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INDEX:
1. Introduction
2. Types of muscle
3. Development of muscle
4. Primary muscles of mastication
5. Suprahyoid muscles
6. Infrahyoid muscles
7. Masticatory system disorders
8. Conclusion
9. References
NOTHING IS MORE FUNDAMENTAL TO
TREATING PATIENTS THAN KNOWING THE
ANATOMY
- JEFFERY P OKESON
Muscle: A tissue composed of contractile cells or fibers that effect movement of an
organ or part of the body.(GPT 9)
Mastication: The process of chewing food for swallowing and digestion.(GPT 9)
There are basically three types of muscles:
1. Skeletal muscle
2. Smooth muscle
3. Cardiac muscle
INTRODUCTION
Glossary of prosthodontic terms-9
1.Have light and dark
bands or striations.
2.Cylindrical
multinucleated
unbranched cells
3.Voluntary
4.Contract quickly but
get fatigued easily
5.Seen in
limbs,tongue,body,face,
neck
1.Do not show light and
dark bands.
2.Spindle shaped
uninucleated cells
3.Involuntary
4.Contract slowly but
don’t get fatigued easily
5.Seen in
ureters,bronchi of
lungs, ailmentary canal
and blood vessels.
1.Structure in between
striated and non
striated muscle
2.Cylinder shaped
uninucleated branched
cells.
3.Involuntary
4.Contract rhythmically
and quickly ,hence do
not get fatigued
5. Seen in walls of heart
DEVELOPMENT
First
pharyngeal
arch
Intra
embryonic
mesoderm
Myoblast
MyotubeMuscle fibre
Textbook Of Embryology- Inderbir Singh
CLASSIFICATION:
Muscles of
mastication
Accesory
1.Masseter
2.Temporalis
3.Lateral
pterygoid
4.Medial pterygoid
Suprahyoid:
1.Digastric
2.Stylohyoid
3.Mylohyoid
4.Geniohyoid
Primary
Infrahyoid:
1.Sternohyoid
2.Thyrohyoid
3.Omohyoid
HUMAN ANATOMY VOL 3- B.D. CHAURASIA
FUNCTIONAL
CLASSIFICATION:
Muscles of
mastication
Elevators
1.Masseter
2.Temporalis
3.Medial pterygoid
4.Upper head of
lateral pterygoid
Depressors
1.Digastric
2.Mylohyoid
3.Geniohyoid
4.Lower head of lateral
pterygoid
Masseter
• Thick quadrilateral muscle
• Second most efficient masticatory muscle
• Multipennate arrangement
• Buccal pad of fat seperates the muscle from
buccinator.
HUMAN ANATOMY VOL 3- B.D. CHAURASIA
ORIGIN:
• By a thick tendinous aponeurosis from the
zygomatic process of maxilla
• From the anterior 2/3rds of the lower border of
zygomatic arch.
Superficial head
• Posterior third of the lower border of zygomatic
arch.
• Whole of the medial surface of the zygomatic
arch
Deep head
HUMAN ANATOMY VOL 3- B.D. CHAURASIA
INSERTION:
Superficial
head
• Middle and lower part of lateral
surface of ramus of mandible
Deep head
• Upper part of ramus and coronoid
process of mandible
HUMAN ANATOMY VOL 3- B.D. CHAURASIA
Actions:
Elevates the mandible
HUMAN ANATOMY VOL 3- B.D. CHAURASIA
Vascular Supply
Vascular
supply
Massetric
artery
Facial
artery
Transverse
facial
artery
HUMAN ANATOMY VOL 3- B.D. CHAURASIA
Nerve Supply:
Massetric nerve
HUMAN ANATOMY VOL 3- B.D. CHAURASIA
Examination:
Burket’s Oral Medicine –Eleventh Edition
Temporalis:
• Largest among all the mastication muscles.
• Broad ,radiating and fan shaped
• Fills the temporal fossa
HUMAN ANATOMY VOL 3- B.D. CHAURASIA
Actions
• Anterior Vertical Fibers
- Elevates the mandible
- Side to side grinding movements
• Posterior Horizontal Fibers
- Retracts the mandible
HUMAN ANATOMY VOL 3- B.D. CHAURASIA
Vascular Supply
• Supplied by deep temporal branches from second part of maxillary artery.
HUMAN ANATOMY VOL 3- B.D. CHAURASIA
Nerve Supply
Deep temporal branches of anterior division of
mandibular nerve.
HUMAN ANATOMY VOL 3- B.D. CHAURASIA
Examination:
Anterior region Middle region Posterior region
BUREKET’S ORAL MEDICINE-ELEVENTH EDITION
Lateral Pterygoid
• Also called as external pterygoid muscle.
• Short and conical
• Has upper and lower heads
Origin:
Upper head Lower head
INSERTION:
• Fibers Run Backward And Laterally And Converge
To Be Inserted Into :
-Pterygoid Fovea On The Anterior Surface Of Neck
Of Mandible
-Anterior And Medial Border Of The Articular Disc
And Capsule Of TMJ.
HUMAN ANATOMY VOL 3- B.D. CHAURASIA
Actions:
Depression
of
mandible
Protrusion
of
mandible
Side to
side
movement
• Vascular supply:
Pterygoid branches from the maxillary artery.
