This document discusses various pests and diseases that affect leguminous vegetable crops. It provides the scientific names of 11 common leguminous crops and then discusses various pests that affect these crops, including their symptoms, damage caused, and management practices. It also lists and describes 11 common diseases of legumes along with their causal organisms. The document aims to educate farmers about identifying and managing key pests and diseases that impact legume production.
What is greenhouse gasses and how many gasses are there to affect the Earth.moosaasad1975
What are greenhouse gasses how they affect the earth and its environment what is the future of the environment and earth how the weather and the climate effects.
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What are greenhouse gasses how they affect the earth and its environment what is the future of the environment and earth how the weather and the climate effects.
Professional air quality monitoring systems provide immediate, on-site data for analysis, compliance, and decision-making.
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Slide 1: Title Slide
Extrachromosomal Inheritance
Slide 2: Introduction to Extrachromosomal Inheritance
Definition: Extrachromosomal inheritance refers to the transmission of genetic material that is not found within the nucleus.
Key Components: Involves genes located in mitochondria, chloroplasts, and plasmids.
Slide 3: Mitochondrial Inheritance
Mitochondria: Organelles responsible for energy production.
Mitochondrial DNA (mtDNA): Circular DNA molecule found in mitochondria.
Inheritance Pattern: Maternally inherited, meaning it is passed from mothers to all their offspring.
Diseases: Examples include Leber’s hereditary optic neuropathy (LHON) and mitochondrial myopathy.
Slide 4: Chloroplast Inheritance
Chloroplasts: Organelles responsible for photosynthesis in plants.
Chloroplast DNA (cpDNA): Circular DNA molecule found in chloroplasts.
Inheritance Pattern: Often maternally inherited in most plants, but can vary in some species.
Examples: Variegation in plants, where leaf color patterns are determined by chloroplast DNA.
Slide 5: Plasmid Inheritance
Plasmids: Small, circular DNA molecules found in bacteria and some eukaryotes.
Features: Can carry antibiotic resistance genes and can be transferred between cells through processes like conjugation.
Significance: Important in biotechnology for gene cloning and genetic engineering.
Slide 6: Mechanisms of Extrachromosomal Inheritance
Non-Mendelian Patterns: Do not follow Mendel’s laws of inheritance.
Cytoplasmic Segregation: During cell division, organelles like mitochondria and chloroplasts are randomly distributed to daughter cells.
Heteroplasmy: Presence of more than one type of organellar genome within a cell, leading to variation in expression.
Slide 7: Examples of Extrachromosomal Inheritance
Four O’clock Plant (Mirabilis jalapa): Shows variegated leaves due to different cpDNA in leaf cells.
Petite Mutants in Yeast: Result from mutations in mitochondrial DNA affecting respiration.
Slide 8: Importance of Extrachromosomal Inheritance
Evolution: Provides insight into the evolution of eukaryotic cells.
Medicine: Understanding mitochondrial inheritance helps in diagnosing and treating mitochondrial diseases.
Agriculture: Chloroplast inheritance can be used in plant breeding and genetic modification.
Slide 9: Recent Research and Advances
Gene Editing: Techniques like CRISPR-Cas9 are being used to edit mitochondrial and chloroplast DNA.
Therapies: Development of mitochondrial replacement therapy (MRT) for preventing mitochondrial diseases.
Slide 10: Conclusion
Summary: Extrachromosomal inheritance involves the transmission of genetic material outside the nucleus and plays a crucial role in genetics, medicine, and biotechnology.
Future Directions: Continued research and technological advancements hold promise for new treatments and applications.
Slide 11: Questions and Discussion
Invite Audience: Open the floor for any questions or further discussion on the topic.
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I will then present some software engineering and AI techniques that can support the strategic exploration of variability spaces. These include the use of abstractions and models (e.g., feature models), sampling strategies (e.g., uniform, random), cost-effective measurements (e.g., incremental build of software configurations), and dimensionality reduction methods (e.g., transfer learning, feature selection, software debloating).
