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ANTEPARTUM
HAEMORRHAGE
BY
COL O NDOLA
2
Definition of APH
 This is defined as bleeding from the
genital tract between the 28th week of
pregnancy and the onset of labour.
3
Causes of APH
 Placenta praevia
 Placental abruption
 Local causes
 Undetermined origin
 Vasa praevia
4
Placenta Praevia
 A placenta that is implanted entirely or
in part, in the lower uterine segment.
5
Grades of Placenta
Praevia
 Type I- placenta encroaches on the
lower uterine segment but does not
reach the internal cervical os.
 Type II- placenta reaches the edge of
the cervix, but does not cover it.
 Type III-placenta covers the cervix but
not at full cervical dilatation.
6
Grades of Placenta
Praevia
 Type IV- placenta is symmetrically
implanted in the lower uterine
segment and covers internal os at full
dilatation
 Types I & II are minor praevias - as
the lower segment develops, may
become normally situated.
 Types III & IV are major praevias
7
Placenta Praevia
 Causes are frequently unclear and the
low site of implantation may merely
represent an accident of nature
 Associations include: older multiparous
women, multiple pregnancy and
previous uterine damage such as C/S
8
Clinical presentation of
Placenta Praevia
 Two classical presentation are of APH
or as fetal malpresentation in late
pregnancy. Can be asymptomatic and
routinely picked up on U/S.
 Recurrent painless bleeding is the
typical history
 Patient is usually stable unless there
has been a major bleed
9
Clinical presentation of
Placenta Praevia
 The uterus is soft and non tender
 High head on presentation or
malpresentation- breech, transverse or
oblique lie
 FHHR
10
Diagnosis of Placenta
Praevia
 History
 Clinical examination
 U/S
 EIT-examination in theatre -full
preparation for C/S
11
Management of Placenta
Praevia
 Depends on stage of pregnancy and
extent of haemorrhage
 Minor praevia with minimal or no
bleeding -conservative management.
Aim to deliver at 38 weeks gestation.
Mode of delivery will depend on EIT
findings. If minor, then vaginal
delivery can be attempted unless it’s a
posterior placenta praevia.
12
Management of Placenta
Praevia
 Major placenta praevia, if
asymptomatic or minimal bleeding -
then aim to deliver at 38 weeks
gestation. EIT? Delivery is by C/S.
 Any type of praevia associated with
severe haemorrhage should be
delivered by the quickest mode - C/S-
regardless of gestation age.
13
Placental Abruption
 The premature separation of a
normally situated placenta
 The basic cause is unknown
 Is a self extending process with the
accumulating blood clot causing more
separation and thus more
haemorrhage, until the edge of the
placenta is reached
14
Placenta Abruption cont’d
 Blood then escapes through the
potential space between the chorion
and decidua until it reaches the cervix
 Blood can also reach the amniotic
cavity (by disrupting the placenta,
producing blood- stained liquor) and
the myometrium (causing a couvelaire
uterus)
15
Complications of
Abruption
 Fetal hypoxia or death - because of
the extent of placental separation
 Haemorrhagic shock
 Renal damage - acute tubular or
cortical necrosis
 DIC
 PPH
16
Associations of Abruption
 Hypertension-cause or consequence?
 Sick placenta-excessive fetomaternal
transfer of AFP in mid pregnancy &
diminished ‘adhesiveness”
 Previous history of abruption-risk of
recurrence ten fold
 Trauma-RTA, assault, ECV,
cordocentesis
17
Associations of Abruption
cont’d
 Fibriods - where site of placental
attachment covers a fibroid, increased
risk of abruption
 PROM esp with sudden decompression
as with polyhydramnios
 Multiple pregnancy - cause is unclear
18
Clinical presentation of
Abruption
 Bleeding may be concealed or
revealed
 Pain over the uterus with no
associated periodicity
 Uterus is extremely hard and tender &
it does not relax. The HOF is large for
dates in concealed haemorrhage
19
Clinical presentation of
Abruption cont’d
 Fetal parts are difficult to palpate and
the FH may be inaudible
 Faintness & collapse may occur, as
may signs of shock
20
Diagnosis of Abruption
 History
 Examination & demonstration after
delivery, of a retroplacental clot
indenting the placental surface
 U/S - has a minimal role in placental
abruption
21
Management of Abruption
cont’d
Principles of management are:
« early delivery
« adequate blood transfusion
« adequate analgesia
« detailed monitoring of maternal & fetal
condition
22
Management of Abruption
cont’d
 Early delivery is vital & if the baby is
alive and the gestation age favors
extra uterine survival, delivery should
be by C/S even if the fetus is not
hypoxic.
 If the fetus is dead, vaginal delivery is
preferred unless there is severe
haemorrhage and mother’s life is at
stake.
23
Management of Abruption
cont’d
 X-match at least 4 units of whole
blood, FFP
 Clotting profile - bedside clotting
- fibrinogen levels
- platelets
 Active management of third stage
ACTIVE MANAGEMENT OF THE
THIRD STAGE OF LABOUR
This has three components:
1. Administration of Oxytocin – to
enhance uterine contractions
2. Clamping the cord early – usually
before, alongside, or immediately
after giving the Oxytocin
24
3. Controlled cord traction is applied
with counter-pressureon the uterus in
order to deliver the placenta
25

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  • 2. 2 Definition of APH  This is defined as bleeding from the genital tract between the 28th week of pregnancy and the onset of labour.