The ascending palatine branch of the facial artery.
• Nerve supply:
Branch from anterior division of mandibular nerve.
HUMAN ANATOMY VOL 3- B.D. CHAURASIA
Examination:
Functional manipulation of Superior lateral
pterygoid :
Contraction – if it is source of pain, clenching
increases pain.
If a tongue blade is placed between posterior
teeth bilaterally and patient clenches on the
separator pain increases with contraction of SLP
Burket’s Oral Medicine- 11th Edition
Functional manipulation of inferior
lateral pterygoid :
Contraction – Ask the patient to
protrude against resistance
provided by the examiner.
If ILP is the source of pain – activity
will increase pain
Burket’s Oral Medicine -11th Edition
Medial Pterygoid
• Quadrilateral muscle
• Has a small superficial and large deep head
• Deepest among the muscles of mastication
• Also known as internal pterygoid muscle
Origin And Insertion:
Actions:
Elevation Protrusion
Contralateral
excursion
• Vascular supply: Pterygoid branch of second part of maxillary artery.
• Innervation: Nerve to medial pterygoid ,branch of main trunk of mandibular nerve.
HUMAN ANATOMY VOL 3- B.D. CHAURASIA
Examination:
• Palpation is done mainly by intra oral approach.
• The region of its attachment to inner aspect of
angle of mandible can be palpated by sliding finger
lingually and applying pressure at the insertion
of muscle and placing one hand extraorally below
the border of mandible
• Functional Manipulation:
1. Contraction: 2.Stretching:
If it is
source of
pain
Clenching
teeth together
wil increase
pain
If it is
source of
pain
Opening
mouth wil
increase pain
ACCESORY MUSCLES OF
MASTICATION
Suprahyoid Muscles
1. Digastric
2. Geniohyoid
3. Mylohyoid
4. Stylohyoid
DIGASTRIC:
Innervation
• Anterior belly: Nerve to
mylohyoid
• Posterior belly: Facial nerve
Actions
• Depresses the mandible
• Elevates hyoid bone
HUMAN ANATOMY VOL 3- B.D. CHAURASIA
GENIOHYOID
Innervation
First cervical nerve
through hypoglossal nerve
Actions
Elevates hyoid bone
When hyoid bone is fixed,
depresses mandible
HUMAN ANATOMY VOL 3- B.D. CHAURASIA
Mylohyoid
• Innervation: Nerve to mylohyoid,branch of inferior alveolar nerve
• Vascular supply: By facial and lingual artery
• This muscle separates sublingual and submandibular salivary glands
Actions:
Actions
Elevates floor of
mouth
Elevation of hyoid
bone
Depression of
mandible
Stylohyoid
•Innervation Facial nerve
•Actions
Elevation of hyoid
Pulls the hyoid
backwards
HUMAN ANATOMY VOL 3- B.D. CHAURASIA
INFRAHYOID MUSCLES:
Infrahyoid group of muscles consist of:
1. Omohyoid
2. Thyrohyoid
3. Sternohyoid
4. Sternothyroid
Omohyoid:
Origin: Upper border of scapula near
suprascapular notch
Insertion: Lower border of body of hyoid bone
Innervation: Superior belly by superior
root of ansa cervicalis
Inferior belly by ansa cervicalis
Action: Depresses the hyoid bone
HUMAN ANATOMY VOL 3- B.D. CHAURASIA
Sternothyroid
Origin: Posterior surface of manubrium
sterni,adjoining parts of first costal cartilage
Insertion: Oblique line of thyroid cartilage
Innervation: Ansa cervicalis
Actions: Depresses the larynx and helps in
swallowing
HUMAN ANATOMY VOL 3- B.D. CHAURASIA
Thyrohyoid
Origin: Oblique line of thyroid cartilage
Insertion: Body of hyoid bone
Innervation: First cervical nerve
through hypoglossal nerve
Actions: Depresses the hyoid bone and
elevates the larnyx
HUMAN ANATOMY VOL 3- B.D. CHAURASIA
Sternohyoid
Origin: Posterior surface of manubrium sterni and
adjoining part of clavicle
Insertion: Medial part of lower border of hyoid bone
Innervation: Ansa cervicalis
Actions: Depresses the hyoid bone
HUMAN ANATOMY VOL 3- B.D. CHAURASIA
Masticatory Muscle
Disorders
• Muscle tissue becomes compromised or unhealthy –
- due to muscle overuse
- physical trauma
- over stretching
- receiving a blow to muscle tissue itself.
• Muscles of mastication become compromised through increased activity.
• Some of the common masticatory disorders are as follows:
1.Trismus
2.Protective co contraction
3. Local muscle soreness
4.Myospasm
5. Myofascial pain
6.Centrally mediated myalgia(chronic myositis)
7.Myofibrotic contracture
Trismus:
What is trismus?
Trismus is inability to open
the jaw
Etiology:
Trauma, infection, drugs,
dental treatment, congenital
problems
Management:
Heat therapy
Analgesics
Physiotherapy
Soft diet
Muscle relaxants
Dhanrajani PJ, Jonaidel O. Trismus: aetiology, differential diagnosis and treatment. Dent update 2002;29:88-94
Protective Co Contraction:
• Protective co-contraction is the initial response of a muscle to altered sensory or
proprioceptive input or injury (or threat of injury).
