Anterior knee pain is one of the most common conditions affecting active young patients. It has many potential causes including patellofemoral imbalance, lower limb structural abnormalities, and overuse. A thorough history focused on pain location and aggravating/relieving factors is important to identify the underlying cause. Physical examination evaluates alignment, patellar tracking, and identifies tenderness. Imaging like x-rays and MRI may help diagnose conditions like patellar tendinopathy, Osgood Schlatter disease, or cartilage lesions. Treatment is usually initially conservative with physical therapy and modifications, while surgery is considered for issues like instability or advanced arthritis. A holistic approach considering multiple factors is important for managing anterior knee pain.
Proximal fibular osteotomy - What is the evidence?Dr Saseendar MD
Proximal fibular osteotomy has been proposed as a simple and inexpensive alternative to high-tibial osteotomy and unicondylar knee arthroplasty and may be useful for low-income populations that cannot afford expensive treatment methods. However, there is no consensus existing regarding the mechanism by which it acts nor the outcome of this procedure. This study was performed to analyze the available evidence on the benefits of proximal fibular osteotomy and to understand the possible mechanisms in play. There are various mechanisms that are proposed to individually or collectively contribute to the outcomes of this procedure, and include the theory of non-uniform settlement, the too-many cortices theory, slippage phenomenon, the concept of competition of muscles, dynamic fibular distalization theory and ground reaction vector readjustment theory. The mechanisms have been discussed and future directions in research have been proposed. The current literature, which mostly consists of case series, suggests the usefulness of the procedure in decreasing varus deformity as well as improving symptoms in medial osteoarthritis. However, large randomised controlled trials with long-term follow-up are required to establish the benefits of this procedure over other established treatment methods.
Proximal Fibular Osteotomy for Knee Osteoarthritis - What is the evidence?Dr Saseendar MD
Proximal Fibular Osteotomy for Knee Osteoarthritis - What is the evidence?
knee osteoarthritis, knee surgery, total knee replacement, osteoarthritis, knee pain, elderly,
https://kneesurgrelatres.biomedcentral.com/articles/10.1186/s43019-019-0016-0
a painful knee can be classified into arthritic and non-arthatic. Many doctor forget non-arthic knee pain. This non-arthritic pain affect many pat.. younger more affected than old pat.,
Osteoarthritis of the knee, OA knee, Osteoarthritis, ankle, shoulder, spine, spondylosis, spondylitis
kellgren lawrence grading, ankle and foot osteoarthritis are explained, subtalar joint osteoarthritis, osteoarthritis of knne, management of osteoarthritis, osteoarthritis of spine, management of osteoarthritis, pathophysiology of osteoarthritis, primary and secondary osteoarthritis, figures and charting, knee scores, validity of knee scores
Proximal fibular osteotomy - What is the evidence?Dr Saseendar MD
Proximal fibular osteotomy has been proposed as a simple and inexpensive alternative to high-tibial osteotomy and unicondylar knee arthroplasty and may be useful for low-income populations that cannot afford expensive treatment methods. However, there is no consensus existing regarding the mechanism by which it acts nor the outcome of this procedure. This study was performed to analyze the available evidence on the benefits of proximal fibular osteotomy and to understand the possible mechanisms in play. There are various mechanisms that are proposed to individually or collectively contribute to the outcomes of this procedure, and include the theory of non-uniform settlement, the too-many cortices theory, slippage phenomenon, the concept of competition of muscles, dynamic fibular distalization theory and ground reaction vector readjustment theory. The mechanisms have been discussed and future directions in research have been proposed. The current literature, which mostly consists of case series, suggests the usefulness of the procedure in decreasing varus deformity as well as improving symptoms in medial osteoarthritis. However, large randomised controlled trials with long-term follow-up are required to establish the benefits of this procedure over other established treatment methods.
Proximal Fibular Osteotomy for Knee Osteoarthritis - What is the evidence?Dr Saseendar MD
Proximal Fibular Osteotomy for Knee Osteoarthritis - What is the evidence?
knee osteoarthritis, knee surgery, total knee replacement, osteoarthritis, knee pain, elderly,
https://kneesurgrelatres.biomedcentral.com/articles/10.1186/s43019-019-0016-0
a painful knee can be classified into arthritic and non-arthatic. Many doctor forget non-arthic knee pain. This non-arthritic pain affect many pat.. younger more affected than old pat.,
Osteoarthritis of the knee, OA knee, Osteoarthritis, ankle, shoulder, spine, spondylosis, spondylitis
kellgren lawrence grading, ankle and foot osteoarthritis are explained, subtalar joint osteoarthritis, osteoarthritis of knne, management of osteoarthritis, osteoarthritis of spine, management of osteoarthritis, pathophysiology of osteoarthritis, primary and secondary osteoarthritis, figures and charting, knee scores, validity of knee scores
In this presentation I’m going to inform you briefly about a novel arthroscopic technique for athletic pubalgia. You may have heard it as “sports hernia or groin injury………” but in fact is a groin pain syndrome, particularly common in sports that require athletes to perform repetitive kicking..
