechanisms of Androgenetic Alopecia, new diagnostic and monitoring methods, prevention/treatments options and management stratagems specifically for patients who are undergoing an Hormone replacement therapy regimen that includes testosterone.
MATERIALS/METHODS:
Chief Complaint & Goals
Medical History
Family history
Medical Conditions
Medications
Surgical History
Hair/Scalp Care
Lab Testing / Measurements
Genetic Testing
Cross Sectional Bundle Trichometry Measurements with Hair Check(TM)
Photography
Standardized Global Photos
Scalp Microscopic Photos
Treatment Plan
Recommended Tracking & Follow-Up Schedule
DISCUSSION
Identifying patients at risk for hereditary hair loss as well as being able to offer guidance and treatment options holds significant benefit for the Age Management (Anti-Aging) Physician and their Hormone Replacement patients because it:
1) enhances the risk/benefit discussion with the testosterone patient.
2) Identifies and addresses a possible side-effect and potential barrier to HRT compliance.
3) Adds a beneficial service to the Age Management Physician’s repertoire that may enhance patient retention and compliance.
4) Successful hair loss management adds a psychological boost to a Androgen Replacement patient’s outlook.
References:
THE ART of SKIN & BODY CARE by ASEPTA LABORATOIRES MONACOAsepta Laboratoires
INNOVATIVE & HIGHLY EFFECTIVE TARGETED PROFESSIONAL TREATMENTS for professional & home use - MADE IN MONACO by ASEPTA LABORATOIRES MONACO | The Laboratory Expert in Dermo-Pharmacy and Leader in Foot Care since 1943.
Discover the benefits of ASEPTA world renown brands: AKILEINE, CYCALEINE, ONYKOLEINE, AKILDIA, AKILWINTER, HELIABRINE, HELIXIENCE, HELIASLIM, COUP D'ECLAT, ECRINAL
Androgenetic Alopecia or hereditary hair loss, considered by many as an outward and undesirable sign of premature aging that can be ‘seen from across the room,’ currently affects an estimated 50 Million men and 30 Million women in the U.S. Approximately 40% of men in their 40’s are experiencing hair loss and 50% of men in their 50’s with over a billion dollars spent in the U.S. annually on medical treatments, hair transplants, non-medical ‘cures’ and cover-ups to address the condition.
Hormone replacement therapy (HRT) that includes androgens/testosterone may accelerate the progression of unwanted androgenetic alopecia, especially in those patients whose hair follicles are highly androgen sensitive.
OBJECTIVE: Patients who undergo testosterone replacement may be at risk for hair loss related side effects. It is our goal to present an overview of the mechanisms of Androgenetic Alopecia, new diagnostic and monitoring methods, prevention/treatments options and management stratagems specifically for patients who are undergoing an Hormone replacement therapy regimen that includes testosterone.
MATERIALS/METHODS:
Chief Complaint & Goals
Medical History
Family history
Medical Conditions
Medications
Surgical History
Hair/Scalp Care
Lab Testing / Measurements
Genetic Testing
Cross Sectional Bundle Trichometry Measurements with Hair Check(TM)
Photography
Standardized Global Photos
Scalp Microscopic Photos
Treatment Plan
Recommended Tracking & Follow-Up Schedule
DISCUSSION
Identifying patients at risk for hereditary hair loss as well as being able to offer guidance and treatment options holds significant benefit for the Age Management (Anti-Aging) Physician and their Hormone Replacement patients because it:
1) enhances the risk/benefit discussion with the testosterone patient.
2) Identifies and addresses a possible side-effect and potential barrier to HRT compliance.
3) Adds a beneficial service to the Age Management Physician’s repertoire that may enhance patient retention and compliance.
4) Successful hair loss management adds a psychological boost to a Androgen Replacement patient’s outlook.
References:
For many, hair loss can be a devastating psychological condition. Unfortunately, dermatologists and primary doctors often fall short when it comes to the state-of-the-art diagnosis and treatment. It's up to hair restoration physicians and integrative practices to take the lead in the medical management of hair loss. Helping patients maintain, enhance and restore their own living and growing hair is a rewarding field to be in. Mismanaging patients expectations or therapies will certainly lead to disappointment. In this webinar, learn an overview of how we diagnose, measure, treat and track hair loss patients on various effective medical regimens. We often prescribe finasteride and FinPlus compounded finasteride, Formula82M minoxidil, LaserCap and in-office laser therapy, PRP with ECM BioD ACell, nutraceuticals and FUE NeoGraft hair transplantation. See some before and afters from various monotherapy patients. HairCheck cross sectional hair bundle trichometry is the key to keeping patients compliant. Thank you to LaserCap
Additional training is available from Dr. Alan J Bauman so visit http://www.haircoach.net for upcoming hands-on PRP and HairCheck classes.