• This response has also been called protective muscle splinting.
• When protective co contraction occurs ,CNS increases activity of antagonistic
muscle during contraction of agonist muscle
• For example,patient who is experiencing protective co contraction wil demonstrate
slightly increased muscle activity in the elevators durimg mouth opening .
• Not a pathological condition but a normal guarding or protective mechanism
Management Of Occlusion & TMD – Okeson
Etiology
1. Altered
sensory or
proprioreceptory
input.
2.Constant deep
pain input
3.Increased
emotional stress
Symptoms
1.Structural
dysfunction
2.No pain at rest
3.Increased pain
with functuon
4.Feeling of
muscle weakness
Treatment
1.No indication to
treat the muscle
condition itself.
Treatment
should instead be
directed toward
the reason for
the co-
contraction
Management Of Occlusion & TMD – Okeson
Local muscle soreness:
• Local muscle soreness is a primary noninflammatory myogenous pain disorder.
• It is often the first response of the muscle tissue to continued protective co-
contraction and therefore the most common acute muscle pain disorder in dental
practice.
• Although co-contraction represents a cns-induced muscle response, local muscle
soreness represents a change in the local environment of the muscle tissues.
• It represents the initial response to overuse, which we think of as fatigue.
Management Of Occlusion & TMD – Okeson
Myospasm
• Involuntary cns-induced tonic muscle contraction often associated with local
metabolic conditions within the muscle tissues.
• Following conditions are etiologic factors in myospasm:
1. Continued deep pain input
2. Local metabolic factors within the muscle tissues associated with fatigue or
overuse
3. Idiopathic myospasm mechanisms
Management Of Occlusion & TMD – Okeson
• The following clinical characteristics are associated with myospasms:
1. Structural dysfunction: There is marked restriction in range of mandibular
movement according to the muscle or muscles involved. Acute malocclusion is common.
2. There is pain at rest.
3. The pain is increased with function.
4. The affected muscle is firm and painful to palpation.
5. There is a generalized feeling of significant muscle tightness
Management Of Occlusion & TMD – Okeson
Management Of Occlusion & TMD – Okeson
Centrally mediated myalgia:
• Chronic, continuous muscle pain disorder originating predominantly from CNS
effects that are felt peripherally in the muscle tissues.
• This disorder presents clinically with symptoms similar to those of an inflammatory
condition of the muscle tissue; therefore it is sometimes referred to as myositis.
Management Of Occlusion & TMD – Okeson
Etiology:
• As the CNS becomes exposed to prolonged nociceptive input, brainstem pathways
can functionally change.
• This can result in an antidromic effect on afferent peripheral neurons.
• When this occurs, the afferent neurons in the periphery can release nociceptive
neurotransmitters such as substance p and bradykinin, which in turn cause
nociception and pain in the peripheral tissues. this process is called neurogenic
inflammation.
Management Of Occlusion & TMD – Okeson
Symptoms:
1. Structural dysfunction: Patients experiencing centrally mediated myalgia present with a
significant decrease in the velocity and range of mandibular movement.
2. There is significant pain at rest.
3. The pain is increased with function.
4. There is a generalized feeling of muscle tightness.
5. There is significant pain to muscle palpation.
6. As chronic centrally mediated myalgia becomes protracted, the lack of use of the muscle
owing to pain may induce muscle atrophy and/or myostatic or myofibrotic contracture.
Management Of Occlusion & TMD – Okeson
Myofascial Pain Dysfunction Syndrome:
When muscle spasm develops ,dysfunction as well as pain occurs and the condition
usually is designated as MPDS.
It is initiated as spasm of one or more masticatory muscle.
Regional myogenous pain condition characterized by local areas of firm,
hypersensitive bands of muscle tissue known as trigger points.
• Textbook of oral medicine- Anil Ghom (Second edition)
• Etiology:
1.Abnormal occlusion
2. Prosthetic problems
3.Orthodontic problems
4.Emotional stress
5. Dental restoration
6. Joint problems
7.Oral habits
Textbook of oral medicine- Anil Ghom (Second edition)
Pathogenesis:
Energy released
during muscle
contraction
Accumulation of
lactic acid
Change in pH
Muscle receptor
prone to impulse
excitation
Infusion of
histamine,bradykinin
Pathological
muscular
derangement
Textbook of oral medicine- Anil Ghom (Second edition)
• Treatment:
1.Educate the patient
2.Muscle relaxation techniques
3. Pharmacotherapy
4. Biofeedback
5. Bruxism prosthesis
6. Anesthesia
7.Physical medication
Textbook of oral medicine- Anil Ghom (Second edition)
Treatment component Description
1.Education • Explanation of the diagnosis and treatment
• Reassurance about the generally good prognosis for recovery and natural
course
• Explanation of patient’s and doctor’s roles in therapy
• Information to enable patient to perform self-care
2.Self care • Eliminate oral habits (eg, tooth clenching, chewing gum)
• Provide information on jaw care associated with daily activities
3. Physical therapy • Education regarding biomechanics of jaw, neck, and head posture
• Passive modalities (heat and cold therapy, ultrasound, laser, TENS)
• Range of motion exercises (active and passive)
• Posture therapy
• Passive stretching, general exercise and conditioning program.
