Basic Life Support
What is CPR
• Cardiopulmonary resuscitation (CPR) is a
series of life saving actions that improve the
chance of survival following cardiac arrest.
• The optimal approach to CPR vary with
rescuer, victim, and available resources
• The fundamental challenge however is to
achieve early and effective CPR.
Chain of survival
Rescuer and Victim
Rescuer – all, regardless of training, should provide
chest compression.
a)Untrained Lay Rescuer–Hands Only CPR
b)Trained Lay Rescuer – Chest compression ± rescue
breaths
c)Health Care Provider– Chest compression + rescue
breaths , coordinate teamwork. Coordinate
teamwork. Likely cause ?
Victim –
Adult : cardiac cause - chest compression
Child/ Drowning : asphyxial cause - ventilation
• CPR can be performed by
ANYONE who knows how to do it,
ANYWHERE,
IMMEDIATELY,
WITHOUT any EQUIPMENT
Basic Life Support (BLS)
1. Ensure scene safety
2. Check for response
3. Activate EMS (Call for help)
4. Check breathing and pulse
5. Chest compressions
6. Open airway and ventilate
7. Defibrillate (when AED arrives)
Steps of BLS
1.Ensure scene safety
• Of Self
• Of Patient
2.Check Responsiveness
• Lay rescuer-
Victim unconscious / unresponsive, with
absent / abnormal breathing
Assume cardiac arrest and begin COCPR
• HCP-
Victim unconscious / unresponsive, with
absent / abnormal breathing check pulse for
no more than 10s - no definite pulse - Assume
cardiac arrest
3.Activate Emergency Medical Services
• Shout for nearby help
• Phone or ask someone to phone EMS
• Phone/caller with the phone remains at
victim’s side- phone on speaker mode
Chest Compressions
• Effectively performed by laying the
patient supine on a relatively hard
surface, allow effective
compression of the sternum
• Select a point that is over the
lower half of sternum
• Place the heel of one hand
• Place the heel of another hand on
top
• Interlace the fingers
• Rescuer’s buttocks above those of
the patient, elbows locked,
shoulders at 90 degrees
• Count 1,2, 3… 30
High Quality CPR!
• Push hard and push fast
• Lower half of sternum, centre of chest, between
the nipples
• Rate : 100 -120 / min
• Depth : atleast 2 inches (5 cm)
• Allow complete chest recoil between compression
• Do not lean on victim
• Minimal interruptions
Correct Technique
• Firm surface
• Kneel beside victimʼs
chest or stand beside bed
• Heel of one hand on
intermammary line
• Heel of other hand on
top
• Lock elbows
Opening the Airway-head tilt chin
lift
• Trained lay rescuer-
confident in
compressions and
ventilation –open
the airway using a
head tilt– chin lift-
when no cervical
spine injury
suspected
HCP – should use
head tilt–chin lift
maneuver to open
the airway of a
patient when no
cervical spine
injury suspected.
Head tilt- chin lift
Jaw thrust
•In trauma patients
•By trained rescuer only
•Use head tilt- chin lift, if jaw
thrust does not adequately
open the airway
Breathing
• Give breaths over 1 sec
• Visible chest rise
• Compression: ventilation ratio - 30 : 2 ( in 18
seconds )
• If no chest rise , re-position head , ensure tight
seal
• Avoid forceful or rapid breaths
• Advanced airway : 10 breaths/min.
Rescue breaths
• Open airway as described
• Deliver 1 tidal breath over 1
second
• Allow the chest to recoil for 1
second
• Deliver the second tidal breath
over another second
• Allow the chest to recoil over
another second
• Do not hyperventilate
Mouth to mouth Mouth to mask
Bag & Mask
IF VICTIM STARTS TO BREATHE NORMALLY
PLACE IN RECOVERY POSITION
Defibrillation Vs CPR first?
• Witnessed arrest, AED available at site -
OHCA - immed CPR , Defib asap !
IHCA - immed CPR , Defib asap !
• AED not available at site - CPR continued till
AED arrives.