The role of Cement Augmentation in the Prevention of Spinal Insufficiency Fra...Winston Rennie
The Role of Cement Augmentation in the Prevention of Spinal Insufficiency Fractures. Spinal Vertebral fractures and percutaneous cement augmentation, vertebroplasty and kyphoplasty. The arguments for a role in preventing new spinal fractures and those against it. The flaws in experimental biomechanical studies and the importance of clinical spinal stability. Biplanar bipedicular percutaneous imaging approaches and formal trainig schemes to be established to train new practitioners with a biomechanically based cement placement.
ACL Injury Hacks covers the entire physiology, etiology,pathology, diagnosis, recent advancements in diagnosis of ACL and focus on how an early and accurate diagnosis can contribute to a better treatment and rehabilitation as well as early return to sport of an athlete.
Evaluation of Lumbar Spine Disease starts with understanding the clinical back grounds. It starts with good history and physical examination. This is a teaching lecture given twice by Prof. Dr. Mohamed Mohi Eldin, professor of neurosurgery, in the Basic Spine Course, Egyptian Medical Syndicate, Cairo, March 2009 and in 2010.
Foot and ankle trauma, common pitfalls, imaging modalities and radiographic occult fractures. The concept of the PITFL or "pitiful injury" an easily overlooked ligamentous injury of the talocrural joint
Adv. biopharm. APPLICATION OF PHARMACOKINETICS : TARGETED DRUG DELIVERY SYSTEMSAkankshaAshtankar
MIP 201T & MPH 202T
ADVANCED BIOPHARMACEUTICS & PHARMACOKINETICS : UNIT 5
APPLICATION OF PHARMACOKINETICS : TARGETED DRUG DELIVERY SYSTEMS By - AKANKSHA ASHTANKAR
In this presentation I’m going to inform you briefly about a novel arthroscopic technique for athletic pubalgia. You may have heard it as “sports hernia or groin injury………” but in fact is a groin pain syndrome, particularly common in sports that require athletes to perform repetitive kicking..
The role of Cement Augmentation in the Prevention of Spinal Insufficiency Fra...Winston Rennie
The Role of Cement Augmentation in the Prevention of Spinal Insufficiency Fractures. Spinal Vertebral fractures and percutaneous cement augmentation, vertebroplasty and kyphoplasty. The arguments for a role in preventing new spinal fractures and those against it. The flaws in experimental biomechanical studies and the importance of clinical spinal stability. Biplanar bipedicular percutaneous imaging approaches and formal trainig schemes to be established to train new practitioners with a biomechanically based cement placement.
ACL Injury Hacks covers the entire physiology, etiology,pathology, diagnosis, recent advancements in diagnosis of ACL and focus on how an early and accurate diagnosis can contribute to a better treatment and rehabilitation as well as early return to sport of an athlete.
Evaluation of Lumbar Spine Disease starts with understanding the clinical back grounds. It starts with good history and physical examination. This is a teaching lecture given twice by Prof. Dr. Mohamed Mohi Eldin, professor of neurosurgery, in the Basic Spine Course, Egyptian Medical Syndicate, Cairo, March 2009 and in 2010.
Foot and ankle trauma, common pitfalls, imaging modalities and radiographic occult fractures. The concept of the PITFL or "pitiful injury" an easily overlooked ligamentous injury of the talocrural joint
Adv. biopharm. APPLICATION OF PHARMACOKINETICS : TARGETED DRUG DELIVERY SYSTEMSAkankshaAshtankar
MIP 201T & MPH 202T
ADVANCED BIOPHARMACEUTICS & PHARMACOKINETICS : UNIT 5
APPLICATION OF PHARMACOKINETICS : TARGETED DRUG DELIVERY SYSTEMS By - AKANKSHA ASHTANKAR
Recomendações da OMS sobre cuidados maternos e neonatais para uma experiência pós-natal positiva.
Em consonância com os ODS – Objetivos do Desenvolvimento Sustentável e a Estratégia Global para a Saúde das Mulheres, Crianças e Adolescentes, e aplicando uma abordagem baseada nos direitos humanos, os esforços de cuidados pós-natais devem expandir-se para além da cobertura e da simples sobrevivência, de modo a incluir cuidados de qualidade.