4.Intra oral appliance therapy • Cover all the teeth on the arch the appliance is seated on
• Adjust to achieve simultaneous contact against opposing teeth
• Adjust to a stable comfortable mandibular posture
• Avoid changing mandibular position
• Avoid long-term continuous use.
5.Pharmacotherapy NSAIDs, acetaminophen, muscle relaxants, antianxiety agents, tricyclic
antidepressants
6. Behavioral/relaxation techniques Relaxation therapy, Hypnosis ,Biofeedback ,Cognitive-behavioral therapy
Conclusion
Nature has blessed us with marvelously
dyamic masticatory system allowing us to
function and therefore exist.
References:
• Glossary of prosthodontic terms-9
• Human anatomy,volume 3 – B.D chaurasia
• Human embryology – Inderbir Singh
• Management of occlusion & Tmd – Okeson
• Burket’s oral medicine –Eleventh edition
• Textbook of oral medicine- Anil Ghom (Second edition)
• De Rossi, Scott S. et al. “Disorders of masticatory muscles”. Dental clinics ,Volume 57(3), 449-
464.
• Dhanrajani PJ, Jonaidel O. “Trismus: aetiology, differential diagnosis and treatment”. Dent update
2002;29:88-94

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Muscles of mastication

  • 1.
  • 2. INDEX: 1. Introduction 2. Types of muscle 3. Development of muscle 4. Primary muscles of mastication 5. Suprahyoid muscles 6. Infrahyoid muscles 7. Masticatory system disorders 8. Conclusion 9. References
  • 3. NOTHING IS MORE FUNDAMENTAL TO TREATING PATIENTS THAN KNOWING THE ANATOMY - JEFFERY P OKESON
  • 4. Muscle: A tissue composed of contractile cells or fibers that effect movement of an organ or part of the body.(GPT 9) Mastication: The process of chewing food for swallowing and digestion.(GPT 9) There are basically three types of muscles: 1. Skeletal muscle 2. Smooth muscle 3. Cardiac muscle INTRODUCTION Glossary of prosthodontic terms-9
  • 5. 1.Have light and dark bands or striations. 2.Cylindrical multinucleated unbranched cells 3.Voluntary 4.Contract quickly but get fatigued easily 5.Seen in limbs,tongue,body,face, neck 1.Do not show light and dark bands. 2.Spindle shaped uninucleated cells 3.Involuntary 4.Contract slowly but don’t get fatigued easily 5.Seen in ureters,bronchi of lungs, ailmentary canal and blood vessels. 1.Structure in between striated and non striated muscle 2.Cylinder shaped uninucleated branched cells. 3.Involuntary 4.Contract rhythmically and quickly ,hence do not get fatigued 5. Seen in walls of heart
  • 8. FUNCTIONAL CLASSIFICATION: Muscles of mastication Elevators 1.Masseter 2.Temporalis 3.Medial pterygoid 4.Upper head of lateral pterygoid Depressors 1.Digastric 2.Mylohyoid 3.Geniohyoid 4.Lower head of lateral pterygoid
  • 9. Masseter • Thick quadrilateral muscle • Second most efficient masticatory muscle • Multipennate arrangement • Buccal pad of fat seperates the muscle from buccinator. HUMAN ANATOMY VOL 3- B.D. CHAURASIA
  • 10.
  • 11. ORIGIN: • By a thick tendinous aponeurosis from the zygomatic process of maxilla • From the anterior 2/3rds of the lower border of zygomatic arch. Superficial head • Posterior third of the lower border of zygomatic arch. • Whole of the medial surface of the zygomatic arch Deep head HUMAN ANATOMY VOL 3- B.D. CHAURASIA
  • 12. INSERTION: Superficial head • Middle and lower part of lateral surface of ramus of mandible Deep head • Upper part of ramus and coronoid process of mandible HUMAN ANATOMY VOL 3- B.D. CHAURASIA
  • 13. Actions: Elevates the mandible HUMAN ANATOMY VOL 3- B.D. CHAURASIA
  • 15. Nerve Supply: Massetric nerve HUMAN ANATOMY VOL 3- B.D. CHAURASIA
  • 17. Temporalis: • Largest among all the mastication muscles. • Broad ,radiating and fan shaped • Fills the temporal fossa
  • 18. HUMAN ANATOMY VOL 3- B.D. CHAURASIA
  • 19. Actions • Anterior Vertical Fibers - Elevates the mandible - Side to side grinding movements • Posterior Horizontal Fibers - Retracts the mandible HUMAN ANATOMY VOL 3- B.D. CHAURASIA
  • 20. Vascular Supply • Supplied by deep temporal branches from second part of maxillary artery. HUMAN ANATOMY VOL 3- B.D. CHAURASIA
  • 21. Nerve Supply Deep temporal branches of anterior division of mandibular nerve. HUMAN ANATOMY VOL 3- B.D. CHAURASIA
  • 22. Examination: Anterior region Middle region Posterior region BUREKET’S ORAL MEDICINE-ELEVENTH EDITION
