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PRESENTED BY:
DR GAYATRI MEHROTRA
ī‚Ą Temporomandibular Disorders (TMDs),
also referred to as craniomandibular
disorders, consist of a group of pathologies
affecting the masticatory muscles, the
tempromandibular joint (TMJ) and related
structures.
ī‚Ą TMD’s RDC groups are:
I Group: Muscle disorders:
Ia) Myofascial pain
Ib) Myofascial pain with limited opening
II Group: Disc Displacement (DD):
IIa) DD with reduction
IIb) DD without reduction with limited
opening
IIc) DD without reduction without limited
opening
III Group: Other common joint disorders:
IIIa) Arthralgia
IIIb) Osteoarthritis
IIIc) Ostoarthrosis
“ TMD is a complex and multifactorial
condition; so also our patients”- JPO
Disc derangement
disorder
Masticatory muscle
disorder
Inflammatory
disorder
Mandibular
hypmobility disorder
Trauma
īļThose which provide
basically conservative
reversible therapy
īļNon conservative,
irreversible therapy
All treatment methods
used for TMD can be
categorized into:
1) Definitive
treatment
2) Supportive
therapy
DEFINITIVE
THERAPY
ī‚Ą Intended to directly eliminate or alter the
causes of a disorder
Normal
function
An
event
Physiologic
tolerance
Temporomandibular
disorder symptoms
Definitive
treatment
For
occlusal
factors
For
emotional
stress
For trauma
For deep pain
input and
parafunction
al activity
DEFINITIVE TREATMENT CONSIDERATIONSFOR OCCLUSAL
FACTORS
ī‚Ą Reversible Occlusal therapy:
ī‚Ą Best accompalished with occlusal appliance
ī‚Ą Irreversible occlusal therapy:
Selective
grinding of
teeth
Restorative
procedures
Orthodontic
treatment
Surgical
procedures
DEFINITIVE TREATMENT CONSIDERATIONSFOR
EMOTIONAL STRESS
ī‚Ą Emotional stress is one of several
pyschologic factors that need to be
considered
ī‚Ą Increase emotional stress Increase
resting activity
alters muscle function or bruxism
Common
personality traits:
īļPerfectionistic
īļCompulsive
īļIntrovert
īļNeurotic
īļDis-satisfied and
self destructive
Common emotional
stress:
īļHigh level of
anxiety
īļApprehension
īļFrustation
īļAnger
īļFear
īļDepression
īļHostility
TMD patients with chronic fear
Increase level of anxiety,
frustation and anger
Increased level of emotional
stress
Contribute to TMD
ī‚Ą Types of emotional stress therapy:
1) Patient awareness-
ī‚Ą Educate the patient and make the patient
aware that stress is a common everyday
experience
2) Restrictive use-
ī‚Ą Painful movements should be avoided as
it may further lead to cyclic muscle pain
ī‚Ą Alter the diet
3) Voluntary avoidance-
ī‚Ą Patient should be instructed that teeth
should be quickly disengaged any time
they come in contact.
ī‚Ą Other oral habits like biting on objects or
cradling telephone between mandible and
shoulder aggravate TMD symptom
â€ĸ Buffering a little air between lips and teeth
â€ĸ Allows jaw to assume a relaxed position
â€ĸ Lips brought together and teeth left slightly apart
â€ĸ This position decreases muscle activity
â€ĸ Minimizes interarticular pressure
â€ĸ Promotes joint repair
4) Relaxation Therapy -
īļ Substitutive Relaxation therapy:
ī‚§ Substitution for stress (any activity that is
enjoyable- sports, hobbies or recreational
activity)
īļ Active relaxation therapy:
ī‚§ Technique used in dentistry are
modification of Jacobsons method,
developed in 1968.
Pt is trained to
relax symptomatic
muscles voluntarily
Blood flow
to these
tissues is
increased
Metabolic
waste
substances
that
stimulate
pain
receptors
are
eliminated
Diminishes
pain
ī‚Ą Patient tenses the muscles and then relaxes
them untill relaxed state can be felt and
maintained
ī‚Ą Another form uses a reverse approach
( muscles are passsively stretched and
relaxed)
ī‚Ą This technique has one major advantage
over Jacobson technique
ī‚Ą Self hypnosis, meditation and yoga also
promote relaxation
5) Biofeedback –
ī‚Ą It is a technique that assist
patient in regulating bodily
functions that are generally
controlled unconsciously.
ī‚Ą It is accompalished by
electromyographically monitoring state of
muscles through surface electrodes placed
over muscles to be monitored.
ī‚Ą Biofeedback units give auditory feedback
ī‚Ą When patient clenches, high readings will
appear or elevated tone is heard
ī‚Ą Negative Biofeedback:
ī‚Ą Devices are small and worn throughout day
and night
ī‚Ą If clenching or bruxism occurs, biofeedback
mechanism is activated and loud sound is
heard.
ī‚Ą A review of 30 studies of physical therapy
modalities for treating TMD concluded that
biofeedback may be more effective than
placebo or occlusial splints
ī‚Ą (Medlicott MS, Harris SR. A systematic review of the effectiveness of
exercise, manual therapy, electrotherapy, relaxation training, and
biofeedback in the management of temporomandibular disorder. Phys
Ther 2006;86:955–973.)
DEFINITIVE TREATMENT CONSIDERATIONSFOR trauma
ī‚Ą Trauma is one of the causative factor that
can lead to TMD
īļMacrotrauma:
ī‚Ą Only supportive therapy is helpful (trauma
has already occurred, so definitive therapy
is of no use)
ī‚Ą Preventive measures are helpful
īļMicrotrauma
ī‚Ą Result from repeated loading of joint
structures, such as bruxism or clencing
ī‚Ą Occlusal appliances help in achieving a
favorable condyle disc relationship that will
unload retrodiscal tissues and load the disc.