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Exposé invité Journées Nationales du GDR GPL 2024
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. Our search finds no candidates
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and that the luminosity function normalization and UV luminosity density decline by a factor of ∼ 2.5
from z = 12 to z = 14. We discuss the possible implications of our results in the context of theoretical
models for evolution of the dark matter halo mass function.
DERIVATION OF MODIFIED BERNOULLI EQUATION WITH VISCOUS EFFECTS AND TERMINAL V...Wasswaderrick3
In this book, we use conservation of energy techniques on a fluid element to derive the Modified Bernoulli equation of flow with viscous or friction effects. We derive the general equation of flow/ velocity and then from this we derive the Pouiselle flow equation, the transition flow equation and the turbulent flow equation. In the situations where there are no viscous effects , the equation reduces to the Bernoulli equation. From experimental results, we are able to include other terms in the Bernoulli equation. We also look at cases where pressure gradients exist. We use the Modified Bernoulli equation to derive equations of flow rate for pipes of different cross sectional areas connected together. We also extend our techniques of energy conservation to a sphere falling in a viscous medium under the effect of gravity. We demonstrate Stokes equation of terminal velocity and turbulent flow equation. We look at a way of calculating the time taken for a body to fall in a viscous medium. We also look at the general equation of terminal velocity.
Seminar of U.V. Spectroscopy by SAMIR PANDASAMIR PANDA
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Salas, V. (2024) "John of St. Thomas (Poinsot) on the Science of Sacred Theol...Studia Poinsotiana
I Introduction
II Subalternation and Theology
III Theology and Dogmatic Declarations
IV The Mixed Principles of Theology
V Virtual Revelation: The Unity of Theology
VI Theology as a Natural Science
VII Theology’s Certitude
VIII Conclusion
Notes
Bibliography
All the contents are fully attributable to the author, Doctor Victor Salas. Should you wish to get this text republished, get in touch with the author or the editorial committee of the Studia Poinsotiana. Insofar as possible, we will be happy to broker your contact.
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Functional Magnetic Resonance Imaging (fMRI) provides means to characterize brain activations in response to behavior. However, cognitive neuroscience has been limited to group-level effects referring to the performance of specific tasks. To obtain the functional profile of elementary cognitive mechanisms, the combination of brain responses to many tasks is required. Yet, to date, both structural atlases and parcellation-based activations do not fully account for cognitive function and still present several limitations. Further, they do not adapt overall to individual characteristics. In this talk, I will give an account of deep-behavioral phenotyping strategies, namely data-driven methods in large task-fMRI datasets, to optimize functional brain-data collection and improve inference of effects-of-interest related to mental processes. Key to this approach is the employment of fast multi-functional paradigms rich on features that can be well parametrized and, consequently, facilitate the creation of psycho-physiological constructs to be modelled with imaging data. Particular emphasis will be given to music stimuli when studying high-order cognitive mechanisms, due to their ecological nature and quality to enable complex behavior compounded by discrete entities. I will also discuss how deep-behavioral phenotyping and individualized models applied to neuroimaging data can better account for the subject-specific organization of domain-general cognitive systems in the human brain. Finally, the accumulation of functional brain signatures brings the possibility to clarify relationships among tasks and create a univocal link between brain systems and mental functions through: (1) the development of ontologies proposing an organization of cognitive processes; and (2) brain-network taxonomies describing functional specialization. To this end, tools to improve commensurability in cognitive science are necessary, such as public repositories, ontology-based platforms and automated meta-analysis tools. I will thus discuss some brain-atlasing resources currently under development, and their applicability in cognitive as well as clinical neuroscience.
PESTS AND DISEASES OF LEGUMINOUS VEGETABLE CROPS.pptx
1. TOPIC:- PESTS AND DISEASES OF
LEGUMINOUS VEGETABLE CROPS
PRESENTED BY: Ramesha N M
2. Sl. No Crop Scientific name
1 Lima bean Phaseolus lunatus
2 French bean Phaseolus vulgaris
3 Cluster bean Cyamopsis tetragonolobus
4 Soya bean Glycine max
5 Broad bean Vicia faba
6 Indian bean Lablab purpureus
7 Garden pea Pisum sativum
8 Chick pea Cicer arietinum
9 cowpea Vigna unguiculata
10 Green gram Vigna radiata
11 Black gram Vigna mungo
3. Spiny pod borer: Etiella zinckenella
Damage symptoms:
• Dropping of flowers and young pods.
