Infection of oral mucosa
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The document discusses various viral infections that can affect the oral mucosa, including herpes simplex virus, varicella zoster virus, coxsackievirus (which causes herpangina), enterovirus (hand-foot-and-mouth disease), measles, and human immunodeficiency virus (HIV). It provides details on the presentation, transmission, clinical features, treatment, and progression of each viral infection. Kaposi's sarcoma, a cancer associated with HIV/AIDS, is also mentioned.
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Viral Infections of Oral Mucosa
This document discusses several viral infections that can affect the oral mucosa. It describes the human herpes virus family, including herpes simplex virus types 1 and 2, which can cause gingivostomatitis and recurrent oral herpes infections. It also discusses herpes zoster virus, which causes chickenpox and shingles. Epstein-Barr virus is noted for causing infectious mononucleosis and oral hairy leukoplakia. Cytomegalovirus, coxsackie virus, paramyxoviruses like mumps and measles viruses are also summarized for their oral manifestations. Treatment options are provided for many of these viral infections.
Oral viral infections
- Herpes simplex virus types 1 and 2 are common oral viral infections, usually transmitted via saliva or direct contact. Both viruses establish latency in ganglia. HSV-1 typically causes oral lesions while HSV-2 typically causes genital lesions.
- Varicella zoster virus causes chickenpox in children and shingles in adults via reactivation. It establishes latency in ganglia.
- Epstein-Barr virus causes infectious mononucleosis, commonly known as "mono" or "kissing disease". It is transmitted through saliva and causes fever, lymphadenopathy and pharyngitis.
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Vesiculobullous Lesions -Dr.Aseem Mohammed
This document discusses vesiculobullous lesions, which are elevated fluid-filled blisters on the skin or mucosa that are either vesicles (less than 1cm) or bullae (greater than 1cm). It covers the classification, etiology, pathogenesis, clinical features, diagnosis and management of various conditions that cause vesiculobullous lesions, including herpes simplex virus infections, varicella zoster virus infections, herpangina, hand foot and mouth disease, and more. Key points include how these viruses cause primary infection and can later reactivate to cause characteristic lesions, as well as how their oral manifestations, complications, and treatments are described.
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This Presentation will help ou to compare the spectrum of pathologies in ulcerated versus non-ulcerated exophytic oral mucosal lesions and explore the significance of surface ulceration as an indication of various oral diseases
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The document summarizes information about herpes simplex viruses types 1 and 2 (HSV-1 and HSV-2), including that they cause a variety of illnesses depending on site of infection, host immunity, and whether it is a primary or recurrent infection. Primary infections tend to be more severe while recurrent infections are generally milder. HSV establishes latent infections in sensory ganglia. Infections range from mild, self-limiting oral-facial infections to potentially life-threatening infections in immunocompromised individuals or newborns. Antiviral medications can shorten symptoms of recurrent infections but do not cure the infection.
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Viral Infections of Oral Mucosa
This document discusses several viral infections that can affect the oral mucosa. It describes the human herpes virus family, including herpes simplex virus types 1 and 2, which can cause gingivostomatitis and recurrent oral herpes infections. It also discusses herpes zoster virus, which causes chickenpox and shingles. Epstein-Barr virus is noted for causing infectious mononucleosis and oral hairy leukoplakia. Cytomegalovirus, coxsackie virus, paramyxoviruses like mumps and measles viruses are also summarized for their oral manifestations. Treatment options are provided for many of these viral infections.
Oral viral infections
- Herpes simplex virus types 1 and 2 are common oral viral infections, usually transmitted via saliva or direct contact. Both viruses establish latency in ganglia. HSV-1 typically causes oral lesions while HSV-2 typically causes genital lesions.
- Varicella zoster virus causes chickenpox in children and shingles in adults via reactivation. It establishes latency in ganglia.
- Epstein-Barr virus causes infectious mononucleosis, commonly known as "mono" or "kissing disease". It is transmitted through saliva and causes fever, lymphadenopathy and pharyngitis.
Viral infections of oral cavity - Dr. Abhishek Solanki
This document discusses various viral infections including herpes simplex virus, varicella, herpes zoster, infectious mononucleosis, cytomegalovirus, enteroviruses, rubeola, rubella, mumps, and human immunodeficiency virus. It provides details on the causative viruses, clinical manifestations, histopathological features, diagnosis and treatment of each infection. Complications are also mentioned for some viruses. Classification systems for HIV infected patients based on CD4 count and clinical categories are summarized.