DEFINITIVE TREATMENT CONSIDERATIONSFOR deep pain
input
ī‚Ą Some TMD may be secondary to another
source of deep pain eg cervical injury
ī‚Ą Appropriate history and examination
DEFINITIVE TREATMENT CONSIDERATIONSFOR
parafunctional activity
ī‚Ą For many years, dentists were convinsed
that bruxism and clenching were major
causes of TMD.
ī‚Ą This is true, but not always the case
ī‚Ą Keep the teeth apart
ī‚Ą Nocturnal bruxism- (more due to emotional
stress level and sleep pattern)
ī‚Ą Effectively reduced by occlusal applianec
therapy
ī‚Ą Morning pain associated with nocturnal
bruxism
SUPPORTIVE
THERAPY
ī‚Ą Supportive therapy mainly includes:
īļ Pharmacologic therapy
īļ Physical therapy
PHARMACOLOGICTHERAPY
ī‚§ If medications are prescribed on “take as
needed” basis, then drugs are abused by
patients.
ī‚§ Continued use lead to frequent pain cycles
and less drug effectiveness
ī‚§ Analgesics and Tranquilizers
ī‚Ą Acute TMD pain
īļ Analgesics
īļ Corticosteroids
īļ Anxiolytics
ī‚§ Chronic orofacial pain
īļ Tricyclic
antidepressants
ī‚Ą Both acute and
chronic condition
īļ NSAID
īļ Muscle relaxant
īļ Local Anesthetics
DRUGS USED
1) Analgesics:
ī‚Ą Important part of supportive therapy
ī‚Ą Opiates or non opiates
ī‚Ą First medication of choice for moderate pain
relief is Calpol (acetaminophen)
ī‚Ą Stronger analgesics (codeine combined with
either salicylate or acetaminophen can be
helpful)
2) Non Steroidal antiinflammatory drugs:
ī‚Ą Chief application is in the treatment of
muscoloskeletal pain
ī‚Ą In presence of tissue injury, that certain
chemical mediators are released into the
injured site.
ī‚Ą NSAID inhibit action of cycloxygenase, an
enzyme used to synthesize prostaglandin.
NSAID
INDOLES
â€ĸIndomethacin
PROPIONIC
ACID
DERIVATIVE
â€ĸIbuprofen
â€ĸNaproxen
â€ĸFenoprofen
COX-2
INHIBITOR
S
â€ĸCelecoxib
â€ĸRofecoxib
3) Anti-inflammatory agents:
ī‚Ą These drugs do not immediately achieve
good blood levels, therefore need to be
taken on a regular schedule for a
minimum of 3 weeks
4) Corticosteroids
ī‚Ą Significant dose to be given followed by
gradual reduction in dosage
ī‚Ą Injecting anti-inflammatory such as
hydrocortisone into the
joint has been
advocated
5) Anxiolytic drugs-
ī‚Ą When high level of emotional stress are
thought to contribute to TMD, antianxiety
(anxiolytic agents ) should be used
ī‚Ą Bezodiazepine (Diazepam)- prescribed on
daily basis
ī‚Ą Single dose of diazepam (2.5- 5 mg) at bed
time to relax the muscles and perhaps
lessen nocturnal parafunctional habit.
6) Muscle relaxants:
ī‚Ą They have a central effect that sedates the
patient.
ī‚Ą Perhaps, this sedation is the main
explanation for the positive response of the
patient
ī‚Ą Skeletal muscle relaxants in combination
with analgesics
īļ Chlorzoxazone with acteaminophen
(Parafon Forte)
īļOrphenadrine citrate with aspirin and
caffeine (Norgesic Forte)
īļCyclobezaprine (Flexeril)- single dose of 10
mg before sleep
7) Antidepressants:
ī‚Ą Low dose of amitriptyline (10 mg) just
before sleep can have analgesic on chronic
pain but has little effect on acute pain
8) Local anesthetics:
ī‚Ą Can be used to differentiate true source of
pain from a site of pain
ī‚Ą Two most common local anesthetics drugs
used for short duration pain reduction in TMD
īļ 2% Lidocaine (Xylocaine)
īļ 3% Mepivacaine ( Carbocaine)
ī‚§ Solution without vasoconstrictor should be
used for muscle injections
ī‚§ Intracapsular injection- sodium Hyaluronate
PHYSICAL THERAPY
ī‚Ą It is important part of successful
management of many TMD
ī‚Ą Includes :
ī‚Ą a) Modalities
ī‚Ą b) Manual technique
PHYSICAL THERAPY MOdALITIES
ī‚Ą Thermotherapy
ī‚Ą Coolant therapy
ī‚Ą Ultrasound
ī‚Ą Phonophoresis
ī‚Ą Iontophoresis
ī‚Ą EGS therapy
ī‚Ą TENS
ī‚Ą Acupuncture
ī‚Ą Laser
1) Thermotherapy:
ī‚Ą Uses heat as a prime mechanism as it
increases circulation to the applied area.
ī‚Ą Apply hot moist towel over symptomatic
area.
ī‚Ą Not to exceed 30 min (10-15 min)
2) Coolant therapy:
ī‚Ą Cold encourages relaxation of muscles that
are in spasm and thus relieve the pain
associated.
ī‚Ą Apply directly to affected area
ī‚Ą After a period of warming, second
application may be desirable
Initially, pt experience uncomfortable feeling
Quickly turn into burning sensation
Continued icing
Mild aching
Numbness begins
Remove the ice
ī‚Ą Common coolant therapy uses vapor spray:
īļ Ethylchloride
īļFluoromethane
ī‚Ą Apply the spray to desired area from
a distance of 1 to 2 feet for approx 5 sec
ī‚Ą Action – associated with stimulation of
cutaneous nerve fibers
ī‚Ą In myofascial pain, a technique is used “
Spray and stretch” tech.