• As the larva develops within the pod, faeces accumulate
causing soft, rotten patches on the pod.
• Seeds are either partially or entirely eaten, and considerable
frass and silk are present.
• Older pods marked with a brown spot where a larvae has
entered.
4. Deep ploughing is likely to kill the diapausing pupae.
Hand picking of the caterpillar and pupae during the early
stages of infestation reduces the pest damage.
Malathion 50%EC @ 600 ml in 200- 400 l water per acre.
Egg parasitoids: Trichogrammatoidea armigera.
Larval parasitoids: Bracon hebetor, Phanerotoma sp.,
Tetrastichus sp, Phanerotoma.
5. Damage symptoms:
At early stage they feed on the foliage and sometime cause
serious defoliation.
During reproductive stage they bore the developing pod and
feed on the seeds with its head typically thrust inside and
most of the part of the body outside.
Management
Bird perches @ 50 / ha
Set up light trap
Pheromone traps @ 12 / ha
ETL: 5-6 eggs or 2-3 small larvae / plant
NPV @ 250 LE / ha + 0.1 % teepol
NSKE 5% twice followed by Triazophos 0.05 %
Monocrotophos 36 SC@ 625 ml/ha
6. Damage symptoms:
The maggots bore into the stem thereby causing withering and
ultimate drying of the affected shoots, thus reducing the bearing
capacity of the host plants.
The adults also cause damage by puncturing the leaves, and the
injured parts turn yellow.
The damage is more severe on seedlings than on the grown up
plants.
Management
avoid sowing of the crop earlier than mid-October to check the
attack of the pest.
Sow the crop in the second fortnight of October to escape the
damage of the pest.
Apply 7.5 kg of phorate 10G or 25 kg of carbofuran 3 G per ha
in furrows at the time of sowing On the crop, spray three times
750 ml of oxydemeton methyl 25 EC in 750 L of water per ha.
The first application should be just after germination and the
other two at an interval of 2 weeks each.
7. Damage symptoms:
The larvae burrow straight through the pods to feed on the
seed, so are not readily found for identification until seed is
mature (above) and it is too late for control. Early harvest of
peas also reduces pod shatter and pea splitting losses.
Management
Cut the volunteer and weed plant.
Proper sanitation of godown store house.
Disinfect the gunny bags that carry the pea grains.
Parasitoids : Larval parasitoid: Dinarmus basalis
8. Damage symptoms:
Both nymphs and adults suck the sap from young shoots, ventral surface of tender
leaves, inflorescence and even on stems.
Curling and distortion of leaves, stunting and malformation shoots occur.
Leaves turn pale and dry. Honeydew secretion of aphids leads to sooty mould which
hinders the photosynthetic activity of the plants.
Management
Judicious use of nitrogenous fertilizers
Regular field monitoring for pest & defender population, barrier crops like mustard
crop around the field.
Plant tall border crops like maize, sorghum or millet to reduce pest population.
Release 1st instar larvae of green lacewing (Chrysoperla zastrow sillemi) @ 4000/acre
Carbofuran 3% CG @ 400 g per acre
Parasitoids of aphids: Parasitic wasp, Aphadius sp., Aphalinus sp., Diaeretiella rapae
Predators of pea aphids : Lacewing, ladybird beetles Predatory mite, Syrphid fly
9. Damage symptoms:
The large number of tunnels made by the larvae between the
lower and upper epidermis interferes with photosynthesis and
proper growth of the plants, making them look unattractive.
Leaves with serpentine mines
Drying dropping of leaves in severe case
10. Remove and destroy the infested leaves identified by the mines
and blotches.
Use yellow sticky traps/ cards for leaf miners adult fly.
Maggots are parasitized by Braco nids, Eulophids.
Predators: Green lacewing, ladybird beetle, spider, red ant
Foliar spray with methyl demeton 2 ml/l or dimethoate 2 ml/l
was effective.