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This document discusses vesiculobullous lesions, which are elevated fluid-filled blisters that can be either vesicles (less than 1cm) or bullae (greater than 1cm). It covers the classification, causes, clinical presentations, diagnosis, and management of various conditions that cause these lesions, including herpes simplex virus, varicella zoster virus, coxsackie virus infections like herpangina and hand foot and mouth disease. Specific oral manifestations of each condition are described. Diagnosis involves viral culture, PCR or antigen detection from lesion samples. Management focuses on pain control, antiviral medications, and prevention of complications or dissemination.
Vesiculobullous Lesions -Dr.Aseem Mohammed
This document discusses vesiculobullous lesions, which are elevated fluid-filled blisters on the skin or mucosa that are either vesicles (less than 1cm) or bullae (greater than 1cm). It covers the classification, etiology, pathogenesis, clinical features, diagnosis and management of various conditions that cause vesiculobullous lesions, including herpes simplex virus infections, varicella zoster virus infections, herpangina, hand foot and mouth disease, and more. Key points include how these viruses cause primary infection and can later reactivate to cause characteristic lesions, as well as how their oral manifestations, complications, and treatments are described.
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This Presentation will help ou to compare the spectrum of pathologies in ulcerated versus non-ulcerated exophytic oral mucosal lesions and explore the significance of surface ulceration as an indication of various oral diseases
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The document summarizes information about herpes simplex viruses types 1 and 2 (HSV-1 and HSV-2), including that they cause a variety of illnesses depending on site of infection, host immunity, and whether it is a primary or recurrent infection. Primary infections tend to be more severe while recurrent infections are generally milder. HSV establishes latent infections in sensory ganglia. Infections range from mild, self-limiting oral-facial infections to potentially life-threatening infections in immunocompromised individuals or newborns. Antiviral medications can shorten symptoms of recurrent infections but do not cure the infection.
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This document provides an overview of ulcerative and infective vesicullobular lesions, including their classification, etiology, clinical manifestations, and management. It discusses viral lesions caused by herpes simplex virus, varicella zoster virus, and cytomegalovirus. Fungal infections from histoplasmosis, blastomycosis, and mucormycosis are also reviewed. Bacterial infections from tuberculosis are mentioned. For each condition, the document outlines etiology, pathogenesis, clinical features, investigations, and management. Differential diagnoses are provided for comparison of conditions.
Bacterial infections of oral cavity
Bacterial infections can cause diseases that manifest in the oral cavity. Some diseases like scarlet fever are caused by specific bacteria, while others can be caused by a broad group of microorganisms. Common bacterial infections discussed in the document include scarlet fever caused by Streptococcus pyogenes, diphtheria caused by Corynebacterium diphtheriae, and tuberculosis caused by Mycobacterium tuberculosis. These bacteria can cause lesions, ulcers, and pseudomembranes in the oral cavity. Diagnosis involves identifying the bacteria through cultures or identifying their characteristics through microscopic examination after staining.
Herpetic skin infections
Herpes simplex and herpes zoster are caused by human herpes viruses that cause primary infection followed by lifelong latency in neuronal or lymphoid tissues. Herpes simplex virus types 1 and 2 cause herpes infections including oral and genital lesions. Varicella zoster virus causes chickenpox primary infection and shingles upon reactivation. These viruses are treated with antiviral medications such as acyclovir to reduce symptoms and transmission risk, especially in at-risk groups.
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This document discusses several vesiculo-bullous diseases including those caused by viral infections and those associated with immunologic defects. It provides details on the pathogenesis, clinical features, histopathology, immunopathology, differential diagnosis and treatment of various conditions such as herpes simplex virus infection, varicella-zoster infection, hand foot and mouth disease, herpangina, measles, pemphigus vulgaris, pemphigus vegetans, cicatricial pemphigoid, bullous pemphigoid, dermatitis herpetiformis, and linear IgA disease. The document contains in-depth information on the etiology, characteristics and management of these vesiculo
O.med الجزء الثانيpdf.pdf
This document discusses cytomegalovirus (CMV) infection. It notes that CMV transmission occurs through direct contact with infected white blood cells or blood products. In organ transplant recipients, CMV can be transmitted through the donor organ. CMV infection is usually asymptomatic but can cause mononucleosis-like symptoms. It establishes latency after initial exposure. CMV infection most commonly involves the eyes, gastrointestinal tract, and mucocutaneous sites in immunocompromised people like those with AIDS. Treatment involves antiviral medications like ganciclovir or valganciclovir.