ī‚Ą Spraying over a muscle with a trigger point
and then immediately passively stretching
the muscle.
3) Ultrasound therapy :
ī‚Ą It is a method of producing an increase in
temperature at the interface of the tissues
and therefore affects the deeper tissues
than does surface heat.
ī‚Ą Improves the flexibility and extensibility of
connective tissues.
4) Phonophoresis:
ī‚Ą Ultrasound has been used to administer
drugs through the skin by a process known
as phonophoresis.
ī‚Ą Example- 10% hydrocortisone cream is
applied to an inflammed joint and
ultrasound transducer is then directed at
the joint.
5) IONTOPHORESIS:
ī‚Ą It is a technique by which certain
medications can be introduced into the
tissues without affecting any other organs
ī‚Ą Medication is placed in a pad and the pad
is placed on the desired area. Then, a low
electrical current is passed though the pad
6) ELECTROGALVANIC STIMULATION THERPAY:
ī‚Ą Works on principle that electrical
stimulation of a muscle cause it to contract
ī‚Ą Uses high voltage, low amperage,
monophasic current of varied frequency
ī‚Ą Repeated contraction and relaxation
ī‚Ą Help to breakup the myospasm and
increase blood flow to the muscles
7) TRANSCUTANEOUS ELECTRICAL NERVE
STIMULATION:
ī‚Ą Continuous stimulation of cutaneous nerve
fibers at a subpainful level.
ī‚Ą Uses a low voltage, low amperage, biphasic
current of varied frequency.
8) ACUPUNCTURE:
ī‚Ą Uses bodys own antinociceptive system to
reduce the level of pain felt
Stimulation of certain areas (acupuncture points)
Release of endorphins
Causes flooding of the afferent
interneurons and subthreshold stimuli
Reduce painful sensation
Block transmission of noxious impulses
9) COLDLASER:
ī‚Ą Cold or soft laser has been investigated for
wound healing and pain relief
Cold laser accelerates collagen
synthesis
Increasing vascularity of
healing tissues
Decreases number of
microorganisms
Decrease pain
Manual techniques
ī‚Ą Manual techniques are the “hands on”
therapies provided by
the physiotherapaist
for the reduction of
pain and dysfunction.
3 main categories
Soft tissue
mobilization
Joint mobilization
Muscle
conditioning
1) SOFTTISSUE MOBILIZATION:
ī‚Ą Includes superficial and deep massage
ī‚Ą Helps in mobilizing the tissues, increasing
blood flow to the area and eliminating the
trigger points
Deep heat tends to relax
muscles
Decrease the pain
Enhance effectiveness of deep
massage
2) Joint mobilization:
ī‚Ą Mobilization of TMJ is useful in
decreasing interarticular pressure and
increasing range of joint movement
ī‚Ą Useful in managing acute disc dislocation
without reduction
ī‚Ą If distraction ellicits pain, then therapist
should think of inflammatory disorder and
discontinue distraction procedure.
ī‚Ą When cervical traction is used, care must
be taken not to place usual forces on
condyle
ī‚Ą Keep the teeth in together while undergoing
traction
3) Muscleconditioning:
ī‚Ą Patients who experience TMD symptoms
often decrease use of their jaw because of
pain muscle becomes atrophy
ī‚Ą Instruct self administered exercises to
restore the normal function and range of
movement
Passive muscle
stretching
Assisted muscle
stretching
Resistance
exercises
Postural training
MUSCLE
CONDITIONING
a) Passive muscle stretching-
Shortened muscle length
Decreases blood flow
Accumulation of algogenic
substances
Muscle pain
ī‚Ą Patient is instructed to open the mouth
slowly and deliberately untill pain is felt
ī‚Ą Look in mirror and then perform, so that
patient makes a straight pathway without
deflection or deviation
ī‚Ą Use a vapocoolant spray which will reduce
pain and then perform.
b) Assisted muscle stretching:
ī‚Ą Stretching should be performed with
gentle intermittent force that is gradually
increased.
ī‚Ą Simons and Travelle described spray and
stretch technique.
c) Resistance exercises:
ī‚Ą Use concept of reflex relaxation
ī‚Ą Instruct the patient to place the fist under
the chin and open the mouth against
resistance
ī‚Ą Repeat 10 times each session, 6 sessions
per day
d) Postural training-
ī‚Ą Posture of head, neck and shoulders can
contribute to TMD symptoms.
ī‚Ą Postural self regulation training was
developed by Drs Peter Bertrand and
Charles Carlson in 1993.
A forward head
position has drawn
the most attention
Forward and rotated
head position
Produces elongation of
suprahyoid and
infrahyoid muscles
Closes posterior space
between atlas and axis
Specific
disorders and
their
treatment
1) Disc displacement:
ī‚Ą Splint therapy
ī‚Ą Physical therapy
ī‚Ą Anti-inflammatory drugs
ī‚Ą Arthrocentesis
ī‚Ą Arthroscopic lysis and lavage
ī‚Ą Arthroplasty
ī‚Ą Vertical ramus osteotomy
2) CONNECTIVE TISSUE DISORDERS
(Osteoarthritis, RA, spondyloarthropathies)
ī‚Ą Treatment of systemic disease
ī‚Ą Jaw self care
ī‚Ą Physiotherapy
ī‚Ą Oral appliance therapy
ī‚Ą Topical NSAID
ī‚Ą Intra-articular corticosteroid injection
ī‚Ą Arthrocentesis (conservative procedure)
ī‚Ą Arthroscopy, arthroplasty, arthrotomy
(surgical procedures)
3) DEVELOPMENTAL DEFECTS and fractures:
ī‚Ą Surgical intervention
4) DISLOCATION:
ī‚Ą Surgical intervention
ī‚Ą IV diazepam for severe pain
ī‚Ą Reduction and stabilization
ī‚Ą Caution- not to open the mouth wide open
ī‚Ą Injection of sclerosing agents
5) ANKYLOSIS:
ī‚Ą Severe surgical procedures
ī‚Ą Gap arthroplasty
6) Bruxism:
ī‚Ą Oral appliance therapy
ī‚Ą Biofeedback
ī‚Ą Injecting botulinum toxin
7) ORALDYSKINESIA:
ī‚Ą Clonazepam
ī‚Ą Baclofen
ī‚Ą Tetrabenazine
ī‚Ą Injection of botulinum toxin
ī‚Ą It is important for dentists to rule out
disorders that mimic TMD, to identify non-
TMD disorders that may negatively impact
the patient’s TMD symptoms, and to offer
the patient therapies that will provide the
most cost-effective long-term symptom
relief.