Estas diretrizes visam melhorar a qualidade dos cuidados pós-natais essenciais e de rotina prestados às mulheres e aos recém-nascidos, com o objetivo final de melhorar a saúde e o bem-estar materno e neonatal.
Uma “experiência pós-natal positiva” é um resultado importante para todas as mulheres que dão à luz e para os seus recém-nascidos, estabelecendo as bases para a melhoria da saúde e do bem-estar a curto e longo prazo. Uma experiência pós-natal positiva é definida como aquela em que as mulheres, pessoas que gestam, os recém-nascidos, os casais, os pais, os cuidadores e as famílias recebem informação consistente, garantia e apoio de profissionais de saúde motivados; e onde um sistema de saúde flexível e com recursos reconheça as necessidades das mulheres e dos bebês e respeite o seu contexto cultural.
Estas diretrizes consolidadas apresentam algumas recomendações novas e já bem fundamentadas sobre cuidados pós-natais de rotina para mulheres e neonatos que recebem cuidados no pós-parto em unidades de saúde ou na comunidade, independentemente dos recursos disponíveis.
É fornecido um conjunto abrangente de recomendações para cuidados durante o período puerperal, com ênfase nos cuidados essenciais que todas as mulheres e recém-nascidos devem receber, e com a devida atenção à qualidade dos cuidados; isto é, a entrega e a experiência do cuidado recebido. Estas diretrizes atualizam e ampliam as recomendações da OMS de 2014 sobre cuidados pós-natais da mãe e do recém-nascido e complementam as atuais diretrizes da OMS sobre a gestão de complicações pós-natais.
O estabelecimento da amamentação e o manejo das principais intercorrências é contemplada.
Recomendamos muito.
Vamos discutir essas recomendações no nosso curso de pós-graduação em Aleitamento no Instituto Ciclos.
Esta publicação só está disponível em inglês até o momento.
Prof. Marcus Renato de Carvalho
www.agostodourado.com
Tom Selleck Health: A Comprehensive Look at the Iconic Actor’s Wellness Journeygreendigital
Tom Selleck, an enduring figure in Hollywood. has captivated audiences for decades with his rugged charm, iconic moustache. and memorable roles in television and film. From his breakout role as Thomas Magnum in Magnum P.I. to his current portrayal of Frank Reagan in Blue Bloods. Selleck's career has spanned over 50 years. But beyond his professional achievements. fans have often been curious about Tom Selleck Health. especially as he has aged in the public eye.
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Introduction
Many have been interested in Tom Selleck health. not only because of his enduring presence on screen but also because of the challenges. and lifestyle choices he has faced and made over the years. This article delves into the various aspects of Tom Selleck health. exploring his fitness regimen, diet, mental health. and the challenges he has encountered as he ages. We'll look at how he maintains his well-being. the health issues he has faced, and his approach to ageing .
Early Life and Career
Childhood and Athletic Beginnings
Tom Selleck was born on January 29, 1945, in Detroit, Michigan, and grew up in Sherman Oaks, California. From an early age, he was involved in sports, particularly basketball. which played a significant role in his physical development. His athletic pursuits continued into college. where he attended the University of Southern California (USC) on a basketball scholarship. This early involvement in sports laid a strong foundation for his physical health and disciplined lifestyle.
Transition to Acting
Selleck's transition from an athlete to an actor came with its physical demands. His first significant role in "Magnum P.I." required him to perform various stunts and maintain a fit appearance. This role, which he played from 1980 to 1988. necessitated a rigorous fitness routine to meet the show's demands. setting the stage for his long-term commitment to health and wellness.
Fitness Regimen
Workout Routine
Tom Selleck health and fitness regimen has evolved. adapting to his changing roles and age. During his "Magnum, P.I." days. Selleck's workouts were intense and focused on building and maintaining muscle mass. His routine included weightlifting, cardiovascular exercises. and specific training for the stunts he performed on the show.
Selleck adjusted his fitness routine as he aged to suit his body's needs. Today, his workouts focus on maintaining flexibility, strength, and cardiovascular health. He incorporates low-impact exercises such as swimming, walking, and light weightlifting. This balanced approach helps him stay fit without putting undue strain on his joints and muscles.
Importance of Flexibility and Mobility
In recent years, Selleck has emphasized the importance of flexibility and mobility in his fitness regimen. Understanding the natural decline in muscle mass and joint flexibility with age. he includes stretching and yoga in his routine. These practices help prevent injuries, improve posture, and maintain mobilit
micro teaching on communication m.sc nursing.pdfAnurag Sharma
Microteaching is a unique model of practice teaching. It is a viable instrument for the. desired change in the teaching behavior or the behavior potential which, in specified types of real. classroom situations, tends to facilitate the achievement of specified types of objectives.
