2. INTRODUCTION
ā¢ Temporomandibular joint (TMJ) is a unique joint in which translatory as
well as rotational movements are possible & where both the ends of bone
articulate, in the same plane, with that of other bone.
ā¢ It is also called as ginglymodiarthrodial type of joint, meaning that it has a
relatively sliding type of movement between bony surfaces, in addition to
hinge movement, common to diarthrodial joint.
3. COMPONENTS OF TEMPOROMANDIBULAR JOINT
a) Glenoid Fossa & Articular Eminence/ Protuberance
b) Mandibular Condyle
c) Articular Capsule and Articular Disk
d) Synovial Fluid
e) Discal Ligaments
f) Posterior Attachment or Retrodiscal Tissue or Bilaminar Zone
g) Ligaments Associated with TMJ
h) Muscles of Mastication
i) Arterial Supply, Venous Drainage & Sensory Innervation of TMJ
4. ā¢ The structure of the TMJā¢
(A) Upper joint space;
(B) lower joint space;
(C) interarticular disc;
(D) condylar head;
(E) lateral pterygoid muscle superior head;
(F) lateral pterygoid muscle inferior head
(G) mandibular fossa;
(H) articular eminence;
(I) external auditory canal
8. Schematic diagram showing the
location of the temporalis & masseter
muscle
Schematic diagram
showing the medial
pterygoid muscle
Schematic diagram showing the
medial pterygoid muscle
9.
10.
11.
12. TMJD
ā¢ Temporomandibular disorders (TMD) are characterized by craniofacial pain
involving the joint, masticatory muscles, or muscle innervations of the head
& neck. TMD is a major cause of nondental pain in the orofacial region.
ā¢ TMD affects 10% to 15% of adults, but only 5% seek treatment.
ā¢ The incidence of TMD peaks from 20 to 40 years of age; it is twice as
common in women than in men & carries a significant financial burden
from loss of work.
13. ā¢ Symptoms can range from mild discomfort to debilitating pain, including
limitations of jaw function.
ā¢ The most common syndromes are: myofascial pain disorder, disk derangement
disorders, osteoarthritis, & autoimmune disorders.
14. Etiology
ā¢ The etiology of TMD is multifactorial & includes:
1. Biologic,
2. Environmental,
3. Social,
4. Emotional,
5. Cognitive triggers.
ā¢ Factors consistently associated with TMD include other pain conditions (e.g., chronic
headaches), fibromyalgia, autoimmune disorders, sleep apnea, and psychiatric illness.
15. ļ± A study showed a twofold increase in TMD in persons with depression
& an increase in myofascial pain in persons with anxiety.
ļ± Smoking is associated with an increased risk of TMD in females
younger than 30 years.
17. ļ± In 2013, the International Research Diagnostic Criteria for TMD
Consortium Network published an updated classification structure for
TMD.
ļ± TMD is categorized as:
1. Intra-articular (within the joint)
2. Extra-articular (involving the surrounding musculature)
18. 1. Articular disorders (intra-articular)
1. Congenital/developmental disorders
2. Degenerative joint disorders
3. Disk derangement disorders
4. Infection
5. Neoplasia
6. TM hypermobility
7. TM hypomobility
8. Trauma
a) Condylar hyperplasia
b) First and second branchial arch disorders
c) Idiopathic condylar resorption
a) Inflammatory: capsulitis, synovitis,
polyarthritides (rheumatoid arthritis,
psoriatic arthritis, ankylosing spondylitis,
Reiter syndrome, gout)
b) Noninflammatory: osteoarthritis
a) Displacement with reduction
b) Displacement without reduction (closed lock)
c) Perforation
a) Dislocation
b) Joint laxity
c) Subluxationa) Ankylosis: true ankylosis (bony or
fibrous) or pseudoankylosis
b) Postradiation fibrosis
c) Trismus
a) Contusion
b) Fracture
c) Intracapsular
hemorrhage
21. A. Clinical Examination:
The dental examination should be systematic & include the TMJ & the
masticatory muscles.
1) Joint examination
2) Muscle examination
22. Joint examination
Movement: Face the patient and ask him/her to open slowly to maximum.
Normal range (interincisal) is 35ā40 mm. If opening is thought to be
reduced, ask whether the limiting factor is pain or an obstruction. Note the
path of opening and any lateral deviation.
Pain on palpation: Palpate in front of the ear & within the external auditory
meatus.
23. Joint examination
Auscultation: This needs a stethoscope to be done properly. However, clicks
may well be audible without a stethoscope. A click implies a disc
displacement that reduces into a normal position on opening. Crepitus
(cracking/grating noise) implies degenerative change or, sometimes, acute
inflammation.
24. Muscle examination
ļ± Muscle tenderness suggests some abnormal function (clenching, bruxism).
I. Masseter & temporalis muscles are assessed by direct palpation.
II. The lateral pterygoid is indirectly examined by noting the response (in
terms of any preauricular pain) to attempted opening against the restriction
of the examinerās hand below the chin.