  • 23. Lateral Pterygoid • Also called as external pterygoid muscle. • Short and conical • Has upper and lower heads
  • 25. INSERTION: • Fibers Run Backward And Laterally And Converge To Be Inserted Into : -Pterygoid Fovea On The Anterior Surface Of Neck Of Mandible -Anterior And Medial Border Of The Articular Disc And Capsule Of TMJ. HUMAN ANATOMY VOL 3- B.D. CHAURASIA
  • 27. • Vascular supply: Pterygoid branches from the maxillary artery. The ascending palatine branch of the facial artery. • Nerve supply: Branch from anterior division of mandibular nerve. HUMAN ANATOMY VOL 3- B.D. CHAURASIA
  • 28. Examination: Functional manipulation of Superior lateral pterygoid : Contraction – if it is source of pain, clenching increases pain. If a tongue blade is placed between posterior teeth bilaterally and patient clenches on the separator pain increases with contraction of SLP Burket’s Oral Medicine- 11th Edition
  • 29. Functional manipulation of inferior lateral pterygoid : Contraction – Ask the patient to protrude against resistance provided by the examiner. If ILP is the source of pain – activity will increase pain Burket’s Oral Medicine -11th Edition
  • 30. Medial Pterygoid • Quadrilateral muscle • Has a small superficial and large deep head • Deepest among the muscles of mastication • Also known as internal pterygoid muscle
  • 33. • Vascular supply: Pterygoid branch of second part of maxillary artery. • Innervation: Nerve to medial pterygoid ,branch of main trunk of mandibular nerve. HUMAN ANATOMY VOL 3- B.D. CHAURASIA
  • 34. Examination: • Palpation is done mainly by intra oral approach. • The region of its attachment to inner aspect of angle of mandible can be palpated by sliding finger lingually and applying pressure at the insertion of muscle and placing one hand extraorally below the border of mandible
  • 35. • Functional Manipulation: 1. Contraction: 2.Stretching: If it is source of pain Clenching teeth together wil increase pain If it is source of pain Opening mouth wil increase pain
  • 37. Suprahyoid Muscles 1. Digastric 2. Geniohyoid 3. Mylohyoid 4. Stylohyoid
  • 39. Innervation • Anterior belly: Nerve to mylohyoid • Posterior belly: Facial nerve Actions • Depresses the mandible • Elevates hyoid bone HUMAN ANATOMY VOL 3- B.D. CHAURASIA
  • 41. Innervation First cervical nerve through hypoglossal nerve Actions Elevates hyoid bone When hyoid bone is fixed, depresses mandible HUMAN ANATOMY VOL 3- B.D. CHAURASIA
  • 43. • Innervation: Nerve to mylohyoid,branch of inferior alveolar nerve • Vascular supply: By facial and lingual artery • This muscle separates sublingual and submandibular salivary glands
  • 44. Actions: Actions Elevates floor of mouth Elevation of hyoid bone Depression of mandible
  • 46. •Innervation Facial nerve •Actions Elevation of hyoid Pulls the hyoid backwards HUMAN ANATOMY VOL 3- B.D. CHAURASIA
  • 47. INFRAHYOID MUSCLES: Infrahyoid group of muscles consist of: 1. Omohyoid 2. Thyrohyoid 3. Sternohyoid 4. Sternothyroid
  • 48. Omohyoid: Origin: Upper border of scapula near suprascapular notch Insertion: Lower border of body of hyoid bone Innervation: Superior belly by superior root of ansa cervicalis Inferior belly by ansa cervicalis Action: Depresses the hyoid bone HUMAN ANATOMY VOL 3- B.D. CHAURASIA
  • 49. Sternothyroid Origin: Posterior surface of manubrium sterni,adjoining parts of first costal cartilage Insertion: Oblique line of thyroid cartilage Innervation: Ansa cervicalis Actions: Depresses the larynx and helps in swallowing HUMAN ANATOMY VOL 3- B.D. CHAURASIA
  • 50. Thyrohyoid Origin: Oblique line of thyroid cartilage Insertion: Body of hyoid bone Innervation: First cervical nerve through hypoglossal nerve Actions: Depresses the hyoid bone and elevates the larnyx HUMAN ANATOMY VOL 3- B.D. CHAURASIA
  • 51. Sternohyoid Origin: Posterior surface of manubrium sterni and adjoining part of clavicle Insertion: Medial part of lower border of hyoid bone Innervation: Ansa cervicalis Actions: Depresses the hyoid bone HUMAN ANATOMY VOL 3- B.D. CHAURASIA
  • 53. • Muscle tissue becomes compromised or unhealthy – - due to muscle overuse - physical trauma - over stretching - receiving a blow to muscle tissue itself. • Muscles of mastication become compromised through increased activity.