These lecture slides, by Dr Sidra Arshad, offer a quick overview of the physiological basis of a normal electrocardiogram.
Learning objectives:
1. Define an electrocardiogram (ECG) and electrocardiography
2. Describe how dipoles generated by the heart produce the waveforms of the ECG
3. Describe the components of a normal electrocardiogram of a typical bipolar lead (limb II)
4. Differentiate between intervals and segments
5. Enlist some common indications for obtaining an ECG
6. Describe the flow of current around the heart during the cardiac cycle
7. Discuss the placement and polarity of the leads of electrocardiograph
8. Describe the normal electrocardiograms recorded from the limb leads and explain the physiological basis of the different records that are obtained
9. Define mean electrical vector (axis) of the heart and give the normal range
10. Define the mean QRS vector
11. Describe the axes of leads (hexagonal reference system)
12. Comprehend the vectorial analysis of the normal ECG
13. Determine the mean electrical axis of the ventricular QRS and appreciate the mean axis deviation
14. Explain the concepts of current of injury, J point, and their significance
Study Resources:
1. Chapter 11, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 9, Human Physiology - From Cells to Systems, Lauralee Sherwood, 9th edition
3. Chapter 29, Ganong’s Review of Medical Physiology, 26th edition
4. Electrocardiogram, StatPearls - https://www.ncbi.nlm.nih.gov/books/NBK549803/
5. ECG in Medical Practice by ABM Abdullah, 4th edition
6. Chapter 3, Cardiology Explained, https://www.ncbi.nlm.nih.gov/books/NBK2214/
7. ECG Basics, http://www.nataliescasebook.com/tag/e-c-g-basics
Integrating Ayurveda into Parkinson’s Management: A Holistic ApproachAyurveda ForAll
Explore the benefits of combining Ayurveda with conventional Parkinson's treatments. Learn how a holistic approach can manage symptoms, enhance well-being, and balance body energies. Discover the steps to safely integrate Ayurvedic practices into your Parkinson’s care plan, including expert guidance on diet, herbal remedies, and lifestyle modifications.
Rasamanikya is a excellent preparation in the field of Rasashastra, it is used in various Kushtha Roga, Shwasa, Vicharchika, Bhagandara, Vatarakta, and Phiranga Roga. In this article Preparation& Comparative analytical profile for both Formulationon i.e Rasamanikya prepared by Kushmanda swarasa & Churnodhaka Shodita Haratala. The study aims to provide insights into the comparative efficacy and analytical aspects of these formulations for enhanced therapeutic outcomes.
Title: Sense of Taste
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the structure and function of taste buds.
Describe the relationship between the taste threshold and taste index of common substances.
Explain the chemical basis and signal transduction of taste perception for each type of primary taste sensation.
Recognize different abnormalities of taste perception and their causes.
Key Topics:
Significance of Taste Sensation:
Differentiation between pleasant and harmful food
Influence on behavior
Selection of food based on metabolic needs
Receptors of Taste:
Taste buds on the tongue
Influence of sense of smell, texture of food, and pain stimulation (e.g., by pepper)
Primary and Secondary Taste Sensations:
Primary taste sensations: Sweet, Sour, Salty, Bitter, Umami
Chemical basis and signal transduction mechanisms for each taste
Taste Threshold and Index:
Taste threshold values for Sweet (sucrose), Salty (NaCl), Sour (HCl), and Bitter (Quinine)
Taste index relationship: Inversely proportional to taste threshold
Taste Blindness:
Inability to taste certain substances, particularly thiourea compounds
Example: Phenylthiocarbamide
Structure and Function of Taste Buds:
Composition: Epithelial cells, Sustentacular/Supporting cells, Taste cells, Basal cells
Features: Taste pores, Taste hairs/microvilli, and Taste nerve fibers
Location of Taste Buds:
Found in papillae of the tongue (Fungiform, Circumvallate, Foliate)
Also present on the palate, tonsillar pillars, epiglottis, and proximal esophagus
Mechanism of Taste Stimulation:
Interaction of taste substances with receptors on microvilli
Signal transduction pathways for Umami, Sweet, Bitter, Sour, and Salty tastes
Taste Sensitivity and Adaptation:
Decrease in sensitivity with age
Rapid adaptation of taste sensation
Role of Saliva in Taste:
Dissolution of tastants to reach receptors
Washing away the stimulus
Taste Preferences and Aversions:
Mechanisms behind taste preference and aversion
Influence of receptors and neural pathways
Impact of Sensory Nerve Damage:
Degeneration of taste buds if the sensory nerve fiber is cut
Abnormalities of Taste Detection:
Conditions: Ageusia, Hypogeusia, Dysgeusia (parageusia)
Causes: Nerve damage, neurological disorders, infections, poor oral hygiene, adverse drug effects, deficiencies, aging, tobacco use, altered neurotransmitter levels
Neurotransmitters and Taste Threshold:
Effects of serotonin (5-HT) and norepinephrine (NE) on taste sensitivity
Supertasters:
25% of the population with heightened sensitivity to taste, especially bitterness
Increased number of fungiform papillae
Title: Sense of Smell
Presenter: Dr. Faiza, Assistant Professor of Physiology
Qualifications:
MBBS (Best Graduate, AIMC Lahore)
FCPS Physiology
ICMT, CHPE, DHPE (STMU)
MPH (GC University, Faisalabad)
MBA (Virtual University of Pakistan)
Learning Objectives:
Describe the primary categories of smells and the concept of odor blindness.