III. The medial pterygoid cannot be examined.
25. Radiology
ļ± Most clinical problems related to the TMJ are caused by muscular
parafunction (e.g. bruxism) or internal disc derangements. Neither is
likely to be associated with any relevant bony abnormalities.
ļ± So, radiography is not normally indicated unless there is any suggestion of
bony abnormality, such as might be the case in rheumatoid arthritis or
osteoarthrosis.
26. Radiology
A clinical diagnosis of suspected internal derangement might lead to a
requirement for imaging of the disc.
1) Panoramic X-ray
2) magnetic resonance imaging (MRI)
27. Arthroscopy
ļ± Arthroscopy allows visual examination of the upper joint space and an
opportunity for minor surgical treatment. A small arthroscope can be used
to facilitate lavage & division of joint adhesions.
ļ± The lower joint space is difficult to access without risk of damage to the
articular disc.
ļ± Arthroscopy is undertaken under local anesthesia; however, if lengthy
arthroscopic surgery is to be undertaken, then a conscious sedation
technique would be appropriate or even general anesthesia.
29. 1) Pain/dysfunction
ā¢ The most common TMJ disorder is pain or dysfunction.
Clinical features Symptoms are a combination of:
ā¢ Headache
ā¢ Limitation/deviation of jaw opening
ā¢ Joint sounds
ā¢ Pain on palpation of the TMJ
ā¢ Pain on palpation of the associated muscles.
Joint sounds alone, or with
headache, are not
diagnostic of TMJ
pain/dysfunction.
30. Radiology
There is no abnormality visible.
Management
ā¢ Reassurance & explanation to patients.
ā¢ Jaw rest & soft diet.
ā¢ Analgesics/anti-inflammatory drugs.
ā¢ Occlusal splints to interfere with parafunction may offer some help.
ā¢ Physiotherapy.
ā¢ Muscle relaxants.
31. 2) Internal derangement
ļ§ The articular disc normally sits above the anterior aspect of the condylar
head, with the disc posterior attachment lying within 10Ā° of the vertical.
ļ§ A disc may be anterior to this ānormalā position in asymptomatic individuals,
suggesting that an anterior disc position is a normal variant.
32. 2) Internal derangement
ļ§ Thus, an internal derangement is best thought of as an abnormality in
position that interferes with function & that may be associated with other
symptoms.
ļ§ An anterior disc ādisplacementā is the most common internal derangement,
but anteromedial, medial, & anterolateral displacements are all seen.
33.
34. 3) Disc displacement with reduction
ā¢ Reduction means that a displaced disc āreducesā into a normal position
on opening but reverts to an abnormal position on closing (reciprocal
click).
ā¢ Clinical features:
1. Clicking on opening.
2. Clicking on closing.
3. Transient jaw deviation during opening/closing
35. Radiology
ā¢ No abnormalities are apparent on plain radiographs.
ā¢ MR imaging shows the displaced disc in a closed/rest position
36. (A) A displaced disc with reduction showing the movement diagrammatically
(B) On MRI in closed & open positions
closed
open
(B)
(A)
37. Management
1. Consider no treatment other than reassurance & explanation.
2. Occlusal splints to interfere with parafunction may offer some help.
3. Physiotherapy:
ļ It should be emphasized that treatment should only be considered where
the abnormality is affecting the patientās quality of life; a clicking joint
may be considered as normal
38. 4) Disc displacement without reduction
ā¢ If there is no reduction, a displaced disc remains in a displaced position
regardless of the stage of opening.
ā¢ This interferes with movement & may cause pain.
ā¢ Clinical features:
1. Reduction in opening.
2. In unilateral cases, lasting deviation on opening.
3. No click.
4. Pain may be present in front of the ear
39. Radiology
ā¢ Plain films usually show nothing.
ā¢ In long-standing cases, there may be signs of osteoarthrosis.
ā¢ MR imaging shows an abnormal disc position in all movements
ā¢ In long-standing cases, perforation of the disc may be seen & joint space
adhesions inferred
40. (A) A displaced disc without reduction showing the movement diagrammatically &
(B) on MR in closed and open positions
41. Management
ā¢ Explanation of the condition and reassurance.
ā¢ Muscle relaxants and physiotherapy.
ā¢ Manipulation under anesthetic.
ā¢ TMJ surgery.
44. Osteoarthrosis
ļ± Osteoarthrosis is a non-inflammatory disorder of joints in which there is
joint deterioration with bony proliferation.
ļ± The deterioration leads to loss of articular cartilage & bone erosions.
ļ± The proliferation manifests as new bone formation at the joint periphery
and subchondrally.
45. It has an unknown etiology, but previous trauma, parafunction & internal
derangements are all suggested as etiological factors.
C/F:
1) Pain localized to the TMJ region.