11. BEAN APHIDS : Aphis craccivora
Damage symptoms:
Twisting of leaves, poor pod development,
devitalization of plants and sooty mould.
It acts as a vector of Rosette disease in groundnut
and broad bean virus in pea.
Management :
Spraying with tobacco decoction (1 kg tobacco
boiled in 10 lit of water of 1⁄2 hour and make up to
30 lit + 100 g soap) and systemic insecticides like
dimethoate 2 ml/l or phosphamidon 2 ml/l or
malathion 2 ml/l are effective.
12. POD BUG : Cavigralla gibbosa, Riptortus linearis
Damage symptoms:
Shoots fade, pods shrivel and seeds with dark patch.
loose germination capacity due to the feeding of
bugs.
Management :
Collection of bugs and their destruction by dipping
into kerosinized water and dusting or spraying with
carbaryl 10D 10 kg/ac or foliar spraywith dimethoate 2
ml/l or monocrotophos 1.5 ml /l are effective.
13. LEAFHOPPER: Empoasca kerri
Damage symptoms :
The leaves turn brown, dry and brittle, a condition called “hopper burn”.
Attacked leaflets become cup shaped and yellow at edges.Heavy attack
result in the leaflets turning red-brown with subsequent defoliation and
stunting.
Management
Collection of bugs and their destruction of infested leaves, seed
treatment with carbosulfan @ 30-40g/kg seed, spraying with
thiamethoxam 0.4g/l or imidacloprid 0.4ml/l have been found effective.
14. GREEN PLANT BUG/STINK BUG : Nezara viridula
Damage symptoms :
suck sap from tendershoots and developing pods in large
numbers, due to that shoots fade.
Adults aestivate during April to June and with first rains they
mate andoviposit.
Management
Mechanical collection of nymphs and adults and destroying them
and
foliar spray with carbaryl 3 g/l or endosulfan 2 ml/l is
recommended.
15. GRAM POD BORER: Helicoverpa armigera
Symptoms of damage:
Defoliation in early stages.
Larva’s head alone thrust inside the pods and the
rest of the body hanging out. Pods with round
holes.
Management
Bird perches @ 50 / ha
Set up light trap
Pheromone traps @ 12 / ha
ETL: 5-6 eggs or 2-3 small larvae / plant
NPV @ 250 LE / ha + 0.1 % teepol
NSKE 5% twice followed by Triazophos 0.05 %
Monocrotophos 36 SC@ 625 ml/ha
16. SPOTTED POD BORER: Maruca testulalis
Symptoms of damage
Bore holes on the buds, flower or pods
Infested pods and flowers are webbed together.
Management:
ETL: 3/plant
(Spray fluid 625 ml/ha)
Note : When the activity of coccinellid predator (both
grubs and adults) is seen, insecticide application
should be avoided.
17. Deep summer ploughing in 2-3 years to eliminate quiescent pupa.
Collect and destroy larvae and adults to the extent possible.
Install pheromone traps at a distance of 50 m @ 5 traps/ha for each
insect pest.
Install Bird perches @ 50/ha.
Setting of light traps (1 light trap/5 acre) to kill moth population.
Control is achieved by releasing of Trichogramma chlionis at weekly
intervals @1.5 lakh/ha/ week for four times.
Conserve green lacewing, predatory stink bugs, spider, ants
Application of NPV 250 LE /ha with teepol 0.1% and Jaggery 0.5%
thrice at 10 – 15 days interval commencing from flowering stage.
Bt @ 600 g, neem oil/ pungum oil 80 EC @ 2ml/lit
Spray NSKE 5% twice followed by triazophos 0.05%.
Apply any one of insecticides at 25 kg/ha. quinalphos 4D, carbaryl
5D
Quinalphos 25 EC @ 1000 ml/ha.
18. WHITEFLY: Bemisia tabaci
Symptoms of damage
Leave mottled and yellowish in colour
vector of yellow mosaic virus
Management
• Shaking the infested plants over the vessels of oil
and water or oily cloth gives most effective
•Spray methyl demeton 25 EC 500ml or dimethoate 30
EC 500 ml or phosphomidon 85 WSC 250 ml/ha
19. GREEN POD BORING CATERPILLAR OR LENTIL POD BORER :
Damage symptoms :
The larvae feed on floral parts, newly formed pods and seeds
in developing pods.