Explain the structure and location of the olfactory membrane and mucosa, including the types and roles of cells involved in olfaction.
Describe the pathway and mechanisms of olfactory signal transmission from the olfactory receptors to the brain.
Illustrate the biochemical cascade triggered by odorant binding to olfactory receptors, including the role of G-proteins and second messengers in generating an action potential.
Identify different types of olfactory disorders such as anosmia, hyposmia, hyperosmia, and dysosmia, including their potential causes.
Key Topics:
Olfactory Genes:
3% of the human genome accounts for olfactory genes.
400 genes for odorant receptors.
Olfactory Membrane:
Located in the superior part of the nasal cavity.
Medially: Folds downward along the superior septum.
Laterally: Folds over the superior turbinate and upper surface of the middle turbinate.
Total surface area: 5-10 square centimeters.
Olfactory Mucosa:
Olfactory Cells: Bipolar nerve cells derived from the CNS (100 million), with 4-25 olfactory cilia per cell.
Sustentacular Cells: Produce mucus and maintain ionic and molecular environment.
Basal Cells: Replace worn-out olfactory cells with an average lifespan of 1-2 months.
Bowman’s Gland: Secretes mucus.
Stimulation of Olfactory Cells:
Odorant dissolves in mucus and attaches to receptors on olfactory cilia.
Involves a cascade effect through G-proteins and second messengers, leading to depolarization and action potential generation in the olfactory nerve.
Quality of a Good Odorant:
Small (3-20 Carbon atoms), volatile, water-soluble, and lipid-soluble.
Facilitated by odorant-binding proteins in mucus.
Membrane Potential and Action Potential:
Resting membrane potential: -55mV.
Action potential frequency in the olfactory nerve increases with odorant strength.
Adaptation Towards the Sense of Smell:
Rapid adaptation within the first second, with further slow adaptation.
Psychological adaptation greater than receptor adaptation, involving feedback inhibition from the central nervous system.
Primary Sensations of Smell:
Camphoraceous, Musky, Floral, Pepperminty, Ethereal, Pungent, Putrid.
Odor Detection Threshold:
Examples: Hydrogen sulfide (0.0005 ppm), Methyl-mercaptan (0.002 ppm).
Some toxic substances are odorless at lethal concentrations.
Characteristics of Smell:
Odor blindness for single substances due to lack of appropriate receptor protein.
Behavioral and emotional influences of smell.
Transmission of Olfactory Signals:
From olfactory cells to glomeruli in the olfactory bulb, involving lateral inhibition.
Primitive, less old, and new olfactory systems with different path
These simplified slides by Dr. Sidra Arshad present an overview of the non-respiratory functions of the respiratory tract.
Learning objectives:
1. Enlist the non-respiratory functions of the respiratory tract
2. Briefly explain how these functions are carried out
3. Discuss the significance of dead space
4. Differentiate between minute ventilation and alveolar ventilation
5. Describe the cough and sneeze reflexes
Study Resources:
1. Chapter 39, Guyton and Hall Textbook of Medical Physiology, 14th edition
2. Chapter 34, Ganong’s Review of Medical Physiology, 26th edition
3. Chapter 17, Human Physiology by Lauralee Sherwood, 9th edition
4. Non-respiratory functions of the lungs https://academic.oup.com/bjaed/article/13/3/98/278874
NVBDCP.pptx Nation vector borne disease control programSapna Thakur
NVBDCP was launched in 2003-2004 . Vector-Borne Disease: Disease that results from an infection transmitted to humans and other animals by blood-feeding arthropods, such as mosquitoes, ticks, and fleas. Examples of vector-borne diseases include Dengue fever, West Nile Virus, Lyme disease, and malaria.