Oral ulcersppt
This document discusses oral ulcers caused by various infectious and non-infectious conditions. It describes the clinical features and management of several specific conditions that can cause oral ulcers, including herpes simplex virus infections (primary and recurrent), varicella-zoster virus infections (chickenpox, herpes zoster), hand-foot-and-mouth disease, herpangina, tuberculosis, and syphilis. For each condition, it covers the presentation of oral ulcers, pathogenesis, diagnosis, and treatment approaches.
Lect.1.mouth cavity
The document discusses various ulcerative lesions that can occur in the oral cavity. It describes acute lesions including those caused by trauma, recurrent aphthous stomatitis, and herpesvirus infections. It then discusses recurrent aphthous stomatitis in more detail, describing the three subtypes. It also covers chronic lesions caused by factors like infection, neoplasms, and nectrotizing sialometaplasia. Fungal infections like candidiasis are discussed. It concludes by mentioning lesions seen in HIV/AIDS patients such as Kaposi's sarcoma and hairy leukoplakia.
Herpes simplex in oral cavity
1) Herpes simplex virus type 1 and 2 are common viruses that cause infections both above and below the waist. They establish latent infections in sensory ganglia.
2) Primary HSV infections typically cause oral lesions and gingivostomatitis with symptoms like fever and pain. Recurrent infections can be triggered by factors like stress and menstruation, causing lesions like recurrent herpes labialis.
3) HSV infections in immunocompromised patients can cause large, painful ulcers over nonkeratinized sites that may disseminate without treatment. Diagnosis involves virus culture, PCR, or biopsy while management focuses on antiviral therapy.
Infective stomatitis
This document discusses infective stomatitis, primarily focusing on viral causes. It defines stomatitis and infective stomatitis. It discusses laboratory diagnosis of viral infections and lists three main methods. It then lists several viruses associated with infective stomatitis and discusses primary herpetic gingivostomatitis caused by HSV-1 in more detail, covering clinical features, histopathology, diagnosis, differential diagnosis, and treatment. It also briefly discusses herpes labialis, herpes zoster of the trigeminal nerve, and viral transmission.
Herpes simplex virus
to download this presentation from this link
https://mohmmed-ink.blogspot.com/2020/11/herpes-simplex-virus.html
Herpes simplex virus
1. Vesiculobullous lesions.pdf by dr jamal
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Inflammation milind
This document discusses various types of gingivitis and inflammation that can occur in children. It defines inflammation and lists its causes. The four classic signs of inflammation are redness, swelling, heat, and pain, with a fifth sign of loss of function added later. Acute inflammation comes on rapidly and is short-lived, while chronic inflammation has a gradual onset and longer duration. Specific types of gingivitis and inflammation discussed in children include eruption gingivitis, dental plaque-induced gingivitis, herpes simplex virus infection, acute necrotizing ulcerative gingivitis, acute candidiasis, chronic nonspecific gingivitis, and ascorbic acid deficiency ging
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5.GINGIVAL_INFECTIONS.ppt dental patholo
The normal periodontium consists of the investing and supporting tissues of the tooth, including the gingiva, periodontal ligament, alveolar bone, and cementum. It is divided into the gingiva, whose main function is protection of the underlying tissues, and the attachment apparatus, composed of the periodontal ligament, cementum, and alveolar bone. Periodontitis is an inflammatory disease of the supporting tissues caused by specific microorganisms, resulting in progressive destruction of the periodontal ligament and alveolar bone.
Primary oral ulcerative lesions new
This document discusses various oral ulcerative diseases including traumatic ulcers, aphthous stomatitis, Behcet's disease, viral infections, and bacterial infections. It provides details on the characteristics, causes, and clinical features of minor, major, and herpetiform aphthous ulcers, which are the most common oral mucosal lesions. Treatment options are also summarized for recurring aphthous ulcers, which primarily involve using mouthwashes to relieve pain and promote healing.
Acute gingival infections and desquamative gingivitis
This document provides an overview of various acute gingival infections and desquamative gingivitis conditions. It discusses necrotizing ulcerative gingivitis in depth, covering its historical background, clinical features, histopathology, relation to bacteria, diagnosis, differential diagnosis, etiology, and management. It also summarizes primary herpetic gingivostomatitis, pericoronitis, and desquamative gingivitis, outlining their key clinical features, diagnoses, and treatment approaches for each condition. The document serves as a reference for dental students to understand these oral infections and inflammatory conditions.
269890879-Hand-Foot-and-Mouth-Disease.pptx
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Primary herpetic gingivostomatitis
This document describes primary herpes simplex infection, commonly known as cold sores. It is usually caused by HSV-1 and presents with fever, headache, malaise and painful sores in the mouth. Lesions start as vesicles that rupture, leaving shallow ulcers that heal within 10-14 days. Diagnosis is made through clinical examination, with viral culture and biopsy used for confirmation. Treatment focuses on pain relief and short term use of antivirals or steroids to reduce symptoms.