Faecal pellets inside damaged pods and small round holes on
pods plugged with excreta can be noticed.
Management
Chlorpyriphos 2.5 ml/l at initiation of flowers
Quinalphos 2 ml/l or acephate 1.5 g/l at flowering and
fruiting using 750 –1000 l of spray fluid with High Volume
sprayer.
In severe incidence, indoxacarb 1 ml/l or spinosad 0.3 ml/l
20. BLUE BUTTERFLIES : Lampides boeticus
Damage symptoms
It is seen on redgram, cowpea, lab lab, niger etc .
The eggs are laid on flower buds.
After hatching the tiny caterpillars enter into
unopened flower bud and feedinside.
Afterwards they may attack another flower or enter
a pod and feed on the developing seeds.
Management :
Foliar spray with carbaryl 3 g/l or endosulfan 2
ml/l is effective.
21. STEM FLY: Ophiomyia phaseoli
Damage symptoms
Yellowish maggots bore into nearest vein, reach the stem
through petiole, bore down the stem and feed on cortical layers
and may extend to tap root.
Distinct tunnel of stem split open. Death of plant or branches.
Pupation is at ground level within the stem. Adult fly exits
through a thin semi transparent window.
MANAGEMENT
Seed treatment with imidacloprid 3 g/kg seed gives protection
upto 30 days.
Foliar spray with acephate 1.5g/l or dimethoate 2 ml/l or
monocrotophos 1.6ml/l
23. Disease symptoms
A grayish white, moldy growth appears on the lower
leaf surface, and a yellowish area appears on the
opposite side of the leaf.
Infected leaves can turn yellow and die if weather is
cool and damp.
Stems may be distorted and stunted.
Brown blotches appear on pods, and mold may grow
inside pods.
24. Favourable conditions:
High humidity and low temperatures (5- 15°C) for few days are
ideal for infection and development of disease.
Survival and spread:
Primary infection by soil, seed and water Secondary infection by
sporangia through rain splash or wind
Management:
• The diseased plants should be removed and burnt soon after
detecting in the field.
• The disease can be effectively managed by giving 3-5
prophylactic sprays with 1% B.M or Fosetyl -Al (Aliette) 0.2% or
metalaxyl + mancozeb 0.3 to 0.4% or Azoxystrobin or
Dimethomorph.
25. Disease symptoms:
It attacks leaves first producing faint, slightly
discolored specks from which grayish white
powdery growth of mycelium develop.
Powdery growth spread over leaf, stem and pod.
The leaves turn yellow and die.
The fruits do not either set or remain very small.
It causes defoliation.
Later stages, powdery growth also covers the pods.
26. Survival and spread:
Powdery mildew spores are carried by air and once
active, will continue to spread in dry conditions.
Favourable conditions:
Warm (temperature 15-25°C), humid (over 70%
relative humidity) conditions for 4-5 days late in the
growing season, during flowering and pod filling,
favour disease development
27. Cultural control:
Use resistant varieties.
Burn infected pea stubble soon after harvest where
practicable.
Avoid sowing field pea crops adjacent to last season’s
stubble.
Control volunteer field peas which can harbour disease.
Adopt bower system.
Chemical control:
Benomyl 50 % WP @ 80g in 240 l water per acre or
carbendazim 50% WP@ 100g in 240 l water per acre or as
required depending on stage of crop.
Second spray after 15 days of interval or sulphur 40% WP
2.26- 3.00 Kg in 300- 400 l of water per acre. Second spray
after 25 days of interval
28. Disease symptoms:
Leaves of infected plants exhibit many small,
orange-brown pustules usually at the lower
surface.
Severely infected leaves wither and may drop from
the plant.
Larger pustules occur on the stems and isolated
pustules may be found on the pods.
Severe infection may result in reduced seed size
and may cause yield losses of up to 30%.
29. Management
• Adjust sowing dates to avoid severe infection of plants
in the field
• Protect the crop with mancozeb or zineb @ 2 kg/ha or
wettable sulphur @0.3%.