Looking for reviews and ratings for hair transplant surgeon Dr. Alan Bauman?
For many years, Dr. Alan J. Bauman and Bauman Medical Group has used a robust patient feedback process to continually improve patient care in hair restoration and their treatment of hair loss patients. Aside from written feedback forms and surveys, Real Patient Ratings is a google-verified HIPAA-compliant 3rd-party electronic feedback system that allows actual patients of Dr. Alan Bauman to provide feedback, complaints, concerns and reviews regarding consultations, treatments, procedures, hair transplants, PRP as well as patient care and results. Dr. Alan Bauman and his team strive daily to provide exceptional results and patient care above and beyond the accepted norms in the industry.
Visit Real Patient Ratings for authentic reviews from real patients of board certified hair restoration doctor, Dr. Alan J. Bauman of Bauman Medical Group in Boca Raton, Florida.
Approach to a case of diffuse hair loss in females
. Anagen effluvium-
(a)Dystrophic
(b)Loose anagen hair
2. Telogen effluvium –
(a)acute telogen effluvium
(b)Chronic telogen effluvium
3. Female pattern hair loss
Primary CTE –represents a primary disorder and is a diagnosis of exclusion.
Secondary CTE- secondary to variety of systemic disorders.
Iron deficiency
Other deficiency –protein calorie malnutrition ,zinc deficiency
Thyroid diseases
Metabolic diseases-chronic liver or renal failure, advanced malignancy, pancreatic disease and upper GI disorder with malabsorption
SLE and other connective tissue disorders.
HIV infection
Drug induced
Lecture by Dr. Patrick Treacy from Ailesbury Hair Clinics to ICAD 2014 Brazil on the reasons and treatments for female hair loss. Courtesy given at lecture to some other doctors and clinics for some images used. Images related to Ailesbury Hair Clinics were added at a alter time.
- Video recording of this lecture in English language: https://youtu.be/kqbnxVAZs-0
- Video recording of this lecture in Arabic language: https://youtu.be/SINlygW1Mpc
- Link to download the book free: https://nephrotube.blogspot.com/p/nephrotube-nephrology-books.html
- Link to NephroTube website: www.NephroTube.com
- Link to NephroTube social media accounts: https://nephrotube.blogspot.com/p/join-nephrotube-on-social-media.html
Basavarajeeyam is a Sreshta Sangraha grantha (Compiled book ), written by Neelkanta kotturu Basavaraja Virachita. It contains 25 Prakaranas, First 24 Chapters related to Rogas& 25th to Rasadravyas.
Muktapishti is a traditional Ayurvedic preparation made from Shoditha Mukta (Purified Pearl), is believed to help regulate thyroid function and reduce symptoms of hyperthyroidism due to its cooling and balancing properties. Clinical evidence on its efficacy remains limited, necessitating further research to validate its therapeutic benefits.
Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
263778731218 Abortion Clinic /Pills In Harare ,sisternakatoto
263778731218 Abortion Clinic /Pills In Harare ,ABORTION WOMEN’S CLINIC +27730423979 IN women clinic we believe that every woman should be able to make choices in her pregnancy. Our job is to provide compassionate care, safety,affordable and confidential services. That’s why we have won the trust from all generations of women all over the world. we use non surgical method(Abortion pills) to terminate…Dr.LISA +27730423979women Clinic is committed to providing the highest quality of obstetrical and gynecological care to women of all ages. Our dedicated staff aim to treat each patient and her health concerns with compassion and respect.Our dedicated group ABORTION WOMEN’S CLINIC +27730423979 IN women clinic we believe that every woman should be able to make choices in her pregnancy. Our job is to provide compassionate care, safety,affordable and confidential services. That’s why we have won the trust from all generations of women all over the world. we use non surgical method(Abortion pills) to terminate…Dr.LISA +27730423979women Clinic is committed to providing the highest quality of obstetrical and gynecological care to women of all ages. Our dedicated staff aim to treat each patient and her health concerns with compassion and respect.Our dedicated group of receptionists, nurses, and physicians have worked together as a teamof receptionists, nurses, and physicians have worked together as a team wwww.lisywomensclinic.co.za/
Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
These lecture slides, by Dr Sidra Arshad, offer a quick overview of the physiological basis of a normal electrocardiogram.