NVBDCP.pptx Nation vector borne disease control program
Approach to anterior knee pain.pptx
1. Approach to anterior knee
pain
Dr. Nishchal Rijal
Fellow, Arthroscopy and Sports injuries
AKB Education Foundation
2. Introduction
• one of the most common conditions to
bring active young patients to a sports
injury clinic
• Incidence variable – 22.7 % in general
population
• F>M
• Challenging to treat
• 60-70% recurrent or chronic pain
Smith et al. Incidence and prevalence of patellofemoral pain:
a systematic review and meta-analysis. PLoS ONE. 2018;13: e0190892
3. Anterior knee pain
Instability Chondropathy OA
Osseous/ cartilage overload
Retinacular overload
PF imbalance
Lower limb structural abnormalities Lower limb lack of dynamic control
Mechanical stimulation of patellar/ trochlear intraosseous nerves
Increment intraosseous pressure Increment subchondral bone stress
Loss of vascular homeostasis
Focal supraphysiological loading of anatomical normal
knee components and soft tissue homeostasis
Ischemia Overuse
Decrease envelope of function
Osseous hypertension
Sanchis-Alfonso V. Holistic approach to understanding anterior knee pain. Clinical implications. Knee Surgery, Sports Traumatology, Arthroscopy. 2014 Oct;22(10):2275-85.
4. History - Pain
• Insidious onset, gradually
progressive
• Hx of trauma is uncommon
• two patterns: retro-patellar or
peripatellar
• Localization of pain
• tibial tuberosity - OSD
• inferior pole of patella - SLJ
D’Ambrosi R, Meena A, Raj A, Ursino N, Hewett TE. Anterior knee pain: state of the art. Sports Medicine-Open. 2022 Dec;8(1):98.
5. History - Pain
• Aggravated on
• climbing up or down stairs
• squatting and kneeling
• prolonged flexion of the knee joint
• Movie theater sign
• May be a/w hyperalgesia,
allodynia and other psychological
factors
Sanchis-Alfonso V, McConnell J, Monllau JC, Fulkerson JP. Diagnosis and treatment of anterior knee pain. Journal of ISAKOS.
2016 May 1;1(3):161-73.
6. History
• Swelling
• Instability – giving way
• functional impairment
• Sometimes, crepitus and limitation of extension may be present
Sanchis-Alfonso V, McConnell J, Monllau JC, Fulkerson JP. Diagnosis and treatment of anterior knee pain. Journal of ISAKOS.
2016 May 1;1(3):161-73.
7. History
• Aggravation of symptoms on
• overuse of knee
• new activity
• increased performance of an accustomed activity
• Psychiatric issues
• depression and catastrophization
• kinesiophobia
• any previous surgery on the symptomatic knee
Sanchis-Alfonso V, McConnell J, Monllau JC, Fulkerson JP. Diagnosis and treatment of anterior knee pain. Journal of ISAKOS.
2016 May 1;1(3):161-73.
8. Clinical examination
• Evaluation of lower limb alignment
• genu valgum
• excessive femoral anteversion
• tibial external rotation
Neal BS, Lack SD, Lankhorst NE, Raye A, Morrissey D, van Middelkoop M. Risk factors for patellofemoral pain: a systematic review
and metaanalysis. Br J Sports Med. 2019;53:270–81
Genu Valgum
Excessive
femoral
anteversion
External tibial
torsion
9. Q angle
• Represents vector of quadriceps
pull
• Normal
• 8-10 degrees in male
• 10-20 degrees in females
• Increased Q angle – increased risk
of PF instability
D’Ambrosi R, Meena A, Raj A, Ursino N, Hewett TE. Anterior knee pain: state of the art. Sports Medicine-Open. 2022 Dec;8(1):98.
10. Clinical examination
• Localization of pain
• Patellar tendinopathy (Jumper’s knee)
• Tenderness of the medial-distal patellar tip
• Hoffa impingement syndrome
• Tenderness of the lateral–distal patellar tip
• Osgood Schlatter disease
• Tenderness in tibial tuberosity
D’Ambrosi R, Meena A, Raj A, Ursino N, Hewett TE. Anterior knee pain: state of the art. Sports Medicine-Open. 2022 Dec;8(1):98.
11. Hoffa’s test
• To test impingement of
Hoffa’s fat pad
• Frequently a/w Patella
alta
Dragoo JL, Johnson C, McConnell J. Evaluation and treatment of disorders of the infrapatellar fat pad. Sports Med 2012;42:51–67.
12. • + Patellar facet
tenderness
• To evaluate PF
chondropathy
Clark’s test/ Patellar grind test
D’Ambrosi R, Meena A, Raj A, Ursino N, Hewett TE. Anterior knee pain: state of the art. Sports Medicine-Open. 2022 Dec;8(1):98.