Survival and spread:
Euphorbia and infected vegetation residues are sources of the infection.
The agent is not transferred by seeds.
Favourable conditions:
Frequent precipitations, plentiful dews and air temperature of 20-
25°Cpromote development.
Dry and hot weather restrains the disease development.
30. Disease symptoms:
Symptoms first appear as small, purplish-brown
and irregular flecks.
Under continued humid conditions, the flecks
enlarge and coalesce, resulting the lower leaves
becoming completely blighted.
Severe infections may lead to girdling of the stem
near the soil line, which is known as foot rot.
Foot rot lesions are purplish-black in colour and
may extend above and below the soil line.
31. Survival and spread:
Ascospores carried long distances by wind.
The asexual conidia travel short distances to new
hosts via water splashes from rain.
Infection originates from diseased seed or from
spores growing on debris in the soil near pea plants.
Favourable conditions:
Favorable conditions are warm humid conditions
with a temperature is about 15to 25 degree celsius.
32. Sow disease-free seed.
Follow rotation crop.
Seed treatment with Carbendazim @ 1g/kg of seed.
or Hot water seed treatment (52 C for 10 min) to
lower the infestation.
Spray the crop with Mancozeb @ 2.5g/lit if noticed
during the growth period.
or Spray Wettable sulphur at the rate of 2.3g/lit of
water.
33. Symptoms
•It comes in the early stages i.e up to six weeks from sowing.
•Drying plants whose foliage turns slightly yellow before death,
scattered in the field is an indication of the disease.
•Seedling become chlorotic.
•The joint of stem & root turns soft slightly contracts and begins
to decay.
• Infected parts turn brown white.
•Black dots, like mustard in shape known as sclerotia are seen
appearing on the white infected plant parts
Survival and spread:
The infection caused by hard sclerotia left in soil or in plant
debris.
Favourable conditions:
When adequate moisture is available and temperature ranges
between 4 and 20°C. However, light is essential for stimulation of
apothecial production
34. Management
•Deep ploughing in summer.
•Avoid high moisture at the sowing time.
•Seedlings should be protected from excessive moisture.
•Destroy the residues of last crop and weed before sowing and
after harvest.
•All un decomposed matter should be removed from the field
before land preparation.
•Treat the seeds with a mixture of Carbendazim 1g per kg of
seed.
35. Survival and spread:
Primary infection by Soil, Seed, Water
Secondary infection by Conidia through rain
splash.
Favourable conditions:
A soil temperature of 23° to 27°C is most
favourable for Fusarium wilt.
Hot weather and warm soils.
36. • In peas, PSbMV causes stunting, reduced internode lengths
and malformation.
• Often results in the formation of malformed terminal rosettes
.The virus can delay plant maturity, leading to uneven crop
maturation .
• Infected leaves can exhibit clearing and swelling of veins,
slight downward curling of leaf margins, chlorosis and/or a
mottled or mosaic discoloration . Pods often are deformed ,
and seeds produced from infected plants can exhibit
pronounced discoloration , splitting of seed coats .shriveling
and reduced size.
• Secondary spread of PSbMV is facilitated by aphids
Management
• pest management
• Use of resistant varities like North Dakota
37. Symptoms
Leaves and seed –necrotic area , discolorations
Dwarfing and early senescence.
Management:
Removal of infected seeds
Avoid cultivation in land with vector infested soil.
38. Vector : aphid
Symtoms :
Chloratic or necrotic lessions, malformation of
leaves and plants.
Enations due to hyperplasia.
Management
Resistant / tolerant cultivars.
Vector control
39. Disease symptoms:
Symptom of the disease is more pronounced in 3
to 5 week old plants. In young seedlings,
cotyledons droop and wither.
Yellowing of lower leaves and stunting of plants.
The xylem vessels develop brown discoloration and
get distorted.
Leaflet margins curl downward and inward.
The stem may be slightly swollen and brittle near
the soil.
Internal woody stem tissue often is discolored,
turning lemon brown to orange brown.
Externally, the root system appears healthy;
however, secondary root rots are likely to occur on
plants wilted for long periods.