Learning objectives:
1. Define an electrocardiogram (ECG) and electrocardiography
2. Describe how dipoles generated by the heart produce the waveforms of the ECG
3. Describe the components of a normal electrocardiogram of a typical bipolar lead (limb II)
4. Differentiate between intervals and segments
5. Enlist some common indications for obtaining an ECG
6. Describe the flow of current around the heart during the cardiac cycle
7. Discuss the placement and polarity of the leads of electrocardiograph
8. Describe the normal electrocardiograms recorded from the limb leads and explain the physiological basis of the different records that are obtained
9. Define mean electrical vector (axis) of the heart and give the normal range
10. Define the mean QRS vector
11. Describe the axes of leads (hexagonal reference system)
12. Comprehend the vectorial analysis of the normal ECG
13. Determine the mean electrical axis of the ventricular QRS and appreciate the mean axis deviation
14. Explain the concepts of current of injury, J point, and their significance
Study Resources:
1. Chapter 11, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 9, Human Physiology - From Cells to Systems, Lauralee Sherwood, 9th edition
3. Chapter 29, Ganong’s Review of Medical Physiology, 26th edition
4. Electrocardiogram, StatPearls - https://www.ncbi.nlm.nih.gov/books/NBK549803/
5. ECG in Medical Practice by ABM Abdullah, 4th edition
6. Chapter 3, Cardiology Explained, https://www.ncbi.nlm.nih.gov/books/NBK2214/
7. ECG Basics, http://www.nataliescasebook.com/tag/e-c-g-basics
Adv. biopharm. APPLICATION OF PHARMACOKINETICS : TARGETED DRUG DELIVERY SYSTEMSAkankshaAshtankar
MIP 201T & MPH 202T
ADVANCED BIOPHARMACEUTICS & PHARMACOKINETICS : UNIT 5
APPLICATION OF PHARMACOKINETICS : TARGETED DRUG DELIVERY SYSTEMS By - AKANKSHA ASHTANKAR
For many, hair loss can be a devastating psychological condition. Unfortunately, dermatologists and primary doctors often fall short when it comes to the state-of-the-art diagnosis and treatment. It's up to hair restoration physicians and integrative practices to take the lead in the medical management of hair loss. Helping patients maintain, enhance and restore their own living and growing hair is a rewarding field to be in. Mismanaging patients expectations or therapies will certainly lead to disappointment. In this webinar, learn an overview of how we diagnose, measure, treat and track hair loss patients on various effective medical regimens. We often prescribe finasteride and FinPlus compounded finasteride, Formula82M minoxidil, LaserCap and in-office laser therapy, PRP with ECM BioD ACell, nutraceuticals and FUE NeoGraft hair transplantation. See some before and afters from various monotherapy patients. HairCheck cross sectional hair bundle trichometry is the key to keeping patients compliant. Thank you to LaserCap
Additional training is available from Dr. Alan J Bauman so visit http://www.haircoach.net for upcoming hands-on PRP and HairCheck classes.
Looking for reviews and ratings for hair transplant surgeon Dr. Alan Bauman?
For many years, Dr. Alan J. Bauman and Bauman Medical Group has used a robust patient feedback process to continually improve patient care in hair restoration and their treatment of hair loss patients. Aside from written feedback forms and surveys, Real Patient Ratings is a google-verified HIPAA-compliant 3rd-party electronic feedback system that allows actual patients of Dr. Alan Bauman to provide feedback, complaints, concerns and reviews regarding consultations, treatments, procedures, hair transplants, PRP as well as patient care and results. Dr. Alan Bauman and his team strive daily to provide exceptional results and patient care above and beyond the accepted norms in the industry.
Visit Real Patient Ratings for authentic reviews from real patients of board certified hair restoration doctor, Dr. Alan J. Bauman of Bauman Medical Group in Boca Raton, Florida.
Approach to a case of diffuse hair loss in females
. Anagen effluvium-
(a)Dystrophic
(b)Loose anagen hair
2. Telogen effluvium –
(a)acute telogen effluvium
(b)Chronic telogen effluvium
3. Female pattern hair loss
Primary CTE –represents a primary disorder and is a diagnosis of exclusion.
Secondary CTE- secondary to variety of systemic disorders.
Iron deficiency
Other deficiency –protein calorie malnutrition ,zinc deficiency
Thyroid diseases
Metabolic diseases-chronic liver or renal failure, advanced malignancy, pancreatic disease and upper GI disorder with malabsorption
SLE and other connective tissue disorders.