13. Tests for PF instability
• Patellar glide test
• Patellar tilt test
• Patellar apprehension test and J sign
• Tenderness at insertion MPFL
• Beighton score for GLL
14. Clinical examination
• Restricted ROM of knee
• Hip examination- Hip pain radiating to anterior knee
• Muscle weakness
• Hip abduction, extension and external rotation
• core muscles
Sanchis-Alfonso V, McConnell J, Monllau JC, Fulkerson JP. Diagnosis and treatment of anterior knee pain. Journal of ISAKOS.
2016 May 1;1(3):161-73.
15. Investigation
• X ray
• 1st line investigation
• Weight bearing AP, True lateral and Axial views
• CT
• Bone morphology, TT-TG, torsional anomalies of lower limb
• MRI
• Cartilage lesion, plica, soft tissue impingement
16. MRI – Patellar tendinitis
• focal thickening of the proximal one-
third of the tendon
• AP diameter greater than 7 mm
• focal T2 hyperintensity within the
proximal tendon (eps medial third)
• an indistinct posterior tendon border
• edema in the adjacent Hoffa’s fat pad
Samim M, Smitaman E, Lawrence D, Moukaddam H. MRI of anterior knee pain. Skeletal Radiol. 2014;43:875–93.
17. MRI - OSD
• enlarged patellar tendon with T1 and
T2 hyperintensity at its insertion on
TT
• Edema of the deep infrapatellar bursa
with surrounding soft tissue edema
• marrow edema in TT
Samim M, Smitaman E, Lawrence D, Moukaddam H. MRI of anterior knee pain. Skeletal Radiol. 2014;43:875–93.
18. MRI - SLJ
• Bony avulsion injury at proximal
patellar tendon insertion
• No injury to cartilage
• Patellar sleeve avulsion
• Avulsion of inferior pole cartilage
Samim M, Smitaman E, Lawrence D, Moukaddam H. MRI of anterior knee pain. Skeletal Radiol. 2014;43:875–93.
19. MRI – medial plica
• Hyperintense on T2 sequence
when symptomatic
MRI – Hoffa’s disease
• T2 hyperintensity at inferolateral
aspect of the patellofemoral joint
and the lateral portion of the
Hoffa’s fat pad
Samim M, Smitaman E, Lawrence D, Moukaddam H. MRI of anterior knee pain. Skeletal Radiol. 2014;43:875–93.
20. Treatment
• Usually conservative, according
to cause
• Multimodal physio
• Operative indications
• PF instability
• PF cartilage injury
• Symptomatic plica
• Advanced PF arthritis
Adapted from Dye SF. The pathophysiology of patellofemoral pain:
a tissue homeostasis perspective. Clin Orthop Relat Res
2005;436:100–10.
21. Anterior knee pain
Classical peripatellar or
retropatellar pain
History
1
Pain not localized to a specific
landmark
3
Pain localized to a specific landmark
2
Adapted from D’Ambrosi R, Meena A, Raj A, Ursino N, Hewett TE. Anterior knee pain: state of the art. Sports Medicine-Open. 2022
Dec;8(1):98.
22. Classical peripatellar or
retropatellar pain
1
Giving way while climbing or
descending stairs or uneven
surfaces
Giving way while turning or
pivoting
Consider PF instability
Consider ACL insufficiency
Abnormal limb alignment
Trochlea dysplasia
Soft tissue problem (Patella
alta, MPFL insufficiency)
PF chondral lesion Surgery if conservative fails
Treat accordingly
‘a la carte’
Adapted from D’Ambrosi R, Meena A, Raj A, Ursino N, Hewett TE. Anterior knee pain: state of the art. Sports Medicine-Open. 2022
Dec;8(1):98.
23. Pain localized to a specific landmark
2
Tenderness localized to a specific landmark
Confirmation by X ray and/or MRI
Plica syndrome OSD or SLJ
Patellar tendinopathy
Arthroscopic
resection
Rest, activity
modification and
physical therapy
Taping and physical
therapy
Rest, activity
modification and
physical therapy
Adapted from D’Ambrosi R, Meena A, Raj A, Ursino N, Hewett TE. Anterior knee pain: state of the art. Sports Medicine-Open. 2022
Dec;8(1):98.
Fat pad syndrome
24. Pain not localized to a specific
landmark
3
Strengthening exercises and
physical therapy
Assess hip abduction, extension,
external rotation and core strength
Adapted from D’Ambrosi R, Meena A, Raj A, Ursino N, Hewett TE. Anterior knee pain: state of the art. Sports Medicine-Open. 2022
Dec;8(1):98.
