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d rot of sugarcane
Presented by Nisha nag
MSc 4th sem
Department of microbiology
Content:-
1.Introduction
2.History
3.Casual organism
4.Pathogen character
5.Etiology
6.transmission
7.Disease cycle
8.Symptoms
9.Survival and spread
10.Favorable conditions
11.Disease management
12.Integrated disease management
13.Reference
Introduction
Red rot is one of the major
constraints in the profitable
cultivation of sugarcane in
many states of India.
• Except Maharashtra the
disease has been recorded in
all the state.
• This disease drastically
retards the yield and
considerably deteriorates the
juice quantity and quality
thus hitting both the cane
growers and millers
History:-
• First reported in 1893 from Java (Indonesia).
• In India first reported in 1901 in Andhra Pradesh.
• Disease epidemic occured in 1939-1940 in UP & BIHAR.
• Disease is soil and seed borne.
• This accepted name, ‘Red rot’ was given to this cane
disease in 1906 by sir
E.J Butler.
Casual organism:-
Kingdom :- fungi
Phylum :- Ascomycota
Class :- sordariomcetes
Order :- phyllachorales
Family :- Glomerellaceae
Genus :- Colletotrichum
Species :- falcatum
Pathogen Character:-
1.Anamorph :- colletotrichum fakatum
2.Teleomorph :- Glomerella tacumanensis.
Etiology :-
Caused by collectotrichum falcatum, a facultative
saprophyte.
• Sexual stage known as Glomerella tucumanensis.
• Mycellium:- both intracellular and intercellular.
• Asexual fruiting bodies known as Acervuli.
• Conidiophores:- linear or club shaped,
hyallne,septate.
• Conidia:- elongated,single celled, thin walled,
uninucleate, colourless, sickle shaped (falcate)
with granuar protoplasm with a large oil globule.
Transmission:-
• Primary transmission through soil and disease
sets while the secondary transmission through
air, rain splash and soil.
Disease cycle:-
• The fungus is sett born.
• The fungus also persist in the soil on the
disease clumps and stubble as acervulli.
• The primary infection is mainly from infected
setts.
• Secondary spread in the field maybe through
irrigation water and cultivation tools.
• The rain splash air currents and dew drops also
help in the spread of conidia from the disease
to healthy plant in the field.
• The fungus also survives on collateral host
like Sorghum vulgare, S. Haleperse and
saccharum spontaneum.
Symptoms:-
Two types of symptoms:-
1.Stalk symptoms
2.leaf symptoms
Stalk symptoms:-
.The first symptom seen after the rainy
season when the normal plant growth stops
and sucrose formation was started.
• Loose of colour and dropping of third or
fourth leaves from the top are the early
symptoms the intert weakness withers.
• In letter States the canes become
shriveled the red shrinks and become
longitudinally wrinkled. Such canes are
lighter invade and easily broken.
• If the disease canes are split open
longitudinally, especially when withering
of leaves started the pic took red
Continue-
• In very advance stage, red colour maybe
replaced by dirty brown cavities filled with
Greyish mycelium and the juice gives a bad
smell due to conversion of sucrose into glucose
and alcohol as a result of enzymetic action of
pathogen.
Leaf symptoms:-
• Tini reddish lesion of the
upper surface of lamina these
lesions are 2 to 3 mm in
length and about 0.5 mm in
width.
• Menu trade rot on upper
surface of midrib of leaf
later on become straw coloured
in centre with development of
darker acervuli.
Survival and spread:-
• They reply heavily on water, particularly rainfall, for
dissemination.
• The fungus is sett borne and also perists in the soil on
the diseased clumps and stubbles.
• Spread Of the fungus occurs in Planting material, and
this can be over
both short and long distance.
• Spread also occurs as spore produced on infected plants,
water – splashed to those nearby.
• The fungus can survive for a few months in the soil on
plant debris & in sugarcane stems.
Favorable conditions:-
• Successive raton cropping
• Water logged conditions and injuries caused by insects.
• Mean temperature range from 29.4 to 31 degree c for
disease development.
• pH 5-6.
• Drought condition during the initial growth is
favorable.
• High humidity.
• Water logged condition of the soil.
Disease management:-
• Use always disease free setts.
• Hot water treatment- 52°c for 8hrs
54°c for 2hrs.
• Hot air treatment- 54°c for 6 hrs.
• Removal of infected stools .
• Crop rotation 2-3 yrs.
• Use disease res.varaties- cos-767,co-840,co-
1148,co-1158, Bo-32, co-975,1148,1158,1336 & 6611,
co-5-561,544,B.O-3,10,47.
Integrated disease
management:-
Reference:-
• Bagyraj and Rangasamy.
• Slideshare
• Biology notes
• https://agritech.tnau.ac.in/crop_protection/sug
arcane_diseases/sugarcane_d4.html

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Red rot of sugarcane and there causes by clostridium falcatum virus.