ī‚Ą Temporomandibular disorders (TMD) are
the most prevalent orofacial pain conditions
for which patients seek treatment.
ī‚Ą A multidisciplinary pain management
approach should be considered for the
optimal treatment of orofacial pain
disorders including both non-
pharmacological and pharmacological
modalities.
1) Textbook of mangement of temporomandibular
disorders and occlusion - Okeson Jeffrey P- 5 th
edition
2) Manual of Temporomandibular joint- Shivalal M
Rawlani
3) Burkets Oral Medicine-11 Edition-by Greenberg
And Glick And Ship
4) Mina et al. Effectiveness of Dexamethasone
Iontophoresis for Temporomandibular Joint
Involvement in Juvenile Idiopathic Arthritis.
Arthritis care an research; Nov 2011
5) Orofacial pain management: current perspectives
6) Sala Horowitz. Biofeedback Applications- A survey of
clinical research; Alternative and complimentary
therapies – Dec 2006
7) Wright E et al. Management and Treatment of
Temporomandibular Disorders: A clinical Perspective.
The journal of manual and manipulative therapy
8) Temporomandibular joint exercises. Therapies
Directorate
9) Chin S et al. Application of acupuncture in
temporomandibular disorder. Journal of prosthodontics
and Implantology 2013
10) Ardehali et al. Temporomandibular joint dislocation
reduction technique. Annals of plastic surgery 2009
ī‚Ą Is osteoarthritis an inflammatory or non-
inflammatory disorder. Justify your answer
ī‚Ą Role of Iontophoresis in treatment of TMD
ī‚Ą Classification of TMD
ī‚Ą Signs and symptoms of TMD
ī‚Ą Function of occlusal splints
ī‚Ą What is biofeedback
ī‚Ą Surgical treatment modalities for TMD
Management of temporomandibular disorders
Management of temporomandibular disorders

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Management of temporomandibular disorders

  • 2. ī‚Ą Temporomandibular Disorders (TMDs), also referred to as craniomandibular disorders, consist of a group of pathologies affecting the masticatory muscles, the tempromandibular joint (TMJ) and related structures.
  • 3. ī‚Ą TMD’s RDC groups are: I Group: Muscle disorders: Ia) Myofascial pain Ib) Myofascial pain with limited opening II Group: Disc Displacement (DD): IIa) DD with reduction IIb) DD without reduction with limited opening IIc) DD without reduction without limited opening
  • 4. III Group: Other common joint disorders: IIIa) Arthralgia IIIb) Osteoarthritis IIIc) Ostoarthrosis
  • 5.
  • 6.
  • 7. “ TMD is a complex and multifactorial condition; so also our patients”- JPO
  • 9. īļThose which provide basically conservative reversible therapy īļNon conservative, irreversible therapy All treatment methods used for TMD can be categorized into: 1) Definitive treatment 2) Supportive therapy
  • 11. ī‚Ą Intended to directly eliminate or alter the causes of a disorder Normal function An event Physiologic tolerance Temporomandibular disorder symptoms
  • 13. DEFINITIVE TREATMENT CONSIDERATIONSFOR OCCLUSAL FACTORS ī‚Ą Reversible Occlusal therapy: ī‚Ą Best accompalished with occlusal appliance
  • 14. ī‚Ą Irreversible occlusal therapy: Selective grinding of teeth Restorative procedures Orthodontic treatment Surgical procedures
  • 15. DEFINITIVE TREATMENT CONSIDERATIONSFOR EMOTIONAL STRESS ī‚Ą Emotional stress is one of several pyschologic factors that need to be considered ī‚Ą Increase emotional stress Increase resting activity alters muscle function or bruxism
  • 16. Common personality traits: īļPerfectionistic īļCompulsive īļIntrovert īļNeurotic īļDis-satisfied and self destructive Common emotional stress: īļHigh level of anxiety īļApprehension īļFrustation īļAnger īļFear īļDepression īļHostility
  • 17. TMD patients with chronic fear Increase level of anxiety, frustation and anger Increased level of emotional stress Contribute to TMD
  • 18. ī‚Ą Types of emotional stress therapy: 1) Patient awareness- ī‚Ą Educate the patient and make the patient aware that stress is a common everyday experience 2) Restrictive use- ī‚Ą Painful movements should be avoided as it may further lead to cyclic muscle pain
  • 19. ī‚Ą Alter the diet 3) Voluntary avoidance- ī‚Ą Patient should be instructed that teeth should be quickly disengaged any time they come in contact. ī‚Ą Other oral habits like biting on objects or cradling telephone between mandible and shoulder aggravate TMD symptom
  • 20. â€ĸ Buffering a little air between lips and teeth â€ĸ Allows jaw to assume a relaxed position â€ĸ Lips brought together and teeth left slightly apart â€ĸ This position decreases muscle activity â€ĸ Minimizes interarticular pressure â€ĸ Promotes joint repair
  • 21. 4) Relaxation Therapy - īļ Substitutive Relaxation therapy: ī‚§ Substitution for stress (any activity that is enjoyable- sports, hobbies or recreational activity) īļ Active relaxation therapy: ī‚§ Technique used in dentistry are modification of Jacobsons method, developed in 1968.