Resuscitate until
• Arrival of qualified help arrives
• Victim starts breathing normally
• Rescuer becomes exhausted
• Scene becomes unsafe
• Physician directs to stop CPR
IHCA BLS & ACLS
2020 American Heart Association
Chains of Survival for IHCA
BLS & ACLS Survey in In-Hospital
Cardiac Arrest
• Healthcare providers (HCPs) must be able to
competently and quickly assist victims who are in
cardiac arrest.
• The aim is to:
1. improve survival rates
2. ensure positive, high-quality outcomes.
• Systematic strategies should be formulated that
allow HCPs to provide immediate care using BLS
and ACLS surveys.
• If a patient is unresponsive the BLS survey
should be utilized at first.
• After the completion of the BLS survey or
when the victim is responsive and awake, the
ACLS survey is the next step that entails
providing advanced treatment approaches.
The BLS Survey
• HCPs must be able to demonstrate both one-
and two-responder resuscitation skills.
• Emphasis should be placed on performing
several actions simultaneously during the
resuscitation process as two responders are
often available.
ACLS Survey
• After completing the BLS survey, or if the
victim is responsive, conscious, or awake, the
responder should begin the ACLS survey.
• Focus needs to be placed on identifying and
treating the underlying cause of the victim’s
problem.
Drug therapy
• Epinephrine IV/IO dose:
- 1mg every 3-5 minutes,
- Early administration improves survival in non-
shockable rhythms
• Amiodarone IV/IO dose:
- First dose: 300 mg bolus
- Second dose: 150 mg
- Recommended for refractory VF/VT
Or
• Lidocaine IV/IO dose:
- First dose: 1-1.5 mg/kg
- Second dose: 0.5-0.75 mg/kg
- Recommended for refractory VF/VT
Reversible causes of cardiac arrest
5H&5T ACC/AHA guidelines
recommendation
• Reversible causes of cardiac arrest
5H 5T
Hypothermia Tension Pneumothorax
Hypovolemia Cardiac tamponade
Hypoxia Toxins
Hydrogen ion (Acidosis) Thrombosis,
pulmonary
Hypo/ hyperkalemia Thrombosis , cardiac
Anaphylaxis
• Removal of the inciting cause
• Call for help
• Intramuscular (IM) injection of epinephrine at the
earliest, followed by additional epinephrine by IM or
intravenous (IV) injection as needed.
• Place patient in supine position with the lower
extremities elevated
• Supplemental oxygen
• Two wide bore iv lines for volume resuscitation with IV
fluids (1-2 lt NS)
• The recommended dose of epinephrine in
anaphylaxis is 0.2 to 0.5 mg (1:1000)
intramuscularly, to be repeated every 5 to 15 min as
needed
• In patients with anaphylactic shock, close
hemodynamic monitoring is recommended.
• When an IV line is in place, it is reasonable to
consider the IV route for epinephrine in anaphylactic
shock, at a dose of infusion at 0.1 mcg/kg/minute
• IV infusion of epinephrine is a reasonable
alternative to IV boluses for treatment of
anaphylaxis in patients not in cardiac arrest.
• There is development of oropharyngeal or
laryngeal edema, immediate referral to a health
professional with expertise in advanced airway
placement, including surgical airway
management, is recommended.
• Adjunct drugs
– H1 antihistamines Eg Cetirizine Diphenhydramine
– H2 antihistamines Eg famotidine
– Bronchodilators Eg theophylline
– Glucocorticoids
Toxicity: Local Anesthetics
Central Nervous System Toxicity
Early Stage
• Lightheadedness and dizziness
• Visual disturbances
• Disorientation
• Drowsiness
Late stage
• Shivering muscular twitching & tremors
• Generalized tonic clonic convulsions
• If sufficiently large or rapid dose is administered
intravenously, the initial signs of CNS excitation
are rapidly followed by state of generalized CNS
depression.
• Respiratory depression and later on respiratory
arrest.
Cardiovascular Toxicity
• Direct action on both the heart and the
peripheral blood vessels.
• Indirect action on the circulation by blockade
of sympathetic and parasympathetic efferent
activity.
• Initial phase- hypertension and tachycardia
• Intermediate phase- myocardial depression
and hypotension.