25. Take home message
• Anterior knee pain has diverse etiology
• Meticulous history is crucial for management
• Diagnosis is essentially clinical with focus on identifying cause
• Investigations aim to support clinical findings
• Treatment is mainly nonoperative with surgery for selective
indications
• Patient education and counselling are crucial to success of treatment
Dey et al. reported the annual prevalence of patellofemoral pain (PFP) in the general population as 22.7%
Patellofemoral imbalance arising from a disorder of the retinacular structures;
loss of vascular homoeostasis secondary to patellofemoral imbalance or direct trauma
overuse (Dye’s theory of envelope of load acceptance)
Thorough and meticulous history-taking is the foundation of an accurate diagnosis
A history of trauma is uncommon, except in cases which may present post-traumatic patellar instability where we need to investigate for recurrent patellar instability
Pain usually occurs in response to activities that burden the patellofemoral joint, such as climbing up or down stairs, squatting, kneeling, and prolonged flexion of the knee joint
The so-called movie theater sign is observed when the patient experiences knee pain upon sitting with their knees flexed for a continuous period, such as while watching a movie in the theater or long car drives. The pain may improve on knee extension. This points to a pathology of the extensor mechanism and not the ftibiofemoral joint joint.
Swelling -Post-traumatic cases will offer a history of painful knee swelling after the antecedent trauma. Other patients may provide a history of knee swelling that was resolved after a period of avoiding pain-provoking activity. There may be multiple episodes of knee joint swelling.
Instability - sensation of their knee joint “giving way” while walking, which is related to reflex inhibition and/or atrophy of quadriceps. The patient is apprehensive about walking up or down stairs or on uneven surfaces
Commonly, the appearance or worsening of the symptoms is related to the overuse of the knee
The pain might also be brought on by a new activity that the patient is not used to performing or increased performance of an accustomed activity
patients with more severe symptoms have been found to suffer from depression and catastrophization
May be apprehensive about clinical examination maneuvers, that is, kinesiophobia (the fear that a manoeuvre will cause more injury or a reinjury and pain)
catastrophising (the belief that pain will worsen and cannot be relieved)
A genu valgum, excessive femoral anteversion, and consecutive tibial external rotation are independent risk factors for patellar instability
Craig’s test - Performed with the patient prone with knees flexed to 90°. The examiner palpates the greater trochanter and rotates the hip internally and externally until the greater trochanter lies at the lateral-most aspect of the hip (parallel to the examination table or bed), thereby projecting the femoral head into the center of the acetabulum. Interpretation: (1) Normal: At birth, the mean anteversion angle is 30°; it decreases to 8–15° in adults (angle of internal rotation). (2) Angle > 15°: Increased anteversion leads to squinting patellae and pigeon-toed walking (in-toeing), which is twice as common in girls. (3) Angle < 8°: Retroversion
Q-angle (quadriceps angle) is the angle between the quadriceps tendon and the patellar tendon. It provides useful information about the knee joint’s alignment. The Q-angle is formed
in the frontal plane by two line segments—one drawn from the anterior superior iliac spine (ASIS) to the center of the patella, and the other drawn from the center of the patella to the tibial tubercle. An increased Q-angle is a risk factor for patellar subluxation
The next step is to localize the painful area to identify the injured or pathological structure, followed by palpation of the important patellofemoral and tibiofemoral landmarks.
In patients with impingement of the Hoffa fat pad, pain is dramatically exacerbated by quadriceps contraction (B) or passive knee extension (C), while applying pressure of the fat pad with the fingers (A,B,C), because this movement causes a small posterior tilt of the inferior pole of the patella, which impinges on an inflamed and sensitised infrapatellar fat pad.
Restricted range of motion of the knee due to any postoperative stiffness tends to increase the patellofemoral contact pressure and cause AKP
Hip abduction, hip extension, and hip external rotation weakness can be associated with AKP, as can core muscle weakness
These patients with AKP tend to activate their quadriceps less, leading to a quadriceps avoidance gait pattern
The diagnosis of AKP is essentially a clinical one. Imaging plays a role in complementing the clinical examination by confirming the diagnosis and estimates the pathology quantitatively and qualitatively, ruling out others.
When no significant abnormality is detected, or the patient’s symptoms are refractory, more detailed studies of the knee such as computed tomography (CT) and magnetic resonance imaging (MRI) are obtained
On MRI, the normal plica has low signal on both T1- and T2-weighted images and is easily identified with some degree of joint distention
The clinician needs to decrease the strain of excessively loaded and painful soft tissues around the PFJ, improving the seating of the patella in the trochlea, as well as to optimise the lower limb mechanics, which should decrease the patient’s symptoms and, if maintained, will minimise any recurrences of symptoms.
To maintain the envelope of function and remain symptom free the patient must keep the intensity and frequency of the load below the threshold.