  • 22. Pt is trained to relax symptomatic muscles voluntarily Blood flow to these tissues is increased Metabolic waste substances that stimulate pain receptors are eliminated Diminishes pain
  • 23. ī‚Ą Patient tenses the muscles and then relaxes them untill relaxed state can be felt and maintained ī‚Ą Another form uses a reverse approach ( muscles are passsively stretched and relaxed) ī‚Ą This technique has one major advantage over Jacobson technique
  • 24. ī‚Ą Self hypnosis, meditation and yoga also promote relaxation 5) Biofeedback – ī‚Ą It is a technique that assist patient in regulating bodily functions that are generally controlled unconsciously.
  • 25. ī‚Ą It is accompalished by electromyographically monitoring state of muscles through surface electrodes placed over muscles to be monitored. ī‚Ą Biofeedback units give auditory feedback ī‚Ą When patient clenches, high readings will appear or elevated tone is heard
  • 26.
  • 27.
  • 28. ī‚Ą Negative Biofeedback: ī‚Ą Devices are small and worn throughout day and night ī‚Ą If clenching or bruxism occurs, biofeedback mechanism is activated and loud sound is heard.
  • 29. ī‚Ą A review of 30 studies of physical therapy modalities for treating TMD concluded that biofeedback may be more effective than placebo or occlusial splints ī‚Ą (Medlicott MS, Harris SR. A systematic review of the effectiveness of exercise, manual therapy, electrotherapy, relaxation training, and biofeedback in the management of temporomandibular disorder. Phys Ther 2006;86:955–973.)
  • 30. DEFINITIVE TREATMENT CONSIDERATIONSFOR trauma ī‚Ą Trauma is one of the causative factor that can lead to TMD īļMacrotrauma: ī‚Ą Only supportive therapy is helpful (trauma has already occurred, so definitive therapy is of no use) ī‚Ą Preventive measures are helpful
  • 31. īļMicrotrauma ī‚Ą Result from repeated loading of joint structures, such as bruxism or clencing ī‚Ą Occlusal appliances help in achieving a favorable condyle disc relationship that will unload retrodiscal tissues and load the disc.
  • 32. DEFINITIVE TREATMENT CONSIDERATIONSFOR deep pain input ī‚Ą Some TMD may be secondary to another source of deep pain eg cervical injury ī‚Ą Appropriate history and examination
  • 33. DEFINITIVE TREATMENT CONSIDERATIONSFOR parafunctional activity ī‚Ą For many years, dentists were convinsed that bruxism and clenching were major causes of TMD. ī‚Ą This is true, but not always the case
  • 34. ī‚Ą Keep the teeth apart ī‚Ą Nocturnal bruxism- (more due to emotional stress level and sleep pattern) ī‚Ą Effectively reduced by occlusal applianec therapy ī‚Ą Morning pain associated with nocturnal bruxism
  • 36. ī‚Ą Supportive therapy mainly includes: īļ Pharmacologic therapy īļ Physical therapy
  • 37. PHARMACOLOGICTHERAPY ī‚§ If medications are prescribed on “take as needed” basis, then drugs are abused by patients. ī‚§ Continued use lead to frequent pain cycles and less drug effectiveness ī‚§ Analgesics and Tranquilizers
  • 38. ī‚Ą Acute TMD pain īļ Analgesics īļ Corticosteroids īļ Anxiolytics ī‚§ Chronic orofacial pain īļ Tricyclic antidepressants ī‚Ą Both acute and chronic condition īļ NSAID īļ Muscle relaxant īļ Local Anesthetics DRUGS USED
  • 39. 1) Analgesics: ī‚Ą Important part of supportive therapy ī‚Ą Opiates or non opiates ī‚Ą First medication of choice for moderate pain relief is Calpol (acetaminophen) ī‚Ą Stronger analgesics (codeine combined with either salicylate or acetaminophen can be helpful)
  • 40. 2) Non Steroidal antiinflammatory drugs: ī‚Ą Chief application is in the treatment of muscoloskeletal pain ī‚Ą In presence of tissue injury, that certain chemical mediators are released into the injured site. ī‚Ą NSAID inhibit action of cycloxygenase, an enzyme used to synthesize prostaglandin.
  • 42. 3) Anti-inflammatory agents: ī‚Ą These drugs do not immediately achieve good blood levels, therefore need to be taken on a regular schedule for a minimum of 3 weeks
  • 43. 4) Corticosteroids ī‚Ą Significant dose to be given followed by gradual reduction in dosage ī‚Ą Injecting anti-inflammatory such as hydrocortisone into the joint has been advocated
  • 44.