  • 1. d rot of sugarcane Presented by Nisha nag MSc 4th sem Department of microbiology
  • 2. Content:- 1.Introduction 2.History 3.Casual organism 4.Pathogen character 5.Etiology 6.transmission 7.Disease cycle 8.Symptoms 9.Survival and spread 10.Favorable conditions 11.Disease management 12.Integrated disease management 13.Reference
  • 3. Introduction Red rot is one of the major constraints in the profitable cultivation of sugarcane in many states of India. • Except Maharashtra the disease has been recorded in all the state. • This disease drastically retards the yield and considerably deteriorates the juice quantity and quality thus hitting both the cane growers and millers
  • 4. History:- • First reported in 1893 from Java (Indonesia). • In India first reported in 1901 in Andhra Pradesh. • Disease epidemic occured in 1939-1940 in UP & BIHAR. • Disease is soil and seed borne. • This accepted name, ‘Red rot’ was given to this cane disease in 1906 by sir E.J Butler.
  • 5. Casual organism:- Kingdom :- fungi Phylum :- Ascomycota Class :- sordariomcetes Order :- phyllachorales Family :- Glomerellaceae Genus :- Colletotrichum Species :- falcatum
  • 6. Pathogen Character:- 1.Anamorph :- colletotrichum fakatum 2.Teleomorph :- Glomerella tacumanensis. Etiology :- Caused by collectotrichum falcatum, a facultative saprophyte. • Sexual stage known as Glomerella tucumanensis. • Mycellium:- both intracellular and intercellular. • Asexual fruiting bodies known as Acervuli. • Conidiophores:- linear or club shaped, hyallne,septate. • Conidia:- elongated,single celled, thin walled, uninucleate, colourless, sickle shaped (falcate) with granuar protoplasm with a large oil globule.
  • 7.
  • 8. Transmission:- • Primary transmission through soil and disease sets while the secondary transmission through air, rain splash and soil.
  • 9. Disease cycle:- • The fungus is sett born. • The fungus also persist in the soil on the disease clumps and stubble as acervulli. • The primary infection is mainly from infected setts. • Secondary spread in the field maybe through irrigation water and cultivation tools. • The rain splash air currents and dew drops also help in the spread of conidia from the disease to healthy plant in the field. • The fungus also survives on collateral host like Sorghum vulgare, S. Haleperse and saccharum spontaneum.
  • 10.
  • 11. Symptoms:- Two types of symptoms:- 1.Stalk symptoms 2.leaf symptoms
  • 12. Stalk symptoms:- .The first symptom seen after the rainy season when the normal plant growth stops and sucrose formation was started. • Loose of colour and dropping of third or fourth leaves from the top are the early symptoms the intert weakness withers. • In letter States the canes become shriveled the red shrinks and become longitudinally wrinkled. Such canes are lighter invade and easily broken. • If the disease canes are split open longitudinally, especially when withering of leaves started the pic took red
  • 13. Continue- • In very advance stage, red colour maybe replaced by dirty brown cavities filled with Greyish mycelium and the juice gives a bad smell due to conversion of sucrose into glucose and alcohol as a result of enzymetic action of pathogen.
  • 14. Leaf symptoms:- • Tini reddish lesion of the upper surface of lamina these lesions are 2 to 3 mm in length and about 0.5 mm in width. • Menu trade rot on upper surface of midrib of leaf later on become straw coloured in centre with development of darker acervuli.
  • 15. Survival and spread:- • They reply heavily on water, particularly rainfall, for dissemination. • The fungus is sett borne and also perists in the soil on the diseased clumps and stubbles. • Spread Of the fungus occurs in Planting material, and this can be over both short and long distance. • Spread also occurs as spore produced on infected plants, water – splashed to those nearby. • The fungus can survive for a few months in the soil on plant debris & in sugarcane stems.
  • 16. Favorable conditions:- • Successive raton cropping • Water logged conditions and injuries caused by insects. • Mean temperature range from 29.4 to 31 degree c for disease development. • pH 5-6. • Drought condition during the initial growth is favorable. • High humidity. • Water logged condition of the soil.
  • 17. Disease management:- • Use always disease free setts. • Hot water treatment- 52°c for 8hrs 54°c for 2hrs. • Hot air treatment- 54°c for 6 hrs. • Removal of infected stools . • Crop rotation 2-3 yrs. • Use disease res.varaties- cos-767,co-840,co- 1148,co-1158, Bo-32, co-975,1148,1158,1336 & 6611, co-5-561,544,B.O-3,10,47.
  • 19. Reference:- • Bagyraj and Rangasamy. • Slideshare • Biology notes • https://agritech.tnau.ac.in/crop_protection/sug arcane_diseases/sugarcane_d4.html