HIV infection
Drug induced
Lecture by Dr. Patrick Treacy from Ailesbury Hair Clinics to ICAD 2014 Brazil on the reasons and treatments for female hair loss. Courtesy given at lecture to some other doctors and clinics for some images used. Images related to Ailesbury Hair Clinics were added at a alter time.
- Video recording of this lecture in English language: https://youtu.be/kqbnxVAZs-0
- Video recording of this lecture in Arabic language: https://youtu.be/SINlygW1Mpc
- Link to download the book free: https://nephrotube.blogspot.com/p/nephrotube-nephrology-books.html
- Link to NephroTube website: www.NephroTube.com
- Link to NephroTube social media accounts: https://nephrotube.blogspot.com/p/join-nephrotube-on-social-media.html
Basavarajeeyam is a Sreshta Sangraha grantha (Compiled book ), written by Neelkanta kotturu Basavaraja Virachita. It contains 25 Prakaranas, First 24 Chapters related to Rogas& 25th to Rasadravyas.
Muktapishti is a traditional Ayurvedic preparation made from Shoditha Mukta (Purified Pearl), is believed to help regulate thyroid function and reduce symptoms of hyperthyroidism due to its cooling and balancing properties. Clinical evidence on its efficacy remains limited, necessitating further research to validate its therapeutic benefits.
Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
263778731218 Abortion Clinic /Pills In Harare ,sisternakatoto
263778731218 Abortion Clinic /Pills In Harare ,ABORTION WOMEN’S CLINIC +27730423979 IN women clinic we believe that every woman should be able to make choices in her pregnancy. Our job is to provide compassionate care, safety,affordable and confidential services. That’s why we have won the trust from all generations of women all over the world. we use non surgical method(Abortion pills) to terminate…Dr.LISA +27730423979women Clinic is committed to providing the highest quality of obstetrical and gynecological care to women of all ages. Our dedicated staff aim to treat each patient and her health concerns with compassion and respect.Our dedicated group ABORTION WOMEN’S CLINIC +27730423979 IN women clinic we believe that every woman should be able to make choices in her pregnancy. Our job is to provide compassionate care, safety,affordable and confidential services. That’s why we have won the trust from all generations of women all over the world. we use non surgical method(Abortion pills) to terminate…Dr.LISA +27730423979women Clinic is committed to providing the highest quality of obstetrical and gynecological care to women of all ages. Our dedicated staff aim to treat each patient and her health concerns with compassion and respect.Our dedicated group of receptionists, nurses, and physicians have worked together as a teamof receptionists, nurses, and physicians have worked together as a team wwww.lisywomensclinic.co.za/
Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
These lecture slides, by Dr Sidra Arshad, offer a quick overview of the physiological basis of a normal electrocardiogram.
Learning objectives:
1. Define an electrocardiogram (ECG) and electrocardiography
2. Describe how dipoles generated by the heart produce the waveforms of the ECG
3. Describe the components of a normal electrocardiogram of a typical bipolar lead (limb II)
4. Differentiate between intervals and segments
5. Enlist some common indications for obtaining an ECG
6. Describe the flow of current around the heart during the cardiac cycle
7. Discuss the placement and polarity of the leads of electrocardiograph
8. Describe the normal electrocardiograms recorded from the limb leads and explain the physiological basis of the different records that are obtained
9. Define mean electrical vector (axis) of the heart and give the normal range
10. Define the mean QRS vector
11. Describe the axes of leads (hexagonal reference system)
12. Comprehend the vectorial analysis of the normal ECG
13. Determine the mean electrical axis of the ventricular QRS and appreciate the mean axis deviation
14. Explain the concepts of current of injury, J point, and their significance
Study Resources:
1. Chapter 11, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 9, Human Physiology - From Cells to Systems, Lauralee Sherwood, 9th edition
3. Chapter 29, Ganong’s Review of Medical Physiology, 26th edition
4. Electrocardiogram, StatPearls - https://www.ncbi.nlm.nih.gov/books/NBK549803/
5. ECG in Medical Practice by ABM Abdullah, 4th edition
6. Chapter 3, Cardiology Explained, https://www.ncbi.nlm.nih.gov/books/NBK2214/
7. ECG Basics, http://www.nataliescasebook.com/tag/e-c-g-basics
Adv. biopharm. APPLICATION OF PHARMACOKINETICS : TARGETED DRUG DELIVERY SYSTEMSAkankshaAshtankar
MIP 201T & MPH 202T
ADVANCED BIOPHARMACEUTICS & PHARMACOKINETICS : UNIT 5
APPLICATION OF PHARMACOKINETICS : TARGETED DRUG DELIVERY SYSTEMS By - AKANKSHA ASHTANKAR
ABDOMINAL TRAUMA in pediatrics part one.drhasanrajab
Abdominal trauma in pediatrics refers to injuries or damage to the abdominal organs in children. It can occur due to various causes such as falls, motor vehicle accidents, sports-related injuries, and physical abuse. Children are more vulnerable to abdominal trauma due to their unique anatomical and physiological characteristics. Signs and symptoms include abdominal pain, tenderness, distension, vomiting, and signs of shock. Diagnosis involves physical examination, imaging studies, and laboratory tests. Management depends on the severity and may involve conservative treatment or surgical intervention. Prevention is crucial in reducing the incidence of abdominal trauma in children.