  • 45. 5) Anxiolytic drugs- ī‚Ą When high level of emotional stress are thought to contribute to TMD, antianxiety (anxiolytic agents ) should be used ī‚Ą Bezodiazepine (Diazepam)- prescribed on daily basis
  • 46. ī‚Ą Single dose of diazepam (2.5- 5 mg) at bed time to relax the muscles and perhaps lessen nocturnal parafunctional habit. 6) Muscle relaxants: ī‚Ą They have a central effect that sedates the patient. ī‚Ą Perhaps, this sedation is the main explanation for the positive response of the patient
  • 47. ī‚Ą Skeletal muscle relaxants in combination with analgesics īļ Chlorzoxazone with acteaminophen (Parafon Forte) īļOrphenadrine citrate with aspirin and caffeine (Norgesic Forte) īļCyclobezaprine (Flexeril)- single dose of 10 mg before sleep
  • 48. 7) Antidepressants: ī‚Ą Low dose of amitriptyline (10 mg) just before sleep can have analgesic on chronic pain but has little effect on acute pain 8) Local anesthetics: ī‚Ą Can be used to differentiate true source of pain from a site of pain
  • 49. ī‚Ą Two most common local anesthetics drugs used for short duration pain reduction in TMD īļ 2% Lidocaine (Xylocaine) īļ 3% Mepivacaine ( Carbocaine) ī‚§ Solution without vasoconstrictor should be used for muscle injections ī‚§ Intracapsular injection- sodium Hyaluronate
  • 50. PHYSICAL THERAPY ī‚Ą It is important part of successful management of many TMD ī‚Ą Includes : ī‚Ą a) Modalities ī‚Ą b) Manual technique
  • 51. PHYSICAL THERAPY MOdALITIES ī‚Ą Thermotherapy ī‚Ą Coolant therapy ī‚Ą Ultrasound ī‚Ą Phonophoresis ī‚Ą Iontophoresis ī‚Ą EGS therapy ī‚Ą TENS ī‚Ą Acupuncture ī‚Ą Laser
  • 52. 1) Thermotherapy: ī‚Ą Uses heat as a prime mechanism as it increases circulation to the applied area. ī‚Ą Apply hot moist towel over symptomatic area. ī‚Ą Not to exceed 30 min (10-15 min)
  • 53. 2) Coolant therapy: ī‚Ą Cold encourages relaxation of muscles that are in spasm and thus relieve the pain associated. ī‚Ą Apply directly to affected area ī‚Ą After a period of warming, second application may be desirable
  • 54. Initially, pt experience uncomfortable feeling Quickly turn into burning sensation Continued icing Mild aching Numbness begins Remove the ice
  • 55. ī‚Ą Common coolant therapy uses vapor spray: īļ Ethylchloride īļFluoromethane ī‚Ą Apply the spray to desired area from a distance of 1 to 2 feet for approx 5 sec ī‚Ą Action – associated with stimulation of cutaneous nerve fibers
  • 56. ī‚Ą In myofascial pain, a technique is used “ Spray and stretch” tech. ī‚Ą Spraying over a muscle with a trigger point and then immediately passively stretching the muscle.
  • 57. 3) Ultrasound therapy : ī‚Ą It is a method of producing an increase in temperature at the interface of the tissues and therefore affects the deeper tissues than does surface heat. ī‚Ą Improves the flexibility and extensibility of connective tissues.
  • 58.
  • 59. 4) Phonophoresis: ī‚Ą Ultrasound has been used to administer drugs through the skin by a process known as phonophoresis. ī‚Ą Example- 10% hydrocortisone cream is applied to an inflammed joint and ultrasound transducer is then directed at the joint.
  • 60. 5) IONTOPHORESIS: ī‚Ą It is a technique by which certain medications can be introduced into the tissues without affecting any other organs ī‚Ą Medication is placed in a pad and the pad is placed on the desired area. Then, a low electrical current is passed though the pad
  • 61.
  • 62. 6) ELECTROGALVANIC STIMULATION THERPAY: ī‚Ą Works on principle that electrical stimulation of a muscle cause it to contract ī‚Ą Uses high voltage, low amperage, monophasic current of varied frequency
  • 63. ī‚Ą Repeated contraction and relaxation ī‚Ą Help to breakup the myospasm and increase blood flow to the muscles
  • 64. 7) TRANSCUTANEOUS ELECTRICAL NERVE STIMULATION: ī‚Ą Continuous stimulation of cutaneous nerve fibers at a subpainful level. ī‚Ą Uses a low voltage, low amperage, biphasic current of varied frequency.
  • 65. 8) ACUPUNCTURE: ī‚Ą Uses bodys own antinociceptive system to reduce the level of pain felt
  • 66.
  • 67.
  • 68. Stimulation of certain areas (acupuncture points) Release of endorphins Causes flooding of the afferent interneurons and subthreshold stimuli Reduce painful sensation Block transmission of noxious impulses
  • 69. 9) COLDLASER: ī‚Ą Cold or soft laser has been investigated for wound healing and pain relief Cold laser accelerates collagen synthesis Increasing vascularity of healing tissues Decreases number of microorganisms Decrease pain
  • 70. Manual techniques ī‚Ą Manual techniques are the “hands on” therapies provided by the physiotherapaist for the reduction of pain and dysfunction. 3 main categories Soft tissue mobilization Joint mobilization Muscle conditioning
  • 71. 1) SOFTTISSUE MOBILIZATION: ī‚Ą Includes superficial and deep massage ī‚Ą Helps in mobilizing the tissues, increasing blood flow to the area and eliminating the trigger points
  • 72. Deep heat tends to relax muscles Decrease the pain Enhance effectiveness of deep massage
  • 73. 2) Joint mobilization: ī‚Ą Mobilization of TMJ is useful in decreasing interarticular pressure and increasing range of joint movement ī‚Ą Useful in managing acute disc dislocation without reduction
  • 74.
  • 75. ī‚Ą If distraction ellicits pain, then therapist should think of inflammatory disorder and discontinue distraction procedure. ī‚Ą When cervical traction is used, care must be taken not to place usual forces on condyle ī‚Ą Keep the teeth in together while undergoing traction
  • 76. 3) Muscleconditioning: ī‚Ą Patients who experience TMD symptoms often decrease use of their jaw because of pain muscle becomes atrophy ī‚Ą Instruct self administered exercises to restore the normal function and range of movement
  • 78. a) Passive muscle stretching- Shortened muscle length Decreases blood flow Accumulation of algogenic substances Muscle pain
  • 79. ī‚Ą Patient is instructed to open the mouth slowly and deliberately untill pain is felt ī‚Ą Look in mirror and then perform, so that patient makes a straight pathway without deflection or deviation ī‚Ą Use a vapocoolant spray which will reduce pain and then perform.
