Cerebral palsy (CP) is a disorder that affects muscle tone, movement, and motor skills (the ability to move in a coordinated and purposeful way). CP usually is caused by brain damage that happens before or during a baby's birth, or during the first 3 to 5 years of a child's life.
Cerebral palsy (CP) is a disorder that affects muscle tone, movement, and motor skills (the ability to move in a coordinated and purposeful way). CP usually is caused by brain damage that happens before or during a baby's birth, or during the first 3 to 5 years of a child's life.
Meaning of Cerebral Palsy , Definition of Cerebral Palsy , Areas affected by Cerebral Palsy , Causes of Cerebral Palsy , Types of Cerebral Palsy ( many basis ) , Signs and Symptoms of Cerebral Palsy , Developmental Milestones of Cerebral Palsy child , Associated problems of Cerebral Palsy , Treatment of Cerebral Palsy ,
CP
Non-specific term that include disorders characterized by early onset and impaired movement and posture.
Non-progressive and may include perceptual problems, language deficits, and intellectual involvement.
Incidence
Most common physical disability of childhood.
Incidence has increased since the 60’s, maybe due to improved survival of VLBW infants.
Etiology
Variety of perinatal, prenatal, and postnatal factors contribute, either singly or multifactorily to CP.
Commonly thought to be due to birth asphyxia; now known to be due to existing prenatal brain abnormalities.
Premature delivery is the single most important determinant of CP.
In 24% of cases, no cause is found.
Spina bifida/ dysraphism - assessment and physiotherapy management Susan Jose
refrences kessler tecklin darcy.
a all round description of assesment in physiotherapeutic methods and management techniques.
participationn increasing measures and limitation reduction stratergies
Evaluation of an infant with hypotonia is described including history, examination and investigations. Clinical algorithm for such evaluation is presented.
Meaning of Cerebral Palsy , Definition of Cerebral Palsy , Areas affected by Cerebral Palsy , Causes of Cerebral Palsy , Types of Cerebral Palsy ( many basis ) , Signs and Symptoms of Cerebral Palsy , Developmental Milestones of Cerebral Palsy child , Associated problems of Cerebral Palsy , Treatment of Cerebral Palsy ,
CP
Non-specific term that include disorders characterized by early onset and impaired movement and posture.
Non-progressive and may include perceptual problems, language deficits, and intellectual involvement.
Incidence
Most common physical disability of childhood.
Incidence has increased since the 60’s, maybe due to improved survival of VLBW infants.
Etiology
Variety of perinatal, prenatal, and postnatal factors contribute, either singly or multifactorily to CP.
Commonly thought to be due to birth asphyxia; now known to be due to existing prenatal brain abnormalities.
Premature delivery is the single most important determinant of CP.
In 24% of cases, no cause is found.
Spina bifida/ dysraphism - assessment and physiotherapy management Susan Jose
refrences kessler tecklin darcy.
a all round description of assesment in physiotherapeutic methods and management techniques.
participationn increasing measures and limitation reduction stratergies
Evaluation of an infant with hypotonia is described including history, examination and investigations. Clinical algorithm for such evaluation is presented.
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2. • In 1860s, known as “cerebral palsy” or “little’s
disease” after an English surgeon William John Little
wrote the 1st medical description..
• Cerebral word is derived from Latin cerebrum : affected
part of brain and palsy from Greek para – beyond , lysis
– loosening i.e. lack of muscle control.
3. • Cerebral palsy is a motor function disorder :-
• Caused by permanent, non-progressive brain lesion.
• It can be present at birth or shortly thereafter.
• It is incurable, lifelong condition, damage doesn’t worsens and it may
be congenital or acquired.
• It is a heterogenous group of movement disorders . C.P. is an umbrella
term not a single diagnosis.
• In C.P. muscles are unaffected, brain is unable to send the appropriate
signals necessary to instruct muscles when to contract and relax.
4.
5.
6. • CAUSES :-
• 1. An insult or injury to the brain –
• Fixed, static lesion(s), in single or multiple areas of the motor centers of the brain.
• Early in CNS deviation
• 2. Developmental malformations –
• The brain fails to develop correctly.
• 3. Neurological damage –
• Can occur before , during or after delivery.
• Rh incompatibility, illness, severe lack of oxygen etc.
• 4. Severe deprivation of oxygen or blood flow to the brain –
• HIE (hypoxic-ischemic encephalopathy or intrapartal asphyxia)
7.