  • 54. • Some of the common masticatory disorders are as follows: 1.Trismus 2.Protective co contraction 3. Local muscle soreness 4.Myospasm 5. Myofascial pain 6.Centrally mediated myalgia(chronic myositis) 7.Myofibrotic contracture
  • 55. Trismus: What is trismus? Trismus is inability to open the jaw Etiology: Trauma, infection, drugs, dental treatment, congenital problems Management: Heat therapy Analgesics Physiotherapy Soft diet Muscle relaxants Dhanrajani PJ, Jonaidel O. Trismus: aetiology, differential diagnosis and treatment. Dent update 2002;29:88-94
  • 56. Protective Co Contraction: • Protective co-contraction is the initial response of a muscle to altered sensory or proprioceptive input or injury (or threat of injury). • This response has also been called protective muscle splinting. • When protective co contraction occurs ,CNS increases activity of antagonistic muscle during contraction of agonist muscle • For example,patient who is experiencing protective co contraction wil demonstrate slightly increased muscle activity in the elevators durimg mouth opening . • Not a pathological condition but a normal guarding or protective mechanism Management Of Occlusion & TMD – Okeson
  • 57. Etiology 1. Altered sensory or proprioreceptory input. 2.Constant deep pain input 3.Increased emotional stress Symptoms 1.Structural dysfunction 2.No pain at rest 3.Increased pain with functuon 4.Feeling of muscle weakness Treatment 1.No indication to treat the muscle condition itself. Treatment should instead be directed toward the reason for the co- contraction Management Of Occlusion & TMD – Okeson
  • 58. Local muscle soreness: • Local muscle soreness is a primary noninflammatory myogenous pain disorder. • It is often the first response of the muscle tissue to continued protective co- contraction and therefore the most common acute muscle pain disorder in dental practice. • Although co-contraction represents a cns-induced muscle response, local muscle soreness represents a change in the local environment of the muscle tissues. • It represents the initial response to overuse, which we think of as fatigue. Management Of Occlusion & TMD – Okeson
  • 59. Myospasm • Involuntary cns-induced tonic muscle contraction often associated with local metabolic conditions within the muscle tissues. • Following conditions are etiologic factors in myospasm: 1. Continued deep pain input 2. Local metabolic factors within the muscle tissues associated with fatigue or overuse 3. Idiopathic myospasm mechanisms Management Of Occlusion & TMD – Okeson
  • 60. • The following clinical characteristics are associated with myospasms: 1. Structural dysfunction: There is marked restriction in range of mandibular movement according to the muscle or muscles involved. Acute malocclusion is common. 2. There is pain at rest. 3. The pain is increased with function. 4. The affected muscle is firm and painful to palpation. 5. There is a generalized feeling of significant muscle tightness Management Of Occlusion & TMD – Okeson
  • 61. Management Of Occlusion & TMD – Okeson
  • 62. Centrally mediated myalgia: • Chronic, continuous muscle pain disorder originating predominantly from CNS effects that are felt peripherally in the muscle tissues. • This disorder presents clinically with symptoms similar to those of an inflammatory condition of the muscle tissue; therefore it is sometimes referred to as myositis. Management Of Occlusion & TMD – Okeson
  • 63. Etiology: • As the CNS becomes exposed to prolonged nociceptive input, brainstem pathways can functionally change. • This can result in an antidromic effect on afferent peripheral neurons. • When this occurs, the afferent neurons in the periphery can release nociceptive neurotransmitters such as substance p and bradykinin, which in turn cause nociception and pain in the peripheral tissues. this process is called neurogenic inflammation. Management Of Occlusion & TMD – Okeson
  • 64. Symptoms: 1. Structural dysfunction: Patients experiencing centrally mediated myalgia present with a significant decrease in the velocity and range of mandibular movement. 2. There is significant pain at rest. 3. The pain is increased with function. 4. There is a generalized feeling of muscle tightness. 5. There is significant pain to muscle palpation. 6. As chronic centrally mediated myalgia becomes protracted, the lack of use of the muscle owing to pain may induce muscle atrophy and/or myostatic or myofibrotic contracture. Management Of Occlusion & TMD – Okeson
  • 65. Myofascial Pain Dysfunction Syndrome: When muscle spasm develops ,dysfunction as well as pain occurs and the condition usually is designated as MPDS. It is initiated as spasm of one or more masticatory muscle. Regional myogenous pain condition characterized by local areas of firm, hypersensitive bands of muscle tissue known as trigger points. • Textbook of oral medicine- Anil Ghom (Second edition)
  • 66. • Etiology: 1.Abnormal occlusion 2. Prosthetic problems 3.Orthodontic problems 4.Emotional stress 5. Dental restoration 6. Joint problems 7.Oral habits Textbook of oral medicine- Anil Ghom (Second edition)
  • 67. Pathogenesis: Energy released during muscle contraction Accumulation of lactic acid Change in pH Muscle receptor prone to impulse excitation Infusion of histamine,bradykinin Pathological muscular derangement Textbook of oral medicine- Anil Ghom (Second edition)
  • 68. • Treatment: 1.Educate the patient 2.Muscle relaxation techniques 3. Pharmacotherapy 4. Biofeedback 5. Bruxism prosthesis 6. Anesthesia 7.Physical medication Textbook of oral medicine- Anil Ghom (Second edition)
  • 69. Treatment component Description 1.Education • Explanation of the diagnosis and treatment • Reassurance about the generally good prognosis for recovery and natural course • Explanation of patient’s and doctor’s roles in therapy • Information to enable patient to perform self-care 2.Self care • Eliminate oral habits (eg, tooth clenching, chewing gum) • Provide information on jaw care associated with daily activities 3. Physical therapy • Education regarding biomechanics of jaw, neck, and head posture • Passive modalities (heat and cold therapy, ultrasound, laser, TENS) • Range of motion exercises (active and passive) • Posture therapy • Passive stretching, general exercise and conditioning program. 4.Intra oral appliance therapy • Cover all the teeth on the arch the appliance is seated on • Adjust to achieve simultaneous contact against opposing teeth • Adjust to a stable comfortable mandibular posture • Avoid changing mandibular position • Avoid long-term continuous use. 5.Pharmacotherapy NSAIDs, acetaminophen, muscle relaxants, antianxiety agents, tricyclic antidepressants 6. Behavioral/relaxation techniques Relaxation therapy, Hypnosis ,Biofeedback ,Cognitive-behavioral therapy
  • 70. Conclusion Nature has blessed us with marvelously dyamic masticatory system allowing us to function and therefore exist.