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Infection of oral mucosa
- 1. INFECTIONS OF THE ORAL MUCOSA VIRAL INFECTION BACTERIAL INFECTION FUNGAL INFECTION
- 3. Herpes simplex virus(HSV) A. Primary herpetic gingivostomatitis (type I H.S.V) In a person without circulating antibodies. It’s developed in children and young adult, rarely in children under 6 months and mainly transmitted by saliva, incubation period is 5 days.
- 4. In the oral cavity is characterized by multiple vesicular eruptions located in the all attached oral mucosa, gingival and movable mucosa, chiefly lips. Buccal mucosa, pharynx and tonsils may be involved .
- 8. Histologically intraepithelial vesicles formations with necrotic exudates and neutrophil infiltrate accompanied by epithelial cells showing nuclear ballooning degeneration.
- 11. Treatment supported care consist of the prescribing a soft diet, analgesic to reduce pain and fever with antibiotic to prevent secondary infection, soothing mouth wash to wet the lesion.
- 12. B. Secondary herpes simplex infection: 1.Recurrent (Herpes Labialis) Occurs mostly on the firmly attached mucosa such as hard palate and attached gingiva and not movable mucosa. Heals in 10 days and no treatment is applied, with reassurance of patient. 2. Herpetic whitlow infection: is viral dermatitis of finger and also called terminal pulp infection.
- 16. Varicella-zoster infection • Primary Varicella zoster(chicken pox) It’s an acute infectious viral disease the incubation period last for 2-5 weeks, appears as vesicles with an erythematous area on boundary and are extremely pruritic, fever and mild generalized lymphadenopathy are also present. Resolved within 4-8 days.
- 17. • Secondary varicella zoster infection along the peripheral nervous system commonly from dorsal root ganglia. It starts as painful vesicular eruption on skin and also may affect oral cavity when the trigeminal ganglia is infected with varicella. Patient gets malaise, fever and enlarged lymph node at area and runs in a course of about 2 weeks.
- 21. Herpangina Viral disease caused by “coxsackie group A virus” there will be fever and ill-feeling followed by vesicle rupture, lesion occurs mainly in soft palate and tonsilar area. The disease run a course of (7-10) days.
- 22. Hand-foot &mouth disease is a highly contagious viral infection The virus is transferred from one individual to another through airborne spread or fecal-oral contamination.
- 23. Clinical Features: about 90% affects children younger than 5 years of age. After a short incubation period, the condition resolves spontaneously in 1 to 2 weeks. Signs and symptoms are usually mild to moderate in intensity and include low-grade fever, malaise, lymphadenopathy, and sore mouth. Pain from oral lesions is often the patient's chief complaint.
- 24. Measles a highly contagious viral infection caused by a member of the paramyxovirus family. Typically, oral eruptions consist of early pinpoint elevations over the soft palate that combines with ultimate involvement of the pharynx with bright erythema. German measles share some clinical features with measles, such as fever, respiratory symptoms
- 25. Clinical Featurs: After an incubation period of 7 to 10 days, prodromal symptoms of fever, malaise, conjunctivitis, photophobia, and cough develop. In 1 to 2 days small erythematous macules with white necrotic centers appear in the buccal mucosa, these lesion spots, known as Koplik's spots. precede the skin rash by 1 to 2 days.
- 26. HUMAN IMMUNODEFICIENCY VIRUS INFECTION (HIV) is transmitted by exchange of blood or body fluids principally through sexual contact. The injection of blood or from mother to child. Transmission of the virus may be followed by infection which is detected by the appearance of HIV antibodies in the blood.
- 27. This generally occurs within 3 months of exposure. A few patients have an acute HIV infection at this time, the clinical features of which include pyrexia, skin rash, headache, diarrhea, sore throat, and erythema of the buccal and palatal mucosa.
- 28. EXPOSURE TO INFECTION INFECTION NO INFECTION
- 29. INFECTION HIV SEROPOSITIVE asymptomatic ACUTE HIV INFECTION Pyrexia,skin rash,headache ,diarrhoea,sore throat &erythema of buccal&palatal mucosa
- 30. HIV SEROPOSITIVE asymptomatic PGL ARC l.Adenopathy p.pyrexia Diarr.,weight loss Fatique &malaise AIDS
- 33. Kaposi sarcoma
- 34. Kaposi Sarcoma. Classic Kaposi sarcoma in an older man presenting as multiple purple macules and plaques on the lower leg.