Basavarajeeyam is an important text for ayurvedic physician belonging to andhra pradehs. It is a popular compendium in various parts of our country as well as in andhra pradesh. The content of the text was presented in sanskrit and telugu language (Bilingual). One of the most famous book in ayurvedic pharmaceutics and therapeutics. This book contains 25 chapters called as prakaranas. Many rasaoushadis were explained, pioneer of dhatu druti, nadi pareeksha, mutra pareeksha etc. Belongs to the period of 15-16 century. New diseases like upadamsha, phiranga rogas are explained.
1. CROSS SECTION
TRICHOMETRY
UPDATE
A NEW TOOL FOR MANAGING
PATIENTS WITH HAIR LOSS
ISHRS – ANNUAL SCIENTIFIC MEETING
Nassau, Bahamas – October 18-21, 2012
Bernard Cohen, MD
Diplomate, American Board of Dermatology
Diplomate, American Board of Hair Restoration Surgery
18. HMI
(BUNDLE CROSS SECTION)
will change if or when
A full-sized hair falls out
A miniaturized hair eventually vanishes
A full-sized hair emerges from the skin
A hair diameter increases
A hair diameter decreases
22. With CST you’ll be able to answer these questions:
How quickly is untreated AGA getting worse? What percent per year?
Is it time to advance my patient from MNX to FIN?
Does FIN work as well if taken 2x per week?
Is MNX 2% bid equivalent to MNX 5% daily?
Is generic MNX equivalent to Rogaine?
Is MNX more effective with added RetinA?
Do HairMax and LaserCap work better than MNX?
Is there an added benefit of adding MNX and/or laser to FIN?
If I correct a patient’s iron deficiency, does her shedding improve?
24. Questions that CST can easily answer:
How quickly is untreated AGA getting worse? What percent per year?
Should I advance my patient from MNX to FIN?
Does FIN work as well if taken 2x per week?
Is MNX 2% bid equivalent to MNX 5% daily?
Is generic MNX equivalent to Rogaine?
Is MNX more effective with added RetinA?
Do HairMax and LaserCap work better than MNX?
Is there an added benefit of adding MNX and/or laser to FIN?
If I correct a patient’s iron deficiency, does her shedding improve?
26. Questions that CST can easily answer:
How quickly is untreated AGA getting worse? What percent per year?
Is it time to advance my patient from MNX to FIN?
Does FIN work as well if taken 2x per week?
Is MNX 2% bid equivalent to MNX 5% daily?
Is generic MNX equivalent to Rogaine?
Is MNX more effective with added RetinA?
Do HairMax and LaserCap work better than MNX?
Is there an added benefit of adding MNX and/or laser to FIN?
If I correct a patient’s iron deficiency, does her shedding improve?
27. on FINASTERIDE 1mgm daily
reduce to1mgm 2x/wk
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Theoretical example – NOT clinical observation
One year later
28. Questions that CST can easily answer:
How quickly is untreated AGA getting worse? What percent per year?
Is it time to advance my patient from MNX to FIN?
Does FIN work as well if taken 2x per week?
Is MNX 2% bid equivalent to MNX 5% daily?
Is generic MNX equivalent to Rogaine?
Is MNX more effective with added RetinA?
Do HairMax and LaserCap work better than MNX?
Is there an added benefit of adding MNX and/or laser to FIN?
If I correct a patient’s iron deficiency, does her shedding improve?
29. MNX 2% bid x 1 year
Change to MNX 5% once
daily
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MNX 5% once daily
at end of following year
Theoretical example – NOT clinical observation
30. Questions that CST can easily answer:
How quickly is untreated AGA getting worse? What percent per year?