• Terminal phase- peripheral vasodilatation,
severe hypotension, and variety of
arrhythmias.
Bupivacaine induced cardiotoxicity
• It depresses the rapid phase of depolarization
in purkinje fibers & ventricular muscle more
than lignocaine.
• The rate of recovery from a use-dependent
block is slower in bupivacaine treated
papillary muscles than in lignocaine treated
muscles.
• The ratio of dose required for irreversible
cardiovascular collapse and the dose that will
produce CNS toxicity is lower for bupivacaine than
lignocaine.
• Ventricular arryrthmias can occur more often
after the rapid iv administration of a large dose of
bupivacaine but far less frequent with lignocaine.
• Cardiac resuscitation is more difficult after
bupivacaine induced CV collapse
Mx for LA toxicity
• STOP injecting local anaesthetic
• Get help
• Initial focus
– Airway Mx- ventilate with 100% O2
– Control Seizures- BZD preferred
– Avoid propofol sp in hemodynamically unstable pt
Mx of cardiac arrythmias
• Basic and advanced cardiac life support
• Avoid vasopresin, CCB, beta blockers & LA
• Reduce individual epinephrine dose to less
than 1mcg/Kg
• Lipid emulsion
Diamox (Acetazolamide)
Contraindications Associated With Acetazolamide
– Poor LFT or Renal Impairment
– Hypokalemia
– Hyperchloremic acidosis
– Sulfonamide allergy
– patients using anti-folate medications such as Mtx
and Trimethoprim
Symptoms of sulfa allergy
– skin rash or hives
– itchy eyes and skin
– congestion
– swelling of the mouth and throat
– Breathing difficulty
– serious complications, including anaphylaxis and
Stevens-Johnson syndrome.
Management of toxicity
– no particular antidote for Acetazolamide, treatment
is supportive
– Maintain iv line
– Antihistaminics (Pheniramine)
– Corticosteroids (Hydrocortisone)
– Bronchodilator (Theophylline)
No diagnostic tests for sulfa allergy -- just
thorough history
THANK YOU!

Basic life support presentation for doctors

  • 1.
  • 2.
    What is CPR •Cardiopulmonary resuscitation (CPR) is a series of life saving actions that improve the chance of survival following cardiac arrest. • The optimal approach to CPR vary with rescuer, victim, and available resources • The fundamental challenge however is to achieve early and effective CPR.
  • 3.
  • 4.
    Rescuer and Victim Rescuer– all, regardless of training, should provide chest compression. a)Untrained Lay Rescuer–Hands Only CPR b)Trained Lay Rescuer – Chest compression ± rescue breaths c)Health Care Provider– Chest compression + rescue breaths , coordinate teamwork. Coordinate teamwork. Likely cause ? Victim – Adult : cardiac cause - chest compression Child/ Drowning : asphyxial cause - ventilation
  • 5.
    • CPR canbe performed by ANYONE who knows how to do it, ANYWHERE, IMMEDIATELY, WITHOUT any EQUIPMENT
  • 6.
    Basic Life Support(BLS) 1. Ensure scene safety 2. Check for response 3. Activate EMS (Call for help) 4. Check breathing and pulse 5. Chest compressions 6. Open airway and ventilate 7. Defibrillate (when AED arrives)
  • 7.
  • 10.
    1.Ensure scene safety •Of Self • Of Patient
  • 11.
    2.Check Responsiveness • Layrescuer- Victim unconscious / unresponsive, with absent / abnormal breathing Assume cardiac arrest and begin COCPR • HCP- Victim unconscious / unresponsive, with absent / abnormal breathing check pulse for no more than 10s - no definite pulse - Assume cardiac arrest
  • 12.
    3.Activate Emergency MedicalServices • Shout for nearby help • Phone or ask someone to phone EMS • Phone/caller with the phone remains at victim’s side- phone on speaker mode
  • 13.
    Chest Compressions • Effectivelyperformed by laying the patient supine on a relatively hard surface, allow effective compression of the sternum • Select a point that is over the lower half of sternum • Place the heel of one hand • Place the heel of another hand on top • Interlace the fingers • Rescuer’s buttocks above those of the patient, elbows locked, shoulders at 90 degrees • Count 1,2, 3… 30
  • 14.