8. • RISK FACTORS :-
• 1. Prenatal factors –
• Hemorrhage/bleeding –
• Abruptio placetna
• Infections –
• Rubella, cytomegalovirus, toxoplasmosis
• Environmental factors
• Maternal characteristics -
• Age
• Multiple births
• History of fetal deaths/ miscarriages
• Cigarette smoking >30 sticks per day
• Alcoholism and drug addiction
• Social status, mother with MR
• Mother’s medical condition.
9. • 2.Perinatal factors –
• High or low BP
• Umbilical cord coil
• Breech delivery
• Oversedation of drugs
• Trauma i.e. forceps or vacuum delivery
• Complications of birth
• 3.Postnatal causes –
• Trauma, head injury
• Infections
• Hypoxia
• Stroke in young age
• Tumor, cyst
10. • TYPES OF CEREBRAL PALSY –
• According to :
• 1.Neurological deficits
• 2.Types of movement involved
• 3.Areas of affected limbs
• 1.According to Neurological deficits –
• Based on the – extent and area of brain damage
• Each type involves the way a person moves.
• 3 main types are :-
• Pyramidal
• Originates from motor areas of the cerebral cortex
• Spastic CP
13. • SPASTIC CP :-
• Increased muscle tone, tense and contracted muscles.
• Have stiff jerky or awkward movements.
• Limbs are usually underdeveloped,
• Increased deep tendon reflexes.
• Most common from 70-80 % of all affected children.
• Types of spastic CP –
• According to affected limbs – plegia or paresis – meaning paralyzed or weak.
• Paraplegia
• Diplegia
• Hemiplegia
• Quadriplegia
• Monoplegia – one limb, extremely rare
• Triplegia – three limbs extremely rare.
14. • Diplegia / paraplegia –
• Both legs with slight involvement elsewhere both legs. May also have contracture of hips and knees and
talipes equinovarus ( clubfoot).
•
• Hemiplegia –
• Limbs only on one side affected,
• Hip and knee contractures
• Quadriplegia –
• Upper body usually normal or with very minor signs
• Child may develop contractures of ankles and feet.
• When the child walks his arms, head and even his mouth may twist strangely.
• Children with all 4 limbs affected often have such severe brain damage that they never are able to walk.
• Legs and feet turned inward.
15.
16. • Spastic quadriplegia –
• Characteristic “scissors” position of limbs due to adductor spasm.
• Dyskinesia –
• Dyskinetic movement of mouth
• Grimacing, drooling and dysarthria.
• Adductor spasm.
• Ataxic CP –
• Poor balance and lack of coordination.
• Wide based gait, depth perception usually affected
• Tendency to fall and stumble.
• Inability to walk straight line.
• Least common 5-10% of cases
17.
18.
19. • Mixed CP –
• A common combination is spastic and athethoid
• Spastic muscle tone and involuntary movements.
• 25% 0f CP cases, fairly common.
• Degree of severity –
• 1.Mild CP -20% of cases
• Not require help for assisting their impaired ambulation capacity.
• 2.Moderate CP -50%
• Requires self help for assisting their impaired ambulation capacity.
• 3.Severe CP –
• Totally incapacitated and bedridden and they always need care from others.
20. • Sign and symptoms –
• Posture or balance problem’
• Loss of control or coordination
• Abnormal tone
• Abnormal strength
• Abnormal reflexes
• Persistent motor delay
• Cognitive deficit
• Associated handicaps.
21.
22. • Early signs –
• Infancy (0-3 months ) –
• Stiff or floppy posture
• Excessive lethargy or irritability
• High pitched cry
• Poor head control
• Weak suck/ tongue thrust / tonic bite/ feeding difficulties
• Late infancy –
• Inability to perform motor skills as indicated :
• Control hand grasp by 3 months
• Rolling over by 5 months
• Independent sitting by 7 months
23. • Abnormal developmental patterns :-
• Hand preference by 12 months
• Excessive arching of back
• Log rolling
• Abnormal or prolonged parachute response.
• Abnormal developmental patterns after 1 year of age :-
• W Sitting – knees flexed, legs extremely rotated.
• Bottom shuffling – scoots along the floor
• Walking on tip toe or hopping
• Behavioural symptoms –
• Poor ability to concentrate
• Unusual tenseness, irritability.
24. • CP Main problem:-
• Mentation and thought processes are not always affected; child is trapped in their body with their disabilities ,
• Ability to express their intelligence may be limited by difficulty In communicating.
• Associated problems –
• Hearing and visual problems
• Sensory integration problems
• FTT (failure to thrive)
• Feeding problems
• Behavioural / emotional difficulties
• Communication disorders
• Bladder and bowel control problem
• GERD
• Skeletal deformities
• Mental retardation and learning disabilities in some
• Seizures/ epilepsy
25. • Diagnosis :-
• A useful diagnosis is when the specific type, affected limb , severity, and
cause , if known are identified.