2) Limitation of opening, worse with prolonged function.
3) Crepitus.
4) Tender on palpation of TMJ.
46. Radiology
ā¢ Plain films show:
1. Erosions of the articular surfaces of the condyle &, less commonly seen, of
the mandibular fossa.
2. Sclerosis of the bone & marginal bony proliferation (ālippingā or
osteophytes) are & narrowing of the radiographic joint space.
3. Bony proliferations may break away & be seen as loose bodies in the joint
space.
48. Management
ā¢ Explanation & reassurance.
ā¢ Anti-inflammatory drugs.
ā¢ Physiotherapy.
ā¢ Restore deficiencies in the posterior occlusion to reduce loading on TMJs.
ā¢ Intra-articular steroid injections (advanced disease).
ā¢ Surgery (advanced disease; final option) to smooth irregular condylar head
where there are osteophytes or irregularities.
50. C/F:
ā¢ Pain over TMJs.
ā¢ Tenderness over TMJs.
ā¢ Swelling over TMJs.
ā¢ Stiffness & limitation of opening.
ā¢ Crepitus.
ā¢ Developing anterior open bite & retrusion of chin in advanced disease.
ā¢ Joints of hands, wrists, knees & feet commonly involved.
51. Radiology
Radiology demonstrates reduction in bone density in the TMJ. There is
marked erosion of the condylar head & articular fossa & narrowing of the
joint space.
In long-standing disease, there is:
ā¢ Destruction of entire condyle
ā¢ Anterior open bite
ā¢ Secondary osteoarthrosis
ā¢ Ankylosis.
53. Juvenile chronic (rheumatoid) arthritis
ļ± Juvenile chronic rheumatoid arthritis differs from rheumatoid arthritis in
the age of onset (mean age 5 years), the severe systemic involvement & the
absence (in some cases) of rheumatoid factor.
ļ± While it shares clinical & radiological features with rheumatoid arthritis,
the age of onset means that there is often a severe effect on mandibular
growth, leading to a ābird faceā appearance owing to the mandibular
retrusion, often accompanied by an anterior open bite.
54. ļ± The disease often has periods of remission/quiescence, during which
time the erosions of the joint may āsmooth overā with formation of a new
cortex.
ļ± Ankylosis may occur.
55. Effusion
ļ± Effusion is influx of fluid into the joint, usually either bleeding
following trauma or inflammatory exudate.
ļ± C/F:
ā¢ Pain over joint.
ā¢ Swelling over joint.
ā¢ Limitation of movement.
ā¢ Sensation of a blocked ear.
ā¢ Difficulty in occluding posterior teeth.
56. Radiology
There is a widened joint space.
Management
ā¢ Anti-inflammatory drugs.
ā¢ Rarely, surgical drainage may be needed.
57. Dislocation
ļ In dislocation of the TMJ, the condyle is abnormally positioned outside
the mandibular fossa but within the joint capsule.
ļ Dislocation may occur during trauma or be caused by failure of
muscular coordination.
59. Radiology
Radiography confirms a clinical diagnosis.
ā¢ The condyle may translate beyond the articular eminence normally,
without a dislocation, so clinical information is essential.
ā¢ The condyle will be anterior and superior to the āsummitā of the
articular eminence.
60. Management
ā¢ Manual manipulation to reduce the dislocation.
ā¢ Intravenous sedation with midazolam provides muscle relaxation &
greatly facilitates this maneuver.
ā¢ The patient should avoid wide mouth opening for some days & use the
hand to prevent this when yawning.
62. Ankylosis
ļ¶ Fusion across a TMJ may occur as a result of trauma, mastoid infection
or juvenile chronic arthritis.
ļ¶ Surgical treatment is by joint replacement with a prosthetic joint unless
the patientās facial development is not yet complete, when a
costochondral (rib) graft is used in an attempt to provide a bony
replacement that may grow.
63. MCQs
ā¢ Temporomandibular joint is a unique of
(a) Ginglymodiarthrodial type
(b) Diarthrodial type
(c) Synarthrodial
(d) Amphiarthrodial
65. Short Question
ā¢ Classify TMJ disorders.
ā¢ Enumerate various traumatic disorders of TMJ.
ā¢ Write in detail about clinical features, radiological features and
management of Disc displacement without reduction.
66. Case -1-
An 11-year-old patient, following trauma to the jaw as an infant,
developed a worsening facial asymmetry and now has extreme limitation of
opening. He has no other joint problems in the body.
1. What diagnosis is suggested?
2. What would be the management?
67. Case -2-
A 35-year-old woman with a long history of clicking and occasional pain from her
right TMJ presents with an acute event, consisting of limited opening, deviation of
the jaw towards the affected side on attempted opening and pain localized to the
joint region. These symptoms were present upon waking up in the morning and have
been unchanged since. She mentions that the click that has always been there is no
longer present.
1. What diagnosis is suggested?
2. What would be the management?