    High Quality CPR! •Push hard and push fast • Lower half of sternum, centre of chest, between the nipples • Rate : 100 -120 / min • Depth : atleast 2 inches (5 cm) • Allow complete chest recoil between compression • Do not lean on victim • Minimal interruptions
  • 15.
    Correct Technique • Firmsurface • Kneel beside victimʼs chest or stand beside bed • Heel of one hand on intermammary line • Heel of other hand on top • Lock elbows
  • 16.
    Opening the Airway-headtilt chin lift • Trained lay rescuer- confident in compressions and ventilation –open the airway using a head tilt– chin lift- when no cervical spine injury suspected HCP – should use head tilt–chin lift maneuver to open the airway of a patient when no cervical spine injury suspected.
  • 17.
  • 18.
    Jaw thrust •In traumapatients •By trained rescuer only •Use head tilt- chin lift, if jaw thrust does not adequately open the airway
  • 19.
    Breathing • Give breathsover 1 sec • Visible chest rise • Compression: ventilation ratio - 30 : 2 ( in 18 seconds ) • If no chest rise , re-position head , ensure tight seal • Avoid forceful or rapid breaths • Advanced airway : 10 breaths/min.
  • 20.
    Rescue breaths • Openairway as described • Deliver 1 tidal breath over 1 second • Allow the chest to recoil for 1 second • Deliver the second tidal breath over another second • Allow the chest to recoil over another second • Do not hyperventilate
  • 21.
    Mouth to mouthMouth to mask Bag & Mask
  • 22.
    IF VICTIM STARTSTO BREATHE NORMALLY PLACE IN RECOVERY POSITION
  • 24.
    Defibrillation Vs CPRfirst? • Witnessed arrest, AED available at site - OHCA - immed CPR , Defib asap ! IHCA - immed CPR , Defib asap ! • AED not available at site - CPR continued till AED arrives.
  • 25.
    Resuscitate until • Arrivalof qualified help arrives • Victim starts breathing normally • Rescuer becomes exhausted • Scene becomes unsafe • Physician directs to stop CPR
  • 26.
  • 27.
    2020 American HeartAssociation Chains of Survival for IHCA
  • 28.
    BLS & ACLSSurvey in In-Hospital Cardiac Arrest • Healthcare providers (HCPs) must be able to competently and quickly assist victims who are in cardiac arrest. • The aim is to: 1. improve survival rates 2. ensure positive, high-quality outcomes. • Systematic strategies should be formulated that allow HCPs to provide immediate care using BLS and ACLS surveys.
  • 29.
    • If apatient is unresponsive the BLS survey should be utilized at first. • After the completion of the BLS survey or when the victim is responsive and awake, the ACLS survey is the next step that entails providing advanced treatment approaches.
  • 30.
    The BLS Survey •HCPs must be able to demonstrate both one- and two-responder resuscitation skills. • Emphasis should be placed on performing several actions simultaneously during the resuscitation process as two responders are often available.
  • 32.
    ACLS Survey • Aftercompleting the BLS survey, or if the victim is responsive, conscious, or awake, the responder should begin the ACLS survey. • Focus needs to be placed on identifying and treating the underlying cause of the victim’s problem.
  • 34.
    Drug therapy • EpinephrineIV/IO dose: - 1mg every 3-5 minutes, - Early administration improves survival in non- shockable rhythms • Amiodarone IV/IO dose: - First dose: 300 mg bolus - Second dose: 150 mg - Recommended for refractory VF/VT
  • 35.
    Or • Lidocaine IV/IOdose: - First dose: 1-1.5 mg/kg - Second dose: 0.5-0.75 mg/kg - Recommended for refractory VF/VT
  • 36.
    Reversible causes ofcardiac arrest 5H&5T ACC/AHA guidelines recommendation
  • 37.
    • Reversible causesof cardiac arrest 5H 5T Hypothermia Tension Pneumothorax Hypovolemia Cardiac tamponade Hypoxia Toxins Hydrogen ion (Acidosis) Thrombosis, pulmonary Hypo/ hyperkalemia Thrombosis , cardiac
  • 38.