Eventually, wilted plants may die.
40. Deep summer ploughing
Follow crop rotation measures continuously.
Always use disease free seeds.
Avoid sowing when temperatures are high.
Apply FYM 10-15 cart load/ha.
Seed treatment with T. viride @4g/kg or P. fluorescens @
10g/ kg of seed or Carbendazim or Thiram 2g/kg of seed.
Spot drenching with Carbendazim 1g/lit or P.
fluorescens / T. viride 2.5 kg/ha with 50 kg FYM.
Seed treatment with Carbendazim at the rate of 1g/kg of
seed .
Seed treatment with Thiram + Carbandizm @ 1g+2g per
kg of seed.
41. Symptoms
Pods with black, sunken lesions or reddish-brown
blotches.
Black, sunken lesions about ½ inch in diameter
develop on stems, pods and seedling leaves
(cotyledons) but are most prominent on pods.
A salmon colored ooze on lesions and the veins on
lower leaf surfaces turns black. On lima beans,
symptoms are sooty- appearing spots on leaves
and pods.
Anthracnose develops primarily during the spring
and fall when the weather is cool and wet, and not
during our hot, dry summers. Lima beans are
particularly susceptible.
42. Mode of spread and survival
The fungus is seed borne and can survive from one
season to another in debris from infected plant as
well as in diseased seed. The fungus can remain
alive in seeds even after the seeds are dead
Management
• Prevent this disease by using certified disease-
free seed for planting and removing all plant
debris after harvest.
• Anthracnose can survive in the soil for two years
on plant debris or be brought to the garden on
infected seeds.
43. Symptoms
• Symptoms vary and include rapid death of young
succulent plants.
• Discoloration of taproots, longitudinal cracks of
the stems, stunting, wilting and poor yields.
• Complete control of root rot and damping off is
difficult, and no variety of cowpea is resistant to
root rot.
• Persistent damp weather prior to development of
the first true leaf and also the crowding of
seedlings due to poor seed spacing may increase
damping off.
44. Survival and spread:
Primary infection by soil, seed and water
Secondary infection by conidia through rain splash
or wind
Favourable conditions:
Cool, wet weather conditions.
Favourable conditions are higher soil temperatures
25° to 30°C and moderate soil moisture
45. Do not plant bean seeds in an area that had
disease for two to three years. Avoid overhead
watering and avoid splashing soil onto the plants
when watering. Fungicide sprays of fixed copper
are the only recommended chemical that can be
used on lima beans for anthracnose control.
Use healthy seed for planting
Seed treatment with Carbendazim@2g/kg seed
Protect the crop by spraying 0.2 % Benlate or
Bavistin or Zineb or Maneb
@2Kg/ha at 7-10 days interval
46. Symptoms
Bean rust is mainly a disease of bean leaves that
causes rust-colored spots to form on the lower leaf
surfaces. Severely infected leaves turn yellow, wilt,
and then drop off of the plant. Stems and pods
may also be infected.
This disease is caused by the fungus Uromyces
appendiculaters. It affects most types of beans
under humid conditions.
47. Mode of spread and survival
The rust fungus is not seed borne, but can be disseminated
locally by farm tools, insects, animals or other moving bodies.
However wind is the principal agent for long distance spore
dissemination.
Management
The fungus survives the winter in the soil, on plant debris and
even on poles used the previous year.
In gardens where rust has been severe, crop rotation is
important. As plants begin to bloom, sulfur or chlorothalonil
can be sprayed weekly on snap and green beans only.
Do not apply chlorothalonil to lima (butter) beans. Wait seven
days between spraying and harvest when using chlorothalonil
on beans, and 14 days on Southern peas. Apply chemicals
according to directions on the label.
48. Do not plant beans in low, poorly drained areas.
Plant on raised beds.
Plant after the soil has warmed to 69° F at a 4 inch depth.
Reduce disease buildup in the soil by rotating locations in
the garden where you plant bean or pea with other
vegetables.
Try to avoid injury to the root system, which often occurs
during planting, through cultivation or due to a large
population of nematodes in the soil.