  • 3. 3 Causes of APH  Placenta praevia  Placental abruption  Local causes  Undetermined origin  Vasa praevia
  • 4. 4 Placenta Praevia  A placenta that is implanted entirely or in part, in the lower uterine segment.
  • 5. 5 Grades of Placenta Praevia  Type I- placenta encroaches on the lower uterine segment but does not reach the internal cervical os.  Type II- placenta reaches the edge of the cervix, but does not cover it.  Type III-placenta covers the cervix but not at full cervical dilatation.
  • 6. 6 Grades of Placenta Praevia  Type IV- placenta is symmetrically implanted in the lower uterine segment and covers internal os at full dilatation  Types I & II are minor praevias - as the lower segment develops, may become normally situated.  Types III & IV are major praevias
  • 7. 7 Placenta Praevia  Causes are frequently unclear and the low site of implantation may merely represent an accident of nature  Associations include: older multiparous women, multiple pregnancy and previous uterine damage such as C/S
  • 8. 8 Clinical presentation of Placenta Praevia  Two classical presentation are of APH or as fetal malpresentation in late pregnancy. Can be asymptomatic and routinely picked up on U/S.  Recurrent painless bleeding is the typical history  Patient is usually stable unless there has been a major bleed
  • 9. 9 Clinical presentation of Placenta Praevia  The uterus is soft and non tender  High head on presentation or malpresentation- breech, transverse or oblique lie  FHHR
  • 10. 10 Diagnosis of Placenta Praevia  History  Clinical examination  U/S  EIT-examination in theatre -full preparation for C/S
  • 11. 11 Management of Placenta Praevia  Depends on stage of pregnancy and extent of haemorrhage  Minor praevia with minimal or no bleeding -conservative management. Aim to deliver at 38 weeks gestation. Mode of delivery will depend on EIT findings. If minor, then vaginal delivery can be attempted unless it’s a posterior placenta praevia.
  • 12. 12 Management of Placenta Praevia  Major placenta praevia, if asymptomatic or minimal bleeding - then aim to deliver at 38 weeks gestation. EIT? Delivery is by C/S.  Any type of praevia associated with severe haemorrhage should be delivered by the quickest mode - C/S- regardless of gestation age.
  • 13. 13 Placental Abruption  The premature separation of a normally situated placenta  The basic cause is unknown  Is a self extending process with the accumulating blood clot causing more separation and thus more haemorrhage, until the edge of the placenta is reached
  • 14. 14 Placenta Abruption cont’d  Blood then escapes through the potential space between the chorion and decidua until it reaches the cervix  Blood can also reach the amniotic cavity (by disrupting the placenta, producing blood- stained liquor) and the myometrium (causing a couvelaire uterus)
  • 15. 15 Complications of Abruption  Fetal hypoxia or death - because of the extent of placental separation  Haemorrhagic shock  Renal damage - acute tubular or cortical necrosis  DIC  PPH
  • 16. 16 Associations of Abruption  Hypertension-cause or consequence?  Sick placenta-excessive fetomaternal transfer of AFP in mid pregnancy & diminished ‘adhesiveness”  Previous history of abruption-risk of recurrence ten fold  Trauma-RTA, assault, ECV, cordocentesis
  • 17. 17 Associations of Abruption cont’d  Fibriods - where site of placental attachment covers a fibroid, increased risk of abruption  PROM esp with sudden decompression as with polyhydramnios  Multiple pregnancy - cause is unclear
  • 18. 18 Clinical presentation of Abruption  Bleeding may be concealed or revealed  Pain over the uterus with no associated periodicity  Uterus is extremely hard and tender & it does not relax. The HOF is large for dates in concealed haemorrhage
  • 19. 19 Clinical presentation of Abruption cont’d  Fetal parts are difficult to palpate and the FH may be inaudible  Faintness & collapse may occur, as may signs of shock
  • 20. 20 Diagnosis of Abruption  History  Examination & demonstration after delivery, of a retroplacental clot indenting the placental surface  U/S - has a minimal role in placental abruption
  • 21. 21 Management of Abruption cont’d Principles of management are: « early delivery « adequate blood transfusion « adequate analgesia « detailed monitoring of maternal & fetal condition
  • 22. 22 Management of Abruption cont’d  Early delivery is vital & if the baby is alive and the gestation age favors extra uterine survival, delivery should be by C/S even if the fetus is not hypoxic.  If the fetus is dead, vaginal delivery is preferred unless there is severe haemorrhage and mother’s life is at stake.
  • 23. 23 Management of Abruption cont’d  X-match at least 4 units of whole blood, FFP  Clotting profile - bedside clotting - fibrinogen levels - platelets  Active management of third stage
  • 24. ACTIVE MANAGEMENT OF THE THIRD STAGE OF LABOUR This has three components: 1. Administration of Oxytocin – to enhance uterine contractions 2. Clamping the cord early – usually before, alongside, or immediately after giving the Oxytocin 24
  • 25. 3. Controlled cord traction is applied with counter-pressureon the uterus in order to deliver the placenta 25