    Anaphylaxis • Removal ofthe inciting cause • Call for help • Intramuscular (IM) injection of epinephrine at the earliest, followed by additional epinephrine by IM or intravenous (IV) injection as needed. • Place patient in supine position with the lower extremities elevated • Supplemental oxygen • Two wide bore iv lines for volume resuscitation with IV fluids (1-2 lt NS)
  • 39.
    • The recommendeddose of epinephrine in anaphylaxis is 0.2 to 0.5 mg (1:1000) intramuscularly, to be repeated every 5 to 15 min as needed • In patients with anaphylactic shock, close hemodynamic monitoring is recommended. • When an IV line is in place, it is reasonable to consider the IV route for epinephrine in anaphylactic shock, at a dose of infusion at 0.1 mcg/kg/minute
  • 40.
    • IV infusionof epinephrine is a reasonable alternative to IV boluses for treatment of anaphylaxis in patients not in cardiac arrest. • There is development of oropharyngeal or laryngeal edema, immediate referral to a health professional with expertise in advanced airway placement, including surgical airway management, is recommended.
  • 41.
    • Adjunct drugs –H1 antihistamines Eg Cetirizine Diphenhydramine – H2 antihistamines Eg famotidine – Bronchodilators Eg theophylline – Glucocorticoids
  • 42.
    Toxicity: Local Anesthetics CentralNervous System Toxicity Early Stage • Lightheadedness and dizziness • Visual disturbances • Disorientation • Drowsiness
  • 43.
    Late stage • Shiveringmuscular twitching & tremors • Generalized tonic clonic convulsions • If sufficiently large or rapid dose is administered intravenously, the initial signs of CNS excitation are rapidly followed by state of generalized CNS depression. • Respiratory depression and later on respiratory arrest.
  • 44.
    Cardiovascular Toxicity • Directaction on both the heart and the peripheral blood vessels. • Indirect action on the circulation by blockade of sympathetic and parasympathetic efferent activity.
  • 45.
    • Initial phase-hypertension and tachycardia • Intermediate phase- myocardial depression and hypotension. • Terminal phase- peripheral vasodilatation, severe hypotension, and variety of arrhythmias.
  • 46.
    Bupivacaine induced cardiotoxicity •It depresses the rapid phase of depolarization in purkinje fibers & ventricular muscle more than lignocaine. • The rate of recovery from a use-dependent block is slower in bupivacaine treated papillary muscles than in lignocaine treated muscles.
  • 47.
    • The ratioof dose required for irreversible cardiovascular collapse and the dose that will produce CNS toxicity is lower for bupivacaine than lignocaine. • Ventricular arryrthmias can occur more often after the rapid iv administration of a large dose of bupivacaine but far less frequent with lignocaine. • Cardiac resuscitation is more difficult after bupivacaine induced CV collapse
  • 48.
    Mx for LAtoxicity • STOP injecting local anaesthetic • Get help • Initial focus – Airway Mx- ventilate with 100% O2 – Control Seizures- BZD preferred – Avoid propofol sp in hemodynamically unstable pt
  • 49.
    Mx of cardiacarrythmias • Basic and advanced cardiac life support • Avoid vasopresin, CCB, beta blockers & LA • Reduce individual epinephrine dose to less than 1mcg/Kg
  • 50.
  • 51.
    Diamox (Acetazolamide) Contraindications AssociatedWith Acetazolamide – Poor LFT or Renal Impairment – Hypokalemia – Hyperchloremic acidosis – Sulfonamide allergy – patients using anti-folate medications such as Mtx and Trimethoprim
  • 52.
    Symptoms of sulfaallergy – skin rash or hives – itchy eyes and skin – congestion – swelling of the mouth and throat – Breathing difficulty – serious complications, including anaphylaxis and Stevens-Johnson syndrome.
  • 53.
    Management of toxicity –no particular antidote for Acetazolamide, treatment is supportive – Maintain iv line – Antihistaminics (Pheniramine) – Corticosteroids (Hydrocortisone) – Bronchodilator (Theophylline) No diagnostic tests for sulfa allergy -- just thorough history
  • 54.