Is it time to advance my patient from MNX to FIN?
Does FIN work as well if taken 2x per week?
Is MNX 2% bid equivalent to MNX 5% daily?
Is generic MNX equivalent to Rogaine?
Is MNX more effective with added RetinA?
Do HairMax and LaserCap work better than MNX?
Is there an added benefit of adding MNX and/or laser to FIN?
If I correct a patient’s iron deficiency, does her shedding improve?
31. MNX 5% generic
at end of year #1
Switch to Rogaine 5%
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Rogaine 5%
at end of following year
Theoretical example – NOT clinical observation
32. Questions that CST can easily answer:
How quickly is untreated AGA getting worse? What percent per year?
Is it time to advance my patient from MNX to FIN?
Does FIN work as well if taken 2x per week?
Is MNX 2% bid equivalent to MNX 5% daily?
Is generic MNX equivalent to Rogaine?
Is MNX more effective with added RetinA?
Do HairMax and LaserCap work better than MNX?
Is there an added benefit of adding MNX and/or laser to FIN?
If I correct a patient’s iron deficiency, does her shedding improve?
34. Questions that CST can easily answer:
How quickly is untreated AGA getting worse? What percent per year?
Is it time to advance my patient from MNX to FIN?
Does FIN work as well if taken 2x per week?
Is MNX 2% bid equivalent to MNX 5% daily?
Is generic MNX equivalent to Rogaine?
Is MNX more effective with added RetinA?
Do HairMax and LaserCap work better than MNX?
Is there an added benefit of adding MNX and/or laser to FIN?
If I correct a patient’s iron deficiency, does her shedding improve?
35. On MNX 5% bid x 3 years
then switch patient
to Laser Rx
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24 MONTHS LATER
Theoretical example – NOT clinical observation
36. Questions that CST can easily answer:
How quickly is untreated AGA getting worse? What percent per year?
Is it time to advance my patient from MNX to FIN?
Does FIN work as well if taken 2x per week?
Is MNX 2% bid equivalent to MNX 5% daily?
Is generic MNX equivalent to Rogaine?
Is MNX more effective with added RetinA?
Do HairMax and LaserCap work better than MNX?
Is there an added benefit of adding MNX and/or laser to FIN?
If I correct a patient’s iron deficiency, does her shedding improve?
37. FIN 1mgm daily
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FIN 1 mgm daily
plus Laser Rx
Theoretical example – NOT clinical observation
38. Questions that CST can easily answer:
How quickly is untreated AGA getting worse? What percent per year?
Is it time to advance my patient from MNX to FIN?
Does FIN work as well if taken 2x per week?
Is MNX 2% bid equivalent to MNX 5% daily?
Is generic MNX equivalent to Rogaine?
Is MNX more effective with added RetinA?
Do HairMax and LaserCap work better than MNX?
Is there an added benefit of adding MNX and/or laser to FIN?
If I correct a patient’s iron deficiency, does her shedding improve?
40. Questions that CST can easily answer:
When has the woman with telogen effluvium stabilized?
My patient is convinced that MNX has made her “worse.”
How does FUE affect donor site density?
What percent improvement does my patient have after HT surgery?
Is this woman’s hair loss caused by shedding or AGA?
.
42. Questions that CST can easily answer:
When has the woman with telogen effluvium stabilized?
My patient is convinced that MNX has made her “worse.”
How does FUE affect donor site density?
What percent improvement does my patient have after HT surgery?
Is this woman’s hair loss caused by shedding or AGA?
.
43. Baseline HMI, then
4 months of MNX.
Phone call: “I’m much worse!”
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On re-exam
the next day
she is no worse.
44. Questions that CST can easily answer:
When has the woman with telogen effluvium stabilized?
My patient is convinced that MNX has made her “worse.”
How does FUE affect donor site density?
What percent improvement does my patient have after HT surgery?
Is this woman’s hair loss caused by shedding or AGA?
.
46. Questions that CST can easily answer:
When has the woman with telogen effluvium stabilized?
My patient is convinced that MNX has made her “worse.”
How does FUE affect donor site density?
What percent improvement does my patient have after HT surgery?
Is this woman’s hair loss caused by shedding or AGA?
.
48. Questions that CST can easily answer:
When has the woman with telogen effluvium stabilized?
My patient is convinced that MNX has made her “worse.”
How does FUE affect donor site density?
What percent improvement does my patient have after HT surgery?
Is this woman’s hair loss caused by shedding or AGA?
.