  • 71. References: • Glossary of prosthodontic terms-9 • Human anatomy,volume 3 – B.D chaurasia • Human embryology – Inderbir Singh • Management of occlusion & Tmd – Okeson • Burket’s oral medicine –Eleventh edition • Textbook of oral medicine- Anil Ghom (Second edition) • De Rossi, Scott S. et al. “Disorders of masticatory muscles”. Dental clinics ,Volume 57(3), 449- 464. • Dhanrajani PJ, Jonaidel O. “Trismus: aetiology, differential diagnosis and treatment”. Dent update 2002;29:88-94

Editor's Notes

  1. All muscles develop from mesoderm except muscles of iris Nerve supply
  2. Helps occlusion in mastication: its superficial fibres, running obliquely help to protract the mandible and acting alternately, the 2 muscles i.e(right & left) swing the chin from one side to the other producing a grinding movement of the teeth which is assisted by the pterygoid muscles.
  3. Massetric artery: branch of second part of maxillary artery Facial artery: branch of eca Transerve facial artery: branch of superficial temporal artery
  4. A branch of the madibular nerve which enters its deep surface by passing through the mandibular notch immediately anterior to the capsule of the TMJ.
  5. The patient is asked to clench the teeth and using both hands practitioner palpates the masseter muscle on both sides extra orally. Palpate the origin of masseter bilaterally along the zygomatic arch and continue to paplate down the body of mandible where masseter is attached
  6. The fibres emerge in a afan like fashion ,anterior fibres run vertically ,middle obliquely and posterior horizontally and converge & descend into a tendon that passes between the zygomatic arch & cranial wall & is then attached to the medial surface, apex, anterior and posterior borders of the coronoid process & anterior border  of the mandibular ramus almost to the third molar tooth. Also few fibres are attached to articular disc.
  7. Each of the three areas of temporal muscles are palpated independently. Anterior Region Palpated above zygomatic arch and anterior to TMJ. Middle Region is palpated directly above the TMJ and superior to zygomatic arch. Posterior Region palpated above and behind the ear. If uncertainty arises – ask the patient to clench teeth together. Temporalis contracts and fibers are felt beneath the fingertips. Fibers run in an oblique direction across the lateral aspect of skull.
  8. Upper head:infra temporal surface and crest of greater wing of sphenioid . Lower head: main and larger. -arises from lateral surface of lateral pterygoid plate. Lower head:
  9. The primary function of the lateral pterygoid muscle is to pull the head of the condyle out of the mandibular fossa along the articular eminence to protrude the mandible. The lateral pterygoid is the only muscle of mastication that assists in depressing the mandible (opening the jaw). Side to side movement is carried out by alternate contraction of medial and lateral pterygoid. Different actions have been ascribed to the 2 parts of the lateral pterygoid, the superior head being involved in chewing and the inferior head in protrusion..  When both the sides of lateral and medial pterygoids act together protrudes the mandible. When medial and lateral pterygoid muscles of two sides contract alternately produces side to side movements of mandible.
  10. Muscle that is compromised by excess activity is painful both during contraction and stretching . In order to differentiate between the pain arising from elevator muscle patient is asked to open jaw wide. This will stretch the elevator muscle but not SLP. If opening elicits no pain If pain during opening Pain of clenching is Both SLP and elevators from SLP may be involved
  11. When the inferior lateral pterygoid contracts,the mandible is protruded. Functional manipulstion is best achieved by having patient make a protrusive movement since this muscle is primary protruding muscle Stretching – ILP stretches when teeth are in maximum intercuspation. If it is the source of pain ,when teeth are clenched, pain increases. When a tongue blade is placed between posterior teeth, ICP cannot be reached, thus ILP does not stretch. Biting on a separator does not increase pain but may decrease it.
  12. Superficial head: from tuberosity of maxilla and adjoining bone Deep head: from medial surface of lateral pterygoid plate and adjoining process of palatine bone Fibres run nbackward ,downward and laterally Insertion: atmedial surface of angle of mandible below and behind mandibukar foramen.