Remove crop debris immediately after harvest. Plant seeds
previously treated with captan.
• Apply chemicals according to directions on the label. Soil
application P. fluorescens or T. viride– 2.5 kg / ha + 50 kg
of well decomposed FYM or sand
• Spot drenching with Carbendazim @ 1 gm/ litre
49. Symptoms
The stems, leaves and fruits of bean plants can be infected by
either disease. Rain and damp weather favor disease
development.
Halo blight occurs primarily when temperatures are cool.
Light greenish-yellow circles that look like halos form around
a brown spot or lesion on the plant.
With age, the lesions may join together as the leaf turns
yellow and slowly dies. Stem lesions appear as long, reddish
spots.
Leaves infected with common blight turn brown and drop
quickly from the plant. Common blight infected pods do not
have the greenish-yellow halo around the infected spot or
lesion. Common blight occurs mostly during warm weather.
50. Mode of spread and survival
The pathogen is seed borne and the disease spread through
wind splashed rains from diseased to healthy plants.
In new area disease spreads through infected seeds.
Management
Both of these diseases come from infected seeds. The
diseases spread readily when moisture is present.
Avoid overhead watering and do not touch plants when the
foliage is wet.
The bacteria can live in the soil for two years on plant debris.
Do not plant beans in the same location more frequently than
every third year. Buy new seeds each year.
Fixed copper can be applied at ten day intervals. Wait one day
between spraying and harvest
51. Symptoms
Affects only beans (Phaeolus vulgaris & other Phaseolus
species)
Symptoms vary according to the variety of bean affected, time
of infection and
environmental conditions
Leaves show mosaic pattern, i.e., light green areas alternate
with dark green areas.
Diseased leaves become rough, and show blisters on the leaf
lamina
Leaf size, petiole length and plant height reduced. Leaves
curl downward
Diseased plants produce fewer pods which are smaller in size
Seeds become smaller, malformed and aborted
Pathogen
Cucumber mosaic virus
52. Mode of spread and survival
Seed, sap, graft and aphid transmissible. Infected seeds are
primary means of spread.
Aphis craccivora, transmit the virus in a non-persistent
manner
Management
There are no recommended chemical controls for these
problems. Many of these viruses are transmitted by aphids
and are also transmitted through seed. For this reason it is
unwise to save seeds from year to year.
Roguing out of cowpea mosaic virus diseased plants in the
early stage of growth up to 30 days and spraying twice at
fortnightly intervals with Monocrotophos 500 ml/ ha (or)
Methyldemeton 25 EC 500 ml/ha.
53. Bean yellow mosaic is caused by Bean yellow mosaic
virus, The virus is vectored by several aphid species (at
least 20) in a non-persistent manner; It is also
transferred efficiently by mechanical inoculation.
Bean yellow mosaic symptoms appear as distinct dark
and yellowish green areas. Infected plants often have
bright yellow spots that intensify in color with plant
age. a faint yellow mosaic and stunting or distortion of
leaves and severe stunting. Pod symptoms appear at a
light green mottle and may be malformed.
Management
Use of resistant variety
Cultural control practices
54. Symptoms
This fungal disease, caused by Cercospora species,
occurs primarily on the lower leaves of plants as
irregular, tan spots.
Severe infection causes excessive leaf drop and
stunting of the plant.
Infection is worse during periods of extended rainfall,
high humidity and temperatures between 75 to 85° F.
Management
Use disease-free seed for planting. Remove all debris in
the garden after harvest. Do not plant beans in the
same area for two to three years. There are no resistant
varieties or recommended chemicals for this disease in
the home garden.
55. Symptoms
Small, soft, watery spots that are caused by the fungus
Sclerotinia sclerotiorum occur on the stems, leaves and pods
of beans.
These spots enlarge rapidly under cool, moist conditions, and
run together, girdling the stem.
Infected pods turn into a soft, watery mass, before dying out
and turning brown.
Soon infected areas are covered by a white fungal growth.
Management
Improve air circulation between plants and rows. Too much
fertilizer favors heavy vine growth, creating areas for the
disease to develop. There are no recommended chemical
controls for the home garden.