  • 80. b) Assisted muscle stretching: ī‚Ą Stretching should be performed with gentle intermittent force that is gradually increased. ī‚Ą Simons and Travelle described spray and stretch technique.
  • 81.
  • 82. c) Resistance exercises: ī‚Ą Use concept of reflex relaxation ī‚Ą Instruct the patient to place the fist under the chin and open the mouth against resistance ī‚Ą Repeat 10 times each session, 6 sessions per day
  • 83.
  • 84.
  • 85.
  • 86.
  • 87.
  • 88.
  • 89.
  • 90.
  • 91.
  • 92. d) Postural training- ī‚Ą Posture of head, neck and shoulders can contribute to TMD symptoms. ī‚Ą Postural self regulation training was developed by Drs Peter Bertrand and Charles Carlson in 1993. A forward head position has drawn the most attention
  • 93. Forward and rotated head position Produces elongation of suprahyoid and infrahyoid muscles Closes posterior space between atlas and axis
  • 95. 1) Disc displacement: ī‚Ą Splint therapy ī‚Ą Physical therapy ī‚Ą Anti-inflammatory drugs ī‚Ą Arthrocentesis ī‚Ą Arthroscopic lysis and lavage ī‚Ą Arthroplasty ī‚Ą Vertical ramus osteotomy
  • 96. 2) CONNECTIVE TISSUE DISORDERS (Osteoarthritis, RA, spondyloarthropathies) ī‚Ą Treatment of systemic disease ī‚Ą Jaw self care ī‚Ą Physiotherapy ī‚Ą Oral appliance therapy ī‚Ą Topical NSAID ī‚Ą Intra-articular corticosteroid injection
  • 97. ī‚Ą Arthrocentesis (conservative procedure) ī‚Ą Arthroscopy, arthroplasty, arthrotomy (surgical procedures) 3) DEVELOPMENTAL DEFECTS and fractures: ī‚Ą Surgical intervention
  • 98. 4) DISLOCATION: ī‚Ą Surgical intervention ī‚Ą IV diazepam for severe pain ī‚Ą Reduction and stabilization ī‚Ą Caution- not to open the mouth wide open ī‚Ą Injection of sclerosing agents
  • 99. 5) ANKYLOSIS: ī‚Ą Severe surgical procedures ī‚Ą Gap arthroplasty 6) Bruxism: ī‚Ą Oral appliance therapy ī‚Ą Biofeedback ī‚Ą Injecting botulinum toxin
  • 100. 7) ORALDYSKINESIA: ī‚Ą Clonazepam ī‚Ą Baclofen ī‚Ą Tetrabenazine ī‚Ą Injection of botulinum toxin
  • 101. ī‚Ą It is important for dentists to rule out disorders that mimic TMD, to identify non- TMD disorders that may negatively impact the patient’s TMD symptoms, and to offer the patient therapies that will provide the most cost-effective long-term symptom relief.
  • 102. ī‚Ą Temporomandibular disorders (TMD) are the most prevalent orofacial pain conditions for which patients seek treatment. ī‚Ą A multidisciplinary pain management approach should be considered for the optimal treatment of orofacial pain disorders including both non- pharmacological and pharmacological modalities.
  • 103. 1) Textbook of mangement of temporomandibular disorders and occlusion - Okeson Jeffrey P- 5 th edition 2) Manual of Temporomandibular joint- Shivalal M Rawlani 3) Burkets Oral Medicine-11 Edition-by Greenberg And Glick And Ship 4) Mina et al. Effectiveness of Dexamethasone Iontophoresis for Temporomandibular Joint Involvement in Juvenile Idiopathic Arthritis. Arthritis care an research; Nov 2011 5) Orofacial pain management: current perspectives
  • 104. 6) Sala Horowitz. Biofeedback Applications- A survey of clinical research; Alternative and complimentary therapies – Dec 2006 7) Wright E et al. Management and Treatment of Temporomandibular Disorders: A clinical Perspective. The journal of manual and manipulative therapy 8) Temporomandibular joint exercises. Therapies Directorate 9) Chin S et al. Application of acupuncture in temporomandibular disorder. Journal of prosthodontics and Implantology 2013 10) Ardehali et al. Temporomandibular joint dislocation reduction technique. Annals of plastic surgery 2009
  • 105. ī‚Ą Is osteoarthritis an inflammatory or non- inflammatory disorder. Justify your answer ī‚Ą Role of Iontophoresis in treatment of TMD ī‚Ą Classification of TMD
  • 106. ī‚Ą Signs and symptoms of TMD ī‚Ą Function of occlusal splints ī‚Ą What is biofeedback ī‚Ą Surgical treatment modalities for TMD

Editor's Notes

  1. Pt symptoms fall into more than one category. First give example of traumatic joint. Masticatory- increase tonicity of elevator muscles- increase interarticluar pressure - hyperactivity of superior lateral pterygoid.- accentuate disc derangement. Disc derangement- muscle co-cntraction-result in an attempt tp prevent painful movements- if muscle co-contraction becomes protracted- local muscle soreness resiults
  2. The treatment goals for TMD are decreasing pain, restoring normal ROM, and restoring normal masticatory and jaw function. Relieving the muscle spasm and pain is the main aim of any treatment modality in order to improve the quality of life. Tell about logical approach.
