This document discusses a case of hepatic lipidosis in a 5-year-old Shetland pony. Biochemistry results showed markedly increased levels of GLDH, triglycerides, bilirubin, ALKP, AST, GGT, and CK, as well as moderately decreased sodium, chloride, and urea levels. Hepatic lipidosis was diagnosed and thought to be caused by poor quality feed or decreased intake during a period of high energy demand, leading to fatty infiltration of the liver. Treatment involved addressing hepatic failure, improving energy balance, eliminating stress, and increasing triglyceride uptake in peripheral tissues. Prognosis for hyperlipidemia is poor, with mortality reported in up to 80%