• Physical evaluation, interview
• MRI, CT scan EEG
• Laboratory and radiologic work up
• Assessment tools
• i.e. Peabody development motor skills, Denver test 2 etc.
26. • the Peabody developmental motor scales :=
• in depth assessment 6 subtests include:
• Reflexes
• Stationary
• Locomotion
• Object manipulation
• Grasping
• Visual-motor integration
• This subtest yields a
• gross motor quotient,
• a fine motor quotient
• and a total motor quotient
• ages covered : from birth through five years of age.
27. • Denver test 2 :-
• Developmental screening test
• Covers 4 general functions –
• Personal social (smiling)
• Fine motor adaptive (eg. Grasping and drawing)
• Language (eg. Combining words)
• Gross motor (eg. Walking)
• Ages covered from birth to 6 years.
• Denver test 2 :-
• 1. subjective (interview)
• a. history taking :- includes all that may predispose an infant to brain damage or CP
• Risk factors
• Psychosocial factors
• Family adaptation
28. • b. child’s health history :- often admitted to hospital for corrective
surgeries and other complications.
• Respiratory status
• Motor functions
• Presence of fever
• Feeding and weight loss
• Any changes in physical state
• Medical regimen.
29. • 2. Objective :-
• Physical examination
• Criteria ---
• P osturing/ poor muscle control and strength
• O ropharyngeal problems
• S trabismus/ squint
• T one (hyper/ hypotonia)
• E volutional maldeveloment
• R eflexes (e.g. increased deep tendon )
• Abnormalities 4/6 strongly point to CP
30.
31. • Oropharyngeal problems –
• Speech, swallowing, breathing, drooling and poor feeding.
• Appearance of strabismus (impaired side to side movement )
• Evolutional maldevelopment –
• Delay in motor skills , such as rolling over, sitting , crawling and walking.
• Size for age
• Persistence of primitive reflexes or parachute reflex fail to develop
• Asymmetric tonic neck reflex “fencing position” – head to one side, arm and leg on that side extended ,
opposite limbs flexed.
• Test for ataxia – hold a toy or finger in front of the child and ask him to touch it on the first try. The child
with ataxia cannot do it.
• Increased or exaggerated deep tendon reflex
32.
33. • Treatment :-
• No treatment to cure CP.
• Brain damage cannot be corrected.
• Crucial for children with CP:
• Early identification
• Multidisciplinary care and
• Support
• 1. Nonphysical therapy
• A. General management
• - Proper nutrition and personal care
• B. Pharmacologic
• Botox, intrathecal, baclofen
• - Controls muscle spasm and seizures
• - Delivered directly to the spinal fluid to avoid brain effects.
• Glycopyrrolate – control drooling
• Pamidronate – may help with osteoporosis.
34. • C. Surgery :-
• - To loosen joints,
• - Relieve muscle tightness
• - Straightening of different twists or unusual curvatures of leg muscles
• - Improves the ability to sit , stand and walk.
• Selective posterior rhizotomy :-
• Is used to improve spasticity (muscle stiffness) in cerebral palsy. In some cases
nerves need to be served to decrease muscle tension of inappropriate contraction.
• Intensive rehabilitation is required after the surgery , usually up to 6 weeks, followed
by physical therapy on an ongoing basis.
35. • D. Physical aids :-
• Orthosis, braces and splints
• - Keeps limbs In correct alignment
• - Prevent deformities
• Positioning devices
• - Enable better postures
• Walkers, special scooters, wheel chairs
• - Makes it easier to move about.
• E. Special education
• - To meet the child’s special needs.
• - Improve learning
• - Vocational training can help prepare young adults for jobs.
36. • F.Rehabilitation services –
• Speech and occupational therapies may improve the ability to speak, and perform activities of daily living and to do some
suitable works to have their own income.
• G. Family services –
• Professional support helps a patient and family cope with cerebral palsy.
• Counsellors help parents learn how to modify behaviours.
• Caring for a child with CP can be very stressful .
• Some families find support groups help.
• H. Other treatments –
• Therapeutic electrical stimulation ,
• Acupuncture
• Hyperbaric therapy
• Massage therapy might help.
37. • 2. Physical therapy
• The ultimate long term goal is realistic independence. To get there we have to have some
short term goals . those being a working communication system, education to his potential,
computer skills and , above all, friends.
• A. Sitting
• - Vertical head control and control of head and trunk.
• B. Standing and walking
• - Establish an equal distribution of weight on each foot, train to use steps or inclines.
• C. Prone development
• D. Supine development
• - Head control on supine and positions.