  3. Events -1) Local trauma to tissues 2) Increased emotional stress 3) Acute changes in occlusion 4) orthopaedic instablility Definitive therapy are those that alter factors associated with event that has interrupted norml function of masticatory system.
  4. Altering mandibular position 2) occlusal contact points or both Stabilization appliances are used for the purpose of equally distributing jaw parafunctional forces, reducing the forces placed on the masticatory muscles, and protecting the occlusal surfaces of the teeth from chronic nocturnal bruxing. Tell about the device, types, orhtopaedic stability.
  5. Apart from this, it may also activate the sympathetic nervous system
  6. Painful movements are avoided bcoz they are associated with further damage to structures. “if it hurts, den don’t do it”
  7. Discontinue the habits or other therapy is use of occlusal appliance
  8. Pts with masticatory muscle disorder often report pain when asked to contract their muscles . This increase in pain makes the relaxation difficult. In contrast, gentle stretching of muscle seems to assist in relaxation
  9. Facia muscles- masster isc hosen. For full body relaxation- frontalis musles.
  10. When this appliance is used, mandible is stabilized with maxilla which minimizes injury to masticatory structure
  11. When deep source is resolced , TMD alos resolve
  12. Supportive therapy is directed towards altering the patients symptoms and often has no effect on the cause of the disorder. The clinician should always remember that supportive therapy is only symptomatic ; it is not a replacement therapy for definitive treatment.
  13. Prsecribe at regular intervals for a specific period.
  14. Brand names calpol,crocin
  15. It is indicated for acute and painful arthritic TMJ that has not responded to other modalities of treatment and when the joint is still acutely inflamed, such as in the case of polyarthritic disorders and in acute discdisplacements without reduction. The use of triamcinolone or dexamethasone, in addition to 2% lidocaine without epinephrine, is generally used for TMJ injections
  16. Do not eliminate stress but merely alters patient perception or reaction to stress. Another drug is clonazepam. Diazepam (valium, diastat)
  17. For muscle relaxants to reach therspeutic effect on muslces of mastication, dose must often be raised to a level that dos not allow the pt to carry out normal activities.
  18. Improves the quality of sleep.
  19. Injection of LA into myofascial trigger points Can result in significant pain reduction long after the anestic solu has beeen metabolized. Another use of LA in the management of some chronic TMD is related to pain management. Therapeutic effect is achieved by breaking the pain cycle.
  20. Most theories consider that decrease blood flow to the tissue is responsible of mylagia, thermotherpay counteracts this by creating vasodilation nd in turn increase blood supply to the affected area.
  21. Ice not to be left more than 5-7 min. It is thought that during warming, increase in blood flow assist in tissue repair
  22. Ethrlychloride- flammable and if inhaled caused cardiac depressant
  23. Vapoccolant spary does not penetrate tissue like ice, pain is associated with stimulation of cutaneous nerve fibers that in turn shut down the smaller pain fibers.
  24. Therapeutic ultrasound consist of inaudible high frequency mechanical vibrations created when a generator produces electrical energy that is converted into acoustic energy through mechanical deformation of piezoelectrical crystal located within transducer. Tell about 1 and 3 MHz and also precautions to be taken while using transducer
  25. Increase in tissue extensibility, increase in blood flow, mild inflammatory response, reduction in joint stiffness, reduction in muscle spasm
  26. Pts having juvenile rheumatoid arthritis. DIP proved to be an effective and safe initial treatment Of TMJ involvement (journal of arthritis care and research 2011)
  27. Rhythmic electrical pulse – repeated inviluntary contraction nd relaxation.
  28. Based on theories of pain- gate control theory and endogenous opiod theory.
  29. Analgesic effect of acupuncture comes from its ability to elevate the pain threshold, release neurotransmitters in local regions, and modulate pathways in the central nervous system.
  30. 10- 15 min of moist heat should be applied before beginning deep massage
  31. Keep teeth in contact- this helps to stabilize and control loading to joint structures
  32. 1) Osteoarthritis is a chronic non-inflammatory degenerative condition of joints also known as degenerative joint disease, degenerative arthritis or osteoarthrosis, reflects both the non-inflammatory and inflammatory changes that may take place in the temporomandibular joint (TMJ). The word ‘osteoarthritis’ originated from the Greek word “osteo” meaning “bone”, “arthro” meaning “joint” and “itis” meaning “inflammation”. Although the “itis” in the term osteoarthritis is a misnomer, as inflammation is not a conspicuous feature as seen in rheumatoid or autoimmune types of arthritis. Some clinicians term this condition as osteoarthrosis to denote the lack of inflammatory response. It is defined pathologically and radiologically by reduced joint spaces secondary to loss of cartilage due to sclerosis of subchondral bone and osteophyte formation. It is one of the most common forms of arthritis affecting the TMJ. 2) It is a technique by which certain medications can be introduced into the tissues without affecting any other organs . Medication is placed in a pad and the pad is placed on the desired area. Then, a low electrical current is passed though the pad 3) research diagnsotic criteria
  33. 4) Costen was the first to recognize signs and symptoms of temporomandibular disorders (TMD) in 1934.22 Temoromandibular disorder is characterized by one or more of the following signs or symptoms: pain, joint sounds, limitation in joint movement, muscle tenderness and joint tenderness.23 Apart from these, it is also associated with other symptoms such as headache, ear related symptoms (tinnitus in which there is ringing of ears), facial pain, neck and shoulder pain and cervical spine disorders 5) Arthrocentesis, Arthroscopy and Arthrotomy 6) 7) It is a technique that assist patient in regulating bodily functions that are generally controlled unconsciously. It is accompalished by electromyographically monitoring state of muscles through surface electrodes placed over muscles to be monitored. Biofeedback units give auditory feedback. When patient clenches, high readings will appear or elevated tone is heard