  13. Elevates the mandible with bilateral contraction Acting with lateral pterygoid it protrudes the mandible.The insertion of muscle is posterior to origin hence it protrudes the mandible When medial and lateral pterygoids contract together corresponding sides of mandible is rotated forwards & to the opposite side. Alternating activity in left and right sides of muscles produces side to side movements
  14. Stretching on opening mouth wide
  15. Origin: digastric fossa of mandible Insertion: mastoid notch of temporal bone
  16. Double innervation …due to different origin Depresses mamndible when mouth is opened widely or against resistance.sec to lateral pterygoid
  17. Short and narrow suprahyoid muscle Origin:mental spine i.e genial tubercle Runs backward and downward. And inserts at anterior surface of body of hyoid bone.
  18. As the geniohyoid muscle contracts it pulls the hyoid bone and with it the attached larynx upwards and forwards during swallowing. It pulls the elevated hyoid bone directly forwards, hence increases the anterior posterior diameter of the pharynx to receive the bolus in swallowing. Conversely when the hyoid bone is fixed the geniohyoid muscles acts synergistically with the mylohyoid muscles to depress the mandible and pull it inwards thus opening the mouth.
  19. It is paired muscle. The mylohoid muscle forms, anatomically & functionally the floor of the oral cavity, hence the old term for it was oral diaphragm. The right & left muscles are united in the mid line between the mandible & hyoid bone by a tendinous slip, the mylohyoid raphe.  Origin and Insertion      The muscle arises from the myohyoid line on the inner surface of the mandible. Posterior fibres take their origin from the region of the alveolus of the lower third molar. The origin of the anterior fibres descend towards the lower border of the mandible. The posterior fibres of the coarsely bundled muscle run steeply downward, medially, & slightly forward & are attached to the body of the hyoid bone; the majority of the fibres, join those of the contralateral muscle in the mylohyoid raphe. Innervated by nerve to mylohyoid
  20. Originates before it enters inferior alveolar canal
  21. Small muscle lies on upper border of posterior digastric Origin: Posterior surface of styloid process and inserts at junction of body and greater cornu of hyoid bone.
  22. Thus helps in keeping the mouth open
  23. It has inferiot brlly ,a commn tendon and a superior belly .it arises from inferior belly and is inserted thru superior belly. The central tendon lies on internal jugular vein at level of cricioid cartilage and is bound to clavicle by fascial pulley
  24. Origin: and sternoclavicular ligament Only muscle which is not directly attached to hyoid bone
  25. Continuation of sternothyroid Elevates the larynx when the hyoid is fixed
  26. Following elevation duting swallowing and vocal movements
  27. Etiology: tmd,tumors, Trauma: Fracture mandible * Fracture zygomatic arch * Incorporation of foreign bodies Infection: Heat therapy consists of placing moist hot towels on the affected area for 15–20 minutes every hour. Aspirin is usually adequate in managing the pain associated with trismus; its antiinflammatory properties are also beneficial. A narcotic analgesic may be required if the discomfort is more intense. If necessary, diazepam (2.5–5 mg three times daily) or other benzodiazepine may be prescribed for muscle relaxation.3 When the acute phase is over the patient should be advised to initiate physiotherapy for opening and closing the jaws and to perform lateral excursions of the mandible for 5 minutes every 3–4 hours.35 Sugarless chewing gum is another means of providing lateral movement of the TMJ. . If pain and dysfunction continue unabated beyond 48 hours, the possibility of infection should be considered. Antibiotics should be added to the treatment regimen and continued for 7 days
  28. The key to identifying protective co-contraction is that it immediately follows an event; therefore the history is very important. Protective co-contraction remains for only a few days. If it is not resolved, local muscle soreness is likely to follow. The history will reveal one of the following: 1. A recent alteration in local structures 2. A recent source of constant deep pain 3. A recent increase in emotional stress
  29. he patient reports a sudden onset of restricted jaw movement usually accompanied by muscle tightness
  30. Myospasms are best treated by reducing the pain and then passively lengthening or stretching the involved muscle. Reduction of the pain can be achieved by manual massage (Figure 12-8), vapocoolant spray, ice, or even an injection of local anesthetic into the muscle in spasm. Once the pain is reduced, the muscle is passively stretched to full length. If an injection is used (and often it is the most effective manner to stop a persistent spasm), 2% lidocaine without a vasoconstrictor is recommended. 2. When obvious etiologic factors are present (e.g., deep pain input), attempts to eliminate these factors should be made so as to lessen the likelihood of recurrent myospasms. When the myospasms are secondary to fatigue and overuse (e.g., due to prolonged exercise), the patient is advised to rest the muscle or muscles and reestablish normal electrolyte balance. n occasion myospasms can occur repeatedly with no identifiable etiologic factors. When this occurs in the same muscle, the condition may actually represent an oromandibular dystonia. Dystonias are repeated uncontrolled spastic contractions of muscles; these conditions are often thought to have central etiologies
  31. in other words, neurons that normally carry information only from the periphery into the cns can now be reversed to carry information from the cns out to the peripheral tissues. this is likely to occur through the axonal transport system.61
  32. The patient reports a constant primary myogenous pain condition usually associated with a prolonged history of muscle complaints (for months and even years).